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Books on the topic 'Neurocognitive disorder'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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1

Nash, Maureen, and Sarah Foidel, eds. Neurocognitive Behavioral Disorders. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11268-4.

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2

Lishman, William Alwyn. Organic psychiatry: The psychological consequences of cerebral disorder. 3rd ed. Blackwell Scientific, 1987.

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3

J, Vinken P., and Frederiks J. A. M, eds. Neurobehavioural disorders. Elsevier Science, 1985.

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4

1958-, Barnes Marcia A., ed. Genes, brain, and development: The neurocognition of genetic disorders. Cambridge University Press, 2010.

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5

P, Tsai James, ed. Leading-edge cognitive disorders research. Nova Science Publishers, 2008.

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6

C, Yudofsky Stuart, and Hales Robert E, eds. Synopsis of neuropsychiatry. American Psychiatric Press, 1994.

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7

C, Yudofsky Stuart, Hales Robert E, and American Psychiatric Press, eds. The American Psychiatric Press textbook of neuropsychiatry. 2nd ed. American Psychiatric Press, 1992.

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8

Luthra, Atul Sunny. The meaning of behaviors in dementia/neurocognitive disorders: New terminology, classification, and behavioral management. Common Ground, 2014.

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9

C, Yudofsky Stuart, Hales Robert E, and American Psychiatric Press, eds. The American Psychiatric Press textbook of neuropsychiatry. 3rd ed. American Psychiatric Press, 1997.

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10

E, Hales Robert, and Yudofsky Stuart C, eds. The American Psychiatric Press textbook of neuropsychiatry. The Press, 1987.

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11

Neurocognitive Disorder: Interventions to Support Occupational Performance. American Occupational Therapy Association, Incorporated, 2014.

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12

Balanzá-Martínez, Vicent, Sofia Brissos, Maria Lacruz, and Rafael Tabarés-Seisdedos. Pharmacotherapy of bipolar disorder: impact on neurocognition. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198748625.003.0025.

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Neurocognitive dysfunction is a core feature of bipolar disorder (BD), which may be further compounded by several clinical factors, such as medications. There is growing interest on the potential impact of pharmacotherapy (lithium, anticonvulsants, antipsychotics, and other) on neurocognition. This chapter summarizes a critical, descriptive update of the literature, mostly focused on human data. Based on current studies, medication-associated neurocognitive side effects cannot be clearly distinguished from those intrinsic to BD. Moreover, available research is limited by several methodological

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13

Alexander, Joshua, David J. Croteau, and Ronald J. Ellis. HIV-Associated Neurocognitive Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0147.

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Three subclassifications of HIV Associated Neurocognitive Disorders (HAND) can be delineated: asymptomatic neurocognitive impairment (ANI), mild neurocognitive disorder (MND), and HIV-associated dementia (HAD). HAND occurs in about half of patients infected with HIV. All forms, including ANI and MND, are associated with unemployment and reduced antiretroviral adherence, which can lead to loss of virologic suppression, further neurocognitive decline, and systemic illness. Fortunately, combined antiretroviral therapy can stabilize and even reverse cognitive impairment.

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14

Letendre, Scott, Jennifer Iudicello, Beau Ances, Thomas D. Marcotte, Serena Spudich, and Mary Ann Cohen. HIV-Associated Neurocognitive Disorders. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0016.

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The human immunodeficiency virus (HIV) enters the central nervous system soon after infection; can infect glia and tissue macrophages in the brain; and can injure neurons, resulting in loss of dendrites. These and other processes underpin a syndrome of cognitive and motor impairment termed HIV-associated neurocognitive disorder (HAND). This chapter principally focuses on HAND, although delirium and other neurocognitive disorders are also discussed and should remain in the differential diagnosis of cognitive impairment in persons with HIV. A differential diagnosis of cognitive impairment in HIV

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15

Hasbun, Rodrigo, Richard Dunham, Joseph S. Kass, et al. HIV-Associated Neurocognitive Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0038.

