Relevant bibliographies by topics / Myos

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
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Academic literature on the topic 'Myos'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "Myos"

1

Jung, G., X. Wu, and J. A. Hammer. "Dictyostelium mutants lacking multiple classic myosin I isoforms reveal combinations of shared and distinct functions." Journal of Cell Biology 133, no. 2 (April 15, 1996): 305–23. http://dx.doi.org/10.1083/jcb.133.2.305.

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Dictyostelium cells that lack the myoB isoform were previously shown to exhibit reduced efficiencies of phagocytosis and chemotactic aggregation ("streaming") and to crawl at about half the speed of wild-type cells. Of the four other Dictyostelium myosin I isoforms identified to date, myoC and myoD are the most similar to myoB in terms of tail domain sequence. Furthermore, we show here that myoC, like myoB and myoD, is concentrated in actin-rich cortical regions like the leading edge of migrating cells. To look for evidence of functional overlap between these isoforms, we analyzed myoB, myoC, and myoD single mutants, myoB/myoD double mutants, and myoB/myoC/myoD triple mutants, which were created using a combination of gene targeting techniques and constitutive expression of antisense RNA. With regard to the speed of locomoting, aggregation-stage cells, of the three single mutants, only the myoB mutant was significantly slower. Moreover, double and triple mutants were only slightly slower than the myoB single mutant. Consistent with this, the protein level of myoB alone rises dramatically during early development, suggesting that a special demand is placed on this one isoform when cells become highly motile. We also found, however, that the absolute amount of myoB protein in aggregation-stage cells is much higher than that for myoC and myoD, suggesting that what appears to be a case of nonoverlapping function could be the result of large differences in the amounts of functionally overlapping isoforms. Streaming assays also suggest that myoC plays a significant role in some aspect of motility other than cell speed. With regard to phagocytosis, both myoB and myoC single mutants exhibited significant reductions in initial rate, suggesting that these two isoforms perform nonredundant roles in supporting the phagocytic process. In triple mutants these defects were not additive, however. Finally, because double and triple mutants exhibited significant and progressive decreases in doubling times, we also measured the kinetics of fluid phase endocytic flux (uptake, transit time, efflux). Not only do all three isoforms contribute to this process, but their contributions are synergistic. While these results, when taken together, refute the simple notion that these three "classic" myosin I isoforms perform exclusively identical functions, they do reveal that all three share in supporting at least one cellular process (endocytosis), and they identify several other processes (motility, streaming, and phagocytosis) that are supported to a significant extent by either individual isoforms or various combinations of them.
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Kaarakka, Terhi Elina, Tero Frondelius, Osmo Kaleva, Reijo Kouhia, Heikki Orelma, and Joona Vaara. "Jännitysväsymisen kontinuumimalli." Rakenteiden Mekaniikka 52, no. 4 (December 31, 2019): 236–43. http://dx.doi.org/10.23998/rm.76262.

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Artikkelissa tarkastellaan evoluutioyhtälöpohjaisen jännitysväsymismallin stokastista laajennusta. Esitetty malli on muodostettu yleisten kontinuumimekaniikan periaatteiden mukaisesti ja on siten luonnostaan moniakselinen ja käsittelee kaikki jännityskomponentit ekvivalentilla tavalla. Malli soveltuu myos mielivaltaiselle kuormitushistorialle. Esimerkkinä tarkastellaan yksinkertaista valkoisella kohinalla häirityn säännollisen kuormituksen aiheuttaman elinikäaennusteen jakaumaa.
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Whitcomb, Donald. "Quseir al-Qadim and the location of Myos Hormos." Topoi 6, no. 2 (1996): 747–72. http://dx.doi.org/10.3406/topoi.1996.1693.

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4

Peacock, D. P. S. "The site of Myos Hormos: a view from space." Journal of Roman Archaeology 6 (1993): 226–32. http://dx.doi.org/10.1017/s1047759400011557.

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5

Haarer, B. K., A. Petzold, S. H. Lillie, and S. S. Brown. "Identification of MYO4, a second class V myosin gene in yeast." Journal of Cell Science 107, no. 4 (April 1, 1994): 1055–64. http://dx.doi.org/10.1242/jcs.107.4.1055.

