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Books on the topic 'Nerve injury'

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Published: 4 June 2021
Last updated: 30 January 2023

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1

Lundborg, Göran. Nerve injury and repair. Edinburgh: Churchill Livingstone, 1988.

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2

Hirasawa, Yasusuke, ed. Treatment of Nerve Injury and Entrapment Neuropathy. Tokyo: Springer Japan, 2002. http://dx.doi.org/10.1007/978-4-431-67883-0.

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3

Facial danger zones: Avoiding nerve injury in facial plastic surgery. St. Louis, Mo: Quality Medical Pub., 1994.

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4

Seckel, Brooke R. Facial danger zones: Avoiding nerve injury in facial plastic surgery. 2nd ed. St. Louis, Mo: Quality Medical Pub., 2010.

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5

Skouras, Emmanouil, Stoyan Pavlov, Habib Bendella, and Doychin N. Angelov. Stimulation of Trigeminal Afferents Improves Motor Recovery After Facial Nerve Injury. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-662-45789-4.

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6

Skouras, Emmanouil, Stoyan Pavlov, Habib Bendella, and Doychin N. Angelov. Stimulation of Trigeminal Afferents Improves Motor Recovery After Facial Nerve Injury. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-33311-8.

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7

1952-, Marwah J., Teitelbaum Herman, and Prasad Kedar N, eds. Neural transplantation, CNS neuronal injury, and regeneration: Recent advances. Boca Raton: CRC Press, 1994.

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8

Skouras, Emmanouil. Stimulation of trigeminal afferents improves motor recovery after facial nerve injury: Functional, electrophysiological and morphological proofs. Heidelberg: Springer, 2013.

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9

Enescu, Cristina. Methods of enhancing mechanical properties of hydrogel tubes used as nerve guidance channels in rat spinal cord injury. Ottawa: National Library of Canada, 2003.

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10

Steve, Parker. Nerves & spinal cord: Injury, illness and health. Oxford: Heinemann Library, 2003.

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11

Steve, Parker. Spinal cord and nerves: Injury, illness, and health. 2nd ed. Chicago, Ill: Heinemann Library, 2009.

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12

Dejarnette, Bertrand. Neurological reflex first aid: A reference manual based upon Chiropractic first aid. [Leadwood, KS]: Sacro Occipital Research Society, 2006.

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13

Hanna, Amgad S., Lisa M. Block, and A. Neil Salyapongse. Emergent Nerve Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0027.

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Injuries to peripheral nerves must be assessed and treated in a thorough and timely manner to achieve optimal outcomes. Physical examination is the cornerstone in diagnosing acute nerve injuries and includes careful inspection as well as precise motor and sensory testing. Nerve conduction studies and electromyography are often more useful in the setting of delayed presentation. Microsurgical repair techniques differ for clean versus ragged lacerations, and resultant nerve gaps will require a conduit or graft to achieve the necessary tension-free closure. The surgeon and patient should be prepared for a lengthy postoperative course and possible complications as the nerve regenerates and function returns.
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14

Payne, Russell A., and Elias B. Rizk. Axillary Nerve Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0024.

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Axillary nerve injury has been associated with sports injuries, especially those involving anterior shoulder dislocation. The nerve injury leads to weakness of the deltoid and teres minor muscles, which impairs abduction and external rotation of the arm at the shoulder. Electrodiagnostic studies are helpful for determining extent of reinnervation and recovery after injury. In the absence of clinical or electrodiagnostic signs of recovery 3 to 6 months after injury, it is appropriate to offer surgical exploration. The options for surgical repair include direct nerve repair, nerve grafting, and nerve transfer. In appropriately selected individuals, outcomes are favorable.
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15

Socolovsky, Mariano, Rafael Torino, and Leandro Pretto Flores. Facial Nerve Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0026.

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This chapter focuses on the clinical and surgical management of facial nerve palsy that occurs as a consequence of injury during resection of a vestibular schwannoma. If the facial nerve is damaged during cerebellopontine angle (CPA) tumor resection, a first attempt to repair it at the skull base should be made. Because this is commonly infeasible, a nerve transfer—scheduled as an elective procedure after the patient has completely recovered from the resection procedure—is mandatory. Hemihypoglossal, masseter, and cross-facial nerve transfers are the techniques most widely used. The authors’ preferred technique is hemihypoglossal nerve transfer, and the surgical technique is described. By contrast, when the facial nerve is preserved during surgery, but complete facial palsy develops afterward, postoperative rehabilitation should be started and continued for up to 1 year. If, however, facial palsy persists beyond 1 year, then the patient should be offered the option of a nerve transfer.
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16

Nerve injury and repair. Churchill Livingstone, 1988.

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17

Carlstedt, Thomas. Central Nerve Plexus Injury. Imperial College Press, 2007.

