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Journal articles on the topic 'Neuroprotector'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Sutherland, Stephani. "Creatine as a neuroprotector." Drug Discovery Today 9, no. 18 (2004): 776. http://dx.doi.org/10.1016/s1359-6446(04)03243-x.

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Buneeva, Olga, Arthur Kopylov, Inga Kapitsa, Elena Ivanova, Victor Zgoda, and Alexei Medvedev. "The Effect of Neurotoxin MPTP and Neuroprotector Isatin on the Profile of Ubiquitinated Brain Mitochondrial Proteins." Cells 7, no. 8 (2018): 91. http://dx.doi.org/10.3390/cells7080091.

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Mitochondria are a crucial target for the actions of neurotoxins, causing symptoms of Parkinson’s disease in various experimental animal models, and also neuroprotectors. There is evidence that mitochondrial dysfunction induced by the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) influences functioning of the ubiquitin-proteasomal system (UPS) responsible for selective proteolytic degradation of proteins from various intracellular compartments (including mitochondria) and neuroprotective effects of certain anti-Parkisonian agents (monoamine oxidase inhibitors) may be associate

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3

Schonhoff, Aubrey M., and Ashley S. Harms. "Glial GLP1R: A novel neuroprotector?" Movement Disorders 33, no. 12 (2018): 1877. http://dx.doi.org/10.1002/mds.27547.

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4

Surov, Artem O., Andrei V. Churakov, Alexey N. Proshin, Xia-Lin Dai, Tongbu Lu, and German L. Perlovich. "Cocrystals of a 1,2,4-thiadiazole-based potent neuroprotector with gallic acid: solubility, thermodynamic stability relationships and formation pathways." Physical Chemistry Chemical Physics 20, no. 21 (2018): 14469–81. http://dx.doi.org/10.1039/c8cp02532k.

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5

Jiménez, Erika V., Jennifer Tovar, Oscar M. Mosquera, and Fernando Cardozo. "Actividad neuroprotectora de Solanum ovalifolium (Solanaceae) contra la toxicidad inducida por rotenona en Drosophila melanogaster." Revista Facultad de Ciencias Básicas 13, no. 1 (2017): 26–34. http://dx.doi.org/10.18359/rfcb.2751.

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Los objetivos de este trabajo fueron evidenciar la presencia de flavonoides en el extracto metanólico de Solanum ovalifolium por cromatografía líquida de alta eficiencia (HPLC-UV), analizar la actividad antioxidante por los métodos de 1,1-difenil-2-picrilhidrazilo (DPPH•) y ácido 2,2'-azinobis-(3-etilbenzotiazolina)-6-sulfónico (ABTS•+), cuantificar el glutatión (GSH), las unidades de enzima antioxidante superóxido dismutasa (SOD) y determinar el efecto neuroprotector contra la toxicidad inducida por rotenona (100 µM) con el modelo in vivo de Drosophila melanogaster mediante geotaxis negativa

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6

Cryan, Michael T., and Ashley E. Ross. "Subsecond detection of guanosine using fast-scan cyclic voltammetry." Analyst 144, no. 1 (2019): 249–57. http://dx.doi.org/10.1039/c8an01547c.

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7

Ramos Ramos, Victoria, Pablo Mesa Suárez, José Diego Santotoribio, María Ángela González García, and Antonio Muñoz Hoyos. "Efecto neuroprotector del sevoflurano en anestesia general." Medicina Clínica 148, no. 4 (2017): 158–60. http://dx.doi.org/10.1016/j.medcli.2016.10.039.

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8

Antonenko, Yuri N., Stepan S. Denisov, Denis N. Silachev, et al. "Mitofluorescein as mitochondria-targeted uncoupler and neuroprotector." Biochimica et Biophysica Acta (BBA) - Bioenergetics 1857 (August 2016): e117. http://dx.doi.org/10.1016/j.bbabio.2016.04.248.

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9

Tsiumpala, S. A., K. M. Starchevska, and V. I. Lushchak. "POTENTIAL USE OF SULFORAPHANE AS A NEUROPROTECTOR." Medical and Clinical Chemistry, no. 2 (August 4, 2021): 125–36. http://dx.doi.org/10.11603/mcch.2410-681x.2021.i2.12048.