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HIV causes a chronic form of encephalitis (HIVE) that is clinically characterized by either dementia or mild neurocognitive impairment. Since the introduction of antiretroviral therapy in 1996, the incidence of HIV dementia has decreased by 50%, but the prevalence of mild neurocognitive disorder has increased up to 39%. HIVE is the result of direct microglial infection, interruption of trophic factors, or caused by inflammatory cytokines. HIV enters the brain primarily by the “Trojan horse mechanism”; it is carried by monocytes and lymphocytes that cross the blood–brain barrier. HIV has a pred

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16

Comorbidity and Developmental Neurocognitive Disorders: A Special Issue of developmental Neuropsychology. Lawrence Erlbaum, 1995.

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17

Sinha, Shirshendu, and Kristina Zdanys. Clinical Aspects of Neuropsychiatric Disorders. Edited by Shirshendu Sinha. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190265557.003.0010.

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In this chapter clinical aspects of neuropsychiatric disorders including delirium, mild neurocognitive disorders, major neurocognitive disorders, major neurocognitive disorder due to Parkinson’s disease, major neurocognitive disorder due to multiple etiologies and sleep wake disorders will be reviewed

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18

Drane, Daniel L., and Dona E. C. Locke. Mechanisms of Possible Neurocognitive Dysfunction. Edited by Barbara A. Dworetzky and Gaston C. Baslet. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190265045.003.0005.

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This chapter covers what is known about the possible mechanisms of neurocognitive dysfunction in patients with psychogenic nonepileptic seizures (PNES). It begins with a review of all research examining possible cognitive deficits in this population. Cognitive research in PNES is often obscured by noise created by a host of comorbid conditions (e.g., depression, post-traumatic stress disorder, chronic pain) and associated issues (e.g., effects of medications and psychological processes that can compromise attention or broader cognition). More recent studies employing performance validity tests

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19

Torrent, Carla, Caterina del Mar Bonnin, and Anabel Martinez-Arán. Functional remediation therapy for bipolar disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198748625.003.0014.

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Impairment in functional outcome is commonly observed even when patients are euthymic and includes multiple areas of functioning. Many factors such as sociodemographic, clinical, pharmacological, and neurocognitive variables have been associated with functional impairment. The term ‘functional remediation’ has been coined to define an innovative strategy aimed at targeting the critical factors for full psychosocial adjustment and functional recovery in the context of psychoses and more specifically bipolar disorders. Functional remediation involves not only neurocognitive techniques and traini

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20

Autism: An integrated view from neurocognitive, clinical, and intervention research. Blackwell Pub., 2008.

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21

Rucci, Jennifer M., and Robert E. Feinstein. Neurocognitive Disorders and Mental Disorders Due to Another Medical Condition. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199326075.003.0005.

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The defining feature of neurocognitive disorders is a decline in cognitive functioning. Patients suffering from delirium experience an acute change in mental status, fluctuating levels of consciousness, and an inability to acquire new information. Patients with major neurocognitive disorder experience significant cognitive decline in complex attention, executive function, learning and memory, language, perceptual-motor, and social cognition. The chapter also discusses mental disorders due to another medical condition. These patients can experience psychotic, mood, or anxious symptoms or a pers

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22

Newman, Jennifer, and Charles R. Marmar. Executive Function in Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0015.

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This chapter discusses the role of executive function in post-traumatic stress disorder (PTSD), which is far from fully understood. Deficits are subtle and findings are often inconsistent. Impairments have been related to worsening of psychological symptoms, functioning, and quality of life. They can also negatively impact treatment. Functional imaging shows that neurocognitive deficits in PTSD may be related to an imbalance in brain connectivity, where emotion processing is enhanced and control is reduced. Structural findings show abnormalities in brain regions involved in higher-level functi

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23

D), Daniels Hommes (Ph. CBD Oil Neurocognitive Disorder : Neurodegenerative Disorders: Alzheimer Disease; Movement Disorders; Multiple Sclerosis; Parkinson & Prion Disease; How CBD Oil Is the Cure. Independently Published, 2019.

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24

Mahon, Katie, Manuela Russo, and M. Mercedes Perez-Rodriguez. Cognitive Enhancement in Bipolar Disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190214401.003.0011.

