Relevant bibliographies by topics / Nicotine

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Nicotine'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Nicotine"

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Zenkner, Fernanda Fleig, Márcia Margis-Pinheiro, and Alexandro Cagliari. "Nicotine Biosynthesis in Nicotiana: A Metabolic Overview." Tobacco Science 56, no. 1 (2019): 1–9. http://dx.doi.org/10.3381/18-063.

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Alkaloids are important compounds found in Nicotiana plants, essential in plant defense against herbivores. The main alkaloid of Nicotiana tabacum, nicotine, is produced in roots and translocated to the leaves. Nicotine is formed by a pyrrolidine and a pyridine ring in a process involving several enzymes. The pyridine ring of nicotine is derived from nicotinic acid, whereas the pyrrolidine ring originates from polyamine putrescine metabolism. After synthesis in root cortical cells, a set of transporters is known to transport nicotine upward to the aerial part and store it in leaf vacuoles. Mor
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Page, Stephen J., Mingyan Zhu, and Suzanne M. Appleyard. "Effects of acute and chronic nicotine on catecholamine neurons of the nucleus of the solitary tract." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 316, no. 1 (2019): R38—R49. http://dx.doi.org/10.1152/ajpregu.00344.2017.

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Nicotine is an addictive drug that has broad effects throughout the brain. One site of action is the nucleus of the solitary tract (NTS), where nicotine initiates a stress response and modulates cardiovascular and gastric function through nicotinic acetylcholine receptors (nAChRs). Catecholamine (CA) neurons in the NTS influence stress and gastric and cardiovascular reflexes, making them potential mediators of nicotine’s effects; however nicotine’s effect on these neurons is unknown. Here, we determined nicotine’s actions on NTS-CA neurons by use of patch-clamp techniques in brain slices from
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Liu, Zhi, Yo Otsu, Cristina Vasuta, Hiroyuki Nawa, and Timothy H. Murphy. "Action-Potential-Independent GABAergic Tone Mediated by Nicotinic Stimulation of Immature Striatal Miniature Synaptic Transmission." Journal of Neurophysiology 98, no. 2 (2007): 581–93. http://dx.doi.org/10.1152/jn.00768.2006.

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Stimulation of presynaptic nicotinic acetylcholine receptors (nAChRs) increases the frequency of miniature excitatory synaptic activity (mEPSCs) to a point where they can promote cell firing in hippocampal CA3 neurons. We have evaluated whether nicotine regulation of miniature synaptic activity can be extended to inhibitory transmission onto striatal medium spiny projection neurons (MSNs) in acute brain slices. Bath application of micromolar nicotine typically induced 12-fold increases in the frequency of miniature inhibitory synaptic currents (mIPSCs). Little effect was observed on the amplit
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DeVito, Elise E., Kevin P. Jensen, Stephanie S. O’Malley, et al. "Modulation of “Protective” Nicotine Perception and Use Profile by Flavorants: Preliminary Findings in E-cigarettes." Nicotine & Tobacco Research 22, no. 5 (2019): 771–81. http://dx.doi.org/10.1093/ntr/ntz057.

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Abstract Introduction Characterizing flavors are widely available in e-cigarettes and motivate initiation and continued use. Flavors may enhance appeal and facilitate development of addiction to tobacco products through modulation of tobacco products’ reinforcing or aversive actions. Palatable flavors (eg, fruit) may increase appeal through primary reinforcing properties. Menthol’s cooling and anesthetic effects may increase appeal by counteracting nicotine’s aversive effects. Genetics provide a method for modeling individual differences in sensitivity to nicotine’s effects. A common polymorph
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Sansone, Luigi, Francesca Milani, Riccardo Fabrizi, et al. "Nicotine: From Discovery to Biological Effects." International Journal of Molecular Sciences 24, no. 19 (2023): 14570. http://dx.doi.org/10.3390/ijms241914570.

