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Relevant bibliographies by topics / Neurotoxic Shellfish Poisoning

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Academic literature on the topic 'Neurotoxic Shellfish Poisoning'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Neurotoxic Shellfish Poisoning"

1

Watkins, Sharon M. "Neurotoxic Shellfish Poisoning." Marine Drugs 6, no. 3 (September 2008): 430–55. http://dx.doi.org/10.3390/md20080021.

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Watkins, Sharon, Andrew Reich, Lora Fleming, and Roberta Hammond. "Neurotoxic Shellfish Poisoning." Marine Drugs 6, no. 3 (July 12, 2008): 431–55. http://dx.doi.org/10.3390/md6030431.

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Abraham, Ann, Steven M. Plakas, Leanne J. Flewelling, Kathleen R. El Said, Edward L. E. Jester, Hudson R. Granade, Kevin D. White, and Robert W. Dickey. "Biomarkers of Neurotoxic Shellfish Poisoning." Toxicon 52, no. 2 (August 2008): 237–45. http://dx.doi.org/10.1016/j.toxicon.2008.04.175.

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Martín, R., T. García, B. Sanz, and P. E. Hernández. "Biotoxinas marinas: intoxicaciones por el consumo de moluscos bivalvos/Seafood toxins: poisoning by bivalve consumption." Food Science and Technology International 2, no. 1 (February 1996): 13–22. http://dx.doi.org/10.1177/108201329600200102.

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Seafood toxins are becoming increasingly important as etiologic agents of foodborne diseases around the world. This is partly because of greater awareness of the potential problems of the paralytic shellfish poisoning (PSP), neurotoxic shellfish poisoning (NSP), diarrheic shellfish poisoning (DSP) and more recently, a new type of seafood toxicity, called amnesic shellfish poisoning (ASP). This review describes the molluskan shellfish and biotoxins implicated, the development of standardized methods for detecting and quantifying these toxins, the importance of the economic loss resulting from their presence and the establishment of regular chemical monitoring for marine toxins.

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Garthwaite, Ian, Kathryn M. Ross, Christopher O. Miles, Lyn R. Briggs, Neale R. Towers, Teresa Borrell, and Phil Busby. "Integrated Enzyme-Linked Immunosorbent Assay Screening System for Amnesic, Neurotoxic, Diarrhetic, and Paralytic Shellfish Poisoning Toxins Found in New Zealand." Journal of AOAC INTERNATIONAL 84, no. 5 (September 1, 2001): 1643–48. http://dx.doi.org/10.1093/jaoac/84.5.1643.

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Abstract Enzyme-linked immunosorbent assays (ELISAs) were developed for amnesic, neurotoxic, and diarrhetic shellfish poisoning (ASP, NSP, and DSP) toxins and for yessotoxin. These assays, along with a commercially available paralytic shellfish poisoning (PSP) ELISA, were used to test the feasibility of an ELISA-based screening system. It was concluded that such a system to identify suspect shellfish samples, for subsequent analysis by methods approved by international regulatory authorities, is feasible. The assays had sufficient sensitivity and can be used on simple shellfish extracts. Alcohol extraction gave good recovery of all toxin groups. The ease of ELISAs permits the ready expansion of the system to screen for other toxins, as new ELISAs become available.

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Lefebvre, Kathi A., Betsy Jean Yakes, Elizabeth Frame, Preston Kendrick, Sara Shum, Nina Isoherranen, Bridget E. Ferriss, et al. "Discovery of a Potential Human Serum Biomarker for Chronic Seafood Toxin Exposure Using an SPR Biosensor." Toxins 11, no. 5 (May 23, 2019): 293. http://dx.doi.org/10.3390/toxins11050293.

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Domoic acid (DA)-producing harmful algal blooms (HABs) have been present at unprecedented geographic extent and duration in recent years causing an increase in contamination of seafood by this common environmental neurotoxin. The toxin is responsible for the neurotoxic illness, amnesic shellfish poisoning (ASP), that is characterized by gastro-intestinal distress, seizures, memory loss, and death. Established seafood safety regulatory limits of 20 μg DA/g shellfish have been relatively successful at protecting human seafood consumers from short-term high-level exposures and episodes of acute ASP. Significant concerns, however, remain regarding the potential impact of repetitive low-level or chronic DA exposure for which there are no protections. Here, we report the novel discovery of a DA-specific antibody in the serum of chronically-exposed tribal shellfish harvesters from a region where DA is commonly detected at low levels in razor clams year-round. The toxin was also detected in tribal shellfish consumers’ urine samples confirming systemic DA exposure via consumption of legally-harvested razor clams. The presence of a DA-specific antibody in the serum of human shellfish consumers confirms long-term chronic DA exposure and may be useful as a diagnostic biomarker in a clinical setting. Adverse effects of chronic low-level DA exposure have been previously documented in laboratory animal studies and tribal razor clam consumers, underscoring the potential clinical impact of such a diagnostic biomarker for protecting human health. The discovery of this type of antibody response to chronic DA exposure has broader implications for other environmental neurotoxins of concern.

