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Journal articles on the topic 'Neurasthenia'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 June 2025

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1

Goetz, Christopher G. "Poor Beard!!" Neurology 57, no. 3 (2001): 510.1–514. http://dx.doi.org/10.1212/wnl.57.3.510.

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Objective: To analyze the role of the seminal 19th-century neurologist, Jean-Martin Charcot, in the internationalization of neurasthenia, previously known as “the American disease.”Background: The New York neurologist, George Beard, first described neurasthenia in 1869 and considered it a disorder related to the particular stress of modern civilization, most typified in the United States.Methods: Charcot’s personal files on neurasthenia from the Bibliothèque Charcot, Salpêtrière Hospital, Paris, were studied and his teaching lessons and lectures were examined.Results: Charcot presented numerous cases of neurasthenia, always crediting Beard with the original name and description. Calling Beard’s 1880 work a “remarkable monograph,” Charcot emphasized that patients with neurasthenia also comprised the bulk of his own private practice. Focusing on the signs of sexual impotency, fatigue, and a tight band of pressure around the head, Charcot categorically distinguished patients with neurasthenica from patients with hysteria. Like Beard, Charcot concluded that the origin of neurasthenia was psychological stress and felt the European society also fostered the environment to precipitate the disease. Charcot adamantly opposed extrapolations that called for early childhood educational reforms to reduce current classroom stress. Charcot sympathized more with the treating physician than the patient, calling neurasthenics insufferable (insupportables). On the front sheet of his neurasthenia file, he wrote in large script, “Poor Beard!!”Conclusion: By emphasizing the prevalence of neurasthenia and extending Beard’s observations, Charcot internationally legitimized the new diagnosis. Adding neurasthenia to the other neurologic descriptions from the United States by Hammond, Mitchell, and Dana, Charcot helped to foster the recognition of the American Neurologic School.

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2

Merikangas, K., and J. Angst. "Neurasthenia in a longitudinal cohort study of young adults." Psychological Medicine 24, no. 4 (1994): 1013–24. http://dx.doi.org/10.1017/s0033291700029093.

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SynopsisThis study examines the concept of neurasthenia in a longitudinal cohort of young adults selected from a community sample of the canton of Zurich, Switzerland. The major focus is on the validity of the case definition of neurasthenia. Close approximations of the proposed descriptive and research definitions of the ICD-10 are employed as well as the concept of ‘irritable weakness’ as described in 1831 by Kraus (1926–1932).The prevalence of neurasthenia defined according to the ICD-10 criteria was: 1% across 10 years and 0·9% in 1988 for a duration criterion of ≥ 3 months; and 8·1% across 10 years and 12% in 1988 for a duration criterion of ≥ 1 month. The duration criterion of ≥ 3 months appeared to be excessively restrictive to represent individuals with neurasthenia in the community. Subjects with 1 month episodes of neurasthenia exhibited sufficient differences from controls and similarities to subjects with anxiety or depressive disorders to justify a 1 month duration criterion for neurasthenia in community samples.The clinical significance of neurasthenia was indicated by the magnitude of subjective distress, and occupational and social impairment reported by the majority of the cases. Prospective assessment of the longitudinal course of neurasthenia revealed that approximately 50% of the cases continued to exhibit this disorder at follow-up. Our findings suggest that neurasthenia is equally likely to represent an early manifestation of affective illness as it is a consequence in those neurasthenic subjects who exhibited comorbid affective disorders. The magnitude, chronicity, impairment, longitudinal stability and distinction from anxiety and depression associated with this condition in the general population, suggest that neurasthenia is an important diagnostic entity for which additional validation studies should be undertaken.

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3

Hall, Wayne, and Donald Macphee. "Do Vietnam Veterans Suffer from Toxic Neurasthenia?" Australian & New Zealand Journal of Psychiatry 19, no. 1 (1985): 19–29. http://dx.doi.org/10.3109/00048678509158810.

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This paper evaluates the claim that Vietnam veterans with psychiatric disorders are suffering from toxic neurasthenia — a neurasthenic syndrome caused by exposure to pesticides while serving in Vietnam.

