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Journal articles on the topic 'Non-lymphoid cells'

Author: Grafiati

Published: 7 June 2025

Last updated: 16 July 2025

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1

Crouse, David A., James B. Turpen, and J. Graham Sharp. "Thymic non-lymphoid cells." Survey of Immunologic Research 4, no. 2 (1985): 120–34. http://dx.doi.org/10.1007/bf02918808.

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2

Deyev, Sergey M., Andre Lieber, Boris V. Radko, and Oleg L. Polanovsky. "Production of recombinant antibodies in lymphoid and non-lymphoid cells." FEBS Letters 330, no. 2 (1993): 111–13. http://dx.doi.org/10.1016/0014-5793(93)80253-q.

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3

Kuper, C. F., D. M. H. Hameleers, J. P. Bruijntjes, I. van der Ven, J. Biewenga, and T. Sminia. "Lymphoid and non-lymphoid cells in nasal-associated lymphoid tissue (NALT) in the rat." Cell and Tissue Research 259, no. 2 (1990): 371–77. http://dx.doi.org/10.1007/bf00318460.

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4

Abdulla, Zainalabideen, Helen Turley, Kevin Gatter, and Francesco Pezzella. "Immunohistological recognition of cyclin D1 expression by non-lymphoid cells among lymphoid neoplastic cells." APMIS 122, no. 3 (2013): 183–91. http://dx.doi.org/10.1111/apm.12123.

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5

PALMEN, M. J. H. J., L. A. DIELEMAN, M. B. ENDE, et al. "Non-lymphoid and lymphoid cells in acute, chronic and relapsing experimental colitis." Clinical & Experimental Immunology 99, no. 2 (2008): 226–32. http://dx.doi.org/10.1111/j.1365-2249.1995.tb05537.x.

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6

Vezzoni, Paolo, Sonia Levi, Elena Gabri, Maria Rosa Pozzi, Silvia Spinazze, and Paolo Arosio. "Ferritins in malignant and non-malignant lymphoid cells." British Journal of Haematology 62, no. 1 (1986): 105–10. http://dx.doi.org/10.1111/j.1365-2141.1986.tb02905.x.

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7

Dugast, A. S., and B. Vanhove. "Immune regulation by non-lymphoid cells in transplantation." Clinical & Experimental Immunology 156, no. 1 (2009): 25–34. http://dx.doi.org/10.1111/j.1365-2249.2009.03877.x.

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8

SMINIA, T., A. VANASSELT, M. VDENDE, D. OFHISTOLOGYMEDICAL, and F. FREEUNIVERSITYAMSTERDAMTHEN. "Ontogeny of non-lymphoid cells in rat thymus." Developmental & Comparative Immunology 10, no. 1 (1986): 119. http://dx.doi.org/10.1016/0145-305x(86)90094-7.

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9

Dijkstra, C. D., and G. Kraal. "Non-lymphoid cells in the splenic white pulp." Research in Immunology 142, no. 4 (1991): 325–28. http://dx.doi.org/10.1016/0923-2494(91)90083-u.

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10

Huggins, Matthew A., Changwei Peng, Kelsey M. Wanhainen, Abigail R. Gress, Stephen C. Jameson, and Sara E. Hamilton. "Circulating KLRG1+ memory CD8 T cells support immune responses in non-lymphoid tissues." Journal of Immunology 206, no. 1_Supplement (2021): 98.25. http://dx.doi.org/10.4049/jimmunol.206.supp.98.25.

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Abstract The memory T cell population must be durable and capable of responding with rapid effector functions during re-encounter with pathogens. This is accomplished by forming a memory CD8 T cell pool containing multiple distinct subsets of cells that combine varying functional and homing characteristics. At steady-state, memory T cells expressing KLRG1 are restricted to circulation and patrol the host from within the vasculature. These cells convey robust protection during acute rechallenge with pathogens delivered intravenously. Given their localization pattern, we questioned whether KLRG1

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11

Li, Chaofan, Reshma Taneja, and Jie Sun. "Bhlhe40 maintains CD8+ T cell fitness and functionality in non-lymphoid tissues and tumors." Journal of Immunology 200, no. 1_Supplement (2018): 111.4. http://dx.doi.org/10.4049/jimmunol.200.supp.111.4.