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Introduction. Under normal conditions, oxidative stress and proinflammatory processes are tightly controlled. However, during neuroinflammation and overproduction of reactive oxygen species (ROS), homeostasis is disrupted, which may lead to development of Alzheimer’s disease, Parkinson’s disease and other neurodegenerative disorders. Inflammatory processes may result in neurodegenerative disorders. Sulforaphane is an isothiocyanate compound which has potential for treatment of neurodegenerative disorders. Its therapeutic potential is based on the ability to activate transcription of genes, th

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10

Gemiralda, Rika Mutiara, Marlaokta Marlaokta, and Marlaokta Marlaokta. "Efek Neuroprotektor Kunyit pada Pasien Alzheimer." Jurnal Ilmu Keperawatan Jiwa 2, no. 3 (2019): 171. http://dx.doi.org/10.32584/jikj.v2i3.431.

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Alzheimer merupakan penyakit neurogeneratif kronis progresif. Penyakit ini ditandai dengan gangguan fungsi luhur seperti bahasa, kalkulasi, kapasitas belajar dan sulit mengambil keputusan. ‘Pikun’ merupakan istilah yang digunakan masyarakat untuk menggambarkan penyakit ini. Kehilangan memori jangka pendek merupakan kekhasan dari penyakit ini yang menyebabkan penurunkan produktivitas. Alzheimer sering terjadi pada usia > 65 tahun dan juga ditemukan pada sekitar usia 40 tahun. Kunyit (Curcuma longa L.) merupakan tanaman yang berfungsi sebagai neuroprotektor karena dapat menghambat Protein Tau

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11

Melero Fernández de Mera, Raquel M., Eva García Martínez, Francisco José Fernández Gómez, et al. "¿Es la vieja minociclina un nuevo fármaco neuroprotector?" Revista de Neurología 47, no. 01 (2008): 31. http://dx.doi.org/10.33588/rn.4701.2008065.

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12

Yang, Ming-Hui, Yi-Ling Chen, Chi-Yu Lu, et al. "Is NAP an additional neuroprotector in Alzheimer's disease?" Biomarkers and Genomic Medicine 5, no. 3 (2013): 103–6. http://dx.doi.org/10.1016/j.bgm.2013.07.004.

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13

Picano, Eugenio, and Maria P. Abbracchio. "Adenosine, the imperfect endogenous anti-ischemic cardio-neuroprotector." Brain Research Bulletin 52, no. 2 (2000): 75–82. http://dx.doi.org/10.1016/s0361-9230(00)00249-5.

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14

López-Miranda González, Visitación, María Luisa Soto Montenegro, Gema Vera Pasamontes, Esperanza Herradón Pliego, Manuel Desco Menéndez, and Raquel Abalo Delgado. "Resveratrol: un polifenol neuroprotector de la dieta mediterránea." Revista de Neurología 54, no. 06 (2012): 349. http://dx.doi.org/10.33588/rn.5406.2011611.

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15

Wu, Heng, Ying Jin, Jianning Wei, Hong Jin, Di Sha, and Jang-Yen Wu. "Mode of action of taurine as a neuroprotector." Brain Research 1038, no. 2 (2005): 123–31. http://dx.doi.org/10.1016/j.brainres.2005.01.058.

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16

Jaya, Made Krisnaadi, and Nimade Oka Dwicandra. "EFFECTIVITY ANALYSIS OF NEUROPROTECTOR (VITAMIN B COMPLEX AND MECOBALAMIN) AS NEUROPATHIC PAIN SUPPORTIVE THERAPY IN ELDERLY WITH TYPE 2 DIABETES MELLITUS." Asian Journal of Pharmaceutical and Clinical Research 10, no. 12 (2017): 320. http://dx.doi.org/10.22159/ajpcr.2017.v10i12.21845.

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Objective: Neuroprotector (Vitamin B complex or mecobalamin)is often used as a supportive neuropathic pain therapy. The effectiveness of this drug remains controversial, especially in geriatrics with Type 2 diabetic neuropathy pains. The aim of this study was to compare the diabetes neuropathic pain reduction in elderly with and without neuroprotective supplementation.Method: The study was conducted by prospective cohort design. 132 agings were observed during 4 weeks at Neurology Polyclinic, Sanglah Public Hospital, Denpasar, Bali-Indonesia. Individuals undergoing the first-line neuropathic p

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17

Buneeva, O. A., O. V. Gnedenko, M. V. Medvedeva, A. S. Ivanov, and A. E. Medvedev. "Oxidative modification of glyceraldehyde-3-phosphate dehydrogenase influences its interaction with endogenous neuroprotector isatin." Biomeditsinskaya Khimiya 62, no. 2 (2016): 160–63. http://dx.doi.org/10.18097/pbmc20166202160.

