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Journal articles on the topic 'Non-compacted cardiomyopathy'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Di Toro, Alessandro, Lorenzo Giuliani, Alexandra Smirnova, et al. "Myths to debunk: the non-compacted myocardium." European Heart Journal Supplements 22, Supplement_L (2020): L6—L10. http://dx.doi.org/10.1093/eurheartj/suaa124.

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Abstract Left ventricular non-compaction (LVNC) is defined by the triad: prominent trabecular anatomy, thin compacted layer, and deep inter-trabecular recesses. No person, sick or healthy, demonstrates identical anatomy of the trabeculae; their configuration represents a sort of individual dynamic ‘cardiac fingerprinting’. LVNC can be observed in healthy subjects with normal left ventricular (LV) size and function, in athletes, in pregnant women, as well as in patients with haematological disorders, neuromuscular diseases, and chronic renal failure; it can be acquired and potentially reversibl

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Rodriguez-Fanjul, Javier, Sheila Tubio-Gómez, Juan Manuel Carretero Bellón, Carles Bautista-Rodríguez, and Joan Sanchez-de-Toledo. "Neonatal Non-compacted Cardiomyopathy: Predictors of Poor Outcome." Pediatric Cardiology 41, no. 1 (2019): 175–80. http://dx.doi.org/10.1007/s00246-019-02241-w.

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Nikolayeva, N. V., and O. V. Zotova. "NON-COMPACTED MYOCARDIUM OF THE LEFT VENTRICULAR." Health and Ecology Issues, no. 3 (September 28, 2015): 93–97. http://dx.doi.org/10.51523/2708-6011.2015-12-3-21.

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Non-compacted myocardium of the left ventricular refers to poorly studied diseases of the heart muscle. According to the WHO classification, it belongs to the category of unclassified cardiomyopathy. It develops in congenital heart disease, neuromuscular pathology, defects of the facial skull and has no specific clinical manifestations. The main signs are heart failure, ventricular and supraventricular arrhythmias, systemic and pulmonary embolism.

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Casanova, José David, Josefa González Carrillo, Jesús Martín Jiménez, et al. "Trabeculated Myocardium in Hypertrophic Cardiomyopathy: Clinical Consequences." Journal of Clinical Medicine 9, no. 10 (2020): 3171. http://dx.doi.org/10.3390/jcm9103171.

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Aims: Hypertrophic cardiomyopathy (HCM) is often accompanied by increased trabeculated myocardium (TM)—which clinical relevance is unknown. We aim to measure the left ventricular (LV) mass and proportion of trabeculation in an HCM population and to analyze its clinical implication. Methods and Results: We evaluated 211 patients with HCM (mean age 47.8 ± 16.3 years, 73.0% males) with cardiac magnetic resonance (CMR) studies. LV trabecular and compacted mass were measured using dedicated software for automatic delineation of borders. Mean compacted myocardium (CM) was 160.0 ± 62.0 g and trabecul

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Loo-Valverde, María, Vivian Tuesta-García, Michael Zárate-Quispe, Consuelo Luna-Muñoz, Lucy Correa-Lopez, and Willer Chanduví-Puicón. "Echocardiographic Monitoring of Anatomical Changes in the Myocardium of Pediatric Patients with a Diagnosis of Non-Compaction Cardiomyopathy. 2017 – 2021." Revista de la Facultad de Medicina Humana 24, no. 4 (2024): 138–44. http://dx.doi.org/10.25176/rfmh.v24i4.6688.

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Background: Non-compaction cardiomyopathy (NCM) is known as the lack of maturation of the myocardium that is evidenced by prominent trabeculations and the presence of recesses in newborns and infants that can manifest clinically as heart failure. The purpose of the research is to evaluate the anatomical development of pediatric patients diagnosed with NCM at an early age. Clinical Case: Retrospective case series study of patients diagnosed with non-compaction cardiomyopathy during the first year of life, confirmed according to Jenni criteria, with echocardiographic follow-up every 6 months, fr

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Shlopak, L. B. "A case of lef t ventricular non-compaction (differential diagnosis with hypertrophic cardiomyopathy)." Spravočnik vrača obŝej praktiki (Journal of Family Medicine), no. 7 (July 8, 2023): 46–51. http://dx.doi.org/10.33920/med-10-2307-06.

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Non-compacted myocardium is a condition characterized by changes in the myocardium in the form of a slight thickening of the epicardial layer with pronounced trabecularity and deep spaces that communicate with the left ventricle cavity, but not with the coronary arteries. The non-compacted myocardium is characterized by the presence of areas of a two-layer myocardium structure with a pronounced spongy layer and a large number of trabeculae and intertrabecular spaces freely communicating with the heart cavity according to imaging study methods.