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Abstract Recent evidence has suggested that tissue-resident and tumor-infiltrating CD8 T cells share common molecular signatures for their maintenance in the non-lymphoid environment. Currently, the molecular mechanisms regulating the residency, health and function of effector and memory CD8 T cells in non-lymphoid tissue remain largely elusive. Here we have identified that the transcription factor Bhlhe40 is critical for the fitness and function of effector & memory CD8 T cells in the non-lymphoid tissue and tumor. Bhlhe40 and its associated gene signature are highly expressed in CD8 T ce

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12

Zhenilo, Svetlana, Igor Deyev, Sergey Serov, and Oleg Polanovsky. "Regulation of oct-1 gene transcription is different in lymphoid and non-lymphoid cells." Biochimie 85, no. 7 (2003): 715–18. http://dx.doi.org/10.1016/s0300-9084(03)00121-4.

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13

Foxwell, Brian M. J., Gaëtane Woerly, Holger Husi, et al. "Identification of several cyclosporine binding proteins in lymphoid and non-lymphoid cells in vivo." Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease 1138, no. 2 (1992): 115–21. http://dx.doi.org/10.1016/0925-4439(92)90050-w.

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14

ROWISKI, J., P. SICISKI, and J. WARCHO. "Non-random distribution of lymphoid cells within the follicle-associated epithelium is caused by lymphoid cells associated with M cells." Cell Biology International Reports 9, no. 2 (1985): 115. http://dx.doi.org/10.1016/0309-1651(85)90084-0.

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15

Chen, Zhengshan, Xiaoyan Qiu, and Jiang Gu. "Immunoglobulin Expression in Non-Lymphoid Lineage and Neoplastic Cells." American Journal of Pathology 174, no. 4 (2009): 1139–48. http://dx.doi.org/10.2353/ajpath.2009.080879.

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16

Inman, Charlotte F., Tamsin Zangerle Murray, Mick Bailey, and Stephen Cose. "Most B cells in non‐lymphoid tissues are naïve." Immunology & Cell Biology 90, no. 2 (2011): 235–42. http://dx.doi.org/10.1038/icb.2011.35.

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17

van den Oord, J. J., R. De Vos, F. Facchetti, C. De Wolf-Peeters, and V. J. Desmet. "Identification of non-lymphoid cells in inflammatory liver disease." Biochimica et Biophysica Acta (BBA) - Bioenergetics 971, no. 1 (1988): S190. http://dx.doi.org/10.1016/s0005-2728(88)80018-5.

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18

Peng, Changwei, Matthew A. Huggins, Kelsey M. Wanhainen, et al. "Engagement of ICOS in tissues promotes establishment of CD8+ tissue-resident memory T cells." Journal of Immunology 206, no. 1_Supplement (2021): 24.06. http://dx.doi.org/10.4049/jimmunol.206.supp.24.06.

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Abstract Resident memory T cells (TRM) provide localized protective immunity in non-lymphoid tissues, but the signals and interactions within non-lymphoid tissues involved in generating TRM are incompletely defined. Inducible T-cell costimulator (ICOS) is a costimulatory molecule with well elaborated roles in regulating differentiation of CD4+ T cell populations, its function in CD8+ T cells is largely unexplored. With acute infection models, we found that ICOS-deficient CD8+ T cells showed minimal defects in the initial effector expansion and the generation of recirculating memory subsets. Ho

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19

Payne, C. M., A. Linde, R. Kibler, B. Poulos, L. Glasser, and R. Fiederlein. "Surface features of human natural killer cells and antibody-dependent cytotoxic cells." Journal of Cell Science 77, no. 1 (1985): 27–46. http://dx.doi.org/10.1242/jcs.77.1.27.

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The purpose of the present study was to examine the surface features of purified large granular lymphocytes (LGLs) (natural killer (NK) cells, antibody-dependent cytotoxic lymphoid (ADCL) cells, K-cells, Fc gamma (+) third population (non-T, non-B) lymphoid cells, T gamma cells) by scanning electron microscopy (SEM) and to compare their surface features with granulocytes, monocytes and Fc gamma (-) lymphoid cells that were all fixed for SEM under identical conditions. We have determined that 72–80% of LGLs enriched by rosette formation with sensitized erythrocytes or using Percoll gradients, h

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20

Lorenzo-Anota, Helen Yarimet, Diana G. Zarate-Triviño, Jorge Alberto Uribe-Echeverría, et al. "Chitosan-Coated Gold Nanoparticles Induce Low Cytotoxicity and Low ROS Production in Primary Leucocytes, Independent of Their Proliferative Status." Pharmaceutics 13, no. 7 (2021): 942. http://dx.doi.org/10.3390/pharmaceutics13070942.