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4

JUNG, Yeonsik. "“Our one great national malady”: Neurasthenia and American Imperial and Masculine Anxiety at the Turn of the Twentieth Century." Korean Journal of Medical History 30, no. 2 (2021): 393–432. http://dx.doi.org/10.13081/kjmh.2021.30.393.

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White upper middle-class Americans at the turn of the twentieth century were entrenched in a battle with a newly discovered, or invented, mental illness called neurasthenia. This essay examines the ways in which the medical discourse of neurasthenia reflected late nineteenth- and early twentieth-century white Anglo-Saxon men’s belief in, as well as anxiety over, American values bolstered by their idea of cultural, racial, and sexual superiority and consolidated through a conjunction of medicine and politics. The idea of neurasthenia as white American men’s malady functioned as a mark both of whites’ racial superiority to the “new” immigrants and African Americans as well as of women’s intellectual inferiority to the opposite sex of their own race. Imposing a subtle distortion on the etiology and diagnosis of neurasthenia and associating it with specific groups of people, the “American disease” constituted the era’s representative pathological symptoms which addressed Anglo-Saxon American men’s anxieties about overcivilized effeminacy and racial and national decadence which was originated as a response to the racial and sexual heterogeneity. This essay also argues that neurasthenia was an imagined disease which addressed late nineteenth-century American men’s spatial anxiety about the decline of the American pastoral ideal caused by the closure of the frontier. Given that the treatment for neurasthenic men was an escape to the frontier in the West in which they could rejuvenate withered American masculinity, their uneasiness about barbarous, unhygienic, and prolific immigrants and unruly white women, in fact, was tied to their spatial anxiety which symptomatically signifies the crisis of American masculinity. Channeled through the medical knowledge of neurology, it made American men’s racial, sexual, and spatial anxieties function to act out their racist, misogynist, nativist, and imperialist impulses which legitimized exclusionary political techniques toward the racial and sexual others such as the U.S. imperial expansion in the 1890s and 1900s and a eugenic-influenced immigration policy from the 1900s through the1920s. In this sense, the decline of neurasthenia around 1920 should not be attributed solely to the continued efforts to professionalize American medicine accompanied by recent discoveries of chemical factors such as hormones and vitamins and the rise of psychiatry and psychology which offered physicians with a more specific theory of health built on clinical laboratory science. Like its rise, the decision to move away from the neurasthenic diagnosis was rather a cultural phenomenon, which reflected the American ascendancy to global power in the early twentieth century, particularly after the First World War. Sustaining a political order rested on racial and sexual hierarchies both within and outside the American continent, American men felt that they were no longer liable to specific, time-tested anxiety and somatic symptoms of neurasthenia, which was more an ideological and cultural construct than a clinical entity that dramatizes the racial, sexual, and imperial politics of the-turn-of-the-twentieth-century America.

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5

Creed, Francis, Joy Ratcliffe, Lakshmi Fernandes, et al. "Outcome in severe irritable bowel syndrome with and without accompanying depressive, panic and neurasthenic disorders." British Journal of Psychiatry 186, no. 6 (2005): 507–15. http://dx.doi.org/10.1192/bjp.186.6.507.

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BackgroundIrritable bowel syndrome often leads to impaired functioning.AimsTo assess the contribution of psychiatric disorders to impaired outcome in severe irritable bowel syndrome.MethodPatients with severe irritable bowel syndrome entering a psychological treatment trial (n=257) were interviewed using the Schedules for Clinical Assessment in Neuropsychiatry. Outcomes were number of days of restricted activity, role limitation (physical) score of the Short Form Health Survey and costs.ResultsAt baseline, depressive disorder (29% of patients), panic (12%) and neurasthenia (35%) were associated with impairment; number of psychiatric disorders was associated in a dose–response fashion (P=0. 005). At follow-up, depressive disorder and neurasthenia were associated with role limitation score. Improved depression was associated with improved role functioning.ConclusionsDepressive, panic and neurasthenic disorders contribute to poor outcomes in severe irritable bowel syndrome, and appropriate treatment should be available to these patients.

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6

Bynum, Bill. "Neurasthenia." Lancet 361, no. 9370 (2003): 1753. http://dx.doi.org/10.1016/s0140-6736(03)13332-6.

