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Journal articles on the topic 'NAHS'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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1

Coitinho, Juliana, Debora Costa, Samuel Guimaraes, et al. "Structural characterization of Polycyclic Aromatic Hydrocarbon degrading enzymes." Acta Crystallographica Section A Foundations and Advances 70, a1 (2014): C472. http://dx.doi.org/10.1107/s2053273314095278.

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Polycyclic aromatic hydrocarbons (PAHs) are a group of aromatic hydrocarbons composed of two or more fused rings and include naphthalene, phenanthrene, pyrene and benzo[a]pyrene amongst others. Exposure to naphthalene has been associated with several toxic manifestations in humans and laboratory animals, with the lens of the eye and the lungs being most sensitive [1]. Additionally, this compound has been reclassified as a possible human carcinogen by the US Environmental Protection Agency and the International Agency for Research on Cancer. It has recently been suggested that naphthalene under

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2

Jupiter, Ryan C., Daniel Yoo, Edward A. Pankey, et al. "Analysis of erectile responses to H2S donors in the anesthetized rat." American Journal of Physiology-Heart and Circulatory Physiology 309, no. 5 (2015): H835—H843. http://dx.doi.org/10.1152/ajpheart.00293.2015.

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Hydrogen sulfide (H2S) is a biologically active endogenous gasotransmitter formed in penile tissue that has been shown to relax isolated cavernosal smooth muscle. In the present study, erectile responses to the H2S donors sodium sulfide (Na2S) and sodium hydrosulfide (NaHS) were investigated in the anesthetized rat. Intracavernosal injections of Na2S in doses of 0.03–1 mg/kg increased intracavernosal pressure and transiently decreased mean arterial pressure in a dose-dependent manner. Blood pressure responses to Na2S were rapid in onset and short in duration. Responses to Na2S and NaHS were si

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3

NAKATA, Masataka, Masato OSAWA, Naotatsu SHIKAZONO, Hisahide HONMA, and Noriko TSUKAMOTO. "Corrosion of Au-Ag Alloys in NaHS and Na2S Solutions." Journal of the Surface Finishing Society of Japan 45, no. 1 (1994): 98–102. http://dx.doi.org/10.4139/sfj.45.98.

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4

Reátegui-Romero, Warren, Fredy Castillejo-Melgarejo, and María E. King-Santos. "Industrial Manufacturing of Aqueous Solutions of Sodium Sulfhydrate (NaHS 43%) in a Multi-Phase Reactor." Open Chemical Engineering Journal 13, no. 1 (2019): 46–67. http://dx.doi.org/10.2174/1874123101913010046.

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Background: This research deals with the manufacture of sodium sulphides and sodium sulfhydrate in an isothermal multiphase chemical reactor to produce concentrated aqueous solutions of sodium sulfhydrate (greater than 43%) through Gas-Liquid-Solid reactions from hydrogen sulfide and hydroxide of sodium at 50%. Methods: A method is proposed that integrates the recovery of hydrogen sulfide from an industrial chemical process where the H2S gas is generated as a sub product, the strategy of the developed process was integrated into a manufacturing plant of dithiophosphoric acids (ADTF) where it w

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5

Matsuoka, Hidekazu, Kohei Mitsuhashi, Masanobu Kawata, and Chiharu Tokoro. "Derivation of Flotation Kinetic Model for Activated and Depressed Copper Sulfide Minerals." Minerals 10, no. 11 (2020): 1027. http://dx.doi.org/10.3390/min10111027.

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The wettability and floatability of oxidized chalcocite, bornite, and chalcopyrite with the conditions of sodium hydrosulfide (NaHS) dosages and pHs were studied by contact angle measurements, and single and mixture mineral flotation tests. To evaluate the results of the flotation, the flotation kinetic model for copper sulfide minerals treated by NaHS was derived. In this study, we focused on the activation and depression by NaHS, a well-known activator and depressant of copper minerals. The flotation results showed that there can be a threshold NaHS dosage to activate the mineral surfaces, a

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6

Zhao, Lanfu, Yuan Wang, Quan Yan, Wenhai Lv, Yufu Zhang, and Shiming He. "Exogenous hydrogen sulfide exhibits anti-cancer effects though p38 MAPK signaling pathway in C6 glioma cells." Biological Chemistry 396, no. 11 (2015): 1247–53. http://dx.doi.org/10.1515/hsz-2015-0148.

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Abstract It has been documented that H2S, in some types of cancer, promotes tumor proliferation, whereas, in the other types, it inhibits the tumor cell growth. In the present study, we investigated the anti-cancer effects and relevant mechanisms of NaHS in C6 glioma cells. C6 cells were subjected to different concentrations of NaHS, then cell viability and morphological changes were examined by MTT assay and Hoechst staining. The protein expression of Caspase-3, Bcl-2, Bax, p38 MAPK (mitogen-activated protein kinase), and p53 was measured by Western blotting. This work demonstrated that NaHS

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7

Yu, Qian, Zhihong Lu, Lei Tao, et al. "ROS-Dependent Neuroprotective Effects of NaHS in Ischemia Brain Injury Involves the PARP/AIF Pathway." Cellular Physiology and Biochemistry 36, no. 4 (2015): 1539–51. http://dx.doi.org/10.1159/000430317.

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Background/Aims: Stroke is among the top causes of death worldwide. Neuroprotective agents are thus considered as potentially powerful treatment of stroke. Methods: Using both HT22 cells and male Sprague-Dawley rats as in vitro and in vivo models, we investigated the effect of NaHS, an exogenous donor of H2S, on the focal cerebral ischemia-reperfusion (I/R) induced brain injury. Results: Administration of NaHS significantly decreased the brain infarcted area as compared to the I/R group in a dose-dependent manner. Mechanistic studies demonstrated that NaHS-treated rats displayed significant re

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8

Ke, Xiao, Jun Zou, Qingsong Hu, et al. "Hydrogen Sulfide-Preconditioning of Human Endothelial Progenitor Cells Transplantation Improves Re-Endothelialization in Nude Mice with Carotid Artery Injury." Cellular Physiology and Biochemistry 43, no. 1 (2017): 308–19. http://dx.doi.org/10.1159/000480411.

