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Journal articles on the topic 'N-Acylethanolamine metabolism'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

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1

Ueda, Natsuo, Kazuhito Tsuboi, and Toru Uyama. "N-acylethanolamine metabolism with special reference to N-acylethanolamine-hydrolyzing acid amidase (NAAA)." Progress in Lipid Research 49, no. 4 (2010): 299–315. http://dx.doi.org/10.1016/j.plipres.2010.02.003.

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2

Natarajan, Viswanathan, Patricia C. Schmid, and Harald H. O. Schmid. "N-Acylethanolamine phospholipid metabolism in normal and ischemic rat brain." Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism 878, no. 1 (1986): 32–41. http://dx.doi.org/10.1016/0005-2760(86)90341-3.

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3

Teaster, Neal D., Christy M. Motes, Yuhong Tang, et al. "N-Acylethanolamine Metabolism Interacts with Abscisic Acid Signaling in Arabidopsis thaliana Seedlings." Plant Cell 19, no. 8 (2007): 2454–69. http://dx.doi.org/10.1105/tpc.106.048702.

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4

Gao, Wei, Kornelius Schmidt, Sören Enge, and Clemens Kirschbaum. "Intra-individual stability of hair endocannabinoid and N-acylethanolamine concentrations." Psychoneuroendocrinology 133 (November 2021): 105395. http://dx.doi.org/10.1016/j.psyneuen.2021.105395.

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5

Pai, Alex Y., Cachet Wenziger, Elani Streja, et al. "Impact of Circulating N-Acylethanolamine Levels with Clinical and Laboratory End Points in Hemodialysis Patients." American Journal of Nephrology 52, no. 1 (2021): 59–68. http://dx.doi.org/10.1159/000513381.

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Background: Patients with ESRD on maintenance hemodialysis (MHD) are particularly susceptible to dysregulation of energy metabolism, which may manifest as protein energy wasting and cachexia. In recent years, the endocannabinoid system has been shown to play an important role in energy metabolism with potential relevance in ESRD. N-acylethanolamines are a class of fatty acid amides which include the major endocannabinoid ligand, anandamide, and the endogenous peroxisome proliferator-activated receptor-α agonists, oleoylethanolamide (OEA) and palmitoylethanolamide (PEA). Methods: Serum concentr

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6

Dalle Carbonare, M., E. Del Giudice, A. Stecca, et al. "A Saturated N-Acylethanolamine Other than N-Palmitoyl Ethanolamine with Anti-inflammatory Properties: a Neglected Story…" Journal of Neuroendocrinology 20, s1 (2008): 26–34. http://dx.doi.org/10.1111/j.1365-2826.2008.01689.x.

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7

Zhao, Li-Ying, Kazuhito Tsuboi, Yasuo Okamoto, Shunichiro Nagahata, and Natsuo Ueda. "Proteolytic activation and glycosylation of N-acylethanolamine-hydrolyzing acid amidase, a lysosomal enzyme involved in the endocannabinoid metabolism." Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids 1771, no. 11 (2007): 1397–405. http://dx.doi.org/10.1016/j.bbalip.2007.10.002.

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8

Pandey, Mukesh K., Timothy R. DeGrado, Kun Qian, et al. "Synthesis and Preliminary Evaluation of N-(16-18F-Fluorohexadecanoyl)ethanolamine (18F-FHEA) as a PET Probe of N-Acylethanolamine Metabolism in Mouse Brain." ACS Chemical Neuroscience 5, no. 9 (2014): 793–802. http://dx.doi.org/10.1021/cn400214j.

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9

Lefort, Charlotte, Martin Roumain, Matthias Van Hul, et al. "Hepatic NAPE-PLD Is a Key Regulator of Liver Lipid Metabolism." Cells 9, no. 5 (2020): 1247. http://dx.doi.org/10.3390/cells9051247.

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Diverse metabolic disorders have been associated with an alteration of N-acylethanolamine (NAE) levels. These bioactive lipids are synthesized mainly by N-acylphosphatidylethanolamine-selective phospholipase D (NAPE-PLD) and influence host metabolism. We have previously discovered that NAPE-PLD in the intestine and adipose tissue is connected to the pathophysiology of obesity. However, the physiological function of NAPE-PLD in the liver remains to be deciphered. To study the role of liver NAPE-PLD on metabolism, we generated a new mouse model of inducible Napepld hepatocyte-specific deletion (

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10

D’Aloia, Alessia, Federica Arrigoni, Renata Tisi, et al. "Synthesis, Molecular Modeling and Biological Evaluation of Metabolically Stable Analogues of the Endogenous Fatty Acid Amide Palmitoylethanolamide." International Journal of Molecular Sciences 21, no. 23 (2020): 9074. http://dx.doi.org/10.3390/ijms21239074.

