Relevant bibliographies by topics / Nibrin

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  2. Dissertations / Theses
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Academic literature on the topic 'Nibrin'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Nibrin"

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Cerosaletti, Karen, Jocyndra Wright, and Patrick Concannon. "Active Role for Nibrin in the Kinetics of Atm Activation." Molecular and Cellular Biology 26, no. 5 (March 1, 2006): 1691–99. http://dx.doi.org/10.1128/mcb.26.5.1691-1699.2006.

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ABSTRACT The Atm protein kinase is central to the DNA double-strand break response in mammalian cells. After irradiation, dimeric Atm undergoes autophosphorylation at Ser 1981 and dissociates into active monomers. Atm activation is stimulated by expression of the Mre11/Rad50/nibrin complex. Previously, we showed that a C-terminal fragment of nibrin, containing binding sites for both Mre11 and Atm, was sufficient to provide this stimulatory effect in Nijmegen breakage syndrome (NBS) cells. To discriminate whether nibrin's role in Atm activation is to bind and translocate Mre11/Rad50 to the nucleus or to interact directly with Atm, we expressed an Mre11 transgene with a C-terminal NLS sequence in NBS fibroblasts. The Mre11-NLS protein complexed with Rad50, localized to the nucleus in NBS fibroblasts, and associated with chromatin. However, Atm autophosphorylation was not stimulated in cells expressing Mre11-NLS, nor were downstream Atm targets phosphorylated. To determine whether nibrin-Atm interaction is necessary to stimulate Atm activation, we expressed nibrin transgenes lacking the Atm binding domain in NBS fibroblasts. The nibrin ΔAtm protein interacted with Mre11/Rad50; however, Atm autophosphorylation was dramatically reduced after irradiation in NBS cells expressing the nibrin ΔAtm transgenes relative to wild-type nibrin. These results indicate that nibrin plays an active role in Atm activation beyond translocating Mre11/Rad50 to the nucleus and that this function requires nibrin-Atm interaction.
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Desai-Mehta, Ami, Karen M. Cerosaletti, and Patrick Concannon. "Distinct Functional Domains of Nibrin Mediate Mre11 Binding, Focus Formation, and Nuclear Localization." Molecular and Cellular Biology 21, no. 6 (March 15, 2001): 2184–91. http://dx.doi.org/10.1128/mcb.21.6.2184-2191.2001.

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ABSTRACT The inherited chromosomal instability disorder Nijmegen breakage syndrome (NBS) results from truncating mutations in theNBS1 gene, which encodes the protein nibrin. Nibrin is part of a nuclear multiprotein complex that also contains the DNA repair proteins Mre11 and Rad50. Upon irradiation, this complex redistributes within the nucleus, forming distinct foci that have been implicated as sites of DNA repair. In NBS cells, nibrin is absent and Mre11 and Rad50 are cytoplasmic. In this study, the interacting domains on nibrin and Mre11 were mapped using the yeast two-hybrid system and expression of epitope-tagged constructs in NBS fibroblasts. Deletion of the carboxy-terminal 101 amino acids of nibrin eliminated its ability to interact with Mre11 and to complement the radiation sensitivity of NBS cells. However, this truncated form of nibrin could localize to the nucleus and form radiation-inducible foci. Expression of a carboxy-terminal 354-amino-acid fragment of nibrin was sufficient to direct the nuclear localization of nibrin, as well as that of Mre11 and Rad50. Despite providing some partial complementation of the radiation-sensitive phenotype, the nibrin-Mre11-Rad50 complexes in these cells were unable to form foci. These results indicate that nibrin directs not only the nuclear localization of the nibrin-Mre11-Rad50 complexes but also radiation-induced focus formation. However, direct interaction between nibrin and Mre11 is required for normal cellular survival postirradiation. Distinct domains of nibrin are required for each of these functions, focus formation, nuclear localization, and Mre11 interaction.
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Vissinga, Christine S., Tiong C. Yeo, Sarah Warren, James V. Brawley, Jennifer Phillips, Karen Cerosaletti, and Patrick Concannon. "Nuclear Export of NBN Is Required for Normal Cellular Responses to Radiation." Molecular and Cellular Biology 29, no. 4 (December 15, 2008): 1000–1006. http://dx.doi.org/10.1128/mcb.01131-08.

