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Books on the topic 'Nerve action'

Author: Grafiati

Published: 27 July 2024

Last updated: 31 July 2025

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1

Michael, Conn P., ed. Hormone action. Academic Press, 1986.

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2

Michael, Conn P., ed. Hormone action. AcademicPress, 1989.

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3

Stanton, Susan Gay. Cochlear nerve compound action potential changes during surgery. National Library of Canada, 1990.

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4

F, Stanley Elis, Nowycky Martha C, Triggle D. J, and New York Academy of Sciences., eds. Calcium entry and action at the presynaptic nerve terminal. New York Academy of Sciences, 1991.

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5

Matthews, Gary G. Cellular physiology of nerve & muscle. Blackwell Scientific Publications, 1986.

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6

Matthews, Gary G. Cellular physiology of nerve and muscle. 2nd ed. Blackwell Scientific Publications, 1991.

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7

Garraway, Richard. The action of semiochemicals on olfactory nerve activity and behaviour of Deroceras reticulatum (Müll.). Portsmouth Polytechnic, School of Biological Sciences, 1992.

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8

Gatti, Andrea. Involvement of protein kinase C (PKC) in the short- and long-term action of nerve growth factor in PC12 cells. Brunel University, 1993.

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9

Borgenicht, Joe, and David Borgenicht. The Action Hero's Handbook: How to Catch a Great White Shark, Perform the Vulcan Nerve Pinch, Track a Fugitive, and Dozens of Other TV and Movie Skills. Quirk Books, 2002.

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10

R, Fozard John, ed. The Peripheral actions of 5-hydroxytryptamine. Oxford University Press, 1989.

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11

Richard, Fozard John, ed. The Peripheral actions of 5-hydroxytryptamine. Oxford University Press, 1989.

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12

E, Bloom Floyd, and Roth Robert H. 1939-, eds. The biochemical basis of neuropharmacology. 5th ed. Oxford University Press, 1986.

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13

E, Bloom Floyd, and Roth Robert H. 1939-, eds. The biochemical basis of neuropharmacology. 6th ed. Oxford University Press, 1991.

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14

E, Bloom Floyd, ed. The biochemical basis of neuropharmacology. 7th ed. Oxford University Press, 1996.

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15

E, Bloom Floyd, and Roth Robert H. 1939-, eds. The biochemical basis of neuropharmacology. 8th ed. Oxford University Press, 2003.

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16

International Symposium on Ground Freezing and Frost Action in Soils (2000 Louvain-la-Neuve, Belgium). Ground freezing 2000: Frost action in soils : proceedings of the International Symposium on Ground Freezing and Frost Action in Soils / Louvain-la-Neuve / Belgium / 11-13 September 2000. A.A. Balkema, 2000.

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17

Spitzer, Manfred. The mind within the net: Models of learning, thinking, and acting. The MIT Press, 1999.

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18

1941-, Szelenyi I., ed. Inhibitors of monoamine oxidase B: Pharmacology and clinical use in neurodegenerative disorders. Birkhäuser, 1993.

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19

Bar, Ḥavivah. The School for Peace at Neve Shalom/[Wāḥat al-Salām]: Description and analysis of an action research : on-going interaction between groups of Jews and Arabs : a 3-year experimental research project. The Israel Institute of Applied Social Research, 1986.

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20

Nerve and muscle excitation. 3rd ed. Sinauer Associates, 1992.

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21

Kat, A. Inferno: Test of Nerve. Krzton, Alicia, 2022.

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22

Lofthouse, Rob. Sheer Nerve: An Action-Packed War Thriller. Lume Books, 2022.

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23

Pitt, Matthew. Nerve physiology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198754596.003.0003.

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The chapter begins with a description of the normal findings in healthy sensory and motor nerves. The distribution of nerve fibres by diameter in the sensory nerve and its effect on the recorded action potential is outlined. The method by which velocity and compound muscle action potential are derived from motor stimulation follows. H-reflex studies and F-wave identification are described. A section on the strategies used for nerve conduction study in children and the nerves chosen for examination leads on to a description of the difficulties of deriving normative data in children. Next follow

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24

(Editor), Nathan P. Colowick, Nathan P. Kaplan (Editor), and P. Michael Conn (Editor), eds. Hormone Action, Part J: Neuroendocrine Peptides, Volume 124: Volume 124: Hormone Action Part J (Methods in Enzymology). Academic Press, 1986.

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25

Mechanisms of insulin action on sympathetic nerve activity. National Aeronautics and Space Administration, 1996.

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26

Galvani's spark: The story of the nerve impulse. Oxford University Press, 2011.

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27

Conn, P. Michael. Methods in Enzymology, Volume 168: Hormone Action, Part K: Neuroendocrine Peptides. Academic Press, 1989.

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28

King, Michael P. Hunt for the Hijacked Nerve Agent: A KD Thorne Thriller. Blurred Lines Press, 2021.

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29

King, Michael P. Hunt for the Hijacked Nerve Agent: A KD Thorne Thriller. Blurred Lines Press, 2021.

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30

Barkley, Harry. Lyrica: Super Action Medication for Treatment of Fibromyalgia, Nerve Pain and Epilepsy. Independently Published, 2019.

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31

King, Michael P. The Hunt for the Hijacked Nerve Agent: A KD Thorne Thriller. Blurred Lines Press, 2021.

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32

Matthews, Gary G. Cellular Physiology of Nerve and Muscle. 4th ed. Blackwell Publishing Limited, 2002.

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33

Zwarts, Machiel J. Nerve, muscle, and neuromuscular junction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0001.

