Academic literature on the topic 'Neisseria gonorrhϾ'

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Journal articles on the topic "Neisseria gonorrhϾ"

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Tabize Olivier, Mutendela, Freddy Munyololo Muganza, Leshweni Jeremia Shai, and Stanley Sechene Gololo. "Rutin Inhibits F, G, N and O gonorrhea strains, 2008 WHO N-gonorrhea Reference strains, in vitro." Interdisciplinary Research Journal and Archives 1 (December 16, 2020): 41–49. http://dx.doi.org/10.36966/irjar2020.13.

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Rutin was isolated from methanol extract of the aerial part of Asparagus suaveolens using precipitation method. South Africans use Asparagus suaveolens to treat gonorrhea infections. The obtained Nuclear Magnetic Resonance (NMR) and Liquid Chromatography-Mass Spectroscopy (LC-MS) data and visiting the published data on the isolation of rutin confirmed the structure. The 2008 WHO Neisseria gonorrhea reference strains were used to evaluate microbial activity of rutin against the gonorrhea strains. Rutin found to be bacteriostatic against WHO 2008 Neisseria gonorrhoea F, G, N and O strains with the minimum inhibition concentration of 0.40, 0.65, 0.22 and 0.65 mg/ml, respectively. In addition, rutin fare better than the reference drugs and bactericidal against K, L, M, and P strains. These results support the traditional use of Asparagus suaveolens against gonorrhea infections by South African indigenous people. To our knowledge, this is the first study indicating the activity of rutin against N.gonorrhea strains. Résumé: La rutine a été isolée à partir d'un extrait au méthanol de la partie aérienne d'Asparagus suaveolens en utilisant la méthode de précipitation. Les SudAfricains utilisent Asparagus suaveolens pour traiter les infections gonorrhées. Les données obtenues par résonance magnétique nucléaire (RMN) et par chromatographie liquide-spectroscopie de masse (LCMS) et la consultation des données publiées sur l'isolement de la rutine ont confirmé la structure. Les souches de référence OMS de Neisseria gonorrhea de 2008 ont été utilisées pour évaluer l'activité microbienne de la rutine contre les souches de gonorrhée. La rutine s'est révélée bactériostatique contre les souches de Neisseria gonorrhea F, G, N et O de l'OMS 2008 avec une concentration minimale d'inhibition de 0,40, 0,65, 0,22 et 0,65 mg/ml, respectivement. De plus, la rutine se porte mieux que les médicaments de référence et bactéricide contre les souches K, L, M et P. Ces résultats soutiennent l'utilisation traditionnelle d'Asparagus suaveolens contre les infections gonorrhées par les populations autochtones sud-africaines. À notre connaissance, il s'agit de la première étude indiquant l'activité de la rutine contre les souches de N. gonorrhée.
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DE, Bitet. "Neisseria Gonorrheae: A Cause of Male Infertility." Open Access Journal of Microbiology & Biotechnology 6, no. 1 (2021): 1–7. http://dx.doi.org/10.23880/oajmb-16000183.

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Background: Gonorrhoea is a sexually transmitted infection that is commonly related to male infertility. The infection affects sperm transport through the urinary tract and subsequent damage of the testicular tubes. The infection also impair sperm production as the infection is rarely asymptomatic and can be difficult to diagnose, it is possible that its contribution to male infertility is underestimated. Infection of the genitals results in a purulent (pus-like) discharge from the genitals which may be foul smelling, inflammation, redness, swelling, dysuria, and a burning sensation during urination. As with Chlamydia, it is possible to have a Gonorrhea infection without noticeable symptoms, and which can cause permanent scarring and blockage in the sperm production duct. Gonorrhoea is a bacterial infection and is treatable with antibiotics. Laboratory studies reveal that N. gonorrhoeae infection can impair motility, viability and spermatogenesis; increase anti-sperm antibodies are associated with a decrease in semen parameters as a result of the production of anti-sperm antibodies in the genital tract. Aim: The study aimed at reviewing the possible role of gonorrhoea in male infertility. Method: Research publications such as Pubmed, Scopus, Medline etc. Results: over 115 journals of international repute were obtained, out of which 50 were found to be closely relevant such as; the implication of N. gonorrheae in male infertility, complication of N. gonorrhea infection, N. gonorrhea e and male infertility and were reviewed. Conclusion: N. gonorrhoae was found implicative in male infertility and the need for comprehensive modern laboratory methods for the diagnosis of the infection and also to included routine laboratory tests.
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Suay-García, Beatriz, and María-Teresa Pérez-Gracia. "Neisseria gonorrhoeae Infections." Pathogens 9, no. 8 (August 12, 2020): 647. http://dx.doi.org/10.3390/pathogens9080647.

