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Journal articles on the topic 'N-methylnicotinamide'

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Published: 4 June 2021
Last updated: 5 February 2022

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1

Percival, Darryl, John M. D. Storey, and William T. A. Harrison. "N-(2-Bromophenyl)-N-methylnicotinamide." Acta Crystallographica Section E Structure Reports Online 63, no. 4 (2007): o1853—o1854. http://dx.doi.org/10.1107/s1600536807010707.

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2

Srikrishnan, T., and R. Parthasarathy. "Structure of N-methylnicotinamide." Acta Crystallographica Section C Crystal Structure Communications 46, no. 9 (1990): 1723–25. http://dx.doi.org/10.1107/s0108270190000658.

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3

Martsynkevich, A. M., V. V. Zakharychev, and N. I. Sharanina. "Synthesis of N,N-disubstituted (Z)-O-methylnicotinamide oximes." Russian Chemical Bulletin 60, no. 3 (2011): 521–25. http://dx.doi.org/10.1007/s11172-011-0081-3.

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4

Bendayan, Reina, Edward M. Sellers, and Melvin Silverman. "Inhibition kinetics of cationic drugs on N′-methylnicotinamide uptake by brush border membrane vesicles from the dog kidney cortex." Canadian Journal of Physiology and Pharmacology 68, no. 4 (1990): 467–75. http://dx.doi.org/10.1139/y90-066.

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The effect of cationic drugs on the uptake of the prototypical organic cation N′-methylnicotinamide has been evaluated. Using purified brash border membrane vesicles prepared from dog kidney cortex and applying a rapid Millipore filtration technique, cationic drugs apparent inhibitory constants (Ki) were calculated from kinetic analysis of N′-methylnicotinamide uptake corrected for noncarrier-mediated transport (10 s uptake; outwardly directed H+ gradient; pH 7.4, 25 °C). All of the cationic drugs tested exhibited competitive inhibition of N′-methylnicotinamide uptake suggesting that they all
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5

SHIBATA, Katsumi. "Fates of N'-methylnicotinamide and nicotinamide N-oxide in mice." Agricultural and Biological Chemistry 54, no. 7 (1990): 1739–44. http://dx.doi.org/10.1271/bbb1961.54.1739.

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6

Uzbekov, M., and E. Misionzhnik. "Potential neurobiological ADHD biomarkers." European Psychiatry 33, S1 (2016): S361. http://dx.doi.org/10.1016/j.eurpsy.2016.01.1293.

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ObjectivesPathogenetic mechanisms of hyperkinetic syndrome (HKS) or attention deficit hyperactivity disorder (ADHD) are not clear.AimTo elucidate some aspects of monoamine involvement in pathogenesis of disorder and response of monoaminergic systems to psychostimulant medication.MethodsLevels of different monoamines, their metabolites and N-methylnicotinamide (end product of kynurenine pathway) were measured in daily samples of urine from children (7–11 years old) with mild and severe HKS using fluorimetric and chromatographic methods as well as platelet monoamine oxidase (MAO) activity. Thirt
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7

Martsynkevich, A. M., V. V. Zakharychev, and N. I. Sharanina. "ChemInform Abstract: Synthesis of N,N-Disubstituted (Z)-O-Methylnicotinamide Oximes." ChemInform 43, no. 7 (2012): no. http://dx.doi.org/10.1002/chin.201207133.

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8

Kuchtanin, Vladimír, Jan Moncol, Jerzy Mroziński, et al. "Study of copper(II) thiophenecarboxylate complexes with N-methylnicotinamide." Polyhedron 50, no. 1 (2013): 546–55. http://dx.doi.org/10.1016/j.poly.2012.11.041.

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9

Marín Galvín, R., and J. M. Rodríguez Mellado. "Ec mechanisms: electrodimerization of n-methylnicotinamide on mercury electrode." Electrochimica Acta 33, no. 12 (1988): 1795–96. http://dx.doi.org/10.1016/0013-4686(88)85017-5.

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10

Parsons, Richard B., Shylesh Aravindan, Anusha Kadampeswaran, et al. "The expression of nicotinamide N-methyltransferase increases ATP synthesis and protects SH-SY5Y neuroblastoma cells against the toxicity of Complex I inhibitors." Biochemical Journal 436, no. 1 (2011): 145–55. http://dx.doi.org/10.1042/bj20101685.