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Jagminas, Rokas, Rokas Šerpytis, Pranas Šerpytis, and Sigita Glaveckaitė. "Left Ventricular Hypertrabeculation (LVHT) in Athletes: A Negligible Finding?" Medicina 61, no. 1 (2024): 32. https://doi.org/10.3390/medicina61010032.

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Left ventricular hypertrabeculation (LVHT) used to be a rare phenotypic trait. With advances in diagnostic imaging techniques, LVHT is being recognised in an increasing number of people. The scientific data show the possibility of the overdiagnosis of this cardiomyopathy in a population of people who have very high levels of physical activity. We describe the case of a young athlete with no medical history, who presented with syncope during a marathon running race. Initial evaluation showed elevated troponin I; transthoracic echocardiography showed a trabeculated ventricle and subsequent cardi

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Fatima, Safa, Lintu Ramachandran, and Mohammad Khan. "COMPACTED CARDIA: LEFT VENTRICULAR NON-COMPACTION CARDIOMYOPATHY IN A YOUNG MALE." Journal of the American College of Cardiology 79, no. 9 (2022): 2605. http://dx.doi.org/10.1016/s0735-1097(22)03596-3.

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Rovira, I., G. Fita, P. Matute, C. Roux, J. Fontanals, and C. Gomar. "Non-compacted cardiomyopathy associated with acute mitral chordae rupture: case report." European Journal of Anaesthesiology 22, Supplement 35 (2005): 42. http://dx.doi.org/10.1097/00003643-200506001-00108.

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MJ, Tino-Martinez, Lerma-Espinosa R, Llaguno-López R, Trejo-Bahena NI, Ocaña-Sánchez MA, and Cortés-Benítez A. "Efecto de la rehabilitación cardiaca fase II en pacientes con insuficiencia cardiaca diagnosticados con miocardio no compacto." Revista de Sanidad Militar 72, no. 2 (2018): 84–89. http://dx.doi.org/10.56443/rsm.v72i2.157.

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Background: Non-compacted myocardium is a recently described cardiomyopathy. It can present itself at any age as different clinical entities, such as heart failure syndrome, arrhythmias, systemic embolism, or sudden death. Scientifi c evidence demonstrates a decrease in mortality in patients with heart failure who are admitted to cardiac rehabilitation programs. Objective: To evaluate the effect of phase II cardiac rehabilitation programs in patients diagnosed with heart failure secondary to non-compacted myocardium. Material and methods: An observational and retrospective study was performed,

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Kádár, Krisztina, Anna Zsuzsa Bagoly, and Zita Sikos. "Correlations between B-type natriuretic peptide levels and nongeometric echocardiographic parameters in pediatric cardiomyopathies." Orvosi Hetilap 154, no. 11 (2013): 409–14. http://dx.doi.org/10.1556/oh.2013.29541.

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Introduction: B-type natriuretic peptide is a conventional cardiac biomarker in adult cardiology, however, it is not commonly used in pediatric cases. Aims: After introducing B-type natriuretic peptide measurements in pediatric patients for the evaluation of systemic right ventricular function, the authors wanted to evaluate the value of plasma B-type natriuretic peptide and compare the results with systolic and diastolic myocardial parameters obtained with conventional echocardiography and tissue Doppler imaging in children with cardiomyopathy. Methods: Between 2007 and 2010, 58 plasma B-type

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Seo, Jun-young, Ji Hoon Yu, Ji Young Choi, Ji Young Ko, and Min Seok Oh. "Dilated Cardiomyopathy Mimicking Arrhythmogenic Right Ventricular Cardiomyopathy in a 35-Year-Old Woman." Clinical Ultrasound 7, no. 2 (2022): 118–23. http://dx.doi.org/10.18525/cu.2022.7.2.118.

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While dilated cardiomyopathy (DCMP) typically involves the left ventricle (LV), it can also impact the right ventricle (RV). Arrhythmogenic right ventricular cardiomyopathy is a heritable myocardial disorder that mainly affects the RV. A 35-year-old woman presented to the emergency department complaining of chest discomfort and palpitations. The patient had ingested 100 g of alcohol daily for more than 5 years. Transthoracic echocardiography showed dilation of the four cardiac chambers with global systolic dysfunction of the prominent myocardial recesses of both ventricles. Non-sustained ventr

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Nemes, Attila. "Myocardial Mechanics and Associated Valvular and Vascular Abnormalities in Left Ventricular Noncompaction Cardiomyopathy." Journal of Clinical Medicine 13, no. 1 (2023): 78. http://dx.doi.org/10.3390/jcm13010078.