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Palmitoylethanolamide (PEA) belongs to the class of N-acylethanolamine and is an endogenous lipid potentially useful in a wide range of therapeutic areas; products containing PEA are licensed for use in humans as a nutraceutical, a food supplement, or food for medical purposes for its analgesic and anti-inflammatory properties demonstrating efficacy and tolerability. However, the exogenously administered PEA is rapidly inactivated; in this process, fatty acid amide hydrolase (FAAH) plays a key role both in hepatic metabolism and in intracellular degradation. So, the aim of the present study wa

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11

Chapman, Kent D. "Occurrence, metabolism, and prospective functions of N-acylethanolamines in plants." Progress in Lipid Research 43, no. 4 (2004): 302–27. http://dx.doi.org/10.1016/j.plipres.2004.03.002.

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12

Coulon, Denis, Lionel Faure, Magali Salmon, Valerie Wattelet, and Jean-Jacques Bessoule. "N-Acylethanolamines and related compounds: Aspects of metabolism and functions." Plant Science 184 (March 2012): 129–40. http://dx.doi.org/10.1016/j.plantsci.2011.12.015.

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13

BERDYSHEV, Evgueni V., Patricia C. SCHMID, Randy J. KREBSBACH, et al. "Cannabinoid-receptor-independent cell signalling by N-acylethanolamines." Biochemical Journal 360, no. 1 (2001): 67–75. http://dx.doi.org/10.1042/bj3600067.

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Anandamide and other polyunsaturated N-acylethanolamines (NAEs) exert biological activity by binding to cannabinoid receptors. These receptors are linked to Gi/o proteins and their activation leads to extracellular-signal-regulated protein kinase (ERK) and c-Jun N-terminal kinase (JNK) mitogen-activated protein kinase (MAP kinase) activation, inhibition of cAMP-dependent signalling and complex changes in the expression of various genes. Saturated and monounsaturated NAEs cannot bind to cannabinoid receptors and may thus mediate cell signalling through other targets. Here we report that both sa

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14

Ferrara, Anne Lise, Fabiana Piscitelli, Angelica Petraroli, et al. "Altered Metabolism of Phospholipases, Diacylglycerols, Endocannabinoids, and N-Acylethanolamines in Patients with Mastocytosis." Journal of Immunology Research 2019 (July 1, 2019): 1–14. http://dx.doi.org/10.1155/2019/5836476.

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Background. Mastocytosis is a condition characterized by the expansion and accumulation of mast cells (MCs) in various organs. The symptoms are related to the increased release of MC-derived mediators that exert local and distant effects. MCs are a source and target of phospholipase enzymes (PLs), which catalyze the cleavage of membrane phospholipids releasing lipid mediators (e.g., diacylglycerols (DAGs) and the endocannabinoid (EC) 2-arachidonoylglycerol (2-AG)). To date, there are no data on the role of these lipid mediators in mastocytosis. Here, we analyzed plasma levels of PLA2, PLC, DAG

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15

Schmid, H. H. O., and E. V. Berdyshev. "Cannabinoid receptor-inactive N -acylethanolamines and other fatty acid amides: metabolism and function." Prostaglandins, Leukotrienes and Essential Fatty Acids (PLEFA) 66, no. 2-3 (2002): 363–76. http://dx.doi.org/10.1054/plef.2001.0348.

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16

Rankin, Linda, and Christopher J. Fowler. "The Basal Pharmacology of Palmitoylethanolamide." International Journal of Molecular Sciences 21, no. 21 (2020): 7942. http://dx.doi.org/10.3390/ijms21217942.

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Palmitoylethanolamide (PEA, N-hexadecanoylethanolamide) is an endogenous compound belonging to the family of N-acylethanolamines. PEA has anti-inflammatory and analgesic properties and is very well tolerated in humans. In the present article, the basal pharmacology of PEA is reviewed. In terms of its pharmacokinetic properties, most work has been undertaken upon designing formulations for its absorption and upon characterising the enzymes involved in its metabolism, but little is known about its bioavailability, tissue distribution, and excretion pathways. PEA exerts most of its biological eff

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17

Lambert, Didier M., and Giulio G. Muccioli. "Endocannabinoids and related N-acylethanolamines in the control of appetite and energy metabolism: emergence of new molecular players." Current Opinion in Clinical Nutrition and Metabolic Care 10, no. 6 (2007): 735–44. http://dx.doi.org/10.1097/mco.0b013e3282f00061.