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Gonorrhea is a sexually transmitted disease with a high morbidity burden. Despite having guidelines for its treatment, the incidence of the disease follows an increasing trend worldwide. This is mainly due to the appearance of antibiotic-resistant strains, inefficient diagnostic methods and poor sexual education. Without an effective vaccine available, the key priorities for the control of the disease include sexual education, contact notification, epidemiological surveillance, diagnosis and effective antibiotic treatment. This Special Issue focuses on some of these important issues such as the molecular mechanisms of the disease, diagnostic tests and different treatment strategies to combat gonorrhea.
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Burhannuddin, Burhannuddin. "IDENTIFIKASI DAN UJI SENSITIVITAS BAKTERI Neisseria gonorrhoeae TERHADAP ANTIBIOTIK SEFIKSIM PADA PEKERJA SEKS KOMERSIAL DI PUSKESMAS II DENPASAR SELATAN." Meditory : The Journal of Medical Laboratory 9, no. 1 (June 25, 2021): 1–11. http://dx.doi.org/10.33992/m.v9i1.1306.

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Gonorrhea is a sexually transmitted disease caused by Neisseria gonorrhoeae. Gonorrhea infection varies greatly in the community, especially in sexually active women. Infection cases in women are often asymptomatic, that can causes complication can easily occur. The aims of this study are to identify and determine sensitivity of Neisseria gonorrhoeae bacteria against cefixime antibiotics. This study was using a descriptive method and used 30 samples. Neisseria gonorrhoeae was isolatd from the vaginal swab sample of Commercial Sex Workers at Puskesmas II Denpasar Selatan. The cultured bacteria on Thayer Martin media were then identified by gram staining, oxidase test, and catalase test. Neisseria gonorrhoeae sensitivity test against cefixime was performed by disc diffusion method. The results of identification showed that 6 (20 %) of 30 samples were Neisseria gonorrhoeae bacteria with cell characteristics in the form of diplococcus, gram-negative, oxidase and catalase positive. The results of the sensitivity test showed that all isolats of Neisseria gonorrhoeae were resistant to cefixime
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Setyowatie, Lita, Tantari SHW, and Inneke Yulian. "Susceptibility Pattern of Neisseria gonorrhoeae towards Cefixime and Ceftriaxone using Kirby-Bauer Method in Dr. Saiful Anwar General Hospital Malang." Berkala Ilmu Kesehatan Kulit dan Kelamin 32, no. 2 (July 31, 2020): 103. http://dx.doi.org/10.20473/bikk.v32.2.2020.103-110.

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Background: Gonorrhea remains the second most common sexually transmitted infection (STI) in the world with an increasing number of cases. Oral cefixime and IM ceftriaxone are still the mainstay therapy for gonorrhea in Indonesia. However, previous studies suggested possible resistance to ceftriaxone and cefixime, which are the first-line treatment of gonorrhea. To date, there are no data available regarding the susceptibility of these antibiotics for the treatment of gonorrhea in Dr. Saiful Anwar General Hospital (RSSA) Malang. Purpose: Determine susceptibility pattern of Neisseria gonorrhoeaetowards cefixime and ceftriaxone in RSSA Malang. Methods: The samples were patients of the Outpatient Clinic of Dermatology and Venereology Department with a symptom of discharge which contained Gram-negative diplococcus after Gram staining and had a positive culture of Neisseria gonorrhoeae. Susceptibility testing for cefixime and ceftriaxone antibiotics were performed using the Kirby-Bauer method. The data are presented in percentages. Result: Antibiotic susceptibility test results showed that 80.77% of Neisseria gonorrhoeaeisolates were still susceptible to cefixime, and 80.77% of isolates were still susceptible to ceftriaxone. Conclusion: Cefixime and ceftriaxone are still effective as gonorrhea therapy in RSSA Malang.
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Nepal, Anand, Kapil Subedi, Jitendra Shah, and Manoj Koirala. "Clinical and Bacteriological Profile of Male Patients with Urethral Discharge at Western Regional Hospital, Nepal." Medical Journal of Pokhara Academy of Health Sciences 2, no. 2 (December 31, 2019): 178–82. http://dx.doi.org/10.3126/mjpahs.v2i2.28088.

