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Journal articles on the topic 'Neurology; Magnetic resonance imaging'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Trevisan, C., M. Spagnoli, G. Crisi, and L. Mavilla. "Magnetic Resonance Imaging." European Neurology 29, no. 2 (1989): 33–35. http://dx.doi.org/10.1159/000116464.

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2

Kuzniecky, Ruben. "Magnetic resonance and functional magnetic resonance imaging." Current Opinion in Neurology 10, no. 2 (1997): 88–91. http://dx.doi.org/10.1097/00019052-199704000-00003.

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3

Humberstone, Miles R., and Guy V. Sawle. "Functional Magnetic Resonance Imaging in Clinical Neurology." European Neurology 36, no. 3 (1996): 117–24. http://dx.doi.org/10.1159/000117227.

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4

Herzog, Richard J., Alexander J. Ghanayem, Richard D. Guyer, Arnold Graham-Smith, Edward D. Simmons, and Alexander Vaccaro. "Magnetic resonance imaging." Spine Journal 3, no. 3 (2003): 6–10. http://dx.doi.org/10.1016/s1529-9430(02)00559-4.

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5

Elster, Allen D. "Magnetic resonance imaging." Surgical Neurology 32, no. 6 (1989): 478–79. http://dx.doi.org/10.1016/0090-3019(89)90018-9.

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6

Farrall, A. J. "Magnetic resonance imaging." Practical Neurology 6, no. 5 (2006): 318–25. http://dx.doi.org/10.1136/jnnp.2006.091843.

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7

Penry, K. "Magnetic resonance imaging." Electroencephalography and Clinical Neurophysiology 61, no. 3 (1985): S2. http://dx.doi.org/10.1016/0013-4694(85)90053-7.

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8

Zhong, J., and D. Bavelier. "Functional Magnetic Resonance Imaging." Neurology 64, no. 7 (2005): 1323. http://dx.doi.org/10.1212/01.wnl.0000164847.45244.a1.

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9

Bendersky, Mariana, Inés Tamer, Juan Van Der Velde, et al. "Prenatal cerebral magnetic resonance imaging." Journal of the Neurological Sciences 275, no. 1-2 (2008): 37–41. http://dx.doi.org/10.1016/j.jns.2008.07.012.

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10

Jackson, Graeme D., and Alan Connelly. "Magnetic resonance imaging and spectroscopy." Current Opinion in Neurology 9, no. 2 (1996): 82–88. http://dx.doi.org/10.1097/00019052-199604000-00004.

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11

Katscher, Ulrich, and Peter Börnert. "Parallel magnetic resonance imaging." Neurotherapeutics 4, no. 3 (2007): 499–510. http://dx.doi.org/10.1016/j.nurt.2007.04.011.

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12

Firsching, Raimund P., Walter Heindel, Ralf-Ingo Ernestus, Reinhold A. Frowein, and Jürgen Bunke. "Postoperative magnetic resonance imaging artifacts." Journal of Neurosurgery 67, no. 5 (1987): 776–78. http://dx.doi.org/10.3171/jns.1987.67.5.0776.

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✓ Artifacts are occasionally encountered on magnetic resonance imaging after operation. These may be due to minute metallic particles from neurosurgical instruments. Particles not detectable on plain x-ray films or computerized tomography scans may cause local change of magnetic resonance activity, resulting in a deceptive magnetic resonance appearance. Three illustrative case reports are presented.

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13

Olanow, C. W. "Magnetic Resonance Imaging in Parkinsonism." Neurologic Clinics 10, no. 2 (1992): 405–20. http://dx.doi.org/10.1016/s0733-8619(18)30218-4.

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14

Aichner, Franz T., Stephan R. Felber, Günther G. Birbamer, and Andrea Posch. "Magnetic Resonance Imaging and Magnetic Resonance Angiography of Vertebrobasilar Dolichoectasia." Cerebrovascular Diseases 3, no. 5 (1993): 280–84. http://dx.doi.org/10.1159/000108716.

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15

Kuyumcu, Gokhan, Carlos Zamora, Noushin Yahyavi-Firouz-Abadi, and Marinos Kontzialis. "Magnetic resonance imaging of ataxia-telangiectasia." Neurology India 64, no. 7 (2016): 129. http://dx.doi.org/10.4103/0028-3886.178058.

