Relevant bibliographies by topics / Neurospecific markers

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Neurospecific markers'

Author: Grafiati
Published: 28 May 2022
Last updated: 31 July 2025

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Journal articles on the topic "Neurospecific markers"

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Levchuk, L. A., N. A. Bokhan, and S. A. Ivanova. "Neurospecific Proteins as Transdiagnostic Markers of Affective Disorders." Нейрохимия 40, no. 1 (2023): 30–34. http://dx.doi.org/10.31857/s1027813323010119.

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Mental disorders have many differences in pathogenesis and clinical symptoms, however, they are characterized by general neurobiological processes that occur with a damage of nervous tissue, disturbance of blood-brain barrier, inclusion of autoimmune mechanisms, neurodegenerative processes and release of neurospecific proteins into the liquor and into the blood. We presented a review of current literature devoted to studies of the role of neurospecific proteins in the pathogenesis of affective disorders. Glial fibrillar acidic protein (GFAP), S-100 protein, myelin basic protein (MBP), and neur
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Panova, M. S., A. S. Panchenko, A. M. Ziganshin, and V. A. Mudrov. "Neurospecific markers of brain damage in infants." Rossiyskiy Vestnik Perinatologii i Pediatrii (Russian Bulletin of Perinatology and Pediatrics) 67, no. 5 (2022): 55–61. http://dx.doi.org/10.21508/1027-4065-2022-67-5-55-61.

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Timely diagnosis of brain lesions in infants is important for preventing the development and progression of neurological diseases.Purpose. The study aimed at investigation of neurospecific laboratory parameters in children of 1 year of age with structural changes in the brain.Material and methods. The clinical and laboratory examination of 50 children of 1 year of age, born at full term, was carried out. Serum concentrations of neurotrophic growth factor (BDNF), fractalkine (CX3CL-1), monocytic chemotactic protein (CCL2), trigger receptor expressed on myeloid cells-1, 2 (TREM-1, TREM-2), trans
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Zhukova, I. A., V. M. Alifirova, and N. G. Zhukova. "Neurospecific enolase as a nonspecific neurodegenerative process marker." Bulletin of Siberian Medicine 10, no. 2 (2011): 15–21. http://dx.doi.org/10.20538/1682-0363-2011-2-15-21.

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For the diagnosis and prognosis of various diseases is essential to determine specific markers in blood serum. Neuronspecific enolase (NSE) is a common marker of differentiatedneurons. The aim of the study was to evaluate the activity of NSE in patients with Parkinson's disease (PD) and chronic cerebral ischemia. The study results showed that the activity of NSE varied dependingon sex and age in PD patients, the stage and rate of progression of disease, age debut and severity of cognitive impairment. Thus, determination of NSE in the serum of PD patients should be included in the complex exami
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Stepanova, T. V., E. B. Popyhova, and A. N. Ivanov. "LABORATORY INDICATORS OF DIABETIC POLYNEUROPATHY." Medical academic journal 19, no. 1S (2019): 112–14. http://dx.doi.org/10.17816/maj191s1112-114.

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This study is devoted to diabetic polyneuropathy (DPN) - one of the common complications of diabetes mellitus (DM). The possibilities of laboratory diagnosis of this condition are shown. The purpose of this study was to examine the pathogenetic and diagnostic significance of certain neurospecific proteins and cytokines in DPN. The analysis of the literature data on the possibility of using neurospecific proteins and some cytokines in patients with diabetes as markers of neural tissue damage has been carried out.
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Levchuk, L. A., N. A. Bokhan, and S. A. Ivanova. "Neurospecific Proteins as Transdiagnostic Markers of Affective Disorders." Neurochemical Journal 17, no. 1 (2023): 25–29. http://dx.doi.org/10.1134/s1819712423010117.

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Samoylova, Yulia Gennad'evna, Maria Vladimirovna Novoselova, Natalya Grigor'evna Zhukova, and Olga Sergeevna Tonkikh. "Analysis of the Role of Neurospecific Proteins in the Diagnosis of Cognitive Dysfunction in Patients with Type 1 Diabetes Mellitus." Diabetes mellitus 17, no. 2 (2014): 83–90. http://dx.doi.org/10.14341/dm2014283-90.

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Background. Impairment of the central nervous system manifested as cognitive dysfunction caused by metabolic or structural changes is a severe progressive vascular complication of type 1 diabetes mellitus (T1DM). Significant difficulties in the diagnosis of cognitive dysfunction are associated with subjective diagnostic techniques. Objective. To identify the role of neurospecific markers in the diagnosis of cognitive dysfunction in patients with T1DM. Materials and Methods. A total of 58 patients with T1DM aged 16?30 years were included in this study. The control group included 29 healthy youn
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Karkashadze, George A., Leyla S. Namazova-Baranova, Leonid M. Yatsik, et al. "Levels of Neurospecific Peptides, Neurotransmitters and Neuroreceptor Markers in the Serum of Children with Various Sensory Disorders, Mild Cognitive Impairments and Other Neuropathology." Pediatric pharmacology 19, no. 6 (2023): 459–78. http://dx.doi.org/10.15690/pf.v19i6.2486.

