Relevant bibliographies by topics / Neoplasms - in ingancy and childhood

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Neoplasms - in ingancy and childhood'

Author: Grafiati
Published: 4 June 2021
Last updated: 3 February 2022

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Journal articles on the topic "Neoplasms - in ingancy and childhood"

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Geller, Evan, and Polly S. Kochan. "Renal Neoplasms of Childhood." Radiologic Clinics of North America 49, no. 4 (July 2011): 689–709. http://dx.doi.org/10.1016/j.rcl.2011.05.003.

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Hancock, Betty Jean, Maria Di Lorenzo, Sami Youssef, Salam Yazbeck, Jacques-Edouard Marcotte, and Pierre-Paul Collin. "Childhood primary pulmonary neoplasms." Journal of Pediatric Surgery 28, no. 9 (September 1993): 1133–36. http://dx.doi.org/10.1016/0022-3468(93)90147-d.

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Lala, Shailee V., and Naomi Strubel. "Ovarian neoplasms of childhood." Pediatric Radiology 49, no. 11 (October 2019): 1463–75. http://dx.doi.org/10.1007/s00247-019-04456-8.

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Maniar, Tapan N., Inbal Braunstein, Stephen Keefe, Sofia Hussen, Tara Abrams, Angela De Michele, and Wafik S. El-Deiry. "Childhood ALL and second neoplasms." Cancer Biology & Therapy 6, no. 10 (October 2007): 1525–31. http://dx.doi.org/10.4161/cbt.6.10.4928.

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Lee, Christina Y., Lauren M. Sholl, Bin Zhang, Emily A. Merkel, Sapna M. Amin, Joan Guitart, and Pedram Gerami. "Atypical Spitzoid Neoplasms in Childhood." American Journal of Dermatopathology 39, no. 3 (March 2017): 181–86. http://dx.doi.org/10.1097/dad.0000000000000629.

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Chung, C. J., V. Bui, Lynn A. Fordham, Jeannie Hill, and Dorothy Bulas. "Malignant intraperitoneal neoplasms of childhood." Pediatric Radiology 28, no. 5 (May 6, 1998): 317–21. http://dx.doi.org/10.1007/s002470050363.

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Eryilmaz Polat, Sanem, Mina Gharibzadeh Hizal, Beste Ozsezen, Gokcen Dilsa Tugcu, and Guzin Cinel. "Childhood Pulmonary Neoplasms in Two Cases." Turkish Thoracic Journal 20, no. -1 (September 9, 2019): 375. http://dx.doi.org/10.5152/turkthoracj.2019.375.

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Armata, Jerzy, and Walentyna Balwierz. "Prognosis in Childhood Second Malignant Neoplasms." Leukemia & Lymphoma 7, no. 4 (January 1992): 341–42. http://dx.doi.org/10.3109/10428199209049788.

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Kennedy, Alexander. "Ovarian Neoplasms in Childhood and Adolescence." Seminars in Reproductive Medicine 6, no. 01 (February 1988): 79–90. http://dx.doi.org/10.1055/s-2007-1021344.

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Varan, Ali, and Rejin Kebudi. "Secondary Malignant Neoplasms After Childhood Cancer." Pediatric Hematology and Oncology 28, no. 5 (March 17, 2011): 345–53. http://dx.doi.org/10.3109/08880018.2011.553879.

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Dissertations / Theses on the topic "Neoplasms - in ingancy and childhood"

1

Janson, Christopher M. "Marriage and Divorce in Survivors of Childhood Cancer: A Report from the Childhood Cancer Survivor Study." Yale University, 2008. http://ymtdl.med.yale.edu/theses/available/etd-08092007-145913/.

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In this report from the Childhood Cancer Survivor Study (CCSS), we described marriage and divorce rates in survivors of childhood cancer, as compared to a sibling control group and the general U.S. population. We also sought to identify patient and treatment characteristics that were associated with survivor marital status. This study included 8,930 five-year survivors of childhood malignancy and 2,855 sibling controls participating in the CCSS. Data on marital status, sociodemographic factors, and current health status were obtained from questionnaires; detailed disease and treatment histories were available from medical records. Marital status of the U.S. population was obtained from the 2002 Current Population Survey of the U.S. Census. We found that survivors were more likely to have never married than both sibling (odds ratio [OR] = 1.79; 95 % CI = 1.65-1.94; p < 0.0001) and population controls (OR = 2.29; 95 % CI = 2.19-2.38; p < 0.0001), with persistence of trends across age and gender strata. Once married, survivors divorced at rates equivalent to controls. In adjusted analysis, we found that several survivor characteristics predicted never-married status, including treatment involving cranial radiation (OR = 2.41; p < 0.0001), CNS tumor diagnosis (OR = 2.05; p < 0.0001), history of growth hormone deficiency (OR = 2.02; p < 0.0001), and unemployment secondary to disability (OR = 1.78; p = 0.0001). Survivor characteristics predictive of divorce included unemployment (OR = 1.91; p < 0.0001, for unemployed or disabled), lower educational achievement (OR = 1.74; p < 0.0001, for non-college graduates), and psychological distress (OR = 1.60; p < 0.0001). This study confirms prior reports of lower marriage rates in survivors of childhood cancer, providing further evidence that this population struggles with psychosocial adjustment to adult life.
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Sköldenberg, Erik. "Angiogenesis in childhood malignancies /." Uppsala : Institutionen för kirurgiska vetenskaper, Uppsala universitet, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3481.

