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Relevant bibliographies by topics / Neurotrophic drugs

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Academic literature on the topic 'Neurotrophic drugs'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Neurotrophic drugs"

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Gupta, Akash, Jeremias G. Galletti, Zhiyuan Yu, Kevin Burgess, and Cintia S. de Paiva. "A, B, C’s of Trk Receptors and Their Ligands in Ocular Repair." International Journal of Molecular Sciences 23, no. 22 (2022): 14069. http://dx.doi.org/10.3390/ijms232214069.

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Neurotrophins are a family of closely related secreted proteins that promote differentiation, development, and survival of neurons, which include nerve growth factor (NGF), brain-derived neurotrophic factor, neurotrophin-3, and neurotrophin-4. All neurotrophins signal through tropomyosin receptor kinases (TrkA, TrkB, and TrkC) which are more selective to NGF, brain-derived neurotrophic factor, and neurotrophin-3, respectively. NGF is the most studied neurotrophin in the ocular surface and a human recombinant NGF has reached clinics, having been approved to treat neurotrophic keratitis. Brain-d

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Hassan, Khadija, Blondelle Matio Kemkuignou, Marco Kirchenwitz, et al. "Neurotrophic and Immunomodulatory Lanostane Triterpenoids from Wood-Inhabiting Basidiomycota." International Journal of Molecular Sciences 23, no. 21 (2022): 13593. http://dx.doi.org/10.3390/ijms232113593.

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Neurotrophins such as nerve growth factor (ngf) and brain-derived neurotrophic factor (bdnf) play important roles in the central nervous system. They are potential therapeutic drugs for the treatment of neurodegenerative diseases, including Alzheimer’s disease and Parkinson’s disease. In this study, we investigated the neurotrophic properties of triterpenes isolated from fruiting bodies of Laetiporus sulphureus and a mycelial culture of Antrodia sp. MUCL 56049. The structures of the isolated compounds were elucidated based on nuclear magnetic resonance (NMR) spectroscopy in combination with hi

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Khadija, Hassan, Matio Kemkuignou Blondelle, Kirchenwitz Marco, et al. "Neurotrophic and Immunomodulatory Lanostane Triterpenoids from Wood-Inhabiting Basidiomycota." Int. J. Mol. Sci. 23 (March 26, 2023): 13593. https://doi.org/10.3390/ ijms232113593.

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Neurotrophins such as nerve growth factor (ngf) and brain-derived neurotrophic factor (bdnf) play important roles in the central nervous system. They are potential therapeutic drugs for the treatment of neurodegenerative diseases, including Alzheimer’s disease and Parkinson’s disease. In this study, we investigated the neurotrophic properties of triterpenes isolated from fruiting bodies of Laetiporus sulphureus and a mycelial culture of Antrodia sp. MUCL 56049. The structures of the isolated compounds were elucidated based on nuclear magnetic resonance (NMR) spectroscopy in combina

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4

CASTREN, E. "Neurotrophic effects of antidepressant drugs." Current Opinion in Pharmacology 4, no. 1 (2004): 58–64. http://dx.doi.org/10.1016/j.coph.2003.10.004.

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Chaldakov, George N., Luigi Aloe, Stanislav G. Yanev, et al. "Trackins (Trk-Targeting Drugs): A Novel Therapy for Different Diseases." Pharmaceuticals 17, no. 7 (2024): 961. http://dx.doi.org/10.3390/ph17070961.

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Many routes may lead to the transition from a healthy to a diseased phenotype. However, there are not so many routes to travel in the opposite direction; that is, therapy for different diseases. The following pressing question thus remains: what are the pathogenic routes and how can be they counteracted for therapeutic purposes? Human cells contain >500 protein kinases and nearly 200 protein phosphatases, acting on thousands of proteins, including cell growth factors. We herein discuss neurotrophins with pathogenic or metabotrophic abilities, particularly brain-derived neurotrophic factor (

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Baazaoui, Narjes, and Khalid Iqbal. "Alzheimer’s Disease: Challenges and a Therapeutic Opportunity to Treat It with a Neurotrophic Compound." Biomolecules 12, no. 10 (2022): 1409. http://dx.doi.org/10.3390/biom12101409.