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Background: Urethral discharge is a symptom of urethritis. Common organisms causing urethral discharge are Neisseria gonorrhea and Chlamydia trachomatis. Cephalosporines, Macrolides and Tetracycline groups of drugs are commonly used for the treatment. Materials and Methods: This was a prospective, cross sectional, observational study. The patients were enrolled from the Sexually Transmitted Infections clinic and also from the hospital laboratory in between the period of one year. All the male cases with urethral discharge at STI clinic in between this period were included for clinical examination and laboratory testing. All urine and discharge specimens of urethral discharge patients as referred from other departments directly to the hospital laboratory for relevant tests were also included for the study. Results: Neisseria gonorrhea was the most common agent of urethral discharge in male patients. Urethral discharge, dysuria and inguinal lymphadenopathy were common symptoms. All the Neisseria gonorrhea isolates were sensitive to Ceftriaxone and Imipenam. Conclusion: Continuous monitoring with culture sensitivity testing of urethral discharge cases is required.
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Isa Adamu, Ibrahim, and Sulaiman Usman. "Mathematical Model for the Dynamics of Neisseria Gonorrhea Disease with Natural Immunity and Treatment Effects." Journal of Mathematics Research 10, no. 2 (March 29, 2018): 151. http://dx.doi.org/10.5539/jmr.v10n2p151.

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Neisseria gonorrhea infection; a sexually transmitted disease, is caused primarily by a type of germ; a bacteria called neisseria gonorrhea. The infection is a major public health challenge today due to the high incidence of infections accompanied by a dwindling number of treatment options especially in developing and underdeveloped countries. In this paper, we developed a mathematical model for the transmission dynamics of neisseria gonorrhea infection and studied the effect of natural immunity and treatment as the only available control interventions on the spread of the disease in a population. We computed the model disease-free equilibrium and analyzed its local and global stability in a well-defined positively invariant and attracting set Ω using the next-generation matrix plus linearization method and the comparison theorem respectively. The disease-free equilibrium was proved to be both locally and globally asymptotically stable if $R_0<1$ and unstable if $R_0>1$. We conducted sensitivity analysis of parameters in the basic reproduction number $R_0$ using the normalized forward sensitivity index method. Results of the analysis revealed that $R_0$ decreases with increase in treatment and natural immunity rates. The results of the numerical simulations carried out using MATLAB R2012B showed that there is increase in new infections due to increased contact with infected individuals in the susceptible population and that, with increased treatment rate and controlled death due to the disease in the population, neisseria gonorrhea infection would be wiped out within 300 days of the treatment intervention.
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Nadir qızı Kərimova, Sevinc. "Gonorrhea in women." NATURE AND SCIENCE 07, no. 02 (April 23, 2021): 16–18. http://dx.doi.org/10.36719/2707-1146/07/16-18.

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Gonorrhea is a sexually transmitted infection. It is caused by gonococci of the genus Neisseria. The literal translation of the disease means "semen flow". Hygienic rules must be followed. Treatment must be carried out correctly. Key words: gonorrhea, preqnancy, gonococcus
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Hamid, Hamid, Dirk Runtuboi, and Lucky V. Waworuntu. "Uji Sensitivitas Neisseria gonorrhoeae terhadap Beberapa Antibiotik Pada Wanita Penjaja Seks (WPS) di Lokalisasi Tanjung Elmo Kabupaten Jayapura." JURNAL BIOLOGI PAPUA 6, no. 2 (August 1, 2018): 60–69. http://dx.doi.org/10.31957/jbp.460.

