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Relevant bibliographies by topics / Neisseria infections

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Academic literature on the topic 'Neisseria infections'

Author: Grafiati

Published: 4 June 2021

Last updated: 12 February 2022

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Journal articles on the topic "Neisseria infections"

1

Suay-García, Beatriz, and María-Teresa Pérez-Gracia. "Neisseria gonorrhoeae Infections." Pathogens 9, no. 8 (August 12, 2020): 647. http://dx.doi.org/10.3390/pathogens9080647.

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Gonorrhea is a sexually transmitted disease with a high morbidity burden. Despite having guidelines for its treatment, the incidence of the disease follows an increasing trend worldwide. This is mainly due to the appearance of antibiotic-resistant strains, inefficient diagnostic methods and poor sexual education. Without an effective vaccine available, the key priorities for the control of the disease include sexual education, contact notification, epidemiological surveillance, diagnosis and effective antibiotic treatment. This Special Issue focuses on some of these important issues such as the molecular mechanisms of the disease, diagnostic tests and different treatment strategies to combat gonorrhea.

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Lee, Hoan Jong. "Neisseria meningitidis Infections." Korean Journal of Pediatric Infectious Diseases 10, no. 1 (2003): 13. http://dx.doi.org/10.14776/kjpid.2003.10.1.13.

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Smith, Roxanne P., Biswaranjan Mohanty, Shakeel Mowlaboccus, Jason J. Paxman, Martin L. Williams, Stephen J. Headey, Geqing Wang, et al. "Structural and biochemical insights into the disulfide reductase mechanism of DsbD, an essential enzyme for neisserial pathogens." Journal of Biological Chemistry 293, no. 43 (September 4, 2018): 16559–71. http://dx.doi.org/10.1074/jbc.ra118.004847.

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The worldwide incidence of neisserial infections, particularly gonococcal infections, is increasingly associated with antibiotic-resistant strains. In particular, extensively drug-resistant Neisseria gonorrhoeae strains that are resistant to third-generation cephalosporins are a major public health concern. There is a pressing clinical need to identify new targets for the development of antibiotics effective against Neisseria-specific processes. In this study, we report that the bacterial disulfide reductase DsbD is highly prevalent and conserved among Neisseria spp. and that this enzyme is essential for survival of N. gonorrhoeae. DsbD is a membrane-bound protein that consists of two periplasmic domains, n-DsbD and c-DsbD, which flank the transmembrane domain t-DsbD. In this work, we show that the two functionally essential periplasmic domains of Neisseria DsbD catalyze electron transfer reactions through unidirectional interdomain interactions, from reduced c-DsbD to oxidized n-DsbD, and that this process is not dictated by their redox potentials. Structural characterization of the Neisseria n- and c-DsbD domains in both redox states provides evidence that steric hindrance reduces interactions between the two periplasmic domains when n-DsbD is reduced, thereby preventing a futile redox cycle. Finally, we propose a conserved mechanism of electron transfer for DsbD and define the residues involved in domain–domain recognition. Inhibitors of the interaction of the two DsbD domains have the potential to be developed as anti-neisserial agents.

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Jacinto, Teresa, Helena Rego, Juan Gonçalves, and Virgílio Paz Ferreira. "Artrite Séptica Meningocócica Primária em Lactente de Dois Meses." Acta Médica Portuguesa 28, no. 1 (February 27, 2015): 117. http://dx.doi.org/10.20344/amp.4976.

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Meningococcal septic arthritis, ocurring without signs of sepsis, is rare, including at pediatric age. The confinement of infection to a single articulation (monoarticular form) is even rarer in infections by Neisseria spp. We discuss the case of a two month-old caucasian girl, presenting with fever and persistent crying during nappy change. Absence of clinical sepsis was notable. She had had no previous anti-meningococcal immunizations. The ultrasound of the left hip revealed the presence of intra-articular fluid. Culture of the purulent<br />sample after drainage by arthrotomy produced Neisseria meningitidis. She had a good clinical response to the antibiotics. Follow-up showed no sequels. Albeit a rare entity, primary meningococcal arthritis is a mandatory differential diagnosis concerning a feverish child with articular complaints.<br /><strong>Keywords:</strong> Arthritis, Infectious; Infant; Meningococcal Infections; Neisseria meningitidis.