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16

Novotny Jr, Edward J. "Metabolic brain imaging by magnetic resonance." Future Neurology 1, no. 5 (2006): 659–63. http://dx.doi.org/10.2217/14796708.1.5.659.

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17

Koltzenburg, Martin, and Tarek Yousry. "Magnetic resonance imaging of skeletal muscle." Current Opinion in Neurology 20, no. 5 (2007): 595–99. http://dx.doi.org/10.1097/wco.0b013e3282efc322.

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18

Ciumas, Carolina, Alexandra Montavont, and Philippe Ryvlin. "Magnetic resonance imaging in clinical trials." Current Opinion in Neurology 24, no. 4 (2008): 431–36. http://dx.doi.org/10.1097/wco.0b013e3283056a3c.

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19

Schenck, John F. "Magnetic resonance imaging of brain iron." Journal of the Neurological Sciences 207, no. 1-2 (2003): 99–102. http://dx.doi.org/10.1016/s0022-510x(02)00431-8.

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20

Clanet, Michel, and Isabelle Berry. "Magnetic resonance imaging in multiple sclerosis." Current Opinion in Neurology 11, no. 4 (1998): 299–303. http://dx.doi.org/10.1097/00019052-199808000-00004.

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21

Roob, Gudrun, and Franz Fazekas. "Magnetic resonance imaging of cerebral microbleeds." Current Opinion in Neurology 13, no. 1 (2000): 69–73. http://dx.doi.org/10.1097/00019052-200002000-00013.

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22

Tackley, George, Wilhelm Kuker, and Jacqueline Palace. "Magnetic resonance imaging in neuromyelitis optica." Multiple Sclerosis Journal 20, no. 9 (2014): 1153–64. http://dx.doi.org/10.1177/1352458514531087.

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Neuromyelitis optica (NMO), or Devic’s disease, is a rare demyelinating disorder of the central nervous system that has a predilection for the optic nerve and spinal cord. Magnetic resonance imaging (MRI) is required to diagnose NMO. Longitudinally extensive transverse myelitis is NMO’s imaging hallmark and the presence of a brain MRI that is not diagnostic of multiple sclerosis (MS) also remains part of the diagnostic criteria. It is increasingly recognised that MS and NMO brain imaging can, however, have similar appearances but differences do exist: hypothalamic, periaqueductal grey and area postrema lesions implicate NMO whilst cortical, U-fibre or Dawson’s finger lesions are suggestive of MS. The timing of image acquisition, age, ethnicity and aquaporin-4 antibody status are all likely to alter the findings at MRI. This review therefore aims to overview and update the reader on NMO imaging, to provide clinically relevant guidance for diagnosing NMO and differentiating it from MS in order to guide management, and to highlight recent research insights.

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23

Maravilla, Kenneth, and W. Sory. "Magnetic Resonance Imaging of Brain Tumors." Seminars in Neurology 6, no. 01 (1986): 33–42. http://dx.doi.org/10.1055/s-2008-1041445.

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24

Waluch, Victor. "Magnetic Resonance Imaging of Blood Flow." Seminars in Neurology 6, no. 01 (1986): 65–71. http://dx.doi.org/10.1055/s-2008-1041448.

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25

Rossi, Dennis, and A. Charney. "Magnetic Resonance Imaging of the Spine." Seminars in Neurology 6, no. 01 (1986): 84–93. http://dx.doi.org/10.1055/s-2008-1041451.

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26

Turner, Robert. "Magnetic resonance imaging of brain function." Annals of Neurology 35, no. 6 (1994): 637–38. http://dx.doi.org/10.1002/ana.410350602.

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27

Sethi, Kapil D. "Magnetic resonance imaging in huntington's disease." Movement Disorders 6, no. 2 (1991): 186. http://dx.doi.org/10.1002/mds.870060223.

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28

Filipek, Pauline A. "Quantitative magnetic resonance imaging in autism." Current Opinion in Neurology 8, no. 2 (1995): 134–38. http://dx.doi.org/10.1097/00019052-199504000-00009.

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29

Döhlinger, Susanne, Till-Karsten Hauser, Johannes Borkert, Andreas R. Luft, and Jörg B. Schulz. "Magnetic resonance imaging in spinocerebellar ataxias." Cerebellum 7, no. 2 (2008): 204–14. http://dx.doi.org/10.1007/s12311-008-0025-0.

