Relevant bibliographies by topics / Nontypeable Haemophilus influenzae

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Academic literature on the topic 'Nontypeable Haemophilus influenzae'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Nontypeable Haemophilus influenzae"

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Bakaletz, Lauren O., and Laura A. Novotny. "Nontypeable Haemophilus influenzae (NTHi)." Trends in Microbiology 26, no. 8 (2018): 727–28. http://dx.doi.org/10.1016/j.tim.2018.05.001.

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Clemans, Daniel L., Carl F. Marrs, Mayuri Patel, Michelle Duncan, and Janet R. Gilsdorf. "Comparative Analysis of Haemophilus influenzae hifA(Pilin) Genes." Infection and Immunity 66, no. 2 (1998): 656–63. http://dx.doi.org/10.1128/iai.66.2.656-663.1998.

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ABSTRACT Adherence of Haemophilus influenzae to epithelial cells plays a central role in colonization and is the first step in infection with this organism. Pili, which are large polymorphic surface proteins, have been shown to mediate the binding of H. influenzae to cells of the human respiratory tract. Earlier experiments have demonstrated that the major epitopes of H. influenzae pili are highly conformational and immunologically heterogenous; their subunit pilins are, however, immunologically homogenous. To define the extent of structural variation in pilins, which polymerize to form pili,
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Rubin, L. G., K. Staiman, and N. Kamani. "Occult bacteremia with nontypeable Haemophilus influenzae." Journal of Clinical Microbiology 25, no. 7 (1987): 1314–15. http://dx.doi.org/10.1128/jcm.25.7.1314-1315.1987.

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LaCross, Nathan C., Carl F. Marrs, and Janet R. Gilsdorf. "Population structure in nontypeable Haemophilus influenzae." Infection, Genetics and Evolution 14 (March 2013): 125–36. http://dx.doi.org/10.1016/j.meegid.2012.11.023.

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Foxwell, A. Ruth, Jennelle M. Kyd, and Allan W. Cripps. "Nontypeable Haemophilus influenzae: Pathogenesis and Prevention." Microbiology and Molecular Biology Reviews 62, no. 2 (1998): 294–308. http://dx.doi.org/10.1128/mmbr.62.2.294-308.1998.

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SUMMARY In this paper, we describe the ability of nontypeable Haemophilus influenzae (NTHi) to coexist with the human host and the devastating results associated with disruption of the delicate state of balanced pathogenesis, resulting in both acute and chronic respiratory tract infections. It has been seen that the strains of NTHi causing disease show a marked genetic and phenotypic diversity but that changes in the lipooligosaccharide (LOS) and protein size and antigenicity in chronically infected individuals indicate that individual strains of NTHi can remain and adapt themselves to avoid e
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Kubiet, Martin, Reuben Ramphal, Allan Weber, and Arnold Smith. "Pilus-Mediated Adherence of Haemophilus influenzae to Human Respiratory Mucins." Infection and Immunity 68, no. 6 (2000): 3362–67. http://dx.doi.org/10.1128/iai.68.6.3362-3367.2000.

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ABSTRACT Haemophilus influenzae, especially the nontypeable strains, are among the most common pathogens encountered in patients with chronic lung disease and otitis media. We and others have demonstrated that respiratory isolates of nontypeable H. influenzae bind to human mucins, but the mechanism of binding is not entirely clear. We have therefore examined the role of pili in the adherence of both type b and nontypeable H. influenzae to human respiratory mucins. We used isogenic H. influenzaestrains with a mutation in the structural gene for pilin (hifA), a laboratory H. influenzae strain tr
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Williams, Bryan J., Miriam Golomb, Thomas Phillips, Joshua Brownlee, Maynard V. Olson, and Arnold L. Smith. "Bacteriophage HP2 of Haemophilus influenzae." Journal of Bacteriology 184, no. 24 (2002): 6893–905. http://dx.doi.org/10.1128/jb.184.24.6893-6905.2002.