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Neurocognitive deficits are acknowledged as integral features of bipolar disorder (BD) and are known to contribute to the compromised level of functioning in individuals with BD. This chapter provides an overview of the current state of cognitive enhancement in BD. Few pharmacological agents have been investigated with regard to their potential for pro-cognitive effects in BD. Dopaminergic agents (pramipexole) and stimulants (modafinil, armodafinil, and amphetamine) as adjunctive treatment in BD appear to be promising cognitive enhancers, and there are few ongoing randomized clinical trials ta

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25

Oladeji, Bibilola D., and Kevin R. Robertson. Neuropsychological Evaluation for Persons with HIV and AIDS. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0012.

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With recent developments in the classification and definitions for HIV-associated neurocognitive disorders (HAND), neuropsychological assessments have become central to the diagnostic process. The pattern and manifestation of neurocognitive dysfunction associated with HIV have changed since the introduction of effective antiretroviral medications and their increasingly widespread use. Prior to the introduction of antiretroviral therapy, severe cognitive disorder presenting as HIV-associated dementia (HAD) was a common manifestation of HIV-associated neurocognitive disorders. In more recent tim

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26

(), Daniels HOMMES. Cbd Oil for Neurocognitive Disorder: The Easy to Read Guide to Understanding and Curing All Kinds of Nuerocognitive Disorder with Cbd Oil. Independently Published, 2019.

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27

Doyle, Lauren R., and Sarah N. Mattson. Behavioral Teratogenic Effects of Alcohol: Focus on Neurobehavioral Disorder Associated With Prenatal Alcohol Exposure. Edited by Thomas H. Ollendick, Susan W. White, and Bradley A. White. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190634841.013.39.

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Prenatal exposure to teratogens may alter fetal development and significantly impact later life. Perhaps the best known teratogen is alcohol; prenatal alcohol exposure causes a broad range of effects that can cause lifelong impairment. Of greatest significance are the functional impairments in behavior and cognition. Recognition of these impairments led to the inclusion of the neurobehavioral disorder associated with prenatal alcohol exposure (ND-PAE) in the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders under “conditions for further study.” This proposed diagnosis

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28

Eckenhoff, Roderic G., and Niccolò Terrando. Perioperative Neurocognitive Disorders. Cambridge University Press, 2019.

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29

Eckenhoff, Roderic G., and Niccolò Terrando. Perioperative Neurocognitive Disorders. Cambridge University Press, 2019.

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30

Eckenhoff, Roderic G., and Niccolò Terrando. Perioperative Neurocognitive Disorders. Cambridge University Press, 2019.

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31

Hsu, David C., and James M. Ellison. Mild Cognitive Impairment. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199374656.003.0032.

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Mild cognitive impairment (MCI), and the newly described DSM-5 diagnosis of mild neurocognitive disorder, often lie at the intersection of geriatric and forensic psychiatry. The concept of MCI as a distinct syndrome between normal cognitive aging and major neurocognitive disorder (dementia) has continued to evolve over the past decade. Given MCI’s range of potential etiologies, as well as its increasing prevalence in an aging population, its evaluation and management can perplex the seasoned clinician and complicate legal proceedings. This chapter describes the clinical phenomenon of MCI, as w

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32

Lishman, William Alwyn. Organic Psychiatry: The Psychological Consequences of Cerebral Disorder. 2nd ed. Blackwell Science Inc, 1988.

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33

Hu, Guoku, Shilpa Buch, and Huangui Xiong. HIV-Associated Neurocognitive Disorders. Elsevier Science & Technology Books, 2024.

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34

Nie, Pei Huey, and David L. Sultzer. Treatments for Neurocognitive Disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780199342211.003.0026.

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Dementia, or neurocognitive disorders, refers to a number of clinical syndromes originating in brain pathology and characterized by cognitive deficits and functional impairment. This chapter provides an update on treatment options in addition to a brief summary of dementia types and an overview of the diagnostic criteria for cognitive disorders. The diagnosis of dementia is ultimately a clinical one and includes a multidimensional perspective; as such, treatment requires a comprehensive approach. This chapter addresses two aspects of the treatment of neurocognitive disorders: pharmacological i

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35

Eckenhoff, Roderic G., and Niccolò Terrando, eds. The Perioperative Neurocognitive Disorders. Cambridge University Press, 2019. http://dx.doi.org/10.1017/9781316402504.

