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Bogolepova, Irina N., Marina V. Krotenkova, Rodion N. Konovalov, Pavel A. Agapov, Irina G. Malofeeva, and Alexander T. Bikmeev. "Neuroplasticity, music, and human brain." Annals of Clinical and Experimental Neurology 18, no. 1 (2024): 72–78. http://dx.doi.org/10.54101/acen.2024.1.8.
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Introduction. Studying the influence of music on the human brain is one of the key topics in neuroscience as it allows extending our understanding of brain neuroplasticity.
 This study aimed to investigate structural brain organization in professional musicians.
 Materials and methods. We investigated 27 brains (i.e. 54 hemispheres) of male musicians, female musicians, male non-musicians, and female non-musicians by magnetic resonance imaging. All study participants were aged 20 to 30 years and did not have any mental or neurological disorders. Gray matter volume and cortex thickness in different cortical structures of the right and left hemispheres were measured.
 Results. We found major changes in the brain structure in professional musicians (both male and female) vs. non-musicians. We found differences in the macroscopic structure of the triangular region in the Broca’s motor speech area in musicians’ brain. Increases in gray matter volume in the brain of musicians and its individual cortical structures were shown in the superior temporal region, Broca’s motor speech area, hippocampus, superior parietal lobule, and other structures. We found increased thickness of cortical structures in musicians vs. non-musicians.
 Conclusions. Practicing music regularly was shown to change structural brain organization; we found significant increases in gray matter volume and cortex thickness in various cortical structures in the right and left brain hemispheres of musicians vs. non-musicians.
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Rachel, Yun. "Understanding Neuroplasticity and the Brain's Potential for Change." International Journal of Healthcare Sciences 10, no. 1 (2022): 263–71. https://doi.org/10.5281/zenodo.7043253.
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<strong>Abstract</strong><strong>:</strong> Neuroplasticity is a relatively new concept about the brain’s ability to rewire itself in certain areas. The general idea was developed in the 20th century, and it has become an intriguing field of study due to the superhuman-like feats of the brain that allows people with missing parts of the brain, impaired neurological functions, and certain mental disorders to live with more function. Neuroplasticity occurs in both animals and humans, as both react similarly to the environment, and results can be compared to reach a deeper understanding about how stress, which is a consequence of internal and external pressures, affects neuroplasticity. Other external stimuli, however, such as music, can improve the rate of neuroplasticity in people or undo damage in the nervous system. On the other hand, neuroplasticity does also have its downsides, which must be carefully avoided to prevent any issues related to mentality. In other words, while neuroplasticity can cure people of disorders, it may also be the exact reason why it developed in the first place. This curious phenomenon explores the true capabilities of the brain. This paper will describe the effect neuroplasticity has on people and the ways people can affect their rates of neuroplasticity. <strong>Keywords:</strong> neuroplasticity, neuroscience, growth mindset, fixed mindset, nervous system, depression, mental disorder, brain, digital media, gene expression. <strong>Title:</strong> Understanding Neuroplasticity and the Brain’s Potential for Change <strong>Author:</strong> Rachel Yun <strong>International Journal of Healthcare Sciences</strong> <strong>ISSN 2348-5728 (Online)</strong> <strong>Vol. 10, Issue 1, April 2022 - September 2022</strong> <strong>Page No: 263-271</strong> <strong>Research Publish Journals</strong> <strong>Website: www.researchpublish.com</strong> <strong>Published Date: 02-September-2022</strong> <strong>DOI: https://doi.org/10.5281/zenodo.7043253</strong> <strong>Paper Download Link (Source)</strong> <strong>https://www.researchpublish.com/papers/understanding-neuroplasticity-and-the-brains-potential-for-change</strong>
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Spytska, Liana. "The Impact of Physical Activity on Brain Neuroplasticity, Cognitive Functions and Motor Skills." OBM Neurobiology 08, no. 02 (2024): 1–10. http://dx.doi.org/10.21926/obm.neurobiol.2402219.
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The research aims to study the mechanisms and factors contributing to brain neuroplasticity. To achieve this goal, the following methods were used: analysis and synthesis, hermeneutic method, psychological testing, and comparative and generalization methods. The research results revealed the nature of the concept of brain neuroplasticity and types of neuroplasticity, analyzed the process of redistribution of brain functions, determined the role of compensatory plasticity, revealed methods of studying brain neuroplasticity, investigated the influence of brain processes on the course of learning, memory development, awareness, concentration, speech; identified factors that can affect brain neuroplasticity revealed the role of genetic factors, analyzed stimulation and rehabilitation methods to promote neuroplasticity. The findings may aid in developing novel rehabilitation techniques, specifically for stroke patients, by utilizing the brain’s compensatory abilities through physical activity, pharmacological interventions, and stimulation techniques. The practical significance of the research is determined by the current disclosure of the features of brain neuroplasticity to understand its ability to reorganize the sensory and perceptual systems.
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Palm, Ulrich, Moussa A. Chalah, and Samar S. Ayache. "Brain Stimulation and Neuroplasticity." Brain Sciences 11, no. 7 (2021): 873. http://dx.doi.org/10.3390/brainsci11070873.
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Electrical or magnetic stimulation methods for brain or nerve modulation have been widely known for centuries, beginning with the Atlantic torpedo fish for the treatment of headaches in ancient Greece, followed by Luigi Galvani’s experiments with frog legs in baroque Italy, and leading to the interventional use of brain stimulation methods across Europe in the 19th century [...]
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Drigas, Athanasios, and Angeliki Sideraki. "Brain Neuroplasticity Leveraging Virtual Reality and Brain–Computer Interface Technologies." Sensors 24, no. 17 (2024): 5725. http://dx.doi.org/10.3390/s24175725.
