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Relevant bibliographies by topics / Nitric Oxide Synthase Type II

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Academic literature on the topic 'Nitric Oxide Synthase Type II'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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Journal articles on the topic "Nitric Oxide Synthase Type II"

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Olson, Susan, Richard Oeckler, Xinmei Li, et al. "Angiotensin II stimulates nitric oxide production in pulmonary artery endothelium via the type 2 receptor." American Journal of Physiology-Lung Cellular and Molecular Physiology 287, no. 3 (2004): L559—L568. http://dx.doi.org/10.1152/ajplung.00312.2003.

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We previously reported that angiotensin II stimulates an increase in nitric oxide production in pulmonary artery endothelial cells. The aims of this study were to determine which receptor subtype mediates the angiotensin II-dependent increase in nitric oxide production and to investigate the roles of the angiotensin type 1 and type 2 receptors in modulating angiotensin II-dependent vasoconstriction in pulmonary arteries. Pulmonary artery endothelial cells express both angiotensin II type 1 and type 2 receptors as assessed by RT-PCR, Western blot analysis, and flow cytometry. Treatment of the e

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Togashi, H., M. Sasaki, E. Frohman, et al. "Neuronal (type I) nitric oxide synthase regulates nuclear factor B activity and immunologic (type II) nitric oxide synthase expression." Proceedings of the National Academy of Sciences 94, no. 6 (1997): 2676–80. http://dx.doi.org/10.1073/pnas.94.6.2676.

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Tomimoto, Hidekazu, Masaki Nishimura, Toshihiko Suenaga, et al. "Distribution of Nitric Oxide Synthase in the Human Cerebral Blood Vessels and Brain Tissues." Journal of Cerebral Blood Flow & Metabolism 14, no. 6 (1994): 930–38. http://dx.doi.org/10.1038/jcbfm.1994.124.

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The distribution of nitric oxide synthase was investigated in human cerebral blood vessels and brain tissues. NADPH-diaphorase histochemistry, which is a marker for nitric oxide synthase in neurons and endothelial cells, revealed periadventitial nerve fibers in the arteries of the circle of Willis and their cortical branches, as well as the common carotid and subclavian arteries. The fibers were mostly nonvaricose in the periadventitial nerve trunk and were varicose within the adventitia. Patchy reaction products were distributed in the perinuclear region of each endothelial cell. Smooth muscl

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Cuzzocrea, Salvatore, Tiziana Persichini, Laura Dugo, Marco Colasanti, and Giovanni Musci. "Copper induces type II nitric oxide synthase in vivo." Free Radical Biology and Medicine 34, no. 10 (2003): 1253–62. http://dx.doi.org/10.1016/s0891-5849(03)00110-2.

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Togashi, H., M. Sasaki, E. Frohman та ін. "NEURONAL (TYPE I) NITRIC OXIDE SYNTHASE REGULATES NUCLEAR FACTOR ϰ B ACTIVITY AND IMMUNOLOGIC (TYPE II) NITRIC OXIDE SYNTHASE EXPRESSION". Japanese Journal of Pharmacology 75 (1997): 6. http://dx.doi.org/10.1016/s0021-5198(19)31275-2.

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Radionova, T. O., І. М. Skrypnyk, О. Ye Akimov, V. О. Kostenko, and V. І. Virchenko. "CORRECTION OF NITRIC OXIDE SYSTEM IN PATIENTS WITH HELICOBACTER PYLORI-ASSOCIATED CHRONIC GASTRITIS AND CONCOMITANT TYPE 2 DIABETES MELLITUS." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 20, no. 2 (2020): 79–85. http://dx.doi.org/10.31718/2077-1096.20.2.79.