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7

Morris, P. D., D. S. Campbell, T. J. Taylor, and J. I. Freeman. "Clinical and epidemiological features of neurotoxic shellfish poisoning in North Carolina." American Journal of Public Health 81, no. 4 (April 1991): 471–74. http://dx.doi.org/10.2105/ajph.81.4.471.

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8

Poli, Mark A., Steven M. Musser, Robert W. Dickey, Paul P. Eilers, and Sherwood Hall. "Neurotoxic shellfish poisoning and brevetoxin metabolites: a case study from Florida." Toxicon 38, no. 7 (July 2000): 981–93. http://dx.doi.org/10.1016/s0041-0101(99)00191-9.

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9

Cuypers, Eva, Angel Yanagihara, Jon D. Rainier, and Jan Tytgat. "TRPV1 as a key determinant in ciguatera and neurotoxic shellfish poisoning." Biochemical and Biophysical Research Communications 361, no. 1 (September 2007): 214–17. http://dx.doi.org/10.1016/j.bbrc.2007.07.009.

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Truman, Penelope, David J. Stirling, Peter Northcote, Robin J. Lake, Catherine Seamer, and Donald J. Hannah. "Determination of Brevetoxins in Shellfish by the Neuroblastoma Assay." Journal of AOAC INTERNATIONAL 85, no. 5 (September 1, 2002): 1057–63. http://dx.doi.org/10.1093/jaoac/85.5.1057.

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Abstract A neuroblastoma assay for determination of brevetoxins in shellfish was developed together with a method for sample cleanup that allows separation of brevetoxins from most of the components that cause matrix interference in the assay. This improved assay method was applied to a range of shellfish samples with different characteristics. Extracts of naturally contaminated and nontoxic shellfish together with extracts spiked with known amounts of toxin were tested. The results demonstrated that brevetoxins could be reliably detected in shellfish extracts at concentrations below the regulatory limit. Brevetoxin activity was detected in 15 of 23 samples from 5 separate toxicity incidents in which shellfish tested positive in the neurotoxic shellfish poisoning (NSP) mouse bioassay. Twelve of these positive NSP results came from 2 toxicity incidents. Yessotoxin was the major contributor to toxicity in 2 other incidents, although some samples contained both yessotoxin and brevetoxin. The sample from the remaining incident contained an unidentified toxin bioactivity, together with gymnodimine. In contrast to earlier versions of the neuroblastoma assay, gymnodimine was not detected by this modified method.

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Dissertations / Theses on the topic "Neurotoxic Shellfish Poisoning"

1

Atwood, Karen E. "Brevetoxin body burdens in seabirds of Southwest Florida." [Tampa, Fla.] : University of South Florida, 2008. http://purl.fcla.edu/usf/dc/et/SFE0002341.

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Allen, Sara E. "Florida Red Tides: Public Perceptions of Risk." [Tampa, Fla.] : University of South Florida, 2007. http://purl.fcla.edu/usf/dc/et/SFE0002267.

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Ogle, Marcus 1982. "Physical Mechanisms Driving Harmful Algal Blooms Along the Texas Coast." Thesis, 2012. http://hdl.handle.net/1969.1/148405.

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Commonly referred to as “red tide”, harmful algal blooms (HABs) formed by Karenia brevis occur frequently in the Gulf of Mexico (GOM). A bloom is defined as cell abundances >105 cells L-1. This thesis will focus primarily on Karenia brevis, formerly known as Gymnodinium breve, in the Gulf of Mexico. K. brevis is harmful because it produces brevetoxin, a ladder-frame polyether that acts as a potent neurotoxin in vertebrates. K. brevis commonly causes fish kills, respiratory irritation in humans, and Neurotoxic Shellfish Poisoning (NSP) if ingested. Blooms of K. brevis occur almost annually along the West Florida Shelf (WFS) in the late summer and early fall, when the coastal current is favorable for bloom initiation. Along the Texas-Louisiana shelf (TLS) however, blooms of K. brevis are infrequent and sporadic. While much is known of the blooms along the WFS due to their frequent presence, little is known of the mechanisms driving the blooms along the TLS due to their inconsistent presence. To understand the stochastic nature of HABs along the TLS, historical data of bloom occurrences from 1996 to present were compared with NOAA station PTAT2 wind, sea-level pressure, air and water temperature data and NCEP NARR-A sea-level pressure data. The difference in the monthly-mean along-shore component of the wind was statistically significant between bloom and non-bloom years in September (p<<0.001) and April (p=0.0015), with bloom years having a strong downcoast current. Monthly mean water temperature values yielded similar results between bloom and non-bloom years. Both March and September monthly-mean water temperature values were lower during non-bloom years with p-values of 0.01 and 0.048, respectively. These results suggest the possibly of forecasting for HABs along the TLS with currently measured, publicly available data.

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Book chapters on the topic "Neurotoxic Shellfish Poisoning"

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Chand, Pratap. "Seafood Neurotoxins I: Shellfish Poisoning and the Nervous System." In Clinical Neurotoxicology, 441–47. Elsevier, 2009. http://dx.doi.org/10.1016/b978-032305260-3.50046-0.

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