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7

Overholser, James C., and Eleanor E. Beale. "Neurasthenia." Journal of Nervous and Mental Disease 207, no. 9 (2019): 731–39. http://dx.doi.org/10.1097/nmd.0000000000000943.

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8

LOVE, I. N. "NEURASTHENIA." JAMA: The Journal of the American Medical Association 271, no. 16 (1994): 1242. http://dx.doi.org/10.1001/jama.1994.03510400028025.

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9

Ye, Lin, Gu Zhu, Nick Martin, and Yangyang Liu. "The Relationship Between Adolescents’ Personality and Neurasthenia: A Comparison of Australian and Chinese." Journal of Early Adolescence 39, no. 9 (2019): 1337–42. http://dx.doi.org/10.1177/0272431618824710.

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The aim of the present study was to examine the cross-cultural differences in the relationship between personality traits and neurasthenia across early adolescence. The participants were from Australia and China. Adolescents’ personality was measured by the Junior Eysenck Personality Questionnaire, and neurasthenia was measured by the Somatic and Psychological Health Report. Structural equation modeling showed that neuroticism significantly predicted neurasthenia for both Chinese and Australian adolescents. Multigroup comparisons indicated that the strength of the relationship between neuroticism and neurasthenia was consistent across Australian and Chinese adolescents. Our findings imply that the relationship between personality traits and neurasthenia is consistent across different cultures.

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10

Wang, Wen-Ji. "Neurasthenia, psy sciences and the ‘great leap forward’ in Maoist China." History of Psychiatry 30, no. 4 (2019): 443–56. http://dx.doi.org/10.1177/0957154x19859204.

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The present study looks into the much-neglected history of neurasthenia in Maoist China in relation to the development of psy sciences. It begins with an examination of the various factors that transformed neurasthenia into a major health issue from the late 1950s to mid-1960s. It then investigates a distinctive culture of therapeutic experiment of neurasthenia during this period, with emphasis on the ways in which psy scientists and medical practitioners manoeuvred in a highly politicized environment. The study concludes with a discussion of the legacy of these neurasthenia studies – in particular, the experiment with the famous ‘speedy and synthetic therapy’ – and of the implications the present study may have for future historical study of psychiatry and science.

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11

Taylor, Ruth E. "Death of neurasthenia and its psychological reincarnation." British Journal of Psychiatry 179, no. 6 (2001): 550–57. http://dx.doi.org/10.1192/bjp.179.6.550.

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BackgroundThe diagnosis of neurasthenia appeared in 1869 and rapidly became fashionable and highly prevalent. It disappeared almost completely, producing ongoing debates about what happened to the disease, which have not so far been informed by empirical data.AimsTo use empirical historical hospital data from one specific hospital to explore several controversies about neurasthenia, including what happened to the disorder.MethodThe annual reports of Queen Square Hospital were examined from 1870 to 1947. The prevalence of neurasthenia diagnoses as a proportion of total discharges was recorded. The possible diagnostic categories into which neurasthenia could have been reclassified were identified. Textbooks and writing by neurologists working at the hospital during this period were examined.ResultsNeurasthenia accounted for 6–11% of total discharges from the late 1890s to 1930, when it virtually disappeared. Men accounted for 33–50% of cases.ConclusionsNeurasthenia affected both the upper and working classes and both men and women. Neurologists, not psychiatrists, continued to see the disorder well into the 20th century. Neurasthenia did not disappear, but was reclassified into psychological diagnoses.

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12

Hickie, Ian, Tracey Davenport, Cathy Issakidis, and Gavin Andrews. "Neurasthenia: Prevalence, disability and health care characteristics in the Australian community." British Journal of Psychiatry 181, no. 1 (2002): 56–61. http://dx.doi.org/10.1192/bjp.181.1.56.

