Relevant bibliographies by topics / NEUROBLASTOMA, HIF-2α

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'NEUROBLASTOMA, HIF-2α'

Author: Grafiati
Published: 11 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "NEUROBLASTOMA, HIF-2α"

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Pini, Nicola, Zihe Huo, Urs Kym, Stefan Holland-Cunz, and Stephanie J. Gros. "AQP1-Driven Migration Is Independent of Other Known Adverse Factors but Requires a Hypoxic Undifferentiated Cell Profile in Neuroblastoma." Children 8, no. 1 (2021): 48. http://dx.doi.org/10.3390/children8010048.

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Neuroblastoma is a biologically very heterogeneous tumor with its clinical manifestation ranging from spontaneous regression to highly aggressive metastatic disease. Several adverse factors have been linked to oncogenesis, tumor progression and metastases of neuroblastoma including NMYC amplification, the neural adhesion molecule NCAM, as well as CXCR4 as a promoter of metastases. In this study, we investigate to what extent the expression of AQP1 in neuroblastoma correlates with changing cellular factors such as the hypoxic status, differentiation, expression of known adverse factors such as
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Holmquist-Mengelbier, Linda, Erik Fredlund, Tobias Löfstedt та ін. "Recruitment of HIF-1α and HIF-2α to common target genes is differentially regulated in neuroblastoma: HIF-2α promotes an aggressive phenotype". Cancer Cell 10, № 5 (2006): 413–23. http://dx.doi.org/10.1016/j.ccr.2006.08.026.

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Montalto, Angela Simona, Monica Currò, Tiziana Russo, et al. "CO2 Pneumoperitoneum Effects on Molecular Markers of Tumor Invasiveness in SH-SY5Y Neuroblastoma Cells." European Journal of Pediatric Surgery 30, no. 06 (2019): 524–28. http://dx.doi.org/10.1055/s-0039-1700547.

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Abstract Introduction CO2 pneumoperitoneum can influence the biological behavior of neuroblastoma (NB). Angiogenesis and genetic features are responsible for malignant phenotype of this tumor. We examined the CO2 effects on N-Myc, vascular endothelial growth factor (VEGF), and matrix metalloproteinase-2 (MMP-2) expression as critical biomarkers of tumor invasiveness, in NB cells without N-Myc amplification. Materials and Methods SH-SY5Y cells were exposed to CO2 (100%) at 15 mm Hg pressure for 4 hours and then moved to normal condition for 24 hours. Control cells were incubated with 5% CO2 for
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Cimmino, F., M. Avitabile, L. Pezone та ін. "CD55 is a HIF-2α marker with anti-adhesive and pro-invading properties in neuroblastoma". Oncogenesis 5, № 4 (2016): e212-e212. http://dx.doi.org/10.1038/oncsis.2016.20.

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Hafizi, Redona, Faik Imeri, Roland H. Wenger та Andrea Huwiler. "S1P Stimulates Erythropoietin Production in Mouse Renal Interstitial Fibroblasts by S1P1 and S1P3 Receptor Activation and HIF-2α Stabilization". International Journal of Molecular Sciences 22, № 17 (2021): 9467. http://dx.doi.org/10.3390/ijms22179467.

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Erythropoietin (Epo) is the critical hormone for erythropoiesis. In adults, Epo is mainly produced by a subset of interstitial fibroblasts in the kidney, with minor amounts being produced in the liver and the brain. In this study, we used the immortalized renal interstitial fibroblast cell line FAIK F3-5 to investigate the ability of the bioactive sphingolipid sphingosine 1-phosphate (S1P) to stimulate Epo production and to reveal the mechanism involved. Stimulation of cells with exogenous S1P under normoxic conditions (21% O2) led to a dose-dependent increase in Epo mRNA and protein levels an
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Nilsson, Helén, Annika Jögi, Siv Beckman, Adrian L. Harris, Lorenz Poellinger та Sven Påhlman. "HIF-2α expression in human fetal paraganglia and neuroblastoma: relation to sympathetic differentiation, glucose deficiency, and hypoxia". Experimental Cell Research 303, № 2 (2005): 447–56. http://dx.doi.org/10.1016/j.yexcr.2004.10.003.

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Nilsson, M. B., P. E. Zage, L. Zeng та ін. "Multiple receptor tyrosine kinases regulate HIF-1α and HIF-2α in normoxia and hypoxia in neuroblastoma: implications for antiangiogenic mechanisms of multikinase inhibitors". Oncogene 29, № 20 (2010): 2938–49. http://dx.doi.org/10.1038/onc.2010.60.

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Hamidian, Arash, Kristoffer von Stedingk, Matilda Munksgaard Thorén, Sofie Mohlin та Sven Påhlman. "Differential regulation of HIF-1α and HIF-2α in neuroblastoma: Estrogen-related receptor alpha (ERRα) regulates HIF2A transcription and correlates to poor outcome". Biochemical and Biophysical Research Communications 461, № 3 (2015): 560–67. http://dx.doi.org/10.1016/j.bbrc.2015.04.083.

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Pietras, A., D. Gisselsson, I. Øra та ін. "High levels of HIF-2α highlight an immature neural crest-like neuroblastoma cell cohort located in a perivascular niche". Journal of Pathology 214, № 4 (2007): 482–88. http://dx.doi.org/10.1002/path.2304.

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Chen, Shu-jen, Nicholas E. Hoffman, Santhanam Shanmughapriya та ін. "A Splice Variant of the Human Ion Channel TRPM2 Modulates Neuroblastoma Tumor Growth through Hypoxia-inducible Factor (HIF)-1/2α". Journal of Biological Chemistry 289, № 52 (2014): 36284–302. http://dx.doi.org/10.1074/jbc.m114.620922.

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Dissertations / Theses on the topic "NEUROBLASTOMA, HIF-2α"

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PEZONE, Lucia. "Cellular proteome alterations in response to hypoxia inducible factor HIF-2α in normoxic neuroblastoma cells". Doctoral thesis, 2016. http://hdl.handle.net/11562/938441.

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Il neuroblastoma (NB) è un tumore embrionale del sistema nervoso simpatico, che deriva da cellule della cresta neurale. È il tumore extracranico più diffuso tra i bambini di età inferiore a un anno e rappresenta circa il 7% di tutti i tumori infantili. L'ipossia si sviluppa comunemente durante la crescita tumorale ed è associata ad una prognosi infausta con resistenza ai trattamenti terapeutici. Molte evidenze suggeriscono una correlazione tra i fattori ipossia-inducibile (HIF), HIF-1α e HIF-2α, con il grado di differenziamento e quindi l’aggressività tumorale. In particolare, nel Neuroblastom
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