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We have isolated a fourth myosin gene (MYO4) in yeast (Saccharomyces cerevisiae). MYO4 encodes a approximately 170 kDa (1471 amino acid) class V myosin, using the classification devised by Cheney et al. (1993a; Cell Motil. Cytoskel. 24, 215–223); the motor domain is followed by a neck region containing six putative calmodulin-binding sites and a tail with a short potential ‘coiled-coil’ domain. A comparison with other myosins in GenBank reveals that Myo4 protein is most closely related to the yeast Myo2 protein, another class V myosin. Deletion of MYO4 produces no detectable phenotype, either alone or in conjunction with mutations in myo2 or other myosin genes, the actin gene, or secretory genes. However, overexpression of MYO4 or MYO2 results in several morphological abnormalities, including the formation of short strings of unseparated cells in diploid strains, or clusters of cells in haploid strains. Alterations of MYO4 or MYO2 indicate that neither the motor domains nor tails of these myosins are required to confer the overexpression phenotype, whereas the neck region may be required. Although this phenotype is similar to that seen upon MYO1 deletion, we provide evidence that the overexpression of Myo4p or Myo2p is not simply interfering with Myo1p function.
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Whitewright, Julian. "Roman Rigging Material from the Red Sea Port of Myos Hormos." International Journal of Nautical Archaeology 36, no. 2 (May 23, 2007): 282–92. http://dx.doi.org/10.1111/j.1095-9270.2007.00150.x.

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7

Temesvari, L. A., J. M. Bush, M. D. Peterson, K. D. Novak, M. A. Titus, and J. A. Cardelli. "Examination of the endosomal and lysosomal pathways in Dictyostelium discoideum myosin I mutants." Journal of Cell Science 109, no. 3 (March 1, 1996): 663–73. http://dx.doi.org/10.1242/jcs.109.3.663.

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The role of myosin Is in endosomal trafficking and the lysosomal system was investigated in a Dictyostelium discoideum myosin I double mutant myoB-/C-, that has been previously shown to exhibit defects in fluid-phase endocytosis during growth in suspension culture (Novak et al., 1995). Various properties of the endosomal pathway in the myoB-/C- double mutant as well as in the myoB- and myoC- single mutants, including intravesicular pH, and intracellular retention time and exocytosis of a fluid phase marker, were found to be indistinguishable from wild-type parental cells. The intimate connection between the contractile vacuole complex and the endocytic pathway in Dictyostelium, and the localization of a myosin I to the contractile vacuole in Acanthamoeba, led us to also examine the structure and function of this organelle in the three myosin I mutants. No alteration in contractile vacuole structure or function was observed in the myoB-, myoC- or myoB-/C- cell lines. The transport, processing, and localization of a lysosomal enzyme, alpha-mannosidase, were also unaltered in all three mutants. However, the myoB- and myoB-/C- cell lines, but not the myoC- cell line, were found to oversecrete the lysosomal enzymes alpha-mannosidase and acid phosphatase, during growth and starvation. None of the mutants oversecreted proteins following the constitutive secretory pathway. Two additional myosin I mutants, myoA- and myoA-/B-, were also found to oversecrete the lysosomally localized enzymes alpha-mannosidase and acid phosphatase. Taken together, these results suggest that these myosins do not play a role in the intracellular movement of vesicles, but that they may participate in controlling events that occur at the actin-rich cortical region of the cell. While no direct evidence has been found for the association of myosin Is with lysosomes, we predict that the integrity of the lysosomal system is tied to the fidelity of the actin cortex, and changes in cortical organization could influence lysosomal-related membrane events such as internalization or transit of vesicles to the cell surface.
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Peterson, M. D., K. D. Novak, M. C. Reedy, J. I. Ruman, and M. A. Titus. "Molecular genetic analysis of myoC, a Dictyostelium myosin I." Journal of Cell Science 108, no. 3 (March 1, 1995): 1093–103. http://dx.doi.org/10.1242/jcs.108.3.1093.

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The protozoan myosin Is are widely expressed actin-based motors, yet their in vivo roles remain poorly understood. Molecular genetic studies have been carried out to determine their in vivo function in the simple eukaryote Dictyostelium, an organism that contains a family of four myosin Is. Here we report the characterization of myoC, a gene that encodes a fifth member of this family. Analysis of the deduced amino acid sequence reveals that the myoC gene encodes a myosin that is homologous to the well-described Acanthamoeba myosin Is as well as to Dictyostelium myoB and -D. The expression pattern of the myoC mRNA is similar to that of myoB and myoD, with a peak of expression at times of maximal cell migration, around 6 hours development. Deletion of the myoB gene has been previously shown to result in mutant cells that are defective in pseudopod extension and phagocytosis. However, no obvious differences in cell growth, development, phagocytosis or motility were detected in cells in which the myoC gene had been disrupted by homologous recombination. F-actin localization and ultrastructural organization also appeared unperturbed in myoC- cells. This apparent ‘lack’ of phenotype in a myosin I single knockout cannot be simply explained by redundancy of function. Our results rather suggest that the present means of assessing myosin I function in vivo are insufficient to identify the unique roles of these actin-based motors.
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Novak, K. D., M. D. Peterson, M. C. Reedy, and M. A. Titus. "Dictyostelium myosin I double mutants exhibit conditional defects in pinocytosis." Journal of Cell Biology 131, no. 5 (December 1, 1995): 1205–21. http://dx.doi.org/10.1083/jcb.131.5.1205.