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The purpose of this research is to study the sensitivity of quinolon antibiotic group against Neisseria gonorrhea. The bacteria which were found from endoservic vaginal from local sexual worker of Tanjung Elmo, Jayapura were isolated using neck duck sterile. This study was done on Health Laboratory of Jayapura from June to September 2013. The swab was planted on Thayer-Martin medium and incubated at 37 ºC for 2 x 24 hours. The grown colonies were sub-cultured to get the specific colonies. The bacterial identification is done by some rapid test including examination of grams, oxidation tests, biochemical tests andsensitivitytest. The result showed that there were six (6) specimen of 52 samples positively contained N. gonorrhea. The sensitivity test of quinolone group indicated that the levofloxacin, cefixime, ciprofoxacin, ofloxacin, azytromisin have the sensivity of 83.3%, 66.6%, 66.6%, 66.6% and 50% respectively.Key words: Neisseria gonorrhea, female commercial sex workers, antibiotics
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Queirós, Catarina Soares, and João Borges da Costa. "Gonorrhea: Antimicrobial Resistance and New Drugs." Journal of the Portuguese Society of Dermatology and Venereology 77, no. 3 (October 10, 2019): 233–38. http://dx.doi.org/10.29021/spdv.77.3.1089.

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The global burden of sexually transmitted infections remains high, with significant associated morbidity and mortality. Gonorrhea is the second most notified sexually transmitted infection in Europe, and its incidence has been increasing in the last years. Although traditionally considered a treatable infection, antimicrobial resistance of Neisseria gonorrhoeaeincludes at present also macrolides, tetracyclines, sulfonamides and trimethoprim combinations, quinolones, and even cephalosporins. These high levels of gonococcal resistance to antimicrobials resulting in untreatable infections in the future may become one of the greatest challenges to the prevention and control of sexually transmitted infections, which may be a significant major public health issue. Therefore, the development of novel antimicrobials and/or new dual antimicrobial therapy regimens is urgently needed. In this paper, evolution of antimicrobial resistance of Neisseria gonorrhoeae is reviewed, along with new drugs currently under development for the treatment of this infection.
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Dissertations / Theses on the topic "Neisseria gonorrhϾ"

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Gbenafa-Agossa, Clémence. "Prévalence et facteurs de risque des infections génitales à Neisseria gonorrhoeae et Chlamydia trachomatis chez les travailleuses de sexe au Bénin en 2003-2004 et évaluation d'un test rapide dans le dépistage de la gonococcie génitale." Master's thesis, Université Laval, 2006. http://hdl.handle.net/20.500.11794/18455.

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Berglund, Torsten. "Recent trends in the epidemiology of gonorrhoea in Sweden : the role of importation and core groups /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-692-1/.

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ROCK, JOHN PATRICK. "MOLECULAR BIOLOGY OF GONOCOCCAL DEATH FOLLOWING EXPOSURE TO THE GRANULE EXTRACTS OF HUMAN NEUTROPHILS (NEISSERIA GONORRHOEAE, PEPTIDOGLYCAN, MEMBRANE DAMAGE)." Diss., The University of Arizona, 1986. http://hdl.handle.net/10150/183844.

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Gonococci that have been phagocytized by human neutrophils are killed very effectively. While much research has focused on defining the microbicidal mechanisms of the neutrophil arsenal, substantially less is known regarding why phagocytized bacteria die. Gonococci were examined, at the molecular level, following exposure to human neutrophil granule extracts (GE) in an effort to discover the "lethal lesion", that injury to the bacterial cell which results in its death. The cytoplasm-based metabolism of GE-treated gonococci continues to function normally for at least 30 minutes, although these same cells have lost the ability to divide and are reproductively dead. GE-treated gonococci were found to utilize less oxygen than control cells, indicative of damage to the cytoplasmic membrane. Visual examination of GE-treated gonococci by light microscopy revealed that the cells undergo very minimal division once exposed to GE. GE-treated gonococci visualized by transmission electron microscopy had outer membranes which suffered time-related disorganization and disruption; the effects began immediately upon contact with GE. GE-treatment was also observed to cause aberrant structure and orientation of forming bacterial septa. Investigation of gonococcal peptidoglycan, the structural component of the bacterial membrane, yielded interesting results when the effects of GE were scrutinized. GE caused subnormal incorporation of peptidoglycan precursors, and also induced a twofold higher rate of release of peptidoglycan turnover fragments than was seen from control cells. After analysis of peptidoglycan fragments released into culture supernatants by thin-layer chromatography and high-pressure liquid chromatography, it was found that the small amount of high-molecular-weight fragments exhausted by control cells was not present with treated cells. Investigation of the cell-associated peptidoglycan, by the above methods, after exposure to GE revealed differences in the digestion products. There was a distinct reduction in the amount of penicillin bound by the penicillin-binding proteins of GE-treated cells. There was, however, no observed change in the electrophoretic mobility between the PBPs of control and treated cells.
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McMillan, Noto Jennifer. "Analysis of the mechanism of transferrin-iron acquisition by Neisseria gonorrhoeae." Online version unavailable until 9/16/2013, 2008. http://hdl.handle.net/10156/2294.