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The current data suggest that Helicobacter pylori infection and type 2 diabetes mellitus may affect the state of nitric oxide system, which significantly influences stomach functioning. The aim of the research was to study the state of nitric oxide system in patients with Helicobacter pylori-associated chronic gastritis and concomitant type 2 diabetes mellitus, and to investigate the potential of eupatilin in its correction. 172 patients with confirmed chronic gastritis were enrolled into the study. They were divided into 4 groups: І (n=71) included individuals with Helicobacter pylori-positiv

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Oydanich, Marko, Denis Babici, Jie Zhang, Nicole Rynecki, Dorothy E. Vatner, and Stephen F. Vatner. "Mechanisms of sex differences in exercise capacity." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 316, no. 6 (2019): R832—R838. http://dx.doi.org/10.1152/ajpregu.00394.2018.

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Sex differences are an important component of National Institutes of Health rigor. The goal of this investigation was to test the hypothesis that female mice have greater exercise capacity than male mice, and that it is due to estrogen, nitric oxide, and myosin heavy chain expression. Female C57BL6/J wild-type mice exhibited greater ( P < 0.05) maximal exercise capacity for running distance (489 ± 15 m) than age-matched male counterparts (318 ± 15 m), as well as 20% greater work to exhaustion. When matched for weight or muscle mass, females still maintained greater exercise capacity than ma

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Tracey, W. R., J. S. Pollock, F. Murad, M. Nakane, and U. Forstermann. "Identification of an endothelial-like type III NO synthase in LLC-PK1 kidney epithelial cells." American Journal of Physiology-Cell Physiology 266, no. 1 (1994): C22—C28. http://dx.doi.org/10.1152/ajpcell.1994.266.1.c22.

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Porcine kidney tubular epithelial cells (LLC-PK1) produce nitric oxide or a related compound (e.g., a nitrosothiol) after stimulation with various agonists. We now report the identification and characterization of a constitutive, particulate nitric oxide (NO) synthase from LLC-PK1 cells. After partial purification on adenosine 2',5'-bisphosphate-Sepharose, the particulate NO synthase activity eluted anomalously from Superose 6 gel permeation columns near the total included volume, similar to that observed for the endothelial (type III) NO synthase. Substrate/cofactor requirements of the epithe

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Nicholls, David J., Andrew Kirk, and Alan V. Wallace. "Reversibility of inhibition of human type II nitric oxide synthase." Biochemical Society Transactions 24, no. 1 (1996): 20S. http://dx.doi.org/10.1042/bst024020s.

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Moritoki, Hideki, Tetsuhiro Hisayama, Wataru Kondoh, and Kann Kida. "Protein kinases in induction of type II nitric oxide synthase." Japanese Journal of Pharmacology 71 (1996): 6. http://dx.doi.org/10.1016/s0021-5198(19)33973-3.

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Dissertations / Theses on the topic "Nitric Oxide Synthase Type II"

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Broadbelt, Nalini V. "Regulation of iNOS expression : in response to pressure in proximal tubule epithelial cells /." Access full-text from WCMC, 2008. http://proquest.umi.com/pqdweb?did=1619205731&sid=2&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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Olekhnovitch, Romain. "The antimicrobial activity of nitric oxide at the site of Leishmania infection." Sorbonne Paris Cité, 2015. http://www.theses.fr/2015USPCC152.

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La production d'oxyde nitrique (NO) par l'enzyme iNOS (inducible NO synthase) est impliquée dans le contrôle de nombreuses infections causées par des pathogènes intracellulaires. Cependant, la nature des signaux nécessaires à l'induction d'iNOS in vivo et les mécanismes responsables de son activité antimicrobienne restent à définir. En particulier, plusieurs études suggèrent que le NO exerce son activité de manière intrinsèque : l'induction d'iNOS par les cellules infectées leur permettrait donc de contrôler individuellement leur charge microbienne. Alternativement, la capacité du NO à diffuse

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Al-Dhaher, Zainab. "Angiotensin II produces endothelial dysfunction by simultaneously activating eNOS and NAD(P)H oxidase." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112371.