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Background/Aims: The aim of present study was to test the hypothesis that preconditioning with sodium hydrosulfide (NaHS) could enhance the capacity of migration, adhesion and proliferation of endothelial progenitor cells (EPCs) in vitro, and also could improve the efficacy of EPCs transplantation for re-endothelialization in nude mice with carotid artery injury. The paper further addressed the underlying mechanisms. Methods: EPCs were isolated from peripheral blood mononuclear cells of healthy male volunteers and the markers of EPCs were analyzed by flow cytometry. Thereafter, different conce

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9

Zuidema, Mozow Y., Kelly J. Peyton, William P. Fay, William Durante, and Ronald J. Korthuis. "Antecedent hydrogen sulfide elicits an anti-inflammatory phenotype in postischemic murine small intestine: role of heme oxygenase-1." American Journal of Physiology-Heart and Circulatory Physiology 301, no. 3 (2011): H888—H894. http://dx.doi.org/10.1152/ajpheart.00432.2010.

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We recently demonstrated that preconditioning with an exogenous hydrogen sulfide donor (NaHS-PC) 24 h before ischemia and reperfusion (I/R) causes postcapillary venules to shift to an anti-inflammatory phenotype in C57BL/6J wild-type (WT) mice such that these vessels fail to support increases in postischemic leukocyte rolling (LR) and leukocyte adhesion (LA). The objective of the present study was to determine whether heme oxygenase-1 (HO-1) is a mediator of these anti-inflammatory effects noted during I/R in mice preconditioned with NaHS. Intravital fluorescence microscopy was used to visuali

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10

Possomato-Vieira, José Sérgio, Victor Hugo Gonçalves-Rizzi, Regina Aparecida do Nascimento, et al. "Clinical and Experimental Evidences of Hydrogen Sulfide Involvement in Lead-Induced Hypertension." BioMed Research International 2018 (2018): 1–13. http://dx.doi.org/10.1155/2018/4627391.

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Lead- (Pb-) induced hypertension has been shown in humans and experimental animals and cardiovascular effects of hydrogen sulfide (H2S) have been reported previously. However, no studies examined involvement of H2S in Pb-induced hypertension. We found increases in diastolic blood pressure and mean blood pressure in Pb-intoxicated humans followed by diminished H2S plasmatic levels. In order to expand our findings, male Wistar rats were divided into four groups: Saline, Pb, NaHS, and Pb + NaHS. Pb-intoxicated animals received intraperitoneally (i.p.) 1st dose of 8 μg/100 g of Pb acetate and subs

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11

Lin, Jian-qing, Hui-qin Luo, Cai-zhu Lin, Jin-zhuan Chen, and Xian-zhong Lin. "Sodium Hydrosulfide Relieves Neuropathic Pain in Chronic Constriction Injured Rats." Evidence-Based Complementary and Alternative Medicine 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/514898.

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Aberrant neuronal activity in injured peripheral nerves is believed to be an important factor in the development of neuropathic pain (NPP). Channel protein pCREB of that activity has been shown to mitigate the onset of associated molecular events in the nervous system, and sodium hydrosulfide (NaHS) could inhibit the expression of pCREB. However, whether NaHS could relieve the pain, it needs further experimental research. Furthermore, the clinical potential that NaHS was used to relieve pain was limited so it would be required. To address these issues, the rats of sciatic nerve chronic constri

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12

Shi, Ying-Xian, Ying Chen, Yi-Zhun Zhu, et al. "Chronic sodium hydrosulfide treatment decreases medial thickening of intramyocardial coronary arterioles, interstitial fibrosis, and ROS production in spontaneously hypertensive rats." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 4 (2007): H2093—H2100. http://dx.doi.org/10.1152/ajpheart.00088.2007.

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Hydrogen sulfide (H2S) is a gasotransmitter that regulates cardiovascular functions. The present study aimed to examine the hypothesis that chronic treatment with sodium hydrosulfide (NaHS, an H2S donor) is able to prevent left-ventricular remodeling in spontaneously hypertensive rats (SHR). Four-week-old SHR were treated with NaHS (10, 30, and 90 μmol·kg−1·day−1), a combination of NaHS (30 μmol·kg−1·day−1) and glibenclamide (5 mg·kg−1·day−1), glibenclamide alone (5 mg·kg−1·day−1), hydralazine alone (10 mg·kg−1·day−1), and placebo for 3 mo. At the end of the treatment period, variables such as

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13

Wu, Dongdong, Jianmei Li, Qianqian Zhang, et al. "Exogenous Hydrogen Sulfide Regulates the Growth of Human Thyroid Carcinoma Cells." Oxidative Medicine and Cellular Longevity 2019 (May 16, 2019): 1–18. http://dx.doi.org/10.1155/2019/6927298.

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Hydrogen sulfide (H2S) is involved in the development and progression of many types of cancer. However, the effect and mechanism of H2S on the growth of human thyroid carcinoma cells remain unknown. In the present study, we found that the proliferation, viability, migration, and invasion of human thyroid carcinoma cells were enhanced by 25–50 μM NaHS (an H2S donor) and inhibited by 200 μM NaHS. However, H2S showed no obvious effects on the proliferation, viability, and migration of human normal thyroid cells. Administration of 50 μM NaHS increased the expression levels of CBS, SQR, and TST, wh

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14

Bai, ChengPing, and ChenLiang Zhao. "Sodium Hydrosulfide Post-conditioning Protects Hippocampal CA1 Neurons from Neuronal Cell Injury in the Rat Model of Transient Global Cerebral Ischemia Through Activation of Extracellular-regulated Kinases Signaling." Current Neurovascular Research 16, no. 2 (2019): 156–65. http://dx.doi.org/10.2174/1567202616666190618114250.

