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Relevant bibliographies by topics / Neurotransmitters; Dopamine release

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Academic literature on the topic 'Neurotransmitters; Dopamine release'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Neurotransmitters; Dopamine release"

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Stern, Peter R. "Daylight Determines Dopamine." Science Signaling 6, no. 273 (2013): ec95-ec95. http://dx.doi.org/10.1126/scisignal.2004276.

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Expression of the appropriate neurotransmitters is essential for the function of neural circuits. Can neurons change their transmitter phenotype to deal with alterations in the environment? Dulcis et al. (see the Perspective by Birren and Marder) exposed adult rats to different photoperiods mimicking summer and winter daylengths. Neurotransmitter expression switched between dopamine and somatostatin in hypothalamic neurons that regulate release of corticotropin-releasing factor. Transmitter switching occurred at the transcriptional level and was accompanied by changes in postsynaptic receptors

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Kolbinger, Walter, and Reto Weiler. "Modulation of endogenous dopamine release in the turtle retina: Effects of light, calcium, and neurotransmitters." Visual Neuroscience 10, no. 6 (1993): 1035–41. http://dx.doi.org/10.1017/s0952523800010142.

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AbstractIn the turtle retina, dopamine has been observed in a small population of amacrine cells. Whereas the effect of dopamine has been intensively studied, knowledge about the release of this transmitter and the neuronal control of its release are still poorly understood. We therefore decided to study the release of endogenous dopamine. Isolated retinas were superfused with Ringer’s solutions and stimulated with increased potassium, light, or drugs which interfere with neurotransmitter systems. Dopamine was analyzed by using aluminum-oxide extraction and high-pressure liquid chromatography

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Myslivecek, Jaromir. "Two Players in the Field: Hierarchical Model of Interaction between the Dopamine and Acetylcholine Signaling Systems in the Striatum." Biomedicines 9, no. 1 (2021): 25. http://dx.doi.org/10.3390/biomedicines9010025.

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Tight interactions exist between dopamine and acetylcholine signaling in the striatum. Dopaminergic neurons express muscarinic and nicotinic receptors, and cholinergic interneurons express dopamine receptors. All neurons in the striatum are pacemakers. An increase in dopamine release is activated by stopping acetylcholine release. The coordinated timing or synchrony of the direct and indirect pathways is critical for refined movements. Changes in neurotransmitter ratios are considered a prominent factor in Parkinson’s disease. In general, drugs increase striatal dopamine release, and others ca

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Paterson, Louise M., Robin J. Tyacke, David J. Nutt, and Gitte M. Knudsen. "Measuring Endogenous 5-HT Release by Emission Tomography: Promises and Pitfalls." Journal of Cerebral Blood Flow & Metabolism 30, no. 10 (2010): 1682–706. http://dx.doi.org/10.1038/jcbfm.2010.104.

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Molecular in vivo neuroimaging techniques can be used to measure regional changes in endogenous neurotransmitters, evoked by challenges that alter synaptic neurotransmitter concentration. This technique has most successfully been applied to the study of endogenous dopamine release using positron emission tomography, but has not yet been adequately extended to other neurotransmitter systems. This review focuses on how the technique has been applied to the study of the 5-hydroxytryptamine (5-HT) system. The principles behind visualising fluctuations in neurotransmitters are introduced, with refe

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Wurtman, RJ. "Presynaptic control of Release of Amine Neurotransmitters by Precursor Levels." Physiology 3, no. 4 (1988): 158–63. http://dx.doi.org/10.1152/physiologyonline.1988.3.4.158.

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The amounts of such aminergic neurotransmitters as serotonin, dopamine, norepinephrine, and acetylcholine that are released into synapses, spontaneously and when the neurons fire, can be affected by the concentrations of their nutrient precursors tryptophan, tyrosine, and dopamine and can thus be influenced by eating "real" foods or taking the pure precursors. Simple laws can apparently enable the investigator to predict when precursor levels will or will not have such effects.

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Mora, Francisco, and Alberto Porras. "Effects of amphetamine on the release of excitatory amino acid neurotransmitters in the basal ganglia of the conscious rat." Canadian Journal of Physiology and Pharmacology 71, no. 5-6 (1993): 348–51. http://dx.doi.org/10.1139/y93-054.