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18

Lin, Lin, Adam H. Metherel, Alex P. Kitson, et al. "Dietary fatty acids augment tissue levels of n-acylethanolamines in n-acylphosphatidylethanolamine phospholipase D (NAPE-PLD) knockout mice." Journal of Nutritional Biochemistry 62 (December 2018): 134–42. http://dx.doi.org/10.1016/j.jnutbio.2018.08.015.

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19

De Luca, Lucia, and Paola Vitaglione. "Food database of endocannabinoids, N-acylethanolamines and N-acylphosphatidylethanolamines and daily intake in Western, Mediterranean and Vegetarian diets." Nutrition, Metabolism and Cardiovascular Diseases 29, no. 8 (2019): 884. http://dx.doi.org/10.1016/j.numecd.2019.05.046.

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20

Keereetaweep, Jantana, and Kent D. Chapman. "Lipidomic Analysis of Endocannabinoid Signaling: Targeted Metabolite Identification and Quantification." Neural Plasticity 2016 (2016): 1–13. http://dx.doi.org/10.1155/2016/2426398.

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The endocannabinoidsN-arachidonoylethanolamide (or anandamide, AEA) and 2-arachidonoylglycerol (2-AG) belong to the larger groups ofN-acylethanolamines (NAEs) and monoacylglycerol (MAG) lipid classes, respectively. They are biologically active lipid molecules that activate G-protein-coupled cannabinoid receptors found in various organisms. After AEA and 2-AG were discovered in the 1990s, they have been extensively documented to have a broad range of physiological functions. Along with AEA, several NAEs, for example,N-palmitoylethanolamine (PEA),N-stearoylethanolamine (SEA), andN-oleoylethanola

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21

Fernández-Irigoyen, Joaquín, Paz Cartas-Cejudo, Marta Iruarrizaga-Lejarreta, and Enrique Santamaría. "Alteration in the Cerebrospinal Fluid Lipidome in Parkinson’s Disease: A Post-Mortem Pilot Study." Biomedicines 9, no. 5 (2021): 491. http://dx.doi.org/10.3390/biomedicines9050491.

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Lipid metabolism is clearly associated to Parkinson’s disease (PD). Although lipid homeostasis has been widely studied in multiple animal and cellular models, as well as in blood derived from PD individuals, the cerebrospinal fluid (CSF) lipidomic profile in PD remains largely unexplored. In this study, we characterized the post-mortem CSF lipidomic imbalance between neurologically intact controls (n = 10) and PD subjects (n = 20). The combination of dual extraction with ultra-performance liquid chromatography-electrospray ionization quadrupole-time-of-flight mass spectrometry (UPLC-ESI-qToF-M

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22

Forteza, Fabiola, Giada Giorgini, and Frédéric Raymond. "Neurobiological Processes Induced by Aerobic Exercise through the Endocannabinoidome." Cells 10, no. 4 (2021): 938. http://dx.doi.org/10.3390/cells10040938.

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Evidence suggesting the triangulation of the endocannabinoid system, exercise, and neurological health is emerging. In addition to the endocannabinoids N-arachidonoylethanolamine (anandamide; AEA) and 2-arachidonoylglycerol (2-AG), the expanded endocannabinoid system, known as the endocannabinoidome (eCBome), appears to be an important player in this relationship. The eCBome includes several endocannabinoid-like mediators such as N-acylethanolamines and 2-monoacylglycerols, the enzymes involved in their biosynthesis and degradation, and the receptors they affect. This review aims to relate the

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23

Kostrzewa, Magdalena, Ali Mokhtar Mahmoud, Roberta Verde, et al. "Modulation of Endocannabinoid Tone in Osteoblastic Differentiation of MC3T3-E1 Cells and in Mouse Bone Tissue over Time." Cells 10, no. 5 (2021): 1199. http://dx.doi.org/10.3390/cells10051199.

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Bone is a highly complex and metabolically active tissue undergoing a continuous remodeling process, which endures throughout life. A complex cell-signaling system that plays role in regulating different physiological processes, including bone remodeling, is the endocannabinoid system (ECS). Bone mass expresses CB1 and CB2 cannabinoid receptors and enzymatic machinery responsible for the metabolism of their endogenous ligands, endocannabinoids (AEA and 2-AG). Exogenous AEA is reported to increase the early phase of human osteoblast differentiation in vitro. However, regarding this cell context

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24

Joosten, Michel M., Michiel GJ Balvers, Kitty CM Verhoeckx, Henk FJ Hendriks, and Renger F. Witkamp. "Plasma anandamide and other N-acylethanolamines are correlated with their corresponding free fatty acid levels under both fasting and non-fasting conditions in women." Nutrition & Metabolism 7, no. 1 (2010): 49. http://dx.doi.org/10.1186/1743-7075-7-49.