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This study explores neuroplasticity through the use of virtual reality (VR) and brain–computer interfaces (BCIs). Neuroplasticity is the brain’s ability to reorganize itself by forming new neural connections in response to learning, experience, and injury. VR offers a controlled environment to manipulate sensory inputs, while BCIs facilitate real-time monitoring and modulation of neural activity. By combining VR and BCI, researchers can stimulate specific brain regions, trigger neurochemical changes, and influence cognitive functions such as memory, perception, and motor skills. Key findings indicate that VR and BCI interventions are promising for rehabilitation therapies, treatment of phobias and anxiety disorders, and cognitive enhancement. Personalized VR experiences, adapted based on BCI feedback, enhance the efficacy of these interventions. This study underscores the potential for integrating VR and BCI technologies to understand and harness neuroplasticity for cognitive and therapeutic applications. The researchers utilized the PRISMA (Preferred Reporting Items for Systematic Reviews and Meta-Analyses) method to conduct a comprehensive and systematic review of the existing literature on neuroplasticity, VR, and BCI. This involved identifying relevant studies through database searches, screening for eligibility, and assessing the quality of the included studies. Data extraction focused on the effects of VR and BCI on neuroplasticity and cognitive functions. The PRISMA method ensured a rigorous and transparent approach to synthesizing evidence, allowing the researchers to draw robust conclusions about the potential of VR and BCI technologies in promoting neuroplasticity and cognitive enhancement.
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Rodrigues, Ana Carolina, Maurício Alves Loureiro, and Paulo Caramelli. "Musical training, neuroplasticity and cognition." Dementia & Neuropsychologia 4, no. 4 (2010): 277–86. http://dx.doi.org/10.1590/s1980-57642010dn40400005.
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Abstract The influence of music on the human brain has been recently investigated in numerous studies. Several investigations have shown that structural and functional cerebral neuroplastic processes emerge as a result of long-term musical training, which in turn may produce cognitive differences between musicians and non-musicians. Musicians can be considered ideal cases for studies on brain adaptation, due to their unique and intensive training experiences. This article presents a review of recent findings showing positive effects of musical training on non-musical cognitive abilities, which probably reflect plastic changes in brains of musicians.
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Peluffo, Lucas. "Crucial Philosophical Implications of Neuroplasticity." Journal of NeuroPhilosophy 3, no. 1 (2024): 96–110. https://doi.org/10.5281/zenodo.10875054.
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This article briefly reviews neuroplasticity's basic terms and mechanisms and then emphasizes three crucial philosophical implications. (1) Considering the relationship of epistemology with the brain, the main organ of human intelligence is now proven to have the capacity to reorganize itself structurally. (2) Neuroplasticity has startled metaphysicians by embodying a mechanism that appears to challenge any strict—non-interactive—interpretations of the controversial term mind-body dualism. (3) Within morality and ethics, many neuroscientific studies performed on Buddhist meditators of Indic meditative traditions have linked positive neuroplasticity with empathy, compassion, and loving-kindness, indicating that these qualities can be developed consciously and suggesting that they may be intrinsic to human beings.
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Murciano-Brea, Julia, Martin Garcia-Montes, Stefano Geuna, and Celia Herrera-Rincon. "Gut Microbiota and Neuroplasticity." Cells 10, no. 8 (2021): 2084. http://dx.doi.org/10.3390/cells10082084.
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The accumulating evidence linking bacteria in the gut and neurons in the brain (the microbiota–gut–brain axis) has led to a paradigm shift in the neurosciences. Understanding the neurobiological mechanisms supporting the relevance of actions mediated by the gut microbiota for brain physiology and neuronal functioning is a key research area. In this review, we discuss the literature showing how the microbiota is emerging as a key regulator of the brain’s function and behavior, as increasing amounts of evidence on the importance of the bidirectional communication between the intestinal bacteria and the brain have accumulated. Based on recent discoveries, we suggest that the interaction between diet and the gut microbiota, which might ultimately affect the brain, represents an unprecedented stimulus for conducting new research that links food and mood. We also review the limited work in the clinical arena to date, and we propose novel approaches for deciphering the gut microbiota–brain axis and, eventually, for manipulating this relationship to boost mental wellness.
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Cramer, S. C., M. Sur, B. H. Dobkin, et al. "Harnessing neuroplasticity for clinical applications." Brain 134, no. 6 (2011): 1591–609. http://dx.doi.org/10.1093/brain/awr039.
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Wolpaw, J. R. "Harnessing neuroplasticity for clinical applications." Brain 135, no. 4 (2012): e215-e215. http://dx.doi.org/10.1093/brain/aws017.
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Németh, Viktor. "Neuroplasticity." Belügyi Szemle 69, no. 6. ksz. (2021): 124–27. http://dx.doi.org/10.38146/bsz.spec.2021.6.8.
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As editor Bruce Tidor sets it in the preface of the book, published in the volume of the MIT Essential Knowledge series: ‘Synthesizing specialized subject matter for non-specialists and engaging critical topics through fundamentals, each of these compact volumes offers readers a point of access to complex ideas.’ (Costandi, 2016). In this book of the series Moheb Costandi provides the reader with a celar and coherent picture about neuroplasticity and neurogenesis . Not just at the level of theories and research results, but also regarding various stages of practical application. It is equally applicable for average people in areas of everyday life- adult education, lifelong learning, and mental training, too. Costandi’s book is decidedly good background material for Anders Hansen’s practical book ‘The Real Happy Pill: Power Up Your Brain by Moving Your Body’ (Németh, 2020).
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Farzan, Vahedifard, and Sadeghniiat Haghighi Atieh. "The role of Neuroradiology in Neuroplasticity: New advancements." World Journal of Advanced Research and Reviews 14, no. 2 (2022): 156–60. https://doi.org/10.5281/zenodo.7186250.
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Neuroplasticity, the brain’s capacity to adapt to internal and external environmental changes, occurs physiologically throughout growth and in reaction to damage. Many MRI studies of neuroplasticity have shown strong evidence that the brain changes quickly and extensively when people have new experiences. · In this paper, we review the most advancement in the role of neuroradiology in neuroplasticity and using biomarkers. o Detecting neuroplasticity in global brain circuits in vivo is critical for understanding various processes such as memory, learning, and injury healing. o MRI-biomarkers can be used to check for corticospinal integrity and how well motor resources are used. White matter neuroplasticity is studied via MRI. It has been used to study structural changes using diffusion tensor imaging (DTI) o The ultrafast fMRI (ufMRI) technique allows for high spatiotemporal sensitivity and resolution in dispersed brain circuits to detect fMRI signals more connected with the underlying neural dynamics. White matter hemodynamics may change over time, explaining functional neuroplasticity in this tissue.
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Moya, Roni. "Brain nutrition, aging and neuroplasticity." Free Radical Biology and Medicine 120 (May 2018): S146. http://dx.doi.org/10.1016/j.freeradbiomed.2018.04.481.