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BackgroundNeurasthenia imposes a high burden on primary medical health care systems in all societies.AimsTo determine the prevalence of ICD-10 neurasthenia and associated comorbidity, disability and health care utilisation.MethodUtilisation of a national sample of Australian households previously surveyed using the Composite International Diagnostic Interview and other measures.ResultsProlonged and excessive fatigue was reported by 1465 people (13.29% of the sample). Of these, one in nine people meet current ICD-10 criteria for neurasthenia. Comorbidity was associated with affective, anxiety and physical disorders. People with neurasthenia alone (< 0.5% of the population) were less disabled and used less services than those with comorbid disorders.ConclusionsFatigue is frequent in the Australian community and is common in people attending general practice. Neurasthenia is disabling and demanding of services largely because of its comorbidity with other mental and physical disorders. Until a remedy for persistent fatigue is provided, doctors should take an active psychological approach to treatment.

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13

Wessely, Simon. "Old wine in new bottles: neurasthenia and ‘ME’." Psychological Medicine 20, no. 1 (1990): 35–53. http://dx.doi.org/10.1017/s0033291700013210.

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SYNOPSISThe history of neurasthenia is discussed in the light of current interest in chronic fatigue, and in particular the illness called myalgic encephalomyelitis (‘ ME ’). A comparison is made of the symptoms, presumed aetiologies and treatment of both illnesses, as well as their social setting. It is shown that neurasthenia remained popular as long as it was viewed as a non-psychiatric, neurological illness caused by environmental factors which affected successful people and for which the cure was rest. The decline in neurasthenia was related to the changes which occurred in each of these views. It is argued that similar factors are associated with the current interest in myalgic encephalomyelitis. It is further argued that neither neurasthenia nor ‘ ME ’ can be fully understood within a single medical or psychiatric model. Instead both have arisen in the context of contemporary explanations and attitudes involving mental illness. Future understanding, treatment and prevention of these and related illnesses will depend upon both psychosocial and neurobiological explanations of physical and mental fatigability.

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14

Qin, Yan He. "Neurasthenia in China." Psychiatric Annals 22, no. 4 (1992): 188–89. http://dx.doi.org/10.3928/0048-5713-19920401-07.

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Machizawa, Shizuo. "Neurasthenia in Japan." Psychiatric Annals 22, no. 4 (1992): 190–91. http://dx.doi.org/10.3928/0048-5713-19920401-08.

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16

Groenendijk, Leendert F. "Masturbation and Neurasthenia." Journal of Psychology & Human Sexuality 9, no. 1 (1997): 71–94. http://dx.doi.org/10.1300/j056v09n01_05.

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17

Angst, J. "Epidemiology of neurasthenia." European Psychiatry 13, S4 (1998): 140s. http://dx.doi.org/10.1016/s0924-9338(99)80034-0.

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18

Wilkinson, J. B., J. King, and P. G. Nixon. "Hyperventilation and neurasthenia." BMJ 298, no. 6687 (1989): 1577. http://dx.doi.org/10.1136/bmj.298.6687.1577.

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19

Frühstück, Sabine. "Male Anxieties: Nerve Force, Nation, and the Power of Sexual Knowledge." Journal of the Royal Asiatic Society of Great Britain & Ireland 15, no. 1 (2005): 71–88. http://dx.doi.org/10.1017/s1356186304004717.

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AbstractIn the autumn of 1929, a Kyôto-based journal for popular medicine reported that the dean of sexology, Habuto Eiji, had committed suicide after having suffered from neurasthenia (shinkei suijaku) for a long time. A practicing gynaecologist, Habuto had been the editor of the sexological journal Seiyoku to Jinsei (Sexual Desire and Humankind), the author of numerous books on sexual issues, and the co-author, together with Sawada Junjirô, of an abridged Japanese version of Richard von Krafft-Ebing's Psychopathia Sexualis, entitled Hentai Seiyokuron (1915). He also was involved in the translation of Havelock Ellis's Studies in the Psychology of Sex (1901–1928), the twenty Japanese-language volumes which were advertised under the title Sei no Shinri as early as in 1927. Among other sexologists, Habuto had been a chief theorist on the causes of neurasthenia. Physicians, psychiatrists, psychologists, pedagogues, and sexologists agreed with him that neurasthenia primarily afflicted men and was caused by overpowering exhaustion that was in turn the result of certain sexual practices. Modern commentators like Habuto speculated that neurasthenia was the result of masturbation or – even worse – homosexuality.

