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Books on the topic 'Neuroni'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 March 2023

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1

Craighero, Laila. Neuroni specchio: [vedere è fare]. Bologna: Il mulino, 2010.

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2

Gobbi, Sandro. L'azione efficace: Quanti e neuroni in scena. Roma: Armando, 2011.

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3

Pali. Kumpalibre: Capsula e Nucleo : due neuroni a piede libero. Milano: Kowalski, 2003.

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4

La mente incorporata: La lezione di J. Kim sino ai neuroni specchio. Roma: Aracne, 2012.

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5

Dai neuroni alle parole: Come l'accesso al linguaggio ha riconfigurato l'esperienza sensibile. Milano: Mimesis, 2010.

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6

Gerstner, Wulfram. Spiking neuron models: Single neurons, populations, plasticity. Cambridge, U.K: Cambridge University Press, 2002.

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7

Plasticity in nerve cell function. Oxford: Clarendon Press, 1998.

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8

Andrew, Bulloch, ed. The Cellular basis of neuronal plasticity: Physiology, morphology, and biochemistry of molluscan neurons. Manchester: Manchester University Press, 1989.

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9

Bazterra, Lilia García. Hola neurona. Quito, Ecuador: Libresa, 2005.

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10

Noradrenergic neurons. Cambridge [England]: Cambridge University Press, 1990.

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11

Fillenz, Marianne. Noradrenergic neurons. Cambridge [England]: Cambridge University Press, 1990.

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12

James, Hollenbeck Peter, Bamburg James R, and American Society for Cell Biology., eds. Neurons: Methods and applications for the cell biologist. San Diego: Academic Press, 2003.

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13

Taylor, John Gerald. Coupled Oscillating Neurons. London: Springer London, 1992.

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14

David, Oliver. Motor neurone disease. 2nd ed. London: Royal College of General Practitioners, 1994.

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15

Colombo, Bruno, ed. The Musical Neurons. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-08132-3.

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16

Taylor, John Gerald, and C. L. T. Mannion, eds. Coupled Oscillating Neurons. London: Springer London, 1992. http://dx.doi.org/10.1007/978-1-4471-1965-4.

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17

1948-, Hunter Maggie, ed. Motor neurone disease. London: Routledge, 1998.

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18

Colecchia, Nicola. I Neuroni Di Monna Lisa. Independently Published, 2017.

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19

Colecchia, Nicola. Cosa Sono I Neuroni Artificiali: Campi Di Applicazione Delle Reti Neurali. Independently Published, 2018.

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20

Nostoi-Ritorni: Cinema, comunicazione, neuroni specchio. Lungavilla (PV) [i.e. Pavia, Italy]: Altravista, 2014.

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21

Neuroni Specchio: Basi Biologiche e Comprensione Del Funzionamento Dei Neuroni Specchio, Dell'empatia e Del Comportamento Sociale. Independently Published, 2022.

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22

Arnold, Nick. Neuroni, ipofisi, meningi: E altri cervellotici elementi. Salani Editore, 2002.

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23

Gerstner, Wulfram, and Werner M. Kistler. Spiking Neuron Models: Single Neurons, Populations, Plasticity. Cambridge University Press, 2002.

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24

Gerstner, Wulfram, and Werner M. Kistler. Spiking Neuron Models: Single Neurons, Populations, Plasticity. Cambridge University Press, 2012.

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25

Montgomery, Erwin B. Controlling the Flow of Electrical Charges. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190259600.003.0004.

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In depolarization to effect neuronal activations,, electrical charges are delivered to the neuron to affect the electrical potential across the neuronal membrane to subsequently affect voltage-gated ionic conductance channels. The orientation of the field of electrical charges to the neuronal membrane is critical. Electrical charges flow from the negative contact to the positive contact. The negative electrostatic charge “pushes” negative charges onto the outer surface of the neuron, which results in depolarization of the neuronal membrane. Neurons near the positive contact will not have negative electrical charges deposited on the outer surface, will not be depolarized, and thus, are not activated. Likewise, neurons whose membranes are oriented parallel to the lines of electrical forces that move electrical charges will not receive the electrical charges and, consequently, will not be activated. The electronics of the DBS systems are designed to control the electrostatic forces so as to control the activations of the nervous system to generate benefit and avoid adverse effects.
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26

Sada, Nagisa, and Tsuyoshi Inoue. Lactate Dehydrogenase. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0029.