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Catalan, F., J. M. Bohbot, P. Sednaoui, and J. Y. Riou. "Infections génitales à Neisseria meningitidis." Médecine et Maladies Infectieuses 21, no. 3 (March 1991): 212–15. http://dx.doi.org/10.1016/s0399-077x(05)80040-x.

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MORAN, JOHN S. "Treating Uncomplicated Neisseria gonorrhoeae Infections." Sexually Transmitted Diseases 22, no. 1 (January 1995): 39–47. http://dx.doi.org/10.1097/00007435-199501000-00007.

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Valejo Coelho, Margarida Moura, Eugénia Matos-Pires, Vasco Serrão, Ana Rodrigues, and Cândida Fernandes. "Extragenital Gonorrhoea in Men Who Have Sex with Men: A Retrospective Study in a STI Clinic in Lisbon, Portugal." Acta Médica Portuguesa 31, no. 5 (May 30, 2018): 247. http://dx.doi.org/10.20344/amp.10146.

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Introduction: Recent studies worldwide reveal a significant prevalence of extragenital infections by Neisseria gonorrhoeae among men who have sex with men. We aimed to analyse the frequency and characteristics of extragenital gonococcal infections diagnosed in men who have sex with men in a walk-in Sexually Transmitted Infection clinic in Lisbon, Portugal.Material and Methods: We conducted a cross-sectional, retrospective study of the anorectal and/or oropharyngeal Neisseria gonorrhoeae infections in men who have sex with men, diagnosed in our Sexually Transmitted Infection clinic between January 2014 and December 2016.Results: We found extragenital infection in 87 cases of gonorrhoea identified in men who have sex with men in this period, including: 49 cases of anorectal disease, 9 of oropharyngeal disease, 13 cases of infection at both extragenital sites, and 16 of simultaneous extragenital and urogenital gonorrhoea. Patients’ ages ranged from 17 to 64 years (median: 28 years). Forty-seven (54%) of the patients did not present with any extragenital symptoms. Thirty (35%) were human immunodeficiency virus-1-positive.Discussion: Since most extragenital Neisseria gonorrhoeae infections are asymptomatic, they may be missed and go untreated unless actively investigated. Current international guidelines recommend the screening of gonorrhoea at extragenital sites in men who have sex with men because anorectal and oropharyngeal infections constitute a potential disease reservoir, and may facilitate transmission and/or acquisition of human immunodeficiency virus infection.Conclusion: Our results highlight the relevance of testing men who have sex with men for Neisseria gonorrhoeae at extragenital sites, regardless of the existence of local complaints. The implementation of adequate screening programmes in Portugal should be considered. We also reinforce the need to raise awareness in the population regarding the adoption of prophylactic measures against transmission of sexually transmitted infections during anal and/or oral sexual exposure.

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Tinoco, I., A. Jarrell, L. Correa, J. Bissler, J. DeVincenzo, Ivan Tinoco, Amber Jarrell, Lauren Correa, John J. Bissler, and John P. DeVincenzo. "#92: What Is the Optimal Management of Patients Immunosuppressed with the Anti-compliment Monoclonal Antibody, Eculizumab? A Case Report and Review." Journal of the Pediatric Infectious Diseases Society 10, Supplement_1 (March 1, 2021): S12. http://dx.doi.org/10.1093/jpids/piaa170.034.