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ABSTRACT Temperate bacteriophages effect chromosomal evolution of their bacterial hosts, mediating rearrangements and the acquisition of novel genes from other taxa. Although the Haemophilus influenzae genome shows evidence of past phage-mediated lateral transfer, the phages presumed responsible have not been identified. To date, six different H. influenzae phages are known; of these, only the HP1/S2 group, which lyosogenizes exclusively Rd strains (which were originally encapsulated serotype d), is well characterized. Phages in this group are genetically very similar, with a highly conserved
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Winter, Linda E., and Stephen J. Barenkamp. "HumanAntibodies Specific for the High-Molecular-Weight Adhesion Proteins ofNontypeable Haemophilus influenzae Mediate OpsonophagocyticActivity." Infection and Immunity 71, no. 12 (2003): 6884–91. http://dx.doi.org/10.1128/iai.71.12.6884-6891.2003.

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ABSTRACT The HMW1- and HMW2-like adhesion proteins of nontypeable Haemophilus influenzae are expressed by 75% of these strains, and antibodies directed against these proteins are protective in animal models of infection. The purpose of the present study was to define the functional activity of human antibodies specific for these proteins in an in vitro complement-dependent opsonophagocytic assay. Human promyelocytic cell line HL-60 served as the source of phagocytic cells, and a commercial preparation of intravenous immunoglobulin (IVIG) served as the source of human antibodies. High-molecular
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Ketterer, Margaret R., Jian Q. Shao, Douglas B. Hornick, Ben Buscher, Venkata K. Bandi, and Michael A. Apicella. "Infection of Primary Human Bronchial Epithelial Cells by Haemophilus influenzae: Macropinocytosis as a Mechanism of Airway Epithelial Cell Entry." Infection and Immunity 67, no. 8 (1999): 4161–70. http://dx.doi.org/10.1128/iai.67.8.4161-4170.1999.

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ABSTRACT Nontypeable Haemophilus influenzae is an exclusive human pathogen which infects the respiratory epithelium. We have initiated studies to explore the interaction of the nontypeableH. influenzae strain 2019 with primary human airway epithelial cells by electron and confocal microscopy. Primary human airway cell cultures were established as monolayers on glass collagen-coated coverslips or on semipermeable membranes at an air-fluid interface. Scanning electron microscopy indicated that bacteria adhered to nonciliated cells in the population. The surface of infected cells showed evidence
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SUNAKAWA, Keisuke, Yuriko TAKEUCHI, and Satoshi IWATA. "Nontypeable Haemophilus influenzae （NTHi） Epidemiology." Kansenshogaku Zasshi 85, no. 3 (2011): 227–37. http://dx.doi.org/10.11150/kansenshogakuzasshi.85.227.

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Dissertations / Theses on the topic "Nontypeable Haemophilus influenzae"

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Tsao, David L. "Serum resistance of an invasive nontypeable H. influenzae." Diss., Columbia, Mo. : University of Missouri-Columbia, 2004. http://hdl.handle.net/10355/5808.

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Thesis (M.S.)--University of Missouri-Columbia, 2004.<br>"December, 2004." The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Includes bibliographical references.
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Puig, Pitarch Carmen. "Nontypeable Haemophilus influenzae: colonization, infection and biofilm formation." Doctoral thesis, Universitat de Barcelona, 2015. http://hdl.handle.net/10803/311616.

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Haemophilus influenzae is an opportunistic pathogen that forms part of the human nasopharyngeal microbiota. This microorganism is classified into encapsulated and nonencapsulated or nontypeable (NTHi) isolates, depending on the presence of a polysaccharide capsule. Although H. influenzae is a common respiratory commensal, it is also able to cause several infections, especially in patients with comorbidities. The most common respiratory infections in which H. influenzae can be identified as the main etiological agent are exacerbations in patients with Chronic Obstructive Pulmonary Disease (COPD
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Kunthalert, Duangkamol, and n/a. "Immunological and structural characterisation of the nontypeable Haemophilus influenzae vaccine protein OMP26." University of Canberra. Health Sciences, 2004. http://erl.canberra.edu.au./public/adt-AUC20060406.101830.

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Nontypeable Haemophilus influenzas (NTHi) is recognised as a significant human pathogen causing mild to severe respiratory tract infections. At present, no vaccine is available for prevention of infection caused by this pathogen. Several outer membrane proteins (OMPs) of NTHi and its lipooligosaccharide have been investigated as possible vaccine antigens against NTHi infections. Previous investigations in our laboratory have shown that OMP26 from an NTHi 289 strain was able to significantly enhance pulmonary clearance of NTHi in a rat model in which animals were immunised via intestinal Peyer'
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Foxwell, Alice Ruth, and n/a. "Mechanisms of immunity to nontypeable Haemophilus influenzae in the lung." University of Canberra. Applied Science, 1998. http://erl.canberra.edu.au./public/adt-AUC20060710.142114.

