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Relevant bibliographies by topics / N-Acylethanolamine metabolism

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Journal articles

Academic literature on the topic 'N-Acylethanolamine metabolism'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

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Journal articles on the topic "N-Acylethanolamine metabolism"

1

Ueda, Natsuo, Kazuhito Tsuboi, and Toru Uyama. "N-acylethanolamine metabolism with special reference to N-acylethanolamine-hydrolyzing acid amidase (NAAA)." Progress in Lipid Research 49, no. 4 (2010): 299–315. http://dx.doi.org/10.1016/j.plipres.2010.02.003.

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2

Natarajan, Viswanathan, Patricia C. Schmid, and Harald H. O. Schmid. "N-Acylethanolamine phospholipid metabolism in normal and ischemic rat brain." Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism 878, no. 1 (1986): 32–41. http://dx.doi.org/10.1016/0005-2760(86)90341-3.

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3

Teaster, Neal D., Christy M. Motes, Yuhong Tang, et al. "N-Acylethanolamine Metabolism Interacts with Abscisic Acid Signaling in Arabidopsis thaliana Seedlings." Plant Cell 19, no. 8 (2007): 2454–69. http://dx.doi.org/10.1105/tpc.106.048702.

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4

Gao, Wei, Kornelius Schmidt, Sören Enge, and Clemens Kirschbaum. "Intra-individual stability of hair endocannabinoid and N-acylethanolamine concentrations." Psychoneuroendocrinology 133 (November 2021): 105395. http://dx.doi.org/10.1016/j.psyneuen.2021.105395.

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5

Pai, Alex Y., Cachet Wenziger, Elani Streja, et al. "Impact of Circulating N-Acylethanolamine Levels with Clinical and Laboratory End Points in Hemodialysis Patients." American Journal of Nephrology 52, no. 1 (2021): 59–68. http://dx.doi.org/10.1159/000513381.

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Background: Patients with ESRD on maintenance hemodialysis (MHD) are particularly susceptible to dysregulation of energy metabolism, which may manifest as protein energy wasting and cachexia. In recent years, the endocannabinoid system has been shown to play an important role in energy metabolism with potential relevance in ESRD. N-acylethanolamines are a class of fatty acid amides which include the major endocannabinoid ligand, anandamide, and the endogenous peroxisome proliferator-activated receptor-α agonists, oleoylethanolamide (OEA) and palmitoylethanolamide (PEA). Methods: Serum concentr

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6

Dalle Carbonare, M., E. Del Giudice, A. Stecca, et al. "A Saturated N-Acylethanolamine Other than N-Palmitoyl Ethanolamine with Anti-inflammatory Properties: a Neglected Story…" Journal of Neuroendocrinology 20, s1 (2008): 26–34. http://dx.doi.org/10.1111/j.1365-2826.2008.01689.x.

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7

Zhao, Li-Ying, Kazuhito Tsuboi, Yasuo Okamoto, Shunichiro Nagahata, and Natsuo Ueda. "Proteolytic activation and glycosylation of N-acylethanolamine-hydrolyzing acid amidase, a lysosomal enzyme involved in the endocannabinoid metabolism." Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids 1771, no. 11 (2007): 1397–405. http://dx.doi.org/10.1016/j.bbalip.2007.10.002.

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8

Pandey, Mukesh K., Timothy R. DeGrado, Kun Qian, et al. "Synthesis and Preliminary Evaluation of N-(16-18F-Fluorohexadecanoyl)ethanolamine (18F-FHEA) as a PET Probe of N-Acylethanolamine Metabolism in Mouse Brain." ACS Chemical Neuroscience 5, no. 9 (2014): 793–802. http://dx.doi.org/10.1021/cn400214j.

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9

Lefort, Charlotte, Martin Roumain, Matthias Van Hul, et al. "Hepatic NAPE-PLD Is a Key Regulator of Liver Lipid Metabolism." Cells 9, no. 5 (2020): 1247. http://dx.doi.org/10.3390/cells9051247.

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Diverse metabolic disorders have been associated with an alteration of N-acylethanolamine (NAE) levels. These bioactive lipids are synthesized mainly by N-acylphosphatidylethanolamine-selective phospholipase D (NAPE-PLD) and influence host metabolism. We have previously discovered that NAPE-PLD in the intestine and adipose tissue is connected to the pathophysiology of obesity. However, the physiological function of NAPE-PLD in the liver remains to be deciphered. To study the role of liver NAPE-PLD on metabolism, we generated a new mouse model of inducible Napepld hepatocyte-specific deletion (

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10

D’Aloia, Alessia, Federica Arrigoni, Renata Tisi, et al. "Synthesis, Molecular Modeling and Biological Evaluation of Metabolically Stable Analogues of the Endogenous Fatty Acid Amide Palmitoylethanolamide." International Journal of Molecular Sciences 21, no. 23 (2020): 9074. http://dx.doi.org/10.3390/ijms21239074.

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Palmitoylethanolamide (PEA) belongs to the class of N-acylethanolamine and is an endogenous lipid potentially useful in a wide range of therapeutic areas; products containing PEA are licensed for use in humans as a nutraceutical, a food supplement, or food for medical purposes for its analgesic and anti-inflammatory properties demonstrating efficacy and tolerability. However, the exogenously administered PEA is rapidly inactivated; in this process, fatty acid amide hydrolase (FAAH) plays a key role both in hepatic metabolism and in intracellular degradation. So, the aim of the present study wa

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