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Books on the topic 'Myopathes'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 February 2022

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1

Delaporte, François. Histoire des myopathies. Bibliothèque scientifique Payot, 1998.

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2

The inflammatory myopathies. Humana Press, 2009.

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3

Kagen, Lawrence J., ed. The Inflammatory Myopathies. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-827-0.

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4

i15, Institut Garches Entretiens. Myopathies de Duchenne-Becker. Frison-Roche, 2002.

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5

1942-, Mendell Jerry R., and Miller Robert G. 1942-, eds. Evaluation and treatment of myopathies. F.A. Davis, 1995.

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6

Babu, S. Suresh. Myopathy ayurvedic perspective. Chaukhambha Orientalia, 2002.

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7

Askanas, Valerie, and W. King Engel. Muscle aging: Inclusion-body myositis and myopathies. Wiley-Blackwell, 2012.

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8

Parker, James N., and Philip M. Parker. The official parent's sourcebook on mitochondrial myopathies. Edited by Icon Group International Inc. Icon Health Publications, 2002.

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9

Askanas, Valerie, and W. King Engel, eds. Muscle Aging, Inclusion-Body Myositis and Myopathies. Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781444398311.

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10

Das, Sudip Kumar. Mechanisms of anticholnesterase-induced myopathy and its prevention. Aston University. Departmentof Pharmaceutical Sciences, 1989.

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11

comptes, France Cour des. Observations de la Cour des comptes sur le compte d'emploi pour 1993 des ressources collectées auprès du public par l'Association française contre les myopathies (AFM): Articles L. 111-8 et L. 135-2 du Code des juridictions financières. Direction des journaux officiels, 1996.

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12

Steinau, Hans-Ulrich. Major limb replantation and postischemia syndrome: Investigation of acute ischemia-induced myopathy and reperfusion injury. Springer-Verlag, 1987.

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13

(Editor), L. P. Rowland, and Salvatore DiMauro (Editor), eds. Myopathies. Elsevier, 1992.

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14

L, Mastaglia Frank, and Hilton-Jones David, eds. Myopathies. Elsevier, 2007.

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15

L, Mastaglia Frank, and Hilton-Jones David, eds. Myopathies. Elsevier, 2007.

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16

Myopathies. Elsevier, 2007.

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17

Myopathies. Elsevier, 2007. http://dx.doi.org/10.1016/s0072-9752(07)x8600-7.

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18

L, Mastaglia Frank, ed. Inflammatory myopathies. Baillière, 1993.

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19

Metabolic myopathies. W.B. Saunders, 2000.

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20

Lilleker, James B., and Mark E. Roberts. Metabolic myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0005.

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Metabolic myopathies are caused by defects in the metabolic processes of energy storage and utilization, and can present with exercise intolerance, fatigue, muscle pain, and weakness. Metabolic myopathies are rare and can be difficult to diagnose. However, the clinical presentation can be similar to, and thus mimic, both the idiopathic inflammatory myopathies and other genetic muscle disorders including the muscular dystrophies. Careful enquiry about the nature and timing of muscle pain, as well as identification of other clinical ‘red-flags’, can highlight the possibility of a metabolic myopa

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21

Lahouti, Arash H., and Lisa Christopher-Stine. Toxic myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0009.

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Toxic myopathy symptoms range from myalgia and muscle cramps to severe weakness, bearing similarities to a number of other muscle conditions. Thus, when evaluating patients with muscle symptoms, an iatrogenic muscle problem should always be considered, to be able to distinguish a toxic from any other myopathy early on, preventing further muscle damage and to potentially reverse muscle injury by withdrawal of the toxic agent. Various commonly prescribed medications, as well as illicit drugs, may cause muscle damage. These substances may cause muscle injury through direct myotoxic effects, or in

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22

Katirji, Bashar. Case 20. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0024.

