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Relevant bibliographies by topics / Myofibrome

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Academic literature on the topic 'Myofibrome'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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Journal articles on the topic "Myofibrome"

1

Maradeix, S., and B. Cribier. "Myofibrome et myofibromatose." Annales de Dermatologie et de Vénéréologie 132, no. 3 (March 2005): 271–75. http://dx.doi.org/10.1016/s0151-9638(05)79263-2.

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Vitrey, F., J. P. Ory, D. Bourscheid, and C. Lebrun. "Polyglobulie et myofibrome utérin." La Revue de Médecine Interne 22 (June 2001): 125. http://dx.doi.org/10.1016/s0248-8663(01)83537-2.

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Ben Salem, A., S. Rammeh, S. Ben Taazayat, N. Znaidi, B. Fazaa, and R. Zermani. "Myofibrome solitaire de l’adulte." Annales de Dermatologie et de Vénéréologie 139, no. 11 (November 2012): 765–66. http://dx.doi.org/10.1016/j.annder.2012.04.190.

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Ben Haj Amor, M., E. Nectoux, D. Basraoui, M. Cagneaux, X. Leroy, B. Herbaux, F. Gabor, and N. Boutry. "Myofibrome calcifié solitaire de la jambe : à propos d’un cas." Journal de Radiologie 92, no. 3 (March 2011): 243–46. http://dx.doi.org/10.1016/j.jradio.2011.02.006.

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Huet, F., P. Plantin, C. Le Rouzic, L. Carausu, L. Misery, and C. Abasq-Thomas. "Myofibrome solitaire cervicofacial de découverte anténatale et de régression spontanée." Annales de Dermatologie et de Vénéréologie 143, no. 12 (December 2016): S306—S307. http://dx.doi.org/10.1016/j.annder.2016.09.461.

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Koo, Selene C., Katherine A. Janeway, Marian H. Harris, Christy J. Fryer, Jon C. Aster, Alyaa Al-Ibraheemi, and Alanna J. Church. "A Distinctive Genomic and Immunohistochemical Profile for NOTCH3 and PDGFRB in Myofibroma With Diagnostic and Therapeutic Implications." International Journal of Surgical Pathology 28, no. 2 (September 29, 2019): 128–37. http://dx.doi.org/10.1177/1066896919876703.

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Introduction. Myofibromas are rare tumors of pericytic lineage, typically affecting children, and are sometimes aggressive. A subset of sporadic and familial myofibromas have activating variants in PDGFRB. The relationship of myofibroma and PDGFRB to the NOTCH pathway has not yet been described. Methods. Ten myofibroma cases were sequenced with a targeted panel of 447 genes, including copy number variation and selected fusions. Immunohistochemical analysis of total NOTCH3 and activated NOTCH3 was assessed for all 10 myofibroma cases, and a series of histologic mimics (n = 20). Results. Alterations identified by next-generation sequencing included PDGFRB sequence variants in 8/10 cases (80%), a NOTCH3 variant in 1/10 cases (10%), and a NOTCH2 variant in 1/10 cases (10%). All 10 cases also showed a pattern of low-amplitude (1.5- to 2-fold) copy number alterations including gains in PDGFRB and NOTCH3. Ten of 10 myofibromas (100%) showed cytoplasmic staining for total NOTCH3 and 9 of 10 cases (90%) showed nuclear staining for activated NOTCH3. Within the control cohort of histologic mimics, 3 of 3 nodular fasciitis cases (100%) were positive for activated and total NOTCH3, and the remaining 17 cases were negative for pan NOTCH3, while 3 of 3 desmoid-type fibromatosis cases (100%) showed patchy weak nuclear staining for activated NOTCH3. Discussion. Our findings suggest a common pathway of PDGFRB/NOTCH3 activation in myofibromas, even in cases that lack PDGFRB sequence variants. These results support the pericytic lineage of myofibroma. Identification of the characteristic genomic alterations or immunohistochemical staining pattern may facilitate a difficult pathologic diagnosis, and support the use of targeted treatments.