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Pulmonary infection caused by nontypeable Haemophilus influenzae (NTHi) is a significant cause of morbidity and mortality in both industrialised and developing countries. Previous work from this group resulted in the development of a respiratory model in rodents which has precipitated studies into the pathogenesis of infection by NTHi and investigation of the humoral and cellular mechanisms by which the bacteria are cleared from the lung. Comparison of mucosally immunised with non-immunised animals has demonstrated that not only are bacteria cleared more rapidly from the lungs, but there is a
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Staffen, Dana Jean. "Environmental Factors Influence Nontypeable Haemophilus influenzae Biofilm Formation, Maturation and Gene Expression." The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1385497645.

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Ho, Derek K. "lgtC expression mediates complement resistance in nontypeable Haemophilus influenzae strain R2866 /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/9308.

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McGrath, John Francis, and n/a. "Immunomodulation in the context of developing a nontypeable Haemophilus influenzae vaccine." University of Canberra. Health Sciences, 2007. http://erl.canberra.edu.au./public/adt-AUC20070726.152419.

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One of the major challenges of vaccine development is the conservation of immunogenicity and protective efficacy through the stages of design, production, formulation and delivery. The critical issue is that how and in what form an antigen is taken up by antigen presenting cells for proteolytic processing and presentation to the immune system bound to MHC can have dramatic effects on the activation of Th cells to drive clonal responses and induction of immunological memory. Nontypeable Haemophilus influenzae (NTHi) is a pathogenic commensal of the human respiratory tract that causes diseases w
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Spessot, Pietro. "Nontypeable Haemophilus influenzae eludes complement-mediated killing seizing C4b-binding protein." Doctoral thesis, Università di Siena, 2019. http://hdl.handle.net/11365/1078633.

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Nontypeable Haemophilus influenzae (NTHi) is a Gram negative, non-capsulated coccobacillus commensal of the nasopharyngeal tract. Acting as an opportunistic pathogen is responsible for different pathologies such as exacerbation of COPD, Otitis Media and in worst cases meningitis and sepsis. Given the great genetic variability among NTHi strains, the greatest challenge in the formulation of a vaccine against NTHi is the identification of cross protective antigens. Under the Novartis legacy (now GSK), several antigens were identified thanks to a reverse vaccinology and proteomic approaches. The
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Webb, Dianne, and n/a. "Nontypeable Haemophilus influenzae outer membrane protein analysis, isolation, characterisation and vaccine potential." University of Canberra. Applied Science, 1998. http://erl.canberra.edu.au./public/adt-AUC20061110.105953.

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Heterogeneity in immunodominant outer membrane proteins has been proposed as a significant factor in the failure of an NTHi infection to induce immune protection against subsequent infections. This study has examined the vaccine potential of three outer membrane proteins in an attempt to identify conserved regions that could be targeted by an immune response after vaccination. The three proteins investigated were: TbpB, P5 and P48 (HI0164). The optimal route of immunisation in clearing a bolus inoculum of NTHi to the lung in the rat has been shown to be a combination of gut sensitisation with
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Raffel, Forrest K. "Haemophilus Sap transporter dependent mechanisms to resist host innate and nutritional immunity influences pathogenesis." The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1388669277.

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Book chapters on the topic "Nontypeable Haemophilus influenzae"

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Thanavala, Yasmin, and Amit A. Lugade. "Role of Nontypeable Haemophilus influenzae in Otitis Media and Chronic Obstructive Pulmonary Disease." In Recent Advances in Tonsils and Mucosal Barriers of the Upper Airways. KARGER, 2011. http://dx.doi.org/10.1159/000324785.

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Spallone, Amy, and Daniel Musher. "Haemophilus." In Schlossberg's Clinical Infectious Disease, edited by Cheston B. Cunha. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190888367.003.0138.