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The functional relationship between three Dictyostelium myosin Is, myoA, myoB, and myoC, has been examined through the creation of double mutants. Two double mutants, myoA-/B- and myoB-/C-, exhibit similar conditional defects in fluid-phase pinocytosis. Double mutants grown in suspension culture are significantly impaired in their ability to take in nutrients from the medium, whereas they are almost indistinguishable from wild-type and single mutant strains when grown on a surface. The double mutants are also found to internalize gp126, a 116-kD membrane protein, at a slower rate than either the wild-type or single mutant cells. Ultrastructural analysis reveals that both double mutants possess numerous small vesicles, in contrast to the wild-type or myosin I single mutants that exhibit several large, clear vacuoles. The alterations in fluid and membrane internalization in the suspension-grown double mutants, coupled with the altered vesicular profile, suggest that these cells may be compromised during the early stages of pinocytosis, a process that has been proposed to occur via actin-based cytoskeletal rearrangements. Scanning electron microscopy and rhodamine-phalloidin staining indicates that the myosin I double mutants appear to extend a larger number of actin-filled structures, such as filopodia and crowns, than wild-type cells. Rhodamine-phalloidin staining of the F-actin cytoskeleton of these suspension-grown cells also reveals that the double mutant cells are delayed in the rearrangement of cortical actin-rich structures upon adhesion to a substrate. We propose that myoA, myoB, and myoC play roles in controlling F-actin filled membrane projections that are required for pinosome internalization in suspension.
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Leppälä, Jarkko, Antero Olakivi, and Kari Mikko Vesala. "Palkkatyövoiman käyttö työnjohdon apuna puutarha- ja maatilayrityksissä." Suomen Maataloustieteellisen Seuran Tiedote, no. 28 (January 31, 2012): 1–7. http://dx.doi.org/10.33354/smst.75532.

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Palkkatyövoiman johtaminen on yksi nousevista haasteista kasvavien puutarha- ja maatilayritysten toiminnassa. Kasvavilla tiloilla käytetään yhä enemmän erityisesti osa-aikaista työvoimaa ja kausityövoimaa kesän sesonkiluonteisiin töihin. Tällöin työnjohdon haasteet oletettavasti kasvavat. Työn johtamiseen liittyy monia osatehtäviä, jotka yrittäjän on hallittava: työvoiman hankkiminen, työn organisointi, työntekijoiden työtyytyväisyys ja motivointi, hallinto ja palkanlaskenta seka itse työnjohto ja siihen liittyvät vuorovaikutustilanteet. Ei kuitenkaan ole tiedossa, ovatko palkkatyövoiman ohjaamisen ja työnjohdon käytännöt uudistuneet tilanjohtamisen muuttuvissa tilanteissa, kuten tilan ulkopuolisen tai ulkomaalaisen työvoiman määrän kasvaessa. Yrittäjälle itselleen palkkatyövoima on tarpeellinen resurssi tuotannon ja yritystoiminnan volyymin kasvattamisessa. Olennaista yrittäjän kannalta on myos estää yrittäjän oman tyomäärän liikakasvu. Tässä artikkelissa käsiteltiin palkkatyövoiman käyttöä työnjohdon apuna eli työnjohdollisten tehtävien delegoimista palkkatyöntekijöille. Tutkimuskysymyksenä oli, käyttävätko puutarha- ja maatilayrittäjät palkkatyövoimaa työnjohtotehtävissä, ja minkälaisena johtamiskeinona se tässä yhteydessä näyttäytyy.Työnjohtotehtäviä on mielekästä delegoida työntekijöille, kun yrityskokoa kasvatetaan. Tyypillisiä tällaisia yrityksiä ovat puutarhayritykset, joilla on paljon kausityövoimaa. Tutkimuksessa haastateltiin tutkimustapauksena itäsuomalaista puutarha- ja maatalousyritystä, jossa kausityövoiman määrä ylitti sata työntekijää. Tilalla oli myos ulkomaalaista työvoimaa. Tutkimustapauksen käytäntöja verrattiin MTT:n lähettämän kyselyn (N=228) tuloksiin. Kyselyn vastaajat olivat Maaseudun Työnantajaliittoon kuuluvia puutarha- ja maatilatyönantajia. Kyselyn perusteella työnjohtotehtävien delegoimista työntekijoille ennustivat työnantajavastaajan naissukupuoli ja nuori ikä, tilan suuri kausittaisen työvoiman määrä sekä puutarhaviljely tuotantosuuntana. Työnjohdon delegointia tekeville työnantajille delegointi ilmeni myös haasteellisena. Puutarha- ja maatilan työt ovat turvallisuusmielessä hyvin riskialtis ala. Kyselyn perusteella näyttää silti, että suurten työvoimamäärien ja osaamisen hallinnan avuksi puutarha- ja maatilayrityksissä tarvitaan uusia johtamisen käytäntöja, niihin paneutuvaa tutkimusta, koulutusta ja tiedotusta.
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Dissertations / Theses on the topic "Myos"