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(1) Background: Chitosan-coated gold nanoparticles (CH-AuNPs) have important theranostic applications in biomedical sciences, including cancer research. However, although cell cytotoxicity has been studied in cancerous cells, little is known about their effect in proliferating primary leukocytes. Here, we assessed the effect of CH-AuNPs and the implication of ROS on non-cancerous endothelial and fibroblast cell lines and in proliferative lymphoid cells. (2) Methods: The Turkevich method was used to synthetize gold nanoparticles. We tested cell viability, cell death, ROS production, and cell cy

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21

Boss, Valerie, Deepa J. Talpade, and Thomas J. Murphy. "Induction of NFAT-mediated Transcription by G-coupled Receptors in Lymphoid and Non-lymphoid Cells." Journal of Biological Chemistry 271, no. 18 (1996): 10429–32. http://dx.doi.org/10.1074/jbc.271.18.10429.

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22

Műzes, Györgyi, Bettina Bohusné Barta, and Ferenc Sipos. "Colitis and Colorectal Carcinogenesis: The Focus on Isolated Lymphoid Follicles." Biomedicines 10, no. 2 (2022): 226. http://dx.doi.org/10.3390/biomedicines10020226.

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Gut-associated lymphoid tissue is one of the most diverse and complex immune compartments in the human body. The subepithelial compartment of the gut consists of immune cells of innate and adaptive immunity, non-hematopoietic mesenchymal cells, and stem cells of different origins, and is organized into secondary (and even tertiary) lymphoid organs, such as Peyer’s patches, cryptopatches, and isolated lymphoid follicles. The function of isolated lymphoid follicles is multifaceted; they play a role in the development and regeneration of the large intestine and the maintenance of (immune) homeost

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23

Borikar, Sneha, Vivek Philip, Lauren Kuffler, and Jennifer J. Trowbridge. "Lysine Methyltransferase Kmt5a Restricts Myeloid-Biased Output of Lymphoid-Primed Multipotent Progenitors." Blood 128, no. 22 (2016): 1487. http://dx.doi.org/10.1182/blood.v128.22.1487.1487.

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Abstract Distinct, lineage-biased subsets of multipotent progenitor cells (MPP) dynamically respond to the demands of the hematopoietic system to replenish mature hematopoietic cells as needed. It currently remains unclear as to whether distinct epigenetic mechanisms regulate lineage-specific expansion and differentiation from MPPs. Focusing on lymphoid-primed multipotent progenitor cells (LMPP/MPP4), we performed a lentiviral shRNA screen of 15 epigenetic factors, selected based on differential expression between myeloid-restricted and lymphoid-restricted progenitors. Following a 48 hour infe

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24

Niedobitek, G., H. Herbst, LS Young, et al. "Patterns of Epstein-Barr virus infection in non-neoplastic lymphoid tissue." Blood 79, no. 10 (1992): 2520–26. http://dx.doi.org/10.1182/blood.v79.10.2520.2520.

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Abstract Taking advantage of the abundant expression of the small Epstein-Barr virus (EBV)-encoded RNAs (EBERs) in latently infected cells, we have analyzed 72 normal and hyperplastic lymph nodes and three tonsils of acute infectious mononucleosis (IM) for the presence and distribution of EBV+ cells using EBER-specific in situ hybridization, in some cases combined with immunohistologic demonstration of cell type- characteristic antigens. In IM, large numbers of EBV+ lymphoid B blasts were detectable in extrafollicular areas, whereas germinal centers were generally free of EBV+ cells. In reacti

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25

Niedobitek, G., H. Herbst, LS Young, et al. "Patterns of Epstein-Barr virus infection in non-neoplastic lymphoid tissue." Blood 79, no. 10 (1992): 2520–26. http://dx.doi.org/10.1182/blood.v79.10.2520.bloodjournal79102520.