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The effects of systemic injections of amphetamine sulfate on the release of aspartic acid, glutamic acid, and glutamine were studied using a push–pull perfusion system in the conscious rat. Amphetamine produced a dose-related increase of the extracellular levels of aspartic acid and glutamic acid. The mean time effect of amphetamine was 40 min, followed by a recovery to baseline levels. The mean percentage increase in amino acids released by the highest dose of amphetamine (5 mg/kg) was as follows: Asp, 334.6%; Glu, 224.5%; and Gln, 317.6%. All these effects were blocked by the dopamine D1–D2

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Gubernator, N. G., H. Zhang, R. G. W. Staal, et al. "Fluorescent False Neurotransmitters Visualize Dopamine Release from Individual Presynaptic Terminals." Science 324, no. 5933 (2009): 1441–44. http://dx.doi.org/10.1126/science.1172278.

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Trouillon, Raphaël, and Martin A. M. Gijs. "Dynamic electrochemical quantitation of dopamine release from a cells-on-paper system." RSC Advances 6, no. 37 (2016): 31069–73. http://dx.doi.org/10.1039/c6ra02487d.

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Lacoste, A., S. K. Malham, A. Cueff, F. Jalabert, F. Gelebart, and S. A. Poulet. "Evidence for a form of adrenergic response to stress in the mollusc Crassostrea gigas." Journal of Experimental Biology 204, no. 7 (2001): 1247–55. http://dx.doi.org/10.1242/jeb.204.7.1247.

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Catecholamines and pro-opiomelanocortin (POMC)-derived peptides, some of the central regulators of the stress-response systems of vertebrates, are also present in invertebrates. However, studies are needed to determine how these hormones participate in the organisation of neuroendocrine stress-response axes in invertebrates. Our present work provides evidence for the presence of an adrenergic stress-response system in the oyster Crassostrea gigas. Noradrenaline and dopamine are released into the circulation in response to stress. Storage and release of these hormones take place in neurosecreto

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Agnesi, Filippo, Susannah J. Tye, Jonathan M. Bledsoe, et al. "Wireless Instantaneous Neurotransmitter Concentration System–based amperometric detection of dopamine, adenosine, and glutamate for intraoperative neurochemical monitoring." Journal of Neurosurgery 111, no. 4 (2009): 701–11. http://dx.doi.org/10.3171/2009.3.jns0990.

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Object In a companion study, the authors describe the development of a new instrument named the Wireless Instantaneous Neurotransmitter Concentration System (WINCS), which couples digital telemetry with fast-scan cyclic voltammetry (FSCV) to measure extracellular concentrations of dopamine. In the present study, the authors describe the extended capability of the WINCS to use fixed potential amperometry (FPA) to measure extracellular concentrations of dopamine, as well as glutamate and adenosine. Compared with other electrochemical techniques such as FSCV or high-speed chronoamperometry, FPA o

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Dissertations / Theses on the topic "Neurotransmitters; Dopamine release"

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Phillips, Paul Edward Mackenzie. "Presynaptic control of striatal dopamine release in vitro." Thesis, Queen Mary, University of London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.313364.

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Cragg, Stephanie Jane. "Electrochemical studies of somatodendritic dopamine release in midbrain." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337781.

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Daniel, James St Vincent Clinical School UNSW. "Studies of neurotransmitter release mechanisms in dopamine neurons." Awarded by:University of New South Wales. St. Vincent Clinical School, 2007. http://handle.unsw.edu.au/1959.4/31934.

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Medications that treat diseases such as Parkinson???s disease work by regulating dopamine transmission at synapses. Surprisingly, little is known about the mechanisms regulating dopamine release at synapses. In this thesis, we study mechanisms that regulate vesicle recycling in axons and dendrites of dopamine neurons. Key questions we addressed were: (1) Are vesicles in axons and dendrites associated with the same regulatory proteins, and thus by implication the same regulatory mechanisms, as in excitatory neurons; (2) Do vesicles undergo recycling, and (3) if so, are they characterised by a d

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Moss, Sharon Helen. "Neurotensin : an ontogenic study." Thesis, University of Nottingham, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.319620.

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Fairbrother, Iain Simon. "The relationship between intraterminal pools of dopamine and its release by chemical stimuli : an in vivo microdialysis study." Thesis, University of Edinburgh, 1988. http://hdl.handle.net/1842/18871.

