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Journal articles on the topic 'Neuro-immune diseases'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Kerschensteiner, M., E. Meinl, and R. Hohlfeld. "Neuro-immune crosstalk in CNS diseases." Neuroscience 158, no. 3 (2009): 1122–32. http://dx.doi.org/10.1016/j.neuroscience.2008.09.009.

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Morris, Gerwyn, Michael Berk, Piotr Galecki, Ken Walder, and Michael Maes. "The Neuro-Immune Pathophysiology of Central and Peripheral Fatigue in Systemic Immune-Inflammatory and Neuro-Immune Diseases." Molecular Neurobiology 53, no. 2 (2015): 1195–219. http://dx.doi.org/10.1007/s12035-015-9090-9.

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3

He, Cuiying, Qian Wang, Jinyan Gao, Hongbing Chen, and Ping Tong. "Neuro-immune regulation in allergic Diseases: Role of neuropeptides." International Immunopharmacology 145 (January 2025): 113771. https://doi.org/10.1016/j.intimp.2024.113771.

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4

Camp, Belinda, Sabine Stegemann-Koniszewski, and Jens Schreiber. "Infection-Associated Mechanisms of Neuro-Inflammation and Neuro-Immune Crosstalk in Chronic Respiratory Diseases." International Journal of Molecular Sciences 22, no. 11 (2021): 5699. http://dx.doi.org/10.3390/ijms22115699.

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Chronic obstructive airway diseases are characterized by airflow obstruction and airflow limitation as well as chronic airway inflammation. Especially bronchial asthma and chronic obstructive pulmonary disease (COPD) cause considerable morbidity and mortality worldwide, can be difficult to treat, and ultimately lack cures. While there are substantial knowledge gaps with respect to disease pathophysiology, our awareness of the role of neurological and neuro-immunological processes in the development of symptoms, the progression, and the outcome of these chronic obstructive respiratory diseases,

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Reale, Marcella, and Erica Costantini. "Cholinergic Modulation of the Immune System in Neuroinflammatory Diseases." Diseases 9, no. 2 (2021): 29. http://dx.doi.org/10.3390/diseases9020029.

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Frequent diseases of the CNS, such as Alzheimer’s disease, Parkinson’s disease, multiple sclerosis, and psychiatric disorders (e.g., schizophrenia), elicit a neuroinflammatory response that contributes to the neurodegenerative disease process itself. The immune and nervous systems use the same mediators, receptors, and cells to regulate the immune and nervous systems as well as neuro-immune interactions. In various neurodegenerative diseases, peripheral inflammatory mediators and infiltrating immune cells from the periphery cause exacerbation to current injury in the brain. Acetylcholine (ACh)

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Chu, Coco, David Artis, and Isaac M. Chiu. "Neuro-immune Interactions in the Tissues." Immunity 52, no. 3 (2020): 464–74. http://dx.doi.org/10.1016/j.immuni.2020.02.017.

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Voisin, Tiphaine, Amélie Bouvier, and Isaac M. Chiu. "Neuro-immune interactions in allergic diseases: novel targets for therapeutics." International Immunology 29, no. 6 (2017): 247–61. http://dx.doi.org/10.1093/intimm/dxx040.

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8

Bjørklund, Geir, Lili Zou, Massimiliano Peana, et al. "The Role of the Thioredoxin System in Brain Diseases." Antioxidants 11, no. 11 (2022): 2161. http://dx.doi.org/10.3390/antiox11112161.

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The thioredoxin system, consisting of thioredoxin (Trx), thioredoxin reductase (TrxR), and NADPH, plays a fundamental role in the control of antioxidant defenses, cell proliferation, redox states, and apoptosis. Aberrations in the Trx system may lead to increased oxidative stress toxicity and neurodegenerative processes. This study reviews the role of the Trx system in the pathophysiology and treatment of Alzheimer’s, Parkinson’s and Huntington’s diseases, brain stroke, and multiple sclerosis. Trx system plays an important role in the pathophysiology of those disorders via multiple interaction

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Vojvodic, Aleksandra, Zorica Peric-Hajzler, Dusica Matovic, et al. "Gut Microbiota and the Alteration of Immune Balance in Skin Diseases: From Nutraceuticals to Fecal Transplantation." Open Access Macedonian Journal of Medical Sciences 7, no. 18 (2019): 3034–38. http://dx.doi.org/10.3889/oamjms.2019.827.

