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Books on the topic 'Nicotinic acetylcholine receptors α7'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Thany, Steeve Hervé, ed. Insect Nicotinic Acetylcholine Receptors. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6445-8.

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2

1944-, Barrantes Francisco J., ed. The nicotinic acetylcholine receptors: Current views and future trends. Springer, 1998.

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3

Clementi, Francesco, Cecilia Gotti, and Emanuele Sher, eds. Nicotinic Acetylcholine Receptors in the Nervous System. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-74167-8.

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4

Francesco, Clementi, Gotti Cecilia, Sher Emanuele, and North Atlantic Treaty Organization. Scientific Affairs Division., eds. Nicotinic acetylcholine receptors in the nervous system. Springer-Verlag, 1988.

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5

Slater, E. Yvonne. The effects of novel alkaloid derivatives on human nicotinic acetylcholine receptors. Oxford Brookes University, 2000.

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6

Jackson, Charles E. P. The pharmacology of nicotinic acetylcholine receptors in Heliothis virescens and Locusta migratoria neurones in vitro. Oxford Brookes University, 1998.

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7

A, Nordberg, and International Symposium on Nicotinic Receptors on the CNS - Their Role in Synaptic Transmission (1988 : Uppsala, Sweden), eds. Nicotinic receptors in the CNS: Their role in synaptic transmission. Elsevier, 1989.

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8

1958-, Parhar Ishwar S., ed. Gonadotropin-releasing hormone: Molecules and receptors. Elsevier, 2002.

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9

P, Illes, and Zimmermann Herbert 1944-, eds. Nucleotides and their receptors in the nervous system. Elsevier, 1999.

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10

G, Holstege, Bandler Richard, and Saper C. B, eds. The emotional motor system. Elsevier, 1996.

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11

H, Yu Albert C., ed. Neuronal-astrocytic interactions: Implications for normal and pathological CNS function. Elsevier, 1992.

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12

M, Gerrits N., Ruigrok T. J. H, and Zeeuw C. I. de, eds. Cerebellar modules: Molecules, morphology, and function. Elsevier, 2000.

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13

Jochen, Klein, Löffelholz Konrad, and International Cholinergic Symposium (9th : 1995 : Mainz, Rhineland-Palatinate, Germany), eds. Cholinergic mechanisms: From molecular biology to clinical significance. Elsevier, 1996.

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14

Luigi, Aloe, and Calzà Laura, eds. NGF and related molecules in health and disease. Elsevier, 2004.

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15

1957-, Castellano Bernardo, and Nieto-Sampedro Manuel 1944-, eds. Glial cell function. Elsevier, 2001.

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16

S, Martinez-Conde, and European Conference on Visual Perception (28th : 2005 : La Coruna, Spain), eds. Visual Perception. Elsevier, 2006.

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17

Masao, Norita, Bando Takehiko, and Stein Barry E, eds. Extrageniculostriate mechanisms underlying visually-guided orientation behavior. Elsevier, 1996.

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18

J, Allum J. H., ed. Natural and artificial control of hearing and balance. Elsevier, 1993.

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19

C, Polosa, and Weaver Lynne C. 1945-, eds. Autonomic dysfunction after spinal cord injury. Elsevier, 2006.

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20

S, Martinez-Conde, and European Conference on Visual Perception (28th : 2005 : La Coruña, Spain), eds. Visual Perception. Elsevier, 2006.

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21

B, Dunnett S., and Björklund Anders 1945-, eds. Functional neural transplantation II: Novel cell therapies for CNS disorders. Elsevier, 2000.

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22

1935-, Barnes Charles D., Pompeiano O, Universitá di Pisa. Dipartimento di Fisiologia e Biochimica., and Washington State University. Dept. of Veterinary and Comparative Anatomy, Pharmacology, and Physiology., eds. Neurobiology of the locus coeruleus. Elsevier, 1991.

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23

H, Kolb, Ripps Harris, Wu Samuel Miao-sin, and Dowling John E, eds. Concepts and challenges in retinal biology. Elsevier, 2003.

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24

Nobel Symposium (76th 1989 Lidingö, Sweden). Cholinergic neurotransmission: Functional and clinical aspects : proceedings of Nobel Symposium 76. Elsevier, 1990.

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25

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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26

Nicotinic Acetylcholine. Springer Verlag, 1988.

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27

Insect nicotinic acetylcholine receptors. Springer Science+Business Media, 2010.

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28

Thany, Steeve Hervé. Insect Nicotinic Acetylcholine Receptors. Springer New York, 2016.

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29

Thany, Steeve Hervé. Insect Nicotinic Acetylcholine Receptors. Springer, 2011.

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30

Maelicke, Alfred. Nicotinic Acetylcholine Receptor: Structure and Function. Springer, 2011.

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31

Maelicke, Alfred. Nicotinic Acetylcholine Receptor: Structure and Function. Springer London, Limited, 2013.