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Wilson, Carmen Louise Children's Cancer Institute Australia for Medical Research UNSW. "The late effects of therapy in an Australian cohort of childhood cancer survivors." Awarded By:University of New South Wales. Children's Cancer Institute Australia for Medical Research, 2008. http://handle.unsw.edu.au/1959.4/43794.

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In Australia, up to 80% of individuals diagnosed with childhood cancer are now expected to survive for more than five years after their initial diagnosis. However, survivors of childhood cancer are at risk of developing late sequelae as a consequence of therapies received during childhood. The aim of this study was to determine the incidence of selected late sequelae in a cohort of Australian childhood cancer survivors and identify treatment and genetic factors that may modify the risk of late sequelae in survivors. Our study included 1150 individuals treated for childhood cancer at the Sydney Children??s Hospital between 1962 and 1999, who had remained in remission >3 years and were confirmed to be alive. Rates of mortality and second cancers among survivors were compared against population rates to determine standardised mortality and incidence ratios. Survivors completed a questionnaire on the incidence of adverse health conditions and provided a buccal specimen. Real time PCR was used to detect polymorphisms in genes involved in drug detoxification and transport. Rates of mortality and secondary cancers were found to be 7.5-fold (95%CI 5.4-10.1) and 4.9-fold (95%CI 2.9-8.0) higher among survivors of childhood cancer relative to the general population, respectively, with the highest risks observed for those survivors previously treated for Hodgkin??s disease. Over 60% of survivors reported at least one cardiopulmonary, endocrine or sensory-motor condition following diagnosis of childhood cancer; the most frequently observed conditions included growth hormone deficiency, hypothyroidism, and hypertension. Late sequelae were most frequently reported by females and survivors of brain tumours. Genetic investigations showed that an increased risk of growth hormone deficiency was associated with homozygosity for the GSTM1 null polymorphism, while no gene associations were observed to influence the risk of second cancers among survivors. Our study demonstrates that survivors of childhood cancer are at risk of developing a variety of health conditions as a result of anti-cancer therapies received during childhood. Determining risk factors for late sequelae based on therapy type, lifestyle and genetic predisposition will enable the optimisation of treatment protocols and promote the future well-being of childhood cancer survivors.
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Dunwell, Thomas Lawson. "Epigenetic analysis of childhood leukaemia and the Hippo pathway." Thesis, University of Birmingham, 2010. http://etheses.bham.ac.uk//id/eprint/991/.

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Hypermethylation of CpG islands is one of the many processes that a developing cancer cell may use for the inactivation of tumour suppressor genes. The Sav/Hippo/Warts pathway was originally identified in Drosophila and shown to be responsible for controlling both growth and apoptosis, implying this is a tumour suppressor pathway. This pathway is both evolutionarily and functionally conserved in mammals. Work presented here shows that apart from FAT1 and YAP other pathway members are not epigenetically silenced in common epithelial or haematological cancers. FAT1 and YAP were frequently methylated in childhood acute lymphoblastic leukaemia (ALL) but unmethylated in epithelial cancers. Childhood ALL is a blood cancer with peak prevalence between the ages of 3-5 years. The epigenetics of this cancer were examined with three separate approaches; the first, a candidate gene approach, second a NotI restriction enzyme based array examining the methylation of genes residing on chromosome 3, and thirdly the methylated-CpG island recovery assay (MIRA) combined with CpG island arrays examining methylation on a genome-wide scale. These approaches identified a large number of novel genes which were frequently methylated in ALL. Many of the identified genes were new methylation targets and were shown to be likely targets for methylation in both common epithelial and haematological cancers. A series of these genes was seen to be specifically methylated in different leukaemia sub types, and to cluster T-ALL and B-ALL samples into high and low methylation clusters. When examined in chronic lymphoblastic leukaemia (CLL) methylation of two of the above genes was associated with disease progression and methylation of another gene was associated with response to clinical treatment.
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Dreifaldt, Ann Charlotte. "Epidemiological aspects on malignant diseases in childhood /." Örebro : Örebro University : Universitetsbiblioteket, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-585.