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Glyceraldehyde-3-phosphate dehydrogenase (GAPDH), a classical glycolytic redox sensitive enzyme, exhibits various non-glycolytic functions, which are considered to be especially important for progression of various neurodegenerative diseases. GAPDH binds isatin (indole-dione-2,3), an endogenous indole often used as a parent component in numerous derivatives demonstrating diverse pharmacological (including neuroprotector) activities. In this study we have investigated binding of intact and mildly oxidized GAPDH to immobilized isatin, using an optical biosensor technique, employing surface plasm

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18

Chitu, Iulia, Ruxandra Tudosescu, Costin Leasu-Branet, and Liliana-Mary Voinea. "Citicoline – a neuroprotector with proven effects on glaucomatous disease." Romanian Journal of Ophthalmology 61, no. 3 (2017): 152–58. http://dx.doi.org/10.22336/rjo.2017.29.

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19

Abreu, Ana, Ana Duque, Carolina Paulino, et al. "Papel neuroprotector da hipotermia terapêutica pós paragem cardio-respiratória." Revista Brasileira de Terapia Intensiva 23, no. 4 (2011): 455–61. http://dx.doi.org/10.1590/s0103-507x2011000400010.

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20

Suero-García, Carlos, Lucia Martín-Banderas, and Mª Ángeles Holgado. "Efecto neuroprotector de los cannabinoides en las enfermedades neurodegenerativas." Ars Pharmaceutica (Internet) 56, no. 2 (2015): 77–87. http://dx.doi.org/10.4321/s2340-98942015000200002.

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21

Alessenko, A. V., S. O. Bachurin, S. V. Gurianova, Y. O. Karatasso, E. F. Shevtsova, and L. N. Shingarova. "Tumor necrosis factor-alpha - potential target for neuroprotector dimebon." Biomeditsinskaya Khimiya 62, no. 4 (2016): 418–25. http://dx.doi.org/10.18097/pbmc20166204418.

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Dimebon (Dimebolin) is an antihistamine drug which has been used in Russia since 1983. Recently Dimebolin has attracted renewed interest after being shown to have positive effects on persons suffering from Alzheimer's disease. Animal studies have shown that dimebon acts through multiple mechanisms, both blocking the action of neurotoxic beta-amyloid peptides and inhibiting L-type calcium channels, modulating the action of AMPA and NMDA glutamate receptors. Our experiments with cell culture L929 and mice have shown that dimebon may exert its neuroprotective effect by blocking cytotoxic signals

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22

Sandoval-Avila, S., N. F. Diaz, U. Gómez-Pinedo, et al. "Efecto neuroprotector de fitoquímicos en cultivo de neuronas dopaminérgicas." Neurología 34, no. 2 (2019): 114–24. http://dx.doi.org/10.1016/j.nrl.2016.04.018.

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23

López-Ogalla, Javier, Gonzalo Saiz, and Francisco E. Palomo. "Process Development of a Potent Neuroprotector Agent: Collismycin A." Organic Process Research & Development 17, no. 1 (2013): 120–26. http://dx.doi.org/10.1021/op3003129.

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24

Villanueva-García, Dina, Daniel Mota-Rojas, Agatha Miranda-Cortés, et al. "Neurobehavioral and neuroprotector effects of caffeine in animal models." Journal of Animal Behaviour and Biometeorology 8, no. 4 (2020): 298–307. http://dx.doi.org/10.31893/jabb.20039.

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25

Mangas, A., J. Yajeya, N. González, et al. "Overexpression of kynurenic acid in stroke: An endogenous neuroprotector?" Annals of Anatomy - Anatomischer Anzeiger 211 (May 2017): 33–38. http://dx.doi.org/10.1016/j.aanat.2017.01.002.

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26

Arwin, Leonardo, and Jihan Nur Pratiwi. "Peran Neuroprotektor Astaxanthin dalam Pencegahan Penyakit Alzheimer." Jurnal Ilmu Keperawatan Jiwa 3, no. 1 (2020): 47. http://dx.doi.org/10.32584/jikj.v3i1.469.