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Background. The role of recently discovered neurospecific peptides in the pathogenesis of acute and progressive neurologic disorders, their neuroprotective features, and possibilities to use them as markers for the course and prognosis of certain diseases have been actively studied in recent decades. However, neurospecific peptides are almost not studied in chronic residual diseases. In our study we measured the levels of neurospecific peptides and some other markers to achieve understanding of general neurophysiological trends in congenital and acquired chronic non-progressive brain pathology
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BYKOV, YU V., and A. A. MURAVYOVA. "LABORATORY BIOMARKERS FOR BRAIN DAMAGE IN DIABETES MELLITUS." AVICENNA BULLETIN 25, no. 3 (2023): 370–79. http://dx.doi.org/10.25005/2074-0581-2023-25-3-370-379.

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Objective: This review outlines the literature data on the main laboratory biomarkers of brain damage in diabetes mellitus (DM) type I and II. Neurospecific proteins: S-100 protein, neurospecific enolase, glial fibrillar acidic protein, myelin basic protein, and brain-derived neurotrophic factor (BDNF) are considered specific markers of cerebral dysfunction in DM. Emphasis is placed on pro-inflammatory cytokines (IL-1, IL-6, tumor necrosis factor-α, C-reactive protein), as blood biomarkers, the increase of which indicates brain damage in DM type I and II. High concentrations of adipokines, inf
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Poluliakh, O. E., T. A. Mityukova, A. I. Martynenko, O. V. Zakharevich, and S. A. Marchuk. "Development of the concepts of childhood autism: pathogenetic mechanisms and markers." Proceedings of the National Academy of Sciences of Belarus, Medical series 16, no. 4 (2019): 499–512. http://dx.doi.org/10.29235/1814-6023-2019-16-4-499-512.

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Today, the study of autism spectrum disorders is relevant due to a high prevalence, an increase in the frequency of occurrence, as well as due to a lack of the unified concept of their etiology and pathogenesis. The purpose of the work is to conduct a brief review of evolution of the childhood autism ideas, including the pathogenesis issues and a search for possible markers of this disease. A brief information on the historical development of the childhood autism ideas, from the pre-Kanner period until now is presented. The modern pathogenesis theories are considered, including heredity, as we
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Samoĭlova, Iu G., M. V. Novoselova, A. K. Kostiunina, É. D. Pirmagomedova, and A. V. Latypov. "Predictors of the development of encephalopathy in the patients presenting with diabetes mellitus." Problems of Endocrinology 59, no. 5 (2013): 67–71. http://dx.doi.org/10.14341/probl201359567-71.

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The present review encompasses the data from the Russian-language and foreign publications. Diabetes mellitus (DM) is currently considered to be a global socio-medical, psychological, and economic problem due to the high frequency of vascular complications resulting in early invalidization of the patients. Encephalopathy (EP) is one of the most severe complications of DM leading to the substantial deterioration of the quality of life. Both the markers and the diagnostic criteria of EP remain to be determined. Certain neurospecific proteins are believed to be predictors of this pathology.
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Dissertations / Theses on the topic "Neurospecific markers"

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Sokhan, A. V. "Neurospecific markers - new opportunities in the diagnosis of bacterial meningitis." Thesis, Sumy State University, 2017. http://essuir.sumdu.edu.ua/handle/123456789/64505.

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У роботі представлені результати дослідження рівнів нейроспецифічних маркерів NSE, протеїну S-100, GFAP та MBP у цереброспінальній рідині хворих на менінгококовий та пневмококовий менінгіти в динаміці хвороби. Визначено наявність уражень нейронів, астрогліальних клітин та мієлінових нервів у всіх хворих та визначена можливість використання цих показників з діагностичною та прогностичною метою.<br>Diagnostic role of neuro specific proteins in assessing the degree of damage to the cells of the central nervous system currently confirmed. It proved that levels of neuron specific proteins
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Ekuase, Philip, and Mhando Catherine Samwel. "Neurospecific enolase - early diagnostical marker of CNS damage." Thesis, Sumy State University, 2015. http://essuir.sumdu.edu.ua/handle/123456789/41257.

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Book chapters on the topic "Neurospecific markers"

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Saadat Arif, Huseynova, Panakhova Nushaba Farkhad, Orujova Pusta Ali, et al. "Endothelial Dysfunction and Intestinal Barrier Injury in Preterm Infants with Perinatal Asphyxia." In Maternal and Child Health [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.110352.

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Perinatal asphyxia is one of the most frequent causes of perinatal morbidity, accounting for approximately 23% of neonatal deaths worldwide. Fetuses that suffer from hypoxia-ischemia are at high risk of developing multiorgan dysfunction, including the gut. Hypoxie-induced gut injury may result in adverse clinical outcomes, such as feeding intolerance and necrotizing enterocolitis. Increased permeability and subsequently an enhanced entry of bacteria and endotoxins into the systemic circulation can contribute to endotoxin aggression and further trigger numerous diseases. The aim of study is to
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