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Gill, Simrandip Kaur. "Single voxel proton magnetic resonance spectroscopy of childhood brain tumours." Thesis, University of Birmingham, 2014. http://etheses.bham.ac.uk//id/eprint/4899/.

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Conventional magnetic resonance imaging (MRI) is essential for the management of childhood brain tumours. However, it is increasingly being supplemented with functional techniques such as magnetic resonance spectroscopy (MRS). This thesis investigates how pre-treatment single voxel MRS can aid in diagnosis and surveillance of paediatric brain tumours and identify prognostic biomarkers. Data from multiple centres, scanners from three leading manufacturers and field strengths of 1.5 T and 3 T are incorporated. MRS was analysed using TARQUIN software with metabolite peaks fitted using a simulated basis set to provide metabolite concentrations. Univariate and multivariate statistical tests were used to compare variables. Multi-scanner spectroscopy detected significant differences in common and rare paediatric brain tumours. Diagnostic metabolite profiles were able to confirm tumour on follow-up imaging. Elevated creatine and total choline determined good prognosis in medulloblastoma. Myo-inositol and citrate aided in the characterisation of diffuse pontine gliomas (DIPG). While conventional MRI was unable to identify prognostic markers for DIPG, elevated taurine was found to be significantly associated with a better prognosis. The results encourage the use of MRS as an adjunct to conventional MRI in routine clinical practice. For future studies, accurate assignment of biomarkers will be determined in tumour tissue using in vitro high-resolution spectroscopy methods.
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Guha, Joyeeta. "Risks of adverse health and social outcomes among childhood cancer survivors." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6612/.

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As a result of improvement in survival after childhood cancer, there are now increasing numbers of long-term survivors of childhood cancer living in the United Kingdom and across Europe. Specific groups of these childhood cancer survivors experience substantial excess risks of adverse health and social outcomes. Using the population-based British Childhood Cancer Survivor Study (BCCSS) the following areas were investigated: (I) The proportion of survivors on regular long-term hospital follow-up using risk stratification levels of care developed by the BCCSS in partnership with the National Cancer Survivorship Initiative. (2) The risks of adverse health and social outcomes using record-linkage and a self-reported questionnaire to assess which survivors of central nervous system tumours were at excess risk compared to the general population. (3) The risk of hospitalisation due to cerebrovascular conditions among childhood cancer survivors by electronic record linkage with Hospital Episode Statistics. Using the European PanCareSurFup cohort, the excess risks of genitourinary subsequent primary neoplasms were investigated among five-year survivors of childhood cancer. This thesis quantifies the risks experienced by childhood cancer survivors in four areas and provides an evidence-base for risk stratification by healthcare professionals caring for survivors.
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Wong, Kwok Fai. "Epidemiological analysis of survivorship after childhood cancer." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6629/.

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This thesis investigates the adverse outcomes amongst survivors of childhood cancer using the British Childhood Cancer Survivor Study (BCCSS) and the Pancare Childhood and Adolescent Cancer Survivor Care and Follow-up studies (PCSF). The specific aims were to investigate (1) adverse outcomes up to 50 years of follow-up in survivors of Wilms’ tumour; (2) risks of hospitalisations due to renal morbidities in childhood cancer survivors; (3) risk of subsequent primary neoplasms arising in the digestive system in survivors of childhood cancer; and (4) adverse outcomes beyond 50 years of follow-up in survivors of heritable retinoblastoma. This thesis demonstrated that survivors of Wilms’ tumour are at substantial risk of premature mortality, particularly for those who have survived 30 years from original diagnosis. This particular group of survivors have the highest risk of hospitalisations due to renal morbidities, such as chronic renal failure, and subsequent primary neoplasms in specific organs in the lower digestive system. Survivors of heritable retinoblastoma who received external beam radiotherapy experienced an increased risk of subsequent primary neoplasms developing above the shoulder, whereas those who received brachytherapy were similar to those who did not receive any radiotherapy and did not experience an increased risk of subsequent primary neoplasms.
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Davis, Emma E. "Neurodevelopmental outcome following cerebellar tumour sustained in early childhood." Thesis, University of Nottingham, 2011. http://eprints.nottingham.ac.uk/12201/.