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Introduction: The effect of hydrogen sulfide (H2S) on global cerebral ischemia remains partially understood. This study aimed to investigate the neuroprotective effect of sodium hydrosulfide (NaHS, a donor of H2S) post-conditioning and its underlying mechanism in a transient global cerebral ischemia (tGCI) model. Materials & Methods: The tGCI rat model was established by the four-vessel occlusion method. Wistar rats were randomly assigned into 6 groups: sham, tGCI, tGCI +NaHS, tGCI+vehicle, tGCI+U0126 and tGCI+U0126+NaHS groups. Neurons survival was assessed by Nissl staining and NeuN immu

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15

Bernardini, Chiara, Debora La Mantia, Salvatore Nesci, et al. "Effects of Hydrogen Sulfide Donor NaHS on Porcine Vascular Wall-Mesenchymal Stem Cells." International Journal of Molecular Sciences 21, no. 15 (2020): 5267. http://dx.doi.org/10.3390/ijms21155267.

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Hydrogen sulfide (H2S) is now considered not only for its toxicity, but also as an endogenously produced gas transmitter with multiple physiological roles, also in maintaining and regulating stem cell physiology. In the present work, we evaluated the effect of a common H2S donor, NaHS, on porcine vascular wall–mesenchymal stem cells (pVW–MSCs). pVW–MSCs were treated for 24 h with increasing doses of NaHS, and the cell viability, cell cycle, and reactive oxygen species (ROS) production were evaluated. Moreover, the long-term effects of NaHS administration on the noteworthy characteristics of pV

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Yusof, Mozow, Kazuhiro Kamada, Theodore Kalogeris, F. Spencer Gaskin, and Ronald J. Korthuis. "Hydrogen sulfide triggers late-phase preconditioning in postischemic small intestine by an NO- and p38 MAPK-dependent mechanism." American Journal of Physiology-Heart and Circulatory Physiology 296, no. 3 (2009): H868—H876. http://dx.doi.org/10.1152/ajpheart.01111.2007.

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Hydrogen sulfide (H2S) is one of three endogenous gases, along with carbon monoxide (CO) and nitric oxide (NO), that exert a variety of important vascular actions in vivo. Although it has been demonstrated that CO or NO can trigger the development of a preconditioned phenotype in postischemic tissues, it is unclear whether H2S may also induce protection in organs subsequently exposed to ischemia-reperfusion (I/R). In light of these observations, we postulated that preconditioning with the exogenous H2S donor sodium hydrosulfide (NaHS-PC) would inhibit leukocyte rolling (LR) and adhesion (LA) i

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Chen, Dan, Yi-Ling Fang, Ling-Ling Zhang, et al. "Hydrogen sulfide ameliorates isoflurane-induced cognitive impairment in mice: Implication of caspase-3 activation." Tropical Journal of Pharmaceutical Research 19, no. 4 (2020): 773–80. http://dx.doi.org/10.4314/tjpr.v19i4.14.

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Purpose: Isoflurane could induce cognitive impairment and activate caspase-3. However, the mechanism of action is unclear and target interventions are unavailable. The present study examined the potential protective function of hydrogen sulfide (H2S) against isoflurane-induced cognitive impairment.Methods: Effects of NaHS (5 mg/kg) on cognitive impairment induced by isoflurane (1.4% for 2 h) were assessed using a fear-conditioning test in a group of 8-month old mice. H4 human neuroglioma cells, which were transfected with upregulated human amyloid precursor protein were treated for 3 or 6 h wi

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Zuidema, Mozow Y., Yan Yang, Meifang Wang, et al. "Antecedent hydrogen sulfide elicits an anti-inflammatory phenotype in postischemic murine small intestine: role of BK channels." American Journal of Physiology-Heart and Circulatory Physiology 299, no. 5 (2010): H1554—H1567. http://dx.doi.org/10.1152/ajpheart.01229.2009.

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The objectives of this study were to determine the role of calcium-activated, small (SK), intermediate (IK), and large (BK) conductance potassium channels in initiating the development of an anti-inflammatory phenotype elicited by preconditioning with an exogenous hydrogen sulfide (H2S) donor, sodium hydrosulfide (NaHS). Intravital microscopy was used to visualize rolling and firmly adherent leukocytes in vessels of the small intestine of mice preconditioned with NaHS (in the absence and presence of SK, IK, and BK channel inhibitors, apamin, TRAM-34, and paxilline, respectively) or SK/IK (NS-3

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Jin, Zhuping, Quanxi Zhang, Eden Wondimu, et al. "H2S-stimulated bioenergetics in chicken erythrocytes and the underlying mechanism." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 319, no. 1 (2020): R69—R78. http://dx.doi.org/10.1152/ajpregu.00348.2019.

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The production of H2S and its effect on bioenergetics in mammalian cells may be evolutionarily preserved. Erythrocytes of birds, but not those of mammals, have a nucleus and mitochondria. In the present study, we report the endogenous production of H2S in chicken erythrocytes, which was mainly catalyzed by 3-mercaptopyruvate sulfur transferase (MST). ATP content of erythrocytes was increased by MST-generated endogenous H2S under normoxic, but not hypoxic, conditions. NaHS, a H2S salt, increased ATP content under normoxic, but not hypoxic, conditions. ATP contents in the absence or presence of

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Liu, Yajun, Theodore Kalogeris, Meifang Wang, et al. "Hydrogen sulfide preconditioning or neutrophil depletion attenuates ischemia-reperfusion-induced mitochondrial dysfunction in rat small intestine." American Journal of Physiology-Gastrointestinal and Liver Physiology 302, no. 1 (2012): G44—G54. http://dx.doi.org/10.1152/ajpgi.00413.2010.