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ABSTRACT Nijmegen breakage syndrome arises from hypomorphic mutations in the NBN gene encoding nibrin, a component of the MRE11/RAD50/nibrin (MRN) complex. In mammalian cells, the MRN complex localizes to the nucleus, where it plays multiple roles in the cellular response to DNA double-strand breaks. In the current study, sequences in mouse nibrin required to direct the nuclear localization of the MRN complex were identified by site-specific mutagenesis. Unexpectedly, nibrin was found to contain both nuclear localizing signal (NLS) sequences and a nuclear export signal (NES) sequence whose functions were confirmed by mutagenesis. Both nuclear import and export sequences were active in vivo. Disruption of either the NLS or NES sequences of nibrin significantly altered the cellular distribution of nibrin and Mre11 and impaired survival after exposure to ionizing radiation. Mutation of the NES sequence in nibrin slowed the turnover of phosphorylated nibrin after irradiation, indicating that nuclear export of nibrin may function, in part, to downregulate posttranslationally modified MRN complex components after DNA damage responses are complete.
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Aracil Avila, Miguel, Antonio Nieto, Adnan Tanovic, Bradley J. Monk, Stanley B. Kaye, Andres Poveda, Thomas J. Herzog, Trilok V. Parekh, Nadia Badri, and Carlos Galmarini. "Exploratory analysis of nibrin in advanced ovarian cancer (AOC) patients treated in the phase III OVA-301 trial." Journal of Clinical Oncology 31, no. 15_suppl (May 20, 2013): 5566. http://dx.doi.org/10.1200/jco.2013.31.15_suppl.5566.

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5566 Background: Nibrin (p95/NBS1) is a protein with an essential function in DNA double-strand break repair by homologous recombination. Therefore, we have investigated its value as a possible biomarker in patients with AOC by immunohistochemistry (IHC). Methods: IHC staining was performed in 138 samples from a subset of patients that have participated in the phase III OVA-301 trial, in which the combination of trabectedin plus pegylated liposomal doxorubicin (PLD) or PLD alone were randomly administered for advanced disease after failure of platinum-based chemotherapy (Monk 2010; Monk 2012). A computerized image analysis system was used to calculate the total percentage of nibrin-positive cells. Nibrin expression was considered as a continuous variable. The analysis of overall response rate (ORR) and progression-free survival (PFS) was based on independent oncologist assessment. Overall survival (OS) was defined from randomization to death/last contact. All the comparisons had an exploratory nature; an alpha cut-off value of 0.05 (two-sided) was established as statistically significant. Results: For PFS, there was a statistically significant correlation between high levels of nibrin and short PFS (HR = 1.014, 95% CI: 1.004-1.024, p=0.0047). Similarly, for OS, there was a statistically significant correlation between high levels of nibrin and worse OS (HR = 1.009, 95% CI: 1.001-1.017, p = 0.0295). A multivariate analysis showed that high levels of nibrin were independently correlated to a worse PFS (HR = 1.012, 95% CI: 1.002-1.022, p = 0.0147) and to a worse OS (HR = 1.010, 95% CI: 1.002-1.018, p=0.0192). After stratification according to platinum-sensitivity, high nibrin showed a significant correlation with lower ORR (ORR = 1.02, 95% CI: 1.01-1.03, p=0.0009), short PFS and OS values only in the platinum-sensitive patients. Conclusions: The results point out the potential importance of nibrin expression in the clinical outcome of patients with AOC. In particular, high protein expression of nibrin seems to be associated with a worse clinical outcome. Prospective clinical trials evaluating the clinical usefulness of this marker with other standard of care treatments are warranted. Clinical trial information: NCT00113607.
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Kracker, S., Y. Bergmann, I. Demuth, P. O. Frappart, G. Hildebrand, R. Christine, Z. Q. Wang, K. Sperling, M. Digweed, and A. Radbruch. "Nibrin functions in Ig class-switch recombination." Proceedings of the National Academy of Sciences 102, no. 5 (January 24, 2005): 1584–89. http://dx.doi.org/10.1073/pnas.0409191102.