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T The P.N.E.I. (Psycho-Neuro-Endocrine-Immunology) approach is represented by the interdisciplinary concept of bidirectional cross-talk between the psycho-neuro-endocrine and immune systems, which can influence the immune response. The well-known Gut-Brain Axis and the Gut-Skin Axis can be merged in a bigger network- the Gut-Brain-Skin Axis, with complex regulation by cytokines, neuro-peptides, neuro-hormones and another messenger (signalling) molecules and maybe the most important modulator of the Gut-Brain-Skin Axis/ the gut microbiota. The role of gut bacterial homeostasis is very imp

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Deczkowska, Aleksandra, and Michal Schwartz. "Targeting neuro–immune communication in neurodegeneration: Challenges and opportunities." Journal of Experimental Medicine 215, no. 11 (2018): 2702–4. http://dx.doi.org/10.1084/jem.20181737.

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Immune cells patrol the brain and can support its function, but can we modulate brain–immune communication to fight neurological diseases? Here, we briefly discuss the mechanisms orchestrating the cross-talk between the brain and the immune system and describe how targeting this interaction in a well-controlled manner could be developed as a universal therapeutic approach to treat neurodegeneration.

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Jacobson, Amanda, Daping Yang, Madeleine Vella, and Isaac M. Chiu. "The intestinal neuro-immune axis: crosstalk between neurons, immune cells, and microbes." Mucosal Immunology 14, no. 3 (2021): 555–65. http://dx.doi.org/10.1038/s41385-020-00368-1.

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AbstractThe gastrointestinal tract is densely innervated by a complex network of neurons that coordinate critical physiological functions. Here, we summarize recent studies investigating the crosstalk between gut-innervating neurons, resident immune cells, and epithelial cells at homeostasis and during infection, food allergy, and inflammatory bowel disease. We introduce the neuroanatomy of the gastrointestinal tract, detailing gut-extrinsic neuron populations from the spinal cord and brain stem, and neurons of the intrinsic enteric nervous system. We highlight the roles these neurons play in

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12

Wang, Tianshu, Hailing Duan, Javier Santos, et al. "Intelligent treatment system based on bioinformatics and neuro-immune-digestive tract diseases." Alexandria Engineering Journal 107 (November 2024): 415–33. http://dx.doi.org/10.1016/j.aej.2024.07.043.

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13

Beopoulos, Athanasios, Manuel Gea, Alessio Fasano, and François Iris. "Autonomic Nervous System Neuroanatomical Alterations Could Provoke and Maintain Gastrointestinal Dysbiosis in Autism Spectrum Disorder (ASD): A Novel Microbiome–Host Interaction Mechanistic Hypothesis." Nutrients 14, no. 1 (2021): 65. http://dx.doi.org/10.3390/nu14010065.

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Dysbiosis secondary to environmental factors, including dietary patterns, antibiotics use, pollution exposure, and other lifestyle factors, has been associated to many non-infective chronic inflammatory diseases. Autism spectrum disorder (ASD) is related to maternal inflammation, although there is no conclusive evidence that affected individuals suffer from systemic low-grade inflammation as in many psychological and psychiatric diseases. However, neuro-inflammation and neuro–immune abnormalities are observed within ASD-affected individuals. Rebalancing human gut microbiota to treat disease ha

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Ueno, Masaki. "Restoring neuro-immune circuitry after brain and spinal cord injuries." International Immunology 33, no. 6 (2021): 311–25. http://dx.doi.org/10.1093/intimm/dxab017.

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Abstract Neuro-immune interactions are essential for our body’s defense and homeostasis. Anatomical and physiological analyses have shown that the nervous system comprises multiple pathways that regulate the dynamics and functions of immune cells, which are mainly mediated by the autonomic nervous system and adrenal signals. These are disturbed when the neurons and circuits are damaged by diseases of the central nervous system (CNS). Injuries caused by stroke or trauma often cause immune dysfunction by abrogation of the immune-regulating neural pathways, which leads to an increased risk of inf

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Fletcher, Kylie, Amin Nassar, Alexander M. Menzies, et al. "Safety and efficacy of immune checkpoint inhibitors (ICI) in patients (pts) with pre-existing neurologic autoimmune diseases (NAID) and Parkinson's disease." Journal of Clinical Oncology 42, no. 16_suppl (2024): 2653. http://dx.doi.org/10.1200/jco.2024.42.16_suppl.2653.