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Hernandez, Diana Raquel. "Regulation of Expression of a Neisseria Gonorrhoeae tRNA-Modification Enzyme (Gcp)." Diss., The University of Arizona, 2012. http://hdl.handle.net/10150/242381.

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Neisseria gonorrhoeae (Ng) encounters different microenvironments during its life-cycle. Some of these niches have different concentrations of oxygen, which influences the rate of Ng growth; as well as iron, an element essential for Ng survival. Differential expression of several proteins allows the bacteria to adapt to the diverse conditions it comes encounters. One protein affected by environmental changes during Ng growth is Gcp, a tRNA-modification enzyme essential for protein synthesis. To study the regulation of expression of Gcp, we first analyzed the sequence of its ORF, gcp. Orthologs of this gene are found in all kingdoms of life. In silico analysis shows that among Neisseria species, gcp ranges in homology from 76% to 99%, at the nucleotide level. Reverse transcription PCR indicates that gcp is expressed as part of an operon, together with three cytochrome-associated genes cyc4, resB and resC. Rapid amplification of complementary DNA ends determined the start of transcription of cyc4 (and possibly of the cyc4-gcp operon) at 95 nucleotides from the gene start codon. Transcriptional fusions determined that the promoter region upstream of cyc4 is the strongest promoter in the operon. However, the region directly upstream of gcp also has low level of promoter activity, suggesting that the gene may be expressed from two different promoters. Semi-quantitative determination of the concentration of gcp mRNA indicates that the transcription of the gene is significantly repressed when Ng is grown under low iron or low oxygen conditions. Analysis of an fnr mutant, grown under the same conditions as its parental wild type, indicates that the FNR transcriptional regulator is involved in the repression of gcp in low iron or low oxygen conditions. Contrary to expectation, the cyc4 promoter is upregulated when Ng is grown under low oxygen or low iron conditions. However, these results cannot be compared to the original promoter strength. Determination of which was performed on bacteria grown in liquid medium. Coregulation of gcp with cytochrome genes can guarantee low levels of protein synthesis when Ng encounters adverse microenvironments and needs its energy redirected to the expression of genes that would allow it to survive.
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Perry, Elizabeth Holly. "Association of ABO, Lewis and Secretor phenotypes and genotypes with Neisseria gonorrhoeae thesis submitted in (partial) fulfilment of the Master of Applied Science, Auckland University of Technology, November 2003." Full thesis. Abstract, 2003. http://puka2.aut.ac.nz/ait/theses/PerryE.pdf.

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DeRocco, Amanda Jean. "Molecular Analysis of Transferrin Binding Protein B in Neisseria Gonorrhoeae." VCU Scholars Compass, 2007. http://scholarscompass.vcu.edu/etd_retro/52.