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Nicotine, the primary psychoactive agent in tobacco leaves, has led to the widespread use of tobacco, with over one billion smokers globally. This article provides a historical overview of tobacco and discusses tobacco dependence, as well as the biological effects induced by nicotine on mammalian cells. Nicotine induces various biological effects, such as neoangiogenesis, cell division, and proliferation, and it affects neural and non-neural cells through specific pathways downstream of nicotinic receptors (nAChRs). Specific effects mediated by α7 nAChRs are highlighted. Nicotine is highly add
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Grieder, Taryn E., Morgane Besson, Geith Maal-Bared, Stéphanie Pons, Uwe Maskos та Derek van der Kooy. "β2* nAChRs on VTA dopamine and GABA neurons separately mediate nicotine aversion and reward". Proceedings of the National Academy of Sciences 116, № 51 (2019): 25968–73. http://dx.doi.org/10.1073/pnas.1908724116.

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Evidence shows that the neurotransmitter dopamine mediates the rewarding effects of nicotine and other drugs of abuse, while nondopaminergic neural substrates mediate the negative motivational effects. β2* nicotinic acetylcholine receptors (nAChR) are necessary and sufficient for the experience of both nicotine reward and aversion in an intra-VTA (ventral tegmental area) self-administration paradigm. We selectively reexpressed β2* nAChRs in VTA dopamine or VTA γ-amino-butyric acid (GABA) neurons in β2−/−mice to double-dissociate the aversive and rewarding conditioned responses to nicotine in n
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Kedmi, Merav, Arthur L. Beaudet та Avi Orr-Urtreger. "Mice lacking neuronal nicotinic acetylcholine receptor β4-subunit and mice lacking both α5- and β4-subunits are highly resistant to nicotine-induced seizures". Physiological Genomics 17, № 2 (2004): 221–29. http://dx.doi.org/10.1152/physiolgenomics.00202.2003.

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Nicotine, the main addictive component of tobacco, evokes a wide range of dose-dependent behaviors in rodents, and when administrated in high doses, it can induce clonic-tonic seizures. Nicotine acts through the nicotinic acetylcholine receptors (nAChRs). Mutations in the human α4- and the β2-nAChR subunit genes cause autosomal dominant nocturnal frontal lobe epilepsy. Using transgenic mice with mutations in nAChR subunits, it was demonstrated previously that the α4-, α5-, and α7-subunits are involved in nicotine-induced seizures. To examine the possibility that the β4-subunit is also involved
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Kizawa, Yasuo, Michiko Shinkai, and Issei Takayanagi. "Effects of chronic nicotine treatment on nicotinic receptors in the rabbit urinary bladder." Canadian Journal of Physiology and Pharmacology 68, no. 1 (1990): 99–103. http://dx.doi.org/10.1139/y90-015.

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Nicotine induced a phasic contraction in the rabbit urinary bladder. The response was abolished by hexamethonium and partially reduced by atropine and capsaicin. Simultaneous atropine and capsaicin treatment did not abolish the contraction. These findings suggest that the response to nicotine is due to acetylcholine, tachykinins, and unknown mediator release. In contrast, nicotine-induced contraction diminished following the chronic nicotine treatment without a change of its pharmacological properties. These results suggest the possibility that chronic nicotine treatment causes a decrease in n
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Bhanu Prakash G, Rajagopalan Vijayaraghavan, Senthilkumar Sivanesan, and Madhankumar Swaminathan. "A study on the agents that reduces the nicotine induced nicotinic receptor density in wistar rats." International Journal of Research in Pharmaceutical Sciences 12, no. 1 (2021): 430–35. http://dx.doi.org/10.26452/ijrps.v12i1.4074.