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Alzheimer merupakan penyakit neurodegeneratif yang terjadi secara bertahap dan progresif disebabkan oleh kematian sel neuron. Bertambahnya usia, cidera kepala traumatis, depresi, penyakit kardiovaskular dan serebrovaskular, usia orang tua yang lebih tinggi, merokok, riwayat keluarga demensia dapat meningkatkan risiko penyakit. Alzheimer tidak dapat disembuhkan, namun terdapat beberapa obat yang dapat mengatasi gejala simptomatis dari penyakit ini seperti inhibitor colinesterase dan N-metil D-aspartat (NMDA) parsial. Astaxanthin diketahui memiliki kandungan antioksidan dan antiinflamasi sepuluh

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27

Panthi, Sandesh, Hyung-Joo Chung, Junyang Jung, and Na Young Jeong. "Physiological Importance of Hydrogen Sulfide: Emerging Potent Neuroprotector and Neuromodulator." Oxidative Medicine and Cellular Longevity 2016 (2016): 1–11. http://dx.doi.org/10.1155/2016/9049782.

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Hydrogen sulfide (H2S) is an emerging neuromodulator that is considered to be a gasotransmitter similar to nitrogen oxide (NO) and carbon monoxide (CO). H2S exerts universal cytoprotective effects and acts as a defense mechanism in organisms ranging from bacteria to mammals. It is produced by the enzymes cystathionineβ-synthase (CBS), cystathionineϒ-lyase (CSE), 3-mercaptopyruvate sulfurtransferase (MST), and D-amino acid oxidase (DAO), which are also involved in tissue-specific biochemical pathways for H2S production in the human body. H2S exerts a wide range of pathological and physiological

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28

Ha, Kee-Yong, Young-Hoon Kim, Kee-Won Rhyu, and Soon-Eok Kwon. "Pregabalin as a neuroprotector after spinal cord injury in rats." European Spine Journal 17, no. 6 (2008): 864–72. http://dx.doi.org/10.1007/s00586-008-0653-6.

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29

Saidasheva, E. I. "NEURORETINOPROTECTION IN TREATMENT AND REHABILITATIONOF EYE DISEASES IN CHILDREN’S AGE." HERALD of North-Western State Medical University named after I.I. Mechnikov 9, no. 3 (2017): 24–31. http://dx.doi.org/10.17816/mechnikov20179324-31.

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Presented of own long-term experience and the generalized results of the researches devoted to use of domestic drugs of peptide structure in complex therapy of a number of diseases of an optic nerve and a retina at patients of children's age. Clinical efficiency of a retinalamin as retinoprotector and cortexin as neuroprotector in treatment and rehabilitation the disabling diseases shown: atrophy of the optic nerve, retinopathy of prematurity and degenerative myopia.

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30

Brazill, Jennifer M., Chong Li, Yi Zhu, and R. Grace Zhai. "NMNAT: It’s an NAD + synthase… It’s a chaperone… It’s a neuroprotector." Current Opinion in Genetics & Development 44 (June 2017): 156–62. http://dx.doi.org/10.1016/j.gde.2017.03.014.

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31

Klein, Christian, Christine Patte-Mensah, Omar Taleb, et al. "The neuroprotector kynurenic acid increases neuronal cell survival through neprilysin induction." Neuropharmacology 70 (July 2013): 254–60. http://dx.doi.org/10.1016/j.neuropharm.2013.02.006.

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32

Fokin, Andrey, Peter Schreiner, Anika Merz, et al. "Synthetic Routes to Aminotriamantanes, Topological Analogues of the Neuroprotector Memantine®." Synthesis 2009, no. 06 (2009): 909–12. http://dx.doi.org/10.1055/s-0028-1087979.

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33

Valcarce, M., A. García-Alix, C. Morales, A. Martín-Ancel, and J. Quero. "NEUROPROTECTOR EFFECTS OF MgSO4 USED IN PIGLETS AFTER HYPOXIC-ISCHEMIC INSULT." Pediatric Research 44, no. 3 (1998): 420. http://dx.doi.org/10.1203/00006450-199809000-00041.

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34

BACHURIN, S., E. BUKATINA, N. LERMONTOVA, et al. "Antihistamine Agent Dimebon As a Novel Neuroprotector and a Cognition Enhancer." Annals of the New York Academy of Sciences 939, no. 1 (2006): 425–35. http://dx.doi.org/10.1111/j.1749-6632.2001.tb03654.x.

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35

Lin, Yu-Fen, Ming-Hui Yang, Yuan-Han Yang, et al. "Activity-dependent neuroprotector homeobox protein level in Alzheimer's disease in Taiwanese." Genomic Medicine, Biomarkers, and Health Sciences 4, no. 1-2 (2012): 48–50. http://dx.doi.org/10.1016/j.gmbhs.2012.04.004.