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Capo, Joseph A., Dina Moubayed, Sami P. Moubayed, Juan C. Hernandez-Prera, Azita Khorsandi, Daniel Buchbinder, and Mark L. Urken. "Pediatric Myofibroma of the Palate with Ulceration and Bone Destruction." Case Reports in Otolaryngology 2016 (2016): 1–3. http://dx.doi.org/10.1155/2016/1432764.

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Myofibroma is a rare benign neoplasm occurring in the head and neck, arising primarily in infants and children. Frequently, myofibromas grow rapidly leading to suspicion of malignancy and the potential for overaggressive surgical excision. We aim to report a rare case of myofibroma with ulceration and bone destruction. A nine-year-old female presented with an ulcerated left hard palate mass. Open biopsy was performed with pathology suggestive of myofibroma. A left partial maxillectomy and reconstruction with a buccal advancement flap were performed. Final pathology confirmed the diagnosis of a benign myofibroma. Myofibroma is a rare benign tumor of the head and neck which must be considered in the differential diagnosis by the clinician and the pathologist in order to prevent inappropriate and/or overaggressive treatment.

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Savithri, Vindhya, Rakesh Suresh, Mahija Janardhanan, and Thara Aravind. "Oral myofibroma presenting as an aggressive gingival lesion." BMJ Case Reports 14, no. 5 (May 2021): e242700. http://dx.doi.org/10.1136/bcr-2021-242700.

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Myofibromas are benign neoplasms of myofibroblastic origin and rarely encountered in the oral cavity. Myofibroma may frequently grow rapidly leading to suspicion of malignancy. This may lead to a tendency for aggressive management. The histopathology of this tumour has similarity with other spindle cell tumours and often requires immunohistochemical staining for diagnosis. Here, we present a case of myofibroma in a 15-year-old female patient who reported with an aggressive gingival swelling and discuss the various histopathological differential diagnosis.

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Davies, Benjamin M., Daniel du Plessis, and Kanna K. Gnanalingham. "Myofibroma of the cervical spine presenting as brachialgia." Journal of Neurosurgery: Spine 21, no. 6 (December 2014): 916–18. http://dx.doi.org/10.3171/2014.8.spine131194.

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Myofibromas are rare, benign tumors of myofibroblasts. Their occurrence in adults, involving bone outside of the head and neck, is especially uncommon. The authors report the case of a 34-year-old woman who presented with left-sided brachialgia. Magnetic resonance imaging identified an expansile soft-tissue lesion of the C6–7 facet joint. En bloc resection via a left posterior midline approach was undertaken. Histopathological analysis confirmed the lesion to be a myofibroma. Brachialgia resolved following surgery and there is no evidence of recurrence at 20 months follow-up. Myofibroma is a rare cause of primary soft-tissue tumor of the spine. Surgical excision remains the mainstay of treatment.

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Narayen, Vaishali, Syed Afroz Ahmed, Charu Suri, and Shahela Tanveer. "Myofibroma of the Gingiva: A Rare Case Report and Literature Review." Case Reports in Dentistry 2015 (2015): 1–4. http://dx.doi.org/10.1155/2015/243894.

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Myofibromas are benign uncommon fibroblastic tumors of the soft tissue, bone, or internal organs affecting all ages. These lesions histopathologically may mimic many other soft tissue tumors of the oral cavity such as spindle cell tumors of neurogenic and smooth muscle cell origin, thus leading to misdiagnosis and mistreatment. This case report describes a rare benign tumor, which presented as a soft tissue swelling on posterior gingiva. Surgical excision of the lesion was carried out under local anaesthesia. Histopathologic and immunohistochemical examination confirmed the diagnosis of myofibroma. Myofibroma should be included in the clinical differential diagnosis of masses of the oral soft tissues; however immunohistochemical examination is essential to establish an accurate diagnosis.