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The most important substance causing addiction towards cigarette is nicotine. Nicotine abstinence causes withdrawal symptoms in smokers. It is not just nicotine, along with it is the upregulation of nicotinic receptor density (NRD) that leads to addiction. All together makes nicotine deaddiction the most difficult aspect. Nicotine receptor density increases as long as the person is exposed to nicotine. When once the NRD is initiated by nicotine, later though you stop smoking, the increased nicotine receptors create an urge to smoke. Hence the person feels to smoke for satisfying the nicotine r
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Wongtrakool, Cherry, Susanne Roser-Page, Hilda N. Rivera та Jesse Roman. "Nicotine alters lung branching morphogenesis through the α7 nicotinic acetylcholine receptor". American Journal of Physiology-Lung Cellular and Molecular Physiology 293, № 3 (2007): L611—L618. http://dx.doi.org/10.1152/ajplung.00038.2007.

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There is abundant epidemiological data linking prenatal environmental tobacco smoke with childhood asthma and wheezing, but the underlying molecular and physiological mechanisms that occur in utero to explain this link remain unelucidated. Several studies suggest that nicotine, which traverses the placenta, is a causative agent. Therefore, we studied the effects of nicotine on lung branching morphogenesis using embryonic murine lung explants. We found that the expression of α7 nicotinic acetylcholine receptors, which mediate many of the biological effects of nicotine, is highest in pseudogland
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Dissertations / Theses on the topic "Nicotine"

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Harrington, Lauriane. "The role of β4-containing nicotinic acetylcholine receptors in nicotine addiction". Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066328/document.

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Le tabac est consommé par environ un milliard de personnes. D'après l'Organisation Mondiale de la Santé, le tabagisme est la première cause évitable de mortalité dans le monde, provocant six millions de morts par an. La nicotine est le composant neuro-actif principal dans le tabac, et exerce ses effets neurologiques via une activation directe des récepteurs nicotiniques de l’acétylcholine (nAChR). Ces récepteurs transmembranaires sont composés de sous-unités alpha, ou alpha plus beta, créant une variété de canaux ioniques ligand-dépendants activés par le neurotransmetteur ACh. Les études génét
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Munhoz, Egberto [UNESP]. "Administração subcrônica de nicotina modifica as respostas neuroendócrina e neuroquímica induzidas pelo teste de natação forçada." Universidade Estadual Paulista (UNESP), 2010. http://hdl.handle.net/11449/104039.

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Made available in DSpace on 2014-06-11T19:32:56Z (GMT). No. of bitstreams: 0 Previous issue date: 2010-12-13Bitstream added on 2014-06-13T18:44:37Z : No. of bitstreams: 1 munhoz_e_dr_arafo.pdf: 3085541 bytes, checksum: f014aca5c4e34e1bd024219270c2acaf (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>O estresse, atualmente, é considerado um fator importante na fisiopatologia de muitos distúrbios psiquiátricos. Embora os efeitos do estresse agudo possam ser contrabalanceados por respostas adaptativas, o estresse intenso, repetido ou prolongado pode eliciar alterações n
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Drasdo, Alison L. "Studies of brain nicotinic acetylchlorine receptors : with respect to nicotine dependence." Thesis, University of Bath, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.332792.

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James, John Randolph. "Evidence That Nicotine Can Acutely Desensitize Central Nicotinic Cholinergic Receptors In Vivo." VCU Scholars Compass, 1992. http://scholarscompass.vcu.edu/etd/3935.

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Current concepts concerning nicotine's central nervous system (CNS) mechanism(s) of action suggest that this drug is producing its effects via an interaction at nicotiniccholinergic receptors (nAChRs) which open a membrane cation channel. Following initial opening of the channel, nicotine appears to induce a rapid desensitization of the nAChRs, closing the channel and resulting in a cessation of nicotine's effects. Research presented here will provide evidence of this secondary desensitization process in vivo by demonstrating nicotine's ability to induce acute tolerance in the discriminative s
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Jones, Jeb. "Nicotine and responding maintained by conditioned reinforcers effects of two nicotinic antagonists /." [Gainesville, Fla.] : University of Florida, 2009. http://purl.fcla.edu/fcla/etd/UFE0041268.

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Larsen, James D. "Nicotinic Acetylcholine Receptor Dependent Effects of Nicotine on HEK293T and HBO Cells." VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5701.