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Blockade of the renin-angiotensin system lowers the rate of cardiovascular events in patients at risk for vascular disease and also improves endothelial function but the mechanism remains unclear. HUVECs were stimulated with Ang II (100 nM). Ang II produced a 2-fold increase in O2- production, which was measured by lucigenin-enhanced chemiluminescence. This increase was blocked by NAD(P)H oxidase inhibitor DPI, but not by eNOS inhibitor L-NAME. Ang II increased monocyte adhesion to ECs by 4.5-fold, and this increase was blocked by candesartan (AT1 receptor antagonist), DPI, L-NAME, wortmannin

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Campbell, Holly R. 1976. "Chlorine-induced lung injury and the role of iNOS." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111574.

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Reactive airways dysfunction syndrome (RADS), a form of irritant-induced asthma (IIA) has been observed in humans following acute chlorine (Cl 2) gas exposure in occupational and domestic settings. Following Cl 2 injury, subepithelial fibrosis, mucous hyperplasia, and non-specific airway hyperresponsiveness have been reported. Based on the disease profile, we hypothesized that pulmonary damage may be oxidative in nature.<br>The aim of this work was to develop a murine model of irritant-induced asthma in order to investigate the pathogenic processes and potential oxidative mechanisms involved i

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Halpern, Ilana. "Células dendríticas plasmocitoides, dendrócitos dérmicos fator XIIIa positivos, macrófagos e expressão da forma induzida da óxido nítrico sintase na resposta tecidual cutânea de leishmaniose tegumentar americana." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-10102012-095334/.

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Em todas as formas clínicas da leishmaniose tegumentar americana os macrófagos são as células efetoras mais importantes na destruição do parasita intracelular. As células dendríticas são células apresentadoras de antígeno localizadas nos sítios de inoculação, como pele e mucosa. Os dendrócitos dérmicos Fator XIIIa positivos são células derivadas de linhagem mielomonocítica e consideradas complementares às células de Langerhans no processo de apresentação de antígenos e indução da resposta imune. As células dendríticas plasmocitoides representam um subgrupo de células dendríticas precursoras pr

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Smith, Adrian Paul Lindsay. "Hypoxic regulation of preproendothelin-1 and nitric oxide synthase type III expression in the lung." Thesis, University of Cambridge, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624334.

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Yannaccone, Andrew. "Spatial distribution and modulation of nitric oxide synthase in a hypertensive rat model." VCU Scholars Compass, 2012. http://scholarscompass.vcu.edu/etd/2649.

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There are gaps in the fundamental understanding of the expression of nitric oxide synthases (NOS) in the microvasculature. We examined co-localization of NOS1 (nNOS), NOS2 (iNOS) and NOS3 (eNOS) in the spinotrapezius muscle of young adult male Wistar-Kyoto (WKY) and Spontaneously Hypertensive (SHR) rats according to fiber type using immunohistochemistry and brightfield microscopy. Data regarding fiber distribution, population and morphology data were collected. Alkaline phosphatase staining was used to determine capillary density and average number of capillaries around a fiber. Gel electr

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Corbin, Karen Davidowitz. "Multi-Level Regulation Of Argininosuccinate Synthase: Significance For Endothelial Nitric Oxide Production." [Tampa, Fla] : University of South Florida, 2008. http://purl.fcla.edu/usf/dc/et/SFE0002692.

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Ollerstam, Anna. "Macula Densa Derived Nitric Oxide and Kidney Function." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2002. http://publications.uu.se/theses/91-554-5293-0/.

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Sangüesa, Ferrer Juan F. "Modulation fonctionnelle et distribution du canal calcique Cav3. 2 : rôle de la nNOS." Montpellier 1, 2008. http://www.theses.fr/2008MON1T015.

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Le canal calcique Cav3. 2 joue un rôle capital dans de nombreux processus physiologiques, notamment dans la transmission de la douleur aigüe et chronique. Cependant, les mécanismes régulant la distribution et les propriétés de ces canaux restent encore mal connus. Au cours de ma thèse, j'ai participé au développement d'un nouveau modèle pour étudier ces mécanismes : la souris knock-in Cav3. 2-GFPécliptique. Cette souris permettra d'étudier la localisation et la dynamique de Cav3. 2 in vivo grâce à une étiquette fluorescente insérée dans la partie extracellulaire du canal. Des sites de recombin

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Books on the topic "Nitric Oxide Synthase Type II"

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Integrated Systems of the CNS, Part II. Elsevier Publishing Company, 1989.

