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Journal articles on the topic 'Nefroida'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

García García, F., L. O. Fontana Compiano, F. Martínez García, et al. "Nefroma multiquístico." Actas Urológicas Españolas 26, no. 3 (2002): 240. http://dx.doi.org/10.1016/s0210-4806(02)72767-8.

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2

Arias Santos, María Dolores, Marta Pavcovich Ruiz, Miguel Andújar Sánchez, David Martínez Lanao, and Laureano León Arencibia. "Nefroma mesoblástico congénito." Revista Española de Patología 39, no. 4 (2006): 243–45. http://dx.doi.org/10.1016/s1699-8855(06)70047-2.

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3

Amico, F., C. Dammino, C. Ninfa, and G. Bartolotta. "Nefroma cistico multiloculare." Urologia Journal 62, no. 4 (1995): 633–34. http://dx.doi.org/10.1177/039156039506200432.

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The authors review two cases of cystic nephroma, highlighting the present preoperative diagnostic possibilities and feasible choices of conservative surgery, even with foci of adenoma and nephroblastoma.
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4

Bahílo Mateu, M. P., A. Budía Alba, F. Boronat Tormo, and F. J. Jiménez-Cruz. "Nefroma multilocular quístico." Actas Urológicas Españolas 34, no. 10 (2010): 921–23. http://dx.doi.org/10.1016/j.acuro.2010.03.036.

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5

Pereda Garay, José. "Nefroma mesoblástico congénito." Revista Peruana de Ginecología y Obstetricia 39, no. 15 (2015): 81–85. http://dx.doi.org/10.31403/rpgo.v39i1593.

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En el niño, el tumor renal más frecuente es el nefroblastoma o tumor de Wilms. Esto no sucede en el recién nacido o en el feto, aunque es cierto que en ellos se puede encontrar lesiones proliferativas que se parecen al tumor de Wilms, tales como la nefroblastomatosis, el nefroblastomas epitelial monomórfico y el nefroma mesoblástico (11) todos los cuales se agrupan bajo la denominación de congéneres infantiles del Wilms. En este grupo, un tumor que tiene características muy definidas, que es pertinente revisar es el llamado nefroma mesoblástico. Se presenta un caso que se observó recientemente
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6

Wilches-Cuadros, María Angélica, Stefania Roca-López, and Luis Miguel Sosa. "Nefronía lobar aguda derecha secundaria a apendicitis aguda no perforada: reporte de caso." Revista Médicas UIS 32, no. 1 (2019): 33–37. http://dx.doi.org/10.18273/revmed.v32n1-2019005.

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La apendicitis aguda en pediatría ocurre generalmente en escolares y adolescentes, siendo su principal complicación el absceso intraabdominal. Por su parte, la nefronía lobar aguda es una infección localizada del parénquima renal, de la cual no existen casos reportados de ésta como secundaria a apendicitis no perforada. Se presenta el caso de unapreadolescente, quien seis días después de una apendicectomía consulta por dolor abdominal, vómito y fiebre. Ingresa con signos de respuesta inflamatoria sistémica, por lo que se inicia terapia antibiótica, y luego de tres días sin mejoría clínica, se
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7

González-Coquel, Suanny Del Carmen, and Emil Julio-Barrios. "Nefronia lobar renal:entidad poco frecuente, diagnosticada en niña de dos años." Revista Ciencias Biomédicas 8, no. 1 (2019): 26–28. http://dx.doi.org/10.32997/rcb-2018-2713.

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Introducción: La nefronia es una infección bacteriana poco frecuente, que se presenta más en niños que en adultos. Afecta a uno o más lóbulos renales. Resulta de una infección de vías urinarias no tratada oportunamente, que evolucionará hasta en aproximadamente el 25 % de los casos a absceso renal.Caso clínico: Preescolar de dos años que presentó dos episodios de infección urinaria en el último mes tratados con antibioticoterapia ambulatoriamente, quien ingresa a urgencias por presentar nuevo episodio febril de cinco días de evolución sin foco aparente; se le practican paraclínicos que evidenc
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8

Senn, E. "Nefrodil-Set: Metallbougies zur perkutanen Nierenfistelung." Aktuelle Urologie 17, no. 01 (1986): 33–34. http://dx.doi.org/10.1055/s-2008-1061636.