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Farzan Vahedifard and Atieh Sadeghniiat Haghighi. "The role of Neuroradiology in Neuroplasticity: New advancements." World Journal of Advanced Research and Reviews 14, no. 2 (2022): 156–60. http://dx.doi.org/10.30574/wjarr.2022.14.2.0420.
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Neuroplasticity, the brain’s capacity to adapt to internal and external environmental changes, occurs physiologically throughout growth and in reaction to damage. Many MRI studies of neuroplasticity have shown strong evidence that the brain changes quickly and extensively when people have new experiences. · In this paper, we review the most advancement in the role of neuroradiology in neuroplasticity and using biomarkers. o Detecting neuroplasticity in global brain circuits in vivo is critical for understanding various processes such as memory, learning, and injury healing. o MRI-biomarkers can be used to check for corticospinal integrity and how well motor resources are used. White matter neuroplasticity is studied via MRI. It has been used to study structural changes using diffusion tensor imaging (DTI) o The ultrafast fMRI (ufMRI) technique allows for high spatiotemporal sensitivity and resolution in dispersed brain circuits to detect fMRI signals more connected with the underlying neural dynamics. White matter hemodynamics may change over time, explaining functional neuroplasticity in this tissue.
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Marzola, Patrícia, Thayza Melzer, Eloisa Pavesi, Joana Gil-Mohapel, and Patricia S. Brocardo. "Exploring the Role of Neuroplasticity in Development, Aging, and Neurodegeneration." Brain Sciences 13, no. 12 (2023): 1610. http://dx.doi.org/10.3390/brainsci13121610.
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Neuroplasticity refers to the ability of the brain to reorganize and modify its neural connections in response to environmental stimuli, experience, learning, injury, and disease processes. It encompasses a range of mechanisms, including changes in synaptic strength and connectivity, the formation of new synapses, alterations in the structure and function of neurons, and the generation of new neurons. Neuroplasticity plays a crucial role in developing and maintaining brain function, including learning and memory, as well as in recovery from brain injury and adaptation to environmental changes. In this review, we explore the vast potential of neuroplasticity in various aspects of brain function across the lifespan and in the context of disease. Changes in the aging brain and the significance of neuroplasticity in maintaining cognitive function later in life will also be reviewed. Finally, we will discuss common mechanisms associated with age-related neurodegenerative processes (including protein aggregation and accumulation, mitochondrial dysfunction, oxidative stress, and neuroinflammation) and how these processes can be mitigated, at least partially, by non-invasive and non-pharmacologic lifestyle interventions aimed at promoting and harnessing neuroplasticity.
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Amaranggani, Anindhita P., Iklima Ritmiani, Rema Vara Indry Dubu, Rima Hariati, and Zararah Yusri Nasution. "Examining the Effects of Exercise on Neuroplasticity and Depression Disorder: Implications for Depression Treatment." Pena Medika : Jurnal Kesehatan 13, no. 2 (2023): 396–406. https://doi.org/10.31941/pmjk.v13i2.3688.
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Depression is a serious public health issue with a high risk of mortality and limited access to pharmacological and psychotherapeutic treatments. Exercise serves as an effective and readily accessible alternative intervention in addressing depression by influencing brain neuroplasticity and various aspects of mental health. This article is an integrative review that analyzes the potential of brain neuroplasticity in reducing depression symptoms through exercise, exploring literature related to depression, exercise, and neuroplasticity to generate implications for depression management. The article was retrieved via Google Scholar, and a synthesis of 38 relevant articles was conducted. The results indicate that depression affects brain structure and function, which are also associated with neuroplasticity. Exercise has the potential to stimulate brain neuroplasticity, particularly in areas affected by depression, such as the hippocampus and prefrontal cortex. This potential makes exercise an effective yet cost-effective and easily accessible alternative for the prevention and treatment of depression, especially mild to moderate depression.
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Cramer, S. C. "Reply: Harnessing neuroplasticity for clinical applications." Brain 135, no. 4 (2012): e216-e216. http://dx.doi.org/10.1093/brain/aws018.
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Chaieb, L., A. Antal, G. G. Ambrus, and W. Paulus. "Brain-derived neurotrophic factor: its impact upon neuroplasticity and neuroplasticity inducing transcranial brain stimulation protocols." neurogenetics 15, no. 1 (2014): 1–11. http://dx.doi.org/10.1007/s10048-014-0393-1.
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Dita, Maria, and Libi Bubuioc. "Neuroplasticity - the metamorphosis of the human brain." Vector European, no. 1 (April 2024): 188–92. http://dx.doi.org/10.52507/2345-1106.2024-1.35.
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The brain's ability to reorganize its neurons to form new neural connections based on life experience and learning is known as neuroplasticity. This process is continuous and allows us to learn new things throughout our lives. In addition, neuroplasticity not only helps us adapt to life and develop culturally and spiritually, but also represents a chance for recovery for neurological conditions that may affect the senses, movements or cognitive functions. Therefore, neuroplasticity allows neurons in the brain to compensate temporarily or permanently by forming new brain connections, fully or partially replacing functions affected by injury or disease.
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Shamay-Tsoory, Simone. "Brains that fire together wire together: interbrain plasticity underlies interaction-based learning." Project Repository Journal 12, no. 1 (2022): 82–85. http://dx.doi.org/10.54050/prj1218392.
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Brains that fire together wire together: interbrain plasticity underlies interaction-based learning Even though social interactions are significant determinants of learning, the field of neuroplasticity doesn’t take this into account. Instead, it is deeply rooted in probing changes occurring in synapses, brain structures, and networks within an individual brain. The ERC funded INTERPLASTIC project, however, will propose a new approach that synthesises disparate findings on network neuroplasticity and mechanisms of social interactions. It will test whether the facilitation effect of social interactions on learning can be explained by interbrain plasticity (short- and long-term experience-dependent changes in interbrain coupling).
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Kadykov, A. S., N. V. Shakhparonova, A. V. Belopasova, and J. V. Prjanikov. "A NEUROPLASTICITY AND FUNCTIONAL RESTORATION AFTER STROKE." Physical and rehabilitation medicine, medical rehabilitation 1, no. 2 (2019): 32–36. http://dx.doi.org/10.36425/2658-6843-19184.