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Tanzarella, Caterina, Antonio Antoccia, Emanuela Spadoni, Alessandra di Masi, Vanna Pecile, Eliana Demori, Raymonda Varon, Gian Luigi Marseglia, Luciano Tiepolo, and Paola Maraschio. "Chromosome instability and nibrin protein variants in NBS heterozygotes." European Journal of Human Genetics 11, no. 4 (April 2003): 297–303. http://dx.doi.org/10.1038/sj.ejhg.5200962.

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Abidi, Fizza, Mervyn Hossein, Saima Akram, and Angabeen Anjum. "Nibrin Double Strand Breakage and its Role in Development of Cancers." Journal of the Pakistan Dental Association 29, no. 02 (May 5, 2020): 94–99. http://dx.doi.org/10.25301/jpda.292.94.

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UZUNOGLU, HAKAN, TUGCAN KORAK, EMEL ERGUL, NIHAL UREN, ALI SAZCI, N. ZAFER UTKAN, ERTUĞRUL KARGI, ÇAĞRI TRIYAKI, and OKTAY YIRMIBESOGLU. "Association of the nibrin gene (NBN) variants with breast cancer." Biomedical Reports 4, no. 3 (January 25, 2016): 369–73. http://dx.doi.org/10.3892/br.2016.579.

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Gatei, Magtouf, David Young, Karen M. Cerosaletti, Ami Desai-Mehta, Kevin Spring, Sergei Kozlov, Martin F. Lavin, Richard A. Gatti, Patrick Concannon, and KumKum Khanna. "ATM-dependent phosphorylation of nibrin in response to radiation exposure." Nature Genetics 25, no. 1 (May 2000): 115–19. http://dx.doi.org/10.1038/75508.

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Erdős, Melinda, Beáta Tóth, Pálma Juhász, Mohamed Mahdi, and László Maródi. "Nijmegen Breakage syndrome." Orvosi Hetilap 151, no. 16 (April 1, 2010): 665–73. http://dx.doi.org/10.1556/oh.2010.28851.

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A Nijmegen–Breakage-szindróma ritka, autoszomális recesszív öröklődésű kórkép, amelyre súlyos kombinált immundeficientia, visszatérő sinopulmonalis fertőzések, a kromoszómainstabilitás és az ionizáló sugárzással szembeni hiperszenzitivitás miatt a malignus betegségek gyakoribb előfordulása, fejlődési rendellenességek, madárarc, progresszív microcephalia, valamint növekedési és mentális retardáció jellemző. A betegség hátterében a DNS-repair-mechanizmusokban fontos szerepet játszó nibrin nevű protein kódolásáért felelős NBS1 gén mutációja áll. A közleményben a szerzők két esetismertetés kapcsán bemutatják a betegség klinikumát, a jellemző laboratóriumi leleteket, és összefoglalják a kórkép molekuláris patomechanizmusával kapcsolatos ismereteket, valamint a kezelés lehetőségeit.
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Dissertations / Theses on the topic "Nibrin"

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Yeo, Tiong Chia. "Nijmegen breakage syndrome : role of nibrin in antigen receptor gene rearrangement and cellular responses to ionizing radiation /." Thesis, Connect to this title online; UW restricted, 2000. http://hdl.handle.net/1773/8340.

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Wessendorf, Petra [Verfasser]. "Deficiency of the DNA repair protein nibrin increases the basal but not the radiation induced mutation frequency in vivo / Petra Wessendorf." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2015. http://d-nb.info/1071088564/34.

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Thierfelder, Nadja Katherina. "Untersuchungen zur Apoptoseinduktion in lymphoblastoiden Zellen von Patienten mit Nijmegen-Breakage-Syndrom." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2006. http://dx.doi.org/10.18452/15496.