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25

Deguchi, Hiroshi, Sunia Trauger, Ewa Kalisiak, et al. "Warfarin Untargeted Metabolomics Study Identifies Novel Procoagulant Ethanolamide Lipids." Blood 118, no. 21 (2011): 1200. http://dx.doi.org/10.1182/blood.v118.21.1200.1200.

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Abstract Abstract 1200 Untargeted metabolomics denotes the profiling of several thousand low molecular weight biochemicals, including lipids, hormones, saccharides, nucleotides, organic acids, and amino acids. The analytes are measured using mass spectrometry (MS) as molecule mass peaks without specific “targeting” of specific metabolites or analytes. We undertook an untargeted metabolomics investigation to define the effect of warfarin on plasma metabolite profiles in a crossover study of 17 venous thrombosis patients (9 male, 8 female) (age 58.1 ± 18.6 yr old; range 27–83 yr old). Blood was

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26

Tsuboi, Kazuhito, Toru Uyama, Yasuo Okamoto, and Natsuo Ueda. "Endocannabinoids and related N-acylethanolamines: biological activities and metabolism." Inflammation and Regeneration 38, no. 1 (2018). http://dx.doi.org/10.1186/s41232-018-0086-5.

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27

Chapman, Kent, Jantana Keereetaweep, and Elison Blancaflor. "Metabolism and Actions of N‐Acylethanolamines in Seedling Development." FASEB Journal 29, S1 (2015). http://dx.doi.org/10.1096/fasebj.29.1_supplement.366.4.

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28

Alexander, Stephen P. H., Patrick Doherty, Christopher J. Fowler, Jürg Gertsch, and Mario Van der Stelt. "Endocannabinoid turnover (version 2019.4) in the IUPHAR/BPS Guide to Pharmacology Database." IUPHAR/BPS Guide to Pharmacology CITE 2019, no. 4 (2019). http://dx.doi.org/10.2218/gtopdb/f943/2019.4.

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The principle endocannabinoids are 2-acylglycerol esters, such as 2-arachidonoylglycerol (2-AG), and N-acylethanolamines, such as anandamide (N-arachidonoylethanolamine, AEA). The glycerol esters and ethanolamides are synthesised and hydrolysed by parallel, independent pathways. Mechanisms for release and re-uptake of endocannabinoids are unclear, although potent and selective inhibitors of facilitated diffusion of endocannabinoids across cell membranes have been developed [19]. FABP5 (Q01469) has been suggested to act as a canonical intracellular endocannabinoid transporter in vivo [12]. For

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29

Rahman, S. M. Khaledur, Toru Uyama, Zahir Hussain, and Natsuo Ueda. "Roles of Endocannabinoids and Endocannabinoid-like Molecules in Energy Homeostasis and Metabolic Regulation: A Nutritional Perspective." Annual Review of Nutrition 41, no. 1 (2021). http://dx.doi.org/10.1146/annurev-nutr-043020-090216.

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The endocannabinoid system is involved in signal transduction in mammals. It comprises principally G protein-coupled cannabinoid receptors and their endogenous agonists, called endocannabinoids, as well as the enzymes and transporters responsible for the metabolism of endocannabinoids. Two arachidonic acid–containing lipid molecules, arachidonoylethanolamide (anandamide) and 2-arachidonoylglycerol, function as endocannabinoids. N-acylethanolamines and monoacylglycerols, in which the arachidonic acid chain is replaced with a saturated or monounsaturated fatty acid, are not directly involved in

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30

Tagliamonte, Silvia, Chris I. R. Gill, Laura Kirsty Pourshahidi, et al. "Endocannabinoids and endocannabinoid-like molecules are present in foods, blood and ileal fluids from ileostomy subjects: insight into possible metabolic implications." Proceedings of the Nutrition Society 79, OCE2 (2020). http://dx.doi.org/10.1017/s0029665120004358.

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AbstractThe endocannabinoid system is a lipid signalling system with several regulatory functions throughout the body including regulation of appetite, food intake, macronutrient metabolism, pain sensation, blood pressure, mood, cognition and immunity. It consists of endocannabinoids (ECs), their receptors and enzymes involved in their synthesis and degradation. The two best-characterized endocannabinoids are N-arachidonoylethanolamide (AEA) and 2-arachidonoylglycerol (2-AG). They are ligands of cannabinoid receptors CB1 and CB2 which are located in the central nervous system (CNS) but also in

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