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9

Muro, Dolores, Cinta Sangüesa, Sara Brugger, Amparo Moreno, and Victoria Castell. "Manifestaciones radiológicas del nefroma mesoblástico." Radiología 46, no. 2 (2004): 77–82. http://dx.doi.org/10.1016/s0033-8338(04)77941-2.

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10

Lorusso, P., I. Cipollini, A. Bottai, and G. Barsotti. "Sindrome nefrosica steroido-resistente trattata con ACTHh." Giornale di Clinica Nefrologica e Dialisi 22, no. 1 (2018): 5–8. http://dx.doi.org/10.33393/gcnd.2010.1189.

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L'effetto antiproteinurico dell'ACTH è stato recentemente dimostrato nei pazienti con glomerulonefrite membranosa resistenti alla terapia convenzionale. Abbiamo praticato tale terapia in una paziente di 17 anni, giunta alla nostra osservazione con sindrome nefrosica (proteinuria 24h: 10 gr) e funzione renale normale. La paziente, con un quadro istologico di glomerulonefrite proliferativa mesangiale, era già stata sottoposta a terapia steroidea con prednisone 1 mg/kg/die per 3 mesi senza successo. Abbiamo impiegato dosi di ACTH più basse rispetto a quelle riportate in letteratura (1 mg i.m. all
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11

Ventura, Alessandro. "La pagina gialla." Medico e Bambino 40, no. 5 (2021): 283–84. http://dx.doi.org/10.53126/meb40283.

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Vaccinazioni in gravidanza: dalla pertosse al Covid - Chirurgia prenatale: efficace a lungo termine nel mielomeningocele - Microbi efficienti… al posto del pane. Ovvero: come i ricchi pensano al bene dei poveri - Rituximab (e sindrome nefrosica corticodipendente) - Anelli vascolari e anomalie dell’arco aortico
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12

Lorusso, P., I. Cipollini, A. Bottai, and G. Barsotti. "Sindrome nefrosica steroido-resistente trattata con ACTHh." Giornale di Tecniche Nefrologiche e Dialitiche 22, no. 1 (2010): 5–8. http://dx.doi.org/10.1177/039493621002200102.

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13

Gentile, M. G. "Ruolo Della Terapia Dietetica Nella Sindrome Nefrosica." Giornale di Tecniche Nefrologiche e Dialitiche 5, no. 3 (1993): 23–27. http://dx.doi.org/10.1177/039493629300500303.

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14

Frieyro Seguí, M. ªI, M. ªJ Martín Aguado, A. Canals Baeza, J. Molla Nicova, J. Camps Herrero, and F. Segarra Aznar. "Nefronía lobar aguda. Aportación de tres nuevos casos." Anales de Pediatría 55, no. 3 (2001): 269–72. http://dx.doi.org/10.1016/s1695-4033(01)77678-3.

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15

Granados Molina, A., M. Espino Hernández, A. Gancedo Baranda, J. C. Albillos Merino, J. F. Álvarez-Cortinas, and C. Molina Amores. "Nefronía focal aguda bacteriana: diagnóstico, tratamiento y evolución." Anales de Pediatría 66, no. 1 (2007): 84–86. http://dx.doi.org/10.1157/13097366.

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16

Preciado-Estrella, D. A., J. Gomez-Sanchez, J. A. Herrera-Muñoz, et al. "Nefroma quístico: tumor benigno de apariencia maligna." Revista Mexicana de Urología 76, no. 2 (2016): 118–22. http://dx.doi.org/10.1016/j.uromx.2015.11.010.

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17

García Rodríguez, Raquel, Elena Cortés Cros, Margarita Medina Castellano, Luisa Gutiérrez García, and Ángel Zubiría Pineda. "Diagnóstico prenatal de un nefroma mesoblástico congénito." Progresos de Obstetricia y Ginecología 52, no. 1 (2009): 61–66. http://dx.doi.org/10.1016/s0304-5013(09)70146-3.

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18

Aguilar Ruiz, A., A. Torramilans Lluis, M. Castells Esteve, and C. Vargas Blasco. "Nefroma multilocular quístico. presentación de un caso." Actas Urológicas Españolas 27, no. 9 (2003): 718–20. http://dx.doi.org/10.1016/s0210-4806(03)73002-2.

