Relevant bibliographies by topics / Nesidioblastose

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Nesidioblastose'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "Nesidioblastose"

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Klaus, Daniele Gehlen, Diélly Cunha de Carvalho, and Jaime César Gelosa Souza. "Hipoglicemia por nesidioblastose: uma complicação rara da cirurgia bariátrica?" ABCD. Arquivos Brasileiros de Cirurgia Digestiva (São Paulo) 20, no. 4 (2007): 280–82. http://dx.doi.org/10.1590/s0102-67202007000400013.

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INTRODUÇÃO: A cirurgia bariátrica tem-se mostrado o método mais eficaz na resolução do diabetes mellitus tipo II, chegando a resultados superiores a 80% de cura da doença. Contudo, alguns estudos têm mostrado estado de hiperinsulinemia com hipoglicemia em certos pacientes obesos mórbidos após a realização da cirurgia bariátrica, ao que se denominou nesidioblastose, que corresponde ao hipercrescimento patológico das células beta pancreáticas. O objetivo desse estudo é realizar revisão bibliográfica sobre nesidioblastose pós-cirurgia bariátrica, através da pesquisa por artigos científicos publicados a partir de 2000. MÉTODOS: Realizou-se revisão bibliográfica de artigos científicos publicados a partir de 2000. A base de dados pesquisada foi o PubMed, através do site www.pubmed.gov. cruzando-se os descritores Nesidioblastose. Hiperinsulinismo. Hipoglicemia. Cirurgia bariátrica, complicações. REVISÃO DE LITERATURA: Revisou-se 14 publicações encontradas com o tema. Dentre elas, Cummings defende que após a realização da cirurgia bariátrica, ocorrem alterações no trânsito intestinal gerando estimulação e crescimento excessivo das células beta-pancreáticas e maior atividade dos hormônios pancreáticos, sugerindo possível causa para a nesidioblastose. Corroborando para desvendar a causa dessa doença, autores sugerem que o rápido trânsito alimentar duodenal após o bypass gástrico, eleva os níveis da peptídeo glucagonóide 1 e estimula, assim, a hiperinsulinemia hipoglicêmica nesses pacientes. De forma semelhante, outros defendem que a perda de peso após a cirurgia bariátrica, reduz muito a resistência à insulina devido à hipertrofia e hiperfunção das células beta-pancreáticas, o que é muito comum na obesidade, gerando a nesidioblastose. CONCLUSÃO: A nesidioblastose após cirurgia bariátrica pode representar o extremo patológico de um fenômeno benéfico para a maioria dos pacientes obesos mórbidos com diabetes do tipo II, porém em alguns casos raros esse fenômeno benéfico gera malefícios, ou seja, a nesidioblastose, que deve ser corrigida pela ressecção parcial do pâncreas, sob pena de ameaçar a vida desses pacientes.
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Hümmer, H., H. Böhles, J. Giedl, and R. Schück. "7/8 Pankreasresektion bei Nesidioblastose?" European Journal of Pediatric Surgery 43, no. 04 (1988): 281–83. http://dx.doi.org/10.1055/s-2008-1043470.

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Costa, Raquel R., Frederico F. R. Maia, and Levimar R. Araújo. "Hipoglicemia hiperinsulinêmica persistente endógena no adulto: relato de caso." Arquivos Brasileiros de Endocrinologia & Metabologia 51, no. 1 (2007): 125–30. http://dx.doi.org/10.1590/s0004-27302007000100020.

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A hipoglicemia hiperinsulinêmica persistente endógena em adultos é, na maioria dos casos, causada por insulinoma. A Nesidioblastose, uma hiperinsulinemia funcional rara por hiperplasia das células beta do pâncreas, tem sido descrita principalmente em neonatos. Apresentamos o caso de uma paciente de 34 anos com quadro clínico-laboratorial compatível com hipoglicemia hiperinsulinêmica endógena (Glicemia jejum: 54 mg/dl / Valor de referência (VR): 6099 mg/dl; Insulina sérica: 70,9 mcU/ml / VR: < 29,1 mcU/ml; e Peptídeo C: 7,1 ng/ml / VR: 1,15,0 ng/ml - simultâneos à glicemia). Foi aventada a hipótese de insulinoma. Em função da ausência de imagem característica aos exames radiológicos (ultra-som e tomografia de abdome), optou-se pela laparotomia exploradora, onde também não foi evidenciado tumor pancreático macroscopicamente. Os exames histopatológico e imuno-histoquímico evidenciaram hiperplasia de células beta, de segmento distal do pâncreas, compatível com nesidioblastose. A paciente evoluiu com estabilidade clínica por cerca de dois meses, quando se verificou recidiva das crises hipoglicêmicas, refratárias ao uso de Octreotide. Optou-se pela realização de "octreosan", que indicou nesidioblastose difusa, sendo procedida pancreatectomia parcial extensa. Seguiu-se o uso contínuo de Octreotide, com controle eficaz das crises hipoglicêmicas. Uma vez que esse é um diagnóstico raro no adulto, objetiva-se, nesse artigo, divulgar o manejo diagnóstico-terapêutico em casos de hipoglicemia hiperinsulinêmica endógena.
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Schatz, M., A. Wolf, K. H. Deeg, R. Meister, and G. Seitz. "Beidseitige Nebennierenblutung und Nesidioblastose eines makrosomen Neugeborenen." Monatsschrift Kinderheilkunde 147, no. 6 (1999): 557–61. http://dx.doi.org/10.1007/s001120050459.