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T2R receptors are the classical bitter taste receptors which detect and transduce bitter taste in a subset of taste receptor cells (TRCs). The TRPM5-dependent T2Rs are G-protein coupled receptors (GPCRs) and are linked to G protein, gustducin to initiate an intracellular signaling cascade for the transduction of bitter tastants. Nicotine is bitter. However, at present the transduction mechanisms for the detection of nicotine in are poorly understood. Previous studies from our laboratory using TRPM5 knockout (KO) mice demonstrated that the T2R pathway is insufficient in explaining the taste per
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DiMaggio, Stassi. "Development of Novel Nicotinic Receptor Mediated Therapeutic Agents: Synthesis and Biological Evaluation of Novel Epibatidine Analogs and the First Total Synthesis of Anabasamine and Related Analogs." ScholarWorks@UNO, 2003. http://scholarworks.uno.edu/td/40.

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In an effort to search for a more selective, less toxic neuronal nicotinic acetylcholine receptor analgesic agent in comparison to epibatidine, a series of analogs with hybrid structures of epibatidine and ABT-594 were designed and synthesized. The 1-(pyridyloxymethyl)-7-azabicyclo[2.2.1]heptane ring systems were furnished via an intramolecular cyclization from a trans 1, 4 disubstitituted amino-cyclohexane derivative. The functionalized cyclohexane ring was formed via a [4+2] Diels-Alder cyclization reaction between the acetamidoacrylate and Danishefsky's diene. These 1- (pyridyloxymethyl)-7-
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Bowker, Katharine. "The effectiveness of nicotine replacement therapy during pregnancy : investigating the role of nicotine substitution, nicotine metabolism and pregnant smokers' experiences." Thesis, University of Nottingham, 2016. http://eprints.nottingham.ac.uk/32138/.

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Nicotine replacement therapy (NRT) is effective in non-pregnant populations at assisting smoking cessation, but there is no evidence that NRT can help pregnant smokers to stop. Nicotine metabolism increases during pregnancy, and so the nicotine dose delivered by NRT could be insufficient for ameliorating withdrawal symptoms. However, little is known about the level of nicotine substitution provided by NRT or the pattern of nicotine metabolism during pregnancy. Also low adherence to NRT may explain why NRT does not appear to be effective. The overall aim of this thesis was to increase understan
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Vallaster, Markus Parzival. "Intergenerational Effects of Nicotine in an Animal Model of Paternal Nicotine Exposure." eScholarship@UMMS, 2017. https://escholarship.umassmed.edu/gsbs_diss/913.

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Environmental conditions imposed onto organisms during certain phases of their life cycles such as embryogenesis or puberty can not only impact the organisms’ own health, but also affect subsequent generations. The underlying mechanisms causing intergenerational phenotypes are not encoded in the genome, but the result of reversible epigenetic modifications. This work investigates in a mouse model the impact of paternal nicotine exposure on the next generation regarding addictive behavior modulation, metabolic changes, and molecular mechanisms. It provides evidence that male offspring from nico
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Vallaster, Markus Parzival. "Intergenerational Effects of Nicotine in an Animal Model of Paternal Nicotine Exposure." eScholarship@UMMS, 2008. http://escholarship.umassmed.edu/gsbs_diss/913.

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Environmental conditions imposed onto organisms during certain phases of their life cycles such as embryogenesis or puberty can not only impact the organisms’ own health, but also affect subsequent generations. The underlying mechanisms causing intergenerational phenotypes are not encoded in the genome, but the result of reversible epigenetic modifications. This work investigates in a mouse model the impact of paternal nicotine exposure on the next generation regarding addictive behavior modulation, metabolic changes, and molecular mechanisms. It provides evidence that male offspring from nico
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Books on the topic "Nicotine"

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Price, Sean. Nicotine. Chelsea House, 2008.

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Wagner, Heather Lehr. Nicotine. Edited by Triggle D. J. Chelsea House Publishers, 2003.