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The objectives of this study were to determine whether neutrophil depletion with anti-neutrophil serum (ANS) or preconditioning with the hydrogen sulfide (H2S) donor NaHS (NaHS-PC) 24 h prior to ischemia-reperfusion (I/R) would prevent postischemic mitochondrial dysfunction in rat intestinal mucosa and, if so, whether calcium-activated, large conductance potassium (BKCa) channels were involved in this protective effect. I/R was induced by 45-min occlusion of the superior mesenteric artery followed by 60-min reperfusion in rats preconditioned with NaHS (NaHS-PC) or a BKCa channel activator (NS-

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Shui, Mengyang, Xiaoyan Liu, Yuanjun Zhu, and Yinye Wang. "Exogenous hydrogen sulfide attenuates cerebral ischemia–reperfusion injury by inhibiting autophagy in mice." Canadian Journal of Physiology and Pharmacology 94, no. 11 (2016): 1187–92. http://dx.doi.org/10.1139/cjpp-2016-0100.

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Hydrogen sulfide (H2S), the third gas transmitter, has been proven to be neuroprotective in cerebral ischemic injury, but whether its effect is mediated by regulating autophagy is not yet clear. The present study was undertaken to explore the underlying mechanisms of exogenous H2S on autophagy regulation in cerebral ischemia. The effects and its connection with autophagy of NaHS, a H2S donor, were observed through neurological deficits and cerebral infarct volume in middle cerebral artery occlusion (MCAO) mice; autophagy-related proteins and autophagy complex levels in the ischemic hemisphere

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Liu, Lulu, Weiwei Gong, Shuping Zhang, et al. "Hydrogen Sulfide Attenuates Angiotensin II-Induced Cardiac Fibroblast Proliferation and Transverse Aortic Constriction-Induced Myocardial Fibrosis through Oxidative Stress Inhibition via Sirtuin 3." Oxidative Medicine and Cellular Longevity 2021 (September 23, 2021): 1–16. http://dx.doi.org/10.1155/2021/9925771.

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Sirtuin 3 (SIRT3) is critical in mitochondrial function and oxidative stress. Our present study investigates whether hydrogen sulfide (H2S) attenuated myocardial fibrosis and explores the possible role of SIRT3 on the protective effects. Neonatal rat cardiac fibroblasts were pretreated with NaHS followed by angiotensin II (Ang II) stimulation. SIRT3 was knocked down with siRNA technology. SIRT3 promoter activity and expression, as well as mitochondrial function, were measured. Male wild-type (WT) and SIRT3 knockout (KO) mice were intraperitoneally injected with NaHS followed by transverse aort

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Majumder, Avisek, Mahavir Singh, Jyotirmaya Behera, et al. "Hydrogen sulfide alleviates hyperhomocysteinemia-mediated skeletal muscle atrophy via mitigation of oxidative and endoplasmic reticulum stress injury." American Journal of Physiology-Cell Physiology 315, no. 5 (2018): C609—C622. http://dx.doi.org/10.1152/ajpcell.00147.2018.

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Although hyperhomocysteinemia (HHcy) occurs because of the deficiency in cystathionine-β-synthase (CBS) causing skeletal muscle dysfunction, it is still unclear whether this effect is mediated through oxidative stress, endoplasmic reticulum (ER) stress, or both. Nevertheless, there is no treatment option available to improve HHcy-mediated muscle injury. Hydrogen sulfide (H2S) is an antioxidant compound, and patients with CBS mutation do not produce H2S. In this study, we hypothesized that H2S mitigates HHcy-induced redox imbalance/ER stress during skeletal muscle atrophy via JNK phosphorylatio

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Zhang, Zhifei, Haixia Huang, Ping Liu, Chaoshu Tang, and Jun Wang. "Hydrogen sulfide contributes to cardioprotection during ischemia–reperfusion injury by opening KATP channels." Canadian Journal of Physiology and Pharmacology 85, no. 12 (2007): 1248–53. http://dx.doi.org/10.1139/y07-120.

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The present study was undertaken to investigate the protective effect of H2S against myocardial ischemia–reperfusion (I/R) injury and its possible mechanism by using isolated heart perfusion and patch clamp recordings. Rat isolated hearts were Langendorff-perfused and subjected to a 30-minute ischemia insult followed by a 30-minute reperfusion. The heart function was assessed by measuring the LVDP, ±dP/dtmax, and the arrhythmia score. The results showed that the treatment of hearts with a H2S donor (40 μmol/L NaHS) during reperfusion resulted in significant improvement in heart function compar

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Zhang, Ying-Xin, Kang-Di Hu, Kai Lv, et al. "The Hydrogen Sulfide Donor NaHS Delays Programmed Cell Death in Barley Aleurone Layers by Acting as an Antioxidant." Oxidative Medicine and Cellular Longevity 2015 (2015): 1–11. http://dx.doi.org/10.1155/2015/714756.