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Abstract Background Patients with deficiencies of terminal components of complement are at hundreds to thousands fold increased risk of severe and fatal Neisseria spp. infections compared with the general population. Eculizumab is a newly approved monoclonal antibody C5 complement inhibitor. It is indicated for the treatment of atypical hemolytic uremic syndrome (atypical HUS), myasthenia gravis, and paroxysmal nocturnal hemoglobinuria. Because of the complement-depleting effect of Eculizumab dosing (Soliris®, Alexion Pharmaceuticals, Munich, Germany), patients are immunosuppressed for specific infectious pathogens (including Neisseria species) against which protection partially relies on normal complement activity. Because Eculizumab treatment is associated with a dramatically increased risk of Neisseria species. infections, recommendations for Neisseria meningitidis vaccination and antibiotic prophylaxis are contained in Eculizumab prescribing information. However, the most appropriate prevention of infections after Eculizumab has yet to be determined. Methods Case report and literature review. Results A previously healthy 7-year-old male was diagnosed with atypical HUS which included renal failure progressing to dialysis, persistent thrombocytopenia, hemolytic anemia, and hemoglobinuria. Stool cultures and a stool multiplex PCR panel did not detect Shiga-like producing E. coli nor E. coli O157/H7. Eculizumab dosing was therefore planned and Infectious Diseases consultation was obtained for appropriate preventions. The FDA Prescribing Information recommends Neisseria meningitidis vaccination before starting Eculizumab or, if immediate Eculizumab is necessary, to use antibiotic prophylaxis until 2 weeks after vaccination. The accepted protective titer after meningococcal vaccination is population based and uses the serum bactericidal assay (SBA). An antibody titer of >1:4 (human compliment) or 1:8 (rabbit complement) is considered protective. However, this “gold standard” assay incorporates the use of exogenous human or rabbit complement. The protective SBA titers in subjects with terminal complement component deficiencies may not be properly assessed using these same SBA titer protective thresholds. Furthermore, serious meningococcal infections have occurred after appropriate vaccination in patients receiving chronic Eculizumab treatments (ie for paroxysmal nocturnal hemoglobinuria). Finally, SBA protective levels after single Neisseria meningitidis vaccination have not been achieved in majorities of patients with renal failure receiving dialysis and or transplant immunosuppression. Conclusions The current Eculizumab prescribing information recommendations for vaccination and antimicrobial prophylaxis may be inadequate to prevent serious Neisseria infections. Repeated Neisseria meningitidis vaccination and extended antibiotic prophylaxis may afford better protection in patients chronically dosed with Eculizumab.

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Alonso, J. M., and M. Taha. "Actualité des infections à Neisseria meningitidis." Revue Française des Laboratoires 2002, no. 344 (June 2002): 14. http://dx.doi.org/10.1016/s0338-9898(02)80011-7.

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Thangkhiew, I., S. M. Drake, M. Walzman, and A. A. Wade. "Genital infections due to Neisseria meningitidis." Sexually Transmitted Infections 66, no. 4 (August 1, 1990): 305–6. http://dx.doi.org/10.1136/sti.66.4.305-b.

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Dissertations / Theses on the topic "Neisseria infections"

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Johansson, Linda. "Host responses and bacterial virulence factors in Neisseria infections /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-017-6/.

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Leyral, Jérôme. "Endocardites à Neisseria Mucosa à propos d'un cas : revue de la littérature." Bordeaux 2, 1997. http://www.theses.fr/1997BOR2M060.

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Levy, Michael. "Impact de la corticothérapie dans les infections invasives à Neisseria meningitidis." Thesis, Sorbonne Paris Cité, 2018. http://www.theses.fr/2018USPCC323.