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Kunz, Von Jochen, Holger Haberland, Andrea Schmidt, and Peter Amendt. "Immunhistochemischer Chromogranin A-Nachweis und morphometrische Befunde bei frühkindlicher Nesidioblastose." Acta Histochemica 89, no. 2 (1990): 131–40. http://dx.doi.org/10.1016/s0065-1281(11)80349-1.

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Henzen. "Hypoglykämien bei der Frau eines Diabetikers." Therapeutische Umschau 69, no. 12 (2012): 667–71. http://dx.doi.org/10.1024/0040-5930/a000346.

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Insulinome sind mit einer Inzidenz von 4 pro 100'000 eine seltene Erkrankung, vermeintliche Hypoglykämie-Symptome werden dagegen häufig auch von Menschen ohne Diabetes mellitus beschrieben. Die Liste der möglichen Ursachen ist sehr umfangreich, weshalb mittels Anamnese, Klinik und laborchemischen Untersuchungen der Verdacht auf ein Insulinom erhärtet werden bzw. ausgeschlossen werden muss. Wie bereits vor über 70 Jahren der New Yorker Chirurge Allen Whipple gefordert hat, ist eine strikte Beachtung der Trias: dokumentierte Hypoglykämie und Neuroglykopenie und Besserung auf Glukosezufuhr Voraussetzung für weitere diagnostische und therapeutische Schritte. Die hyperinsulinämische Hypoglykämie wird im Fastentest bewiesen, Ziel der bildgebenden Methoden ist die Lokalisation des Insulinoms für die chirurgische Resektion bzw. die Abgrenzung zur nicht-Insulinom bedingten pankreatogenen Hypoglykämie (Nesidioblastose). Als neue und hochsensitive Methode hat sich die GLP-1 Rezeptor Szintigraphie erwiesen, womit auch im vorliegenden Fallbeispiel eine zielgerichtete chirurgische Intervention möglich war.
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McElroy, Michele K., Andrew M. Lowy, and Noel Weidner. "Case report: focal nesidioblastosis (“nesidioblastoma”) in an adult." Human Pathology 41, no. 3 (2010): 447–51. http://dx.doi.org/10.1016/j.humpath.2009.09.002.

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Treiber, Gerlies. "Nicht-Diabetes-assoziierte Hypoglykämien." Journal für Klinische Endokrinologie und Stoffwechsel 13, no. 4 (2020): 177–81. http://dx.doi.org/10.1007/s41969-020-00124-7.

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ZusammenfassungDie Symptome einer Hypoglykämie sind häufig und unspezifisch. Echte Hypoglykämien sind selten und kommen meistens im Rahmen einer Insulintherapie vor. Die Diagnose einer Hypoglykämie bei Patienten ohne Diabetes erfordert das Vorliegen der Whipple-Trias mit dokumentierter Blutglukose ≤ 55 mg/dl, hypoglykämischen Symptomen und deren Verschwinden durch Zufuhr von Kohlenhydraten. Eine weitere Abklärung erfolgt mittels standardisiertem 72‑h Fastentest oder bei Patienten mit vorwiegend postprandialen Hypoglykämien einem 5‑h-Mahlzeitentoleranztest. Die parallele Messung von Plasmaglukose, Insulin, C‑Peptid, Proinsulin und Ketonen hilft in der Differenzierung zwischen Insulinom, faktitieller Gabe von Insulin und Sulfonylharnstoffen sowie Nesidioblastose (NIPHS) und Hypoglykämien nach bariatrischen Operationen. Die Messung von Insulin-Autoantikörpern ist hinweisend auf ein autoimmunes Insulinsyndrom und der Nachweis von „big“ IFG‑2 auf eine paraneoplastische Sekretion dieses Proteins bei Tumorerkrankungen. Erst wenn der Nachweis einer autonomen endogenen Hyperinsulinämie vorliegt, erfolgt die Lokalisationsdiagnostik. Dabei sind szintigrafische Untersuchungen mittels radioaktivem 68Gallium und Exendin‑4 und ein selektiver Kalzium-Stimulationstest in der Lokalisation von Insulinomen hilfreich. Therapeutisch ist eine Hypoglykämie akut durch Glukosegabe zu behandeln. Die langfristige Therapie besteht wenn möglich in der Beseitigung der auslösenden Ursache, Ernährungsmaßnahmen sowie medikamentöser Hemmung der Insulinsekretion.
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Caboclo, Edmundo Di Giaimo, and Moacyr Vergara de Godoy Moreira. "Mudança de função para atividade compatível de uma funcionária com restrição: relato de um caso de nesidioblastose após realização de cirurgia bariátrica." Revista Brasileira de Medicina do Trabalho 16, s1 (2018): 62–63. http://dx.doi.org/10.5327/z16794435201816s1p41.