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36

Marques, Eduardo Peil, and Angela T. S. Wyse. "Creatine as a Neuroprotector: an Actor that Can Play Many Parts." Neurotoxicity Research 36, no. 2 (2019): 411–23. http://dx.doi.org/10.1007/s12640-019-00053-7.

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37

Roncati, Luca, Beatrice Lusenti, Federica Pellati, and Lorenzo Corsi. "Micronized / ultramicronized palmitoylethanolamide (PEA) as natural neuroprotector against COVID-19 inflammation." Prostaglandins & Other Lipid Mediators 154 (June 2021): 106540. http://dx.doi.org/10.1016/j.prostaglandins.2021.106540.

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38

Saidasheva, Elvira I. "THE ROLE OF NEURORETINOPROTECTION IN THE PEDIATRIC OPHTHALMOLOGICAL PRACTICE." Russian Pediatric Ophthalmology 12, no. 4 (2017): 204–9. http://dx.doi.org/10.18821/1993-1859-2017-12-4-204-209.

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This article presents the author’s many year experience with the use of domestic medications of peptide structure for the combined treatment of various diseases of the optic nerve and the retina in the children. In addition it summarizes the results of the relevant investigations. The clinical effectiveness of retinalamin used as a neuroprotector for the treatment of the disabling conditions, such as atrophy of the optic nerve, retinopathy of prematurity, and degenerative myopia and post-therapy rehabilitation, is demonstrated.

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39

Sheng, Shiying, Jingzhong Huang, Yi Ren та ін. "Neuroprotection Against Hypoxic/Ischemic Injury: δ-Opioid Receptors and BDNF-TrkB Pathway". Cellular Physiology and Biochemistry 47, № 1 (2018): 302–15. http://dx.doi.org/10.1159/000489808.

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The delta-opioid receptor (DOR) is one of three classic opioid receptors in the opioid system. It was traditionally thought to be primarily involved in modulating the transmission of messages along pain signaling pathway. Although there were scattered studies on its other neural functions, inconsistent results and contradicting conclusions were found in past literatures, especially in terms of DOR’s role in a hypoxic/ischemic brain. Taking inspiration from the finding that the turtle brain exhibits a higher DOR density and greater tolerance to hypoxic/ischemic insult than the mammalian brain,

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40

Nandini, Cinintha, and Lukisiari Agustini. "Neuropati Optik Kompresif Bilateral yang Terinduksi oleh Schwannoma." Jurnal Ilmiah Kedokteran Wijaya Kusuma 9, no. 1 (2020): 40. http://dx.doi.org/10.30742/jikw.v9i1.733.

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The aim of this case report was to present further study about diagnosis and management of bilateral compressive optic neuropathy induced by schwannoma. This case report will discuss about the definition, classification, etiology, clinical diagnosis and treatment for compressive optic neuropathy. A case report. 43 years-old woman complained about blurred vision since 2 years ago. She also complained about pain on her right head while she was walking. There was no history of tinnitus or hearing loss. She had a history of contraception pill uses and progesterone injection every 3 months for 5 ye

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41

Castro Gago, Manuel, Mª Inés Novo Rodríguez, Carmen Gómez Lado, and Jesús Manuel Eirís Puñal. "Efecto neuroprotector de los factores dietéticos pre y perinatales sobre el neurodesarrollo." Revista de Neurología 44, S03 (2007): S001. http://dx.doi.org/10.33588/rn.44s03.2007220.

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42

Teryaeva, N. B. "Leptin as a Neuroprotector and a Central Nervous System Functional Stability Factor." Neuroscience and Behavioral Physiology 45, no. 6 (2015): 612–18. http://dx.doi.org/10.1007/s11055-015-0120-x.

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43

Horvath, Tamas L., Sabrina Diano, and Colin Barnstable. "Mitochondrial uncoupling protein 2 in the central nervous system: neuromodulator and neuroprotector." Biochemical Pharmacology 65, no. 12 (2003): 1917–21. http://dx.doi.org/10.1016/s0006-2952(03)00143-6.

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44

Alessenko, A. V., S. O. Bachurin, S. V. Gurianova, Y. O. Karatasso, E. F. Shevtsova, and L. N. Shingarova. "Tumor necrosis factor-alpha is a potential target for the neuroprotector Dimebon." Biochemistry (Moscow) Supplement Series B: Biomedical Chemistry 9, no. 2 (2015): 189–98. http://dx.doi.org/10.1134/s199075081502002x.