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Left ventricular (LV) non-compaction (LVNC) is a rare genetic cardiomyopathy due to abnormal intra-uterine arrest of compaction of the myocardial fibers during endomyocardial embryogenesis. Due to the partial or complete absence of LV compaction, the structure of the LV wall shows characteristic abnormalities, including a thin compacted epicardium and a thick non-compacted endocardium with prominent trabeculations and deep intertrabecular recesses. LVNC is frequently associated with chronic heart failure, life-threatening ventricular arrhythmias, and systemic embolic events. According to recen

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Paluszkiewicz, Jerzy, Hendrik Milting, Marta Kałużna-Oleksy, et al. "Left Ventricular Non-Compaction Cardiomyopathy-Still More Questions than Answers." Journal of Clinical Medicine 11, no. 14 (2022): 4135. http://dx.doi.org/10.3390/jcm11144135.

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Left ventricular non-compaction (LVNC) describes the phenotypical phenomena characterized by the presence of excessive trabeculation of the left ventricle which forms a deep recess filled with blood. Considering the lack of a uniform definition of LVNC as well as the “golden standard” it is difficult to estimate the actual incidence of the disease, however, seems to be overdiagnosed, due to unspecific diagnostic criteria. The non-compacted myocardium may appear both as a disease representation or variant of the norm or as an adaptive phenomenon. This article covers different approaches to inci

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Fatema, Nilufar, Ahmed Saiful Bari, Naznin Sultana, et al. "Noncompaction of Ventricular Wall - A Threat to Life." University Heart Journal 7, no. 1 (2012): 39–41. http://dx.doi.org/10.3329/uhj.v7i1.10209.

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Ventricular noncompaction is a kind of cardiomyopathy which is called Noncompaction Cardiomyopathy (NCC). In this condition in which the muscular wall of the main pumping chamber of the heart -the left ventricle (LV) appears to be spongy and non-compacted, consisting of a meshwork of numerous muscle bands called trabeculations. Here, anatomically LV wall has deep trabeculations. This condition is associated major clinical problems like systolic and diastolic dysfunction, arrhythmia and even systemic embolism. Sudden cardiac death may occur in this group. Early detection of this condition may

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Nilda, Espínola-Zavaleta 1. 4. ME Soto 2. Eulo Lupi-Herrera 1. Paola Romano 1. Verónica Guarner-Lans 3. Claudia Huesca-Gómez 3. Ricardo Gamboa*3. "THE POLYMORPHISMS LDB3 (rs45618633), TAZ (rs104894941), LMNA (rs28928903), DTNA (rs7243528) AND TNNT2 G/T (rs2365652) AND ITS ASSOCIATION WITH NON-COMPACTED CARDIOMYOPATHY IN MEXICAN MESTIZO FAMILY." Indian Journal of Medical Research and Pharmaceutical Sciences 4, no. 12 (2017): 13–23. https://doi.org/10.5281/zenodo.1095889.

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<strong>Introduc</strong><strong>tion:</strong> Noncompacted cardiomyopathy (NCC) has been attributed to the mutation in multiple genes leading to interruption in the endomyocardial compaction process. <strong>Objective:</strong> The objective of this work was to identify gene variants that participated in the development of NCC in a Mexican mestizo family. <strong>Material and methods</strong>: A total of 36 (85.7%) individuals from a family of 42 members were studied. All patients underwent clinical history, transthoracic echocardiography and the polymorphisms: <em>LDB3, TAZ, LMNA,

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Nimeri, N. A., F. F. Abou Nahia, A. S. Ibrahim, and A. Y. Khella. "The first reported case of non-compacted cardiomyopathy in a preterm infant with Ebstein's anomaly." Case Reports 2012, jul20 1 (2012): bcr0220125861. http://dx.doi.org/10.1136/bcr-02-2012-5861.

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Massara, M. "A RARE CASE OF INTERVENTRICULAR SEPTUM DUPLICATION." European Heart Journal Supplements 26, Supplement_2 (2024): ii193. http://dx.doi.org/10.1093/eurheartjsupp/suae036.462.

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Abstract Case Report A 62–year–old man, history of hypertension. In 2015, there was suspicion of non–compaction cardiomyopathy, and he underwent cardiac MRI at another facility (report not available). The patient reported palpitations and dyspnea with exertion, leading to a follow–up visit. EKG showed sinus rhythm 90/min, nonspecific intraventricular conduction delay and isolated monomorphic ventricular ectopic beats. Echo revealed a mildly dilated left ventricle (end–diastolic volume 88 ml/m), systolic function at the lower limits of normal (EF 53%), and prominent muscular structures near the

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Huertas-Quiñones, Víctor M., Camilo F. Mestra, Valeria Peña-Trujillo, Sebastián Gallo-Bernal, Mariana Villaveces, and Laura C. Alarcón-Forero. "Paediatric cardiomyopathies: echocardiographic diagnosis, clinical profile, and demographic characteristics: the experience of a tertiary referral centre for Latin American paediatric cardiology." Cardiology in the Young 30, no. 4 (2020): 462–67. http://dx.doi.org/10.1017/s1047951120000281.

