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Relevant bibliographies by topics / Myosins; GTP-Binding Proteins; Academic Dissertations

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Dissertations / Theses

Academic literature on the topic 'Myosins; GTP-Binding Proteins; Academic Dissertations'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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Dissertations / Theses on the topic "Myosins; GTP-Binding Proteins; Academic Dissertations"

1

Saeki, Nobutaka. "The Function of Myosin IX: the Ninth Class of Myosin Superfamily: a Dissertation." eScholarship@UMMS, 2005. http://escholarship.umassmed.edu/gsbs_diss/294.

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Among 18 family members in the myosin superfamily, myosin IX is unique by possessing a GTPase activating protein (GAP) for Rho. It is also attention-grabbing since it is a single-headed processive motor, as well as a minus-end directed motor. Although many biochemical properties have been revealed, its physiological function is largely unknown. As an initial step to address this question, I attempted to find the binding partner of myosin IXb using the yeast two-hybrid screen. Through the screen using the tail domain of myosin IXb as bait I found BIG1, a guanine nucleotide exchange factor (GEF)

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2

Woon, Chee-Wai. "Mutations of the Alpha-Subunit of G-Proteins: A Thesis." eScholarship@UMMS, 1988. http://escholarship.umassmed.edu/gsbs_diss/296.

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Signal transduction by G-proteins (a heterotrimer membrane protein composed of an α, β, and γ subunit) requires that the α-subunit undergoes a transition from a GDP-bound inactive state to an activated GTP-bound state. The exchange of GDP for GTP leads to a conformational change in the α-subunit that results in the loss of affinity for the βγ subunits. We predicted that appropriate genetic manipulation of key regions of the α-subunit could result in the induction of the active conformation that would mimic at least in part the activated GTP-bound state. We have demonstrated that the substitu

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3

Zhu, Zhongyuan. "Structural and Mutational Analysis of Rab2A Activation by Mss4: A Dissertation." eScholarship@UMMS, 2000. http://escholarship.umassmed.edu/gsbs_diss/176.

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The function of GTP-binding proteins (G-proteins) in diverse intracellular pathways depends on their ability to switch between two forms, a GDP-bound (inactive) form and a GTP bound (active) form in a highly regulated GTPase cycle. The inactivation step of this cycle is regulated by GTPase-activating proteins (GAPs) which increase the intrinsic rate of hydrolysis of bound GTP; the activation step is regulated by a diverse family of GDP/GTP exchange factors (GEFs). A unique model system, which consists of the 13 kDa GEF Mss4 and the monomeric G protein Rab3A involved in presynaptic neurotransm

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4

Macdonald, Susan G. "G Protein Interactions with the Substance P Receptor in Rat Submaxillary Gland: a Dissertation." eScholarship@UMMS, 1991. http://escholarship.umassmed.edu/gsbs_diss/268.

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Substance P (SP) is an undecapeptide whose functions are as varied as its locations. In the nervous system, it is thought to act as a neurotransmitter. In the peripheral vasculature, it has hypotensive effects and it causes contraction in the smooth muscle of the gut. In salivary gland, it is a potent secretagogue and it is how this effect is transduced that is the subject of this dissertation. Activation of the SP receptor in rat submaxillary gland by SP results in the hydrolysis of inositol phospholipids and the mobilization of intracellular Ca2+. These second messengers are then able to ac

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5

Roche, John Patrick. "The Role of Ca²⁺ Channel Subunit Composition in G Protein-Mediated Inhibition of Ca²⁺ Channels: a Disstertation." eScholarship@UMMS, 1997. http://escholarship.umassmed.edu/gsbs_diss/281.

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Modulation of Ca2+ channels is an important mechanism for regulation of synaptic strength. However, it is clear that some Ca2+ current types are insensitive to inhibitory modulation mediated by heterotrimeric G proteins (G protein inhibition), and among currents which are sensitive to G protein inhibition, there is great variation in the magnitude of Ca2+ current inhibition between cells of different origin. For the experiments in this dissertation, I utilized recently cloned Ca2+ channels to determine the minimal combination of Ca2+ channel subunits which would confer G protein sensitivity to

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6

Adhikari, Anirban. "Regulation of guanine nucelotide exchange in inhibitory G protein alpha subunit by activator of G protein signaling 3 and novel regulatory peptides." Embargoed access until after 12/19/2006, 2005. http://www4.utsouthwestern.edu/library/ETD/etdDetails.cfm?etdID=114.

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7

Raman, Malavika. "Identification of intracellular signaling pathways regulated by the TAO family of mammalian STE20p kinases." Access to abstract only; dissertation is embargoed until after 5/15/2007, 2006. http://www4.utsouthwestern.edu/library/ETD/etdDetails.cfm?etdID=163.

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8

Davis, Tara Lynne. "Heterotrimeric G protein beta : gamma bound to a biologically active peptide : structural definition of a preferred protein interaction surface." 2004. http://edissertations.library.swmed.edu/pdf/DavisT050405/DavisTara.pdf.

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9

Arora, Vivek Kumar. "Identification and characterization of a NEF associated kinase." 2002. http://edissertations.library.swmed.edu/pdf/AroraV050404/AroraVivek.pdf.

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10

Hatley, Mark Edward. "Allosteric determinants of guanine nucleotide binding proteins and methods to crystallize the cytosolic domains of adenylyl cyclase." 2004. http://edissertations.library.swmed.edu/pdf/HatleyM050404/HatleyMark.pdf.

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