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Henningfield, Jack E., Edythe D. London, and Sakire Pogun, eds. Nicotine Psychopharmacology. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-69248-5.

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National Institute on Drug Abuse, ed. Nicotine addiction. National Institute on Drug Abuse, U.S. Dept. of Health and Human Services, National Institutes of Health, 1998.

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DeAngelis, Gina. Nicotine & cigarettes. Chelsea House Publishers, 2000.

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Hofmann, F. B. Nicotine Psychopharmacology. Springer Berlin Heidelberg, 2009.

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National Institute on Drug Abuse., ed. Nicotine addiction. National Institute on Drug Abuse, U.S. Dept. of Health and Human Services, National Institutes of Health, 1998.

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Yann. Nicotine goudron. L'Echo des savanes, 1990.

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National Institute on Drug Abuse., ed. Nicotine addiction. National Institute on Drug Abuse, U.S. Dept. of Health and Human Services, National Institutes of Health, 1998.

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Hasan, Heather. Caffeine and nicotine. Rosen Pub., 2009.

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Book chapters on the topic "Nicotine"

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Andrews, Anne M., Greg A. Gerhardt, Lynette C. Daws, et al. "Nicotine." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_303.

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Davies, Marilyn. "Nicotine." In Encyclopedia of Women’s Health. Springer US, 2004. http://dx.doi.org/10.1007/978-0-306-48113-0_298.

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Sershen, Henry. "Nicotine." In Alterations of Metabolites in the Nervous System. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4757-6740-7_10.

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Nakajima, Motohiro, and Mustafa al’Absi. "Nicotine." In Encyclopedia of Behavioral Medicine. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_268.

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Tiro, Jasmin, Simon J. Craddock Lee, Steven E. Lipshultz, et al. "Nicotine." In Encyclopedia of Behavioral Medicine. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_268.

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Clarke, Paul B. S. "Nicotine." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-36172-2_303.

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Henningfield, J. E., R. M. Keenan, and P. B. S. Clarke. "Nicotine." In Pharmacological Aspects of Drug Dependence. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60963-3_8.

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Hsia, Stephanie L., Anna K. Mischel, and Arthur L. Brody. "Nicotine." In Absolute Addiction Psychiatry Review. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-33404-8_7.

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Milhorn, H. Thomas. "Nicotine." In Drug and Alcohol Abuse. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-6126-6_24.

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George, David J. "Nicotine." In Poisons. CRC Press, 2017. http://dx.doi.org/10.1201/9781315371757-13.

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Conference papers on the topic "Nicotine"

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Campanella, L., R. Cocco, G. Favero, M. P. Sammartino, and M. Tomassetti. "INHIBITION ENZYME SENSOR FOR NICOTINE, NICOTINAMIDE AND NICOTINIC ACID DETERMINATION." In Proceedings of the 5th Italian Conference — Extended to Mediterranean Countries. WORLD SCIENTIFIC, 2000. http://dx.doi.org/10.1142/9789812792013_0005.

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Smith, Danielle, Lynn Kozlowski, Richard O'Connor, Andrew Hyland, Maciej Goniewicz, and Lorraine Collins. "Reasons for individual and concurrent use of vaped nicotine and cannabis: their similarities, differences, and association with product use." In 2021 Virtual Scientific Meeting of the Research Society on Marijuana. Research Society on Marijuana, 2022. http://dx.doi.org/10.26828/cannabis.2022.01.000.15.

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Background: Understanding similarities, differences, and associations between reasons people use vaped nicotine and cannabis may be important for identifying underlying contributors to co-use. Methods: A cross-sectional survey of n=112 co-users of vaped nicotine and cannabis was conducted in 2020. Participants reported on their use of nicotine and cannabis products (vaped and smoked), along with reasons for individual product use, nicotine-cannabis co-use, and engagement with sequential use and co-administration. Results: Cannabis vaping and smoking exhibited similar ratings for user experienc
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Hans, Hesketh, Mila Citrawati, and Citra Ayu Aprilia. "The Effect of Electric Cigarette Nicotine Levels on Peak Expiratory Flow in Vape User Communities, South Jakarta." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.02.14.