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20

Goering, Laura. "“Russian nervousness”: Neurasthenia and National Identity in Nineteenth-Century Russia." Medical History 47, no. 1 (2003): 23–46. http://dx.doi.org/10.1017/s0025727300000065.

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“Nothing dies so hard as a word”, wrote Harry Quilter in 1892, “—particularly a word nobody understands.” At the end of the nineteenth century, one such word—first uttered in America, but soon reverberating across the Western world—was “neurasthenia”. Popularized by the American neurologist George M Beard, this vaguely defined nervous disorder seemed to crop up everywhere, from medical journals to the popular press to belles lettres. Looking back at the years leading up to the Second World War, Paul Hartenberg recalled its remarkable pervasiveness: “It could be found everywhere, in the salons, at the theatre, in novels, at the Palace. It was used to explain the most disparate individual reactions: suicide and decadent art, fashion and adultery; it became the giant of neuropathology.” Its sufferers included American intellectuals from Beard himself to Theodore Roosevelt, Edith Wharton, and Henry Adams; for European commentators less convinced of the disease's modern American pedigree, the list could be expanded to include everyone from Alcibiades to Tiberius to Napoleon. Anybody who was anybody, it seemed, was neurasthenic.

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21

Kocharyan, Garnik. "Hypoactive Sexual Desire Due to Mental and Neurological Disorders." Health of Man, no. 3 (September 30, 2022): 12–24. http://dx.doi.org/10.30841/2307-5090.3.2022.270806.

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The article presents data on hypoactive sexual desire which occurs in neurotic and stress-related disorders (neurasthenia, anxiety-phobic and obsessive-compulsive disorders, including the syndrome of anxious expectation of sexual failure, dissociative disorders, adaptation disorders [prolonged depressive reaction, anxiety-depressive reaction]). In particular, characterizing sexual disorders in patients with neurasthenia, one group of authors believes that these disorders differ depending on the form of this disease (hypersthenic, hyposthenic). In their opinion, in the hypersthenic form increased excitability of the sexual sphere is observed which can lead to the appearance of various sexual disorders, including increased libido. In the hyposthenic form of neurasthenia, according to this group of authors, various hypoactive sexual manifestations can be noted, including a decrease in libido, and in addition, premature ejaculation. Another group of researchers found in patients with neurasthenia, regardless of its form, only hypofunctional sexological manifestations, as well as premature ejaculation (that is, those symptoms that were named by the first group of authors as characteristic of the hyposthenic form of neurasthenia). The article also presents data on hypoactive sexual desire which can be observed in personality disorders and character accentuations (schizoid personality disorder and character schizoid accentuation, accentuations of the cycloid, asthenoneurotic, hysteroid, unstable, psychasthenic, sensitive and infantile-dependent types); mood disorders [affective disorders] (dysthymia [depressive neurosis], bipolar disorder); schizophrenia; mental retardations; dementia due to organic brain damages; epilepsy; organic brain damages; multiple sclerosis. Clinical observations are given; these concern hypoactive sexual desire in patients with schizophrenia and organic brain damage.

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Kocharyan, Garnik. "Hypoactive Sexual Desire Due to Mental and Neurological Disorders." Health of Man, no. 3 (September 30, 2022): 12–24. https://doi.org/10.30841/2307-5090.3.2022.270806.

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The article presents data on hypoactive sexual desire which occurs in neurotic and stress-related disorders (neurasthenia, anxiety-phobic and obsessive-compulsive disorders, including the syndrome of anxious expectation of sexual failure, dissociative disorders, adaptation disorders [prolonged depressive reaction, anxiety-depressive reaction]). In particular, characterizing sexual disorders in patients with neurasthenia, one group of authors believes that these disorders differ depending on the form of this disease (hypersthenic, hyposthenic). In their opinion, in the hypersthenic form increased excitability of the sexual sphere is observed which can lead to the appearance of various sexual disorders, including increased libido. In the hyposthenic form of neurasthenia, according to this group of authors, various hypoactive sexual manifestations can be noted, including a decrease in libido, and in addition, premature ejaculation. Another group of researchers found in patients with neurasthenia, regardless of its form, only hypofunctional sexological manifestations, as well as premature ejaculation (that is, those symptoms that were named by the first group of authors as characteristic of the hyposthenic form of neurasthenia). The article also presents data on hypoactive sexual desire which can be observed in personality disorders and character accentuations (schizoid personality disorder and character schizoid accentuation, accentuations of the cycloid, asthenoneurotic, hysteroid, unstable, psychasthenic, sensitive and infantile-dependent types); mood disorders [affective disorders] (dysthymia [depressive neurosis], bipolar disorder); schizophrenia; mental retardations; dementia due to organic brain damages; epilepsy; organic brain damages; multiple sclerosis. Clinical observations are given; these concern hypoactive sexual desire in patients with schizophrenia and organic brain damage.