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The transferrin iron acquisition system of Neisseria consists of two dissimilar proteins, transferrin binding protein A and B (TbpA and TbpB). TbpA and TbpB both specifically and independently bind human transferrin (Tf). TbpA is a TonB-dependent transporter, expression of which is necessary for Tf iron acquisition. In contrast, the lipoprotein TbpB is not necessary for iron internalization; however it makes this process more efficient. The role of TbpB in the transferrin iron acquisition system has not been completely elucidated. It has been suggested that TbpB is entirely surface exposed and tethered to the outer membrane by its lipid moiety. We inserted the hemagluttinin antigen (HA) epitope into TbpB in an effort to examine surface accessible and functional domains of the lipoprotein. We determined that TbpB was entirely surface exposed from just beyond the mature N-terminus. It was previously reported that the N- and C-terminus of TbpB independently bind Tf. HA epitope analysis defined both the N-terminal and C-terminal binding domains. TbpB was previously reported to play an important role in the release of Tf from the receptor. We established that TbpB exhibited a biphasic dissociation pattern; a C-terminal rapid release followed by a slower N-terminal release. These results suggested that the C-terminus plays a role in ligand turnover of the wild-type receptor. Little is known about the transport of TbpB to the outer membrane. In an attempt to identify the signals/mechanisms required for TbpB localization, the signal sequence of the protein was altered. In the absence of lipid modification, TbpB remained associated with the cell, localized to the periplasm. We also noted that internal cysteine residues were not critical for TbpB localization. Our results suggested that TbpB was transported by a lipoprotein-specific mechanism. Additionally, we demonstrated the major outer membrane secretin, PilQ, was not necessary for proper localization of TbpB. The mechanism responsible for this process remains elusive. This body of work represents the first comprehensive study of TbpB topology and function, utilizing the lipoprotein expressed in its native membrane. These results may translate to other, similar lipoprotein receptors of the pathogenic Neisseria, helping to shed light on these poorly understood proteins.
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Pickel, Donnie. "Investigating Complement Regulator Involvement in Innate Immune Evasion by Neisseria gonorrhoeae." Ohio University / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1628181973757983.

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Wood, Megan Lindsay Falsetta. "Coping with stress: anaerobic respiratory and oxidative stress tolerance mechanisms are critical for Neisseria gonorrhoeae biofilm formation." Diss., University of Iowa, 2009. https://ir.uiowa.edu/etd/450.

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Many illnesses and infections are exacerbated and/or caused by biofilms. Neisseria gonorrhoeae, the etiologic agent of gonorrhea, is frequently asymptomatic in women, which can lead to persistent infection. Persistent infection can result in pelvic inflammatory disease, tubo-ovarian abscesses, infertility, and ectopic pregnancy. N. gonorrhoeae has been shown to form biofilms over glass, primary and immortalized cervical cells, and during natural cervical infection. Asymptomatic infection occurs in only 1% of infected males, and the infection site is subject to periodic rapid fluid flow, which may limit biofilm formation. Thus, biofilm formation may specifically play an important role in the infection of women and could contribute to the infrequent occurrence of symptoms. Prior to work presented in this dissertation, little was known about biofilm formation by N. gonorrhoeae. Therefore, we elected to compare the transcriptional profiles of biofilms to their planktonic counterparts, to identify genetic pathways involved in biofilm formation and maintenance. We found that 3.8% of the genome was differentially regulated, and that genes involved in anaerobic metabolism and oxidative stress tolerance were up-regulated in biofilm, while genes involved in aerobic metabolism were down-regulated. We determined that expression of aniA , ccp, and norB is required for robust biofilm formation over glass and human cervical cells, and anaerobic respiration occurs in the substratum of gonococcal biofilms. Disruption of the norB gene resulted in severe attenuation of biofilm formation. We determined that the accumulation of nitric oxide (NO) contributes to the phenotype of a norB mutant and can retard biofilm formation when present at sublethal concentrations. However, higher concentrations of NO can enhance biofilm formation in the absence of nitrite. NO enhances biofilm formation in an aniA mutant, but cannot completely restore biofilm formation, suggesting that NO can support anaerobic growth, although nitrite is preferred. We determined that the majority of the genes involved in gonococcal oxidative stress tolerance are required for normal biofilm formation, as mutations in the following genes resulted in biofilm attenuation over cervical cells and/or glass: oxyR, gor, prx, mntABC, trxB, and estD. Overall, biofilm formation may represent an adaptation for coping with the stresses present in the female genitourinary tract.
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Bergman, Peter. "Antimicrobial peptides and pathogenic Neisseria : experimental studies in mouse, man and rat /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-428-7/.