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NNMT (nicotinamide N-methyltransferase, E.C. 2.1.1.1) catalyses the N-methylation of nicotinamide to 1-methylnicotinamide. NNMT expression is significantly elevated in a number of cancers, and we have previously demonstrated that NNMT expression is significantly increased in the brains of patients who have died of Parkinson's disease. To investigate the cellular effects of NNMT overexpression, we overexpressed NNMT in the SH-SY5Y cell line, a tumour-derived human dopaminergic neuroblastoma cell line with no endogenous expression of NNMT. NNMT expression significantly decreased SH-SY5Y cell dea
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11

SANO, Akira, Naoto ENDO, and Shoji TAKITANI. "Fluorometric Assay of Rat Brain N-Methyltransferase with 4-Methylnicotinamide." Biological & Pharmaceutical Bulletin 16, no. 3 (1993): 304–6. http://dx.doi.org/10.1248/bpb.16.304.

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12

Jansen, E. H. J. M., and P. De Fluiter. "Determination of N-Methylnicotinamide in Urine with Capillary Zone Electrophoresis." Journal of Liquid Chromatography 17, no. 9 (1994): 1929–39. http://dx.doi.org/10.1080/10826079408013469.

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13

Bujdošová, Zuzana, Juraj Kuchár, and Katarína Györyová. "Diaquabis(5-bromo-2-hydroxybenzoato)bis(N-methylnicotinamide)zinc(II)." Acta Crystallographica Section E Structure Reports Online 66, no. 4 (2010): m394—m395. http://dx.doi.org/10.1107/s1600536810008834.

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14

SHIBATA, Katsumi, Yuko MORI, and Michiko ONODERA. "Nutritional Efficiency of Nicotinamide N-Oxide and N'-Methylnicotinamide as Niacin in Rats." Agricultural and Biological Chemistry 55, no. 10 (1991): 2591–97. http://dx.doi.org/10.1271/bbb1961.55.2591.

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15

Lavelle, Donald, Vinzon Ibanez, Kestis Vaitkus, Yogenthiran Saunthararajah та Robert E. Molokie. "Effect of Nicotinamide, 1-Methylnicotinamide, and N'-Methylnicotinamide on Erythroid Colony Formation and γ-Globin Expression in Cultured Baboon CD34+ Cells". Blood 136, Supplement 1 (2020): 4–5. http://dx.doi.org/10.1182/blood-2020-133974.

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Pharmacological treatments designed to increase Fetal Hemoglobin (HbF) levels offer great promise to alleviate the symptoms and improve the lifespan of the vast numbers of patients afflicted with sickle cell disease (SCD) and β-thalassemia. Hydroxyurea can increase HbF, but a large fraction of patients with SCD do not respond to the drug. DNMT1 and LSD1 inhibitors are the most powerful drugs to increase HbF but are limited by side effects that include neutropenia, thrombophilia and/or thrombocytopenia. The development of new, more effective, and safer pharmacological strategies to augment HbF
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16

Martínez-Aguirre, Mayte A., Jorge M. del Campo, Sigfrido Escalante-Tovar, and Anatoly K. Yatsimirsky. "Self-assembly and recognition properties of a tetraanionic macrocyclic boronate ester in aqueous medium." RSC Advances 5, no. 38 (2015): 30075–83. http://dx.doi.org/10.1039/c5ra03291a.

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A tetraanionic [2 + 2] boronate ester macrocycle is self-assembled in water containing 0–5% vol DMSO and binds efficiently various cationic guests including R<sub>4</sub>N<sup>+</sup> cations, choline, acetylcholine, 1-methylnicotinamide and an alkaloid berberine.
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17

Milani, Zeinab H., David B. Ramsden, and Richard B. Parsons. "Neuroprotective Effects of Nicotinamide N -Methyltransferase and its Metabolite 1-Methylnicotinamide." Journal of Biochemical and Molecular Toxicology 27, no. 9 (2013): 451–56. http://dx.doi.org/10.1002/jbt.21508.

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18

Almeida, Ana R. R. P., Juliana A. S. A. Oliveira, and Manuel J. S. Monte. "Thermodynamic study of nicotinamide, N-methylnicotinamide and N,N-dimethylnicotinamide: Vapour pressures, phase diagrams, and hydrogen bonds." Journal of Chemical Thermodynamics 82 (March 2015): 108–15. http://dx.doi.org/10.1016/j.jct.2014.11.001.