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The article discusses the history of brain neuroplasticity, its effect on the restoration of functions after a stroke. Various mechanisms of neuroplasticity are considered: functions of reorganization, neurogenesis, the effect on neuroplasticity of training, the use of various rehabilitation techniques, and drug therapy.
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Singh, Shailendra. "Neuroplasticity and Rehabilitation: Harnessing Brain Plasticity for Stroke Recovery and Functional Improvement." Universal Research Reports 11, no. 3 (2024): 50–56. http://dx.doi.org/10.36676/urr.v11.i3.1287.
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This paper provides a comprehensive review of the current understanding of neuroplasticity and its application in stroke rehabilitation. Stroke remains a leading cause of disability worldwide, often resulting in motor, sensory, and cognitive impairments. Neuroplasticity, the brain's ability to reorganize and adapt in response to experience and injury, offers promising avenues for recovery. This review discusses key principles of neuroplasticity and explores various rehabilitation strategies aimed at harnessing its potential for stroke recovery. Topics covered include early intervention, task-specific training, intensity and repetition, constraint-induced movement therapy, multimodal approaches, environmental enrichment, and neurostimulation techniques. Additionally, the paper discusses emerging research directions and challenges in optimizing neuroplasticity-based rehabilitation approaches. Understanding the role of neuroplasticity in stroke recovery can inform the development of more effective rehabilitation interventions and improve outcomes for individuals affected by stroke.
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Peregud, D. I., V. Yu Baronets, N. N. Terebilina, and N. V. Gulyaeva. "Role of BDNF in neuroplasticity associated with alcohol dependence." Биохимия 88, no. 3 (2023): 491–507. http://dx.doi.org/10.31857/s0320972523030090.
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Chronic alcohol consumption is characterized by disturbances of neuroplasticity. Brain-derived neurotrophic factor (BDNF) may mechanistically participate in this process. Here we aimed to review actual experimental and clinical data related to BDNF involvement in neuroplasticity in the context of alcohol dependence. As shown in experiments on the rodents alcohol consumption is accompanied brain region-specific changes of BDNF expression and by structural and behavioral impairments. BDNF reverses aberrant neuroplasticity during alcohol intoxication. According to clinical data indices characterized BDNF demonstrate close relationship with consequences of alcohol dependence. Polymorphism rs6265 within BDNF gene interacts with macrostructural changes in the brain, while peripheral BDNF concentration may reflect anxiety, depression and cognitive decline. Thus, BDNF is involved in mechanisms of alcohol-related aberrant neuroplasticity, while polymorphisms within BDNF gene and peripheral BDNF concentration may be biomarkers, diagnostic or prognostic factors in clinics of alcoholism.
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Goldberg, Hagar. "Growing Brains, Nurturing Minds—Neuroscience as an Educational Tool to Support Students’ Development as Life-Long Learners." Brain Sciences 12, no. 12 (2022): 1622. http://dx.doi.org/10.3390/brainsci12121622.
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Compared to other primates, humans are late bloomers, with exceptionally long childhood and adolescence. The extensive developmental period of humans is thought to facilitate the learning processes required for the growth and maturation of the complex human brain. During the first two and a half decades of life, the human brain is a construction site, and learning processes direct its shaping through experience-dependent neuroplasticity. Formal and informal learning, which generates long-term and accessible knowledge, is mediated by neuroplasticity to create adaptive structural and functional changes in brain networks. Since experience-dependent neuroplasticity is at full force during school years, it holds a tremendous educational opportunity. In order to fulfill this developmental and learning potential, educational practices should be human-brain-friendly and “ride” the neuroplasticity wave. Neuroscience can inform educators about the natural learning mechanisms of the brain to support student learning. This review takes a neuroscientific lens to explore central concepts in education (e.g., mindset, motivation, meaning-making, and attention) and suggests two methods of using neuroscience as an educational tool: teaching students about their brain (content level) and considering the neuro-mechanisms of learning in educational design (design level).
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de Sousa Fernandes, Matheus Santos, Tayrine Figueira Ordônio, Gabriela Carvalho Jurema Santos, et al. "Effects of Physical Exercise on Neuroplasticity and Brain Function: A Systematic Review in Human and Animal Studies." Neural Plasticity 2020 (December 14, 2020): 1–21. http://dx.doi.org/10.1155/2020/8856621.
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Background. Physical exercise (PE) has been associated with increase neuroplasticity, neurotrophic factors, and improvements in brain function. Objective. To evaluate the effects of different PE protocols on neuroplasticity components and brain function in a human and animal model. Methods. We conducted a systematic review process from November 2019 to January 2020 of the following databases: PubMed, ScienceDirect, SciELO, LILACS, and Scopus. A keyword combination referring to PE and neuroplasticity was included as part of a more thorough search process. From an initial number of 20,782 original articles, after reading the titles and abstracts, twenty-one original articles were included. Two investigators evaluated the abstract, the data of the study, the design, the sample size, the participant characteristics, and the PE protocol. Results. PE increases neuroplasticity via neurotrophic factors (BDNF, GDNF, and NGF) and receptor (TrkB and P75NTR) production providing improvements in neuroplasticity, and cognitive function (learning and memory) in human and animal models. Conclusion. PE was effective for increasing the production of neurotrophic factors, cell growth, and proliferation, as well as for improving brain functionality.
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Classen, J. "Focal hand dystonia - a disorder of neuroplasticity?" Brain 126, no. 12 (2003): 2571–72. http://dx.doi.org/10.1093/brain/awg290.
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Daneshmandi, Hassan, Mostafa Payandeh, and Zaher Mohammad Ashour. "Brain Neuroplasticity Effects on the Occurrence of Anterior Cruciate Ligament Injury and the Effect of this Injury on Brain Function and Structure: A Systematic Review." Journal of Rehabilitation 23, no. 2 (2022): 162–85. http://dx.doi.org/10.32598/rj.23.2.3377.1.