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L’évolution fatale des infections invasives à méningocoque (IIM) est souvent associée à une réaction inflammatoire systémique exacerbée déclenchée par des souches de Neisseria meningitidis (Nm) hyper-virulente. L’ajout de glucocorticoïdes, puissants anti-inflammatoires, à l’antibiothérapie adaptée pourrait dans ce cas améliorer le pronostic des patients bien que cela reste difficile à évaluer dans les études cliniques. L’objectif de notre travail était d’évaluer l’impact de ce traitement sur l’infection expérimentale par une souche hyper-invasive de Nm de complexe clonal ST-11 dans le modèle de souris transgénique exprimant la transferrine humaine. Dans notre modèle expérimental, l’infection par voie intrapéritonéale des souris transgéniques avec la souche hyper-invasive Nm C LNP24198 de complexe clonal ST-11 était effectivement associée à une réponse inflammatoire intense et provoquait une atteinte multi-organes avec dissémination des bactéries au niveau méningé, cardiaque, hépatique, splénique et rénal.Les traitements par amoxicilline ou amoxicilline et dexaméthasone à partir de trois heures d’infection puis toutes les six heures pendant 48 heures, ont permis une amélioration clinique et biologique nette en comparaison aux souris traitées par sérum physiologique. Une amélioration clinique nette était notée chez les souris traitées par l’association amoxicilline et dexaméthasone au niveau de la survie et de l’état général par rapport au souris traitées par amoxicilline seule. De plus, on notait une diminution significative de la réaction inflammatoire évaluée par le taux de CRP et de Lipocaline 2 ainsi qu’une augmentation précoce et significative du taux sanguin d’IL-10 après six heures d’infection. Il y avait également une tendance à une diminution de la charge bactérienne évaluée par hémoculture et bioluminescence in-vivo.En réalisant une analyse transcriptomique approfondie, l’adjonction de dexaméthasone à l’amoxicilline a entrainé des différences significatives dans l’expression de gènes et ce, particulièrement concernant les voies de signalisation des monocytes-macrophages. La déplétion des monocytes-macrophages par anticorps monoclonal anti-CSF1R chez des souris infectées par la souche de Nm C LNP24198 a par la suite montré que les souris déplétées (que ce soit au préalable de l’infection ou pendant l’infection) présentaient des infections plus sévères avec sécrétion exacerbée de cytokines pro et anti-inflammatoires. En accord avec ces résultats, les enfants hospitalisés pour IIM sévères de type Purpura fulminans avaient des taux significativement plus bas de monocytes à l’admission.En conclusion, nos travaux suggèrent que les corticoïdes pourraient avoir un effet bénéfique dans les IIM et ce, en modulant la réponse inflammatoire avec notamment une régulation des monocytes-macrophages en favorisant un phénotype de type anti-inflammatoire (M2) plutôt qu’un phénotype pro-inflammatoire (M1)
The fatal course of invasive meningococcal disease (IMD) is often associated with an exacerbated systemic inflammatory response triggered by hyper virulent strains of Neisseria meningitidis (Nm). In view of this, the addition of glucocorticoids, strong anti-inflammatory drugs, to the appropriate antibiotherapy could improve the prognosis of patients but remains difficult to evaluate in clinical studies.The objective of our work was to evaluate the impact of this treatment on experimental infection by a hyper-invasive strain of Nm that belongs to the ST-11 clonal complex in transgenic mice expressing human transferrin.In our experimental model, the intraperitoneal infection of transgenic mice with the hyper-invasive strain Nm C LNP24198 belonging to the ST-11 clonal complex was indeed associated with an intense inflammatory response and caused multi-organ invasion with bacteria spreading to the meninges, the heart, the liver, the spleen and the kidneys.Treatments with amoxicillin or amoxicillin and dexamethasone from three hours of infection and then every six hours for 48 hours, allowed a clear clinical and biological improvement compared to mice treated with physiological saline. A clear clinical improvement (better survival and general condition) was noted in mice treated with amoxicillin and dexamethasone compared to mice treated with amoxicillin alone. In addition, there was a significant decrease in the inflammatory response evaluated by CRP and Lipocalin 2 levels as well as an early and significant increase in the blood level of IL-10 after six hours of infection. There was also a decreasing trend in the bacterial load assessed by blood cultures and in-vivo bioluminescence.By performing a thorough transcriptomic analysis, the addition of dexamethasone to amoxicillin resulted in significant differences in gene expression, particularly regarding the monocyte-macrophage signaling pathways.Monocyte-macrophage depletion using monoclonal antibody anti-CSF1R in mice infected with the strain Nm C LNP24198 subsequently showed that depleted mice (whether prior to infection or during infection) had more severe infections with exacerbated secretion of pro and anti-inflammatory cytokines. In accordance with these results, children hospitalized for severe IMD with purpura fulminans had significantly lower rates of monocytes on admission compared with children with meningitis.In conclusion, our work suggests that corticosteroids may have a beneficial effect on IMD by modulating the inflammatory response, including the regulation of monocytes-macrophages by favoring an anti-inflammatory (M2) phenotype rather than a pro-inflammatory phenotype (M1)

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Rytkönen, Anne. "Molecular studies of Neisseria - host cell interactions /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-018-4/.

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Makepeace, Benjamin Lawrence. "The aetiology and cell biology of inflammation in sexually transmitted bacterial infections." Thesis, University of Southampton, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327260.

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Quinternet, Marc Cung Manh Thông. "Analyse structurale et dynamique par RMN des domaines N-terminaux des protéines DsbD et PilB de Neisseria meningitidis et de leur interaction." S. l. : S. n, 2008. http://www.scd.inpl-nancy.fr/theses/2008_QUINTERNET_M.pdf.

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PENARD, MARIE-CHRISTINE. "Prevention des infections a neisseria meningitidis et haemophilus influenzae : connaissances actuelles sur les vaccins polysaccharidiques." Nantes, 1989. http://www.theses.fr/1989NANT151M.

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Bergman, Peter. "Antimicrobial peptides and pathogenic Neisseria : experimental studies in mouse, man and rat /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-428-7/.