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Widén, Jonas. "Automatisk test för myoelektroder." Thesis, Linköping University, Department of Science and Technology, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-2338.

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Denna rapport avhandlar en tio veckors period på Otto Bock Scandinavia AB i Norrköping. Där analyserades en manuell testutrusning för funktionstest av myoelektroder, för att mäta muskelspänningar. Myoelektroderna används till att styra gripfunktionen hos handproteser, för patienter som förlorat en del av sin arm. Analysen ska resultera i att ge ett förslag på en automatiserad test av elektroderna. En stor del av rapporten består av studier kring hur testmetoderna fungerar och elektrodernas användning och funktion. Slutligen behandlas även ett förslag på en automatiserad test för elektroderna.


The present report concerns a ten weeks period at Otto Bock Scandinavia AB in Norrköping. An analyse of a manual test equipment for testing myoelectrodes, who is used to measure muscle potential in the arm. The myoelectrodes are used to control a grip function on hand prostheses, which is used by persons who has lost their lower arm. The analyse should result in a proposal of an automation of the manual test equipment for the electrodes.

A significant part of the report discusses the function of test methods and who the electrodes are used for and their function. Finally, discusses a proposal on an automated test for the electrodes.

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Faralli, Hervé. "Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris." Thesis, Aix-Marseille 2, 2010. http://www.theses.fr/2010AIX22045/document.

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L’unité cellulaire du muscle squelettique est la myofibre, un syncytium hautement spécialisé générant la contraction musculaire. Au cours de la croissance et de la régénération musculaire, les cellules satellites quiescentes (cellules souches) du muscle squelettique adulte sont activées, prolifèrent puis fusionnent formant de nouvelles fibres. A l’aide d’un modèle murin de régénération et de cultures primaires, j’ai identifié TSHZ3 comme un nouveau marqueur des cellules satellites quiescentes et activées. Dans la lignée cellulaire C2C12, j’ai mis en évidence un effet répresseur spécifique de Tshz3 sur la différenciation myogénique. L’entrée des myoblastes dans la voie de différenciation terminale est déclenchée par le facteur Myogenin (MYOG). L’activation de la transcription du gène myogenin (Myog) est dépendante du facteur MYOD et fait intervenir le complexe de remodelage de la chromatine SWI/SNF. In vitro, TSHZ3 interagit avec BAF57 une sous unité du complexe SWI/SNF. TSHZ3 réprime l’activation dépendante de MYOD sur le promoteur proximal de Myog et cette répression dépend en partie de la présence de BAF57. L’activité répressive et la cinétique d’expression de Tshz3, indique que TSHZ3 pourrait empêcher l’activation prématurée du promoteur Myog lors de la prolifération des cellules satellites activées. TSHZ3 pourrait ainsi participer aux mécanismes de régulation permettant de contrôler l’équilibre entre prolifération, différenciation et renouvellement des progéniteurs myogéniques
Skeletal muscles are made of several units called myofibers, a syncitium into which muscular contraction is generated. During the muscle growth and repair, the quiescent Satellite Cells (SCs; adult stem cells) become activated, proliferate and differentiate to form new multinucleated myofibers. In animal model and primary culture, I found that, Tshz3 was strongly expressed in the quiescent and activated satellite cells.In C2C12 myoblast cells, I showed a specific repressive effect of TSHZ3 on the myogenic differentiation. The terminal differentiation of the myoblastes is trigger by Myogenin (Myog). The transcriptional activation of Myog promoter involves MYOD and the SWI/SNF remodelling complex. In vitro, I showed that TSHZ3 interacts with BAF57, a subunit of the SWI/SNF complex. TSHZ3 represses the MYOD-dependant activation on the Myog promoter. This specific repression involves in part BAF57.The repressive activity of and the temporal dynamic of expression of Tshz3, indicated that TSHZ3 potentially is required to impede the premature activation of the Myog promotor during the SCs proliferation. These results suggest that TSHZ3 plays important roles in the molecular mechanisms operating in activated SCs when there are poised between proliferation, differentiation and self renewal of muscular progenitors
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Brunelli, Roberta de Matos 1985. "Os efeitos do laser de baixa potência no processo de reparo muscular após criolesão em ratos = The effects of low-level laser therapy on muscle healing process after cryolesion." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308777.