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Taking advantage of the abundant expression of the small Epstein-Barr virus (EBV)-encoded RNAs (EBERs) in latently infected cells, we have analyzed 72 normal and hyperplastic lymph nodes and three tonsils of acute infectious mononucleosis (IM) for the presence and distribution of EBV+ cells using EBER-specific in situ hybridization, in some cases combined with immunohistologic demonstration of cell type- characteristic antigens. In IM, large numbers of EBV+ lymphoid B blasts were detectable in extrafollicular areas, whereas germinal centers were generally free of EBV+ cells. In reactive lymph

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26

Matsuura, Y., T. Matsuoka, and Y. Fuse. "Ultrastructural and immunohistochemical studies on the ontogenic development of bronchus-associated lymphoid tissue (BALT) in the rat: special reference to follicular dendritic cells." European Respiratory Journal 5, no. 7 (1992): 824–28. http://dx.doi.org/10.1183/09031936.93.05070824.

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The ontogenic development of lymphoid and non-lymphoid cells in bronchus-associated lymphoid tissue (BALT) of the rat was studied ultrastructurally and immunohistochemically. In the late foetal period, only the alveolar macrophages showed a weak positivity for leucocyte common antigen, but no immune region associated (Ia) antigen was detected by monoclonal antibody, MAS 043. Mast cells were present. At 6 days of age, Ia-positive cells were observed in the alveolar wall and peribronchial interstitial tissue, and ultrastructurally there was an aggregation of the fibroblastoid mesenchymal cells.

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27

Smith, Corinne, Holly Turula, and Christopher Snyder. "Systemic hematogenous maintenance of memory inflation by MCMV infection (VIR4P.1013)." Journal of Immunology 192, no. 1_Supplement (2014): 143.8. http://dx.doi.org/10.4049/jimmunol.192.supp.143.8.

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Abstract Cytomegalovirus (CMV) is a β-herpesvirus that establishes a latent infection and supports the life-long maintenance of very large numbers of virus-specific effector T cells. These T cells remain functional and are found in many organs in the body. Current models propose that, to sustain these populations, reservoirs of viral antigen and/or latently infected cells in lymph nodes stimulate T cell proliferation and differentiation, followed by migration of progeny to non-lymphoid tissues. We tested this model using murine CMV (MCMV). While MCMV transcriptional activity during latency was

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28

Pawelec, G., H. ff S. Davies, J. D. Pearson, C. Steele, and G. Brons. "Stimulation of Lymphocyte Proliferation by Non-Lymphoid Porcine Tissue Cells." Tissue Antigens 14, no. 5 (2008): 367–78. http://dx.doi.org/10.1111/j.1399-0039.1979.tb00865.x.

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29

PENDINO, KIMBERLY J., K. P. CHEPENIK, and R. R. SCHMIDT. "Differential eicosanoid synthesis by murine fetal thymic non-lymphoid cells." Immunology and Cell Biology 70, no. 4 (1992): 237–52. http://dx.doi.org/10.1038/icb.1992.31.

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30

Jeurissen, Suzan H. M., E. Marga Janse, Guus Koch, and Gerben F. De Boer. "6.9 Distribution and function of non-lymphoid cells in chickens." Developmental & Comparative Immunology 13, no. 4 (1989): 413. http://dx.doi.org/10.1016/0145-305x(89)90136-5.

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31

van den Oord, Joost J., Rita de Vos, Fabio Facchetti, Jan Delabie, Chris De Wolf-Peeters, and Valeer J. Desmet. "Distribution of non-lymphoid, inflammatory cells in chronic HBV infection." Journal of Pathology 160, no. 3 (1990): 223–30. http://dx.doi.org/10.1002/path.1711600308.

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32

Anubha, Bajaj. "The Sentient Propagation-Reactive B Cell-Rich Lymphoid Proliferation." Annals of Cytology and Pathology 9, no. 1 (2024): 020–24. http://dx.doi.org/10.17352/acp.000032.