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Inflammatory myopathies are a group of disorders characterized by necrotizing myopathies with inflammatory infiltrates. Dermatomyositis, polymyositis, and inclusion body myositis are the classical types although other overlapping disorders are now more commonly diagnosed, including necrotizing autoimmune myopathy and the anti-synthetase syndrome. This case presents a typical patient with polymyositis and outlines the clinical and pathological findings of the various inflammatory myopathies. It highlights the differential diagnosis as well as the differences and similarities among the autoimmun

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23

Kennett, Robin P., and Sidra Aurangzeb. Primary muscle diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0024.

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This chapter on primary muscle diseases explains how analysis of compound muscle action potential (CMAP) amplitude, abnormal spontaneous activity on needle electromyography (EMG), and motor unit action potentials (MUAP) characteristics may be used to give an indication of pathophysiological processes, and goes on to describe the combination and distribution of abnormalities that may be expected in the more commonly encountered myopathies. The conditions considered in detail are inflammatory myopathy (including myositis), critical illness myopathy, disorders with myotonia, inherited myopathy (i

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24

Heene, Rainer, and Adriann M. de Graaf. Experimental Myopathies and Muscular Dystrophy: Studies in the Formal Pathogenesis of the Myopathy of 2,4-Dichlorophenoxyacetate. Springer, 2011.

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25

Kagen, Lawrence J. The Inflammatory Myopathies. Humana, 2014.

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26

Myopathies: New Research. Nova Biomedical, 2012.

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27

Idiopathic inflammatory myopathies. Saunders, 2002.

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28

Limaye, Vidya Sadanand. Overview and epidemiology. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0001.

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The term idiopathic inflammatory myopathies (IIM) encompasses a heterogeneous group of muscle-dominant systemic autoimmune syndromes, including polymyositis (PM), dermatomyositis (DM), sporadic inclusion body myositis (sIBM), and immune-mediated necrotizing myopathy (IMNM). The reported incidence of IIM ranges from 5 to 10 × 10–6. Patients with PM, DM, and IMNM characteristically present with the insidious onset of symmetric proximal weakness, while in sIBM the weakness can be asymmetric, and involve the distal upper limbs and quadriceps. Dermatomyositis may also be accompanied by a range of c

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29

Jenkins, Liberty. Myopathy. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0235.

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30

Kagen, Lawrence J. Myositis & Myopathies: Raynauld's Phenomenon. Rapid Science Publishers, 1994.

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31

Myopathies in Clinical Practice. Informa Healthcare, 2003.

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32

Kaplan, Tamara, and Tracey Milligan. Myopathy (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190650261.003.0017.

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The video in this chapter discusses myopathy, including its symptoms (pain, stiffness, cramps, exertional fatigue, myoglobinuria, shortness of breath, rash), muscular dystrophy and dystrophinopathies (Becker with reduced dystrophin and Duchenne with no dystrophin), polymyositis, dermatomyositis, and inclusion body myositis.

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33

Mendell, Jerry R., Eric L. Logigan, John T., M.D. Kissel, and Robert C. Griggs. Evaluation and Treatment of Myopathies. 2nd ed. Oxford University Press, 2007.

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34

Ciafaloni, Emma, Patrick F. Chinnery, and Robert C. Griggs, eds. Evaluation and Treatment of Myopathies. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199873937.001.0001.

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35

Evaluation and Treatment of Myopathies. Oxford University Press, 2014.

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36

Gran, Jan Tore, ed. Idiopathic Inflammatory Myopathies - Recent Developments. InTech, 2011. http://dx.doi.org/10.5772/814.

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37

Valerie, Askanas, Serratrice Georges, and Engel W. King, eds. Inclusion-body myositis and myopathies. Cambridge University Press, 1997.

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38

Bell, Tanvir K. Musculoskeletal Complications of HIV. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0043.

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Vitamin D levels have been observed to be low in HIV-infected patients. If replacement of low vitamin D is warranted, supplementation is done with vitamin D2 or D3. HIV-infected patients may be at higher risk for osteopenia, osteoporosis, and fragility fractures. Tenofovir alafenamide has been shown to produce less bone loss compared to tenofovir disoproxil fumarate. Muscle disorders can be debilitating in HIV-infected patients. Myopathies can have a range of presentation from myalgias to rhabdomyolysis. HIV myopathy is a rare proximal muscle disorder that can occur in HIV-infected patients. A

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39

Metabolic Myopathies (Major Problems in Neurology). W.B. Saunders Company, 1995.