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H2S is a signaling molecule in plants and animals. Here we investigated the effects of H2S on programmed cell death (PCD) in barley (Hordeum vulgareL.) aleurone layers. The H2S donor NaHS significantly delayed PCD in aleurone layers isolated from imbibed embryoless barley grain. NaHS at 0.25 mM effectively reduced the accumulation of superoxide anion (·O2-), hydrogen peroxide (H2O2), and malondialdehyde (MDA), promoted the activity of superoxide dismutase (SOD), guaiacol peroxidase (POD), catalase (CAT), and ascorbate peroxidase (APX), and decreased those of lipoxygenase (LOX) in isolated aleu

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Yoo, Daniel, Ryan C. Jupiter, Edward A. Pankey, et al. "Analysis of cardiovascular responses to the H2S donors Na2S and NaHS in the rat." American Journal of Physiology-Heart and Circulatory Physiology 309, no. 4 (2015): H605—H614. http://dx.doi.org/10.1152/ajpheart.00171.2015.

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Hydrogen sulfide (H2S) is an endogenous gaseous molecule formed from L-cysteine in vascular tissue. In the present study, cardiovascular responses to the H2S donors Na2S and NaHS were investigated in the anesthetized rat. The intravenous injections of Na2S and NaHS 0.03–0.5 mg/kg produced dose-related decreases in systemic arterial pressure and heart rate, and at higher doses decreases in cardiac output, pulmonary arterial pressure, and systemic vascular resistance. H2S infusion studies show that decreases in systemic arterial pressure, heart rate, cardiac output, and systemic vascular resista

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Hsu, Chun-Chun, Ruei-Lung Lin, Lu-Yuan Lee, and You Shuei Lin. "Hydrogen sulfide induces hypersensitivity of rat capsaicin-sensitive lung vagal neurons: role of TRPA1 receptors." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 305, no. 7 (2013): R769—R779. http://dx.doi.org/10.1152/ajpregu.00202.2013.

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The sensitization of capsaicin-sensitive lung vagal (CSLV) afferents by inflammatory mediators is important in the development of airway hypersensitivity. Hydrogen sulfide (H2S) is an endogenous mediator inducing hyperalgesia through transient receptor potential ankyrin 1 (TRPA1) receptors located on nociceptors. We conducted this study to determine whether H2S elevates the sensitivity of rat CSLV afferents. In anesthetized, artificially ventilated rats, the inhalation of aerosolized sodium hydrosulfide (NaHS, a H2S donor) caused no significant changes in the baseline activity of CSLV afferent

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Castillo, Leticia Nunes Carreras Del, Gustavo Leporace, Themis Moura Cardinot, Roger Abramino Levy, and Liszt Palmeira de Oliveira. "Translation, cross-cultural adaptation and validation of the Brazilian version of the Nonarthritic Hip Score." Sao Paulo Medical Journal 131, no. 4 (2013): 244–51. http://dx.doi.org/10.1590/1516-3180.2013.1314487.

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CONTEXT AND OBJECTIVE The Nonarthritic Hip Score (NAHS) is a clinical evaluation questionnaire that was developed in the English language to evaluate hip function in young and physically active patients. The aims of this study were to translate this questionnaire into the Brazilian Portuguese language, to adapt it to Brazilian culture and to validate it. DESIGN AND SETTING Cohort study conducted between 2008 and 2010, at Universidade do Estado do Rio de Janeiro (UERJ). METHODS Questions about physical activities and household chores were modified to better fit Brazilian culture. Reproducibilit

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Ng, Hooi H., Gunes S. Yildiz, Jacqueline M. Ku, Alyson A. Miller, Owen L. Woodman, and Joanne L. Hart. "Chronic NaHS treatment decreases oxidative stress and improves endothelial function in diabetic mice." Diabetes and Vascular Disease Research 14, no. 3 (2017): 246–53. http://dx.doi.org/10.1177/1479164117692766.

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Hydrogen sulphide (H2S) is endogenously produced in vascular tissue and has anti-oxidant and vasoprotective properties. This study investigates whether chronic treatment using the fast H2S donor NaHS could elicit a vasoprotective effect in diabetes. Diabetes was induced in male C57BL6/J mice with streptozotocin (60 mg/kg daily, ip for 2 weeks) and confirmed by elevated blood glucose and glycated haemoglobin levels. Diabetic mice were then treated with NaHS (100 µmol/kg/day) for 4 weeks, and aortae collected for functional and biochemical analyses. In the diabetic group, both endothelium-depend

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Fang, Liping, Hong Li, Chaoshu Tang, Bin Geng, Yongfen Qi, and Xinmin Liu. "Hydrogen sulfide attenuates the pathogenesis of pulmonary fibrosis induced by bleomycin in rats." Canadian Journal of Physiology and Pharmacology 87, no. 7 (2009): 531–38. http://dx.doi.org/10.1139/y09-039.

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The present study investigated the role of the endogenous cystathionine γ-lyase (CSE) / hydrogen sulfide pathway in the pathogenesis of pulmonary fibrosis. Rats treated with intratracheal bleomycin were exposed either to the H2S donor NaHS or to saline. The results on day 7 showed that plasma H2S concentration and pulmonary CSE activity (H2S production rate) were significantly lower in rats treated with bleomycin and saline (fibrosis-alone) than in controls, whereas on day 28 plasma H2S concentration was higher and pulmonary CSE activity was the same as that of controls. The relative CSE mRNA

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Li, Changxia, Guibao Chen, Dengjing Huang, Ni Wang, and Weibiao Liao. "The Antioxidant Defense System during Lanzhou Lily Scales Storage Is Modulated by Hydrogen Sulfide." Horticulturae 7, no. 7 (2021): 183. http://dx.doi.org/10.3390/horticulturae7070183.

