Academic literature on the topic 'Normal Tension Glaucom'

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Journal articles on the topic "Normal Tension Glaucom"

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Anderson, DouglasR. "Normal-tension glaucoma (Low-tension glaucoma)." Indian Journal of Ophthalmology 59, no. 7 (2011): 97. http://dx.doi.org/10.4103/0301-4738.73695.

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Brodsky, M. C. "Normal tension glaucoma." British Journal of Ophthalmology 89, no. 9 (2005): 1228–29. http://dx.doi.org/10.1136/bjo.2005.073866.

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Anderson, Douglas R., William Feuer, and Joyce Schiffman. "Normal-Tension Glaucoma." Ophthalmology 115, no. 3 (2008): 590–91. http://dx.doi.org/10.1016/j.ophtha.2007.08.017.

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TANNA, A., and H. JAMPEL. "NORMAL-TENSION GLAUCOMA." Ophthalmology Clinics of North America 13, no. 3 (2000): 455–64. http://dx.doi.org/10.1016/s0896-1549(05)70206-6.

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Anderson, Douglas R., Stuart Graham, and Lutz Pillunat. "Normal-Tension Glaucoma." Journal of Glaucoma 12, no. 2 (2003): 164–66. http://dx.doi.org/10.1097/00061198-200304000-00012.

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Fan, Ning, Junkai Tan, and Xuyang Liu. "Is “normal tension glaucoma” glaucoma?" Medical Hypotheses 133 (December 2019): 109405. http://dx.doi.org/10.1016/j.mehy.2019.109405.

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Kuo, Debbie S., and Sanjay Asrani. "Normal-Tension Glaucoma Masqueraders." Journal of Glaucoma 26, no. 4 (2017): e153-e156. http://dx.doi.org/10.1097/ijg.0000000000000578.

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Girkin, Christopher A., Balwantray C. Chauhan, and Felipe A. Medeiros. "Progressive Normal-tension Glaucoma." Journal of Glaucoma 17, no. 7 (2008): 591–93. http://dx.doi.org/10.1097/ijg.0b013e31817efbd7.

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Lichter, Paul R. "Normal-tension glaucoma history." Ophthalmology 109, no. 1 (2002): 2. http://dx.doi.org/10.1016/s0161-6420(01)00906-x.

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Wilson, M. Roy, and M. S. Creighton. "Normal tension glaucomaa." Acta Ophthalmologica Scandinavica 80 (October 2002): 9–11. http://dx.doi.org/10.1034/j.1600-0420.80.s236.1.x.

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Dissertations / Theses on the topic "Normal Tension Glaucom"

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Li, Yuen-mei Emmy, and 李琬微. "Cost-effectiveness of treating normal tension glaucoma." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45173114.

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Mok, Kwok-hei. "The characterization of retinal nerve fiber layer thickness in normal, high-tension and normal-tension glaucoma using optical coherence tomography." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31381005.

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Mok, Kwok-hei, and 莫國熙. "The characterization of retinal nerve fiber layer thickness in normal,high-tension and normal-tension glaucoma using optical coherencetomography." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B31381005.

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Häntzschel, Janek, Naim Terai, Olga Furashova, Karin Pillunat, and Lutz E. Pillunat. "Comparison of Normal- and High-Tension Glaucoma: Nerve Fiber Layer and Optic Nerve Head Damage." Karger, 2014. https://tud.qucosa.de/id/qucosa%3A70550.

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Purpose: The aim of this study was to investigate differences in the nerve fiber layer and glaucoma-induced structural optic nerve head (ONH) damage in patients with normal- (NTG) and high-tension (HTG) glaucoma. Methods: In this retrospective pair-matched comparative study, 22 NTG and 22 HTG eyes were matched according to the same glaucomatous damage based on rim volume, rim area and disk size, as measured by Heidelberg retinal tomography (HRT III). Visual fields (VF) were assessed by Humphrey perimetry, and nerve fiber layer thickness was determined both by scanning laser polarimetry (GDxVCC
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Ekström, Curt. "Studies on the Epidemiology of Open-angle Glaucoma." Doctoral thesis, Uppsala University, Department of Neuroscience, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8323.

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<p>Glaucoma is a common disease in the elderly population. Open-angle glaucoma (OAG) is the predominant form of glaucoma. Chronic simple glaucoma and capsular glaucoma, characterized by the occurrence of pseudoexfoliation in the anterior eye segment, are the most frequent types of OAG. The purpose of the present thesis was to study the epidemiology of OAG in the municipality of Tierp, whose population has a high exposure to pseudoexfoliation.</p><p>In a case-finding study, the prevalence of known cases of OAG by December 31, 1983 was estimated to 1.4% in people ≥45 years of age. Sixty-three pe
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Dong, Jin. "Slope analysis of the optic disc in eyes with ocular hypertension and early normal-tension glaucoma by confocal scanning laser ophthalmoscope." Kyoto University, 2000. http://hdl.handle.net/2433/151429.