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19

Charman, WN, CSC Lai, DJ Craik, BC Finnin, and BL Reed. "Self-Association of Nicotinamide in Aqueous-Solution: N.M.R. Studies of Nicotinamide and the Mono- and Di-methyl-Substituted Amide Analogs." Australian Journal of Chemistry 46, no. 3 (1993): 377. http://dx.doi.org/10.1071/ch9930377.

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The concentration-dependent self-association of nicotinamide in solution has been studied by 1H and 13C n.m.r. spectroscopy, attendant relaxation time measurements, and osmometric techniques. N-Methylnicotinamide and N,N- dimethylnicotinamide were also studied to evaluate the role of the amide group in the association process. The osmometric studies indicated that the dimethyl -substituted analogue underwent little (if any) self-association, whereas nicotinamide and N- methylnicotinamide did self-associate. The concentration-dependent 1H and 13C chemical shift profiles of nicotinamide and the
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20

Erb, Christina, Albrecht Seidel, Heinz Frank, Karl L. Platt, Franz Oesch, and Jochen Klein. "Formation of N-methylnicotinamide in the brain from a dihydropyridine-type prodrug." Biochemical Pharmacology 57, no. 6 (1999): 681–84. http://dx.doi.org/10.1016/s0006-2952(98)00338-4.

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21

Maiza, Amor, and Peter T. Daley-Yates. "Estimation of the renal clearance of drugs using endogenous N-1-methylnicotinamide." Toxicology Letters 53, no. 1-2 (1990): 231–35. http://dx.doi.org/10.1016/0378-4274(90)90135-9.

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22

Krajníková, Annamária, Róbert Gyepes, and Katarína Győryová. "Crystal Structure of [Zn(2-Bromobenzoato)2] n and [Zn(2-Bromobenzoato)2(N-Methylnicotinamide)]2." Journal of Chemical Crystallography 40, no. 8 (2010): 650–55. http://dx.doi.org/10.1007/s10870-010-9712-z.

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23

Melo, Sandra Soares, Mônica Meirelles, Alceu Afonso Jordão Jr, and Helio Vannucchi. "Lipid Peroxidation in Nicotinamide-Deficient and Nicotinamide-Supplemented Rats." International Journal for Vitamin and Nutrition Research 70, no. 6 (2000): 321–23. http://dx.doi.org/10.1024/0300-9831.70.6.321.

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Supplementation or deficiency of nicotinamide in rats may interfere with the oxidative balance, with excess leading to greater lipid peroxidation, measured by TBARS, and deficiency causing a greater consumption of antioxidants such as vitamin E and glutathione. Urinary N-methylnicotinamide excretion was much more marked in the supplemented group, whereas the difference between deficient and control animals was non-significant.
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24

Bujdošová, Zuzana, Katarína Győryová, Milan Melník, Marian Koman, and Jana Kovářová. "Structures and Characterization of [Zn(n-chlorosalicylato)2(N-methylnicotinamide)2(H2O)2] (n = 4 or 5) Ligand Isomers." Journal of Chemical Crystallography 41, no. 4 (2010): 443–48. http://dx.doi.org/10.1007/s10870-010-9898-0.

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25

Soccorsi, Laura, and Maurizio Cignitti. "On the interactions of catecholamines with N-methylnicotinamide cation: a UV spectral study." European Journal of Medicinal Chemistry 24, no. 3 (1989): 307–8. http://dx.doi.org/10.1016/0223-5234(89)90014-7.

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26

Mateuszuk, Łukasz, Tamara I. Khomich, Ewa Słomińska, et al. "Activation of nicotinamide N-methyltrasferase and increased formation of 1-methylnicotinamide (MNA) in atherosclerosis." Pharmacological Reports 61, no. 1 (2009): 76–85. http://dx.doi.org/10.1016/s1734-1140(09)70009-x.

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27

Zabielska, Magdalena A., Jan Adamus, Robert Kowalski, et al. "Cardioprotective effect of N-methylnicotinamide salt of pyruvate in experimental model of cardiac hypoxia." Pharmacological Reports 70, no. 2 (2018): 378–84. http://dx.doi.org/10.1016/j.pharep.2017.09.011.