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Das Nijmegen-Breakage-Syndrom (NBS) ist ein autosomal-rezessiv vererbtes Chromosomenbruchsyndrom, dem in > 90% der Patienten eine 5bp-Deletion im Nbs1-Gen zugrundeliegt. Klinisches Hauptmerkmal ist ein stark erhöhtes Krebsrisiko, insbesondere für B-Zell-Lymphome. Bereits bekannt ist die Funktion des entsprechenden Genprodukts, Nibrin, bei den für die Krebsprävention wichtigen Mechanismen der DNA-Reparatur und der Zellzykluskontrolle. Daneben spielt die Apoptose eine essentielle Rolle bei der Krebsentstehung. Zu untersuchen ob Nibrin auch hier Funktionen übernimmt war Gegenstand dieser Arbeit. Eine Störung der Apoptose könnte dabei mitverantwortlich für das hohe Krebsrisiko der NBS-Patienten sein. Kern der Arbeit war die Untersuchung von NBS-B-Lymphozyten hinsichtlich ihrer Fähigkeit, nach einer DNA-Schädigung die Apoptose zu induzieren. Hierzu wurde in den entsprechenden Zellen mittels Bleomycin der Apoptoseprozess ausgelöst und die prozentualen Apoptoseraten durchflusszytometrisch bestimmt. Die Mehrheit der NBS-Zelllinien zeigte eine Störung in der Apoptoseinduktion im Sinne signifikant verminderter Apoptoseraten. Dies weist auf eine Funktion des Nibrins bei der Induktion der Apoptose hin. Andere NBS-Zelllinien zeigten normale Apoptoseindices. Dies könnte auf dem individuellen genetischen Hintergrund der Zellen beruhen, der auch für die erhebliche klinische Variabilität des Krankheitsbildes verantwortlich ist. Eine Korrelation der Apoptoseraten mit der Krebsinzidenz zeigte, dass alle Patienten mit reduzierten Apoptoseraten bereits Lymphome entwickelt hatten, während Patienten mit normalen Apoptoseindices bisher keine Lymphome aufwiesen. Möglicherweise gibt es also generell zwei Gruppen von NBS-Patienten - Patienten mit höherem und mit niedrigerem Entartungsrisiko, wobei eine verminderte Apoptoseinduktion als Risikofaktor für Krebs angesehen werden könnte.
The human genetic disorder, Nijmegen-Breakage-Syndrome (NBS), is characterised by an in increased risk for cancer, particularly B-cell-lymphoma. The Nbs1-gene codes for a protein, Nibrin, involved in the processing/repair of DNA double strand breaks and in cell cycle checkpoints - mechanisms relevant for cancer-prevention. As a third mechanism, apoptosis is important in preventing cancer. To evaluate whether Nibrin plays a role in this process was the aim of this study. Failure of apoptosis-induction could be another factor responsible for the high cancer risk in NBS. For this purpose we examined a set of NBS-B-cell-lines for their capacitiy to enter into apoptosis after a DNA-damaging treatment with Bleomycin. The majority of NBS-cell-lines showed a deficiency in apoptosis-induction. This may indicate a function of Nibrin in mechanisms of apoptosis-regulation. Some NBS-cell-lines showed a proficient apoptotic response, though. The reason may be found in the variable genetic background of the cell lines, also responsible for the high clinical variability of the disease. Correlation of apoptosis rates with cancer incidence showed that all patients deficient in apoptosis had already developed B-cell-lymphoma, whereas patients with normal rates had not developed lymphoma so far. Possibly there are two groups of NBS-patients- patients with higher and with lower risk of malignancy, with reduced apoptotic rates being a risk-factor for the development of cancer in NBS.
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Deming, Brenda Boon. "Evaluating the role of lymphocyte radiosensitivity and variants in double-strand break repair genes, checkpoint kinase 2 (CHEK2) and nibrin (NBN), in the predisposition to prostate cancer : a dissertation /." San Antonio : UTHSC, 2007. http://proquest.umi.com/pqdweb?did=1425298611&sid=1&Fmt=2&clientId=70986&RQT=309&VName=PQD.

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Cheng, Ka Hei. "Nibiru." Bowling Green State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1586149280428446.

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Grigorescu, Curumlis Ioana Cristina. "Etude structurale et mécanique d'un composite à matrice métallique NiBSi-VC. Comportement en frottement. Contraintes résiduelles." Toulouse, INPT, 1994. http://www.theses.fr/1994INPT030G.