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2653 Background: Patients with pre-existing NAID often flare with ICIs; neurological (neuro) immune related adverse events (irAEs) are often morbid or fatal, and little is known about safety of ICI in pts with NAID and other neuro conditions (e.g. Parkinson’s). Thus, we aimed to determine their safety and efficacy in these contexts. Methods: We retrospectively analyzed 71 pts from 5 institutions receiving ICIs with NAIDs and Parkinson’s disease. NAIDs included multiple sclerosis (MS), myasthenia gravis (MG), inflammatory neuropathy, transverse myelitis, Lambert-Eaton, myotonic dystrophy, and m

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16

Rossi, Elena, Luciano Mutti, Andrea Morrione, and Antonio Giordano. "Neuro–Immune Interactions in Severe COVID-19 Infection." Pathogens 11, no. 11 (2022): 1256. http://dx.doi.org/10.3390/pathogens11111256.

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SARS-CoV-2 is a new coronavirus that has affected the world since 2019. Interstitial pneumonia is the most common clinical presentation, but additional symptoms have been reported, including neurological manifestations. Severe forms of infection, especially in elderly patients, present as an excessive inflammatory response called “cytokine storm”, which can lead to acute respiratory distress syndrome (ARDS), multiorgan failure and death. Little is known about the relationship between symptoms and clinical outcomes or the characteristics of virus–host interactions. The aim of this narrative rev

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Stakenborg, Nathalie, and Guy E. Boeckxstaens. "Shining light on the neuro-immune axis in the gut." Immunity 54, no. 5 (2021): 850–52. http://dx.doi.org/10.1016/j.immuni.2021.04.016.

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18

Korneva, E. A. "Pathways of neuro-immune communication: past and present time, clinical application." Medical Immunology (Russia) 22, no. 3 (2020): 405–18. http://dx.doi.org/10.15789/1563-0625-pon-1974.

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Fundamental studies in neuroimmunophysiology are the keystone for development of new therapeutic approaches to the treatment of infectious, allergic, oncologic and autoimmune diseases. The achievements in this field allowed approving new treatment methods based on irritation of afferent and efferent fibers of autonomic nerves. That became possible due to numerous studies of pathways between the immune and nervous systems performed over last two decades. The milestones in the history of neuroimmune communication research are represented here. The immune system organs – bone marrow, thymus and s

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19

Managlia, Elizabeth, Deborah Carroll, Andrew Zloza, and Lena Al-Harthi. "Immune Modulation of HIV Replication: Relevance to HIV Immuno- and Neuro-Pathogenesis." Current HIV Research 2, no. 4 (2004): 395–401. http://dx.doi.org/10.2174/1570162043351020.

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20

Wang, Tian-Qi, Li-Ming Sun, Le-Xin Hao, Yuan Guo, Yue Zhang, and Xin Feng. "Visualized Analysis of Research Hotspots in Acupuncture-Neuro Immunoregulation Based on Bibliometrics." Acupuncture & Electro-Therapeutics Research 46, no. 4 (2021): 259–70. http://dx.doi.org/10.3727/036012921x16281724938078.

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Objective: To review the development of acupuncture and moxibustion-neural network regulation research systematically and comprehensively in China. Method: This study adopted co-word analysis, cluster analysis and other bibliometric methods such as SATI3.2, Citespace and Gephi to visually demonstrate the annual publication volume, cooperation between institutions and co-authorship in the field of acupuncture-neuro immunoregulation research based on the data of more than 70 papers on related topics covering a period of 24 years from 1997 to 2020 in China. Results: The results show that there ar

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21

Markelov, Vladimir, and Maxim Trushin. "Sympathetic nervous system and neurotransmitters: their possible role in neuroimmunomodulation of multiple sclerosis and some other autoimmune diseases." Open Medicine 1, no. 4 (2006): 313–29. http://dx.doi.org/10.2478/s11536-006-0031-x.

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AbstractMultiple sclerosis is still a disease without a cure. Although intensive research efforts have led to the development of drugs that modify the activity of the disease, most of them have various side effects and are expensive. At the same time it is becoming apparent that some remedies usually used to treat somatic and psychic disorders also have immunomodulating properties, and may help manage multiple sclerosis and other autoimmune diseases. We describe here the role of the sympathetic nervous system in the neuro-immune interaction in multiple sclerosis and other immune diseases with

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22

Murdaca, Giuseppe, Francesca Paladin, Marco Casciaro, Carmelo Mario Vicario, Sebastiano Gangemi, and Gabriella Martino. "Neuro-Inflammaging and Psychopatological Distress." Biomedicines 10, no. 9 (2022): 2133. http://dx.doi.org/10.3390/biomedicines10092133.