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Alzheimer’s disease (AD) is a progressive neurodegenerative disease with an insidious onset and multifactorial nature. A deficit in neurogenesis and synaptic plasticity are considered the early pathological features associated with neurofibrillary tau and amyloid β pathologies andneuroinflammation. The imbalance of neurotrophic factors with an increase in FGF-2 level and a decrease in brain derived neurotrophic factor (BDNF) and neurotrophin 4 (NT-4) in the hippocampus, frontal cortex and parietal cortex and disruption of the brain micro-environment are other characteristics of AD. Neurotrophi

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Gören, Jessica L. "Brain-derived neurotrophic factor and schizophrenia." Mental Health Clinician 6, no. 6 (2016): 285–88. http://dx.doi.org/10.9740/mhc.2016.11.285.

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Abstract Schizophrenia is a severe disorder affecting approximately 1% of the population. Historically, alterations of dopaminergic function were considered the primary cause of schizophrenia. However, for many patients, drugs that alter dopaminergic function do not consistently lead to resolution of the symptoms of schizophrenia. Thus, there is an increased interest in pathophysiologic processes that result in altered neurodevelopment and plasticity associated with schizophrenia. Brain-derived neurotrophic factor (BDNF) is a neurotrophin involved in neurogenesis, synaptic plasticity, cognitio

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Mosiołek, Anna, Jadwiga Mosiołek, Sławomir Jakima, Aleksandra Pięta, and Agata Szulc. "Effects of Antidepressant Treatment on Neurotrophic Factors (BDNF and IGF-1) in Patients with Major Depressive Disorder (MDD)." Journal of Clinical Medicine 10, no. 15 (2021): 3377. http://dx.doi.org/10.3390/jcm10153377.

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Major depressive disorder (MDD) remains the subject of ongoing research as a multifactorial disease and a serious public health problem. There is a growing body of literature focusing on the role of neurotrophic factors in pathophysiology of MDD. A neurotrophic hypothesis of depression proposes that abnormalities of neurotrophins serum levels lead to neuronal atrophy and decreased neurogenesis, resulting in mood disorders. Consequently, in accordance with recent findings, antidepressant treatment modifies the serum levels of neurotrophins and thus leads to a clinical improvement of MDD. The pu

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Borowicz-Reutt, Kinga K. "How Antidepressant Drugs Affect the Antielectroshock Action of Antiseizure Drugs in Mice: A Critical Review." International Journal of Molecular Sciences 22, no. 5 (2021): 2521. http://dx.doi.org/10.3390/ijms22052521.

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Depression coexists with epilepsy, worsening its course. Treatment of the two diseases enables the possibility of interactions between antidepressant and antiepileptic drugs. The aim of this review was to analyze such interactions in one animal seizure model—the maximal electroshock (MES) in mice. Although numerous antidepressants showed an anticonvulsant action, mianserin exhibited a proconvulsant effect against electroconvulsions. In most cases, antidepressants potentiated or remained ineffective in relation to the antielectroshock action of classical antiepileptic drugs. However, mianserin

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Potupchik, T. V., O. F. Veselova, and I. V. Gatskikh. "Pharmacotherapeutic aspects of nootropics use in people with alcohol dependence." Medical alphabet 2, no. 19 (2019): 37–41. http://dx.doi.org/10.33667/2078-5631-2019-2-19(394)-37-41.

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In the pharmacotherapy of chronic alcoholism a significant place is given to nootropic drugs. Currently, neurodegenerative processes are associated with the activity of various neurotrophic factors of the brain and neuropeptides. An important advantage of the use of neuropeptide drugs (cerebrolysin, semax, cortexin) in the treatment of chronic alcoholism is that they have an organ-specific multimodal effect on the brain: provide metabolic regulation, neuroprotection, functional neuromodulation and neurotrophic activity. They are recommended for relief of acute withdrawal syndrome, as well as i

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Dissertations / Theses on the topic "Neurotrophic drugs"

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Sawamura, Megumi. "Development of novel treatment against glaucoma; Intraocular pressure-lowering by cytoskeletal drugs and neuroprotection by neurotrophic factors." Kyoto University, 2001. http://hdl.handle.net/2433/150551.