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Orientadores: Daniela Cristina Carvalho de Abreu, Alberto Cliquet Junior
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
Made available in DSpace on 2018-08-22T07:19:52Z (GMT). No. of bitstreams: 1 Brunelli_RobertadeMatos_M.pdf: 1872583 bytes, checksum: 5ce843f202a01778398ee2807e19dd08 (MD5) Previous issue date: 2013
Resumo: O objetivo deste estudo foi verificar os efeitos da laserterapia de baixa potência no comprimento de onda ?=780nm entre diferentes períodos de tratamento 7, 14 e 21 dias e verificar a dose (10J/cm2 ou 50J/cm2) que promove melhor reparo muscular através das análises histopatológicas e imunohistoquímicas. Foram utilizados 54 ratos machos divididos em 3 grupos: GC: grupo controle (criolesão, sem tratamento); G10: criolesão do músculo tibial anterior (TA) e tratados com laser dose 10J/cm² e G50: criolesão do músculo TA e tratados com laser dose 50J/cm² que foram subdivididos em 3 subgrupos (n=6): 7, 14 e 21 dias de tratamento. Os achados histopatológicos revelaram maior organização das fibras musculares dos grupos tratados com laser 10J/cm² e 50J/cm² durante os períodos 7 e 14 dias em relação ao grupo controle; no período 21 dias os grupos apresentaram semelhanças na reparação tecidual. Em relação à área da lesão os grupos tratados com laser 10J/cm² e 50J/cm² durante 7 dias obtiveram diminuição significativa (p ? 0.05) da área da lesão em relação ao grupo controle, sendo que os grupos 14 e 21 dias não apresentaram diferenças significativas entre eles. Na contagem dos vasos o grupo tratado com laser 10J/cm² no 14° dia apresentou aumento dos vasos em relação ao grupo tratado com dose 50J/cm², mas não em relação ao grupo controle. Nos tempos de 7 e 21 dias os grupos não apresentaram diferença significativa entre si. Com relação às análises imunohistoquímicas da myoD no período de 7 dias os grupos tratados com laser 10J/cm² e 50J/cm² apresentaram maior imunomarcação comparada com o grupo controle, no período 14 e 21 dias a imunomarcação estava ausente. A imunomarcação da miogenina estava presente de forma semelhante nos períodos 7 e 14 dias para os três grupos analisados e no período 21 dias a imunomarcação da miogenina estava ausente em todos os grupos experimentais. Os resultados mostraram que o laser possui efeitos positivos no reparo muscular
Abstract: The objective of this study was to assess the effects of 780nm low-level laser therapy at different periods of 7, 14 and 21 days after cryolesion, including the dose (10 or 50J/cm2) to promote a better muscle repair evidenced by histopathological and immumohistochemical analyses. Fifty-four male rats were divided into three groups: injured control group (CG) - injured animals without any treatment; injured 780nm laser treated group, at 10 J/cm² (G10) and injured 780nm laser treated group, at 50 J/cm² (G50). Each group was divided into 3 subgroups (n=6): 7, 14 and 21 days post-injury. Histopathological findings revealed better-organized muscle fibers in the G10 and G50 during the periods of 7 and 14 days compared to CG. The G10 and G50 during 7 days showed a significant reduction (p? 0.05) of lesion area compared to CG, without differences between groups treated for 14 and 21 days. The G10 showed an increase of the amount of vessels after 14 days compared to the G50, but not in relation to controls. With regard to the immumohistochemical analyses of the MyoD factor, The G10 and G50 during 7 days showed higher concentrations of immunomarkers than controls. Myogenin immunomarkers were similarly observed at days 7 and 14 in all three groups analyzed, whereas immunomarkers were found in none of the groups after 21 days of laser therapy. The results showed that laser has positive effects on muscle repair
Mestrado
Fisiopatologia Cirúrgica
Mestra em Ciências
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4

Sawyer, Debbie Ann. "Analogues of myo-inositol and D-myo-inositol 1,4,5-triphosphate." Thesis, University of Leicester, 1993. http://hdl.handle.net/2381/33883.

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Oelsner, Malte. "Myon-Einfang durch den 3He-Kern." [S.l. : s.n.], 1999. http://deposit.ddb.de/cgi-bin/dokserv?idn=957152892.