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28

Domagala, T. B., M. Celinska-Lowenhoff, J. Dropinski, et al. "Th-P16:302 Fenofibrate increases homocysteine and N-methylnicotinamide levels through PPARalpha-mediated mechanism." Atherosclerosis Supplements 7, no. 3 (2006): 560. http://dx.doi.org/10.1016/s1567-5688(06)82260-9.

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29

Jiang, Nan, Min Wang, Jiayi Song, et al. "N-methylnicotinamide protects against endothelial dysfunction and attenuates atherogenesis in apolipoprotein E-deficient mice." Molecular Nutrition & Food Research 60, no. 7 (2016): 1625–36. http://dx.doi.org/10.1002/mnfr.201501019.

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30

Bi, Hui-Chang, Yu-Zhuo Pan, Jing-Xin Qiu, et al. "N-methylnicotinamide and nicotinamide N-methyltransferase are associated with microRNA-1291-altered pancreatic carcinoma cell metabolome and suppressed tumorigenesis." Carcinogenesis 35, no. 10 (2014): 2264–72. http://dx.doi.org/10.1093/carcin/bgu174.

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31

Moncol, Jan, Marcela Mudra, Peter Lönnecke, et al. "Crystal structures and spectroscopic behavior of monomeric, dimeric and polymeric copper(II) chloroacetate adducts with isonicotinamide, N-methylnicotinamide and N,N-diethylnicotinamide." Inorganica Chimica Acta 360, no. 10 (2007): 3213–25. http://dx.doi.org/10.1016/j.ica.2007.03.027.

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32

Uzbekov, M. "Kynurenine Involvement in Pathogenesis of Hyperkinetic Children Syndrome." European Psychiatry 24, S1 (2009): 1. http://dx.doi.org/10.1016/s0924-9338(09)71225-8.

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Objectives:The aim of the study was to evaluate the response of different monoaminergic systems to psychostimulant treatment in children with mild form of hyperkinetic syndrome (HKS).Methods:The levels of N-methylnicotinamide (end product of kynurenine pathway of tryptophan metabolism), 5-hydroxyindoleacetic (product of serotonin pathway of tryptophan metabolism), homovanillic and vanillylmandelic acids were measured in daily (3 days) urine samples of children (30 patients, 7-11 years old) with mild HKS with normal intellect or borderline mental insufficience. Biochemical measurements were per
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33

Edwards, B. D., A. Maiza, P. T. Daley-Yates, et al. "Altered Clearance of N-1 Methylnicotinamide Associated With the Use of Low Doses of Cyclosporine." American Journal of Kidney Diseases 23, no. 1 (1994): 23–30. http://dx.doi.org/10.1016/s0272-6386(12)80807-9.

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34

Przygodzki, Tomasz, Bartlomiej Grobelski, Piotr Kazmierczak, and Cezary Watala. "N-methyl-2-pyridone-5-carboxamide is 1-methylnicotinamide metabolite of low cyclooxygenase-dependent vasodilating activity." Vascular Pharmacology 56, no. 5-6 (2012): 357. http://dx.doi.org/10.1016/j.vph.2011.08.141.

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35

Domagala, Teresa B., Agata Szeffler, Lawrence W. Dobrucki, et al. "Nitric Oxide Production and Endothelium-Dependent Vasorelaxation Ameliorated by N 1 -Methylnicotinamide in Human Blood Vessels." Hypertension 59, no. 4 (2012): 825–32. http://dx.doi.org/10.1161/hypertensionaha.111.183210.

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36

Maïza, A., S. Waldek, F. W. Ballardie, and P. T. Daley-Yates. "Estimation of Renal Tubular Secretion in Man, in Health and Disease, Using Endogenous N-1-Methylnicotinamide." Nephron 60, no. 1 (1992): 12–16. http://dx.doi.org/10.1159/000186698.

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37

Przygodzki, Tomasz, Bartlomiej Grobelski, Piotr Kazmierczak, and Cezary Watala. "N-Methyl-2-pyridone-5-carboxamide is 1-methylnicotinamide metabolite of low cyclooxygenase-dependent vasodilating activity." Journal of Physiology and Biochemistry 68, no. 3 (2012): 329–34. http://dx.doi.org/10.1007/s13105-012-0144-4.