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Objective: The present study aimed to investigate the effect of brain neuroplasticity on the incidence of anterior cruciate ligament (ACL) injury and on brain function and structure before and after ligament reconstruction and after a period of rehabilitation exercises. Materials & Methods: In this review study, a search was conducted in PubMed, Scopus, Web of Science, Google Scholar, ScienceDirect, MedLine, Pedro, CINAHL, SPORTDiscus, and Cochrane Databases as well as national databases on related studies published from 1970 to 2021 using keywords in Persian and English related to the research topic. Results: The initial search yielded 65 articles. Based on the inclusion and exclusion criteria, 24 articles were selected for review of which 5 articles prospectively examined the effect of brain neuroplasticity on the incidence of ACL injury. Their results showed that the brains of people with ACL injury was different from the uninjured people, especially in the motor-sensory part of the cerebral cortex and cerebellum, which caused errors during movement planning of these persons. Ten articles examined the effect of ACL injury before reconstruction on brain function and structure and reported that changes occur in the level of motor-sensory cortex of the brain at least two weeks after the injury; after one year, these structural and functional changes were widely increased in injured people compared to healthy people. These studies also showed that the ligament dysfunction and the damage to mechanical receptors cause the reorganization of the central nervous system. In injured people, the control activity of motor-visual areas and their need for visual feedback have increased. Seven articles examined these changes after ligament reconstruction and showed that the brain neuroplasticity or functional and structural changes resulting from the injury not only did not return to normal conditions, but also increased after a while despite the reconstruction. Two articles examined these changes after a period of rehabilitation exercises and showed that functional and Conclusion: The changes in the brain after ACL injury not only persist after ligament reconstruction, but also increase after reconstruction. The common rehabilitation exercises whose main focus is not on eliminating these functional and structural changes in the brain cannot overdrive this negative neuroplasticity after injury which is one of the important causes of secondary injury and subsequent complications. In developing exercises to prevent ACL injury and for rehabilitation, it is better to use the new principles of motor learning and exercises related to visual feedback along with conventional exercises to overdrive negative neuroplasticity created in the brain and create positive neuroplasticity to support ACL.
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Palm, Ulrich, Samar S. Ayache, and Moussa A. Chalah. "Brain Stimulation and Neuroplasticity—Series II." Brain Sciences 12, no. 8 (2022): 1084. http://dx.doi.org/10.3390/brainsci12081084.
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Cramer, Steven C., and Jeff D. Riley. "Neuroplasticity and brain repair after stroke." Current Opinion in Neurology 21, no. 1 (2008): 76–82. http://dx.doi.org/10.1097/wco.0b013e3282f36cb6.
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Jasey, Neil, and Irene Ward. "Neuroplasticity in Brain Injury: Maximizing Recovery." Current Physical Medicine and Rehabilitation Reports 7, no. 4 (2019): 333–40. http://dx.doi.org/10.1007/s40141-019-00242-7.
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Davidson, Richard. "Meditation and Neuroplasticity: Training Your Brain." EXPLORE 1, no. 5 (2005): 380–88. http://dx.doi.org/10.1016/j.explore.2005.06.013.
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BULBOACA, Adriana Elena, Ioana STANESCU, Cristina NICULA, and Angelo BULBOACA. "Neuroplasticity pathophysiological mechanisms underlying neuro-optometric rehabilitation in ischemic stroke – a brief review." Balneo and PRM Research Journal 12, Vol.12, no.1 (2021): 16–20. http://dx.doi.org/10.12680/balneo.2021.412.
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Neuroplasticity is an essential phenomenon underlying on neurorehabilitation process, by which the brain can remodel the dysfunction consequent to a lesion. Ischemic brain lesions are the most frequent brain lesions often associated with visual function disability. Experimental and clinical studies established that visual function disability can impede the neurorehabilitation therapy efficiency. Neuro-optometric therapy has been proved to significantly improve the patient outcome after brain lesions. The pathophysiological mechanisms underlying this process are yet to be deciphered. Current knowledge regarding the pathophysiological mechanisms involved in ischemic lesions and neuroplasticity as a reparation process offers real support to a more efficient neurorehabilitation therapy that can contribute to the improvement of life quality in stroke patients. Keywords: neuroplasticity, neuro-optometric rehabilitation, ischemic stroke,
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Romanchuk, N. "Brain Homo sapiens XXI Century: Neurophysiological, Neuroeconomic and Neurosocial Decision-making Mechanisms." Bulletin of Science and Practice 7, no. 9 (2021): 228–70. http://dx.doi.org/10.33619/2414-2948/70/23.
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Systemic neurocognitive and neuroeconomic decision-making is becoming one of the greatest quality life problems of Homo sapiens in the 21st century. Human decision-making at neurocognitive, neurosocial and neuroeconomic levels has been investigated. Neuroplasticity management methods allow timely prevention of factors that reduce neuroplasticity, preserve factors of positive influence on neuroplasticity, and most importantly, timely use of combined methods of preserving and developing neuroplasticity of the human brain in practical healthcare (Romanchuk N. P., Moscow, 2016, Science and Education in the 21st Century). Modern science views man, humanity and the biosphere as a single system, with growing demographic, food and medical problems. The main engine of human longevity is when the microbiological memory of the microbiota remains stable, and the diet of functional (healthy) dietary nutrition and the structure of healthy biomicrobiota function almost unchanged. Healthy biomicrobiota provides stability of functioning and timely reprogramming in the hypothalamic-pituitary-adrenal axis, in the work of bidirectional intestinal-brain connections of the “cognitive and visceral brain”. The role of cortisol, estrogen, testosterone and oxytocin has been established - in age-related changes in brain functions, and in the process of cognitive and socio-emotional aging. Human brains are biological, biophysical, neurophysiological and medico-social paradigms of information exchange. Modern communications are multilevel, multi-paradigm and interdisciplinary models of information exchange. The introduction of copyright developments in the last decade has made it possible to form a system of algorithms and tools for managing neuroplasticity. The new competencies of psychoneuroimmunoendocrinology and psychoneuroimmunology play a strategic role in interdisciplinary science and interdisciplinary planning and decision-making. Qualified mind — creates and improves the cognitive potential of the brain. The “neurointerface stone” of H. sapiens self-esteem for self-actualization and self-realization of personality is self-discovery, self-development, self-control, self-realization. Brain H. sapiens working in the mode of genius (talent, creativity) requires the creation and maintenance of modern neurocommunications between the new cortex and the hippocampus (memory library, memory winchester), the formation of new structural-functional neurocommunications in brain H. sapiens that occur continuously throughout life from birth to super-longevity and have creative advantages in the era of modern neuroscience and neuromarketing.