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Mounting evidence from imaging studies, developmental disorders and typically-developing children suggests that different domains of functioning are more closely related than previously considered. This is reflected in theories of development which are increasingly recognising that developmental progression should be considered as an integrated process, with associations between domains. The extent of the interrelation between cognitive and motor skills remains unclear despite previous investigations. Examination of this relationship in typically-developing children is important to clarify the nature of this link, thereby informing theories of development for both typical and atypical populations. This thesis investigated the underlying nature of the association between cognitive and motor domains to establish the extent of interrelation and whether this link alters across development. As the cerebellum has been hypothesised to be instrumental in this relationship (Diamond, 2000), the role of the cerebellum was investigated by examining cognitive and motor development in children treated for cerebellar tumour in the preschool years (N=15). The impact of cerebellar injury on development of scholastic and attentional skills was also investigated, together with the influence of factors affecting prognosis. The interrelation of cognitive and motor skills in typically-developing children (N=248; 4-11 years) was found to be underpinned by a link between visual processing abilities and fine manual motor skills. Despite fluctuations in correlations between other aspects of cognitive and motor functioning, this core relationship remained constant, furthering evidence that cognitive and motor development are linked from an early age. A similar pattern of correlations was seen for the patient sample, suggesting that development in these domains remains tightly linked despite damage to an underlying component of the anatomical network. This suggests that the patients are demonstrating a developmental delay, rather than deviation; their trajectory does not appear to be qualitatively different from that of typically-developing children, rather development appears to be more constrained than suggested by some hypotheses (e.g. Karmiloff-Smith, 1992). Cerebellar damage was therefore found to impact on the two domains similarly, offering support to a „universal cerebellar transform‟ (Schmahmann, 2000b) conceptualisation of cerebellar functioning. Both cognitive and motor skills were found to be compromised following a cerebellar tumour, although no specific impact of cerebellar damage was reported on scholastic skills, above and beyond general cognitive deficit. Attention was found to be impaired in the patient sample, with sustained attention most closely related to functioning in cognitive, academic and motor skills, suggesting that a deficit in this basic underlying process underlies difficulties in other domains. In addition, sustained attention was implicated in the association between visual processing and fine manual control in the patient sample, suggesting that this core link may be further underpinned by more basic cognitive processes. Effective rehabilitation may therefore target sustained attention, as this appears to be related to functioning in the other domains assessed in this study, as well as recognising that an integrated approach across domains is likely to yield maximum benefits. Of the potential moderating factors investigated, age at diagnosis and tumour type/treatment were found to be the most reliable predictors of outcome. This research highlights the importance of a case-study approach, and the clinical importance of individual investigation of each child‟s needs for rehabilitation.
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Hjalmars, Ulf. "Epidemiological studies including new methods for cluster analysis of acute childhood leukaemia and brain tumours in Sweden /." Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-3264-6/.

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Books on the topic "Neoplasms - in ingancy and childhood"

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Steen, R. G. Childhood Cancer. Cambridge, MA: Harper Collins Trade, 2000.

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Ronald D. [et al.] Barr. Childhood cancer. 2nd ed. Hamilton, Ont: B.C. Decker, 2001.

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National Cancer Institute (U.S.) and International Cancer Research Data Bank., eds. Selected abstracts on childhood nervous system neoplasms. [Bethesda, MD]: U.S. Dept. of Health and Human Services, Public Health Service, National Cancer Institute, 1987.

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Maul-Mellott, Susan K. Childhood cancer: A nursing overview. Boston: Jones and Bartlett, 1987.

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Bain, Lisa J. A parent's guide to childhood cancer. New York, N.Y: Dell, 1995.

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Lederman, Ruth I. Childhood cancer in Massachusetts, 1982-1986. Boston, Mass. (150 Tremont St., Boston 02111): Massachusettes Dept. of Public Health, 1989.

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Shiminski-Maher, Tania. Childhood brain & spinal cord tumors: A guide for families, friends & caregivers. Bejing: O'Reilly, 2002.

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Shiminski-Maher, Tania. Childhood brain & spinal cord tumors: A guide for families, friends & caregivers. Bejing: O'Reilly, 2002.

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Shiminski-Maher, Tania. Childhood brain & spinal cord tumors: A guide for families, friends & caregivers. Bellingham, WA: Childhood Cancer Guides, 2014.

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Connolly, Harry. Fighting chance: Journeys through childhood cancer. Baltimore, Md: Woodholme House, 1998.