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18

Hems, T. E. J. Reconstruction after nerve injury. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.006009.

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♦ Late reconstructive procedures may improve function if there is persisting paralysis after nerve injury♦ Transfer of a functioning musculotendinous unit to the tendon of the paralysed muscle is the most common type of procedure♦ Passive mobility must be maintained in affected joints before tendon transfer can be performed♦ The transferred muscle should be expendable, have normal power, and have properties appropriate to the function it is required to restore♦ Tendon transfers can provide reliable improvement in function after isolated radial nerve palsy♦ A number of procedures have been described for reconstruction of thumb opposition but impaired sensation after median nerve injury may limit gain in function♦ Tendon transfers are possible to improve clawing of fingers and lateral pinch of the thumb after ulnar nerve palsy or other cases of intrinsic paralysis.
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19

Chan, Kevin, Rishi Dihr, and Michael Fox. Spinal Accessory Nerve Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0025.

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Spinal accessory nerve (SAN) injuries can be idiopathic or iatrogenic. Providers who understand the essential anatomy of the SAN can direct the history, physical exam, and ancillary studies to localize the lesion, while considering the differential diagnosis. The differential diagnosis includes both traumatic and atraumatic causes, including penetrating or blunt trauma to the neck, fracture malunion, glenohumeral instability, brachial neuritis, progressive neuromuscular disease, and cerebrovascular accident. The chapter discusses the timing of, and indications for, operative exploration, with or without nerve repair, as well as the details of the surgical procedure. The authors provide instructive pearls for initial management, establishing patient expectations, and potential complications.
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20

Heinen, Christian, and Thomas Kretschmer. Iatrogenic Peripheral Nerve Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0028.

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Iatrogenic nerve lesions are frequently neglected. The chapter stresses the importance of adequate assessment, surgical timing, surgical strategies, follow-up, and results. Using the example of a radial nerve lesion in discontinuity due to osteosynthesis after humeral fracture, the authors describe a typical patient history with delayed presentation, as well as the role of physical examination, electrophysiology, and high-resolution ultrasound in demonstrating substantial nerve damage incompatible with spontaneous recovery. Surgical findings are demonstrated, along with a stepwise approach for nerve reconstruction via sural nerve graft. Clinical approach and surgery for traumatic radial nerve lesions are detailed, as well as general information on iatrogenic nerve lesions.
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21

Nerve Injury and Repair. Elsevier, 2004. http://dx.doi.org/10.1016/b978-0-443-06711-2.x5001-x.

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22

Hafez, Daniel, Adam Bevan, and Wilson Z. Ray. Nerve Transfers for Spinal Cord Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0029.

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In spinal cord injury, nerve transfers represent a potential adjunct in the comprehensive clinical management of patients. Unlike tendon transfers, nerve transfers preserve the native muscle biomechanics and provide greater than a 1:1 functional exchange. Nerve transfers can provide improved upper extremity function by capitalizing on the preserved upper motor neurons below the zone of spinal cord injury. One goal in reconstruction is to restore movement. Major movements that have been targeted for restoration include elbow extension (to allow the patient to assist in transfers) and pinch, grasp, and release, which can aid in controlling a motorized wheelchair as well as in feeding oneself. Other major goals are restoration of hand sensation and diaphragm reinnervation to allow ventilator weaning and spontaneous respiration.
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23

Treatment of Nerve Injury and Entrapment Neuropathy. Springer, 2012.

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24

Peripheral Trigeminal Nerve Injury Repair And Regeneration. W.B. Saunders Company, 2011.

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25

Hirasawa, Y. Treatment of Nerve Injury and Entrapment Neuropathy. Springer London, Limited, 2012.

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26

Hirasawa, Y. Treatment of Nerve Injury and Entrapment Neuropathy. Springer, 2002.

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27

Giddins, Grey. Nerve injuries. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.012025.

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♦ Nerve injuries are common♦ The history is usually clear but the examination may be less so♦ Neurophysiology is only useful after 3 weeks♦ Imaging is of limited value as yet♦ Treatment is removal of the cause/repair as appropriate♦ Recovery is dependent upon many factors especially patient age, severity of the injury, how proximal the injury is and the type of nerve♦ Post operative therapy is crucial♦ Late reconstruction is primarily for motor function.
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28

Humble, Stephen R. Plasticity in somatic receptive fields after nerve injury. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0023.