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Mamczur, Maciej, Michał Szczepański, Dominik Feret, et al. "Neuroplasticity in Depressive Disorders: The Role of BDNF in Linking Pharmacotherapy and Physical Activity." Quality in Sport 36 (December 14, 2024): 56457. https://doi.org/10.12775/qs.2024.36.56457.
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Introduction: Depressive disorders are among the most prevalent mental health conditions globally, affecting millions of people. Despite extensive research, the exact pathophysiology of depression remains unclear. Recent evidence suggests that neuroplasticity, particularly the role of brain-derived neurotrophic factor (BDNF), plays a crucial role in both the onset and treatment of depressive disorders. Aim of Study: This review explores the relationship between neuroplasticity and depression, focusing on BDNF as a central biomarker and potential therapeutic target. It also examines how antidepressant therapies and physical activity influence neuroplasticity and BDNF levels, offering insights into novel approaches for treating depressive disorders. Material and Methods: A literature review was conducted using PubMed and Google Scholar databases, focusing on peer-reviewed articles with the keywords: "neuroplasticity", "BDNF", "depression", "antidepressants" and "physical activity". Conclusions: Neuroplasticity, particularly through BDNF signaling, plays a critical role in the pathogenesis and treatment of depression. Elevated BDNF levels promote synaptic plasticity, neurogenesis, and recovery of brain function. Antidepressant treatments enhance neuroplasticity by increasing BDNF levels. Antidepressants and physical activity both influence neuroplastic processes, with BDNF serving as a key mediator.
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Peregud, Danil I., Valeria Yu Baronets, Natalia N. Terebilina, and Natalia V. Gulyaeva. "Role of BDNF in Neuroplasticity Associated with Alcohol Dependence." Biochemistry (Moscow) 88, no. 3 (2023): 404–16. http://dx.doi.org/10.1134/s0006297923030094.
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Abstract Chronic alcohol consumption is characterized by disturbances of neuroplasticity. Brain-derived neurotrophic factor (BDNF) is believed to be critically involved in this process. Here we aimed to review actual experimental and clinical data related to BDNF participation in neuroplasticity in the context of alcohol dependence. As has been shown in experiments with rodents, alcohol consumption is accompanied by the brain region-specific changes of BDNF expression and by structural and behavioral impairments. BDNF reverses aberrant neuroplasticity observed during alcohol intoxication. According to the clinical data parameters associated with BDNF demonstrate close correlation with neuroplastic changes accompanying alcohol dependence. In particular, the rs6265 polymorphism within the BDNF gene is associated with macrostructural changes in the brain, while peripheral BDNF concentration may be associated with anxiety, depression, and cognitive impairment. Thus, BDNF is involved in the mechanisms of alcohol-induced changes of neuroplasticity, and polymorphisms within the BDNF gene and peripheral BDNF concentration may serve as biomarkers, diagnostic or prognostic factors in treatment of alcohol abuse.
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Duan, Menghan. "Mechanisms of Epilepsy and Efficacy of Neuroplasticity-Based Treatment." Transactions on Environment, Energy and Earth Sciences 3 (November 26, 2024): 294–99. https://doi.org/10.62051/pyrxm188.
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Epilepsy is a neurological disorder characterized by the development of trauma to the nervous system and recurrent seizures. Neuroplasticity, the ability of the nervous system to reorganize its structure and function in response to trauma, plays a crucial role in the development and treatment of epilepsy. Several recent studies have mentioned the potential of treating epilepsy based on neuroplasticity, such as deep brain stimulation (DBS), neurofeedback and non-invasive brain stimulation techniques. While conventional treatments focus primarily on seizure control, neuroplasticity interventions aim to alter neural circuits, reduce seizure frequency and severity, and may ameliorate the effects caused by the sequelae of epilepsy. There are already a number of therapies for epilepsy that incorporate neuroplasticity, which provides a basis for subsequent research. This paper explores the pathogenesis of epilepsy with a focus on the role of neuroplasticity in the onset and progression of epilepsy. However, further research is needed to optimize these approaches and fully understand their therapeutic potential.
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Qiao, Chenye, Zongjian Liu, and Shuyan Qie. "The Implications of Microglial Regulation in Neuroplasticity-Dependent Stroke Recovery." Biomolecules 13, no. 3 (2023): 571. http://dx.doi.org/10.3390/biom13030571.
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Stroke causes varying degrees of neurological deficits, leading to corresponding dysfunctions. There are different therapeutic principles for each stage of pathological development. Neuroprotection is the main treatment in the acute phase, and functional recovery becomes primary in the subacute and chronic phases. Neuroplasticity is considered the basis of functional restoration and neurological rehabilitation after stroke, including the remodeling of dendrites and dendritic spines, axonal sprouting, myelin regeneration, synapse shaping, and neurogenesis. Spatiotemporal development affects the spontaneous rewiring of neural circuits and brain networks. Microglia are resident immune cells in the brain that contribute to homeostasis under physiological conditions. Microglia are activated immediately after stroke, and phenotypic polarization changes and phagocytic function are crucial for regulating focal and global brain inflammation and neurological recovery. We have previously shown that the development of neuroplasticity is spatiotemporally consistent with microglial activation, suggesting that microglia may have a profound impact on neuroplasticity after stroke and may be a key therapeutic target for post-stroke rehabilitation. In this review, we explore the impact of neuroplasticity on post-stroke restoration as well as the functions and mechanisms of microglial activation, polarization, and phagocytosis. This is followed by a summary of microglia-targeted rehabilitative interventions that influence neuroplasticity and promote stroke recovery.
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Taupin, Philippe. "Adult neurogenesis and neuroplasticity." Restorative Neurology and Neuroscience 24, no. 1 (2006): 9–15. https://doi.org/10.3233/rnn-2006-00325.
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After cerebral strokes and traumatic brain injuries (TBIs), there is a striking amount of neurological recovery in the following months and years, despite often-permanent structural damage. Though the mechanisms underlying such recovery are not fully understood, properties of plasticity of the central nervous system (CNS), such as the reorganization of the pre-existing network and axonal sprouting have been implicated in the recovery. With the recent evidences that neurogenesis occurs in the adult brain, and neural stem cells (NSCs) reside in the adult CNS, the involvement of newly generated neuronal cells in the recovery following injury to the CNS remains to be established. Neurogenesis is increased bilaterally in the dentate gyrus (DG) and the subventicular zone (SVZ) after cerebral strokes and TBIs, and new neuronal cells are generated at the sites of injury, where they replace some of the degenerated nerve cells. Newly generated neuronal cells at the sites of injury may represent an attempt by the CNS to regenerate itself after injury, whereas the increased neurogenesis in the DG and SVZ would also contribute to the CNS plasticity. Thus, injury-induced neurogenesis may contribute to the recovery and plasticity of the CNS.