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Folger, Alonzo T. V. "Maternal Chlamydia trachomatis and Neisseria gonorrhoeae Infections and the Outcome of Preterm Birth: The Impact of Early Detection." University of Cincinnati / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1353098416.

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Maurakis, Stavros. "Characterization of the Novel Interaction Between Neisseria gonorrhoeae TdfJ and its Human Ligand S100A7." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5710.

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Neisseria gonorrhoeae is an obligate human pathogen that causes the common STI gonorrhea, which presents a growing threat to global health. The WHO estimated 78 million new cases of gonorrhea worldwide in 2017, with estimates of 820,000 new cases in the United States alone according to the CDC. High-frequency phase and antigenic variation inherent in N. gonorrhoeae, coupled with its natural ability to rapidly acquire and stably integrate antimicrobial resistance factors into its genome, have culminated in an infection against which there is no effective vaccine, and for which the list of viable therapeutic options is quickly shrinking. Moreover, no protective immunity against subsequent infections is elicited upon exposure to N. gonorrhoeae, which highlights the need for research of novel antimicrobial and vaccination strategies. Within the human host, N. gonorrhoeae utilizes a unique strategy to overcome host sequestration of essential nutrients – termed nutritional immunity (NI) – such as ions of trace metals. The pathogen produces a family of outer membrane proteins called TonB-dependent transporters (TdTs) capable of binding to host NI factors and stripping them of their nutritional cargo for use by the pathogen. Importantly, these TdTs are very highly conserved and expressed among Neisseria species. TbpA is a well-characterized TdT that allows N. gonorrhoeae to acquire iron from human transferrin, and recent studies from our lab have shown that TdfH is capable of binding to a zinc-sequestering S100 protein called calprotectin and stripping it of its zinc ion. The S100 proteins are EF-hand calcium-binding proteins that naturally play an integral role in Ca2+ homeostasis, but due to their ability to bind transition metals, they have also demonstrated an innate immunity role by participating in nutrient sequestration. The S100 proteins are expressed in all human cells, and all are capable of binding transition metals including zinc, manganese, and cobalt. Calprotectin, S100A7, and S100A12 have demonstrated an ability to hinder the infection potential of pathogenic E. coli, S. aureus, C. albicans, and various other pathogens via zinc sequestration. Herein, we demonstrate that N. gonorrhoeae is able to overcome this phenomenon and actually utilize these proteins as a zinc source in vitro. Furthermore, we identify S100A7 as the specific ligand for TdfJ, which utilizes this ligand to internalize zinc during infection. S100A7 growth support in vitro is dependent upon a functional TonB, TdfJ, and the cognate ABC transport system ZnuABC, and isogenic mutants incapable of producing znuA or tdfJ recover S100A7 utilization by complementation. Whole-cell binding assays and affinity pulldowns show that S100A7 binds specifically to both gonococcal and recombinant TdfJ, and growth and binding experiments show that these described phenomena are specific to human and not mouse S100A7. Finally, we show that a His-Asn double mutant S100A7 that is incapable of binding zinc cannot be utilized for growth by gonococci. These data illustrate the unique nature of the gonococcus’ ability to co-opt host defense strategies for its own purposes, and further identify the TdTs as promising targets for strategies to combat and prevent gonococcal infection.

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Books on the topic "Neisseria infections"

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Pathogenic Neisseria: Genomics, molecular biology and disease intervention. Norfolk, UK: Caister Academic Press, 2014.

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Genco, C. A. Neisseria: Molecular mechanisms of pathogenesis. Norfolk, UK: Caister Academic Press, 2010.

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Mark, Achtman, ed. Neisseriae 1990: Proceedings of the Seventh InternationalPathogenic Neisseria Conference, Berlin, Federal Republic of Germany, September 9-14, 1990. Berlin: Walter de Gruyter, 1991.

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Neisseria gonorrhoeae Infections. MDPI, 2021. http://dx.doi.org/10.3390/books978-3-0365-0791-0.

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Neisseria gonorrhoeae Antimicrobial Resistance Surveillance: Consolidated Guidance. Organización Panamericana de la Salud, 2020. http://dx.doi.org/10.37774/9789275122365.