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Des composites matrice base nickel (nibsi), renforces par des particules de carbure de vanadium (vc) ont ete prepares par frittage. L'etude metallurgique et structurale a ete menee par microscopie optique (mo), microscopie electronique en balayage et en transmission avec analyse locale (meb-eds+wds, met-eds). Ces examens ont montre l'existence d'un reseau riche en bore englobant les particules des carbures, une forte diffusion du vanadium dans la structure nickel cfc et la bonne adhesion dans l'interface particule vc-matrice, bien que siege d'une deformation plastique. Les essais tribologiques ont ete effectues en utilisant des montages pion-anneau et pion-disque, suivis par des analyses morphologiques et chimiques des surfaces usees. En fonction du materiau du deuxieme corps, dans le couple tribologique des differents mecanismes d'usure se sont developpes. Ainsi, le contact avec l'alumine-zircone frittee favorise l'adhesion entre les deux surfaces. Par contre, l'acier, employe comme deuxieme corps, induit un mecanisme d'usure a plusieurs etapes: compression du reseau riche en b - extrusion de la matrice riche en ni - fracture du reseau riche en b - fluage du volume de materiau, la derniere etape s'accompagnant des chutes transitoires du coefficient de frottement, traitees comme evenements statistiques. Ce comportement est commun pour tous les materiaux etudies et une relation logarithmique a ete etablie entre l'intensite d'usure et le nombre cumule des evenements. L'etat de contraintes residuelles introduites par le frottement a ete etudie par diffraction de rayon x. La methode classique (dite de sinus carre psi) ne permettant pas l'interpretation des mesures, une methode originale a ete utilisee. Il s'agit de comparer la deformation superficielle produite par le test d'usure a celle produite par un essai statique de flexion bi-axiale
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Hoffman, Bruce. "Biology and use of nibbi heteropsis flexuosa (ARACEAE) the source of an aerial root fiber product in Guyana." FIU Digital Commons, 1997. http://digitalcommons.fiu.edu/etd/2716.

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The aerial roots of Heteropsis flexuosa (Kunth) Bunting, a hemi-epiphyte, are harvested by indigenous communities in Guyana for a developing wicker furniture market. Nibbi roots have potential as a sustainably harvested product, but there is little data to guide management. I examined nibbi biology, harvest response, product yield and use at several forest sites. H. flexuosa is a relatively abundant plant and 35% of trees (≥ 10 cm dbh) in plots were hosts. Stems exhibited mean growth rates of 1-3 cm per month. Aerial roots grew a mean 156 cm per month and some reached maturity within 6 months. With present methods, harvest does not decimate populations because 97% of colonized trees possess few harvestable roots. But, only 28% of cut roots re-generated in experiments. For indigenous harvesters at Manawarin village, nibbi harvesting is a primary source of cash income and is important in daily subsistence.
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Fegadel, Averi Rebekah. "Juvenile and Adult Involvement in Double Parricide and Familicide in the U.S.: An Empirical Analysis of 20 Years of Data." Scholar Commons, 2014. https://scholarcommons.usf.edu/etd/5013.

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The killing of parents and stepparents by biological and stepchildren is a rare event. Incidents involving multiple parricide victims and/or multiple parricide offenders are an even rarer occurrence. The majority of studies on parricide involve a single victim and single offender. Using the National Incident-Based Reporting System (NIBRS), this study identified 603 single-victim, single-offender incidents, 22 single-victim, multiple-offender incidents, 60 single-offender double parricide incidents, 17 multiple-offender double parricide incidents, and 15 familicide incidents over the 20 year period 1990 to 2010. Univariate and bivariate analyses examined parricidal incidents involving single or multiple offenders and single or multiple victims with the aim of investigating juvenile and adult involvement in double parricide and familicide. Frequencies reported include victim, offender, and incident characteristics for all types of parricide incidents. Consistent with prior research on single-victim, single-offender parricide, the results indicated that the typical parricide offender was a white male approximately 30 years of age. A firearm predominated as the weapon of choice for all parricide incidents; however, when a biological mother was one of the victims, the offender(s) used more diverse methods. When multiple offenders were involved in double parricides, however, the offenders tended to be younger and were more likely to include a female accomplice. Only one case of familicide involved a female offender, and none of the familicide incidents involved multiple offenders. Study limitations and implications for prevention are also discussed.
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Shubair, Asma. "Tuning the Selectivity of Bimetallic NiBi Catalysts for Glycerol Electrooxidation Into Value-Added Products." Thesis, Université d'Ottawa / University of Ottawa, 2021. http://hdl.handle.net/10393/41882.