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Gupta, Steve. "Neuroplasticity and Recovery from Brain Injury: A Comprehensive Review of Current Research." International Journal of Science and Research (IJSR) 12, no. 8 (2023): 2059–65. http://dx.doi.org/10.21275/sr23802115456.
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Yaneva, Antonia, Kristina Kilova, and Teodora Dimcheva. "SUCCESSFUL COGNITIVE AGING - COGNITIVE RESERVE AND NEUROPLASTICITY." Teacher of the future 31, no. 4 (2019): 1005–8. http://dx.doi.org/10.35120/kij31041005y.
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As people age they tend to experience changes in cognitive function. Cognitive exercise can allow the brain to remain active and dynamic even at a later age. The promotion of successful cognitive aging is a topic of great importance and a challenge to public health considering the growth and the aging of the world population. This article discusses three concepts - the concept of successful cognitive aging, cognitive reserve and neuroplasticity, and their relationship to the overall cognitive functioning of the elderly. The concept of the cognitive reserve explains the discrepancy between the degree of brain damage and the way the individual responds. Cognitive reserve is based on current brain activity which is formed by the experiences and the activities throughout life. Cognitive reserve theory corresponds with the studies of brain plasticity in the elderly and the fact that cognitive interventions can be useful during aging. The concept of brain plasticity or neuroplasticity is the foundation of all brain exercises or games and relates to the changes in the neuronal organization that can lead to behavioral changes and the development of compensatory mechanisms in older people with cognitive dysfunction due to aging or brain pathology. The model of cognitive plasticity in elderly people proposed by Lövden argues that cognitive interventions are effective when there is a mismatch between the cognitive ability of the individual and the difficulty level of the cognitive task. According to the concepts presented in this article, successful cognitive aging can be achieved by the elderly. The brain can continue to adapt and develop new abilities throughout life. The ability of the brain to reorganize and create new roads is at the key of cognitive learning - an instrument that can be used by health professionals to complement and support the improvement of therapeutic approaches. Research has shown that systemic brain training can potentially lead to the improvement of a number of cognitive skills.
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Staneiu, Roxana-Maria. "Nurturing Neuroplasticity as an Enabler for Growth Mindset through Lifelong Learning and Knowledge Dynamics." Proceedings of the International Conference on Business Excellence 17, no. 1 (2023): 1264–74. http://dx.doi.org/10.2478/picbe-2023-0113.
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Abstract In a world where change and uncertainty prevail, one’s ability to take full responsibility for the development of their own brain renders itself as a propensity to increase neuroplasticity by engaging in mentally challenging endeavors to be broader stimulated. Being an in-built human ability, which enables the brain to reorganize pathways and create new synapses at all times in people’s lives by accumulating new information through experiences, neuroplasticity offers people the opportunity to rewire their behaviors by constantly learning to expose to new contexts and be creative in front of challenging and complex situations. Following an empirical analysis of the literature review covering neuroplasticity, growth mindset, lifelong learning and knowledge dynamics, the paper aims to revels the connection between these forces and understand the influence each other plays in stimulating the brain, increasing its capacity and ensuing an enhanced personal and professional development. The results showcase an organic connection between neuroplasticity, growth mindset, lifelong learning and knowledge dynamics which illustrated itself in the shape of a research diagram, emphasizing the interconnections and the prevalent role of knowledge transformations.
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Price, Amy. "Cognitive Rehabilitation Computer Brain Solutions." International Journal of User-Driven Healthcare 2, no. 2 (2012): 77–81. http://dx.doi.org/10.4018/ijudh.2012040111.
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Mild traumatic brain injury (MTBI) survivors claim advantage in retraining their brains with neuroplasticity based cognitive training after trauma. Significant growth in computer based cognitive rehabilitation is spurred on by positive research findings on neuroplasticity and advances in accessible computer technology. Drawbacks include limitations on the part of both patient and therapist in regards to time expenditure, cost of therapy, ease of use/learning curve, and the availability of long-term studies in regards to near and far transfer of training. MTBI patients may have sustained motor, visual, auditory, and chronic pain difficulties that complicate computer use. Benefits and barriers as perceived by patients and psychologists who are using the interventions for patient rehabilitation are critical. MTBI patient and therapist feedback concerning efficacy, usability accessibility, and satisfaction are needed to realize this form of rehabilitation.
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Coderre, Terence J., and Joel Katz. "What exactly is central to the role of central neuroplasticity in persistent pain?" Behavioral and Brain Sciences 20, no. 3 (1997): 483–86. http://dx.doi.org/10.1017/s0140525x97611498.
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The commentaries on our target article have raised important issues about central neuroplasticity and its role in persistent pain states. Some suggest that central neuroplasticity plays nothing more than a minor role in persistent pain, while others argue that persistent pain depends critically on peripheral inputs for its maintenance. Some stress that persistent pain relies to a large extent on changes in the brain and on centrifugal inputs from brain to spinal cord, whereas others argue that it depends on alterations in inhibitory as well as excitatory systems. We attempt to address each of the commentators' points, while defending our position that central neuroplasticity is critical to pathological persistent pain states.
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Noronha, Gustavo de. "O CÉREBRO E A NEUROPLASTICIDADE FUNCIONAL." Revista ft 29, no. 142 (2025): 45–46. https://doi.org/10.69849/revistaft/dt10202501281845.