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Prevention, early diagnosis, and effective treatment are essential for the control and elimination of Neisseria gonorrhoeae as a public health problem. Currently, in Latin America and the Caribbean, treatment for gonorrhea infection is largely empiric and based on clinical diagnosis. In the Americas, the high burden of new N. gonorrhoeae infections (estimated at 11 million new cases a year), the complexity of the disease epidemiology, and in many countries the limited resources, make it difficult to fully understand the burden of disease and the burden of antimicrobial resistance (AMR) in N. gonorrhoeae. PAHO has developed this document to facilitate the navigation of available guidance and recommendations for N. gonorrhoeae AMR surveillance by public health and health care professionals, at the national and subnational levels, involved in designing, implementing, and/or strengthening AMR surveillance of N. gonorrhoeae and overall surveillance of sexually transmitted infections. This document aims to consolidate guidance on AMR surveillance for N. gonorrhoeae from documents published by WHO and PAHO, and strives to assemble relevant information in a summarized manner to help countries in strengthening and/or developing AMR surveillance systems for N. gonorrhoeae.

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Bentley, Tony. Bacterial infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0310.

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Putman, Shannon B., and Arjun S. Chanmugam. Urethritis, Prostatitis, and Epididymitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0039.

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Urethritis, prostatitis, and epididymitis are a constellation of diseases often caused by infections; they can result in dysuria, pain, urethral discharge, and fever. Male dysuria can be the presenting complaint in patients with urethritis, prostatitis, epididymitis, or cystitis. Urethritis is most frequently caused by sexually transmitted infection, including Neisseria gonorrhea, Chlamydia trachomatis, Mycoplasma genitalium, and Trichomonas vaginalis. Prostatitis has four classifications: acute bacterial, chronic bacterial, chronic prostatitis/chronic pelvic pain, and asymptomatic inflammatory prostatitis. Epididymitis is an inflammation of the epididymis, with or without infection lasting less than 6 weeks. Acute epididymitis usually involves the testicles, resulting in an epididymo-orchitis. Although trauma is one example of a noninfectious cause, infectious causes must be considered, especially gonorrhea and chlamydia. Treatment for these diseases is targeted antibiotics based on lab and culture results.

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Lydyard, Peter, Michael Cole, John Holton, Will Irving, Nino Porakishvili, Pradhib Venkatesan, and Kate Ward. Case Studies in Infectious Disease: Neisseria gonorrhoeae. Garland Science, 2009. http://dx.doi.org/10.4324/9780203853962.

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Lydyard, Peter, Michael Cole, John Holton, Will Irving, Nino Porakishvili, Pradhib Venkatesan, and Kate Ward. Case Studies in Infectious Disease: Neisseria meningitidis. Garland Science, 2009. http://dx.doi.org/10.4324/9780203853979.

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Nadel, Simon, and Johnny Canlas. Management of meningitis and encephalitis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0241.

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Management of CNS infections requires specific antimicrobial agents, as well as specific supportive treatment targeted at reducing raised intracranial pressure and other life-threatening complications. It is important that the need for management in an intensive care setting is considered early in the illness. Antibiotic resistance amongst the most common organisms causing bacterial meningitis is becoming more common and antibiotic therapy should be adjusted accordingly. Anti-inflammatory treatment such as steroids should be started as soon as possible in patients with proven acute bacterial meningitis. Optimally, this should be before or with the first dose of antibiotics. Vaccine research is progressing so that effective vaccines should be available in the future against all the common causes of bacterial meningitis and encephalitis, including Neisseria meningitidis serogroup b.

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Book chapters on the topic "Neisseria infections"

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Ketunuti, Melissa, and Matthew P. Kronman. "Neisseria Infections." In Textbook of Clinical Pediatrics, 1011–16. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_86.

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Young, Hugh, and Marie Ogilvie. "Neisseria gonorrhoeae (Gonorrhoea)." In Genitourinary Infections, 275–79. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-017-5080-6_10.

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Cristaudo, Antonio, and Diego Orsini. "Neisseria gonorrhoeae Infections." In Sexually Transmitted Infections, 197–210. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-02200-6_9.

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Taha, Muhamed-Kheir. "Evolution of Neisseria and Neisseria Infections." In Evolutionary Biology of Bacterial and Fungal Pathogens, 465–74. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815639.ch39.

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Taha, Muhamed-Kheir, and Ala-Eddine Deghmane. "Molecular Typing of Neisseria meningitidis." In Molecular Typing in Bacterial Infections, 179–91. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-185-1_12.