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38

Sun, Hongqi, Nan Chen, Xuchen Wang, et al. "The Study on the Pathogenesis of Pediatric Lymphoma Based on the Combination of Pseudotargeted and Targeted Metabolomics." BioMed Research International 2021 (May 25, 2021): 1–10. http://dx.doi.org/10.1155/2021/9984357.

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Pediatric lymphoma is a kind of malignant tumor with high mortality. The complexity of pediatric lymphoma shows a great challenge for effective diagnosis and treatment. In order to meet the challenge, the combination of pseudotargeted and targeted metabolomics was used to analyze the serum metabolites in pediatric lymphoma patients and healthy controls for discovering the metabolites related to pediatric lymphoma. The serum samples were obtained from the treatment group ( n = 43 ), the control group ( n = 26 ), and the patients group ( n = 18 ). A total of 17 serum metabolites, including carni
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39

Halaška, Jozef, Ján Lokaj, Klaudia Jomová, Zdeňka Růžičková, Milan Mazúr, and Ján Moncol. "Formation of supramolecular hydrogen-bonding chains and networks from copper (II) halogenobenzoates with N-methylnicotinamide: Supramolecular isomerism." Polyhedron 175 (January 2020): 114237. http://dx.doi.org/10.1016/j.poly.2019.114237.

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40

Sternak, Magdalena, Tamara I. Khomich, Andrzej Jakubowski, et al. "Nicotinamide N-methyltransferase (NNMT) and 1-methylnicotinamide (MNA) in experimental hepatitis induced by concanavalin A in the mouse." Pharmacological Reports 62, no. 3 (2010): 483–93. http://dx.doi.org/10.1016/s1734-1140(10)70304-2.

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41

Przygodzki, Tomasz, Bartlomiej Grobelski, Piotr Kazmierczak, and Cezary Watala. "Erratum to: N-Methyl-2-pyridone-5-carboxamide is 1-methylnicotinamide metabolite of low cyclooxygenase-dependent vasodilating activity." Journal of Physiology and Biochemistry 68, no. 2 (2012): 305. http://dx.doi.org/10.1007/s13105-012-0151-5.

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42

Dołowy, Małgorzata, and Alina Pyka. "Validation of an RPHPTLC-Densitometric Method Using Silica Gel 60 RP18WF254for Simultaneous Determination of Nicotinamide in Selected Pharmaceutical Formulations." Journal of Analytical Methods in Chemistry 2015 (2015): 1–9. http://dx.doi.org/10.1155/2015/631025.

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This research study describes the applicability of silica gel 60 RPW18F254plates for the development and validation of new, simple, economic, accurate, and precise RPHPTLC-densitometric method suitable for the quantification of nicotinamide (asVitamin PP) in three marketed preparations. The mobile phase used was methanol-water in volume composition 3 : 7. Detection wavelength was 200 nm. The proposed method was validated according to ICH guidelines and also based on Ferenczi-Fodor and Konieczka reports. Results were found to be linear over a range of 1.00 to 2.00 μg/spot. Limit of detection (L
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43

McKinney, T. D., M. B. Scheller, M. Hosford, and J. A. McAteer. "Tetraethylammonium transport by OK cells." Journal of the American Society of Nephrology 1, no. 6 (1990): 902–9. http://dx.doi.org/10.1681/asn.v16902.

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Mechanisms exist in renal proximal tubules for the mediated transepithelial secretion or reabsorption of endogenous and exogenous organic cations. In the studies presented here, the uptake of the organic cation tetraethylammonium (TEA) into confluent monolayers of opossum kidney cells was evaluated to determine if these cells might serve as an in vitro model of this transport pathway. 3H-TEA entered opossum kidney cells in a time-dependent manner. Uptake at early time points was saturable with an apparent Km of 59.1 +/- 11.2 microM and a Vmax of 1,292 +/- 210 fmol/micrograms of DNA. TEA uptake
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44

Kaliňáková, Barbora, Daniela Hudecová, Peter Segľa, Martina Palicová, and Jozef Švorec. "Effects of [Cu(2-MeSNic)2(MeNia)2(H2O)2]·H2O and [Cu(2-MeSNic)2(H2O)]2 on Candida albicans." Acta Chimica Slovaca 8, no. 1 (2015): 39–43. http://dx.doi.org/10.1515/acs-2015-0008.