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As an important gaseous regulator, hydrogen sulfide (H2S) is involved in various aspects of plant processes, including seed germination, stomatal movement, and postharvest senescence. The preservation capacity of Lanzhou lily (Lilium davidii var. unicolor Salisb) scales fumigated with or without exogenously applied sodium hydrosulfide (NaHS, a H2S donor) was investigated in the current study. Results indicate that NaHS fumigation was able to extend storage life and elicit endogenous H2S production of postharvest Lanzhou lily scales with an optimal concentration at 0.8 mM. Moreover, exogenously

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Domb, Benjamin, Cammille Go, David Maldonado, Sarah Chen, and Ajay Lall. "Predictors of Clinical Outcomes After Hip Arthroscopy: 5 Year Follow- Up Analysis of 1038 Patients." Orthopaedic Journal of Sports Medicine 8, no. 7_suppl6 (2020): 2325967120S0038. http://dx.doi.org/10.1177/2325967120s00385.

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Objectives: Identify predictive factors of midterm outcomes after hip arthroscopy in a cohort of 1038 patients, whose outcomes at minimum 2-year follow-up have previously been reported. In addition, to provide a comparison of short-term and midterm predictive factors in outcome measures following hip arthroscopy. Methods: Data were prospectively collected and retrospectively reviewed on all patients undergoing hip arthroscopy between February 2008 and June 2012. Patients were included if they had minimum 5-year follow-up on 3 patient reported outcomes: Nonarthritic Hip Score (NAHS), modified H

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Wang, Xianli, Qian Wang, Wei Guo, and Yi Zhun Zhu. "Hydrogen sulfide attenuates cardiac dysfunction in a rat model of heart failure: a mechanism through cardiac mitochondrial protection." Bioscience Reports 31, no. 2 (2010): 87–98. http://dx.doi.org/10.1042/bsr20100003.

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HF (heart failure) after MI (myocardial infarction) is a major cause of morbidity and mortality worldwide. Recent studies have shown that hydrogen sulfide (H2S) has cardioprotective effects. Hence, we aimed to elucidate the potential effects of H2S on HF after MI in rats. The HF model after MI was made by ligating the left anterior descending coronary artery. HF groups and sham-operated groups of rats were treated with vehicle, sodium hydrosulfide (NaHS) or PAG (propagylglycine). Equal volumes of saline, 3.136 mg·kg−1·day−1 NaHS or 37.5 mg·kg−1·day−1 PAG, were intraperitoneally injected into r

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Roediger, W. E. W., A. Duncan, O. Kapaniris, and S. Millard. "Sulphide Impairment of Substrate Oxidation in Rat Colonocytes: A Biochemical Basis for Ulcerative Colitis?" Clinical Science 85, no. 5 (1993): 623–27. http://dx.doi.org/10.1042/cs0850623.

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1. Isolated colonic epithelial cells of the rat were incubated for 40 min with [6-14C]glucose and n-[1-14C]batyrate in the presence of 0.1-2.0 mmol/l NaHS, a concentration range found in the human colon. Metabolic products, 14CO2, acetoacetate, β-hydroxybutyrate and lactate, were measured and injury to cells was judged by diminished production of metabolites. 2. Oxidation of n-butyrate to CO2 and acetoacetate was reduced at 0.1 and 0.5 mmol/l NaHS, whereas glucose oxidation remained unimpaired. At 1.0-2.0 mmol/l NaHS, n-butyrate and glucose oxidation were dose-dependently reduced at the same r

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Domb, Benjamin G., Sarah L. Chen, Cammille C. Go, et al. "Predictors of Clinical Outcomes After Hip Arthroscopy: 5-Year Follow-up Analysis of 1038 Patients." American Journal of Sports Medicine 49, no. 1 (2020): 112–20. http://dx.doi.org/10.1177/0363546520968896.

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Background: Although hip arthroscopy has been shown to have favorable results, there is a paucity of literature describing predictive factors of 5-year clinical outcomes. Purpose: To identify predictive factors of midterm outcomes after hip arthroscopy in a cohort of 1038 patients whose outcomes at minimum 2-year follow-up were previously reported. In addition, to provide a comparison of short- and midterm predictive factors in outcome measures after hip arthroscopy. Study Design: Case-control study; Level of evidence, 3. Methods: Data were prospectively collected and retrospectively reviewed

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Xiao, Man, Jun Ma, Hongyu Li, Han Jin, and Hanqing Feng. "Effects of Hydrogen Sulfide on Alternative Pathway Respiration and Induction of Alternative Oxidase Gene Expression in Rice Suspension Cells." Zeitschrift für Naturforschung C 65, no. 7-8 (2010): 463–71. http://dx.doi.org/10.1515/znc-2010-7-808.

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The toxic effects of H2S on plants are well documented. However, the molecular mechanisms reponsible for inhibition of plants by H2S are still not completely understood. We determined the effects of NaHS in the range of 0.5 - 10 mM on the growth of rice suspension culture cells, as well as on the expression of the alternative oxidase (AOX) gene. AOX is the terminal oxidase of the alternative pathway (AP) and exists in plant mitochondria. The results showed that H2S treatment enhanced the AP activity. During the process of H2S treatment for 4 h, the AP activity increased dramatically and achiev

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Wu, Ting, Hua Li, Bing Wu, et al. "Hydrogen Sulfide Reduces Recruitment of CD11b+DG Gr-1+DG Cells in Mice with Myocardial Infarction." Cell Transplantation 26, no. 5 (2017): 753–64. http://dx.doi.org/10.3727/096368917x695029.

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The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H2S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H2S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b+Gr-1+ m

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Kristić, Marija, Miroslav Lisjak, Marija Špoljarević, Tihana Teklić, Sanja Grubišić, and Andrijana Rebekić. "THE INFLUENCE OF HYDROGEN SULFIDE ON WHEATGRASS (Triticum æstivum L.) DE‐ETIOLATION." Poljoprivreda 26, no. 2 (2020): 21–29. http://dx.doi.org/10.18047/poljo.26.2.3.