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THE BRAIN AND FUNCTIONAL NEUROPLASTICITY ABSTRACT Neural plasticity, or neuroplasticity, is characterized by the ability of the nervous system to change its structure and function based on patterns of experience. This phenomenon can be evaluated and conceptualized both from a structural perspective that considers the synaptic configuration and from a functional perspective that takes into account behavior modification. The entirety of neuropsychological rehabilitation, as well as psychotherapies in general, is based on the belief that the human brain is a flexible and adaptable organ that can rebuild itself in response to new environmental demands or the restrictions induced by brain injuries. Considering this concept, how does functional neuroplasticity occur? Since the driven unit is no longer centered on neurons, but is conceived as an extensive network of synaptic connections between neuronal units, in addition to glial cells, it may change according to personal experience, i.e., the level of activity and the type of stimulus received. The human brain is made up of about 100 billion neurons. According to researchers, it is believed that neurogenesis, or the production of new neurons, is limited after birth. Given this concept, this research seeks to analyze the brain and its functional neuroplasticity. Whereas, triggered restitution is emphasized at the beginning and compensation acquires greater relevance over time. A developmental perspective is also useful, helping to identify the pathways in which self-actualization remains viable. The entire process of neuropsychological rehabilitation, and psychotherapy in general, is based on the belief that the human brain is a dynamic, adaptable organ, capable of self-acting in response to new environmental demands or functional limitations imposed by brain injury. This research analyzes concepts of injury mechanisms by examining the impact of functional recovery on neuropsychological rehabilitation from a dynamic perspective of the correlation between brain structure and function. The methodology used was qualitative through academic articles and bibliographical references. KEYWORDS: Neuroplasticity. Rehabilitation. Functional restoration. Presynaptic and postsynaptic neuron.
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Teplyashina, E. A., Y. K. Komleva, E. V. Lychkovskaya, A. S. Deikhina, and A. B. Salmina. "Regulation of neurogenesis and cerebral angiogenesis by cell protein proteolysis products." RUDN Journal of Medicine 25, no. 2 (2021): 114–26. http://dx.doi.org/10.22363/2313-0245-2021-25-2-114-126.
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Brain development is a unique process characterized by mechanisms defined as neuroplasticity (synaptogenesis, synapse elimination, neurogenesis, and cerebral angiogenesis). Numerous neurodevelopmental disorders brain damage, and aging are manifested by neurological deficits that are caused by aberrant neuroplasticity. The presence of stem and progenitor cells in neurogenic niches of the brain is responsible for the formation of new neurons capable of integrating into preexisting synaptic assemblies. The determining factors for the cells within the neurogenic niche are the activity of the vascular scaffold and the availability of active regulatory molecules that establish the optimal microenvironment. It has been found that regulated intramembrane proteolysis plays an important role in the control of neurogenesis in brain neurogenic niches. Molecules generated by the activity of specific proteases can stimulate or suppress the activity of neural stem and progenitor cells, their proliferation and differentiation, migration and integration of newly formed neurons into synaptic networks. Local neoangiogenesis supports the processes of neurogenesis in neurogenic niches, which is guaranteed by the multivalent action of peptides formed from transmembrane proteins. Identification of new molecules regulating the neuroplasticity (neurogenesis and angiogenesis). i. e. enzymes, substrates, and products of intramembrane proteolysis, will ensure the development of protocols for detecting the neuroplasticity markers and targets for efficient pharmacological modulation.
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Jena, Prasant Kumar, Tahereh Setayesh, Lili Sheng, Jacopo Di Lucente, Lee Way Jin, and Yu-Jui Yvonne Wan. "Intestinal Microbiota Remodeling Protects Mice from Western Diet-Induced Brain Inflammation and Cognitive Decline." Cells 11, no. 3 (2022): 504. http://dx.doi.org/10.3390/cells11030504.
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It has been shown that the Western diet (WD) induces systemic inflammation and cognitive decline. Moreover, probiotic supplementation and antibiotic treatment reduce diet-induced hepatic inflammation. The current study examines whether shaping the gut microbes by Bifidobacterium infantis (B. infantis) supplementation and antibiotic treatment reduce diet-induced brain inflammation and improve neuroplasticity. Furthermore, the significance of bile acid (BA) signaling in regulating brain inflammation was studied. Mice were fed a control diet (CD) or WD for seven months. B. infantis was supplemented to WD-fed mice to study brain inflammation, lipid, metabolomes, and neuroplasticity measured by long-term potentiation (LTP). Broad-spectrum coverage antibiotics and cholestyramine treatments were performed to study the impact of WD-associated gut microbes and BA in brain inflammation. Probiotic B. infantis supplementation inhibited diet-induced brain inflammation by reducing IL6, TNFα, and CD11b levels. B. infantis improved LTP and increased brain PSD95 and BDNF levels, which were reduced due to WD intake. Additionally, B. infantis reduced cecal cholesterol, brain ceramide and enhanced saturated fatty acids. Moreover, antibiotic treatment, as well as cholestyramine, diminished WD-induced brain inflammatory signaling. Our findings support the theory that intestinal microbiota remodeling by B. infantis reduces brain inflammation, activates BA receptor signaling, and improves neuroplasticity.
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Isel, Frédéric. "Neuroplasticity of second language vocabulary acquisition." Language, Interaction and Acquisition 12, no. 1 (2021): 54–81. http://dx.doi.org/10.1075/lia.20023.ise.
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Abstract The present article reviews a series of selected functional and structural magnetic resonance imaging (MRI) studies focusing on the neuroplasticity of second language vocabulary acquisition as a function of linguistic experience. A clear-cut picture emerging from the review is that brain changes induced by second language vocabulary acquisition are observed at both functional and structural levels. Importantly, second language experience is even able to shape brain structures in short-term training of a few weeks. The evidence that linguistic experience can sculpt the brain in late second language learners, and even solely after a short-term laboratory training, constitutes a strong argument against theoretical approaches postulating that environmental factors are relatively unimportant for language development. Rather, combined neuroimaging data lend support to the determining role of linguistic experience in linguistic knowledge emergence during second language acquisition, at least at the lexical level.
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Marzo, Aude, Jing Bai, and Satoru Otani. "Neuroplasticity Regulation by Noradrenaline in Mammalian Brain." Current Neuropharmacology 7, no. 4 (2009): 286–95. http://dx.doi.org/10.2174/157015909790031193.
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Kays, Jill L., Robin A. Hurley, and Katherine H. Taber. "The Dynamic Brain: Neuroplasticity and Mental Health." Journal of Neuropsychiatry and Clinical Neurosciences 24, no. 2 (2012): 118–24. http://dx.doi.org/10.1176/appi.neuropsych.12050109.
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Wu, Steve W., and Donald L. Gilbert. "Measuring neuroplasticity in children using brain stimulation." Developmental Medicine & Child Neurology 57, no. 6 (2015): 499. http://dx.doi.org/10.1111/dmcn.12716.
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