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Dissertations / Theses on the topic "Myofibrome"

1

CASTEL, VINCENT. "Les analogues de la luteinizing hormone-releasing hormone en pathologie uterine." Angers, 1990. http://www.theses.fr/1990ANGE1046.

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Desguerre, Isabelle. "Subphénotypes de la maladie de Duchenne et caractérisation de la myofibrose dystrophique humaine et expérimentale." Phd thesis, Université Paris-Est, 2008. http://tel.archives-ouvertes.fr/tel-00462105.

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La dystrophie musculaire de Duchenne (DMD) est la maladie neuromusculaire la plus fréquente de l'enfant. Son évolution progressive inexorable conduit habituellement au décès dans la troisième décade. La DMD constitue cependant une affection hétérogène pour la sévérité de l'atteinte musculaire, cognitive et cardiaque, et cette hétérogénéité n'est pas totalement expliquée par la localisation des mutations dans le gène de la dystrophine. Ma thèse comporte trois volets: (1) une analyse clinique multivariée d'une cohorte de DMD suivie à long terme qui nous a permis de définir 4 phénotypes distincts de DMD; (2) une étude de corrélation clinico-pathologique qui a identifié la fibrose endomysiale précoce comme seul facteur histologique prédictif de sévérité motrice; (3) la mise au point d'un modèle murin original de myofibrose dystrophique chez la souris mdx déficiente en dystrophine. 1- Étude multiparamétrique clinique. La saisie par la même équipe des données fonctionnelles musculaires, cardiaques, respiratoires et cognitives de 75 patients atteints de DMD (tous génotypés et présentant une absence complète de dystrophine musculaire), suivis pendant >10 ans, a permis d'établir un modèle multiparamétrique satisfaisant à deux dimensions principales, cognitive et motrice, et de définir 4 clusters phénotypiques : (i) DMD cognitive et motrice congénitale (20%), (ii) DMD classique (28%), (iii) DMD motrice pure modérée (22%), (iv) DMD motrice pure sévère (30%). La corrélation génotypephénotype était restreinte à la seule atteinte cognitive. Des indicateurs pronostics précoce ont été identifiés et validés sur une 2ème série de 34 patients. 2- Étude histopathologique. Les variations de sévérité de l'atteinte musculaire n'étant pas expliquées par la génétique moléculaire, nous avons cherché à corréler les paramètres moteurs et la biopsie musculaire prélevée dans le quadriceps à un stade précoce (3-7 ans) chez 25 patients (analyse stéréologique des images numérisées pour les paramètres élémentaires: nécrose/régénération, fibres hypercontractées, adipocytes, fibrose endomysiale et périmysiale). Seule la fibrose endomysiale était associée à un pronostic moteur défavorable (p<0.002) attesté par l'âge de perte de marche, la force du quadriceps et le testing musculaire global à 10 ans. Cette fibrose endomysiale dissociait les capillaires des myofibres (écartement x 2.5), et s'accompagnait d'une augmentation sélective des macrophages CD206+ activés dans la voie alterne (M2) et d'une diminution relative des cellules satellites musculaires (p<0.0001). Ces données suggèrent un rôle clé de la fibrose endomysiale (et des macrophages M2 profibrosant) et dans la sévérité clinique de la DMD. 3- Étude expérimentale. Ces éléments rendent nécessaire la mise au point d'un modèle expérimental de myofibrose dystrophique, la souris mdx présentant peu de fibrose et un déficit moteur modéré et tardif. Nous avons mis au point une nouvelle méthode de lésion musculaire focale profibrosante du tibialis antérieur chez la souris mdx (piqûres multiples quotidiennes pendant 15 jours). Une fibrose endomysiale attestée par un fort immunomarquage du collagène I (à 8, 30, 60 et 90 jours) a été quantifiée et corrélée à la perte de la force musculaire dans la patte lésée (comparée au muscle contralatéral). Ces résultats légitiment et préparent les futures stratégies thérapeutiques "anti-fibrosantes" dans la DMD

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Aguirre, Sarah E. "Expression of SATB2 and PXDN in Benign Myofibroblastic Proliferations." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1563371581390823.