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19

Arze Aimaretti, Lorena, Aixa Reusmann, Mariano Boglione, Marcelo Barrenechea, María Laura Galluzzo Mutti, and Walter Cacciavillano. "Nefroma mesoblastico congénito: reporte de un caso." Gaceta Medica Boliviana 44, no. 1 (2021): 81–85. http://dx.doi.org/10.47993/gmb.v44i1.217.

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El nefroma mesoblastico congénito es un tumor renal infrecuente, pero común en el periodo neonatal. De comportamiento benigno y pronóstico excelente en su variedad clásica pero en el subtipo celular puede presentar recurrencia local, invasión retroperitoneal y metástasis pulmonares y cerebrales. El tratamiento es la nefrectomía radical. Los factores pronósticos son la edad menor a tres meses y la resección quirúrgica completa. Paciente de 17 días nacida a las 35 semanas por polihidramnios, con diagnóstico prenatal de masa abdominal. Al examen físico masa en flanco derecho e hipertensión arteri
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20

Vergara Espitia, Angie Vanessa. "NEFRONIA LOBAR AGUDA EN UN NIÑO. REPORTE DE CASO." Revista de la Facultad de Medicina 63, no. 2 (2015): 321–24. http://dx.doi.org/10.15446/revfacmed.v63n2.47501.

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<p>La nefronía lobar aguda o nefritis aguda focal es una entidad poco frecuente en los niños, se trata de una infección renal con presentación clínica variada e inusual, su diagnóstico se realiza a través de la tomografía axial computarizada TAC, la cual es considerada actualmente la prueba más sensible y especifica. El tratamiento antibiótico adecuado según el germen aislado es la clave para un tratamiento exitoso evitando así la progresión a absceso renal. </p>
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21

Moriconi, Luigi. "Terapia della sindrome nefrosica idiopatica: ruolo delle tecniche aferetiche." Giornale di Clinica Nefrologica e Dialisi 25, no. 4_suppl (2013): S41—S45. http://dx.doi.org/10.33393/gcnd.2013.1090.

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La Sindrome Nefrosica Idiopatica (Idiopathic Nephrotic Syndrome, INS) ricorre essenzialmente in presenza di due glomerulopatie: la MCN (Minimal Change Nephropathy) e la FSGS (Focal Segmental Glomerular Sclerosis). La prima ha un decorso più benigno ed è più frequente nei bambini, mentre la seconda ha un decorso più severo, può portare a Insufficienza Renale Cronica Terminale e può re-cidivare nel trapianto. Soprattutto per la FSGS sono state identificate possibili eziologie virali o genetiche, oltre a forme secondarie in corso di altre malattie, per cui non è semplice classificare queste glome
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22

Santos, Lina Gomes dos, Juliana de Sousa Ribeiro de Carvalho, Marcela Aguiar Reis, and Rayli Lauro Jennyfer Brandão Sales. "Nefroma mesoblástico congênito subtipo celular: relato de caso." Jornal Brasileiro de Nefrologia 33, no. 1 (2011): 109–12. http://dx.doi.org/10.1590/s0101-28002011000100014.

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23

Cuesta Alcalá, J. A., I. Pascual Piédrola, J. Aldave Villanueva, et al. "Nefroma mesoblástico del adulto. características radiológicas y patológicas." Actas Urológicas Españolas 27, no. 7 (2003): 567. http://dx.doi.org/10.1016/s0210-4806(03)72976-3.

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24

Ferrari, P., F. Tavoni, C. A. Pollastri, B. Palmieri, and D. Grassi. "Nefroma Cistico Multiloculare: Considerazioni Radiologiche Su Un Caso." Urologia Journal 53, no. 6 (1986): 957–63. http://dx.doi.org/10.1177/039156038605300626.

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25

Puy, Ramon. "Casos en imagen 2.—Nefroma mesoblástico (tumor de bolande)." Radiología 46, no. 2 (2004): 120. http://dx.doi.org/10.1016/s0033-8338(04)77949-7.

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26

Paniza, M. Angeles, Enrique Poblet, Beatriz Ocaña, Jose Maria Jimenez, and Francisco Pastor. "Nefroma quístico y angiomiolipoma renal sincrónicos unilaterales: asociación inusual." Revista Española de Patología 48, no. 4 (2015): 245–49. http://dx.doi.org/10.1016/j.patol.2015.04.007.