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Arponen, Felicia. "Mifepristonbehandling vid myom : Effekt- och säkerhetsaspekter." Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-94171.

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Bakgrund: Myom är den vanligaste gynekologiska benigna tumören hos kvinnor i fertil ålder. Det finns idag läkemedelsbehandlingar, invasiva ingrepp och icke-invasiva ingrepp vid behandling av myom. Utvecklingen av läkemedel mot myom är lågprioriterat, eftersom de främst är benigna och snarare leder till sjuklighet än dödlighet. De läkemedel som idag används vid behandling av myom är ulipristal, GnRH-agonister, NSAID, tranexamsyra, p-piller eller hormonspiral. Ulipristal och GnRH-agonister har utöver förbättring av symtom som alla de senast nämnda, en effekt på reducering av myomstorlek. Icke-invasiva ingrepp innefattar idag myolys, embolisering av arteria uterina och fokuserad ultraljudskirurgi under MRI-vägledning, framtagna för att slippa operativa ingrepp. Myomektomi och hysterektomi är två operativa ingrepp som genomförs om inga andra behandlingar fungerar, eftersom de medför en längre återhämtningstid och en större risk för komplikationer. Mifepriston är en antiprogesteron, vilket innebär att den hämmar progesterons effekt. Progesteron i sin tur spelar en stor roll i utvecklingen av ett myom. Behandling med läkemedlet har i många olika studier visat goda resultat både i avseende på myomstorlek, symtom, livskvalité och biverkningar. Syfte: Syftet med examensarbetet var att undersöka om mifepriston är ett säkert och effektivt läkemedel vid behandling av myom hos kvinnor. Metod: Examensarbetet är en litteraturstudie och baseras på 6 olika randomiserade kontrollerade vetenskapliga studier som undersökte mifepristons effekt och säkerhet vid behandling av myom. Studierna hämtades från PubMed. Resultat: Samtliga studier visade en reducering i myomvolym, en förbättring av symtom och milda biverkningar med olika doseringar av mifepriston vid behandling av myom. I 2 av studierna undersöktes livskvalitén vilket ökade hos de personer som behandlades med mifepriston. Slutsats: Det är svårt att dra generella slutsatser vilken dos och behandlingstid som är optimala på grund av de olika behandlingstiderna, doseringarna och studieuppläggen som användes i samtliga studier. Dock har mifepriston en god effekt avseende på reducering i myomstorlek, symtom, biverkningar och livskvalité. Det krävs dock fler studier för att säkerställa dosering och behandlingstid samt fler jämförelser med andra behandlingsalternativ som idag finns för behandling av myom.
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Tripet, Angèle. "The exclusive production of r0 [rho 0] mesons in polarized muon nucleon scattering within the SMC experiment at CERN." [S.l. : s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=968580343.

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ZOUBIAN, MARWAN. "La myose stromale endolymphatique (m. S. E. )." Saint-Etienne, 1988. http://www.theses.fr/1988STET6020.

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Xiao, Lei. "Transcriptional Regulation of the Xenopus MyoD Gene." Diss., lmu, 2003. http://nbn-resolving.de/urn:nbn:de:bvb:19-11960.

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Styer, Jean Christine. "Regulating Inositol Biosynthesis in Plants: Myo-Inositol Phosphate Synthase and Myo-Inositol Monophosphatase." Thesis, Virginia Tech, 2000. http://hdl.handle.net/10919/9870.

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Inositol is important for normal growth and development in plants. The regulation of the inositol biosynthetic enzymes, myo-inositol phosphate synthase (MIPS) and myo-inositol monophosphatase (IMP) was investigated. The specific aims of this research were (1) to develop a tool to study MIPS protein accumulation in a model plant system, Arabidopsis thaliana (At) and potentially other plant species and (2) to determine the spatial expression patterns of Lycopersicon esculentum IMP-2 (LeIMP-2) at the cellular level.

Myo-inositol phosphate synthase (mips) genes have been identified in plants, animals, fungi and bacteria. Alignment of the predicted amino acid sequences of AtMIPS-1, -2 and Glycine max MIPS (GmMIPS) indicated that AtMIPS-1 and GmMIPS are 87% identical, and AtMIPS-2 and GmMIPS are 89% identical. Based on these data, a Gmmips cDNA was fused at the N-terminus to a 6X histidine tag (5' GAC GAC GAC GAC GAC GAC 3'), cloned into an overexpression vector and overexpressed in E. coli. The fusion protein, HISMIPS, was extracted using denaturing conditions and purified using Ni2+ affinity chromatography. Anti-GmMIPS antiserum from rabbit detected the recombinant HISMIPS protein (76 kD), and GmMIPS (64 kD). Affinity purification by subtractive chromatography yielded anti-GmMIPS antibody that detected MIPS (66 kD) and a protein (34 kD) of unknown function. AtMIPS accumulated to high levels in unopened flowers, opened flowers, and immature siliques (6 mm in length or less), but was not detectable in bolts, cauline or rosette leaves.