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Essential to all living creatures is the ability to convey information. In addition motor responses are required, for example running. This all is possible due to the ability of specialized cells to conduct information along the cell membrane by means of action potentials (AP) made possible by the charged cell membrane, which has selective permeability for different ions. Voltage and ligand sensitive ion channels are responsible for sudden changes in selective permeability of the membrane resulting in local depolarization of the membrane. The neuromuscular junction is a highly specialized regi

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34

Owera-Atepo, Johnson Bolomor. The vagal nerve as a model for assessment of drug action on 5-hydroxy tryptamine receptors.: Measurements of drug action to influence 5-hydroxy tryptamine receptor function on the vagal nerve.... 1987.

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35

Cellular physiology of nerve and muscle. 4th ed. Blackwell Science, 2003.

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36

Cellular physiology of nerve and muscle. 3rd ed. Blackwell Science, 1998.

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37

Neural Network Models of Conditioning and Action. Taylor & Francis Group, 2016.

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38

Neural Network Models of Conditioning and Action. Taylor & Francis Group, 2016.

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39

Neural Network Models of Conditioning and Action. Taylor & Francis Group, 2016.

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40

Nurmikko, Turo J. Identification of the target of gabapentinoid action in neuropathic pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0070.

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The landmark paper discussed in this chapter is ‘Identification of the α‎2-δ‎-1 subunit of voltage-dependent calcium channels as a molecular target for pain mediating the analgesic actions of pregabalin’, published by Field et al. in 2006. In this seminal paper, Field et al. demonstrated that the anti-allodynic effect of pregabalin is related to its binding to the α‎2δ‎-1 subunit of the voltage-gated calcium channel. In transgenic mice lacking this subunit, pregabalin had no effect on allodynia induced by sciatic nerve ligation, whereas, in wild-type mice, there was a substantial anti-allodyni

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41

(Editor), Makoto Taketani, and Michel Baudry (Editor), eds. Advances in Network Electrophysiology: Using Multi-Electrode Arrays. Springer, 2006.

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42

Taketani, Makoto, and Michel Baudry. Advances in Network Electrophysiology: Using Multi-Electrode Arrays. Springer London, Limited, 2006.

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43

Taketani, Makoto, and Michel Baudry. Advances in Network Electrophysiology: Using Multi-Electrode Arrays. Springer, 2010.

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44

Katirji, Bashar. Routine Clinical Electromyography. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0002.

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Nerve conduction studies and needle EMG represent the two essential parts of the clinical EMG study. In almost all patients, both studies need to be completed before a final conclusion is made. This chapter outlines the basic concepts of nerve conduction studies including stimulations, recordings, variables and sources of errors. This is followed by detailed discussions of basic pathophysiological changes that accompany peripheral nerve disorders. The chapter then covers the normal needle EMG findings including normal insertional activity, motor unit action potential morphology and recruitment

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45

Katirji, Bashar. Case 9. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0013.

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Ulnar nerve lesions are the second most common mononeuropathies encountered in clinical practice. Although the majority of the lesions are due to entrapment/compression of the ulnar nerve around the elbow (at the cubital tunnel or ulnar groove), it is important to consider and exclude distal ulnar nerve lesions at the wrist or palm. This case highlights the clinical and electrodiagnostic findings of ulnar neuropathies at the wrist, including Guyon canal and pisohamate hiatus. It also discusses the electrodiagnostic challenges in distinguishing distal from proximal ulnar nerve lesions. Emphasis

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46

Rossi, Francesco M. A study on nerve growth factor (NGF) receptor expression in the rat visual cortex: Possible sites and mechanisms of NGF action in cortical plasticity. Edizioni della Normale, 2007.

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47

Borgennicht, D. Action Hero's Handbook: How to Catch a Great White Shark, Perform the Vulcan Nerve Pinch, Track a Fugitive, and Dozens of Other TV and Movie Skills. Quirk Books, 2002.

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48

Wyatt, Tristram D. 2. Sensing and responding. Oxford University Press, 2017. http://dx.doi.org/10.1093/actrade/9780198712152.003.0002.

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How an animal behaves is coordinated by nerves and hormones in different, complementary ways. Stimuli, such as the sound of a predator, cause fast behavioural responses coordinated by nerve signals. The stimuli also cause longer lasting physiological changes via hormones, which release energy sources needed for the muscle action required for escape. ‘Sensing and responding’ considers the sensory responses of bats and moths, and then explains selective sensitivity—how animals evolve to detect only what affects their survival or reproductive success. It also shows how the study of neural circuit

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49

Shaw, Pamela, and David Hilton-Jones. The lower cranial nerves and dysphagia. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0429.

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Disorders affecting the lower cranial nerves – V (trigeminal), VII (facial), IX (glossopharyngeal), X (vagus), XI (accessory) and XII (hypoglossal) – are discussed in the first part of this chapter. The clinical neuroanatomy of each nerve is described in detail, as are disorders – often in the form of lesions – for each nerve.Trigeminal nerve function may be affected by supranuclear, nuclear, or peripheral lesions. Because of the wide anatomical distribution of the components of the trigeminal nerve, complete interruption of both the motor and sensory parts is rarely observed in practice. Howe

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50

Katirji, Bashar. Case 15. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0019.

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Ulnar nerve lesions are the second most common mononeuropathies encountered in clinical practice. The majority of ulnar neuropathies are across the elbow, more specifically due to entrapment or compression of the ulnar nerve at the cubital tunnel or ulnar groove. This case highlights the clinical and electrodiagnostic findings of ulnar neuropathies across the elbow and discusses the challenges in making an accurate diagnosis. Focal slowing of conduction velocities and/or conduction block are the main findings that pinpoint the site of the lesion, while the needle electromyography is poor in ac

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