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27

Bielsa-Gracia, Sara, Luis Miguel Lou, José Antonio Gimeno, Olga Gracia-García, Ignacio López-Alejaldre, and Elvira Fernández. "Hypertriglyceridaemic waist phenotype for Chronic Kidney Disease population: NEFRONA cohort." Nefrología (English Edition) 40, no. 5 (2020): 514–21. http://dx.doi.org/10.1016/j.nefroe.2019.12.007.

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28

POLAT, Mesut, Resul ARISOY, Emre ERDOĞDU, A. Doğukan ANGIN, and Ahmet Semih TUĞRUL. "KONJENİTAL MEZOBLASTİK NEFROMA : OLGU SUNUMU [Congenital Mesoblastic Nephroma: Case Report]." Zeynep Kamil Tıp Bülteni 45, no. 1 (2014): 22. http://dx.doi.org/10.16948/zktb.40317.

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29

Jiménez-Vázquez, I., A. R. Aragón-Tovar, J. C. Sánchez-Venegas, et al. "Nefroma mesoblástico del adulto. Un nuevo caso de un tumor agresivo." Revista Mexicana de Urología 76, no. 3 (2016): 194–98. http://dx.doi.org/10.1016/j.uromx.2016.02.003.

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30

Gracia, Marta, Montserrat Martínez, Àngels Betriu, et al. "FP373FACTORS PREDICTING SUBCLINICAL ATHEROMATOSIS PROGRESSION IN CHRONIC KIDNEY DISEASE. THE NEFRONA STUDY." Nephrology Dialysis Transplantation 30, suppl_3 (2015): iii193. http://dx.doi.org/10.1093/ndt/gfv175.55.

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31

Arroyo, David, María Abajo, Marta Gracia, Àngels Betriu, Jose M. Valdivielso, and Elvira Fernández. "SP357PROGRESSION OF PERIPHERAL ARTERY DISEASE IN CHRONIC RENAL IMPAIRMENT: THE NEFRONA PROJECT." Nephrology Dialysis Transplantation 30, suppl_3 (2015): iii497. http://dx.doi.org/10.1093/ndt/gfv192.23.

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32

Blanco Parra, M. A., J. M. Suárez Peñaranda, D. A. Pérez Fentes, and A. Puñal Pereira. "Nefroma multilocular quístico. Nuevo caso y revisión de la literatura médica reciente." Actas Urológicas Españolas 34, no. 3 (2010): 301–3. http://dx.doi.org/10.1016/j.acuro.2010.01.016.

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33

Travasoni, Francesca, Mariangela Annoloro, Alessia Bortot, Stefano Cantelli, Giorgia Russo, and Yuri Battaglia. "Amiloidosi e Glomerulonefrite Extracapillare: un raro caso clinico di overlap." Giornale di Clinica Nefrologica e Dialisi 25, no. 4 (2013): 310–14. http://dx.doi.org/10.33393/gcnd.2013.1063.

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L'amiloidosi è una malattia caratterizzata dalla deposizione in sede extracellulare di materiale di natura proteica detto amiloide. Questa malattia sistemica può essere sia primitiva AL che secondaria AA. Il coinvolgimento renale è un'evenienza frequente e, occasionalmente, si assiste a un'insufficienza renale rapidamente progressiva sostenuta dalla sovrapposizione di una glomerulonefrite extracapillare. I rari casi riportati in letteratura sono correlati a un'amiloi-dosi secondaria all'artrite reumatoide. Si riporta il caso clinico di una donna di 46 anni tabagista, affetta da ipotiroidismo s
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34

Bielsa-Gracia, Sara, Luis Miguel Lou, José Antonio Gimeno, Olga Gracia-García, Ignacio López-Alejaldre, and Elvira Fernández. "Fenotipo de Cintura Hipertrigliceridémica en la población con Enfermedad Renal Crónica. Cohorte NEFRONA." Nefrología 40, no. 5 (2020): 514–21. http://dx.doi.org/10.1016/j.nefro.2019.12.005.

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35

Monica, B., A. Barbieri, G. P. Incarbone, E. Cudazzo, P. P. Puviani, and M. Franzini. "Su di un caso di carcinoma a cellule chiare in nefroma cistico multiloculare." Urologia Journal 64, no. 1_suppl (1997): 47–49. http://dx.doi.org/10.1177/039156039706401s11.