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Devor and Wall, in a pioneering electrophysiological study, examined the change in somatic receptive fields in the dorsal horn of the spinal cord after nerve injury. Rather than the anticipated loss of an area of electrophysiological perception, the system demonstrated ‘plasticity’ whereby novel receptive fields, remote to the corresponding area of damage, were evident. The authors postulated that this neuroplasticity occurred via a hitherto undefined spinal mechanism, which lead to an explosion of interest and research to elucidate the mechanisms of central plasticity. In this truly landmark paper, the idea of the nervous system being an inherently ‘hard-wired’ structure was made redundant and the concept of neuroplasticity was given robust form.
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29

Spinner, Robert J., Marios Loukas, R. Shane Tubbs, Mohammadali M. Shoja, and Elias Rizk. Nerves and Nerve Injuries : Vol 2: Pain, Treatment, Injury, Disease and Future Directions. Elsevier Science & Technology Books, 2015.

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30

Loukas, Marios, R. Shane Tubbs, Elias Rizk, Nicholas Barbaro, and Mohammadali Shoja. Nerves and Nerve Injuries : Vol 2: Pain, Treatment, Injury, Disease and Future Directions. Elsevier Science & Technology Books, 2015.

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31

Lundborg, Goran. Nerve Injury and Repair: Regeneration, Reconstruction, and Cortical Remodeling. 2nd ed. Churchill Livingstone, 2005.

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32

Echeverry, S. Contribution of Inflammation to Chronic Pain Triggered by Nerve Injury. INTECH Open Access Publisher, 2012.

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33

Seckel, Brooke. Facial Danger Zones: Avoiding Nerve Injury in Facial Plastic Surgery. Thieme Medical Publishers, Incorporated, 2010.

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34

Nadi, Mustafa, and Rajiv Midha. Adult Total Brachial Plexus Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0021.

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Total brachial plexus injury (BPI) typically results from high-energy vehicular accidents, affects mostly young adult males, and produces a flail, insensate limb. Because of the association of total BPI with head and cervical spine injuries, diagnosis might be delayed. Recognizing patients with total BPI and using electrodiagnostic and imaging tests in a timely fashion are critical. Advances in microsurgical techniques, primary nerve transfer, appropriate nerve graft utilization from a remaining intact (often C5) spinal nerve root, and free muscle transfers have improved outcomes. However, limited recovery even after reconstruction and severe deafferentation pain both remain challenging problems that further advancements will need to overcome.
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35

J, Seil Fredrick, ed. Neural injury and regeneration. New York: Raven Press, 1993.

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36

Pisapia, Jared M., Zarina S. Ali, Gregory G. Heuer, and Eric L. Zager. Adult Upper Trunk Brachial Plexus Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0022.

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This chapter takes a case-based approach to the diagnosis and management of adult brachial plexus injury involving the upper trunk. The clinical presentation and differential diagnosis associated with this injury pattern are reviewed, as well as the findings of electrodiagnostic and imaging studies. Preoperative considerations include the timing from initial injury and the difference between pre- and postganglionic injury. Options for nerve reconstruction include nerve grafting, nerve transfer, or a combination of both. The options are compared, and a detailed description of each surgical procedure is provided, along with related complications, alternative repair strategies, and outcomes.
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37

Leung, Albert. Peripheral Nerve Injury and Pain: Epidemiology, Mechanisms, Rehabilitation and Treatment Guidelines. Nova Science Publishers, Incorporated, 2019.

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38

Peripheral Nerve Injury: An Anatomical and Physiological Approach for Physical Therapy Intervention. F.A. Davis Company, 2015.

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39

Filler, Aaron G. Piriformis Syndrome and Other Nerve Entrapments of the Posterior Pelvis. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0011.

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Not every case of neurologically based pelvic/genital numbness/incontinence is due to cauda equina syndrome. Pelvic pain, incontinence, and sexual dysfunction can result from treatable peripheral nerve injury or entrapment affecting the pudendal nerves or impar ganglion. Learning the signs, physical exam findings, tests, and surgical options greatly expands a neurosurgeon’s range. The pudendal nerve and nerve to the obturator internus muscle arise after S2, S3, and S4 spinal nerves traverse the piriformis muscle. They exit the sciatic notch with the sciatic nerve but then re-enter the pelvis, where the pudendal nerve then gives off bladder, rectal, and genital branches.
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40

Crick, Alexandra, David Warwick, and Roderick Dunn. Nerves. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757689.003.0011.

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Examination of the nerves of the upper limb and localization of nerve lesions is mysterious to the unfamiliar. This chapter provides a scheme for the neuroanatomy of the upper limb, and for examination and investigation of nerve pathology including a section on neurophysiology. We discuss nerve injury, including pathophysiology and recovery. We describe common compression neuropathies affecting the median, ulnar, and radial nerves, and the brachial plexus lesions including thoracic outlet syndrome. Common tendon transfers are discussed for reconstruction following peripheral nerve injury or other loss of peripheral nerve function, and also for spinal injury at different levels.
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41

Brown, Matthew. The chronic constriction injury model of neuropathic pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0067.