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AbstractBackground:Although multiple studies have been conducted in the adult population, there is a vast knowledge gap regarding the epidemiologic characteristics of cardiomyopathies in the paediatric population. This issue is even more crucial when the precarious situation of medical research in Latin America is considered. Given the potential impact that these disorders could have on Latin American health systems, a comprehensive epidemiologic study regarding the clinical profile and sociodemographic characteristics of these patients will influence the way we approach paediatric cardiomyopa

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Lopes, Débora, Cátia Gorgulho, Joana Ribeiro, André Neto Real, and Nuno Catorze. "The Crack Lung – A Case Report." Brazilian Journal of Case Reports 5, no. 1 (2025): bjcr49. https://doi.org/10.52600/2763-583x.bjcr.2025.5.1.bjcr49.

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Cocaine is an alkaloid extracted from the leaves of Erythroxylon coca and was first isolated in 1859. Cocaine induces both acute and chronic toxicity, affecting virtually every organ system. A 32-year-old male patient, with a history of post-traumatic epilepsy and substance abuse involving cannabinoids and cocaine, presented to the Emergency Department (ED) following self-limited seizures. Arterial blood gas analysis revealed hypoxemic respiratory failure, while blood tests showed no significant abnormalities. A thoracic computed tomography (CT) scan demonstrated extensive consolidations in mo

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Gurunathan, Sothinathan, and Roxy Senior. "Catastrophic stroke in a patient with left ventricular non-compaction." Echo Research and Practice 5, no. 3 (2018): K59—K62. http://dx.doi.org/10.1530/erp-18-0015.

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Summary We present the case of a 32-year-old man who presented with a remote history of chest pain and was diagnosed with non-compaction cardiomyopathy on echocardiography. On presentation, he was relatively asymptomatic with normal cardiac function. Unfortunately, he presented 1 year later with a catastrophic embolic stroke. Learning points: Left ventricular non-compaction (LVNC) is a myocardial disorder characterised by prominent left ventricular (LV) trabeculae, a thin compacted layer and deep intertrabecular recesses. Two-dimensional echocardiography with colour Doppler is the study of cho

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Manai, R., V. Dusi, S. Pidello, et al. "C27 RBM20 VARIANTS RELATED CARDIOMYOPATHY: AN ITALIAN CASE SERIES." European Heart Journal Supplements 25, Supplement_D (2023): D12. http://dx.doi.org/10.1093/eurheartjsupp/suad111.026.

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Abstract Background RNA–binding motif protein 20 (RBM20) regulates post–transcriptional splicing, particularly in sarcomeric, but also in other cardiac genes crucial for homeostasis. RBM20 variants are a rare cause of cardiomyopathy (CMP), but genotype–phenotype correlation is still elusive, starting from the type of CMP (dilated, non–compacted, hypertrophic, and even restrictive phenotypes have been described), and ranging to sex–dependent and arrhythmic expression. Purpose: To describe cardiac involvement in 8 patients with RBM20 variants. Methods 189 patients (pts) underwent genetic analysi

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FRISCHKNECHT, B. "P1144 Validation of criteria for non-compacted myocardium in dilated cardiomyopathy, valvular and hypertensive heart disease." European Heart Journal 24, no. 5 (2003): 211. http://dx.doi.org/10.1016/s0195-668x(03)94436-8.

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Obramenko, I. E. "Semiotics of apical hypertrophic cardiomyopathy in magnetic resonance Imaging." Regional blood circulation and microcirculation 19, no. 2 (2020): 18–23. http://dx.doi.org/10.24884/1682-6655-2020-19-2-18-23.

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Introduction. About 0.2 % of the adult population all over the world suffers from hypertrophic cardiomyopathy. Early and timely diagnosis of the apical form of hypertrophic cardiomyopathy remains an urgent medical problem, since the disease has a wide variability of clinical manifestations and often occurs asymptomatic or with symptoms of other heart diseases. Magnetic resonance imaging is an informative method of radiation diagnosis of hypertrophic cardiomyopathy. The aim is improving of radiology diagnostics in applying to the apical form of hypertrophic cardiomyopathy. Materials and methods

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Bustea, Cristiana, Alexa Florina Bungau, Delia Mirela Tit, et al. "The Rare Condition of Left Ventricular Non-Compaction and Reverse Remodeling." Life 13, no. 6 (2023): 1318. http://dx.doi.org/10.3390/life13061318.