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Perna, Marla Kay. "Nicotine Sensitization in a Rodent Model of Schizophrenia: A Comparison of Adolescents, Adults, and Neurotrophic Factors." Digital Commons @ East Tennessee State University, 2007. https://dc.etsu.edu/etd/2106.

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The behavioral effects of nicotine on locomotor activity in a rodent model of psychosis were analyzed. This model is based on neonatal quinpriole treatment (a dopamine D2/D3 agonist) which causes increased D2 receptor sensitivity, a phenomenon known as D2 priming that is common in schizophrenia. D2-primed adolescent rats did not demonstrate nicotine-induced hypoactivity early in training, and males demonstrated more rapid sensitization to nicotine as compared to controls administered nicotine. D2-primed females administered nicotine demonstrated increased stereotypic behavior. D2-primed adult

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Förander, Petter. "On the actions of neurotrophic factors on the chromaffin cells of the adrenal medulla /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4145-9/.

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Jain, Anjana. "Delivery of Cdc42, Rac1, and Brain-derived Neurotrophic Factor to Promote Axonal Outgrowth After Spinal Cord Injury." Diss., Georgia Institute of Technology, 2007. http://hdl.handle.net/1853/16210.

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Injury severs the axons in the spinal cord causing permanent functional loss. After injury, a series of events occur around the lesion site, including the deposition of growth cone inhibitory astroglial scar tissue containing chondroitin sulfate proteoglycan (CSPG)- rich regions. It is important to encourage axons to extend through these inhibitory regions for regeneration to occur. The work presented in this dissertation investigates the effect of three proteins, constitutively active (CA)-Cdc42, CA-Rac1, and brain-derived neurotrophic factor (BDNF) on axonal outgrowth through CSPGs-rich i

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Cummins, Elizabeth D., Stephen B. Griffin, Chase M. Duty, Daniel J. Peterson, Katherine C. Burgess, and Russell W. Brown. "The Role of Dopamine D1 and D2 Receptors in Adolescent Methylphenidate Conditioned Place Preference: Sex Differences and Brain-Derived Neurotrophic Factor." Digital Commons @ East Tennessee State University, 2014. https://doi.org/10.1159/000360636.

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This study analyzed the role of dopamine D1 and D2 receptors in methylphenidate (MPH) conditioned place preference (CPP) in adolescent male and female rats, in addition to the role of these receptors in the effects of MPH on brain-derived neurotrophic factor (BDNF) in the dorsal striatum and nucleus accumbens. Using a nonbiased CPP procedure, the animals were conditioned from postnatal day (PD) 33 to 37. On conditioning trials, animals were first administered saline or their respective antagonist (0.1 or 0.2 mg/kg SCH-23390; 0.01 or 0.03 mg/kg eticlopride HCl), followed by MPH (5 mg/kg). Appro

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COA, ROBERTA. "Brain Plasticity induced by Vagal Nerve Stimulation in Patients affected by Drug-resistant Epilepsy: effects on Brain Network and Neurotrophins." Doctoral thesis, Università degli Studi di Cagliari, 2022. http://hdl.handle.net/11584/327392.