The tomato inositol monophosphatase (Leimp) genes are a developmentally regulated multigene family. From analysis of sequences, Leimp-2 is intron-less and has the putative start site of translation located at +108 bp downstream from the putative start site of transcription. Investigation of the 5â UTR revealed the 3' end of a partial open reading frame (338 bp) highly homologous to the gene for calmodulin. Three light responsive elements and a cold responsive element were also identified in the 5' UTR.

Transgenic Leimp-2::uidA plants were produced using the existing construct of the Leimp-2 promoter fused to the uidA gene (J. Keddie, University of California at Berkeley). Seedlings were perserved and sectioned. Using histological techniques, the analysis of the Leimp-2 promoter::uidA transgenic seedlings revealed that the Leimp-2 promoter causes expression at the base of the shoot apex and within leaflets of the first set of fully expanded leaves. Further, Leimp-2 promoter expression was localized to epidermal and cortex cells on the abaxial side of the 1st and 2nd fully expanded compound leaves.

These studies of MIPS and IMP expression lay a foundation for a better understanding of the regulation of inositol biosynthesis in Arabidopsis, tomato, and other plant species.


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Books on the topic "Myos"

1

Sñan rtsom tshaṅs sras myos paʼi drug ʼgyur. Pe-cin: Mi rigs dpe skrun khaṅ, 1996.

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Changgi ŭi myosu: Myosu chung ŭi myosu pʻuri. Sŏul: Usŏng Chʻulpʻansa, 1991.

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Atwood, Margaret Eleanor. Myös sinun nimesi. Helsinki: Werner Söderström Osakeyhtiö, 2001.

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Berberova, Nina Nikolaevna. Mys Burʹ. Moskva: AST, 2011.

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Uri sik ipch'ejŏk myosa. 2nd ed. [P'yŏngyang]: Sahoe Kwahak Ch'ulp'ansa, 2013.

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Chungguk Chosŏnjok Collection (Library of Congress. Asian Division. Korean Section), ed. Ŏhwi myosa pʻyohyŏn pullyujip. Yŏnʼgil: Yŏnbyŏn Inmin Chʻulpʻansa, 1998.

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Zwa-ser cod pan ʼdzin paʼi Paṇḍi-ta Dkon-mchog ʼJigs-med-dbaṅ-poʼam ʼJam-dbyaṅs-legs-bśad-phreṅ-baʼi-blo-gros kyi padmo las byuṅ ʼphrin yig sñan ṅag gi rim pa buṅ ba myos paʼi glu dbyaṅs. Zi-liṅ: Mtsho-sṅon mi rigs dpe skrun khaṅ, 1996.

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Fazin, Zinoviĭ. Khersonesskiĭ mys: Roman. Moskva: Sov. pisatelʹ, 1986.

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Fazin, Z. Khersonesskiĭ mys: Roman. Moskva: Sov. pisatelʹ, 1986.

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Martínek, Lubomír. Mys dobré beznaděje. [Prague]: Český spisovatel, 1994.

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Book chapters on the topic "Myos"

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Strauss, Alexander. "Myom." In Ultraschallpraxis, 383–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-662-10678-5_105.

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Gewies, Andreas, Jürgen Ruland, Alexey Kotlyarov, Matthias Gaestel, Shiri Procaccia, Rony Seger, Shin Yasuda, et al. "Myosin I (Myo1)." In Encyclopedia of Signaling Molecules, 1165–69. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_529.

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Coluccio, Lynne M. "Myosin I (Myo1)." In Encyclopedia of Signaling Molecules, 3305–9. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_529.

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Hild, Manfred, Torsten Siedel, Christian Benckendorff, Christian Thiele, and Michael Spranger. "Myon, a New Humanoid." In Language Grounding in Robots, 25–44. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-3064-3_2.

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Fraser, Gordon, Egil Lillestøl, and Inge Sellevåg. "Das Myon tritt auf." In Auf der Suche nach dem Unendlichen, 52–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59930-9_22.

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Schatz, Günter, and Alois Weidinger. "Myon-Spin-Rotation (μSR)." In Nukleare Festkörperphysik, 176–210. Wiesbaden: Vieweg+Teubner Verlag, 1992. http://dx.doi.org/10.1007/978-3-322-93989-0_8.