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Hydrogen sulfide (H2S) is involved in many physiological processes and responses to the abiotic types of stress. The aim of the study was to determine the effect of sodium hydrogen sulfide (NaHS) and the time of application on the physiological properties of etiolated wheatgrass plants. Two genotypes of wheatgrass were grown under controlled conditions for five days without light and then with a 12-hour photoperiod, watered for three consecutive days with 100, 200, and 500 mM NaHS solutions. The plants were watered in three variants, 7-9, 10-12, and 13-15 days after sowing, respectively. The h

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Lim, Jia Jia, Yi-Hong Liu, Ester Sandar Win Khin, and Jin-Song Bian. "Vasoconstrictive effect of hydrogen sulfide involves downregulation of cAMP in vascular smooth muscle cells." American Journal of Physiology-Cell Physiology 295, no. 5 (2008): C1261—C1270. http://dx.doi.org/10.1152/ajpcell.00195.2008.

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Hydrogen sulfide (H2S), a new endogenous mediator, produces both vasorelaxation and vasoconstriction. This study was designed to examine whether cAMP mediates the vasoconstrictive effect of H2S. We found that NaHS at a concentration range of 10–100 μM (yields ∼3–30 μM H2S) concentration-dependently reversed the vasodilation caused by isoprenaline and salbutamol, two β-adrenoceptor agonists, and forskolin, a selective adenylyl cyclase activator, in phenylephrine-precontracted rat aortic rings. Pretreatment with NaHS (10–100 μM) for 5 min also significantly attenuated the vasorelaxant effect of

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Domb, Benjamin G., Timothy J. Martin, Chengcheng Gui, Sivashankar Chandrasekaran, Carlos Suarez-Ahedo, and Parth Lodhia. "Predictors of Clinical Outcomes After Hip Arthroscopy: A Prospective Analysis of 1038 Patients With 2-Year Follow-up." American Journal of Sports Medicine 46, no. 6 (2018): 1324–30. http://dx.doi.org/10.1177/0363546518763362.

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Background: As hip arthroscopy has expanded in popularity and volume, more information is needed about indications for the procedure and the predictive factors of clinical outcomes. Purpose: To evaluate clinical outcomes of hip arthroscopy in a prospective study and to analyze the cohort to identify factors that are predictive of improvement. Study Design: Case-control study; Level of evidence, 3. Methods: Data were collected prospectively on all patients undergoing hip arthroscopy between February 2008 and June 2012. We included all patients undergoing hip arthroscopy who agreed to participat

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Chung, Chi-Li, You Shuei Lin, Nai-Ju Chan, Yueh-Yin Chen, and Chun-Chun Hsu. "Hypersensitivity of Airway Reflexes Induced by Hydrogen Sulfide: Role of TRPA1 Receptors." International Journal of Molecular Sciences 21, no. 11 (2020): 3929. http://dx.doi.org/10.3390/ijms21113929.

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The activation of capsaicin-sensitive lung vagal (CSLV) afferents can elicit airway reflexes. Hypersensitivity of these afferents is known to contribute to the airway hypersensitivity during airway inflammation. Hydrogen sulfide (H2S) has been suggested as a potential therapeutic agent for airway hypersensitivity diseases, such as asthma, because of its relaxing effect on airway smooth muscle and anti-inflammatory effect. However, it is still unknown whether H2S affects airway reflexes. Our previous study demonstrated that exogenous application of H2S sensitized CSLV afferents and enhanced Ca2

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Zhang, Li, Qi Qi, Jianqiang Yang, et al. "An Anticancer Role of Hydrogen Sulfide in Human Gastric Cancer Cells." Oxidative Medicine and Cellular Longevity 2015 (2015): 1–8. http://dx.doi.org/10.1155/2015/636410.

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Hydrogen sulfide (H2S) can be synthesized in mammalian cells by cystathionineγ-lyase (CSE) and/or cystathionineβ-synthase (CBS). Both CSE and CBS are expressed in rat gastric tissues but their role in human gastric neoplasia has been unclear. The aims of the present study were to detect CSE and CBS proteins in human gastric cancer and determine the effect of exogenous NaHS on the proliferation of gastric cancer cells. We found that both CSE and CBS proteins were expressed in human gastric cancer cells and upregulated in human gastric carcinoma mucosa compared with those in noncancerous gastric

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Gelaude, Armance, Sylvain Slaby, Katia Cailliau, et al. "Hydrogen Sulfide Impairs Meiosis Resumption in Xenopus laevis Oocytes." Cells 9, no. 1 (2020): 237. http://dx.doi.org/10.3390/cells9010237.

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The role of hydrogen sulfide (H2S) is addressed in Xenopus laevis oocytes. Three enzymes involved in H2S metabolism, cystathionine β-synthase, cystathionine γ-lyase, and 3-mercaptopyruvate sulfurtransferase, were detected in prophase I and metaphase II-arrested oocytes and drove an acceleration of oocyte meiosis resumption when inhibited. Moreover, meiosis resumption is associated with a significant decrease in endogenous H2S. On another hand, a dose-dependent inhibition was obtained using the H2S donor, NaHS (1 and 5 mM). NaHS impaired translation. NaHS did not induce the dissociation of the

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Hu, Jiangtao, Yali Li, Ya Liu, Dong Il Kang, Hao Wei, and Byoung Ryong Jeong. "Hydrogen Sulfide Affects the Root Development of Strawberry During Plug Transplant Production." Agriculture 10, no. 1 (2020): 12. http://dx.doi.org/10.3390/agriculture10010012.

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Hydrogen sulfide (H2S) is endogenously produced in plant cells and plays an essential role in root development. Given its potential for future agricultural applications, the optimal concentration of sodium hydrosulfide (NaHS, an H2S donor) and the potential mechanisms for root development in the strawberry ‘Seolhyang’ were investigated in this study. The results showed that NaHS with a concentration of 1.250 mM had a positive effect on root development in strawberry. Further experiments showed that exogenous NaHS elevated the H2S content in the root. The dry root weight was increased by the 1.