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Left ventricular non-compaction (LVNC) is a rare disease defined by morphological criteria, consisting of a two-layered ventricular wall, a thin compacted epicardial layer, and a thick hyper-trabeculated myocardium layer with deep recesses. Controversies still exist regarding whether it is a distinct cardiomyopathy (CM) or a morphological trait of different conditions. This review analyzes data from the literature regarding diagnosis, treatment, and prognosis in LVNC and the current knowledge regarding reverse remodeling in this form of CM. Furthermore, for clear exemplification, we report a c

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Shen, Mylène, Romain Capoulade, Lionel Tastet, et al. "Prevalence of left ventricle non-compaction criteria in adult patients with bicuspid aortic valve versus healthy control subjects." Open Heart 5, no. 2 (2018): e000869. http://dx.doi.org/10.1136/openhrt-2018-000869.

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ObjectiveThe aim of this study was to compare the prevalence of left ventricle non-compaction (LVNC) criteria (or hypertrabeculation) in a cohort of patients with bicuspid aortic valve (BAV) and healthy control subjects (CTL) without cardiovascular disease using cardiovascular MR (CMR).Methods79 patients with BAV and 85 CTL with tricuspid aortic valve and free of known cardiovascular disease underwent CMR to evaluate the presence of LVNC criteria. The left ventricle was assessed at end-systole and end-diastole, in the short-axis, two-chamber and four-chamber views and divided into the 16 stand

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Vaikhanskaya, T. G., L. N. Sivitskaya, T. V. Kurushko, et al. "Non-compaction cardiomyopathy. Part I: clinical and genetic heterogeneity and predictors of unfavorable prognosis." Russian Journal of Cardiology 25, no. 11 (2020): 3872. http://dx.doi.org/10.15829/29/1560-4071-2020-3872.

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Non-compaction cardiomyopathy (NCM) is a rare heart disease characterized by a two-layered ventricular wall, comprising a thinner compact epicardial layer and an inner non-compacted layer. However, only structural and morphological data without a thorough clinical assessment does not determine the NCM (regardless of the diagnostic criterion used).Aim. To study the NCM-related genes, phenotypic and genetic correlations, predictors of life-threatening ventricular tachyarrhythmias (VTA) and adverse clinical outcomes.Material and methods. Of 93 individuals with identified morphological criteria of

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Jaouadi, Hager, Fedoua El Louali, Chloé Wanert, Aline Cano, Caroline Ovaert, and Stéphane Zaffran. "Dilated-Left Ventricular Non-Compaction Cardiomyopathy in a Pediatric Case with SPEG Compound Heterozygous Variants." International Journal of Molecular Sciences 23, no. 9 (2022): 5205. http://dx.doi.org/10.3390/ijms23095205.

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Left Ventricular Non-Compaction (LVNC) is defined by the triad prominent myocardial trabecular meshwork, thin compacted layer, and deep intertrabecular recesses. LVNC associated with dilation is characterized by the coexistence of left ventricular dilation and systolic dysfunction. Pediatric cases with dilated-LVNC have worse outcomes than those with isolated dilated cardiomyopathy and adult patients. Herein, we report a clinical and genetic investigation using trio-based whole-exome sequencing of a pediatric case with early-onset dilated-LVNC. Compound heterozygous mutations were identified i

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Комиссарова, С. М., and Н. М. Ринейская. "Thromboembolic Risk and Anticoagulation Strategies in Non-Compaction Cardiomyopathy." Кардиология в Беларуси 15, no. 4 (2023): 460–71. http://dx.doi.org/10.34883/pi.2023.15.4.003.

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Введение. Некомпактная кардиомиопатия (НКМП) – редкое заболевание левого и/или правого желудочков с наличием двухслойной структуры миокарда, характеризующееся более толстым эндокардиальным слоем с выраженными трабекулами и глубокими межтрабекулярными углублениями и более тонким уплотненным эпикардиальным слоем. Учитывая значительный риск тромбоэмболических осложнений (ТЭО) у пациентов с НКМП (частота тромбоэмболических осложнений составляет от 0 до 38%), важность стратегии антикоагулянтной терапии не вызывает сомнений. Однако до сих пор антикоагулянтная терапия является предметом дискуссий и с

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Jiménez Gómez, Elvira, Isabel Bravo Rey, Rafael Oteros Fernández, and Fernando Delgado Acosta. "Endovascular treatment of stroke in children under 2 years with heart failure and ventricular assist device." Interventional Neuroradiology 25, no. 5 (2019): 516–20. http://dx.doi.org/10.1177/1591019919844946.