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Schatz, Günter, Alois Weidinger, and Manfred Deicher. "Myon-Spin-Rotation (μSR)." In Nukleare Festkörperphysik, 151–82. Wiesbaden: Vieweg+Teubner, 2010. http://dx.doi.org/10.1007/978-3-8348-9835-7_7.

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Nachtmann, Otto, and Roman U. Sexl. "Das Myon und die Myon-Paarproduktion in der Elektron-Positron-Vernichtung." In Phänomene und Konzepte der Elementarteilchenphysik, 121–26. Wiesbaden: Vieweg+Teubner Verlag, 1986. http://dx.doi.org/10.1007/978-3-663-07776-3_11.

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Schomburg, Dietmar, and Dörte Stephan. "myo-Inositol oxygenase." In Enzyme Handbook, 253–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-57942-4_54.

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Schomburg, Dietmar, and Dörte Stephan. "myo-Inositol 1-kinase." In Enzyme Handbook 13, 913–15. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-59176-1_174.

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Conference papers on the topic "Myos"

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Dodds, Zachary, and Michael Erlinger. "MyCS." In Proceeding of the 44th ACM technical symposium. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2445196.2445417.

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Gopu, Arvind, Soichi Hayashi, and Robert Quick. "MyOSG." In the 5th Grid Computing Environments Workshop. New York, New York, USA: ACM Press, 2009. http://dx.doi.org/10.1145/1658260.1658276.

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Dodds, Zachary, and Michael Erlinger. "MyCS." In the 18th ACM conference. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2462476.2465611.

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Schofield, Elizabeth, Michael Erlinger, and Zachary Dodds. "MyCS." In the 45th ACM technical symposium. New York, New York, USA: ACM Press, 2014. http://dx.doi.org/10.1145/2538862.2538901.

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Ashmore, Thomas, Sorathan Chaturapruek, Zachary Dodds, Corinne Druhan, Bridgette Eichelberger, Michael Erlinger, and Elizabeth Schofield. "MyCS." In the 45th ACM technical symposium. New York, New York, USA: ACM Press, 2014. http://dx.doi.org/10.1145/2538862.2544313.

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Liu, Zhihui, Xiyuan Zhang, Haiyan Lei, Norris Lam, Oliver Yockey, Max Xu, Arnulfo Mendoza, et al. "Abstract B42: Mutant RAS represses CASZ1, a novel regulator of MYOD and MYOG, to inhibit embryonal rhabdomyosarcoma differentiation." In Abstracts: AACR Special Conference on the Advances in Pediatric Cancer Research; September 17-20, 2019; Montreal, QC, Canada. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.pedca19-b42.

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Andow, Sam, Kaitlyn Eng, Julia McCarthy, Olivia Palenscar, Thomas Schneider, Adam Schulze, Zachary Dodds, and Bryan Twarek. "Merging MyCS." In SIGCSE '17: The 48th ACM Technical Symposium on Computer Science Education. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3017680.3022414.

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Castro, Brenda, Terrence Diaz, Marissa Gee, Rebekah Justice, David Kwan, Preethi Seshadri, and Zachary Dodds. "MyCS at 5." In the 47th ACM Technical Symposium. New York, New York, USA: ACM Press, 2016. http://dx.doi.org/10.1145/2839509.2844643.

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Kanebako, Junichi, Hiroaki Oishi, Lisako Ishigami, and Hiroko Uchiyama. "myo-skin." In SIGGRAPH Asia 2013 Emerging Technologies. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2542284.2542296.

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McCullough, Morgan, Hong Xu, Joel Michelson, Matthew Jackoski, Wyatt Pease, William Cobb, William Kalescky, Joshua Ladd, and Betsy Williams. "Myo arm." In SAP '15: ACM Symposium on Applied Perception 2015. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2804408.2804416.

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Reports on the topic "Myos"

1

Erdmann, Martin. Lebensdauer des Farbigen Protons in der Myon-Proton-Streuung. Office of Scientific and Technical Information (OSTI), January 1990. http://dx.doi.org/10.2172/1426711.

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Ecker, Uwe. Longitudinale und Transversale Impulsverteilungen der Hadronen im Endzustand der Tiefinelastischen Myon-Nukleon-Streuung. Office of Scientific and Technical Information (OSTI), January 1991. http://dx.doi.org/10.2172/1372870.

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Hantke, Detlev. Untersuchung der Produktion von Neutralen Seltsamen Teilchen in der Tief - Inelastischen Myon - Nukleon - Streuung bei einer Strahlenergie von 490 GeV. Office of Scientific and Technical Information (OSTI), January 1993. http://dx.doi.org/10.2172/1425586.

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