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23

Parnikoza, T. P., та Y. D. Zalesnа. "Акупунктура в лікуванні неврастенії". Likarska sprava, № 7 (19 листопада 2012): 154–55. http://dx.doi.org/10.31640/ls-2012-7-24.

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Neurasthenia – is the exhaustion of the nervous system, mental disorder that belongs to a group of neuroses. Doctors sometimes briefly describing him “irritable weakness” or “irritable fatigue.” This term is justified: a person who suffers asthenic-neurotic syndrome, while experiencing fatigue and increased nervous excitability. Reasons neurasthenia quite a lot. Unfortunately, the lifestyle of modern man gives many reasons for the emergence of asthenic-neurotic syndrome.

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24

Rudenko, Svetlana L. "Emotional factors of social perception deficit in neurasthenia." Izvestiya of Saratov University. Philosophy. Psychology. Pedagogy 23, no. 3 (2023): 331–35. http://dx.doi.org/10.18500/1819-7671-2023-23-3-331-335.

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Introduction. The study is aimed at identifying emotional factors that determine the deficit of social perception in neurasthenia. Theoretical analysis. Insufficient knowledge of the phenomenology of neurasthenia and the difficulties of its differential diagnosis at the stage of pathological personality development is shown. The lack of targeted assistance to patients and their objectively rapid disability is emphasized. The hypothesis is formulated that patients with neurasthenia have a pronounced violation of social perception, determined by social anhedonia. Empirical analysis. In the group of patients with neurasthenia, in comparison with healthy subjects, there are pronounced difficulties in recognizing emotions by verbal and nonverbal manifestations, finding the necessary tone of communication and weakness of analytical abilities that allow to anticipate the dynamics of social situations. Factor analysis has shown the decisive role of emotional determinants in the development of the studied disorders – a high level of social anhedonia and associated personal, reactive anxiety and depression. Conclusion. Emotional factors form a pronounced social anhedonia that determines the formation of social perception deficit. A weak focus on the details of social situations, a tendency to attribute one’s own experiences to others determine the low level of effectiveness of social interaction, gradual withdrawal from contacts and complete social disintegration.

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25

Sherwood, Gayle. "Charles Ives and “Our National Malady”." Journal of the American Musicological Society 54, no. 3 (2001): 555–84. http://dx.doi.org/10.1525/jams.2001.54.3.555.

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Abstract In his psychoanalytical biography of Ives, Charles Ives: “My Father's Song,” Stuart Feder convincingly argued that Ives's health breakdowns in 1906 and 1918 were the result of emotional and psychological factors rather than a physical heart condition. But the Freudian approach and terminology employed by Feder were unknown in American in 1906 and had not been fully accepted even by 1918. Therefore, how would an American doctor in 1906 have diagnosed Ives's “condition”? A careful examination of key events in Ives's life between 1902 and 1908, and a close reading of his correspondence indicate that he may have been recognized as neurasthenic. Ives's neurasthenia locates his identity by nationality, ethnicity, gender, economic and social class, education, profession, environment, and lifestyle. As a result, his artistic values and character traits emerge as remarkably typical of his country, culture, and time.

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26

Yamamoto, Joe. "Psychiatric Diagnoses and Neurasthenia." Psychiatric Annals 22, no. 4 (1992): 171–72. http://dx.doi.org/10.3928/0048-5713-19920401-05.

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27

Rollin, Henry. "The treatment of neurasthenia." British Journal of Psychiatry 163, no. 6 (1993): 840. http://dx.doi.org/10.1192/s0007125000050005.

