Relevant bibliographies by topics / Non-mendelian inheritance

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Non-mendelian inheritance'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Non-mendelian inheritance"

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Clausen, J. "Non-Mendelian Inheritance in Viola." Hereditas 9, no. 1-3 (2010): 245–56. http://dx.doi.org/10.1111/j.1601-5223.1927.tb03526.x.

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Hernandez, Dena G., Xylena Reed, and Andrew B. Singleton. "Genetics in Parkinson disease: Mendelian versus non-Mendelian inheritance." Journal of Neurochemistry 139 (April 18, 2016): 59–74. http://dx.doi.org/10.1111/jnc.13593.

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Aiello, Lisa B., and Beth Desaretz Chiatti. "Primer in Genetics and Genomics, Article 4—Inheritance Patterns." Biological Research For Nursing 19, no. 4 (2017): 465–72. http://dx.doi.org/10.1177/1099800417708616.

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Since the completion of the Human Genome Project, much has been uncovered about inheritance of various illnesses and disorders. There are two main types of inheritance: Mendelian and non-Mendelian. Mendelian inheritance includes autosomal dominant, autosomal recessive, X-linked, and Y-linked inheritance. Non-Mendelian inheritance includes mitochondrial and multifactorial inheritance. Nurses must understand the types of inheritance in order to identify red flags that may indicate the possibility of a hereditary disorder in a patient or family.
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Paul Tian, Jianjun. "Algebraic model of non-Mendelian inheritance." Discrete & Continuous Dynamical Systems - S 4, no. 6 (2011): 1577–86. http://dx.doi.org/10.3934/dcdss.2011.4.1577.

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Vigé, Alexandre, Catherine Gallou-Kabani, and Claudine Junien. "Sexual Dimorphism in Non-Mendelian Inheritance." Pediatric Research 63, no. 4 (2008): 340–47. http://dx.doi.org/10.1203/pdr.0b013e318165b896.

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Wilby, A. S., and J. S. Parker. "Mendelian and non-Mendelian inheritance of newly-arisen chromosome rearrangements." Heredity 60, no. 2 (1988): 263–68. http://dx.doi.org/10.1038/hdy.1988.41.

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Bis-Brewer, Dana M., Ziv Gan-Or, Patrick Sleiman, et al. "Assessing non-Mendelian inheritance in inherited axonopathies." Genetics in Medicine 22, no. 12 (2020): 2114–19. http://dx.doi.org/10.1038/s41436-020-0924-0.

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Lee, Robert W., and Claude Lemieux. "BIPARENTAL INHERITANCE OF NON-MENDELIAN GENE MARKERS IN CHLAMYDOMONAS MOEWUSII." Genetics 113, no. 3 (1986): 589–600. http://dx.doi.org/10.1093/genetics/113.3.589.

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ABSTRACT The first two non-Mendelian gene mutations to be identified in Chlamydomonas moewusii are described. These putative chloroplast gene mutations include one for resistance to streptomycin (sr-nM1) and one for resistance to erythromycin (er-nM1). In one- and two-factor reciprocal crosses, usually over 90% of the germinating zygospores transmitted these mutations and their wild-type alternatives from both parents (biparental zygospores); the remaining zygospores transmitted exclusively the non-Mendelian markers of the mating-type "plus" parent. Among the biparental zygospores, a strong bi
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van Heyningen, V. "Mechanisms of non-Mendelian inheritance in genetic disease." Human Molecular Genetics 13, suppl_2 (2004): R225—R233. http://dx.doi.org/10.1093/hmg/ddh254.

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DINI, FERNANDO, LEA K. BLEYMAN, and PAOLA GIUBBILINI. "Non-Mendelian Inheritance of Early Maturity inEuplotes crassus." Journal of Protozoology 37, no. 6 (1990): 475–78. http://dx.doi.org/10.1111/j.1550-7408.1990.tb01250.x.

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Dissertations / Theses on the topic "Non-mendelian inheritance"

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Deans, Natalie Christine. "Molecular mechanisms that underlie non-Mendelian inheritance patterns in Zea mays." The Ohio State University, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu159541311114259.

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Singh, Deepankar Pratap. "Non-Mendelian inheritance of mating-type determination in Paramecium tetraurelia : Role of the scnRNA pathway." Paris 6, 2013. http://www.theses.fr/2013PA066176.