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45

Arnett, Anne B. "28.3 A Unique ASD Phenotype in Activity-Dependent Neuroprotector Homeobox (ADNP) Syndrome." Journal of the American Academy of Child & Adolescent Psychiatry 57, no. 10 (2018): S313. http://dx.doi.org/10.1016/j.jaac.2018.07.778.

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46

Tarasyuk, A. V., S. V. Pomogaibo, D. V. Kurilov, and T. A. Gudasheva. "Synthesis of BDNF-mimetic dimeric dipeptide GSB-106, a potential neuroprotector drug." Pharmaceutical Chemistry Journal 47, no. 1 (2013): 20–27. http://dx.doi.org/10.1007/s11094-013-0888-3.

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47

Arruda, Gian Lucas M., Hugo Vigerelli, Michelle C. Bufalo, et al. "Box Jellyfish (Cnidaria, Cubozoa) Extract Increases Neuron’s Connection: A Possible Neuroprotector Effect." BioMed Research International 2021 (March 4, 2021): 1–12. http://dx.doi.org/10.1155/2021/8855248.

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Neurodegenerative diseases are one of the major causes of death worldwide, characterized by neurite atrophy, neuron apoptosis, and synapse loss. No effective treatment has been indicated for such diseases so far, and the search for new drugs is being increased in the last years. Animal venoms’ secretion/venom can be an alternative for the discovery of new molecules, which could be the prototype for a new treatment. Here, we present the biochemical characterization and activity of the extract from the box jellyfish Chiropsalmus quadrumanus (Cq) on neurites. The Cq methanolic extract was obtaine

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48

Latypova, E. M., S. I. Timoshenko, G. A. Kislik, M. P. Vitek, A. L. Schwarzman, and S. V. Sarantseva. "Investigation of neuroprotective activity of apolipoprotein E peptide mimetic Cog1410 in transgenic lines of drosophila melanogaster." Biomeditsinskaya Khimiya 60, no. 4 (2014): 515–21. http://dx.doi.org/10.18097/pbmc20146004515.

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The neuroprotective activity of apolipoprotein E (apoE) peptide mimetic Cog1410, containing amino acid sequence of the receptor-binding domain apoE, has been investigated in transgenic lines of Drosophila melanogaster expressing human APP and beta-secretase. Expression of two transgenes caused neuropathological processes attributed to Alzheimer's disease: neurodegeneration, cognitive abnormality and amyloid deposits formation in brain. It was shown that Cog 1410 reduces neurodegeneration in brain of transgenic flies and improves cognitive functions (odor recognition). These data suggest that C

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49

Shcherbak, N. S., M. A. Popovetskiy, G. Yu Yukina, and M. M. Galagudza. "Effect of curcumin in the acute phase of ischemia in chronic cerebral hypoperfusion in rats." Regional blood circulation and microcirculation 17, no. 1 (2018): 69–73. http://dx.doi.org/10.24884/1682-6655-2018-17-1-69-73.

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Curcumin presents antioxidant and anti-inflammatory properties and can be considered as a neuroprotector. Data on doses and duration of application of curcumin to achieve protective effects in various types of ischemic brain injury is controversial. The purpose was to study the neuroprotective properties of curcumin in the acute phase of ischemia in chronic cerebral hypoperfusion in rats. It is shown that a single application of curcumin (300 mg/kg, i.p.) is not has neuroprotective effect in the acute phase of ischemia in chronic hypoperfusion in Wistar rats. The results allow to conclude that

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50

Martínez-Pizarro, Sandra. "Propiedades de la ingesta de aceite de coco en pacientes con Alzheimer." Revista de Discapacidad, Clínica y Neurociencias 7, no. 1 (2020): 1. http://dx.doi.org/10.14198/dcn.2020.7.1.01.

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Abstract:

El aceite de coco está formado por ácidos grasos que se pueden convertir en cetonas. Los cuerpos cetónicos son una fuente de energía alternativa en el cerebro, y pueden ser beneficiosos para los individuos con Alzheimer. En los últimos años se ha sugerido el uso del aceite de coco en dichos enfermos para mejorar sus funciones cognitivas. Los resultados muestran que activa la cetogénesis, mejora la orientación, el lenguaje, memoria y es neuroprotector. No obstante, es fundamental incrementar los estudios para corroborar la eficacia, y efectos a largo plazo; para ofrecer a los pacientes los mejo

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