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28

Greenberg, Donna B. "Neurasthenia in the 1980s." Psychosomatics 31, no. 2 (1990): 129–37. http://dx.doi.org/10.1016/s0033-3182(90)72185-8.

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29

Young, Derson. "Neurasthenia and related problems." Culture, Medicine and Psychiatry 13, no. 2 (1989): 131–38. http://dx.doi.org/10.1007/bf02220657.

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30

Rin, Hsien, and Mei-Gum Huang. "Neurasthenia as nosological dilemma." Culture, Medicine and Psychiatry 13, no. 2 (1989): 215–26. http://dx.doi.org/10.1007/bf02220663.

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31

Rollin, Henry. "The treatment of neurasthenia." British Journal of Psychiatry 163, no. 06 (1993): 840. http://dx.doi.org/10.1017/s0007125000050005.

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32

White, P. "Fatigue syndrome: neurasthenia revived." BMJ 298, no. 6682 (1989): 1199–200. http://dx.doi.org/10.1136/bmj.298.6682.1199.

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33

Trautman, P. D. "The legitimacy of neurasthenia." JAMA: The Journal of the American Medical Association 272, no. 22 (1994): 1719b—1719. http://dx.doi.org/10.1001/jama.272.22.1719b.

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Vielma, Valentina M. "The Legitimacy of Neurasthenia." JAMA: The Journal of the American Medical Association 272, no. 22 (1994): 1719. http://dx.doi.org/10.1001/jama.1994.03520220013004.

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35

Cathébras, Pascal. "Neurasthenia, Spasmophilia and Chronic Fatigue Syndromes in France." Transcultural Psychiatric Research Review 31, no. 3 (1994): 259–70. http://dx.doi.org/10.1177/136346159403100302.

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This paper reviews French concepts of fatigue syn dromes, with a special emphasis on a reputedly unique ly French diagnosis: spasmophilia. Neurasthenia has virtually no place in contemporary French nosology of fatigue states, and chronic fatigue syndrome has not been accepted by all French researchers. The contro versies on neurasthenia and spasmophilia illustrate some of the social uses of diagnostic categories (e.g., legitimation of symptoms) and the ambivalence of physicians toward diagnostic labels based on clinical criteria.

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36

Angst, J. "History and epidemiology of panic." European Psychiatry 10, S2 (1995): 57s—59s. http://dx.doi.org/10.1016/0924-9338(96)80325-7.

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The conceptualisation and investigation of panic disorder have developed on two different axes, medical and psychological, for more than 100 years. In medicine, reports of acute anxiety attacks with cardiac, circulatory, and respiratory symptoms date back to the French Revolution, and terms such as “soldier's heart”, “neurocirculatory neurasthenia”, or “hyperventilation syndrome” have been used to describe them. In psychological medicine, anxiety attacks were first reported by Domrich in 1849. These attacks, which were thought to be caused by strong emotions, were classified mainly within neurasthenia, until Freud created the concept of anxiety neurosis in 1895.

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37

Bailly, L. "Chronic fatigue syndrome or neurasthenia?" British Journal of Psychiatry 181, no. 4 (2002): 350–51. http://dx.doi.org/10.1192/bjp.181.4.350-a.

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38

Starcevic, Vladan. "Neurasthenia in European Psychiatric Literature." Transcultural Psychiatric Research Review 31, no. 2 (1994): 125–36. http://dx.doi.org/10.1177/136346159403100203.

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Zohar, J. "OCD: From neurasthenia to neuroscience." European Psychiatry 13, S4 (1998): 195S. http://dx.doi.org/10.1016/s0924-9338(99)80228-4.

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Peters, U. H. "The Neurasthenia as Eternal Revenant." Fortschritte der Neurologie · Psychiatrie 70, no. 11 (2002): 569. http://dx.doi.org/10.1055/s-2002-35175.

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Veltishchev, Dmitri Yuryevich. "Neurasthenia: history and the present." Neurology, neuropsychiatry, Psychosomatics, no. 4 (December 14, 2011): 9. http://dx.doi.org/10.14412/2074-2711-2011-338.