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30

Baburaj, Remya, Rajeswaran Rangasami, and PS Rajakumar. "Magnetic resonance imaging and magnetic resonance spectroscopy in varicella zoster necrotizing encephalitis." Neurology India 66, no. 3 (2018): 836. http://dx.doi.org/10.4103/0028-3886.232343.

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31

Adler, Charles H., Robert A. Zimmerman, Peter J. Savino, Bruno Bernardi, Thomas M. Bosley, and Robert C. Sergott. "Hemifacial spasm: Evaluation by magnetic resonance imaging and magnetic resonance tomographic angiography." Annals of Neurology 32, no. 4 (1992): 502–6. http://dx.doi.org/10.1002/ana.410320404.

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32

Chiappa, Keith H., Rosamund A. Hill, Frank Huang-Hellinger, and Bruce G. Jenkins. "Photosensitive Epilepsy Studied by Functional Magnetic Resonance Imaging and Magnetic Resonance Spectroscopy." Epilepsia 40, s4 (1999): 3–7. http://dx.doi.org/10.1111/j.1528-1157.1999.tb00899.x.

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33

Hadley, D. M. "Cranial Magnetic Resonance Imaging." Journal of Neurology, Neurosurgery & Psychiatry 52, no. 1 (1989): 151–52. http://dx.doi.org/10.1136/jnnp.52.1.151-b.

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34

Peacock, Warwick J., and Judith A. Murovic. "Magnetic resonance imaging in myelocystoceles." Journal of Neurosurgery 70, no. 5 (1989): 804–7. http://dx.doi.org/10.3171/jns.1989.70.5.0804.

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✓ Two cases of terminal myelocystocele, a rare localized cystic dilatation of the caudal spinal central canal, are reviewed. Magnetic resonance imaging is a useful diagnostic tool for its evaluation. Terminal myelocystocele consists of the following: a myelocystocele which contains a “trumpet-like” flaring of the distal spinal cord central canal and thus is partially lined by ependymal tissue; a meningocele or dilated subarachnoid space located around the myelocystocele, which bulges into the subcutaneous region; and fibrolipomatous tissue surrounding the two cysts. This condition is usually associated with abnormalities of the vertebral column and sacrum as well as compression of the spinal cord and meningocele by a fibrous band. There is a possible relationship of the myelocystocele to teratogens such as loperamide HCl and retinoic acid, although the exact etiology of this entity is not known.

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35

Henriques, Rafael Neto, Sune Nørhøj Jespersen, and Noam Shemesh. "Correlation tensor magnetic resonance imaging." NeuroImage 211 (May 2020): 116605. http://dx.doi.org/10.1016/j.neuroimage.2020.116605.

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36

Kokmen, E., W. R. Marsh, and H. L. Baker. "Magnetic resonance imaging in syringomyelia." Neurosurgery 17, no. 2 (1985): 267???70. http://dx.doi.org/10.1097/00006123-198508000-00003.

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37

Lunsford, L. D. "Magnetic resonance imaging stereotactic thalamotomy." Neurosurgery 23, no. 3 (1988): 363???7. http://dx.doi.org/10.1097/00006123-198809000-00014.

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38

Kokmen, E., W. R. Marsh, and H. L. Baker. "Magnetic Resonance Imaging in Syringomyelia." Neurosurgery 17, no. 2 (1985): 267–70. http://dx.doi.org/10.1227/00006123-198508000-00003.

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Abstract Myelography and myelography assisted with computed tomography have been the most commonly used radiographic methods in the study of syringomyelia. These studies have never been entirely reliable in demonstrating the syrinx cavity and its relationship to other intracranial structures. During the 1st year of operation of the magnetic resonance imaging facility, the syringomyelic cavity was demonstrated in 15 patients who all had typical clinical signs and symptoms associated with syringomyelia. Nine cases were syringomyelia with Chiari malformation. One case showed additional hydrocephalus. Four cases were idiopathic, and 1 case was remotely posttraumatic. Magnetic resonance imaging, although it is in its infancy, already promises to be the most important radiographic technique for syringomyelia because it provides an anatomically truthful visualization of the sagittal plane of the cervical cord and can demonstrate the syrinx cavity and its relationship with the cerebellar tonsils, the 4th ventricle, and other related structures.