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Introduction Strokes in children are characterised by a high mortality rate while, at the same time, the low number of cases makes it difficult to gain practical experience. As heart disease is the most common risk factor, and as more and more cardiological interventions are being carried out, an increase in the incidence of paediatric stroke is expected. In some cases a transplant is required. While waiting for a donor, the use of ventricular assist devices may be necessary. These present with a high rate of neurological complications. We present two cases of children under 2 years of age awa

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Thiene, Gaetano. "Storytelling of Myocardial Biopsy." Biology 14, no. 3 (2025): 306. https://doi.org/10.3390/biology14030306.

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A biopsy involves the removal of a piece or an entire organ from a living patient. The former began with open heart surgery (surgical pathology) and the latter with the recipient heart in cardiac transplantation. Transvenous or transarterial catheterization is the current procedure to performed endomyocardial biopsy with bioptome from the ventricles. This manoeuvre was first carried out by Werner Forssmann through a urological catheter in 1929, which he introduced into his radial left vein until it reached the RV. Then, in London in 1974, Richardson invented a new technique with a catheter via

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Simonenko, M. A., T. M. Pervunina, P. A. Fedotov, et al. "THE EXPERIENCE OF PEDIATRIC HEART TRANSPLANTATION ON NORTH-WEST OF RUSSIAN FEDERATION." Russian Journal of Transplantology and Artificial Organs 20, no. 2 (2018): 37–46. http://dx.doi.org/10.15825/1995-1191-2018-2-37-46.

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Aim:to estimate early and long-term outcomes in recipients under 18 years old who have been heart transplanted in Almazov National Medical Research Centre.Materials and methods. From April 2011 to September 2017 we performed 5 heart transplantations (HTx) in recipients under 18 years old (female) old) from adults donors. The median of age were 15 years (range 10–16 years), LVEF prior HTx – 17% (10–33%). Causes of heart failure were dilated cardiomyopathy (n = 2), non-compacted myocardium (n = 1), arrhythmogenic ventricular dysplasia (n = 1) and Ebstein’s anomaly (n = 1). They spent in HT waiti

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Alieva, I. N., O. V. Blagova, N. V. Gagarina, et al. "Multislice spiral computed tomography of the heart in dilated cardiomyopathy: possibilities in the verification of myocarditis (in comparison with myocardial biopsy) and in the evaluation of prognosis." Terapevticheskii arkhiv 89, no. 12 (2017): 15–27. http://dx.doi.org/10.17116/terarkh2017891215-27.

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Aim. To investigate whether intravenous contrast-enhanced multislice spiral computed tomography (computed tomography) (MSCT) versus myocardial morphological examination can diagnose myocarditis and the non-inflammatory causes of dilated cardiomyopathy (DCM) and evaluate prognosis in patients with the latter. Subjects and methods. A study group consisted of 130 patients, including 95 men (46.8±11.9 years), with DCM (mean left ventricular (LV) end-diastolic dimension (EDD), 6.6±0.8 cm; mean LV ejection fraction (EF), 29.8±9.3%; NYHA functional class (FC) III (II; III)). All the patients underwen

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Espinola-Zavaleta, Nilda, M. Elena Soto, Luis Muñóz Castellanos, Silvio Játiva-Chávez, and Candace Keirns. "Non-compacted cardiomyopathy: clinical-echocardiographic study." Cardiovascular Ultrasound 4, no. 1 (2006). http://dx.doi.org/10.1186/1476-7120-4-35.

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"Mixed phenotype: arrhythmogenic and non-compacted cardiomyopathy." Journal of Clinical and Translational Research, 2016. http://dx.doi.org/10.18053/jctres.aecvp2016.020.

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Bianco, Francesco. "Non-Compacted Cardiomyopathy: Is there a Need of a New Cardiomyopathy?" Advancements in Cardiovascular Research 2, no. 1 (2019). http://dx.doi.org/10.32474/acr.2019.02.000129.

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Bianco, Francesco. "Non-Compacted Cardiomyopathy: Is there a Need of a New Cardiomyopathy?" February 17, 2020. https://doi.org/10.32474/acr.2019.02.000129.

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Kołodzińska, Agnieszka, Anna Heleniak, and Anna Ratajska. "Retinoic acid-induced ventricular non-compacted cardiomyopathy in mice." Kardiologia Polska, May 19, 2013, 447–52. http://dx.doi.org/10.5603/kp.2013.0090.

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Kaliyev, Bauyrzhan Bakhytovich, Tairkhan Bekpolatovich Dautov, and Bibissara Yerekesh. "Left ventricular non-compaction with ventricular aneurysm in 6-year-old patient." Digital Diagnostics, October 17, 2023. http://dx.doi.org/10.17816/dd551837.

