Relevant bibliographies by topics / Neuro-immune diseases

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Neuro-immune diseases'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Neuro-immune diseases"

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Kerschensteiner, M., E. Meinl, and R. Hohlfeld. "Neuro-immune crosstalk in CNS diseases." Neuroscience 158, no. 3 (2009): 1122–32. http://dx.doi.org/10.1016/j.neuroscience.2008.09.009.

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Morris, Gerwyn, Michael Berk, Piotr Galecki, Ken Walder, and Michael Maes. "The Neuro-Immune Pathophysiology of Central and Peripheral Fatigue in Systemic Immune-Inflammatory and Neuro-Immune Diseases." Molecular Neurobiology 53, no. 2 (2015): 1195–219. http://dx.doi.org/10.1007/s12035-015-9090-9.

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He, Cuiying, Qian Wang, Jinyan Gao, Hongbing Chen, and Ping Tong. "Neuro-immune regulation in allergic Diseases: Role of neuropeptides." International Immunopharmacology 145 (January 2025): 113771. https://doi.org/10.1016/j.intimp.2024.113771.

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Camp, Belinda, Sabine Stegemann-Koniszewski, and Jens Schreiber. "Infection-Associated Mechanisms of Neuro-Inflammation and Neuro-Immune Crosstalk in Chronic Respiratory Diseases." International Journal of Molecular Sciences 22, no. 11 (2021): 5699. http://dx.doi.org/10.3390/ijms22115699.

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Chronic obstructive airway diseases are characterized by airflow obstruction and airflow limitation as well as chronic airway inflammation. Especially bronchial asthma and chronic obstructive pulmonary disease (COPD) cause considerable morbidity and mortality worldwide, can be difficult to treat, and ultimately lack cures. While there are substantial knowledge gaps with respect to disease pathophysiology, our awareness of the role of neurological and neuro-immunological processes in the development of symptoms, the progression, and the outcome of these chronic obstructive respiratory diseases,
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Reale, Marcella, and Erica Costantini. "Cholinergic Modulation of the Immune System in Neuroinflammatory Diseases." Diseases 9, no. 2 (2021): 29. http://dx.doi.org/10.3390/diseases9020029.

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Frequent diseases of the CNS, such as Alzheimer’s disease, Parkinson’s disease, multiple sclerosis, and psychiatric disorders (e.g., schizophrenia), elicit a neuroinflammatory response that contributes to the neurodegenerative disease process itself. The immune and nervous systems use the same mediators, receptors, and cells to regulate the immune and nervous systems as well as neuro-immune interactions. In various neurodegenerative diseases, peripheral inflammatory mediators and infiltrating immune cells from the periphery cause exacerbation to current injury in the brain. Acetylcholine (ACh)
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Chu, Coco, David Artis, and Isaac M. Chiu. "Neuro-immune Interactions in the Tissues." Immunity 52, no. 3 (2020): 464–74. http://dx.doi.org/10.1016/j.immuni.2020.02.017.

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Voisin, Tiphaine, Amélie Bouvier, and Isaac M. Chiu. "Neuro-immune interactions in allergic diseases: novel targets for therapeutics." International Immunology 29, no. 6 (2017): 247–61. http://dx.doi.org/10.1093/intimm/dxx040.

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Bjørklund, Geir, Lili Zou, Massimiliano Peana, et al. "The Role of the Thioredoxin System in Brain Diseases." Antioxidants 11, no. 11 (2022): 2161. http://dx.doi.org/10.3390/antiox11112161.

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The thioredoxin system, consisting of thioredoxin (Trx), thioredoxin reductase (TrxR), and NADPH, plays a fundamental role in the control of antioxidant defenses, cell proliferation, redox states, and apoptosis. Aberrations in the Trx system may lead to increased oxidative stress toxicity and neurodegenerative processes. This study reviews the role of the Trx system in the pathophysiology and treatment of Alzheimer’s, Parkinson’s and Huntington’s diseases, brain stroke, and multiple sclerosis. Trx system plays an important role in the pathophysiology of those disorders via multiple interaction
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Vojvodic, Aleksandra, Zorica Peric-Hajzler, Dusica Matovic, et al. "Gut Microbiota and the Alteration of Immune Balance in Skin Diseases: From Nutraceuticals to Fecal Transplantation." Open Access Macedonian Journal of Medical Sciences 7, no. 18 (2019): 3034–38. http://dx.doi.org/10.3889/oamjms.2019.827.