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Mard, Seyyed Ali, Hasan Askari, Niloofar Neisi, and Ali Veisi. "Antisecretory Effect of Hydrogen Sulfide on Gastric Acid Secretion and the Involvement of Nitric Oxide." BioMed Research International 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/480921.

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The present study was designed to investigate the effect of H2S on distention-induced gastric acid secretion. Fifty-two rats were randomly assigned to seven experimental groups. The gastric acid secretion was stimulated by gastric distention. Two groups of rats received L-cysteine or saline for 5 days before stimulation of the gastric acid secretion. Two groups of animals also received NaHS or saline just prior to stimulation of the gastric acid secretion. The effect of L-NAME and propargylglycine was also investigated. The mucosal levels of the gene expression of cyclooxygenase-2 (COX-2), end

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Zhang, Liping, Yanxi Pei, Hongjiao Wang, et al. "Hydrogen Sulfide Alleviates Cadmium-Induced Cell Death through Restraining ROS Accumulation in Roots ofBrassica rapaL. ssp.pekinensis." Oxidative Medicine and Cellular Longevity 2015 (2015): 1–11. http://dx.doi.org/10.1155/2015/804603.

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Hydrogen sulfide (H2S) is a cell signal molecule produced endogenously and involved in regulation of tolerance to biotic and abiotic stress in plants. In this work, we used molecular biology, physiology, and histochemical methods to investigate the effects of H2S on cadmium- (Cd-) induced cell death in Chinese cabbage roots. Cd stress stimulated a rapid increase of endogenous H2S in roots. Additionally, root length was closely related to the cell death rate. Pretreatment with sodium hydrosulfide (NaHS), a H2S donor, alleviated the growth inhibition caused by Cd in roots—this effect was more pr

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Kang, Minho, Atsushi Hashimoto, Aravind Gade, and Hamid I. Akbarali. "Interaction between hydrogen sulfide-induced sulfhydration and tyrosine nitration in the KATP channel complex." American Journal of Physiology-Gastrointestinal and Liver Physiology 308, no. 6 (2015): G532—G539. http://dx.doi.org/10.1152/ajpgi.00281.2014.

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Hydrogen sulfide (H2S) is an endogenous gaseous mediator affecting many physiological and pathophysiological conditions. Enhanced expression of H2S and reactive nitrogen/oxygen species (RNS/ROS) during inflammation alters cellular excitability via modulation of ion channel function. Sulfhydration of cysteine residues and tyrosine nitration are the posttranslational modifications induced by H2S and RNS, respectively. The objective of this study was to define the interaction between tyrosine nitration and cysteine sulfhydration within the ATP-sensitive K+ (KATP) channel complex, a significant ta

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Zhao, Fusheng, Fang Lei, Xiang Yan, Senfeng Zhang, Wen Wang, and Yu Zheng. "Protective Effects of Hydrogen Sulfide Against Cigarette Smoke Exposure-Induced Placental Oxidative Damage by Alleviating Redox Imbalance via Nrf2 Pathway in Rats." Cellular Physiology and Biochemistry 48, no. 5 (2018): 1815–28. http://dx.doi.org/10.1159/000492504.

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Background/Aims: Cigarette smoke exposure (CSE) during pregnancy is a well-recognized health hazard that causes placental damage. Hydrogen sulfide (H2S) has been reported to protect multiple organs from injury. However, the protective effects of H2S have not been tested in the placenta. This study aimed to explore the potential of H2S in protecting placenta against oxidative injury induced by CSE during pregnancy and the possible underlying mechanisms. Methods: Pregnant SD rats were randomly divided into 4 groups: NaCl, NaHS (a donor of H2S), CSE and CSE+NaHS. Placental oxidative damage was de

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Meghpara, Mitchell B., Rishika Bheem, Marshall Haden, et al. "Differences in Clinical Presentations and Surgical Outcomes of Gluteus Medius Tears Between Men and Women." American Journal of Sports Medicine 48, no. 14 (2020): 3594–602. http://dx.doi.org/10.1177/0363546520966335.

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Background: Gluteus medius (GM) tears often occur in women aged >50 years. There is a paucity of literature comparing sex-based differences in those undergoing GM repair. Purpose: To report differences between women and men in clinical presentations and patient-reported outcome (PRO) scores at a minimum 2-year follow-up after undergoing GM repair. Study Design: Cohort study; Level of evidence, 3. Methods: Data were prospectively collected and retrospectively reviewed. All included patients had postoperative scores for the following PROs: modified Harris Hip Score (mHHS), Non-arthritic Hip S

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Ragy, Merhan M., Fatma F. Ali, and Nisreen D. M. Toni. "Comparing the preventive effect of sodium hydrosulfide, leptin, and curcumin against L-arginine induced acute pancreatitis in rats: role of corticosterone and inducible nitric oxide synthase." Endocrine Regulations 53, no. 4 (2019): 221–30. http://dx.doi.org/10.2478/enr-2019-0022.

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AbstractObjectives. Acute pancreatitis (AP) is a life-threatening condition. Using antioxidants in AP is insufficient and conflicting. Therefore, this study compared the effect of hydrogen sulfide (H2S) donor, sodium hydrosulfide (NaHS), leptin or curcumin pretreatment on AP induced by L-arginine.Methods. Forty adult male rats were used and classified into: 1) control; 2) AP group [each rat was intraperitoneally (i.p.) injected with 2 doses of L-arginine of 250 mg/100 g body weight (b.w.) with an interval of 1 h]; 3) NaHS+AP group (each rat was i.p. injected with 10 mg/kg b.w. of NaHS 1 h befo

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