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A case of adenocarcinoma of the kidney in a multilocular cystic disease is reported. The many diagnostic pitfalls of this rare association are underlined and the diagnostic role of selective renal artery angiography, which removed all doubt about the lesion.
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36

Cambray, Serafi, Marcelino Bermudez-Lopez, Milica Bozic, and Jose M. Valdivielso. "Association of a single nucleotide polymorphism combination pattern of the Klotho gene with non-cardiovascular death in patients with chronic kidney disease." Clinical Kidney Journal 13, no. 6 (2020): 1017–24. http://dx.doi.org/10.1093/ckj/sfaa014.

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Abstract Background Chronic kidney disease (CKD) is associated with an elevated risk of all-cause mortality, with cardiovascular death being extensively investigated. However, non-cardiovascular mortality represents the biggest percentage, showing an evident increase in recent years. Klotho is a gene highly expressed in the kidney, with a clear influence on lifespan. Low levels of Klotho have been linked to CKD progression and adverse outcomes. Single nucleotide polymorphisms (SNPs) of the Klotho gene have been associated with several diseases, but studies investigating the association of Klot
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37

Hoşağası, Nihan Hilal, Dilek Dilli, İsmet Faruk Özgüner, et al. "Yenidoğan Döneminde Nöron Spesifik Enolaz Yüksekliği ile Seyreden Atipik (Selüler) Konjenital Mezoblastik Nefroma Olgusu." Haseki Tıp Bülteni 52, no. 3 (2014): 223–26. http://dx.doi.org/10.4274/haseki.1577.

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38

Arroyo, David, María Abajo, Marta Gracia, Àngels Betriu, Jose M. Valdivielso, and Elvira Fernández. "FP359SUBCLINICAL PERIPHERAL ARTERY DISEASE PREDICTS CARDIOVASCULAR EVENTS IN CHRONIC RENAL IMPAIRMENT: THE NEFRONA PROJECT." Nephrology Dialysis Transplantation 30, suppl_3 (2015): iii188—iii189. http://dx.doi.org/10.1093/ndt/gfv175.41.

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39

Gracia, M., J. M. Valdivielso, A. Betriu, M. Abajo, D. Arroyo, and E. D. Fernández. "Subclinical atheromatosis progression and associated risk factors in chronic kidney disease. The nefrona study." Atherosclerosis 241, no. 1 (2015): e14-e15. http://dx.doi.org/10.1016/j.atherosclerosis.2015.04.067.

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40

Betriu, A., M. Martinez-Alonso, M. V. Arcidiacono, et al. "Prevalence of subclinical atheromatosis and associated risk factors in chronic kidney disease: the NEFRONA study." Nephrology Dialysis Transplantation 29, no. 7 (2014): 1415–22. http://dx.doi.org/10.1093/ndt/gfu038.

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41

Piñera, Cecilia, Francisca Loyola, and Pamela Hernández. "Nefronia en pediatría: dentro del espectro de las infecciones urinarias. Serie clínica y revisión de la literatura." Revista chilena de infectología 32, no. 5 (2015): 564–68. http://dx.doi.org/10.4067/s0716-10182015000600011.

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42

Martínez Fernández, Lidia, J. Emilio Sánchez-Alvarez, César Morís de la Tassa, et al. "Risk factors associated with valvular calcification in patients with chronic kidney disease. Analysis of NEFRONA study." Nefrología (English Edition) 41, no. 3 (2021): 337–46. http://dx.doi.org/10.1016/j.nefroe.2021.08.002.

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43

Abajo, M., D. Arroyo, M. Gracia, À. Betriu, J. M. Valdivielso, and E. Fernández. "FP353RISK FACTORS FOR PROGRESSION OF COMMON INTIMA − THICKNESS IN INDIVIDUALS WITH CHRONIC KIDNEY DISEASE: THE NEFRONA STUDY." Nephrology Dialysis Transplantation 30, suppl_3 (2015): iii186. http://dx.doi.org/10.1093/ndt/gfv175.35.

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44

Bermudez-Lopez, M., C. Fornè, S. Cambray, M. Bozic, and J. M. Valdivielso. "PUK17 Outcomes and Healthcare Resources Utilization Differences in Patients with and without Secondary Hyperparathyroidism from the Nefrona Cohort." Value in Health 23 (December 2020): S753. http://dx.doi.org/10.1016/j.jval.2020.08.2060.