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In the process of biodiesel production, glycerol is produced as a byproduct in bulk amounts. The amount of glycerol supplied is larger than its demand thus stockpiling and acting as waste. As a solution, glycerol which is a highly functionalized molecule must be converted to value-added products. Several catalytic routes were thoroughly investigated including, hydrogenolysis, dehydration, pyrolysis, transesterification, etherification, carboxylation and electro-oxidation. All of these routes produce products of high economic interests. However, electro-oxidation seems to be the most promising as it runs under milder conditions and the selectivity may be easily tuned by varying the applied potential and the catalyst type. In addition, the electrical energy required may be provided by renewable energy sources. Some of the value-added products that may be produced by electrooxidation listed from highest economic value to lowest are glyceraldehyde, dihydroxyacetone, lactate, glycerate, tartronate (C₃ products) > mesooxalate, glycolate, oxalate (C₂ products) > and formate (C₁ products). Noble metals (Pt, Pd and Au) are considered to be the best for alcohol electrooxidation reactions as they present high electrocatalytic activity and selectivity. To date, research is focused on enhancing the activity and selectivity of noble metals by changing the nanoparticles morphology and adding adatoms/promoters/supports. On the other hand, these metals are non-abundant and expensive which limits their actual use in the industry. For this reason, non-noble metals (Ni and Co) have gained interest as potential alternatives. Particularly, nickel has proved to have significant activity, high durability and anti-poisoning capability for GEOR. A few studies presented enhancement in catalytic performance by varying the nanoparticles structure and adjusting the surface with a bimetallic promoter. However, there is still so much space for further research to enhance the catalytic performance and selectivity of Ni-based materials. In this thesis, carbon supported bimetallic NiₓBi₁₀₀₋ₓ [x= 100, 95, 90, 80, and 50 at.%] and Ni₉₅Bi₅/C mixed with small amounts of metal oxides (CeO₂, SnO₂ and Sb₂O₃:SnO₂) were studied for GEOR application. All catalysts were synthesized by facile sodium borohydride reduction method which can be easily scaled up. Transmission electron microscopy (TEM) and electron dispersive x-ray spectroscopy (EDS) techniques were implemented to gather physical characterizations of the as-synthesized bimetallic NiBi/C catalyst. Different electrochemical tests such as; cyclic voltammetry, linear sweep voltammetry and chronoamperometry were conducted using a conventional three electrode electrochemical cell and a potentiostat to get insight on the electrochemical performance of all catalysts. Finally, quantitative product analysis was generated by running continuous glycerol electrolysis experiments in a 25 cm2 cell accompanied by HPLC analysis. The nanoparticles size of Ni₉₅Bi₅/C was ≥6nm as determined by TEM images. Results indicated that tuning the nanoparticles size has an impact on both activity and selectivity of bimetallic carbon supported NiBi catalyst. For instance, the NiBi/C (≥6nm NP size) synthesized herein had 40% higher selectivity to C₃ products compared to NiBi/C (≤3nm NP size) reported in literature. Additionally, the selectivity of Ni-based catalysts to C₃ products were largely enhanced by developing bimetallic carbon supported NiBi catalysts of different Ni:Bi atomic ratios and adding metal oxides (CeO₂, SnO₂ and Sb₂O₃.SnO₂) to NiBi/C catalysts. Results indicate that addition of metal oxides greatly enhanced selectivity to C₃ products in the following order; Ni₉₅Bi₅/C-ATO (100%)> Ni/C-ATO (99.17%)> Ni₉₅Bi₅/C-ceria (98.05%)> Ni/C-ceria (78.29%)> Ni₉₅Bi₅/C (41.43%)> Ni/C (34.57%). However, the activity of Ni₉₅Bi₅/C-X [X=CeO₂, SnO₂, and Sb₂O₃:SnO₂] was lower than that of Ni₉₅Bi₅/C and Ni/C which was explained by the strong metal support interactions between metal oxides and nickel.
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Petraglia, Elizabeth Ellen. "Estimating County-Level Aggravated Assault Rates by Combining Data from the National Crime Victimization Survey (NCVS) and the National Incident-Based Reporting System (NIBRS)." The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1439027433.

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Books on the topic "Nibrin"

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Lubbādah, Hānī. Qāmūs al-Nibrās: Injilīzī-ʻArabī = Al-Nibras : English-Arabic dictionary. ʻAmmān: Dār al-Nibrās al-ʻArabī, 1993.