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39

Gulani, Vikas, and Pia C. Sundgren. "Diffusion Tensor Magnetic Resonance Imaging." Journal of Neuro-Ophthalmology 26, no. 1 (2006): 51–60. http://dx.doi.org/10.1097/01.wno.0000205978.86281.3e.

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40

Mukherji, Suresh K., Thomas L. Chenevert, and Mauricio Castillo. "Diffusion-Weighted Magnetic Resonance Imaging." Journal of Neuro-Ophthalmology 22, no. 2 (2002): 118–22. http://dx.doi.org/10.1097/00041327-200206000-00013.

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41

Biousse, Val??rie. "Magnetic Resonance Imaging in Stroke." Journal of Neuro-Ophthalmology 25, no. 1 (2005): 58. http://dx.doi.org/10.1097/00041327-200503000-00019.

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42

Hoggard, Nigel. "Magnetic Resonance Imaging in Stroke." Acta Neurochirurgica 148, no. 8 (2006): 922. http://dx.doi.org/10.1007/s00701-006-0779-4.

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43

Komiyama, Masaki, Akira Hakuba, Yuichi Inoue, et al. "Magnetic resonance imaging: Lumbosacral lipoma." Surgical Neurology 28, no. 4 (1987): 259–64. http://dx.doi.org/10.1016/0090-3019(87)90303-x.

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44

Dobkin, Bruce H. "Book Review: Functional Magnetic Resonance Imaging." Neurorehabilitation and Neural Repair 19, no. 1 (2005): 62–63. http://dx.doi.org/10.1177/1545968304273864.

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45

Roach, E. Steve, Thomas Smith, Charles V. Terry, Anthony R. Riela, and D. Wayne Laster. "Magnetic Resonance Imaging in Pediatric Neurologic Disorders." Journal of Child Neurology 2, no. 2 (1987): 111–16. http://dx.doi.org/10.1177/088307388700200206.

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46

Voss, Henning U., Jonathan P. Dyke, Karsten Tabelow, Nicholas D. Schiff, and Douglas J. Ballon. "Magnetic resonance advection imaging of cerebrovascular pulse dynamics." Journal of Cerebral Blood Flow & Metabolism 37, no. 4 (2016): 1223–35. http://dx.doi.org/10.1177/0271678x16651449.

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We analyze the pulsatile signal component of dynamic echo planar imaging data from the brain by modeling the dependence between local temporal and spatial signal variability. The resulting magnetic resonance advection imaging maps depict the location of major arteries. Color direction maps allow for visualization of the direction of blood vessels. The potential significance of magnetic resonance advection imaging maps is demonstrated on a functional magnetic resonance imaging data set of 19 healthy subjects. A comparison with the here introduced pulse coherence maps, in which the echo planar imaging signal is correlated with a cardiac pulse signal, shows that the magnetic resonance advection imaging approach results in a better spatial definition without the need for a pulse reference. In addition, it is shown that magnetic resonance advection imaging velocities can be estimates of pulse wave velocities if certain requirements are met, which are specified. Although for this application magnetic resonance advection imaging velocities are not quantitative estimates of pulse wave velocities, they clearly depict local pulsatile dynamics. Magnetic resonance advection imaging can be applied to existing dynamic echo planar imaging data sets with sufficient spatiotemporal resolution. It is discussed whether magnetic resonance advection imaging might have the potential to evolve into a biomarker for the health of the cerebrovascular system.

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47

FUKUDA, Aya, Luciano de Souza QUEIROZ, and Fabiano REIS. "Gliosarcomas: magnetic resonance imaging findings." Arquivos de Neuro-Psiquiatria 78, no. 2 (2020): 112–20. http://dx.doi.org/10.1590/0004-282x20190158.