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Stefanelli, Paola, and Anna Carannante. "Antimicrobial Resistance in Neisseria gonorrhoeae: A New Challenge." In Sexually Transmitted Infections, 363–74. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-02200-6_19.

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Knapp, Katherine M. "Listeria monocytogenes, Neisseria gonorrhoeae, and Other Bacteria." In Congenital and Perinatal Infections, 225–32. Totowa, NJ: Humana Press, 2006. http://dx.doi.org/10.1385/1-59259-965-6:225.

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Jerse, Ann E. "Neisseria gonorrhoeae: Adaptation and Survival in the Urogenital Tract." In Persistent Bacterial Infections, 199–227. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818104.ch11.

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Ison, Catherine A., and Jonathan Ross. "The Management of Antibiotic-Resistant Neisseria gonorrhoeae." In Management of Multiple Drug-Resistant Infections, 159–72. Totowa, NJ: Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-738-3_9.

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Meyer, Th F., R. Haas, R. Halter, P. Nickel, J. Pohlner, A. Stern, J. Clarke, H. Delius, and M. So. "Molecular Analysis of Virulence Determinants of Neisseria gonorrhoeae." In The Pathogenesis of Bacterial Infections, 221–34. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70351-5_19.

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Conference papers on the topic "Neisseria infections"

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Wang, Ying, Tianhong Dai, and Ying Gu. "Antimicrobial blue light inactivation of Neisseria gonorrhoeae." In Photonic Diagnosis and Treatment of Infections and Inflammatory Diseases, edited by Tianhong Dai. SPIE, 2018. http://dx.doi.org/10.1117/12.2291545.

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Wang, Ying, Raquel Ferrer-Espada, Yan Baglo, Sharon X. Goh, Kathy D. Held, Yonatan H. Grad, Ying Gu, Jeffrey A. Gelfand, and Tianhong Dai. "Photo-inactivation of Neisseria gonorrhoeae: a paradigm changing approach for combating antibiotic-resistant gonococcal infections." In Photonic Diagnosis, Monitoring, Prevention, and Treatment of Infections and Inflammatory Diseases 2019, edited by Tianhong Dai, Mei X. Wu, and Jürgen Popp. SPIE, 2019. http://dx.doi.org/10.1117/12.2505736.

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Thibeault, R., R. Escobar Careaga, C. Lavallée, A. Labbé, G. Roy, and C. Fortin. "P416 Screening Rates and Follow-up of Chlamydia trachomatis and Neisseria gonorrhoeae Infections During Pregnancy." In Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.441.

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Laumen, J., S. Abdellati, SS Manoharan-Basil, C. Van Dijck, D. Van den Bossche, I. De Baetselier, T. De Block, et al. "P084 Screening of anorectal and oropharyngeal samples fails to detect bacteriophages infecting Neisseria gonorrhoeae." In Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.216.

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Whelan, J., J. Eeuwijk, E. Bunge, and E. Beck. "P151 Systematic literature review and quantitative analysis of health problems associated with sexually transmitted Neisseria gonorrhoeae infection." In Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.259.

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Rowlinson, E., O. Soge, A. Berzkalns, C. Thibault, R. Kerani, M. Golden, and L. Barbee. "O09.2 Lack of association between azithromycin-resistant Neisseria gonorrhoeae infection and prior exposure to azithromycin among persons attending a Sexual Health Clinic." In Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.99.

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Fuller, S., M. Furegato, L. Phillips, E. Harding-Esch, A. Pacho, E. Heming De-Allie, E. Mabonga, et al. "O17.4 First clinical evaluation of a 30-minute point-of-care-test for Chlamydia trachomatis and Neisseria gonorrhoeae infection in UK sexual health clinics." In Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.149.

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Reports on the topic "Neisseria infections"

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Newman, Sara B. An Epidemiologic Analysis of Chlamydia trachomatis and Neisseria gonorrhoeae Infections in Female Federal Prisoners. Fort Belvoir, VA: Defense Technical Information Center, January 2002. http://dx.doi.org/10.21236/ada421099.

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Vonck, Rachel A. Chlamydia muridarum Alters the Immune Environment of the Murine Genital Tract to be More Permissive for Infection with Neisseria gonorrhoeae in a Novel Coinfection Model. Fort Belvoir, VA: Defense Technical Information Center, March 2011. http://dx.doi.org/10.21236/ad1013375.

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