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Cette thèse est une étude des mécanismes moléculaires de la détermination des types sexuels chez le cilié Paramecium tetraurelia, et de leur hérédité non mendélienne. L’identification de mutations affectant les types sexuels montre que le type E dépend de l’expression de la protéine trans-membranaire mtA, elle-même dépendant des facteurs de transcription mtB et mtC. Le type O est déterminé par l’excision du promoteur de mtA pendant le développement du macronoyau somatique à partir du micronoyau germinal, un stade où le génome est entièrement réarrangé pour éliminer éléments transposables et sé
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Schilling, Taylor. "Non-Mendelian Inheritance in C. elegans: A Violation of The Law of Independent Assortment." Cleveland State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=csu1611776149127827.

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Khalid, Aaron Munir. "An assessment of genomic sequence restoration in Arabidopsis thaliana." Thesis, 2009. http://hdl.handle.net/10012/4621.

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A fundamental tenet of classical Mendelian genetics is that allelic information is stably transmitted from parent to progeny. Work in our laboratory has revealed a novel exception to this law where Arabidopsis thaliana plants homozygous for the recessive organ-fusion mutation hothead (hth) gave rise to phenotypically and genotypically wild-type (HTH) progeny at high frequencies. We have coined the term restoration to describe this phenomenon, since the reverted HTH allele was not detectable in the parental genome but was present in a recent ancestor (the grandparent). Recent work in our labora
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Marvanová, Hana. "Úloha energetického matebolismu při odchylce od Mendelovské dědičnosti v případě t-haplotypu u myší." Master's thesis, 2017. http://www.nusl.cz/ntk/nusl-367816.

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When two alleles carried by a heterozygote are transmitted unequally to the zygote at the time of fertilization, transmission ratio distortion occurs. The best studied example of this phenomenon in mammals is t-haplotype in mice. The mouse t-haplotype is a selfish variant region on chromosome 17, in nature transmitted as a unit. Male mice homozygous for t haplotype are sterile, but heterozygotes transmit the t haplotype up to 99% of their progeny. This is believed to be caused by motility differences between sperm carrying the t haplotype and wild-type sperm from the same heterozygous male. Th
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Book chapters on the topic "Non-mendelian inheritance"

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Kowles, Richard. "Non-Mendelian Inheritance." In Solving Problems in Genetics. Springer New York, 2001. http://dx.doi.org/10.1007/978-1-4613-0205-6_10.

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"Non-Mendelian Inheritance." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics. Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_11509.

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Silver, L. "Non-Mendelian Inheritance." In Encyclopedia of Genetics. Elsevier, 2001. http://dx.doi.org/10.1006/rwgn.2001.0904.

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Cuzin, F. "Non-Mendelian Inheritance." In Brenner's Encyclopedia of Genetics. Elsevier, 2013. http://dx.doi.org/10.1016/b978-0-12-374984-0.01058-5.

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"Non-Mendelian Inheritance." In Prenatal Diagnosis: Cases & Clinical Challenges. Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9780470696262.ch3.

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Avise, John C. "1909 Non-Mendelian Inheritance." In Conceptual Breakthroughs in Evolutionary Genetics. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-12-420166-8.00013-0.

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Harel, Tamar, Davut Pehlivan, C. Thomas Caskey, and James R. Lupski. "Mendelian, Non-Mendelian, Multigenic Inheritance, and Epigenetics." In Rosenberg's Molecular and Genetic Basis of Neurological and Psychiatric Disease. Elsevier, 2015. http://dx.doi.org/10.1016/b978-0-12-410529-4.00001-2.

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Butler, Merlin G., Michael Begleiter, Shannon Lillis, Molly Lund, and F. John Meaney. "Patterns of Inheritance: Mendelian and Non-Mendelian." In Genetics of Developmental Disabilities. CRC Press, 2019. http://dx.doi.org/10.1201/9780429264078-2.

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"Patterns of Inheritance: Mendelian and Non-Mendelian." In Genetics of Developmental Disabilities. CRC Press, 2005. http://dx.doi.org/10.1201/b14171-6.

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Harel, Tamar, and James R. Lupski. "Mendelian, non-Mendelian, multigenic inheritance, and epigenetics." In Rosenberg's Molecular and Genetic Basis of Neurological and Psychiatric Disease. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-813955-4.00001-5.

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