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Book chapters on the topic "Nitric Oxide Synthase Type II"

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Werner, E. R., and H. H. H. W. Schmidt. "Nitric-Oxide-Synthase Inhibitors II — Pterin Antagonists/Anti-Pterins." In Nitric Oxide. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_7.

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Permutt, Solbert. "Nitric Oxide in Asthma Is Like Insulin in Type II Diabetes." In Mechanics of Breathing. Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5647-3_6.

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Permutt, S. "Nitric Oxide in Asthma is Like Insulin in Type II Diabetes." In Mechanics of Breathing. Springer Milan, 2002. http://dx.doi.org/10.1007/978-88-470-2916-3_23.

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Soni, Jeesha, J. Selvaraj, S. Kavitha, V. Vishnu Priya, and R. Gayathri. "Effect of Argyreia Nervosa on the Endothelial Nitric Oxide Synthase Activity in Experimentally Induced Type 2 Diabetes." In Case Studies on Holistic Medical Interventions. CRC Press, 2024. https://doi.org/10.1201/9781003596684-163.

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Kiel, Johnathan L. "Nitric Oxide Synthase." In Type-B Cytochromes: Sensors and Switches. CRC Press, 2018. http://dx.doi.org/10.1201/9781351077415-4.

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Bahadoran, Zahra, Mattias Carlström, Parvin Mirmiran, and Asghar Ghasemi. "Impaired Nitric Oxide Metabolism in Type 2 Diabetes: At a Glance." In The Role of Nitric Oxide in Type 2 Diabetes. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815079814122010006.

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Abnormal nitric oxide (NO) metabolism has been associated with the development of insulin resistance and type 2 diabetes (T2D). The concept of NO deficiency is supported by human studies on polymorphisms of endothelial NO synthase (eNOS) gene, animal knockout models for NO synthase isoforms (NOSs), and pharmacological evidence, showing detrimental effects of NOS inhibitors and salutary effects of NO donors on carbohydrate metabolism. On the other hand, T2D and insulin resistance may impair NO homeostasis due to hyperglycemia, oxidative stress, and inflammation. Reduced production of NO [i.e.,

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Bahadoran, Zahra, Mattias Carlström, Parvin Mirmiran, and Asghar Ghasemi. "Asymmetrical Dimethyl Arginine, Nitric Oxide, and Type 2 Diabetes." In The Role of Nitric Oxide in Type 2 Diabetes. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815079814122010007.

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Asymmetric dimethylarginine (ADMA), an endogenous competitive inhibitor of nitric oxide (NO) synthase (NOS) isoenzymes, can substantially inhibit vascular NO production at concentrations that are observed in pathophysiological conditions. Over-production of ADMA (via overexpression and/or activity of class 1 of the protein arginine methyltransferases, PRMT-1) alongside decreased catabolism (due to decreased expression and/or activity of dimethylarginine dimethyloaminohydrolase, DDAH) in type 2 diabetes (T2D) and insulin resistance results in increased circulatory and intracellular ADMA levels.

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Pei, Zhe, Kuo-Chieh Lee, Amber Khan, and Hoau-Yan Wang. "Brain Insulin Resistance, Nitric Oxide and Alzheimer’s Disease Pathology." In The Role of Nitric Oxide in Type 2 Diabetes. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815079814122010014.

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Alzheimer’s disease (AD) is a devastating age-related neurodegenerative disease characterized by progressive pathological changes and functional and cognitive impairments. Brain insulin resistance appears to contribute significantly to the pathology and cognitive deficits among several pathological mechanisms. Brain insulin resistance has been demonstrated in animal models of AD and postmortem human brain tissue from patients with AD dementia. Studies conducted in AD models and humans suggest attenuating brain insulin resistance by agents such as glucagon-like peptide1 (GLP-1) analogs and smal

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Yousefzadeh, Nasibeh, Sajad Jeddi, Khosrow Kashfi, and Asghar Ghasemi. "Role of Nitric Oxide in Type 2 Diabetes-Induced Osteoporosis." In The Role of Nitric Oxide in Type 2 Diabetes. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815079814122010011.