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The landmark paper discussed in this chapter is ‘A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man’, published by Bennett and Xie in 1988. This paper, in which the unilateral sciatic nerve chronic constriction injury (CCI) model was first presented, is one of the earliest and most comprehensive descriptions of a specific animal paradigm that was designed to model human neuropathic pain. The authors realized that human neuropathic pain rarely involves nerve transection but instead involves evoked changes in damaged and preserved nerve fibres. Furthermore, they systematically applied a barrage of sensory testing that demonstrated quantifiable hyperalgesia and cold allodynia reflecting some of the clinical observations of human neuropathic pain phenotype. CCI provided a high-quality template for the development of neuropathic pain models that impelled the subsequent development of other animal models striving to replicate the human condition faithfully and accurately.
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42

Gupta, Rajnish K., and Alexandria N. Nickless. Nerve Injuries from Positioning and Regional Blocks. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0074.

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Peripheral nerve injury in the perioperative period can have a variety of etiologies, including preexisting patient factors and by surgical and anesthetic complications such as intraoperative positioning and nerve blockade. The actual incidence may be difficult to assess, because most nerve injuries resolve with time and frequently require minimal to no intervention. Injuries often manifest more than 48 hours after surgery and have even been noted in patients who undergo awake procedures and in hospitalized patients who never undergo surgery. This should not negate the fact that close attention to detail when positioning patients and performing regional anesthesia may help decrease the overall incidence of nerve injury and should be considered in every anesthesiologist’s perioperative plan. This chapter reviews proper assessment, treatment, and follow-up for peripheral nerve injuries.
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43

Warwick, David, Roderick Dunn, Erman Melikyan, and Jane Vadher. Nerves. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199227235.003.0011.

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Neuroanatomy 298Examination of the nerves of the upper limb 300Clinical assessment 304Neurophysiology tests 306Nerve injury 310Compression neuropathy 314Carpal tunnel syndrome 315Proximal compression of the median nerve 318Anterior interosseous nerve syndrome 319Ulnar nerve compression at the elbow ...
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44

N, Johannessen Jan, ed. Markers of neuronal injury and degeneration. New York, N.Y: New York Academy of Sciences, 1993.

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45

James, Nicholas D., and Elizabeth J. Bradbury. Autotomy. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0065.

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The landmark paper discussed in this chapter is ‘Autotomy following peripheral nerve lesions: Experimental anaesthesia dolorosa’, published by Wall et al. in 1979. This paper was the culmination of a series of studies in which Wall, together with a number of colleagues, investigated the underlying causes of neuropathic pain following peripheral nerve injury. In this paper, the authors used a variety of nerve injury models to show that the extent of resultant anaesthesia combined with ectopic firing from damaged axons in nerve-end neuromas correlated with the severity of self-mutilation (termed ‘autotomy’) observed in the affected hindlimb. The authors therefore suggested that these simple models might be suitable for studies of the prevention of irritations originating from chronic lesions of peripheral nerves. Indeed, this proved to be the case, sparking the development of numerous animal models of spontaneous pain following nerve injury and spawning a new field of neuropathic pain research.
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46

Sears, T. A. Neuronal-Glial Cell Interrelationships : Report of the Dahlem Workshop on Neuronal-glial Cell Interrelationships: Ontogeny, Maintenance, Injury, ... 30 - December 5. Dr. S. Bernhard, Dahlem Konferenzen, 2011.

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47

Mullins, Gerard, and Julian Ray. Neurophysiological investigation of injuries sustained in sport. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199533909.003.0013.

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The continued growth of recreational and competitive sports has been accompanied by an increased incidence of nerve injuries that have been traditionally associated with other types of occupational injury (Krivickas and Wilbourn 1998). Peripheral nerves are susceptible to injury in the athlete because of excessive physiological demands (...
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48

Skouras, Emmanouil, Stoyan Pavlov, and Habib Bendella. Stimulation of Trigeminal Afferents Improves Motor Recovery After Facial Nerve Injury: Functional, Electrophysiological and Morphological Proofs. Springer, 2012.

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49

Manickam, S. Effect of Vata Gajankusha Rasa in Rat Sciatic Nerve Crush Injury: Validating Ayurveda Through Scientific Methods. Notion Press, Inc., 2021.

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50

Katirji, Bashar. Case 1. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0005.

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Peripheral nerve injuries are most prevalent in young adults and are sometimes overlooked by medical staff caring for trauma patients. They are classified based on damage to myelin or axon and degree of supporting structures injury. The diagnosis of these injuries is often aided by electrodiagnostic studies. The case illustrates a patient with peripheral nerve injury (more specifically, a common peroneal [fibular] nerve injury) and highlights the anatomy of peripheral nerve and classification of peripheral nerve injury. It emphasizes the role and challenges of electrodiagnostic studies, including nerve conduction studies and needle electromyography, in the diagnosis, localization, prognosis, and management of peripheral nerve injury.
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