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32

Boyd, R. Thomas. Nicotinic Acetylcholine Receptors in Health and Disease. Elsevier Science & Technology, 2022.

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33

Clementi, Francesco, Cecilia Gotti, and Emanuele Sher. Nicotinic Acetylcholine Receptors in the Nervous System. Springer, 2011.

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34

Clementi, Francesco, Cecilia Gotti, and Emanuele Sher. Nicotinic Acetylcholine Receptors in the Nervous System. Springer London, Limited, 2013.

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35

Clementi, Francesco. Nicotinic Acetylcholine Receptors in the Nervous System. Springer, 2011.

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36

Boyd, R. Thomas. Nicotinic Acetylcholine Receptors in Health and Disease. Elsevier Science & Technology Books, 2022.

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37

Maelicke, Alfred. Nicotinic Acetylcholine Receptor: Structure and Function (Nato Asi Series, Series H : Cell Biology, Vol 3). Springer, 1987.

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38

Clementi, Francesco, and Cecilia Gotti. Nicotinic Acetylcholine: Receptors in the Nervous System (Nato a S I Series Series H, Cell Biology). Springer-Verlag, 1989.

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39

Nicotinic Acetylcholine Receptors: From Molecular Biology to Cognition (Odile Jacob). Odile Jacob, 2005.

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40

Lobel, Peter. Structural studies of a curarimimetic cobra toxin and of the nicotinic acetylcholine receptor. 1986.

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41

A, Nordberg, and InternationalSymposium on Nicotinic Receptors in the CNS, their Role in Synaptic Transmission (1988 : Uppsala, Sweden), eds. Nicotinic receptors in the CNS. Elsevier, 1989.

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42

Royland, Joyce E. Acetylcholine synthesis, storage, degredation and nicotinic receptores in a rat model for space-induced muscle atrophy. 1989.

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43

Leung, Doris G. Other Proven and Putative Autoimmune Disorders of the Peripheral Nervous System. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0098.

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Myasthenia gravis is in most cases an autoimmune disorder of the neuromuscular junction in which antibodies are directed at nicotinic acetylcholine receptors or other synaptic proteins, such as the MusK protein that is involved in the formation of the formation and maturation of the motor endplate. Less commonly, myasthenia gravis can result from antibodies directed to presynaptic calcium channels as a side effect of paraneoplastic antibodies (Lambert-Eaton syndrome) or from a developmental paucity of acetylcholine receptors in the neonatal form of the disease. Treatment is usually a combinati

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44

Mason, Peggy. Receiving the Synaptic Message. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0013.

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Ionotropic and metabotropic receptors differ in their speed of action, the variety of effects produced after ligand-binding, and in the number of types present in the nervous system. The participation of two ionotropic glutamate receptors in synaptic plasticity is thought to be the cellular basis of learning. The actions of acetylcholine on nicotinic acetylcholine receptors present at the neuromuscular junction are described. The pharmacological profile of the GABAA receptor, central to most neural functions, is introduced. The properties of metabotropic receptors that are coupled to G protein

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45

Nordberg, A., K. Fuxe, and B. Holmstedt. Nicotinic Receptors in the Cns: Their Role in Synaptic Transmission (Progress in Brain Research, Vol 79). Elsevier Publishing Company, 1989.

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46

Lambert, David G. Mechanisms and determinants of anaesthetic drug action. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0013.

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This chapter is broken into two main sections: a general description of the principles of ligand receptor interaction and a discussion of the main groups of ‘targets’; and explanation of some common pharmacological interactions in anaesthesia, critical care, and pain management. Agonists bind to and activate receptors while antagonists bind to receptors and block the effects of agonists. Antagonists can be competitive (most common) or non-competitive/irreversible. The main classes of drug target are enzymes, carriers, ion channels, and receptors with examples of anaesthetic relevance interacti

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47

Nutt, David J., and Liam J. Nestor. Nicotine addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0011.

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Cigarette smoking presents with considerable health risks and induces high costs on healthcare resources. People continue to smoke cigarettes in the face of adversity because they contain nicotine, which is highly addictive. Nicotine is a stimulant that exerts its effects within the brain by acting at nicotinic acetylcholine receptors (nAChRs). nAChRs are located in areas of the brain involved in reward processing, motivation, and cognitive control, which results in disruptions to behaviour when nicotine addiction has developed. Disturbances to the brain and behaviour are particularly evident

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48

(Editor), P. Illes, and H. Zimmermann (Editor), eds. Nucleotides and their Receptors in the Nervous System (Progress in Brain Research). Elsevier Science, 1999.

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49

(Editor), J. Klein, and K. Löffelholz (Editor), eds. Cholinergic Mechanisms: From Molecular Biology to Clinical Significance (Progress in Brain Research). Elsevier Science, 1996.

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50

Hertz, Leif, Albert C. H. Yu, and Michael D. Norenberg. Neuronal Astrocytic Interactions: Implications for Normal and Pathological Cns Function (Progress in Brain Research). Elsevier Publishing Company, 1992.

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