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Inflammaging is a low degree of chronic and systemic tissue inflammation associated with aging, and is intimately linked to pro-inflammatory mediators. These substances are involved in the pathogenesis of chronic inflammatory diseases and related psychopathological symptoms. When inflammation and aging affect the brain, we use the term neuro-inflammaging. In this review, we focused on the neuro-inflammatory process typical of advanced ages and the related psychopathological symptoms, with particular attention to understanding the immune-pathogenetic mechanisms involved and the potential use of

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23

Kytikova, Oxana, Tatyana Novgorodtseva, Marina Antonyuk, Yulia Denisenko, and Tatyana Gvozdenko. "Molecular Targets of Fatty Acid Ethanolamides in Asthma." Medicina 55, no. 4 (2019): 87. http://dx.doi.org/10.3390/medicina55040087.

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Asthma is a common allergic pathology of the respiratory tract that requires the study of mechanisms underlying it, due to severe forms of the disease, which are refractory to therapy. The review is devoted to the search for molecular targets of fatty acid ethanolamides in asthma, in particular palmitoylethanolamide (PEA), which has been successfully used in the treatment of chronic inflammatory and neurodegenerative diseases, in the pathogenesis of which the nervous and immune systems are involved. Recently, the potentially important role of neuro-immune interactions in the development of all

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Zusinaite, Eva, Aleksandr Ianevski, Diana Niukkanen, et al. "A Systems Approach to Study Immuno- and Neuro-Modulatory Properties of Antiviral Agents." Viruses 10, no. 8 (2018): 423. http://dx.doi.org/10.3390/v10080423.

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There are dozens of approved, investigational and experimental antiviral agents. Many of these agents cause serious side effects, which can only be revealed after drug administration. Identification of the side effects prior to drug administration is challenging. Here we describe an ex vivo approach for studying immuno- and neuro-modulatory properties of antiviral agents, which may be associated with potential side effects of these therapeutics. The current approach combines drug toxicity/efficacy tests and transcriptomics, which is followed by mRNA, cytokine and metabolite profiling. We demon

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Nhieu, Jennifer, Yu-Lung Lin, and Li-Na Wei. "CRABP1 in Non-Canonical Activities of Retinoic Acid in Health and Diseases." Nutrients 14, no. 7 (2022): 1528. http://dx.doi.org/10.3390/nu14071528.

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In this review, we discuss the emerging role of Cellular Retinoic Acid Binding Protein 1 (CRABP1) as a mediator of non-canonical activities of retinoic acid (RA) and relevance to human diseases. We first discuss the role of CRABP1 in regulating MAPK activities and its implication in stem cell proliferation, cancers, adipocyte health, and neuro-immune regulation. We then discuss an additional role of CRABP1 in regulating CaMKII activities, and its implication in heart and motor neuron diseases. Through molecular and genetic studies of Crabp1 knockout (CKO) mouse and culture models, it is establ

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Nockher, W. A., and H. Renz. "The role of neurotrophins in the pathogenesis of asthma and related diseases: allergy and asthma as prototypic neuro-immune diseases?" Clinical & Experimental Allergy 32, no. 9 (2002): 1266–68. http://dx.doi.org/10.1046/j.1365-2745.2002.01489.x.

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27

Jiang, Yunqiu, Sunny S. Po, Faris Amil, and Tarun W. Dasari. "Non-invasive Low-level Tragus Stimulation in Cardiovascular Diseases." Arrhythmia & Electrophysiology Review 9, no. 1 (2020): 40–46. http://dx.doi.org/10.15420/aer.2020.01.

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Low-level tragus stimulation (LLTS) is a non-invasive approach of transcutaneous vagus nerve stimulation. LLTS has applications in diseases of multiple systems, including epilepsy, depression, headache and potentially several cardiovascular diseases. LLTS has shown promising results in suppressing AF, alleviating post-MI ventricular arrhythmias and ischaemia-reperfusion injury along with improving diastolic parameters in heart failure with preserved left ventricular ejection fraction (HFpEF). Preliminary pilot clinical studies in patients with paroxysmal AF, HFpEF, heart failure with reduced e

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Tamberi, Lorenza, Alessia Belloni, Armanda Pugnaloni, et al. "The Influence of Myeloid-Derived Suppressor Cell Expansion in Neuroinflammation and Neurodegenerative Diseases." Cells 13, no. 7 (2024): 643. http://dx.doi.org/10.3390/cells13070643.