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36

HIV-Associated Neurocognitive Disorders. Elsevier Science & Technology, 2024.

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37

Kempler, Daniel. Neurocognitive Disorders in Aging. Sage Publications, Inc, 2004.

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38

Kempler, Daniel. Neurocognitive Disorders in Aging. Sage Publications, Inc, 2004.

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39

Neurocognitive disorders in aging. Sage Publications, 2005.

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40

Cournos, Francine, Karen McKinnon, and Milton Wainberg. Epidemiology of Psychiatric Disorders Associated with HIV and AIDS. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0003.

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This chapter presents the prevalence of common and severe mental illnesses among people with HIV infection, as well as the prevalence of HIV infection among people with severe mental illness. It begins with a look at population-based studies, which are limited in number, then discusses specific disorders studied in smaller studies with selected populations. While the chapter is largely focused on epidemiology in the United States, selected studies from other regions are cited. Taken together, studies show that people with HIV infection have high rates of HIV-associated neurocognitive disorders

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41

Neurocognitive Development: Disorders and Disabilities. Elsevier, 2020. http://dx.doi.org/10.1016/c2017-0-02918-x.

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42

David, Cohen. Neurocognitive Development: Disorders and Disabilities. Elsevier, 2020.

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43

Bosch, Annet M., and Elaine Murphy. Galactosemia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0002.

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There are three known inherited disorders of galactose metabolism: classic galactosemia (galactose-1-phosphate uridyltransferase deficiency), galactokinase deficiency, and uridine diphosphate galactose 4-epimerase deficiency. Classic galactosemia presents in the newborn period with liver and renal impairment and failure to thrive. Acute symptoms resolve when lactose is excluded from the diet, but long-term complications are frequent and include neurocognitive and social difficulties, speech and language problems, motor problems, and premature ovarian insufficiency. Patients with galactokinase

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44

Galvin, James E., and Jose Tomas Bras. Neurobiology of Lewy Body Dementias. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0055.

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Lewy body dementia (LBD) is the second most common form of neurocognitive disorder after Alzheimer’s disease and covers two related diagnoses: Dementia with Lewy Bodies and Parkinson’s Disease Dementia. Despite being a common disorder, diagnosis outside expert academic centers remains a significant challenge. The core pathological feature of LBD is the cortical Lewy body; however, many cases will have coexistent Alzheimer disease pathology. Genetic risk factors for LBD include mutations in genes for alpha-synuclein (SNCA) and galactocerbrosidase (GBA). Dopamine transporter imaging remains the

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45

Association, Information Resources Management. Neurocognitive Disorders: Breakthroughs in Research and Practice. IGI Global, 2020.

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46

Association, Information Resources Management. Neurocognitive Disorders: Breakthroughs in Research and Practice. IGI Global, 2020.

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47

Association, Information Resources Management. Neurocognitive Disorders: Breakthroughs in Research and Practice. IGI Global, 2020.

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48

Association, Information Resources Management. Neurocognitive Disorders: Breakthroughs in Research and Practice. IGI Global, 2020.

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49

Abnormal Minds: INDEX to the NeuroCognitive Disorders. Lulu Press, Inc., 2022.

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50

Sorscher, Nechama. Your Neurodiverse Child. Rowman & Littlefield Publishers, 2025. https://doi.org/10.5040/9798881846435.

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Provides invaluable information and practical strategies to empower neurodiverse children to thrive. Globally, the prevalence of children with challenges has increased dramatically. Unfortunately, these children are often labeled as “bad,” “stupid,” or “lazy.” They are frequently left feeling totally helpless and despondent about their problems, blaming themselves for their poor performance. It’s essential for parents, caregivers, and teachers to understand how to support these kids to ensure their success. In Your Neurodiverse Child: How to Help Kids with Learning, Attention, and Neurocogniti

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