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Reactive B cell-rich lymphoid proliferation is a heterogeneous group of non-neoplastic lymphoid cell proliferation recapitulating diverse B cell lymphomas. The lesion is commonly categorized as nodal and extra-nodal follicular proliferations, nodal and extra-nodal nodular proliferations or nodal and extra-nodal immunoblastic proliferations. Florid follicular hyperplasia is comprised of quantifiably enhanced, disseminated primary and secondary lymphoid follicles with irregular outlines. Hyperplastic Germinal Centres (GCs) are comprised of admixed centroblasts and centrocytes, reactive T cells,

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33

Caucheteux, Stephan M., Souheil A. Younes, and William E. Paul. "CD45RB expression refines delineation of memory CD4 T cells and aids in understanding their development (91.8)." Journal of Immunology 178, no. 1_Supplement (2007): S161. http://dx.doi.org/10.4049/jimmunol.178.supp.91.8.

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Abstract Upon immunization, CD4 T cell stimulation leads to the development of effectors cells and of 2 major subsets of memory cells: central and effector memory cells, the latter often defined by their differential expression of CD62L and CD44. It is still unclear whether central memory cells derive from effectors or are directly induced. Our results have shown that CD44hi cells can be divided into 2 different subtypes depending on their expression level of CD45RB, providing a precise distinction among central and effector memory CD4 T cells. Using these markers, we show that while central m

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34

Klein, S. C., S. H. van der Burg, L. H. Boer, et al. "Targeting of cytotoxic T cells against leukemic B cells by bispecific antibody (aCD3 x aCD19) does not distract the T cell from its primary target." Journal of Immunology 159, no. 11 (1997): 5545–49. http://dx.doi.org/10.4049/jimmunol.159.11.5545.

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Abstract Bispecific Abs (BsAb) represent a novel format of immunotherapy, recognizing immune effector cells (e.g., T cells), on the one hand, and target cells (e.g., tumor cells), on the other hand. To be successful, cross-linking of the two cell types is necessary for effector cell activation and subsequent killing of the malignant target cells. We asked the question, whether CTL that were incubated with the BsAb aCD3 x aCD19 and malignant B cells and activated to kill the malignant B cells were still able to eliminate their natural target cells (e.g., virus-infected autologous body cells). T

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35

IV, Sainova. "Development of Techniques about Production of Recombinant vaccines and cells with Activated Immunogenic potential." Virology & Immunology Journal 4, no. 1 (2020): 1–4. http://dx.doi.org/10.23880/vij-16000232.

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Methods about derivation of myeloid-like and lymphoid-like cells from normal embryonic fibroblasts were developed and tested. Balb/c mice normal mouse embryonic cells from line 3T3 were co-cultivated with mouse malignant myeloma cells, containing inserted. Murine Leukemia Virus (MuLV) (with RNA-genome, Retroviridae family). For this goal, separate subpopulation of normal mouse embryonic fibroblasts were pre-incubated in cultural fluid, supplemented by previous incubation of myeloma cells in it, after subsequent centrifugation and filtration. Other sub-populations of 3T3 cells were co-cultivate

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36

Sowell, Ryan, Magdalena Rogozinska, and Amanda Marzo. "Rapamycin inhibits generation of memory CD8 T cells that localize to mucosal tissues (46.23)." Journal of Immunology 186, no. 1_Supplement (2011): 46.23. http://dx.doi.org/10.4049/jimmunol.186.supp.46.23.

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Abstract A limitation of current vaccine strategies is the inability to generate effective memory CD8 T cell responses in locations relevant to disease transmission. There is a gap in our knowledge of how memory CD8 T cells are generated and maintained in vivo in different tissues. In response to vaccination, CD8 T cells expand, differentiate into effectors and then undergo contraction. The end result is a population of memory CD8 T cells that survive this process. The mammalian target of rapamycin (mTOR) kinase is a critical regulator of many cell processes including proliferation, differenti

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37

Rothkotter, H. J., T. Kirchhoff, and R. Pabst. "Lymphoid and non-lymphoid cells in the epithelium and lamina propria of intestinal mucosa of pigs." Gut 35, no. 11 (1994): 1582–89. http://dx.doi.org/10.1136/gut.35.11.1582.

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38

van der Brugge-Gamelkoorn, Gerda J., Marja B. van de Ende, and T. Sminia. "Non-lymphoid cells of bronchus-associated lymphoid tissue of the rat in situ and in suspension." Cell and Tissue Research 239, no. 1 (1985): 177–82. http://dx.doi.org/10.1007/bf00214917.

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39

Peng, Changwei, and Stephen C. Jameson. "The relationship between CD4+ follicular helper T cells and CD8+ resident memory T cells: sisters or distant cousins?" International Immunology 32, no. 9 (2020): 583–87. http://dx.doi.org/10.1093/intimm/dxaa045.