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Ventricular noncompaction is a very rare form of cardiomyopathy and its combination with a ventricular aneurysm is even more uncommon. We present a clinical case of a 6-year-old girl, who was admitted to our clinic with complaints of physical exertion. Echocardiography demonstrated prominent trabeculations in the left ventricular wall with a lateral basal part ballooning out, findings consistent with noncompaction of the left ventricular myocardium with an aneurysm. Magnetic resonance imaging confirmed evidence of non-compacted myocardium with a non-compacted /compacted myocardium ratio of 2.7

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Principi, S., M. Alfieri, A. Barbarossa, et al. "Left-ventricular non-compaction and ventricular arrhythmias." Europace 25, Supplement_1 (2023). http://dx.doi.org/10.1093/europace/euad122.262.

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Abstract Funding Acknowledgements Type of funding sources: None. Background Left ventricular non-compaction (LVNC) is a rare, although underdiagnosed, cardiomyopathy deriving from the incomplete development of myocardial tissue. Its presentations range from heart failure to embolism and arrhythmic events with the latter being the most frequent cause of death. Purpose To define the incidence of arrhythmic complications in LVNC and describe the interplay between systolic dysfunction and ventricular events. Methods This is a retrospective evaluation of a prospectively-collected database including

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Quaglia, María Isabel, Carla Gobbi, Carla Alonso, et al. "Non compacted cardiomyopathy as a cause of insufficiency cardiac in Systemic Lupus Erythematosus." Revista Argentina de Reumatología, June 1, 2021, 33–36. http://dx.doi.org/10.47196/rar.v32i2.359.

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Non compacted cardiomyopathy (NCNC) frequently represents an abnormality of myocardial morphology associated with a genetic etiology, however, there are few descriptions of its association with autoimmune diseases. We present a review of the literature and the case of a lupus patient who was admitted with signs of decompensated heart failure with confirmation of VINC by echocardiography and cardiac nuclear magnetic resonance (C-MRI).

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Gastl, Mareike, Alexander Gotschy, Malgorzata Polacin, et al. "Determinants of myocardial function characterized by CMR-derived strain parameters in left ventricular non-compaction cardiomyopathy." Scientific Reports 9, no. 1 (2019). http://dx.doi.org/10.1038/s41598-019-52161-1.

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Abstract Clinical presentation of left ventricular non-compaction cardiomyopathy (LVNC) can be heterogeneous from asymptomatic expression to congestive heart failure. Deformation indices assessed by cardiovascular magnetic resonance (CMR) can determine subclinical alterations of myocardial function and have been reported to be more sensitive to functional changes than ejection fraction. The objective of the present study was to investigate the determinants of myocardial deformation indices in patients with LVNC. Twenty patients with LVNC (44.7 ± 14.0 years) and twenty age- and gender-matched c

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Chandra, S., A. A. Lukito, H. Hutabarat, and R. A. P. Fernandes. "C83. Recurrent Syncope in Isolated Right Ventricular Non-Compaction Cardiomyopathy: A Case Report." European Heart Journal Supplements 23, Supplement_F (2021). http://dx.doi.org/10.1093/eurheartjsupp/suab125.082.

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Abstract Isolated right ventricular non-compaction cardiomyopathy (iRVNC) is a rare disease, and often under diagnosed. The patients might come with right heart failure, ventricular arrhythmias and systemic embolism. A 64 –yo female presented with episodes of recurrent syncope. There is thickened trabecular on apical right ventricular (RV), moderate tricuspid regurgitation and moderate probability of pulmonary hypertension on echocardiography. Furthermore, cardiac MRI on this patient confirned hypertrabeculated and dilatation of RV, spongiform appearance of the RV apical, non-compacted (NC) th

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Chandra, S., A. A. Lukito, H. Hutabarat, and R. A. P. Fernandes. "C83. Recurrent Syncope in Isolated Right Ventricular Non-Compaction Cardiomyopathy: A Case Report." European Heart Journal Supplements 23, Supplement_F (2021). http://dx.doi.org/10.1093/eurheartjsupp/suab124.082.

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Abstract Isolated right ventricular non-compaction cardiomyopathy (iRVNC) is a rare disease, and often under diagnosed. The patients might come with right heart failure, ventricular arrhythmias and systemic embolism. A 64 –yo female presented with episodes of recurrent syncope. There is thickened trabecular on apical right ventricular (RV), moderate tricuspid regurgitation and moderate probability of pulmonary hypertension on echocardiography. Furthermore, cardiac MRI on this patient confirned hypertrabeculated and dilatation of RV, spongiform appearance of the RV apical, non-compacted (NC) th

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Abburu, Alekhya. "A Case of Pregnant Woman with Severe Mitral Regurgitation Demonstrating Left Ventricular Non-compaction." Journal of Clinical Medical Research, July 2, 2024, 1–6. http://dx.doi.org/10.46889/jcmr.2024.5204.