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T
 The P.N.E.I. (Psycho-Neuro-Endocrine-Immunology) approach is represented by the interdisciplinary concept of bidirectional cross-talk between the psycho-neuro-endocrine and immune systems, which can influence the immune response. The well-known Gut-Brain Axis and the Gut-Skin Axis can be merged in a bigger network- the Gut-Brain-Skin Axis, with complex regulation by cytokines, neuro-peptides, neuro-hormones and another messenger (signalling) molecules and maybe the most important modulator of the Gut-Brain-Skin Axis/ the gut microbiota. The role of gut bacterial homeostasis is very imp
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Deczkowska, Aleksandra, and Michal Schwartz. "Targeting neuro–immune communication in neurodegeneration: Challenges and opportunities." Journal of Experimental Medicine 215, no. 11 (2018): 2702–4. http://dx.doi.org/10.1084/jem.20181737.

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Immune cells patrol the brain and can support its function, but can we modulate brain–immune communication to fight neurological diseases? Here, we briefly discuss the mechanisms orchestrating the cross-talk between the brain and the immune system and describe how targeting this interaction in a well-controlled manner could be developed as a universal therapeutic approach to treat neurodegeneration.
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Dissertations / Theses on the topic "Neuro-immune diseases"

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Carvalho, Kévin. "Rôle des récepteurs adénosinergiques A2A dans la maladie d'Alzheimer." Thesis, Lille 2, 2019. http://www.theses.fr/2019LIL2S030.

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L'accumulation neuronale de protéines tau hyperphosphorylées et agrégées (« pathologie tau ») est corrélée au déclin cognitif dans la maladie d'Alzheimer. Les mécanismes liant tau à la perte de mémoire demeurent néanmoins mal connus. Différentes données épidémiologiques et expérimentales suggèrent que la consommation régulière de caféine réduit le risque de développer la maladie d'Alzheimer ainsi que les lésions et déficits cognitifs associés. Les effets protecteurs de la caféine ont été attribués au blocage des récepteurs à l'adénosine A2A (A2ARs), qui sont retrouvés augmentés dans le cerveau
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Paltser, Geoffrey. "Neuro-immune Elements of Inflammatory Disease." Thesis, 2012. http://hdl.handle.net/1807/43706.

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Interactions between the immune system and the nervous system are currently underappreciated, assumed to play minor mechanistic roles in disease pathogenesis. In contrast, our laboratory has demonstrated the importance of this relationship with significant impact, initially in Type 1 Diabetes (T1D). The experiments presented here build on our previous work to provide insights into the etiology of Multiple Sclerosis (MS) and Type 2 Diabetes (T2D). Transient Receptor Potential Vanilloid-1 (TRPV1) is an ion channel expressed on peripheral sensory afferent neurons that fundamentally control T1D
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Books on the topic "Neuro-immune diseases"

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(Editor), Paul H. Patterson, Claude Kordon (Editor), and Yves Christen (Editor), eds. Neuro-Immune Interactions in Neurologic and Psychiatric Disorders. Springer-Verlag Telos, 2000.

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2

Scolding, Neil. Vasculitis and collagen vascular diseases. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0862.

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That part of the clinical interface between neurology and general medicine occupied by inflammatory and immunological diseases is neither small nor medically trivial. Neurologists readily accept the challenges of ‘primary’ immune diseases of the nervous system: these tend to be focussed on one particular target such as oligodendrocytes or the neuro-muscular junction present in predictable ways, and are amenable as a rule to rational, methodological diagnosis, and occasionally even treatment. This is proper neurology.‘Secondary’ neurological involvement in diseases mainly considered systemic in
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Christen, Y., C. Kordon, and P. Patterson. Neuro-Immune Interactions in Neurologic and Psychiatric Disorders. Springer, 2011.

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Christen, Y., C. Kordon, and P. Patterson. Neuro-Immune Interactions in Neurologic and Psychiatric Disorders. Springer, 2012.

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5

Haghighi, Afshin Borhani, and Bernadette Kalman. Other Proven and Putative Autoimmune Disorders of the CNS. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0094.