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Books on the topic "Neisseria gonorrhϾ"

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Mehaffey, Mary A. Performance evaluation critique: Neisseria Gonorrhoeae 1986 I. Atlanta, Ga: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control, 1986.

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Gaboulaud, Valerie. Hayastanum hakamanrēayin mijotsʻneri nkatmamb Neisseria gonorrhoeae-i zgayunutʻyuně in vitro paymannerum. Erevan: Médecins sans frontières, 2005.

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Pathogenic, Neisseria Conference (5th 1986 Noordwijkerhout Netherlands). Gonococci and meningococci: Epidemiology, genetics, immunochemistry, and pathogenesis : proceedings of the 5th International Pathogenic Neisseriae Conference, held in Noordwijkerhout, The Netherlands, 15-18 September, 1986. Dordrecht: Kluwer Academic Publishers, 1988.

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K, Schoolnik Gary, and American Society for Microbiology, eds. The Pathogenic Neisseriae: Proceedings of the fourth international symposium, Asilomar, California, 21-25 October 1984. Washington, D.C: American Society for Microbiology, 1985.

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K, Schoolnik Gary, and Brooks George F, eds. The pathogenic neisseriae: Proceedings of the 4th International Symposium, Asilomar, California, 21-25 October, 1984. Washington: American Society for Microbiology, 1985.

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Antibiotic-resistant strains of neisseria gonorrhoeae: Policy guidelines for detection, management, and control. Atlanta, Ga: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control, Center for Prevention Services, Division of Sexually Transmitted Diseases, 1988.

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Neisseria gonorrhoeae Antimicrobial Resistance Surveillance: Consolidated Guidance. Organización Panamericana de la Salud, 2020. http://dx.doi.org/10.37774/9789275122365.

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Prevention, early diagnosis, and effective treatment are essential for the control and elimination of Neisseria gonorrhoeae as a public health problem. Currently, in Latin America and the Caribbean, treatment for gonorrhea infection is largely empiric and based on clinical diagnosis. In the Americas, the high burden of new N. gonorrhoeae infections (estimated at 11 million new cases a year), the complexity of the disease epidemiology, and in many countries the limited resources, make it difficult to fully understand the burden of disease and the burden of antimicrobial resistance (AMR) in N. gonorrhoeae. PAHO has developed this document to facilitate the navigation of available guidance and recommendations for N. gonorrhoeae AMR surveillance by public health and health care professionals, at the national and subnational levels, involved in designing, implementing, and/or strengthening AMR surveillance of N. gonorrhoeae and overall surveillance of sexually transmitted infections. This document aims to consolidate guidance on AMR surveillance for N. gonorrhoeae from documents published by WHO and PAHO, and strives to assemble relevant information in a summarized manner to help countries in strengthening and/or developing AMR surveillance systems for N. gonorrhoeae.
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Putman, Shannon B., and Arjun S. Chanmugam. Urethritis, Prostatitis, and Epididymitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0039.

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Urethritis, prostatitis, and epididymitis are a constellation of diseases often caused by infections; they can result in dysuria, pain, urethral discharge, and fever. Male dysuria can be the presenting complaint in patients with urethritis, prostatitis, epididymitis, or cystitis. Urethritis is most frequently caused by sexually transmitted infection, including Neisseria gonorrhea, Chlamydia trachomatis, Mycoplasma genitalium, and Trichomonas vaginalis. Prostatitis has four classifications: acute bacterial, chronic bacterial, chronic prostatitis/chronic pelvic pain, and asymptomatic inflammatory prostatitis. Epididymitis is an inflammation of the epididymis, with or without infection lasting less than 6 weeks. Acute epididymitis usually involves the testicles, resulting in an epididymo-orchitis. Although trauma is one example of a noninfectious cause, infectious causes must be considered, especially gonorrhea and chlamydia. Treatment for these diseases is targeted antibiotics based on lab and culture results.
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Book chapters on the topic "Neisseria gonorrhϾ"

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Unemo, Magnus, Daniel Golparian, and David W. Eyre. "Antimicrobial Resistance in Neisseria gonorrhoeae and Treatment of Gonorrhea." In Neisseria gonorrhoeae, 37–58. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9496-0_3.