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Glucose is transported into neurons and used as an energy source. It is also transported into astrocytes, a type of glial cell, and converted to lactate, which is then released to neurons and used as another energy source. The latter is called the astrocyte-neuron lactate shuttle. Although the lactate shuttle is a metabolic pathway, it also plays important roles in neuronal activities and brain functions. We recently reported that this metabolic pathway is involved in the antiepileptic effects of the ketogenic diet. Lactate dehydrogenase (LDH) is a metabolic enzyme that mediates the lactate shuttle, and its inhibition hyperpolarizes neurons and suppresses seizures. This enzyme is also a molecular target of stiripentol, a clinically used antiepileptic drug for Dravet syndrome. This review provides an overview of electrical regulation by the astrocyte-neuron lactate shuttle, and then introduces LDH as a metabolic target against epilepsy.
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27

Moore, John W., and Ann E. Stuart. Neurons in Action 2: Tutorials and Simulations Using NEURON. Oxford University Press, Incorporated, 2017.

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28

Moore, John W., John G. Nicholls, Ann E. Stuart, Robert A. Martin, and Paul A. Fuchs. From Neuron to Brain/ Neurons in Action Version 2. Sinauer Associates, Inc., 2011.

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29

Neurons In Action 2: Tutorials and Simulations using NEURON. Sinauer Associates, Inc., 2007.

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30

Neurotic Turn: Inter-Disciplinary Correspondences on Neurosis. ReadHowYouWant.com, Limited, 2017.

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31

Nicholls, John G., A. Robert Martin, Bruce G. Wallace, and Paul A. Fuchs. From Neuron to Brain & Neurons in Action (bundle with CD ROM). 4th ed. Sinauer Associates, 1999.

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32

Bradbury, Elizabeth J., and Nicholas D. James. Mapping of neurotrophin receptors on adult sensory neurons. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0022.

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The paper discussed in this chapter describes the first mapping of neurotrophin receptors in adult sensory neurons. Neurotrophins and their receptors were a particularly hot topic at the time, but the primary focus of interest had been in their role in development. In this paper, McMahon and colleagues characterized both mRNA and protein expression of the recently discovered trk receptors on defined populations of adult sensory neurons, correlating trk expression with other primary afferent projection neuron properties such as cell size and neuronal function. Furthermore, by showing clear correlations between the expression of different trk receptors and the physical and functional properties of defined primary afferent projections, the authors provided key evidence suggesting that nerve growth factor and neurotrophin-3 acted on functionally distinct populations of adult sensory neurons. This paper provided the basis for subsequent research on neurotrophin signalling and function in both the healthy and the diseased nervous system.
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33

Anderson, James A. An Engineer’s Introduction to Neuroscience. Oxford University Press, 2018. http://dx.doi.org/10.1093/acprof:oso/9780199357789.003.0006.

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When building something, it is essential to know the hardware. This chapter contains key things to know about the active components of the brain: nerve cells (aka neurons). Neurons have severe performance limitations. Problems include high energy consumption, mechanical and physiological sensitivity, unreliability, limited connectivity, and difficulty in wiring neurons together. Neurons are at least a million times slower to “compute” than a modern electronic device. This slow speed cannot be avoided because the neuron has to deal with high electrical capacity and resistance and slow conduction times to move information from neuron to neuron. A specialization called the action potential serves as a long-distance communications mechanism. However, the neuron also has major virtues including the ability to integrate, communicate, and process information from multiple sources, and it acts like a tiny electrochemical analog computer.
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34

Schünemann, Wolf J., and Marianne Kneuer, eds. E-Government und Netzpolitik im europäischen Vergleich. Nomos Verlagsgesellschaft mbH & Co. KG, 2019. http://dx.doi.org/10.5771/9783845291918.

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How are e-government and Internet policy organised and developed in different countries? This comprehensively revised volume answers this question and addresses the latest developments within the amended framework of digitalisation, such as cybersecurity, data protection, open government and e-democracy, among others. With contributions by Ana Azurmendi, Christoph Bieber, Jérôme Brugger, Emiliana De Blasio, Robert Dewar, Myriam Dunn Cavelty, Marianne Fraefel, Annette Knaut, Marianne Kneuer, Stine Marg, Véronique Millim, Manuel Misgeld, Matt Poelmans, Alessia, C. Neuroni, Simon P. Rinas, Patrick Ruestchmann, Ulrich Sarcinelli, Wolf J. Schünemann, Welf Schröter, Michele Sorice, Stefan Steiger, Sebastian Stier, Sophie Valdenaire-Ratto and Maria A. Wimmer.
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35

Thakore, Nimish, and Erik P. Pioro. Types of Motor Neuron Diseases. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0022.

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Disorders of lower motor neurons (LMNs, or anterior horn cells) and upper motor neurons (UMNs), jointly termed motor neuron disorders (MNDs), are diverse and numerous. The prototypical MND, namely amyotrophic lateral sclerosis (ALS), a relentlessly progressive lethal disorder of adults, is the subject of another section and will not be discussed further here. Other MNDs include spinal muscular atrophy (SMA), of which there are four types: Kennedy’s disease, Brown-Violetto-Van Laere, and Fazio-Londe syndromes, lower motor neuron disorders as part of neurodegenerations and secondary motor neuron disease as part of malignancy, radiation and infection.
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36

Nakajima, Kazuyuki, and Shinichi Kohsaka. Interaction of Microglia with Neurons and Astrocytes Under Lesioned Neuronal Conditions. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199794591.003.0053.