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40

Brady, Stefen, and David Hilton-Jones. Muscular dystrophies and other genetic myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0006.

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Muscular dystrophies are a genetically and phenotypically heterogeneous group of progressive muscle diseases. Modern molecular genetic techniques have made it possible to clarify the genetic mutations responsible for most muscular dystrophies. Despite advances in genetics, the importance of the clinical history and physical examination has increased rather than diminished. It is only through correctly identifying the clinical features that the appropriate diagnostic investigations will be performed. Although muscular dystrophies are typically slowly progressive disorders in which muscle atroph

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41

Mann, Heřman, and Jiří Vencovský. Treatment of the idiopathic inflammatory myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0017.

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The management of idiopathic inflammatory myopathies (IIM) poses a challenge due to disease heterogeneity, variable responses to treatment and limited data from controlled studies. High dose glucocorticoids, often combined with other immunosuppressive drugs, represent the most common pharmacotherapeutic approach. The importance of early commencement of supervised exercise is also stressed. In treatment resistant cases, the introduction of drug combinations is recommended, and intravenous immunoglobulins may also be effective. Currently available data do not support use of most biological agent

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42

Heyck, Hartwig, M. Walter, D. Tönnis, et al. Die progressiv-dystrophischen Myopathien. Springer, 2012.

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43

Askanas, Valerie, and W. King Engel. Muscle Aging, Inclusion-Body Myositis and Myopathies. Wiley & Sons, Incorporated, John, 2011.

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44

MD, Robert L. Wortmann. Myopathies, An Issue of Rheumatic Disease Clinics. Saunders, 2011.

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45

Garcia-Trejo, Dr Jose de Jesus. Recent Research Developments in Human Mitochondrial Myopathies. Research Signpost, 2002.

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46

Cardioskeletal Myopathies in Children and Young Adults. Elsevier, 2017. http://dx.doi.org/10.1016/c2013-0-09788-5.

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47

de Visser and Eleonora M.A. Aronica, Marianne. Histopathological features of the idiopathic inflammatory myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0013.

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In adult patients with presumed idipathic inflammatory myopathy (IIM) without a characteristic and diagnostic dermatomyositis rash, muscle biopsy is mandatory to confirm the IIM diagnosis and to exclude a myopathy which would not respond to glucocorticoids or other immunosuppressants, including inclusion body myositis. This chapter discusses when, where, and how to undertake muscle biopsies, when to repeat them, how to interpret their results, and how these relate to IIM subtypes and disease processes.

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48

Ramdass, Ranjit. Neurophysiology in the assessment of inflammatory myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0015.

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Clinical neurophysiology (electrodiagnosis) includes the assessment of peripheral nerves by electrical stimulation (nerve conduction studies, NCS) and needle examination of muscles (electromyography, EMG). Electrodiagnostic assessment is a functional extension of clinical examination into the laboratory. It plays an important role in the investigation of a patient suspected of having myositis, providing valuable information regarding peripheral nerve, neuromuscular junction and muscle functions, to better characterize clinical syndromes. NCS can establish the presence and quantify the severity

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49

Rider, Lisa G., and Frederick W. Miller. Outcome assessment in the idiopathic inflammatory myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0016.

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Due to their rarity, heterogeneity, and multispecialty nature, the myositis syndromes have limited data-driven consensus on appropriate outcome measures. Recently, two international, multispecialty consortia developed new tools and consensus on core set measures of myositis disease activity and damage, as well as response criteria that are now recommended for use as clinical trial endpoints but will also be useful in clinical practice. Magnetic resonance imaging, muscle ultrasound, selected laboratory tests, and immunological biomarkers—including cytokines, chemokines, lymphocyte flow cytometr

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50

Askanas, Valerie, and W. King Engel. Muscle Aging, Inclusion-Body Myositis and Myopathies. Wiley & Sons, Incorporated, John, 2011.

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