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Left ventricular non-compaction is a rare form of cardiomyopathy. It is characterized by a thin compacted epicardial layer and a noncompacted endocardial layer with significant wall trabeculations and intertrabecular recesses that communicate with the ventricular cavity. It could be either in isolated form or coexists with other congenital heart diseases including valvular heart disease. It is associated with congestive heart failure, life threatening cardiac arrhythmia and thromboembolic complications. Adult LVNC has a prevalence of 0.01-0.27%. Diagnostic workup includes echocardiography, con

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Hotta, V. T., L. M. B. Martinelli, F. Fernandes, V. A. Moises, M. L. C. Vieira, and C. Mady. "P697 Left ventricular non compaction highlighted by three-dimensional and speckle tracking echocardiography." European Heart Journal - Cardiovascular Imaging 21, Supplement_1 (2020). http://dx.doi.org/10.1093/ehjci/jez319.371.

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Abstract Introduction Left ventricular non-compaction (LVNC) is a relatively new cardiomyopathy, first reported by Chin et al. in 1990. Since then, much has been learned about this entity, but until now, there are some limitations for the diagnosis of this disease. Cardiac Magnetic Resonance Imaging is considered the gold standard for the diagnosis of LVNC, but echocardiography remains the first line imaging modality due to its availability and cost efficacy. Case report In this case, we report a case of an asymptomatic 21 years old young male with no personal or familiar history of cardiomyop

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Anton, M., S. Boeangiu, C. Maresiu, et al. "45 Severe secondary mitral regurgitation due to left ventricular non- compactation cardiomyopathy- a rare cause of heart failure." European Heart Journal - Cardiovascular Imaging 21, Supplement_1 (2020). http://dx.doi.org/10.1093/ehjci/jez319.007.

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Abstract Introduction Left ventricular non-compaction is a rare form of cardiomyopathy charactherized by the presence of a two layered ventricular wall- a thinner epicardial layer and an inner, non-compacted layer with prominent trabeculations associated with deep, intratrabecular recesses that communicate with left ventricle cavity. Clinical manifestations vary in severity, including symptoms of heart failure, thromboembolic events or arrhythmias. Left ventricular (LV) dysfunction leads to tethering of the mitral apparatus and is a cause for secondary significant mitral regurgitation. Case pr

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Rodrigo, D., U. Estandia, P. Perez, C. Perez, A. Cortes, and E. Panera. "89 Simultaneous ocurrence of hammock mitral valve and no-compaction cardiomyopathy." European Heart Journal - Cardiovascular Imaging 21, Supplement_1 (2020). http://dx.doi.org/10.1093/ehjci/jez319.012.

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Abstract We report a 62-year-old man with a past medical history of dyslipidemia, paranoid schizophrenia and permanent atrial fibrillation. A ATTE performed at his district hospital revealed rheumatic mitral valve disease with double lesion: severe regurgitation and mild stenosis, plus moderate tricuspid regurgitation and a mean PAP of 32mm Hg. Cardiac catheterization showed no abnormalities of the coronary arteries. He was transferred to our hospital and scheduled for mitral valve replacement and tricuspid ring valvuloplasty. Preoperative transesophageal echocardiography showed an abnormal su

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Guedes Ramallo, P., L. Carrillo Aleman, E. Candela Sanchez, and E. Feliu Rey. "P107Prenatal diagnosis of familiar biventricular non-compacted cardiomyopathy with later demonstration of a MYH7 gene mutation." European Heart Journal - Cardiovascular Imaging 20, Supplement_2 (2019). http://dx.doi.org/10.1093/ehjci/jez110.006.

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Kayvanpour, E., F. Sedaghat-Hamedani, W. T. Gi, et al. "P4658Left ventricular non-compaction, trait or cardiomyopathy." European Heart Journal 40, Supplement_1 (2019). http://dx.doi.org/10.1093/eurheartj/ehz745.1040.

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Abstract Background The diagnosis of cardiac hypertrabecularization has increased considerably in recent years. Whether or not the non-compaction is a pathological condition or a physiological trait is still highly debated. We performed a meta-analysis and systematic review on more than 7,000 adult individuals with left ventricular non-compaction to provide a comprehensive overview on its clinical outcome as well as its genetic background. Methods We first retrieved PubMed/Medline literatures in English language between 2000 to 2018 on clinical outcome and genotype of patients with non-compact

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