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R., Madhumathi, Mohan C. N., and Prabhu S. "Islets of hypoglycaemia: a rare case of adult onset nesidioblastosis." International Journal of Advances in Medicine 7, no. 1 (2019): 185. http://dx.doi.org/10.18203/2349-3933.ijam20195669.

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A case of diffuse nesidioblastosis in an adult patient is reported in this study. A 24-year-old female with no known comorbidities presented with multiple episodes of documented recurrent hypoglycaemia and Hypoglycaemia induced seizures both in fasting and postprandial state. Her blood investigations revealed low plasma glucose levels, high insulin and C-peptide levels with positive 72-hour fast test. Her transabdominal USG and CECT abdomen did not reveal any abnormality, 68Ga DOTANOC PET CT done showed ill-defined diffuse somatostatin receptor expression in the pancreatic head and tail suggestive of nesidioblastosis. As patient was not willing for surgical treatment, hence started on medical treatment with oral nifedipine. Nesidioblastosis is very rare in adults. It is an important differential diagnosis in adults with hyperinsulinemic hypoglycemia although the incidence is very rare in adults. PET SCAN was used to non-invasively diagnose nesidioblastosis in this case. Surgery being the preferred choice of treatment in nesidioblastosis, there is limited data on medical line of management in nesidioblastosis.
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Dissertations / Theses on the topic "Nesidioblastose"

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PROTAT, MADAR HELENE. "Nesidioblastose neo-natale : revue generale a propos d'un cas." Nice, 1989. http://www.theses.fr/1989NICE6028.

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Kemper, Ines. "Vergleich der pränatalen, sonografischen Subkutangewebsmessung mit der postnatalen, mechanischen Hautfaltenmessung unter besonderer Berücksichtigung des maternalen Kohlenhydratstoffwechsels /." Hamburg : Akademos Wiss.-Verl, 2003. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=013149099&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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Delfau, Christine. "La nésidioblastose néonatale : à propos de deux observations." Bordeaux 2, 1994. http://www.theses.fr/1994BOR23090.

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Wieben, Daniel. "Pathologie der persistierenden hyperinsulinämischen Hypoglykämie bei Erwachsenen mit diffuser Nesidioblastose." 2006. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=015432723&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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Book chapters on the topic "Nesidioblastose"

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Froehlich, Stephan J., Carlo A. Lackerbauer, Guenter Rudolph, et al. "Nesidioblastosis." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8800.

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Sipos, Bence. "Nesidioblastosis." In Encyclopedia of Pathology. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-28845-1_5279-1.

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Braun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, et al. "Pancreatic Nesidioblastosis." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6564.

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Dobroschke, J., R. Linder, and A. Otten. "Surgical Treatment of Nesidioblastosis in Childhood." In Surgery for Endocrinological Diseases and Malformations in Childhood. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-88324-8_10.

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Willberg, B., and E. Müller. "Surgery for Nesidioblastosis — Indications, Treatment and Results." In Surgery for Endocrinological Diseases and Malformations in Childhood. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-88324-8_9.

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Al-Salem, Ahmed H. "Nesidioblastosis: Persistent Hyperinsulinemic Hypoglycemia of Infancy (PHHI)." In Atlas of Pediatric Surgery. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-29211-9_27.

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Dohrmann, P., W. Mengel, and J. Splieth. "Total Pancreatectomy in a Case of Nesidioblastosis Due to Persisting Hyperinsulinism Following Subtotal Pancreatectomy." In Surgery for Endocrinological Diseases and Malformations in Childhood. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-88324-8_11.

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"Nesidioblastosis." In Diagnostic Pathology: Hepatobiliary and Pancreas. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-323-44307-4.50099-3.

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"Nesidioblastosis." In Diagnostic Pathology: Pediatric Neoplasms. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-54806-9.50149-x.

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Karanchi, Harsha, Omaima M. Sabek, Daniel W. Fraga, et al. "Recurrent Hyperinsulinemic Hypoglycemia after Successful Distal Pancreatectomy for Post Gastric Bypass Nesidioblastosis." In The Endocrine Society's 93rd Annual Meeting & Expo, June 4–7, 2011 - Boston. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part4.p14.p3-738.

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Conference papers on the topic "Nesidioblastose"

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Alves, Matheus Sousa, Juliana de Ponte Souza Pereira, and Salma Saraty Malveira. "HIPOGLICEMIA HIPERINSULINÊMICA PERSISTENTE DA INFÂNCIA - NESIDIOBLASTOSE: RELATO DE CASO NO HOSPITAL REGIONAL PÚBLICO DO ARAGUAIA." In 4º Congresso Internacional Sabará de Saúde Infantil. Editora Blucher, 2020. http://dx.doi.org/10.5151/cissi2020-31.

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