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The neuro-immune axis has a crucial function both during physiological and pathological conditions. Among the immune cells, myeloid-derived suppressor cells (MDSCs) exert a pivotal role in regulating the immune response in many pathological conditions, influencing neuroinflammation and neurodegenerative disease progression. In chronic neuroinflammation, MDSCs could lead to exacerbation of the inflammatory state and eventually participate in the impairment of cognitive functions. To have a complete overview of the role of MDSCs in neurodegenerative diseases, research on PubMed for articles usin

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Van Gerven, L., G. Boeckxstaens, and P. Hellings. "Up-date on neuro-immune mechanisms involved in allergic and non-allergic rhinitis." Rhinology journal 50, no. 3 (2012): 227–35. http://dx.doi.org/10.4193/rhino.11.152.

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Non-allergic rhinitis (NAR) is a common disorder, which can be defined as chronic nasal inflammation, independent of systemic IgE-mediated mechanisms. Symptoms of NAR patients mimic those of allergic rhinitis (AR) patients. However, AR patients can easily be diagnosed with skin prick test or allergen-specific IgE measurements in the serum, whereas NAR patients form a heterogeneous group and are difficult to diagnose because of an extensive list of different phenotypes, all varying in severity, underlying etiology and type of inflammation. Characterization of those phenotypes, mechanisms and ma

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Говш, E. Govsh, Кирюхина, et al. "Some Immunoendocrine Criteria Protracted Forms of Hysterical Disorders." Journal of New Medical Technologies 21, no. 4 (2014): 67–71. http://dx.doi.org/10.12737/7271.

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The purpose of this study was to identify prognostically unfavorable immune and endocrine development criteria hysterical disorders. The study involved patients with hysterical personality development, hysterical neurosis and healthy donors. Immunological reactions were performed by standard methods, hormonal - immune enzyme analysis. Changes in cellular and humoral immunity in the form of a reduction in the absolute number of T- and B-lymphocytes, decrease the metabolic activity of neutrophils, the ratio of circulating immune complexes of different molecular weight, and lower cortisol levels

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Slama Schwok, Anny, and Julien Henri. "Long Neuro-COVID-19: Current Mechanistic Views and Therapeutic Perspectives." Biomolecules 14, no. 9 (2024): 1081. http://dx.doi.org/10.3390/biom14091081.

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Long-lasting COVID-19 (long COVID) diseases constitute a real life-changing burden for many patients around the globe and, overall, can be considered societal and economic issues. They include a variety of symptoms, such as fatigue, loss of smell (anosmia), and neurological–cognitive sequelae, such as memory loss, anxiety, brain fog, acute encephalitis, and stroke, collectively called long neuro-COVID-19 (long neuro-COVID). They also include cardiopulmonary sequelae, such as myocardial infarction, pulmonary damage, fibrosis, gastrointestinal dysregulation, renal failure, and vascular endotheli

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Zouali, Moncef. "B Cells at the Cross-Roads of Autoimmune Diseases and Auto-Inflammatory Syndromes." Cells 11, no. 24 (2022): 4025. http://dx.doi.org/10.3390/cells11244025.

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Whereas autoimmune diseases are mediated primarily by T and B cells, auto-inflammatory syndromes (AIFS) involve natural killer cells, macrophages, mast cells, dendritic cells, different granulocyte subsets and complement components. In contrast to autoimmune diseases, the immune response of patients with AIFS is not associated with a breakdown of immune tolerance to self-antigens. Focusing on B lymphocyte subsets, this article offers a fresh perspective on the multiple cross-talks between both branches of innate and adaptive immunity in mounting coordinated signals that lead to AIFS. By virtue

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Stimmer, Lev, Claire-Maëlle Fovet, and Ché Serguera. "Experimental Models of Autoimmune Demyelinating Diseases in Nonhuman Primates." Veterinary Pathology 55, no. 1 (2017): 27–41. http://dx.doi.org/10.1177/0300985817712794.