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Book chapters on the topic "Neoplasms - in ingancy and childhood"

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Cassady, J. Robert. "Unusual Neoplasms of Childhood." In Radiation Therapy in Pediatric Oncology, 369–78. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-84520-8_24.

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Adzick, N. Scott, and Pablo Laje. "Pancreatic Neoplasms in Children." In The Surgery of Childhood Tumors, 303–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-48590-3_17.

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Bhatia, Smita. "Second Malignant Neoplasms." In Handbook of Long Term Care of The Childhood Cancer Survivor, 209–20. Boston, MA: Springer US, 2015. http://dx.doi.org/10.1007/978-1-4899-7584-3_14.

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Nanda, Arti. "Vascular Malformations and Neoplasms in Childhood." In Textbook of Clinical Pediatrics, 1555–64. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_150.

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Russell, Robert T., and Anna T. Meadows. "Organ Dysfunction, Second Malignant Neoplasms, and Survival." In The Surgery of Childhood Tumors, 615–27. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-48590-3_32.

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Fisher, Kevin E., and M. Monica Gramatges. "Inherited and Acquired Myeloid Neoplasms of Childhood." In Molecular Pathology Library, 281–310. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-62146-3_15.

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Nistal, Manuel, and Pilar González-Peramato. "Germ Cell Tumors of Infancy and Childhood." In Pathology of Testicular and Penile Neoplasms, 69–89. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-27617-5_4.

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Ball, W. S. "Neuroimaging Diagnosis of Primary Brain Neoplasms in Childhood." In Diseases of the Brain, Head and Neck, Spine, 107–11. Milano: Springer Milan, 2004. http://dx.doi.org/10.1007/978-88-470-2131-0_17.

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Barnhill, Raymond L. "Congenital Melanocytic Nevi and Associated Neoplasms, Congenital and Childhood Melanoma." In Pathology of Melanocytic Nevi and Malignant Melanoma, 111–47. New York, NY: Springer New York, 2004. http://dx.doi.org/10.1007/978-0-387-21619-5_6.

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Coffin, C. M., and A. L. Pappin. "Polyps and Neoplasms of the Gastrointestinal Tract in Childhood and Adolescence." In Perspectives in Pediatric Pathology, 127–71. Basel: KARGER, 1997. http://dx.doi.org/10.1159/000061921.

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Conference papers on the topic "Neoplasms - in ingancy and childhood"

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Marron, Manuela, Anja Victor, Eva Weis, Claudia Martina Messow, Brigitte Schneider-Rätzke, Nicolai Kohlschmitd, Thomas Haaf, Claudia Spix, and Danuta Galetzka. "Abstract 5504: Second neoplasms after childhood cancer and gene expression differences in primary fibroblasts." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5504.

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Chopra, Seema. "Sclerosing sex cord stromal tumour of the ovary: A rare variant of ovarian neoplasms in childhood and adolescence." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685321.

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Case Report: 19 yr old unmarried girl c/o abdominal distension, loss of appetite and Irregular menstrual cycles x 5 months. USG: gross ascites, liver, Lobulated isoechoic mass in right adnexa, 7x5 cm, abutting right ovary. CA125: 1297 U/ml. FNAC Degenerated crushed cells & stromal fragments. Few scattered benign oval/spindle cells. Laparoscopy f/b laparotomy: 6 litres of straw colored asciic fluid drained. Uterus, left adnexa normal. Rt ovarian mass 6x7 cm, bilobed, arising from ovary. Solid, stuck in POD Adherent to gut. Right oophrectomy done. CA-125: 22 u/ml on day 6 post op. HPE – Sclerosing stromal tumor. Discussion: Sclerosing sex cord stromal tumour of the ovary is a rare tumor; accounts for 6% of ovarian stromal tumors Over a 100 reported tumors in literature. 80% of SST seen in second and third decade of life. Essentially a benign tumour, Usually a unilateral nonfunctioning tumor. Few cases with elevated serum CA-125 and hormonal abnormalities have been reported. Endocrine alterations caused by secretion of estrogen, progesterone or testosterone; induction of precocious puberty. Conclusion: Unilateral oophrectomy is the treatment. No recurrence of the tumor in the patients treated by oophorectomy or by conservative resection of the tumor. Excision of the tumor isfollowed by normal menses, pregnancy has also been reported.
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Wang, Zhaoming, Carmen L. Wilson, John Easton, Dale Hedges, Qi Liu, Gang Wu, Michael Rusch, et al. "Abstract 3001: Germline mutations in cancer predisposition genes and risk for subsequent neoplasms among long-term survivors of childhood cancer in the St. Jude Lifetime Cohort." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3001.

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