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Books on the topic "Neurotransmitters; Dopamine release"

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Arnold, Monica M., Lauren M. Burgeno, and Paul E. M. Phillips. Fast-Scan Cyclic Voltammetry in Behaving Animals. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199939800.003.0005.

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Gaining insight into the mechanisms by which neural transmission governs behavior remains a central goal of behavioral neuroscience. Multiple applications exist for monitoring neurotransmission during behavior, including fast-scan cyclic voltammetry (FSCV). This technique is an electrochemical detection method that can be used to monitor subsecond changes in concentrations of electroactive molecules such as neurotransmitters. In this technique, a triangular waveform voltage is applied to a carbon fiber electrode implanted into a selected brain region. During each waveform application, specific

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Book chapters on the topic "Neurotransmitters; Dopamine release"

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Zhou, Fu-Ming, and John A. Dani. "Dopamine and Serotonin Crosstalk Within the Dopaminergic and Serotonergic Systems." In Co-Existence and Co-Release of Classical Neurotransmitters. Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-09622-3_9.

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McRae, Amanda, S. Hjorth, D. Mason, L. Dillon, and T. Tice. "Implantable microencapsulated dopamine (DA): prolonged functional release of DA in denervated striatal tissue." In Neurotransmitter Actions and Interactions. Springer Vienna, 1990. http://dx.doi.org/10.1007/978-3-7091-9050-0_20.

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Chéramy, A., L. Barbeito, G. Godeheu, et al. "Respective contributions of neuronal activity and presynaptic mechanisms in the control of the in vivo release of dopamine." In Neurotransmitter Actions and Interactions. Springer Vienna, 1990. http://dx.doi.org/10.1007/978-3-7091-9050-0_18.

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"Nerve and muscle." In Oxford Assess and Progress: Medical Sciences, edited by Jade Chow, John Patterson, Kathy Boursicot, and David Sales. Oxford University Press, 2012. http://dx.doi.org/10.1093/oso/9780199605071.003.0016.

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Higher animals have four basic tissue types: epithelial tissue, connective tissue, nervous tissue, and muscle. Of these, nerve and muscle are grouped together as ‘excitable cells’ because the cell membrane has the ability to vary membrane ion conductance and membrane voltage so as to transmit meaningful signals within and between cells. Within excitable cells information is transmitted using either an amplitude-modulated (AM) code using slow, electrotonic potentials, or a frequency-modulated (FM) code when signalling is by action potentials. Much of the signalling between excitable cells occurs at chemical synapses where a chemical neurotransmitter is released from presynaptic cells and then interacts with postsynaptic membrane receptors. Clinical symptoms can arise when the release of chemical neurotransmitters is disturbed, or when availability of postsynaptic receptors is altered. Thus, a reduction in dopamine release from basal ganglia substantia nigra cells is found in Parkinson’s disease, while myasthenia gravis results from loss of nicotinic acetylcholine receptors at the neuromuscular junction of skeletal muscle. Sometimes transmission from cell to cell is not by chemical neurotransmitter but by electrical synapses, where gap-junctions provide direct electrical connectivity. Transmission between cardiac muscle cells occurs in this way. Some cardiac arrhythmias, such as Wolff –Parkinson–White syndrome, are a consequence of an abnormal path of electrical conduction between cardiac muscle fibres. Sensory cells on and within the body pass information via afferent pathways from the peripheral nervous system into the central nervous system (CNS). CNS processes and sensory information are integrated to produce outputs from the CNS. These outputs pass by various efferent routes to the effector organs: skeletal muscle, cardiac muscle, smooth muscle, and glands. It is through these effectors that the CNS is able to exert control over the body and to interact with the environment. Alterations of function anywhere in the afferent, integrative, or efferent aspects of the system, as well as defects in the effectors themselves, are likely to lead to significant clinical symptoms and signs. The efferent outflow from the CNS has two major components. One, the somatic nervous system, innervates only skeletal muscle. The other is the autonomic nervous system (ANS), which innervates cardiac muscle, smooth muscle, and the glands of the viscera and skin.

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Keni, Jyotsna, and Anna Pawlikowska –. Haddal. "Growth Regulation." In Textbook of Endocrine Physiology. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780199744121.003.0014.