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Islam, Md Moydul. "Characterization of the Effect of Optineurin on Alpha-synuclein Aggregation andToxicity in Yeast." Wright State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=wright1535409230684852.

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Ferreira, Maria Inês Xavier 1992. "Normal tension glaucoma : literature review." Master's thesis, 2016. http://hdl.handle.net/10451/29032.

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Trabalho Final do Curso de Mestrado Integrado em Medicina, Faculdade de Medicina, Universidade de Lisboa, 2016<br>Normal tension glaucoma is a disease that causes glaucomatose damage to the optic nerve without the presence of elevated intraocular tensions. New studies have emerged that shine a new light on possible pathologic mechanisms and present new therapeutic approaches to this disease. We present an inclusive literature review, focusing on these new developments, comparing them with older notions and treatments, and discussing epidemiology data and diagnostic methods.<br>O Glaucoma de Te
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"Breaking the Rules: A Rebuttal of Normal-Tension Glaucoma." TopSCHOLAR, 1999. http://digitalcommons.wku.edu/stu_hon_theses/137.

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Miele, Alba. "Normal Tension Glaucoma Development in two years long-term evolution of in idiophatic Normal Pressure Hydrocephalus patients who received CSF shunting :follow up." Doctoral thesis, 2021. http://hdl.handle.net/2158/1235506.

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Books on the topic "Normal Tension Glaucom"

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Huntbach, Julie, and Amar Alwitry. Glaucoma. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199237593.003.0007.

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The chapter begins by discussing optic nerve head anatomy and aqueous fluid dynamics, before covering the key clinical skills, namely optic nerve head assessment in glaucoma, tonometry and pachymetry , gonioscopy, and perimetry. It also covers the key areas of clinical knowledge, including ocular hypertension, primary open-angle glaucoma, acute angle closure, normal-tension glaucoma, steroid-induced glaucoma, traumatic glaucoma, inflammatory glaucomas, pseudoexfoliative and pigmentary glaucoma, neovascular glaucoma, malignant glaucoma, iridocorneal endothelial syndrome and iridocorneal dysgene
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Tatham, Andrew, and Peng Tee Khaw. Glaucoma. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199672516.003.0008.

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This chapter explores glaucoma. It starts off with an outline of optic nerve head anatomy and then describes aqueous fluid dynamics and the pathogenesis of glaucoma. It then goes on to discuss the clinical skill areas of optic nerve head assessment in glaucoma, glaucoma imaging devices, tonometry and tachymetry, gonioscopy, and perimetry. The chapter also details ocular hypertension, primary open-angle glaucoma, primary angle closure, and secondary angle closure. In addition, it discusses normal tension glaucoma, steroid-induced glaucoma, traumatic glaucoma, inflammatory glaucomas, pseudoexfol
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Book chapters on the topic "Normal Tension Glaucom"

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Sugiyama, Kazuhisa, and Yoshiaki Kitazawa. "Glaucomas: Managing Normal-Tension Glaucoma." In Pearls of Glaucoma Management. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68240-0_43.

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Sugiyama, Kazuhisa, and Yoshiaki Kitazawa. "Glaucomas: Managing Normal-Tension Glaucoma." In Pearls of Glaucoma Management. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-49042-6_43.

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Prum, Bruce E. "Normal-Tension Glaucoma." In Clinical Glaucoma Care. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-4172-4_13.

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Conner, Ian, Kimberly V. Miller, Joel S. Schuman, and David L. Epstein. "Normal-Tension Glaucoma." In Chandler and Grant's Glaucoma, 6th ed. CRC Press, 2024. http://dx.doi.org/10.1201/9781003522980-23.

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Hitchings, R. A. "A Practical Approach to the Management of Normal Tension Glaucoma." In Glaucoma. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18633-2_10.

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Kitazawa, Y., and T. Yamamoto. "Therapeutic Problems of Normal Tension Glaucoma." In Glaucoma: Decision Making in Therapy. Springer Milan, 1996. http://dx.doi.org/10.1007/978-88-470-2196-9_36.

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Khodadadeh, Sarah S., and James C. Tsai. "Surgical Management of Normal Tension Glaucoma." In Essentials in Ophthalmology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-18060-1_10.

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Yu, Jing, Jinghong Sang, and Ningli Wang. "Normal-Tension Glaucoma: A “Qi Deficiency” Disease." In Intraocular and Intracranial Pressure Gradient in Glaucoma. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-2137-5_42.