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Abstract Probable mode of action of new copper complexes of 2-methylthionicotinate (2-MeSNic) of composition [Cu(2-MeSNic)2(H2O)2] and [Cu(2-MeSNic)2(MeNia)2(H2O)2]·H2O (where MeNia is N-methylnicotinamide) is described. Both partial growth inhibition of Candida albicans (IC50 ≥ 1.78 mmol·L−1, MIC ≥ 2.5 mmol·L−1) and leak of proteins into the extracellular space (more than 80 %) were observed in the presence of these copper complexes. The membrane damage was detected by staining with Hoechst 33342, propidium iodide and methylene blue. Ascorbic acid potentiated antifungal activity of copper com
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45

Orlando, Rocco, Maura Floreani, Eleonora Napoli, Roberto Padrini, and Pietro Palatini. "Renal Clearance of N1-Methylnicotinamide: A Sensitive Marker of the Severity of Liver Dysfunction in Cirrhosis." Nephron 84, no. 1 (2000): 32–39. http://dx.doi.org/10.1159/000045536.

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46

Pumpo, Rossella, Giovanni Sarnelli, Aldo Spinella, Gabriele Budillon, and Rosario Cuomo. "The Metabolism of Nicotinamide in Human Liver Cirrhosis: A Study on N-Methylnicotinamide and 2-Pyridone-5-Carboxamide Production." American Journal of Gastroenterology 96, no. 4 (2001): 1183–87. http://dx.doi.org/10.1111/j.1572-0241.2001.03698.x.

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47

Thomas, Martin G., Davide Sartini, Monica Emanuelli та ін. "Nicotinamide N-methyltransferase catalyses the N-methylation of the endogenous β-carboline norharman: evidence for a novel detoxification pathway". Biochemical Journal 473, № 19 (2016): 3253–67. http://dx.doi.org/10.1042/bcj20160219.

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Nicotinamide N-methyltransferase (NNMT) is responsible for the N-methylation of nicotinamide to 1-methylnicotinamide. Our recent studies have demonstrated that NNMT regulates cellular processes fundamental to the correct functioning and survival of the cell. It has been proposed that NNMT may possess β-carboline (BC) N-methyltransferase activity, endogenously and exogenously produced pyridine-containing compounds which, when N-methylated, are potent inhibitors of Complex I and have been proposed to have a role in the pathogenesis of Parkinson's disease. We have investigated the ability of reco
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48

Bendayan, Reina, Edward M. Sellers, and Melvin Silverman. "Erratum: Inhibition kinetics of cationic drugs on N′-methylnicotinamide uptake by brush border membrane vesicles from the dog kidney cortex." Canadian Journal of Physiology and Pharmacology 68, no. 11 (1990): 1469–70. http://dx.doi.org/10.1139/y90-222.

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49

Thomas, D. E., K. O. Chung-A-On, J. W. T. Dickerson, S. F. Tidmarsh, and D. M. Shaw. "Tryptophan and nutritional status of patients with senile dementia." Psychological Medicine 16, no. 2 (1986): 297–305. http://dx.doi.org/10.1017/s0033291700009119.

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SynopsisThe nutritional status of 23 severely demented patients was compared with that of 23 similarly aged controls in the community. A 3-day weighed intake on all subjects showed lower mean intakes of energy, protein, ascorbic acid and nicotinic acid in the patient group. This group had lower levels of plasma ascorbic acid and red cell folate and of urinary N-methylnicotinamide excretion relative to creatinine. Over a third of both controls and patients had evidence of thiamin deficiency, as judged by a raised percentage erythrocyte transketolase activity. An earlier finding in patients with
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50

Miller, D. S., and P. D. Holohan. "Organic cation secretion in flounder renal tissue." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 253, no. 6 (1987): R861—R867. http://dx.doi.org/10.1152/ajpregu.1987.253.6.r861.

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Abstract:
In the winter flounder, Pseudopleuronectes americanus, renal clearance experiments showed that the model organic cations, tetraethylammonium (TEA) and N'-methylnicotinamide (NMN), were strongly secreted; organic cation-to-polyethylene glycol (glomerular filtration rate marker) clearance ratios averaged 130 and 30, respectively. TEA uptake by isolated renal tubular masses was concentrative and saturable. Transport was inhibited by competitor organic cations and reduced by exposure to NaCN,2,4-dinitrophenol, ouabain, and HgCl2. Organic anions did not reduce TEA uptake. NMN was the poorest inhibi
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