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Abstract Independent studies over the last decade have characterized the properties of non-circulating CD8+ ‘resident’ memory T cells (TRM), which offer barrier protective immunity in non-lymphoid tissues and CD4+ follicular helper T cells (TFH), which mediate B-cell help in lymphoid sites. Despite their very different biological roles in the immune system, intriguing parallels have been noted between the trafficking properties and differentiation cues of these populations, parallels which have only sharpened with recent findings. In this review, we explore the features that underlie these sim

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40

Sullivan, Jenna M., Barbara Hoellbacher, and Daniel J. Campbell. "Id3 expression defines anatomically and functionally distinct regulatory T cells." Journal of Immunology 200, no. 1_Supplement (2018): 116.13. http://dx.doi.org/10.4049/jimmunol.200.supp.116.13.

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Abstract The Id (or inhibitor of DNA binding) proteins are small proteins that contain a helix-loop-helix domain and can heterodimerize with E-protein transcription factors. However, the Id proteins lack a DNA binding domain and act as natural dominant negatives of E-protein function. As a powerful transcriptional regulator, Id3 helps control the functional differentiation of CD8+ effector T cells and its expression is essential for regulatory T cell (TR) maintenance and function. Here we show that expression of Id3 is dynamically regulated in TR in a subset- and tissue-specific fashion. Withi

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41

Kinoshita, Kazuo, Munehiro Uemura, Takahiro Shimizu, Shun Kinoshita, and Hiroyuki Marusawa. "Stepwise generation of AID knock-in and conditional knockout mice from a single gene-targeting event." International Immunology 33, no. 7 (2021): 387–98. http://dx.doi.org/10.1093/intimm/dxab019.

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Abstract Activation-induced cytidine deaminase (AID) encoded by the Aicda gene initiates class-switch recombination and somatic hypermutation of immunoglobulin genes. In addition to this function, AID is also implicated in the epigenetic regulation in pluripotent stem cells and in the oncogenesis of lymphoid and non-lymphoid origins. To examine AID’s role in specific cell types, we developed mouse strains of conditional knockout (Aicda-FL) and knock-in with a red fluorescent protein gene (RFP) inserted into the Aicda locus (Aicda-RFP). These two strains were obtained from a single targeting ev

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42

Baghdadi, Hamed, Reza Heidari, Mahdi Zavvar, et al. "Long Non-Coding RNA Signatures in Lymphopoiesis and Lymphoid Malignancies." Non-Coding RNA 9, no. 4 (2023): 44. http://dx.doi.org/10.3390/ncrna9040044.

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Lymphoid cells play a critical role in the immune system, which includes three subgroups of T, B, and NK cells. Recognition of the complexity of the human genetics transcriptome in lymphopoiesis has revolutionized our understanding of the regulatory potential of RNA in normal lymphopoiesis and lymphoid malignancies. Long non-coding RNAs (lncRNAs) are a class of RNA molecules greater than 200 nucleotides in length. LncRNAs have recently attracted much attention due to their critical roles in various biological processes, including gene regulation, chromatin organization, and cell cycle control.

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43

Cooper, Christopher L., Richard R. Hardy, Michael Reth, and Stephen Desiderio. "Non–cell-autonomous hedgehog signaling promotes murine B lymphopoiesis from hematopoietic progenitors." Blood 119, no. 23 (2012): 5438–48. http://dx.doi.org/10.1182/blood-2011-12-397976.

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Abstract The role of hedgehog (Hh) signaling in B lymphopoiesis has remained unclear. We observed that the proliferation of pro-B cells in stromal cocultures was impaired by interruption of Hh signaling, prompting us to investigate whether the target of Hh antagonism was intrinsic or extrinsic to the B-lymphoid compartment. In the present study, using conditional deletion of the pathway activator gene Smo, we found that cell-autonomous Hh signaling is dispensable for B-cell development, B-lymphoid repopulation of the BM, and humoral immune function. In contrast, depletion of the Smo protein fr

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44

Zheng, Mingzhu, Kairui Mao, Difeng Fang, et al. "B cell residency but not T cell–independent IgA switching in the gut requires innate lymphoid cells." Proceedings of the National Academy of Sciences 118, no. 27 (2021): e2106754118. http://dx.doi.org/10.1073/pnas.2106754118.