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Nah kamu nibriyo, nah g̲h̲amu nibriyo. Kanḍiyāro: Roshnī Pablīkeshani, 1993.

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al-Nibrās: Riwāyah. al-Shāriqah: Dāʼirat al-Thaqāfah wa-al-Iʻlām, Ḥukūmat al-Shāriqah, 2003.

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Last train to Nibroc. New York: Dramatists Play Service, 2000.

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Tasaro. Nibiru dan kesatria Atlantis. Solo: Tiga Serangkai, 2010.

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Colen, Dominique. Ferdinand Hart Nibbrig: 1866-1915. Zwolle: Waanders, 1996.

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Shūshishitsu, Sumitomo. Nennen shoyōtome niban, sanban. Kyōto-shi: Shibunkaku Shuppan, 1986.

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Carla, Carotenuto, ed. Gino Nibbi, marchigiano d'Australia. Pesaro: Metauro, 2008.

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Nibbi, Gino. Gino Nibbi, marchigiano d'Australia. Pesaro: Metauro, 2008.

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Nibbi, Gino. Gino Nibbi, marchigiano d'Australia. Pesaro: Metauro, 2008.

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Book chapters on the topic "Nibrin"

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Froehlich, Stephan J., Carlo A. Lackerbauer, Guenter Rudolph, Jan Rémi, Soheyl Noachtar, Werner J. Heppt, Annette Cryer, et al. "Nibrin/NBS1 Deficiency." In Encyclopedia of Molecular Mechanisms of Disease, 1477. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6440.

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Kaneko, T., and T. Kanomata. "5.1.12 NiBr2." In Magnetic Properties of d-Elements, Alloys and Compounds Under Pressure, 250–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-41834-1_137.

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Holze, Rudolf. "Ionic conductance of NiBr2." In Electrochemistry, 1595. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-49251-2_1423.

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Heineke, Hans J., Hans-Ulrich Bartsch, Jan Sbresny, and Udo Müller. "Das Methodenmanagementsystem im Niedersächsischen Bodeninformationssystem NIBIS." In Bodenmanagement, 59–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56284-6_3.

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Blaeser, Kimberly. "A cosmology of nibi." In Geopoetics in Practice, 29–47. Abingdon, Oxon ; New York, NY : Routledge, 2020. | Series: Routledge research in culture, space and identity: Routledge, 2019. http://dx.doi.org/10.4324/9780429032202-4.

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Craft, Aimée, Deborah McGregor, Rayanna Seymour-Hourie, and Sue Chiblow. "Decolonizing Anishinaabe nibi inaakonigewin and gikendaasowin research." In Decolonizing Law, 17–33. Milton Park, Abingdon, Oxon ; New York, NY : Routledge, 2021. | Series: Indigenous peoples and the law: Routledge, 2021. http://dx.doi.org/10.4324/9781003161387-3.

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Addington, Lynn A. "Research Adventures with “Kinda Big” Data: Using NIBRS to Study Crime." In Envisioning Criminology, 157–63. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15868-6_16.

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Addington, Lynn A. "Studying the Crime Problem with NIBRS Data: Current Uses and Future Trends." In Handbooks of Sociology and Social Research, 23–42. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-0245-0_2.

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Addington, Lynn A. "NIBRS as the New Normal: What Fully Incident-Based Crime Data Mean for Researchers." In Handbooks of Sociology and Social Research, 21–33. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-20779-3_2.

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"Nibrin." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics, 1345. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_11397.

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Conference papers on the topic "Nibrin"

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Bahgat, Ahmed, Abdelrahman Adel Mahmoud, Said Elmi Ahmed, Kamran Ali, R. A. Shakoor, Ramazan Kahraman, and F. M. Montemor. "Synthesis and Properties of Novel NiBAlN Nanocomposite Coatings." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2018. http://dx.doi.org/10.5339/qfarc.2018.eepd560.

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Benahmed, A., and R. Alami. "Development of a new radiation measuring instrument for the Column scanning technique - NibraS." In 2013 3rd International Conference on Advancements in Nuclear Instrumentation, Measurement Methods and their Applications (ANIMMA). IEEE, 2013. http://dx.doi.org/10.1109/animma.2013.6727873.