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Behcet’s Disease (BD) is a multiorgan disorder characterized by oral and genital ulceration, uveitis, and dermatological symptoms. BD is most prevalent in the Mediterranean countries and East Asia, but also occurs in Europe and North America. The etiology remains unknown. Evidence suggests that BD is an autoimmune disorder with complex traits. Neuro-Behcet’s Syndome (NBS) develops in about 5% to 30% of patients with BD and presents with parenchymal or nonparenchymal pathology. The course of NBS is highly variable. Treatment strategies include modulations of the immune response and tissue degen
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Book chapters on the topic "Neuro-immune diseases"

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Ibrahim, Iddrisu, Syamala Soumya Krishnan, and Joseph Atia Ayariga. "The Intestinal Neuro-immune Axis." In Neuroimmune Diseases. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-60006-7_28.

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Ibrahim, Iddrisu, Syamala Soumya Krishnan, and Joseph Atia Ayariga. "The Intestinal Neuro-immune Axis." In Neuroimmune Diseases. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-24297-7_28-1.

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Kerschensteiner, Martin, Edgar Meinl, and Reinhard Hohlfeld. "Neuro-Immune Crosstalk in CNS Diseases." In Results and Problems in Cell Differentiation. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/400_2009_6.

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4

Argenzio, Robert A. "Neuro-Immune Pathobiology of Infectious Enteric Disease." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1828-4_2.

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Maltsev, Dmytro. "Efficacy of rituximab in autism spectrum disorders associated with genetic deficiency of the folate cycle with signs of antineuronal autoimmunity." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch12.

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Advances in genetics, molecular biology, and immunology over the past decades have significantly changed our understanding of the etiology and pathogenesis of autism spectrum disorders (ASD) in children. One of the key advances in this direction is the elucidation of the association of genetic deficiency of the folate cycle (GDFC) with ASD, evidence for which is based on the results of at least 5 meta-analyses of randomized controlled clinical trials and a number of additional controlled trials, the data of which have not yet been properly summarized. It has been established that GDFC leads to
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Zilka, Norbert, Zuzana Kazmerova, and Michal Novak. "Neuro-immune proteomic crosstalk in health and disease: partners in love, partners in divorce." In Farm animal proteomics 2013. Wageningen Academic Publishers, 2013. http://dx.doi.org/10.3920/978-90-8686-776-9_7.

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Vizi, Szilveszter. "Neuro-immune responses in different diseases." In Molecular Pathomechanisms and New Trends in Drug Research. CRC Press, 2002. http://dx.doi.org/10.1201/9780203219973.ch13.

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"Neuro-immune responses in different diseases – role of tumor necrosis factor- and IL-I." In Molecular Pathomechanisms and New Trends in Drug Research. CRC Press, 2002. http://dx.doi.org/10.1201/9780203219973-20.

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Wieck, Andrea, Izabela G. Barbosa, Antonio L. Teixeira, and Moises E. Bauer. "Immunology of Post-Traumatic Stress Disorder (PTSD)." In Immunopsychiatry. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190884468.003.0013.

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Following exposure to a severe traumatic event, such as warfare, sexual assault, or traffic accidents, a person may develop post-traumatic stress disorder (PTSD). This psychiatric disorder is characterized by constant re-experiencing of the traumatic event, with disturbing thoughts, feelings, or dreams related to the events, as well as irritability and exaggerated stressor reactivity. The increase of “fight-or-flight” responses leads to chronic dysregulation of stress-responsive mechanisms, including hypothalamic-pituitary-adrenal (HPA) and sympathetic axis activation, as well as immune imbala
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Jana, Rishika, Souvik Karmakar, Bishal Hazra, Subhadeep Roy, and Jayasri Das Sarma. "Mice as an Experimental Model to Understand the Pathobiology of Diseases." In Rodents and Their Role in Ecology, Medicine and Agriculture [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.1001835.

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Murine models are widely used in scientific research because they share many genetic similarities with humans, making them a valuable tool for studying various diseases. C57BL/6 is an experimental mouse model to study the demyelination and inflammation etiology of Multiple Sclerosis (MS). Intracranial inoculation of neurotropic murine β-coronavirus strain of Mouse hepatitis virus in C57BL/6 mice induces demyelination with or without axonal loss, providing many insights regarding the mechanism of MS as well as SARS-CoV-2 mediated pulmonary and neuro pathology in humans. By selectively using kno
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Conference papers on the topic "Neuro-immune diseases"

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Li, Yijie, Tao Ma, Le Lu, and Shao Li. "Networking drugs and diseases in the context of neuro-endocrine-immune system." In 2010 3rd International Conference on Biomedical Engineering and Informatics (BMEI). IEEE, 2010. http://dx.doi.org/10.1109/bmei.2010.5639709.

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Journal articles
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