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Immunoglobulin A (IgA)–producing plasma cells derived from conventional B cells in the gut play an important role in maintaining the homeostasis of gut flora. Both T cell–dependent and T cell–independent IgA class switching occurs in the lymphoid structures in the gut, whose formation depends on lymphoid tissue inducers (LTis), a subset of innate lymphoid cells (ILCs). However, our knowledge on the functions of non-LTi helper-like ILCs, the innate counter parts of CD4 T helper cells, in promoting IgA production is still limited. By cell adoptive transfer and utilizing a unique mouse strain, we

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45

Fix, A. S., and L. H. Arp. "Conjunctiva-associated Lymphoid Tissue (CALT) in Normal and Bordetella avium-infected Turkeys." Veterinary Pathology 26, no. 3 (1989): 222–30. http://dx.doi.org/10.1177/030098588902600306.

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Conjunctiva-associated lymphoid tissue (CALT) was characterized in normal and Bordetella avium-infected turkey poults during the first 5 weeks of life. At 1, 5, 12, 19, 25, and 33 days post-hatching (DPH), upper and lower eyelids were examined by gross, histologic, and electron microscopic techniques. CALT was confined to the proximal part of the lower eyelid near the conjunctival fornix; it appeared by 5 DPH as individual lymphoid nodules and as dense masses by 19 DPH. In the upper eyelid, CALT was present only as isolated nodules. Histologically. CALT was composed of dense lymphocyte infiltr

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46

Carmichael, Catherine L., Donald Metcalf, Katya J. Henley, et al. "Hematopoietic overexpression of the transcription factor Erg induces lymphoid and erythro-megakaryocytic leukemia." Proceedings of the National Academy of Sciences 109, no. 38 (2012): 15437–42. http://dx.doi.org/10.1073/pnas.1213454109.

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The transcription factor encoded by the E-twenty-six (ETS)-related gene, ERG, is an essential regulator of hematopoietic stem cell function and a potent human oncoprotein. Enforced expression of ERG in murine hematopoietic cells leads to the development of a well-characterized lymphoid leukemia and a less well-defined non lymphoid disease. To clarify the latter, we generated murine bone marrow chimeras with enforced Erg expression in engrafted hematopoietic progenitor cells. As expected, these mice developed lymphoid leukemia. However, the previously reported non lymphoid disease that develope

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47

Giorno, R. "Immunohistochemical analysis of human peripheral blood and lymphoid tissues using monoclonal antibodies immunoreactive with non-lymphoid cells." Histochemistry 84, no. 3 (1986): 241–45. http://dx.doi.org/10.1007/bf00495789.

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Chuluunbaatar, Tsolmon, Osamu Ichii, Md Abdul Masum, Takashi Namba, and Yasuhiro Kon. "Morphological Characteristics of Genital Organ-Associated Lymphoid Tissue in the Vaginal Vestibule of Goats and Pigs." Veterinary Sciences 10, no. 1 (2023): 51. http://dx.doi.org/10.3390/vetsci10010051.

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Abstract:

Mucosa-associated lymphoid tissue (MALT) is a specialized form of peripheral lymphoid tissue (LT), which is found on mucosal surfaces exposed to the environment. However, morphological data of these tissues in farm animals are scarce. This study investigated the gross anatomical and histological features of genital organ-associated lymphoid tissues (GOALTs) in the vaginal vestibule (VV) of healthy, non-pregnant, adult goats and pigs. Their VVs were composed of stratified squamous, non-keratinized epithelium, and various-sized dark-blue hematoxylin-positive spots were observed in whole-mount sp

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Cairo, Gaetano, Paolo Vezzoni, Lidia Bardella, et al. "Regulation of ferritin synthesis in malignant and non-malignant lymphoid cells." Biochemical and Biophysical Research Communications 139, no. 2 (1986): 652–57. http://dx.doi.org/10.1016/s0006-291x(86)80040-7.

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Klein, Benjamin Y., and Sherie L. Morrison. "Expression of genes containing the IgH enhancer in non-lymphoid cells." Molecular Immunology 27, no. 8 (1990): 713–22. http://dx.doi.org/10.1016/0161-5890(90)90080-j.

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