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While multiple hormones influence somatic growth, the main regulator of postnatal growth is growth hormone. Growth hormone (GH) is secreted in a pulsatile manner from the anterior pituitary primarily as a 22-kilodalton molecule (although other forms may be found). The development of the pituitary gland as well as GH gene expression is regulated by the multiple pituitary transcription factors listed in Table11-1. The Pit-1 and Prop-1 genes encode proteins that are often mutated or deleted in cases of congenital hypopituitarism. Under normal waking conditions, GH levels are often low or undetectable, but several times during the day, and particularly at night during stage 3 of sleep, surges of GH secretion occur. The pulsatile pattern characteristic of GH secretion largely reflects the interaction of multiple regulators, including two hypothalamic regulatory peptides: GH-releasing hormone (GHRH), which stimulates GH secretion, and somatostatin (somatotropin release–inhibiting factor [SRIF]), which inhibits GH secretion. Multiple neurotransmitters and neuropeptides are involved in regulation of release of these hypothalamic factors, including, but not limited to, serotonin, histamine, norepinephrine, dopamine, acetylcholine, γ -aminobutyric acid (GABA), thyroid-releasing hormone, vasoactive intestinal peptide, gastrin, neurotensin, substance P, calcitonin, neuropeptide Y, vasopressin, corticotropinreleasing hormone, and galanin. Many factors influence GH secretion; notably, glucose that inhibits, and certain amino acids and Ghrelin that stimulate GH secretion. GH secretion is also impacted by a variety of nonpeptide hormones, including androgens, estrogens, thyroxine, and glucocorticoids. The precise mechanisms by which these hormones regulate GH secretion are complex, potentially involving actions at both the hypothalamic and pituitary levels. Exogenous physiological and pharmacological factors are known to stimulate GH secretion. Some of these agents, including clonidine, L-dopa, and exercise, are used in GH stimulation tests. In plasma, the majority of GH is bound with high specificity and affinity, but with relatively low capacity to a carrier protein termed GH binding protein (GHBP). The GHBP is a cleavage product of the extracellular domain of the GH receptor.

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Parker, Linda A. "Cannabinoids, Reward, and Addiction." In Cannabinoids and the Brain. The MIT Press, 2017. http://dx.doi.org/10.7551/mitpress/9780262035798.003.0006.

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Dopamine is a neurotransmitter critical for reward processing and is elevated by most addicting drugs. The effect of THC and other CB<sub>1</sub> agonists moderately elevate dopamine in reward related regions of the rodent brain; however, there is less consistent evidence in humans for marijuana-induced changes in dopamine release or for morphological changes in brain reward areas. In humans, cannabis use disorder has been identified, which shows similar features of other substance use disorders, but not in the same extremes as opiates, psychostimulants or alcohol. This chapter discusses the interaction between cannabis and other drugs in relapse to drugs use, with a special case for the interaction between cannabinoids and opiates. Finally, the relationship between cannabinoid effects on men and women in sexual behaviour is discussed.

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"Effects of Antipsychotic Drugs on Dopamine Release and Metabolism in the Central Nervous System." In Neurotransmitter Receptors in Actions of Antipsychotic Medications. CRC Press, 2000. http://dx.doi.org/10.1201/9781420041774-7.

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David Jentsch, J., and Robert Roth. "Effects of Antipsychotic Drugs on Dopamine Release and Metabolism in the Central Nervous System." In Neurotransmitter Receptors in Actions of Antipsychotic Medications. CRC Press, 2000. http://dx.doi.org/10.1201/9781420041774.ch3.

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Zhu, Gang, Shukuko Yoshida, Sunao Kaneko, Shinichi Hirose, and Motohiro Okada. "Mechanisms of Calcium-Associated Exocytosis of Striatal Dopamine and DOPA Release Studied by In Vivo Microdialysis." In Neurobiology of DOPA as a Neurotransmitter. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023329.ch6.

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Misu, Yoshimi, Masashi Sasa, Kumatoshi Ishihara, and Muhammad Akbar. "Dopamine-Independent Inhibition of Hippocampal CA1 Neurons Produced by Nanomolar Levodopa with Facilitation of Noradrenaline and GABA Release in Rats." In Neurobiology of DOPA as a Neurotransmitter. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023329.ch17.

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