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Tomita, G., Y. Kitazawa, and N. Hayashi. "Changes in the Optic-Nerve Head over Time in Normal-Tension Glaucoma." In Glaucoma Update VI. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57056-8_24.

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Yamamoto, T., Y. Kitazawa, K. Ishida, and L. Daugeliene. "Role of Trabeculectomy and Calcium-Channel Blockers in the Treatment of Normal-Tension Glaucoma." In Glaucoma Update VI. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57056-8_25.

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Conference papers on the topic "Normal Tension Glaucom"

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Eisner, Alvin, John R. Samples, Heidi M. K. Campbell, and George A. Cioffi. "Foveal Adaptation Abnormalities In Early Normal-Tension Glaucoma." In Vision Science and its Applications. Optica Publishing Group, 1995. http://dx.doi.org/10.1364/vsia.1995.mb3.

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The purpose of this study is to test the hypothesis that visual adaptation can be affected in glaucoma, particularly in normal (or "low") tension glaucoma. More specifically, this study aims to test the hypothesis that by taxing the adaptation capabilities of eyes with normal-tension glaucoma but only minimal field loss, time-dependent abnormalities can be elicited at the fovea.
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Chauhan, Balwantray C., Stephen M. Drance, Gordon R. Douglas, and Chris A. Johnson. "Visual Field Damage in Normal- and High-Tension Glaucoma." In Noninvasive Assessment of the Visual System. Optica Publishing Group, 1990. http://dx.doi.org/10.1364/navs.1990.mb2.

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Optic disc and visual field damage in open angle glaucoma occurs under a wide range of intraocular pressure (IOP). The existence of different types of disc and field damage would suggest that factors other than IOP are responsible for the production of glaucoma.
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Feng, Yali, Zhe Cui, and Wenshuang Xu. "Correlation Between Genetic Factors and the Incidence of Ocular Hypertension Glaucoma / Normal Tension Glaucoma." In International Conference on Electronics, Mechanics, Culture and Medicine. Atlantis Press, 2016. http://dx.doi.org/10.2991/emcm-15.2016.104.

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Girard, Michaël J. A., Lyn Zimmo, Edward T. White, Jean Martial Mari, C. Ross Ethier, and Nicholas G. Strouthidis. "Towards a Biomechanically-Based Diagnosis for Glaucoma: In Vivo Deformation Mapping of the Human Optic Nerve Head." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80557.

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Glaucoma is the most common cause of irreversible blindness. It is associated with a progressive loss of cells within the optic nerve head (ONH) at the back of the eye. Glaucoma remains incurable and its exact causes are not well understood. It was once thought to occur only in eyes with elevated pressure (i.e., intraocular pressure or IOP) and to date, lowering IOP is the only clinical treatment proven to be beneficial for slowing the progression of glaucoma. However, the success rate of such therapy is only 50%. Multiple lines of evidence now indicate that IOP is not the only important risk
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Coudrillier, Baptiste, Kristin M. Myers, and Thao D. Nguyen. "The Biomechanical Response of Normal and Glaucoma Human Sclera." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19354.

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By 2010, 60 million people will have glaucoma, the second leading cause of blindness worldwide [1]. The disease is characterized by a progressive degeneration of the retinal ganglion cells (RGC), a type of neuron that transmits visual information to the brain. It is well know that elevated intraocular pressure (IOP) is a risk factor in the damage to the RGCs [3–5], but the relationship between the mechanical properties of the ocular connective tissue and how it affects cellular function is not well characterized. The cornea and the sclera are collage-rich structures that comprise the outer loa
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Eisner, Alvin, and John R. Samples. "Can flicker deficits depend on spectral opponency?" In OSA Annual Meeting. Optica Publishing Group, 1989. http://dx.doi.org/10.1364/oam.1989.fa2.

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While studying the aging visual system for the intended purpose of understanding age-related macular degeneration, we have employed a battery of tests that has included measures of 20-Hz flicker sensitivity on chromatic backgrounds. Unexpectedly, we found that more than 10% of people older than age 70 in a large control group had profound reductions of flicker sensitivity, more than 10 standard deviations from the mean norm, for at least one of the two background conditions used to measure flicker sensitivity. These people were more likely to be taking cardiovascular medication and/or have gla
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Karunaratne, I. K., C. H. C. Lee, Y. Wei, Y. Zhang, M. Yu, and D. C. C. Lam. "Contact Lens Sensor Human Trial and Conceptual in-vivo Validation of Rate-Based Classification Method for Normal Tension Glaucoma (NTG)." In 2021 Global Medical Engineering Physics Exchanges/ Pan American Health Care Exchanges (GMEPE/PAHCE). IEEE, 2021. http://dx.doi.org/10.1109/gmepe/pahce50215.2021.9434865.

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