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Schuster, D. G. "Neurasthenia and a Modernizing America." JAMA: The Journal of the American Medical Association 290, no. 17 (2003): 2327–28. http://dx.doi.org/10.1001/jama.290.17.2327.

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Martensen, Robert L. "The Legitimacy of Neurasthenia-Reply." JAMA: The Journal of the American Medical Association 272, no. 22 (1994): 1719. http://dx.doi.org/10.1001/jama.1994.03520220013005.

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Yao, Yongcheng, Jie Tang, Zhenzhen Li, et al. "Perceived social support mediates the effect of COVID-19 pandemic on job adaptation disorders of workers: An exploratory cross-sectional study." Medicine 103, no. 5 (2024): e37118. http://dx.doi.org/10.1097/md.0000000000037118.

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Abstract:

COVID-19 lockdown can lead to job adaptation disorders, which are heterogeneous among individuals. The purpose of this study was to explore the association between perceived social support and job adaptation disorders among workers in China during the COVID-19 pandemic. The questionnaires of Psychological Questionnaire for Public Health Emergencies, Multidimensional Scale of Perceived Social Support, Work Attitude Scale were used for this cross-section study via an online survey. The study included 626 employees. Hierarchical regression analysis and Bootstrap method were used to investigate the mediation effect of perceived social support between the emergency and job adaptation disorders. The percentages of the 5 dimensions of depression, neurasthenia, fear, compulsion-anxiety, and hypochondria in workers were 59.7%, 56.1%, 92.3%, 42.0%, and 18.7%, respectively. Social support mediated the relationship between depression, neurasthenia, obsessive-compulsive anxiety and job adaptation disorder, accounting for 18.1%, 16.1%, and 17.5% of the total effect (ab/c), respectively. Perceived social support could alleviate COVID-19 pandemic-related depression, neurasthenia, compulsion-anxiety, and job adaptation disorder in Chinese workers. Improving their perception of social support, workers may better adapt themselves to work in the challenging of the public health emergency during COVID-19 pandemic.

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Yang, Qiumei. "Anorexia, self-repression, and neurasthenia: Lucys psychological characteristics from the perspective of spatial theory." Advances in Humanities Research 12, no. 2 (2025): None. https://doi.org/10.54254/2753-7080/2025.23466.

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In Charlotte Bront studies, Villette has received comparatively limited scholarly attention, with most existing research focusing narrowly on womens issues while neglecting the relationship between spatial dimensions and character psychology. This paper draws on the spatial theories of Henri Lefebvre and Edward Soja to explore the psychological characteristics of Lucy Snowe in Villette. Across three layers of spacebodily, private, and urbanLucy displays symptoms of anorexia, self-repression, and neurasthenia, respectively. Her psychological predicament stems from the disciplinary nature of social space. The interrelation between her neurasthenia, anorexia, and self-repression is one of mutual causality, highlighting the broader dilemma of Victorian women in their pursuit of self-worth.

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Skultans, Vieda. "Neurasthenia and Political Resistance in Latvia." Anthropology Today 11, no. 3 (1995): 14. http://dx.doi.org/10.2307/2783366.

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Hong, Walter, Louisa Chan, Dunchun Zheng, and Chao Wang. "Neurasthenia in Chinese Students at UCLA." Psychiatric Annals 22, no. 4 (1992): 199–201. http://dx.doi.org/10.3928/0048-5713-19920401-11.

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Cao, Yuping, Yalin Zhang, Doris F. Chang, Guoqiang Wang, and Xianghui Zhang. "Psychosocial and Immunological Factors in Neurasthenia." Psychosomatics 50, no. 1 (2009): 24–29. http://dx.doi.org/10.1176/appi.psy.50.1.24.

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Huang, Hsuan-Ying. "Revisiting Arthur Kleinman’s Research on Neurasthenia." Taiwanese Journal of Psychiatry 38, no. 3 (2024): 103–6. http://dx.doi.org/10.4103/tpsy.tpsy_21_24.

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King, Charles. "Parallels Between Neurasthenia and Premenstrual Syndrome." Women & Health 15, no. 4 (1990): 1–23. http://dx.doi.org/10.1300/j013v15n04_01.

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