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Background: Electronic Cigarette is one form of many nicotine replacement therapy (NRT) that uses energy from battery to deliver nicotine in gas form and by World Health Organization (WHO) defined as Electronic Nicotine Delivery System (ENDS). This study aimed to investigated the effect of electronic cigarette’s nicotine dosage on the peak expiratory flow (PEF). Subjects and Method: This was a cross sectional study conducted at VH Community-South Jakarta from February to march 2018. A Sample of 72 vapers was selected by consecutive sampling. It was divided into 3 groups: vapers who used 3 mg,
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BOUTHERIN-FALSON, O., and N. BLAES. "EFFECT OF NICOTINE ON HUMAN UMBILICAL VEIN ENDOTHELIAL CELLS IN CULTURE : PROSTACYCLIN PRODUCTION AND PROLIFERATION STUDIES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643378.

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Clinical observations and results from animal studies indicate that nicotine may play a role in vascular disorders related to smoking. It has been reported that nicotine could alter vascular prostacyclin (PGI2) production and increase the number of circulating endothelial cells. In the present study, the direct effect of nicotine on endothelial cells in culture was investigated : Both PGI2 production and proliferative abilities were studied.PGI2 production studies : human umbilical vein endothelial cells (HUVEC) were grown in 4 days to confluency in control medium (RPM/199 1:1 + 20% fetal calf
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Fitzke, Reagan, Jordan Davis, and Eric Pedersen. "Co-use of Tobacco/Nicotine and Cannabis Among Veterans: A Preliminary Investigation of Prevalence and Associations with Mental Health Outcomes." In 2020 Virtual Scientific Meeting of the Research Society on Marijuana. Research Society on Marijuana, 2021. http://dx.doi.org/10.26828/cannabis.2021.01.000.10.

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While tobacco and cannabis use rates remain high in the general U.S. population, veterans from the conflicts in Iraq and Afghanistan (i.e., OEF/OIF veterans) are at particularly high risk of high rates of cannabis and tobacco use. Co-use of tobacco/nicotine and cannabis (i.e., using both substances within a specified period of time or combining the drugs within the same device for use) is of growing prevalence in the United States. Tobacco/nicotine and cannabis use is often associated with poor mental health outcomes such as stress, anxiety, and depression. However, little is understood about
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Nenadović, Milena, Katja Kržišnik, Polonca Trebše, and Mojca Bavcon Kralj. "Effect of Time, pH, Alcohol and Sugar Content on Nicotine Re-lease from Pouches Available on Slovene Market." In Socratic lectures 10. University of Lubljana Press, 2024. http://dx.doi.org/10.55295/psl.2024.ii7.

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Abstract: Tobacco-free nicotine pouches, first introduced in Sweden, are made as a less hazardous product for cigarette consumers. Instead of tobacco leaves, they consist of nicotine-containing powder and other ingredients such as water, salts, natural aromatic oils and others, that will boost the flavor and effect and make it last longer. This research aimed to investigate the extraction efficiency of nicotine from poaches of different brands. The effects of alcohol and sugars present in saliva at different pHs and consumption times were investigated as well. We optimized a High performance l
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Zobena, Aija. "Student Tobacco Use Behaviours: A Qualitative Study of Alternative Tobacco and Nicotine Product Use in Young Adulthood." In 14th International Scientific Conference "Rural Environment. Education. Personality. (REEP)". Latvia University of Life Sciences and Technologies. Faculty of Engineering. Institute of Education and Home Economics, 2021. http://dx.doi.org/10.22616/reep.2021.14.043.