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Chronic Vagal Nerve Stimulation (VNS) is an approved neuromodulation therapy that is effective in the treatment of drug-resistant epilepsy (DRE). Despite the high number of devices implanted and subjects treated (over 100,000), the exact mechanism of action is still debated in the literature: widespread effects on neurotransmitter expression, blood flow, brain network and EEG, neuroinflammation, and neurotrophins (e.g. BDNF and NGF) have been proposed as key to understanding the efficacy of VNS on seizure frequency, psychiatric symptoms, memory, and quality of life of DRE subjects. Neuroplasti

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Brown, Russell W., Marla K. Perna, Tori L. Schaefer, and Michael T. Williams. "The Effects of Adulthood Nicotine Treatment on D2-Mediated Behavior and Neurotrophins of Rats Neonatally Treated with Quinpirole." Digital Commons @ East Tennessee State University, 2006. https://doi.org/10.1002/syn.20237.

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This study was designed to analyze the effects of nicotine on yawning behavior and neurotrophin content in the hippocampus and frontal cortex of D2-receptor primed female adult Sprague-Dawley rats. Animals were neonatally treated with quinpirole, a dopamine (DA) D2/D3 agonist, from postnatal day 1-21 (P1-21) and raised to P60 and administered nicotine tartarate (0.3 mg/kg free base) or saline twice daily for 14 days. One day after nicotine treatment had ceased, the number of yawns was recorded for 1 h in response to an acute injection of quinpirole (i.p., 100 microg/kg). Yawning is a D2-recept

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Kandalam, Saikrishna. "Pharmacologically active microcarriers delivering brainderived neurotrophic factor combined to adult mesenchymal stem cells : novel approach for the treatment of spinal cord injury." Thesis, Angers, 2017. http://www.theses.fr/2017ANGE0050/document.

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Un traumatisme de la moelle épinière (TME) est une condition dévastatrice entraînant la perte permanente de fonctions neuronales. L’objectif de cette thèse est de formuler de microsupports pharmacologiquement actif (MPAs) avec une surface de fibronectine (FN), libérant le« brain-derived neurotrophic factor » (BDNF) de façon controlée. Nous voulons combiner ce système avec des cellules souches mésenchymateuses (CSMs) pour la réparation de TME. Le BDNF nanoprécipité a été encapsulé dans les FN-MPAs et le profil de libération in vitro a été évaluée. Elle a montré une libération biphasique et prol

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Soong, David. "Classifying the manner of death in drug/ethanol overdose in equivocal cases: a suggested future tool for medical examiners using neuroanatomical markers." Thesis, 2014. https://hdl.handle.net/2144/13296.

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The purpose of the present thesis was to propose a guideline to differentiate between an accidental or suicide manner of death when dealing with a drug/ethanol overdose in which all available medical and investigational evidence, including a psychological autopsy, is inconclusive, thereby resulting in an undetermined manner of death. An in-depth literature review was conducted in the field of neuroscience, psychiatry, and pharmacology to discover neuroanatomical markers indicative of suicidal behavior in the context of two major risk factors of suicide, stress and depression, and two hypothese

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Books on the topic "Neurotrophic drugs"

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Workshop on Neurotransmitters and Diseases (3rd 1986 Tokyo, Japan). Psychotropic and neurotropic drugs and neurotransmitter receptors: Proceedings of the Third Workshop on Neurotransmitters and Diseases, Tokyo, June 21, 1986. Edited by Takahashi Ryō 1926- and Kameyama Masakuni 1924-. Excerpta Medica, 1986.

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Gregory, Bock, and Goode Jamie, eds. Growth factors as drugs for neurological and sensory disorders. Wiley, 1996.

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De Leo, Joyce A., 1960-, Sorkin Linda S. 1953-, Watkins Linda R. 1954-, and International Association for the Study of Pain., eds. Immune and glial regulation of pain. IASP Press, 2007.

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Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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Psychotropic & Neurotropic Drugs & Neurotransmitter Receptors: Proceedings of the 3rd Workshop on Neurotransmitters & Diseases, Tokyo, Japan, June 21, (International Congress Series). Elsevier Science Publishing Company, 1987.

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6

Carvalho, André F., Gilberto S. Alves, Cristiano A. Köhler, and Roger S. McIntyre. Cognitive Enhancement in Major Depressive Disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190214401.003.0010.