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Human idiopathic inflammatory demyelinating diseases (IIDD) are a heterogeneous group of autoimmune inflammatory and demyelinating disorders of the central nervous system (CNS). These include multiple sclerosis (MS), the most common chronic IIDD, but also rarer disorders such as acute disseminated encephalomyelitis (ADEM) and neuromyelitis optica (NMO). Great efforts have been made to understand the pathophysiology of MS, leading to the development of a few effective treatments. Nonetheless, IIDD still require a better understanding of the causes and underlying mechanisms to implement more eff

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Pandey, Manoj Kumar. "Exploring Pro-Inflammatory Immunological Mediators: Unraveling the Mechanisms of Neuroinflammation in Lysosomal Storage Diseases." Biomedicines 11, no. 4 (2023): 1067. http://dx.doi.org/10.3390/biomedicines11041067.

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Lysosomal storage diseases are a group of rare and ultra-rare genetic disorders caused by defects in specific genes that result in the accumulation of toxic substances in the lysosome. This excess accumulation of such cellular materials stimulates the activation of immune and neurological cells, leading to neuroinflammation and neurodegeneration in the central and peripheral nervous systems. Examples of lysosomal storage diseases include Gaucher, Fabry, Tay–Sachs, Sandhoff, and Wolman diseases. These diseases are characterized by the accumulation of various substrates, such as glucosylceramide

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Tudorancea, Ivona Maria, Mitică Ciorpac, Gabriela Dumitrița Stanciu, et al. "The Therapeutic Potential of the Endocannabinoid System in Age-Related Diseases." Biomedicines 10, no. 10 (2022): 2492. http://dx.doi.org/10.3390/biomedicines10102492.

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The endocannabinoid system (ECS) dynamically regulates many aspects of mammalian physiology. ECS has gained substantial interest since growing evidence suggests that it also plays a major role in several pathophysiological conditions due to its ability to modulate various underlying mechanisms. Furthermore, cannabinoids, as components of the cannabinoid system (CS), have proven beneficial effects such as anti-inflammatory, immunomodulatory, neuromodulatory, antioxidative, and cardioprotective effects. In this comprehensive review, we aimed to describe the complex interaction between CS and mos

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Lénárt, Nikolett, David Brough, and Ádám Dénes. "Inflammasomes link vascular disease with neuroinflammation and brain disorders." Journal of Cerebral Blood Flow & Metabolism 36, no. 10 (2016): 1668–85. http://dx.doi.org/10.1177/0271678x16662043.

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The role of inflammation in neurological disorders is increasingly recognised. Inflammatory processes are associated with the aetiology and clinical progression of migraine, psychiatric conditions, epilepsy, cerebrovascular diseases, dementia and neurodegeneration, such as seen in Alzheimer’s or Parkinson’s disease. Both central and systemic inflammatory actions have been linked with the development of brain diseases, suggesting that complex neuro-immune interactions could contribute to pathological changes in the brain across multiple temporal and spatial scales. However, the mechanisms throu

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Laurindo, Lucas Fornari, Otávio Augusto Garcia Simili, Adriano Cressoni Araújo, et al. "Melatonin from Plants: Going Beyond Traditional Central Nervous System Targeting—A Comprehensive Review of Its Unusual Health Benefits." Biology 14, no. 2 (2025): 143. https://doi.org/10.3390/biology14020143.

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Melatonin is indispensable for the homeostasis of plants and animals. In humans, it can help prevent or be an adjuvant treatment for several diseases mainly related to the immune system, inflammation, and oxidative stress. Moreover, a melatonin-rich diet is linked to several health benefits, such as regulation of circadian rhythm, regulation of the immunological system, epilepsy control, delaying the aging process, and diminishing hormones related to cancer. This review aimed to show the effects of melatonin in diseases beyond its traditional use. The results showed it can present scavenging o

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Fan, Min, Subash C. B. Gopinath, and Hongjuan Gao. "Gold structured nanourchin as anesthetic biosensor for determining neuroprotective effect." Materials Express 14, no. 4 (2024): 545–50. http://dx.doi.org/10.1166/mex.2024.2640.

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Research has proven that confusion, delirium, and cognitive dysfunction are the common symptoms caused by anesthesia. In general, neuroproteins fluctuation is common during the process of anesthetic treatment. Among others, tau protein was found to the higher fluctuations after the anesthetic process, and it is highly associated with various neuro-related diseases. Monitoring the level of tau protein helps to identify the neuro-related problems during and after the anesthetic processes. This research was focused on developing a tau electrochemical impedance biosensor on aptamer and antibody-mo

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Li, Xi, Guanghua Xiong, Manni Luo, et al. "Peptidoglycan recognition protein PGRP-5 is involved in immune defence and neuro-behavioral disorders in zebrafish embryos." PLOS ONE 20, no. 1 (2025): e0315714. https://doi.org/10.1371/journal.pone.0315714.