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Non-combustible alternative tobacco products such as tobacco-free nicotine pouches, heated tobacco, and electronic cigarettes (e-cigarettes) marketed as less harmful alternatives to cigarettes as smoking cessation aids are becoming increasingly popular among adolescents and young adults. This age group includes individuals still experimenting with and establishing tobacco use. The aim of the study is to investigate student tobacco use behaviours, particularly novel devices, and alternative products to understand how to decrease tobacco initiation and use among adolescents and young adults. In
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Fathi, Raden Muhammad, Ahmad Fauzantoro, Siti Fauziyah Rahman, and Misri Gozan. "Column chromatography isolation of nicotine from tobacco leaf extract (Nicotiana tabaccum L.)." In 2ND BIOMEDICAL ENGINEERING’S RECENT PROGRESS IN BIOMATERIALS, DRUGS DEVELOPMENT, AND MEDICAL DEVICES: Proceedings of the International Symposium of Biomedical Engineering (ISBE) 2017. Author(s), 2018. http://dx.doi.org/10.1063/1.5023958.

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Esther, C. R., W. K. O'Neal, A. L. Koch, et al. "Prolonged, Physiologically Relevant Concentrations of Nicotine and Nicotine Metabolites in Airways of Smokers." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1907.

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Girdhar, Gaurav, Sulan Xu, Jolyon Jesty, and Danny Bluestein. "E-Selectin Expression on Endothelial Cells in the Presence of Platelets and Cigarette Smoke Extract." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192154.

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Cigarette smoking is a risk factor for development of cardiovascular (CV) disease [1], with increased platelet activation due to cigarette smoke involving a major part of this risk.[2] We have shown that this smoke-induced platelet activation is largely due to the non-nicotine smoke components and their effects can be effectively modulated in the presence of nicotine.[3] However, the effects of nicotine and non-nicotine cigarette smoke components need to be confirmed more physiologically in the presence of endothelial cells (ECs). Prior in-vitro studies have shown that high concentrations of c
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Reports on the topic "Nicotine"

1

Lillard, Dean. The Economics of Nicotine Consumption. National Bureau of Economic Research, 2020. http://dx.doi.org/10.3386/w26912.

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Tauras, John, and Frank Chaloupka. The Demand for Nicotine Replacement Therapies. National Bureau of Economic Research, 2001. http://dx.doi.org/10.3386/w8332.

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3

Guillaumier, A., K. McCarter, J. Trigg, et al. Managing nicotine dependence in clinical settings. The Sax Institute, 2023. http://dx.doi.org/10.57022/zuzh3201.

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This Evidence Check aims to identify the most effective clinical interventions for nicotine-dependent patients in clinical settings. The Evidence Check identified 74 eligible studies for inclusion in the review, and the National Health and Medical Research Council (NHMRC) Levels of Evidence were used to assess the robustness of the included studies. According to the evidence, a multi-component approach involving both medications and behavioural interventions remains the best practice for quitting smoking and is recommended to support quitting smoking for nicotine-dependent patients. The medici
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4

Carlezon, Jr, and William A. Nicotine Effects on the Impact of Stress. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada612315.

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Carlezon, Jr, and William A. Nicotine effects on the impact of stress. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada591378.

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Carlezon, Jr, and William A. Nicotine Effects on the Impact of Stress. Defense Technical Information Center, 2015. http://dx.doi.org/10.21236/ada624296.

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7

Roman, Jesse. Prenatal Exposure to Nicotine and Childhood Asthma: Role of Nicotine Acetylcholine Receptors, Neuropeptides, and Fibronectin Expression in Lung. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada452269.

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Roman, Jesse. Prenatal Exposure to Nicotine and Childhood Asthma: Role of Nicotine Acetylcholine Receptors, Neuropeptides and Fibronectin Expression in Lung. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada508588.

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9

Brody, Debra. Data Brief 396: Secondhand Smoke Exposure Among Nonsmoking Adults: United States, 2015-2018. National Center for Health Statistics, 2021. http://dx.doi.org/10.15620/cdc:101197.

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10

Apatov, Nathaniel M. Nicotine-Induced Antinociception in Male and Female Sprague-Dawley Rats. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ad1012097.

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