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Major depressive disorder (MDD) is a chronic and disabling illness often associated with elevated rates of non-recovery and substantial psychosocial burden. Cognitive impairment is a common residual manifestations of MDD. Overactivation of the hypothalamic–pituitary–adrenal axis, along with immune–inflammatory imbalances, a decrease in neurotrophin signaling, and an increase in oxidative and nitrosative stress, leads to neuroprogression and cognitive deterioration in MDD. “Cognitive remission” has been proposed as a novel treatment target for MDD. Cognitive remediation therapy has provided enc

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(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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Book chapters on the topic "Neurotrophic drugs"

1

Hefti, Franz, Mark P. Armanini, Klaus D. Beck, et al. "Development of Neurotrophic Factor Therapy for Alzheimer's Disease." In Ciba Foundation Symposium 196 - Growth Factors as Drugs for Neurological and Sensory Disorders. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514863.ch5.

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Apfel, Stuart C., and John A. Kessler. "Neurotrophic Factors in the Treatment of Peripheral Neuropathy." In Ciba Foundation Symposium 196 - Growth Factors as Drugs for Neurological and Sensory Disorders. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514863.ch8.

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Hilt, Dana C., James A. Miller, and Errol Malta. "Early Experiences with Trophic Factors as Drugs for Neurological Disease: Brain-Derived Neurotrophic Factor and Ciliary Neurotrophic Factor for ALS." In Cell Death and Diseases of the Nervous System. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-4612-1602-5_29.

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Van De Water, Thomas R., Hinrich Staecker, Patrik Ernfors, Gustave Moonen, and Philippe P. Lefebvre. "Neurotrophic Factors as Pharmacological Agents for the Treatment of Injured Auditory Neurons." In Ciba Foundation Symposium 196 - Growth Factors as Drugs for Neurological and Sensory Disorders. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514863.ch11.

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Tolkovsky, Aviva M., and Jack Brelstaff. "Sensory Neurons from Tau Transgenic Mice and Their Utility in Drug Screening." In Neurotrophic Factors. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7571-6_7.

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Vogel, H. Gerhard, Wolfgang H. Vogel, Bernward A. Schölkens, Jürgen Sandow, Günter Müller, and Wolfgang F. Vogel. "Psychotropic and neurotropic activity1." In Drug Discovery and Evaluation. Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/3-540-29837-1_6.

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Vogel, Hans Gerhard. "Psychotropic and Neurotropic Activity." In Drug Discovery and Evaluation. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-70995-4_6.

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Vogel, H. Gerhard, and Wolfgang H. Vogel. "Psychotropic and neurotropic activity." In Drug Discovery and Evaluation. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-03333-3_5.

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Amadoro, G., and P. Calissano. "Chapter 7. Zinc, Copper, Neurotrophic Factors and Neurodegeneration." In Drug Discovery. Royal Society of Chemistry, 2011. http://dx.doi.org/10.1039/9781849733014-00141.

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Krieglstein, J., I. Semkova, C. Culmsee, and P. Wolz. "Neuroprotection by Drug-Induced Neurotrophic Factors." In Maturation Phenomenon in Cerebral Ischemia II. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60546-8_8.

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Conference papers on the topic "Neurotrophic drugs"

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Karpova, Inessa V., Eugeniy R. Bychkov, and Peter D. Shabanov. "Asymmetric monoaminergic effects of neurotropic peptidergic drugs." In II Международная конференция, посвящеенная 100- летию И.А. Држевецкой. СКФУ, 2022. http://dx.doi.org/10.38006/9612-62-6.2022.170.173.

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Reznikov, A., D. Nikerov, and Yu Klimochkin. "ASYMMETRIC SYNTHESIS OF NEUROTROPIC DRUGS – GABA ANALOGS USING NI(II)-CATALYZED MICHAEL ADDITION." In MedChem-Russia 2021. 5-я Российская конференция по медицинской химии с международным участием «МедХим-Россия 2021». Издательство Волгоградского государственного медицинского университета, 2021. http://dx.doi.org/10.19163/medchemrussia2021-2021-31.

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