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Peptidoglycan recognition proteins (PGRPs) are the evolutionarily highly conserved class of pattern recognition receptors, however, their functions on the innate immune system and neuro-inflammatory response in aquatic organism are still poorly understood. In this study, we systematically investigated the molecular functions of PGRPs in zebrafish embryos. Firstly, we identified three PGRPs in zebrafish and phylogenetic analysis suggested that DrPGRP-5 was a novel member of the PGRP superfamily in evolution. Secondly, the endogenous mRNA levels of DrPGRP-5 were highly expressed in brain and mus

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CHEN, JIANXIN, ZHENHUA JIA, XIANGCHUN WU, et al. "SELECTING BIOMARKERS FOR PRIMARY HYPERLIPIDEMIA AND UNSTABLE ANGINA IN THE CONTEXT OF NEURO-ENDOCRINE-IMMUNE NETWORK BY FEATURE SELECTION METHODS." Journal of Biological Systems 18, no. 03 (2010): 605–19. http://dx.doi.org/10.1142/s0218339010003433.

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Hyperlipidemia (HL) and unstable angina (UA) are two sequential diseases that cause more and more morbidity and mortality world-wide. Biomarkers selection in the level of physical and chemical specifications (PCS) plays a key role in understanding the pathology of both diseases. Neuro-Endocrine-Immune (NEI) system is a preferable pathway to investigate the interaction network of related PCS in the context of HL and UA. Data mining approaches are a kind of advanced statistical methods to unravel the "secret" of interaction network of PCS in both diseases. Feature selection methods are a branch

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Gupta, Rakesh Kumar, Poonam Wasnik, Deepankar Mondal, and Dhananjay Shukla. "Critical role of keratinocytes in cutaneous immune responses." Exploration of Immunology 4, no. 4 (2024): 502–22. http://dx.doi.org/10.37349/ei.2024.00155.

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Keratinocytes play an integral role in the human epidermis, serving as a barrier between the internal and external environment. They are immune-competent cells involved in both innate and adaptive cutaneous immune responses, crucial for maintaining skin integrity. Keratinocytes are essential for epidermal repair, facilitating proliferation and re-epithelialization following injury. They secrete pro-inflammatory markers such as cytokines and chemokines, which promote the recruitment of inflammatory cells like polymorphs and macrophages to the site of skin injury. The immune response mediated by

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Tauber, M., and L. Basso. "Neuro-immune interactions in allergic skin diseases." Revue Française d'Allergologie, June 2021. http://dx.doi.org/10.1016/j.reval.2021.05.001.

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Wu, Wanhua, Jianing Li, Su Chen, and Suidong Ouyang. "The airway neuro-immune axis as a therapeutic target in allergic airway diseases." Respiratory Research 25, no. 1 (2024). http://dx.doi.org/10.1186/s12931-024-02702-8.

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AbstractRecent evidence has increasingly underscored the importance of the neuro-immune axis in mediating allergic airway diseases, such as allergic asthma and allergic rhinitis. The intimate spatial relationship between neurons and immune cells suggests that their interactions play a pivotal role in regulating allergic airway inflammation. Upon direct activation by allergens, neurons and immune cells engage in interactions, during which neurotransmitters and neuropeptides released by neurons modulate immune cell activity. Meanwhile, immune cells release inflammatory mediators such as histamin

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Straub, Rainer H., and Maurizio Cutolo. "A history of psycho-neuro-endocrine immune interactions in rheumatic diseases." Neuroimmunomodulation, August 21, 2024. http://dx.doi.org/10.1159/000540959.

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Background All active scientists stand on the shoulders of giants and many other more anonymous scientists, and this is not different in our field of psycho-neuro-endocrine immunology in rheumatic diseases. Too often, the modern world of publishing forgets about the collective enterprise of scientists. Some journals advise the authors to present only literature of the last decade, and it became a natural attitude of many scientists to present only the latest publications. In order to work against this general unempirical behavior, Neuroimmunomodulation devotes the 30-year anniversary issue to

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Zhu, Yan, Shixin Duan, Mei Wang, Zhili Deng, and Ji Li. "Neuroimmune Interaction: A Widespread Mutual Regulation and the Weapons for Barrier Organs." Frontiers in Cell and Developmental Biology 10 (May 11, 2022). http://dx.doi.org/10.3389/fcell.2022.906755.