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Abstract Background: Central nervous system (CNS) gliosarcoma (GSM) is a rare primary neoplasm characterized by the presence of glial and sarcomatous components. Objective: In this report, we describe the clinical and neuroimaging aspects of three cases of GSM and correlate these aspects with pathological findings. We also provide a brief review of relevant literature. Methods: Three patients were evaluated with magnetic resonance imaging (MRI), and biopsies confirmed the diagnosis of primary GSM, without previous radiotherapy. Results: The analysis of conventional sequences (T1, T1 after contrast injection, T2, Fluid attenuation inversion recovery, SWI and DWI/ADC map) and advanced (proton 1H MR spectroscopy and perfusion) revealed an irregular, necrotic aspect of the lesion, peritumoral edema/infiltration and isointensity of the solid component on a T2-weighted image. These features were associated with irregular and peripheral contrast enhancement, lipid and lactate peaks, increased choline and creatine levels in proton spectroscopy, increased relative cerebral blood volume (rCBV) in perfusion, multifocality and drop metastasis in one of the cases. Conclusion: These findings are discussed in relation to the general characteristics of GSM reported in the literature.

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48

Pelz, David M., Stephen J. Karlik, Allan J. Fox, and Fernando Viñuela. "Magnetic Resonance Imaging in Down's Syndrome." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 13, S4 (1986): 566–69. http://dx.doi.org/10.1017/s0317167100037318.

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Abstract:100% of brains of Down's adults over age 40 will show Alzheimer-type neuropathologic changes in the frontal and temporal lobes. In an attempt to image these lesions, magnetic resonance imaging (MRI) was performed in seven patients with Down's syndrome, ranging in age from 17 to 45 years, using a resistive unit operating at 0.15 Tesla. All scans were within normal limits except for one 45 year-old patient with severe left temporal lobe atrophy. No areas of abnormal signal were seen in the frontal or temporal lobes and the white matter lesions commonly seen in elderly demented subjects were not visualized in this group. We conclude that these white matter lesions are likely coincidental and not causally related to Alzheimer's changes. The pathologic process leading to the formation and development of Alzheimer's changes in the brains of Down's adults may not be visible on magnetic resonance images.

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49

Webb, Megan E., Farnaz Amoozegar, and Ashley D. Harris. "Magnetic Resonance Imaging in Pediatric Migraine." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 46, no. 6 (2019): 653–65. http://dx.doi.org/10.1017/cjn.2019.243.

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ABSTRACT:This literature review provides an overview of the research using magnetic resonance imaging (MRI) in pediatric migraine and compares findings with the adult migraine literature. A literature search using PubMed was conducted using all relevant sources up to February 2019. Using MRI methods to categorize and explain pediatric migraine in comparison with adult migraine is important, in order to recognize and appreciate the differences between the two entities, both clinically and physiologically. We aim to demonstrate the differences and similarities between pediatric and adult migraine using data from white matter and gray matter structural studies, cerebral perfusion, metabolites, and functional MRI (fMRI) studies, including task-based and resting-state blood oxygen level-dependent studies. By doing this we identify areas that need further research, as well as possible areas where intervention could alter outcomes.

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50

Baird, Alison E., and Steven Warach. "Magnetic Resonance Imaging of Acute Stroke." Journal of Cerebral Blood Flow & Metabolism 18, no. 6 (1998): 583–609. http://dx.doi.org/10.1097/00004647-199806000-00001.

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In the investigation of ischemic stroke, conventional structural magnetic resonance (MR) techniques (e.g., T1-weighted imaging, T2-weighted imaging, and proton density-weighted imaging) are valuable for the assessment of infarct extent and location beyond the first 12 to 24 hours after onset, and can be combined with MR angiography to noninvasively assess the intracranial and extracranial vasculature. However, during the critical first 6 to 12 hours, the probable period of greatest therapeutic opportunity, these methods do not adequately assess the extent and severity of ischemia. Recent developments in functional MR imaging are showing great promise for the detection of developing focal cerebral ischemic lesions within the first hours. These include (1) diffusion-weighted imaging, which provides physiologic information about the self-diffusion of water, thereby detecting one of the first elements in the pathophysiologic cascade leading to ischemic injury; and (2) perfusion imaging. The detection of acute intraparenchymal hemorrhagic stroke by susceptibility weighted MR has also been reported. In combination with MR angiography, these methods may allow the detection of the site, extent, mechanism, and tissue viability of acute stroke lesions in one imaging study. Imaging of cerebral metabolites with MR spectroscopy along with diffusion-weighted imaging and perfusion imaging may also provide new insights into ischemic stroke pathophysiology. In light of these advances in structural and functional MR, their potential uses in the study of the cerebral ischemic pathophysiology and in clinical practice are described, along with their advantages and limitations.

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