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Book chapters on the topic "Myofibrome"

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Mocellin, Simone. "Myofibroma." In Soft Tissue Tumors, 563–65. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-58710-9_177.

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Cappellesso, Rocco. "Myofibroma/Myofibromatosis." In Encyclopedia of Pathology, 1–3. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-28845-1_5431-1.

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Campanacci, Mario. "Infantile Myofibroma, Myofibromatosis." In Bone and Soft Tissue Tumors, 921–24. Vienna: Springer Vienna, 1999. http://dx.doi.org/10.1007/978-3-7091-3846-5_60.

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Foest, R., S. Weigel, R. Stenger, H. Wiersbitzky, G. Lorenz, and O. A. Festge. "Inflammatorisches Myofibrom des rechten Lungenoberlappens." In Zurück in die Zukunft, 412. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55611-1_232.

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Strocchi, Marina, Christoph M. Augustin, Matthias A. F. Gsell, Elias Karabelas, Aurel Neic, Karli Gillette, Caroline H. Roney, et al. "The Effect of Ventricular Myofibre Orientation on Atrial Dynamics." In Functional Imaging and Modeling of the Heart, 659–70. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-78710-3_63.

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Varela, Marta, Jichao Zhao, and Oleg V. Aslanidi. "Determination of Atrial Myofibre Orientation Using Structure Tensor Analysis for Biophysical Modelling." In Functional Imaging and Modeling of the Heart, 425–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-38899-6_50.

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"Myofibroma." In Diagnostic Pathology: Vascular, 6–56. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-37674-7.50052-0.

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"Myofibroma." In Encyclopedia of Cancer, 2993. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-46875-3_101589.

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"Myofibroma/Myofibromatosis." In Diagnostic Imaging: Musculoskeletal Non-Traumatic Disease, 473. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-39252-5.50115-3.

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"Myofibromas." In Encyclopedia of Cancer, 2441. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_3945.

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Conference papers on the topic "Myofibrome"

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Blein, E., A. Féki, L. Marcellin, CI Gros, and F. Bornert. "Myofibrome mandibulaire : présentation d’un cas et de revue de la littérature." In 64ème Congrès de la SFCO, edited by S. Boisramé, S. Cousty, J. C. Deschaumes, V. Descroix, L. Devoize, P. Lesclous, C. Mauprivez, and T. Fortin. Les Ulis, France: EDP Sciences, 2016. http://dx.doi.org/10.1051/sfco/20166402038.

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de Souza, Anderson, Mário Faria, Victor Barretto, and Camila de Sousa. "Intracranial Myopericytoma/Myofibroma (Haemangioperycitoma) in Pediatric Patient: an Exclusive Case Report with Differential Diagnosis in Meningoangiomatosis and DNET." In XXXII Congresso Brasileiro de Neurocirurgia. Thieme Revinter Publicações Ltda, 2018. http://dx.doi.org/10.1055/s-0038-1672757.

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Koo, Selene C., Calicchio Monica, Benjamin Ferland, Marian H. Harris, Jon C. Aster, Katherine A. Janeway, Alyaa Al-Ibraheemi, and Alanna J. Church. "Abstract A31: A distinctive genomic and immunohistochemical profile for NOTCH3 and PDGFRB in infantile myofibroma with diagnostic and therapeutic implications." In Abstracts: AACR Special Conference: Pediatric Cancer Research: From Basic Science to the Clinic; December 3-6, 2017; Atlanta, Georgia. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.pedca17-a31.

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Reports on the topic "Myofibrome"

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Strub, Marion. Diagnosis and Therapeutic Care of Infantile Myofibroma Of the Jaws: A Case Report and Literature Systematic Review. Science Repository, July 2019. http://dx.doi.org/10.31487/j.dobcr.2019.03.02.

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