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Shihab, Sufyan, Shaikhan Khadhuri, Salim Busaidy, and Solenn Rawnsley Bettembourg. "Real-Time Operations Portal (Nibras): Another Step En Route to a Smart Field Management." In SPE Middle East Oil and Gas Show and Conference. Society of Petroleum Engineers, 2011. http://dx.doi.org/10.2118/141771-ms.

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Gaffney, P. J., L. J. Creighton, A. Curry, B. MacMahon, and R. Thorpe. "MONOCLONAL ANTIBODIES OF THE IgM AND IgG CLASS SPECIFIC FOR CROSSLINKED FIBRIN DEGRADATION PRODUCTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643651.

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Abstract:
Monoclonal antibodies (mabs) to crosslinked fibrin degradation products (XL-FDP) having the general formula D/Y[X]nY/D (known as X-oligomer) and D-D (known as D dimer) have been raised in balb/C mice by both a novel mtrasplenic and a conventional subcutaneous route of immunisation and by combinations of both these procedures. Mabs to X-oligomers (NIBn 52 and NIBn 123) obtained by an intrasplenic procedure have been demonstrated to crossreact only with X-oligomer in a 2-site ELISA procedure and not with D dimer or whole fibrinogen and have been shown to be of value m the examination of clinical material obtained from patients with various types of thrombosis and have also been useful in monitoring the efficacy of thrombolytic therapy. The X-oligomer mabs are immunoglobulins of the M class. It was demonstrated that their unique specificity for conformational epitopes on the large X-oligomer fragments does not reside in the IgM structure since alterative immunisation procedures have been used to generate mabs of the IgG class which have the same specificity. Using immunoglobulin class switching in culture rather than during immunisation was suggested by certain cell lines which produced both IgM and IgG specific for X-oligomer. This latter point needs rigorous validation.Immunoglobulin G type mabs to highly purified D dimer were raised by conventional subcutaneous immunisation of balb/C mice. One of these, NIBn-11, was found to crossreact with PVC-immobilised X-oligomer and D dimer but not with fibrinogen. However NIBn-11 did not bind to D dimer in a 2-site ELISA procedure while crossreactmg quite avidly with X-oligomer. This suggests that the D dimer epitope to which NIBn-11 is directed is expressed in some conformations and not m others and that these conformations are always expressed in the complex X-oligomer group of fragments. These mabs, whilst of value in measuring certain unique fibrin fragments m plasma, are useful in the epitope mapping of fibrinogen/fibrin and their plasmm-mediated
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Korobov, Yury, Alexander Vopneruk, Alexander Kotelnikov, Yulia Khudorozhkova, Sergey Burov, and Prabu Balu. "Structure analysis of laser deposited NiBSi-WC coatings on a Cu-Cr-Zr substrate." In PROCEEDINGS OF THE INTERNATIONAL CONFERENCE ON ADVANCED MATERIALS WITH HIERARCHICAL STRUCTURE FOR NEW TECHNOLOGIES AND RELIABLE STRUCTURES 2017 (AMHS’17). Author(s), 2017. http://dx.doi.org/10.1063/1.5013779.

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Giacomo, Anna M. Di, Alessia Covre, Francesca Finotello, Dietmar Rieder, Luca Sigalotti, Carolina Fazio, Ornella Cutaia, et al. "Abstract CT059: Epigenetic tumor remodelling to improve the efficacy of immune checkpoint blockade: the NIBIT-M4 clinical trial." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-ct059.

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Peng, Grace. "“Funding opportunities at the National Institutes of Health (NIH) and the National Institute of Biomedical Imaging and Bioengineering (NIBIB)”." In Engineering Conference (BSEC): Exploring the Intersections of Interdisciplinary Biomedical Research. IEEE, 2009. http://dx.doi.org/10.1109/bsec.2009.5090471.

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Di Giacomo, Anna Maria, Luana Calabrò, Riccardo Danielli, Monica Valente, Elisabetta Gambale, Sandra Coral, Giovanni Amato, et al. "Abstract CT270: A randomized, multi-center, phase II study of nivolumab combined with ipilimumab and guadecitabine or nivolumab combined with ipilimumab in melanoma and NSCLC patients resistant to anti-PD-1/-PD-L1: The NIBIT-ML1 Study." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-ct270.

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