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This chapter examines Haemophilus influenzae, an extracellular, fastidious Gram-negative coccobacillus that commonly colonizes the human upper respiratory tract. Until the early 1980’s, an encapsulated form, H. influenzae type b, was the prevalent recognized pathogen, causing invasive disease such as meningitis or bacteteremic pneumonia; since that time, widespread use of a protein-conjugated capsular polysaccharide vaccine has largely eliminated this organism from the population. Most Haenophilus disease at the present time is due to unencapsulated (nontypeable) isolates which are common caus
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Pelton, Stephen I. "Otitis." In Schlossberg's Clinical Infectious Disease, edited by Cheston B. Cunha. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190888367.003.0006.

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This chapter looks at otitis. The clinical burden from acute (AOM) and chronic suppurative otitis media (CSOM) and their associated morbidities is substantial, especially in children. In industrialized countries, AOM remains the most frequent reason for pediatric office visits and recurrent otitis media (ROM), or persistent middle ear fluid and associated hearing loss, reduces quality of life. In developing nations, infectious complications of AOM include suppurative intracranial infection and CSOM with severe hearing loss. The prescription of antimicrobials increases bacterial resistance, so
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Kyd, Jennelle M., Ajay Krishnamurthy, and Stephen Kidd. "Interactions and Mechanisms of Respiratory Tract Biofilms Involving Streptococcus Pneumoniae and Nontypeable Haemophilus Influenzae." In Microbial Biofilms - Importance and Applications. InTech, 2016. http://dx.doi.org/10.5772/63500.

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Conference papers on the topic "Nontypeable Haemophilus influenzae"

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Kimura, Genki, Yuki Nishimoto, Takahiro Nakaoki, Kazuhiro Ito, and Yasuo Kizawa. "Establishment of Nontypeable Haemophilus influenzae airway infection murine model." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.2322.

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Ackland, Jodie, David Cleary, Myron Christodoulides, Tom Wilkinson, and Karl Staples. "Strain-dependent effects of Nontypeable Haemophilus influenzae (NTHi) on human macrophage function." In Abstracts from the 17th ERS Lung Science Conference: ‘Mechanisms of Acute Exacerbation of Respiratory Disease’. European Respiratory Society, 2019. http://dx.doi.org/10.1183/23120541.lungscienceconference-2019.pp135.

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Freeman, Christine M., Fernando J. Martinez, MeiLan K. Han, et al. "Lung CD8+ T Cells From COPD Subjects Respond To Nontypeable Haemophilus Influenzae Stimulation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1284.

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Kc, Rajendra, Nicholas M. Harkness, Louise A. Cooley, et al. "A simple workflow for comparative analysis of longitudinal isolates of nontypeable Haemophilus influenzae." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2899.

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Saliu, Fabio, Giulia Rizzo, Alessandra Bragonzi, et al. "The persistence of Nontypeable Haemophilus Influenzae fuels type 17 immunity in the lung." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa2107.

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Lee, J., Y. Lu, N. Mohammad, K. Han, and M. L. Brantly. "The Exacerbation of the Inflammatory Response to Nontypeable Haemophilus Influenzae Bacteria by Alpha-Defensins." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4297.

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Stodden, H. S., F. Ritzmann, C. Herr, R. Bals, and C. Beisswenger. "Nontypeable Haemophilus influenzae supresses SP-B and SP-C expression in alveolar epithelial type II cells." In ERS Lung Science Conference 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.lsc-2021.40.

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Desai, Himanshu, Catherine Wrona, Timothy F. Murphy, and Sanjay Sethi. "Mechanisms Of Decline In Serum Bactericidal Activity For Nontypeable Haemophilus Influenzae (NTHI) In COPD Patients Chronically Colonized With NTHI." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2911.

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Roscioli, E., M. Poh, A. Kicic, et al. "Nontypeable Haemophilus Influenzae (NTHi) Exploits Disease-Related Defects in Xenophagy to Persist and Traffic Within the Epithelial Compartment During COPD." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5376.

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Sethi, Sanjay, Catherine Wrona, Ellana Eberhardt, Lori Grove, Ganapathi I. Parameswaran, and Timothy Murphy. "Distinguishing Infecting (New) From Colonizing (Pre-Existing) Nontypeable Haemophilus Influenzae Strains At Exacerbation Of COPD By Molecular And Immunological Assays." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4452.

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