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45

Cambray, Serafi, Merce Ibarz, Marcelino Bermudez-Lopez, et al. "Magnesium Levels Modify the Effect of Lipid Parameters on Carotid Intima Media Thickness." Nutrients 12, no. 9 (2020): 2631. http://dx.doi.org/10.3390/nu12092631.

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Classical risk factors of atherosclerosis in the general population show paradoxical effects in chronic kidney disease (CKD) patients. Thus, low low-density lipoprotein (LDL) cholesterol levels have been associated with worse cardiovascular outcomes. Magnesium (Mg) is a divalent cation whose homeostasis is altered in CKD. Furthermore, Mg levels have been associated with cardiovascular health. The present study aims to understand the relationships of Mg and lipid parameters with atherosclerosis in CKD. In this analysis, 1754 participants from the Observatorio Nacional de Atherosclerosis en Nefr
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46

Valdivielso, José M., Milica Bozic, Rajesh Kumar Galimudi, et al. "Association of the rs495392 Klotho polymorphism with atheromatosis progression in patients with chronic kidney disease." Nephrology Dialysis Transplantation 34, no. 12 (2018): 2079–88. http://dx.doi.org/10.1093/ndt/gfy207.

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Abstract Background Prevalence of atherosclerotic cardiovascular disease and its rate of progression are higher in patients with chronic kidney disease (CKD) compared with the general population. Mineral metabolism parameters have been shown to be involved in the increased velocity of atheromatosis progression. The aim of this study is to determine the role of 11 single-nucleotide polymorphisms (SNPs) of the Klotho gene on the rate of atherosclerosis progression in CKD. Methods This was a multicentre, prospective, observational study of 1439 CKD patients from the NEFRONA cohort. Carotid and fe
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47

Gracia, M., À. Betriu, M. Martínez, et al. "Klotho gene polymorphisms are associated with progression of atheromatosis in patients with CKD. Results of two years follow-up of the NEFRONA cohort." Atherosclerosis 252 (September 2016): e17. http://dx.doi.org/10.1016/j.atherosclerosis.2016.07.284.

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48

Gracia, Marta, Àngels Betriu, Montserrat Martínez, et al. "MP178KLOTHO GENE POLYMORPHISMS ARE ASSOCIATED WITH PROGRESSION OF ATHEROMATOSIS IN PATIENTS WITH CKD. RESULTS OF TWO YEARS FOLLOW-UP OF THE NEFRONA COHORT." Nephrology Dialysis Transplantation 32, suppl_3 (2017): iii493. http://dx.doi.org/10.1093/ndt/gfx164.mp178.

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49

Cambray, Serafí, Rajesh Kumar Galimudi, Milica Bozic, Marcelino Bermúdez-López, Isabel Rodríguez, and José M. Valdivielso. "The rs1126616 Single Nucleotide Polymorphism of the Osteopontin Gene Is Independently Associated with Cardiovascular Events in a Chronic Kidney Disease Cohort." Journal of Clinical Medicine 8, no. 5 (2019): 592. http://dx.doi.org/10.3390/jcm8050592.

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Abstract:
Chronic kidney disease (CKD) is associated with a higher risk of cardiovascular events (CVE), partly due to the higher burden of atherosclerosis. Circulating Osteopontin (OPN) levels have been also shown to have a potential role in the development of atherosclerosis. Indeed, CKD patients show an increase in circulating OPN levels, but their effect of CKD-related atherosclerosis is not clear. Polymorphisms in the OPN gene (SPP1) have been studied in atheromatous disease, but reported results show conflictive findings. Thus, the main aim of the present study is to analyze the influence of SPP1 p
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50

Valdivielso, José M., Diego Rodríguez-Puyol, Julio Pascual, et al. "Atherosclerosis in Chronic Kidney Disease." Arteriosclerosis, Thrombosis, and Vascular Biology 39, no. 10 (2019): 1938–66. http://dx.doi.org/10.1161/atvbaha.119.312705.

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Abstract:
Patients with chronic kidney disease (CKD) are at an increased risk of premature mortality, mainly from cardiovascular causes. The association between CKD on hemodialysis and accelerated atherosclerosis was described >40 years ago. However, more recently, it has been suggested that the increase in atherosclerosis risk is actually observed in early CKD stages, remaining stable thereafter. In this regard, interventions targeting the pathogenesis of atherosclerosis, such as statins, successful in the general population, have failed to benefit patients with very advanced CKD. This raises the is
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