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Kohl, P. K., M. Kratofiel, K. F. Heck, R. Görner, D. Petzoldt, W. Eggert-Kruse, I. Gerhard, I. Kunze-Janssenharms, A. Senf-Blum, and E. Hoferer. "Studies on natural local immunity in gonorrhea: ELISA reactivities of female local secretions with outer membranes of Neisseria gonorrhoeae." In Neisseriae 1990, edited by Mark Achtman, Peter Kohl, Christian Marchal, Giovanna Morelli, Andrea Seiler, and Burghard Thiesen, 603–8. Berlin, Boston: De Gruyter, 1991. http://dx.doi.org/10.1515/9783110867787-104.

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FAIRBROTHER, R. W. "NEISSERIA: GONORRHŒA: MENINGITIS." In A Text-Book of Medical Bacteriology, 184–97. Elsevier, 2014. http://dx.doi.org/10.1016/b978-1-4832-0032-3.50020-5.

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FAIRBROTHER, R. W. "NEISSERIA: GONORRHŒA: MENINGITIS." In A Text-Book of Bacteriology, 199–214. Elsevier, 2014. http://dx.doi.org/10.1016/b978-1-4832-0031-6.50021-6.

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Marrazzo, Jeanne M., and Michael A. Apicella. "Neisseria gonorrhoeae (Gonorrhea)." In Mandell, Douglas, and Bennett's Principles and Practice of Infectious Diseases, 2446–62. Elsevier, 2015. http://dx.doi.org/10.1016/b978-1-4557-4801-3.00214-9.

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"Gonorrhea (Neisseria Gonorrhoeae)." In The APRN and PA’s Complete Guide to Prescribing Drug Therapy. New York, NY: Springer Publishing Company, 2019. http://dx.doi.org/10.1891/9780826179357.0154.

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"Gonorrhea (Neisseria Gonorrhoeae)." In The APRN and PA’s Complete Guide to Prescribing Drug Therapy. New York, NY: Springer Publishing Company, 2019. http://dx.doi.org/10.1891/9780826179340.0154.

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Schliep, S., and M. Sticherling. "Neisseria-gonorrhoeae-Infektionen – Gonorrhö bei Erwachsenen." In Therapie-Handbuch, 627–28. Elsevier, 2019. http://dx.doi.org/10.1016/b978-3-437-24951-8.00070-1.

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Schliep, S., and M. Sticherling. "Neisseria-gonorrhoeae-Infektionen– Gonorrhö bei Erwachsenen." In Therapie-Handbuch Grundwerk inkl 6. Lfg., 1–2. Elsevier, 2015. http://dx.doi.org/10.1016/b978-3-437-22107-1.50411-8.

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Ramalingam, Karthikeyan, and Sucharithra Ganesh. "In Vitro and in Vivo Models for Pathogenic Neisseria gonorrhoeae Infections." In Epidemiological Research Applications for Public Health Measurement and Intervention, 111–43. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-4414-3.ch008.

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Abstract:
The prevalence of gonorrhea has not discontinued in several countries and still remains as one of the top sexually transmitted diseases (STD) and it's caused by Neisseria gonorrhoeae. This bacterium gains entry into the human host via receptors, and by the usage of several virulence factors, it manages to spread through the cells and leads to severe complications. The study of these bacteria in various in vitro and in vivo models have paved the way for gaining insights on various aspects of bacterial infection, such as the study of pathogenesis of the organism in the host. It also drove the development of more appropriate drugs for the treatment of the gonorrhea illness caused by this ‘superbug'. This chapter focuses on providing a concise overview on the general aspects of N. gonorrhoeaeas an update and the in vitro and in vivo models used for understanding this bacterium over the years. Despite gonorrhea not being a rare STD, it is still a big challenge for researchers, healthcare professionals, and communicators with public awareness to communicate effectively with the general community.
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Conference papers on the topic "Neisseria gonorrhϾ"

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Maatouk, I. "P045 Assessment of Neisseria gonorrhea prevalence and resistance in the Eastern Mediterranean Region." In Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.190.

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