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Since the embryo, the nervous system and immune system have been interacting to regulate each other’s development and working together to resist harmful stimuli. However, oversensitive neural response and uncontrolled immune attack are major causes of various diseases, especially in barrier organs, while neural-immune interaction makes it worse. As the first defense line, the barrier organs give a guarantee to maintain homeostasis in external environment. And the dense nerve innervation and abundant immune cell population in barrier organs facilitate the neuroimmune interaction, which is the p

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Sudeep Kumar Brahma. "NEURO-ENDOCRINE-IMMUNE MODULATION BY AYURVEDIC RASAYANA DRUGS." International Journal of Ayurveda and Pharma Research, January 13, 2021, 51–71. http://dx.doi.org/10.47070/ijapr.v8i12.1734.

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Ayurveda which literally means “the science of life” is the ancient Indian system of medicine. Rasayana is a specialized branch of Ayurveda. The drugs attributed with Rasayana properties are mentioned to delay the process of ageing, enhance the mental and cognitive functions and deliver freedom from a number of diseases including those caused by infectious organisms. All these activities of Rasayana drugs seem to be associated with neuro-endocrine-immune systems. In this study we have reviewed the growing inter-relationship between the neuro-endocrine-immune systems and stress as a neuro-endoc

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De Luca, Ciro, Anna Maria Colangelo, Lilia Alberghina, and Michele Papa. "Neuro-Immune Hemostasis: Homeostasis and Diseases in the Central Nervous System." Frontiers in Cellular Neuroscience 12 (November 26, 2018). http://dx.doi.org/10.3389/fncel.2018.00459.

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Hou, Xiaoxiao, and David Artis. "Neuro-immune cell interactions in the regulation of intestinal immune homeostasis." Current Opinion in Gastroenterology, October 18, 2024. http://dx.doi.org/10.1097/mog.0000000000001065.

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Purpose of the review Bidirectional regulation between neurons and immune cells in the intestine governs essential physiological processes, including digestion, metabolism and motility, while also controlling intestinal inflammation and maintaining tissue homeostasis. This review covers recent advances and future research challenges focused on the regulatory molecules and potential therapeutic targets in neuron-immune interactions within the intestine. Recent findings Recently identified molecular and cellular pathways have been shown to regulate neuron-immune cell cross talk in the context of

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Lepennetier, Gildas, Zsuzsanna Hracsko, Marina Unger, et al. "Cytokine and immune cell profiling in the cerebrospinal fluid of patients with neuro-inflammatory diseases." Journal of Neuroinflammation 16, no. 1 (2019). http://dx.doi.org/10.1186/s12974-019-1601-6.

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Abstract Background Cytokines play multiple roles during neuro-inflammatory processes and several cytokines have been studied in the context of specific diseases. This study provides a comprehensive picture of cerebrospinal fluid (CSF) changes during neuro-inflammation by analyzing multiple cytokines in combination with immune cell subsets and standard CSF parameters. Methods Using multiplex assays, we simultaneously measured 36 cytokines (CCL1–3, CCL7, CCL8, CCL11, CCL13, CCL19, CCL20, CCL22–27, CXCL1, CXCL2, CXCL5, CXCL6, CXCL8, CXCL9, CXCL11–13, CXCL16, CX3CL1, IL2, IL4, IL6, IL10, IL16, GM

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You, Xin-yu, Han-yu Zhang, Xu Han, Fang Wang, Peng-wei Zhuang, and Yan-jun Zhang. "Intestinal Mucosal Barrier Is Regulated by Intestinal Tract Neuro-Immune Interplay." Frontiers in Pharmacology 12 (May 31, 2021). http://dx.doi.org/10.3389/fphar.2021.659716.

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Inflammatory bowel disease, irritable bowel syndrome and severe central nervous system injury can lead to intestinal mucosal barrier damage, which can cause endotoxin/enterobacteria translocation to induce infection and is closely related to the progression of metabolic diseases, cardiovascular and cerebrovascular diseases, tumors and other diseases. Hence, repairing the intestinal barrier represents a potential therapeutic target for many diseases. Enteral afferent nerves, efferent nerves and the intrinsic enteric nervous system (ENS) play key roles in regulating intestinal physiological home

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