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Osteoporosis affects 200 million people worldwide. Osteoporosis in subjects with diabetes is called diabetoporosis, and type 2 diabetes (T2D) contributes to and aggravates osteoporotic fractures. Hyperglycemia, insulin resistance, bone vasculature impairment, increased inflammation, oxidative stress, and bone marrow adiposity contribute to a higher incidence of osteoporotic fractures in T2D. Decreased nitric oxide (NO) bioavailability due to lower endothelial NO synthase (eNOS)-derived NO and higher inducible NOS (iNOS)-derived NO is one of the main mechanisms of the diabetoporosis. Available

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Vincent, S. R. "Chapter II Histochemistry of nitric oxide synthase in the central nervous system." In Handbook of Chemical Neuroanatomy. Elsevier, 2000. http://dx.doi.org/10.1016/s0924-8196(00)80056-1.

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Conference papers on the topic "Nitric Oxide Synthase Type II"

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Yokomizo, Shinya, Dmitriy N. Atochin, and Satoshi Kashiwagi. "Near-infrared II photobiomodulation augments nitric oxide bioavailability via phosphorylation of endothelial nitric oxide synthase (Conference Presentation)." In Biophotonics and Immune Responses XVIII, edited by Wei R. Chen. SPIE, 2023. http://dx.doi.org/10.1117/12.2646735.

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Gredic, M., S. Kraut, C. Y. Wu, et al. "The Role of Myeloid Cell Type-Specific Inducible Nitric Oxide Synthase in Chronic Obstructive Pulmonary Disease." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3837.

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Susanti, Restu, and Yuliarni Syafrita. "Analysis of inducible nitric oxide synthase, CXCL1 serum levels and pericranial tenderness scores in Tension-type headache." In 3RD INTERNATIONAL CONFERENCE OF BIO-BASED ECONOMY FOR APPLICATION AND UTILITY. AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0127764.

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Toukabri, I., and Z. Aouni. "6ER-023 Impact of endothelial nitric oxide synthase gene polymorphism g894t on the development of type-2 diabetes." In Abstract Book, 23rd EAHP Congress, 21st–23rd March 2018, Gothenburg, Sweden. British Medical Journal Publishing Group, 2018. http://dx.doi.org/10.1136/ejhpharm-2018-eahpconf.515.

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Li, Tsai-Kun, Yu-Chen Yang, and Tang-Long Shen. "Abstract LB-394: Type II topoisomerases contribute to nitric oxide-induced DNA breakage during cancer-related inflammation." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-lb-394.

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Elshiekh, Duaa Ibnomer, Hadeel Hendawi, Aya Goul, et al. "Effect of Hyperglycemia on eNOS function in EPCs." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0215.

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Type 2 diabetes mullites (T2DM) results in different cardiovascular complications. The main cause of these complications is endothelial dysfunction, which affects the endothelium physiologically and pathologically. The chronic hyperglycemia introduced by T2DM impacts the pivotal enzyme endothelial nitric oxide synthase (eNOS) in terms of phosphorylation and dimerization, which initiates oxidative stress and reduces the bioavailability of the vasodilator nitric oxide. To overcome endothelial dysfunction, endothelial progenitor cells (EPCs) contribute to vascular repair due to their regenerative

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Ramasamy, Kumaraguruparan, Lori D. Nield, Natalie J. Serkova, et al. "Abstract 948: Silibinin selectively inhibits growth and progression of urethane-induced lung tumors in wild-type but not inducible nitric oxide synthase (-/-) mice: potential usefulness of micro-computed tomography scanning in lung cancer chemoprevention studies." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-948.

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