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This is a digitally enhanced text. Readers can also see the coverage of this topic area in the second edition of Neuroglia. The second edition of Neuroglia was first published digitally in Oxford Scholarship Online and the bibliographic details provided, if cited, will direct people to that version of the text. Readers can also see the coverage of this topic area in the ...
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37

Gerstner, Wulfram, Werner M. Kistler, Richard Naud, and Liam Paninski. Neuronal Dynamics: From Single Neurons to Networks and Models of Cognition. Cambridge University Press, 2014.

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38

Gerstner, Wulfram, Werner M. Kistler, Richard Naud, and Liam Paninski. Neuronal Dynamics: From Single Neurons to Networks and Models of Cognition. Cambridge University Press, 2014.

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39

Neuronal Dynamics: From Single Neurons to Networks and Models of Cognition. Cambridge University Press, 2014.

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40

Seniuk, Nadine Anne *. Neuronal destruction by MPTP: neurochemical and behavioural compensation by surviving neurons. 1989.

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41

Pamela J., M.d. Shaw (Editor) and Michael J. Strong (Editor), eds. Motor Neuron Disorders (Blue Books of Practical Neurology). Butterworth-Heinemann, 2003.

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42

Jef ferys, John G. R. Cortical activity: single cell, cell assemblages, and networks. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0004.

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This chapter describes how the activity of neurons produces electrical potentials that can be recorded at the levels of single cells, small groups of neurons, and larger neuronal networks. It outlines how the movement of ions across neuronal membranes produces action potentials and synaptic potentials. It considers how the spatial arrangement of specific ion channels on the neuronal surface can produce potentials that can be recorded from the extracellular space. Finally, it outlines how the layered cellular structure of the neocortex can result in summation of signals from many neurons to be large enough to record through the scalp as evoked potentials or the electroencephalogram.
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43

Hepoxilins and neuronal repair: Effects on SCG neurons after in vitro injury. Ottawa: National Library of Canada, 2000.

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44

Levitan, Irwin B., and Leonard K. Kaczmarek. The Neuron. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199773893.001.0001.

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The Fourth Edition of The Neuron provides a comprehensive first course in the cell and molecular biology of nerve cells. It begins with properties of the many newly discovered ion channels that have emerged through mapping of the genome and which shape the way a single neuron generates varied patterns of electrical activity. It also covers the molecular mechanisms that convert electrical activity into the secretion of neurotransmitter hormones at synaptic junctions between neurons. It discusses the biochemical pathways that are linked to the action of neurotransmitters and that can alter the cellular properties of neurons or sensory cells that transduce information from the outside world into the electrical code used by neurons, and the rapidly expanding knowledge of the molecular factors that induce an undifferentiated cell to become a neuron, and then guide it to form appropriate synaptic connections with its partners. Also addressed is the role of ongoing experience and activity in shaping these connections, and the mechanisms thought to underlie the phenomena of learning and memory.
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45

1952-, Barnes Michael P., and Johnson Garth R. 1945-, eds. Upper motor neurone syndrome and spasticity: Clinical management and neurophysiology. New York, NY: Cambridge University Press, 2001.

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46

(Editor), Michael P. Barnes, and Garth R. Johnson (Editor), eds. Upper Motor Neurone Syndrome and Spasticity: Clinical Management and Neurophysiology. Cambridge University Press, 2001.

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47

Mason, Peggy. Cells of the Nervous System. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0002.

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The nervous system is made up of neurons and glia that derive from neuroectoderm. Since neurons are terminally differentiated and do not divide, primary intracranial tumors do not arise from mature neurons. Tumors outside the nervous system may metastasize inside the brain or may release a substance that negatively affects brain function, termed paraneoplastic disease. Neurons receive information through synaptic inputs onto dendrites and soma and send information to other cells via a synaptic terminal. Most neurons send information to faraway locations and for this, an axon that connects the soma to synaptic terminals is required. Glial cells wrap axons in myelin, which speeds up information transfer. Axonal transport is necessary to maintain neuronal function and health across the long distances separating synaptic terminals and somata. A common mechanism of neurodegeneration arises from impairments in axonal transport that lead to protein aggregation and neuronal death.
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48

Hawver, David Burke. Selective avoidance and fasciculation in the formation of specific connections between "aplysia" neurons "in vitro". [New York], 1992.

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49

Osborne, Neville N. Biochemistry of Characterised Neurons. Elsevier Science & Technology Books, 2013.

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50

Neuron. Cambridge, Mass: Cell Press, 1988.

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