Academic literature on the topic 'Normotension'
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Journal articles on the topic "Normotension"
Seyde, W. C., and D. E. Longnecker. "NORMOTENSION VERSUS HYPERTENSION." Anesthesiology 63, Supplement (1985): A13. http://dx.doi.org/10.1097/00000542-198509001-00013.
Full textBrunner, M. J., and M. D. Kligman. "Rapid resetting of baroreflexes in hypertensive dogs." American Journal of Physiology-Heart and Circulatory Physiology 262, no. 5 (1992): H1508—H1514. http://dx.doi.org/10.1152/ajpheart.1992.262.5.h1508.
Full textSanz, Jesús, María Paz García‐Vera, Inés Magán, Regina Espinosa, and María Fortún. "Differences in personality between sustained hypertension, isolated clinic hypertension and normotension." European Journal of Personality 21, no. 2 (2007): 209–24. http://dx.doi.org/10.1002/per.605.
Full textHarper, Alexander A., Julian P. L. Davis, and Alan R. Chipperfield. "Activation of Two Inward Chloride Transport Systems in Rat Femoral Arterial Smooth Muscle in Deoxycorticosterone Acetate/Salt Hypertension." Clinical Science 93, no. 4 (1997): 295–98. http://dx.doi.org/10.1042/cs0930295.
Full textde Leeuw, Peter W., and Abraham A. Kroon. "A sluice to normotension?" Journal of Hypertension 36, no. 12 (2018): 2314–15. http://dx.doi.org/10.1097/hjh.0000000000001926.
Full textJannah, Mifthahul, Delmi Sulastri, and Yuniar Lestari. "Perbedaan Asupan Natrium Dan Kalium Pada Penderita Hipertensi Dan Normotensi Masyarakat Etnik Minangkabau di Kota Padang." Jurnal Kesehatan Andalas 2, no. 3 (2013): 132. http://dx.doi.org/10.25077/jka.v2i3.148.
Full textFunder, John W. "Aldosterone, Normotension, and Diastolic Dysfunction." Journal of Clinical Endocrinology & Metabolism 90, no. 9 (2005): 5500–5501. http://dx.doi.org/10.1210/jc.2005-1613.
Full textLeffler, C. W., and D. W. Busija. "Prostanoids and pial arteriolar diameter in hypotensive newborn pigs." American Journal of Physiology-Heart and Circulatory Physiology 252, no. 4 (1987): H687—H691. http://dx.doi.org/10.1152/ajpheart.1987.252.4.h687.
Full textHatt, Andrew. "Phentolamine continuous infusion in a pediatric patient with uncontrolled hypertension." American Journal of Health-System Pharmacy 78, no. 13 (2021): 1195–99. http://dx.doi.org/10.1093/ajhp/zxab133.
Full textWang, Jian-Bing, Qiu-Chi Huang, Shu-Chang Hu, et al. "Baseline and longitudinal change in blood pressure and mortality in a Chinese cohort." Journal of Epidemiology and Community Health 72, no. 12 (2018): 1083–90. http://dx.doi.org/10.1136/jech-2018-211050.
Full textDissertations / Theses on the topic "Normotension"
Guillard, Frédérique. "Devenir de 40 sujets jugés normotendus a l'issue d'un bilan hospitalier d'hypertension : étude cas-contrôle." Lyon 1, 1993. http://www.theses.fr/1993LYO1201B.
Full textExner, Cécile. "Hyperaldostéronisme primaire par adénome de Conn et normotension : à propos d'un cas." Bordeaux 2, 1995. http://www.theses.fr/1995BOR2M061.
Full textCampos, Débora Ribeiro. "Avaliação do efeito vascular do diterpeno esclareol em ratos normotensos e hipertensos." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/17/17137/tde-27072016-152727/.
Full textINTRODUCTION. High Blood Pressure is a common cardiovascular disease that affects about 50 million people in the US, and a proportionate number to this in Brazil. Many studies have shown that many diterpenoid classes exert significant effect on the cardiovascular system. These studies suggest these metabolites class as a promising source prototype for the development of new agents in the cardiovascular therapy. OBJECTIVE. This study was designed to investigate the mechanisms involved in cardiovascular effect (in vitro and in vivo) of diterpene esclareol in normotensive and hypertensive rats. MATERIAL AND METHODS. Male Wistar rats were used, weighing an average of 250 - 300g from the Central Animal Facility of the Campus of Ribeirão Preto, University of São Paulo (USP). The animals were kept in the animal house of the Department of Surgery and Anatomy in temperature environment (22-25ºC) and light / dark cycle (12:12 hours) controlled. For the induction of hypertension, the model was used two kidney - one clip (left renal artery). The tail noninvasive blood pressure was measured using a cuff, connected to a sensor for systolic arterial blood pressure ((Kent Scientific Corporation, Connecticut, USA). The monitoring of mean arterial blood pressure was measured using MP System 100 A (BioPac System, Inc., Santa Barbara, CA, USA). The in vitro vascular reactivity was evaluated by measuring concentration-response curves for sclareol built from the record of isometric forces obtained in aortic rings with or without endothelium precontracted with Phe 10-7M in organ chambers. The indirect plasma measurements of NO were carried out by determination of serum levels of nitrite and nitrate using the Sievers NOAnalizer 280i (Sievers, Boulder, CO, USA). One or two-way analysis of variance (ANOVA) followed by Bonferroni posttests were performed to detect possible differences between the values in the study. P <0.05 was considered significant. RESULTS. The analysis of the effect of Esclareol on ?PAS showed that there was a significant reduction in ?PAS in both groups. The nitric oxide synthase and soluble guanylate cyclase inhibitors were as efficient as the removal of endothelium, to inhibit relaxation induced by the sclareol. Analysis of the strengths of plasmatic showed no significant difference between groups. CONCLUSION. From the data obtained in this study, it is concluded that the sclareol showed promise as to their vasodilator effects, both in vivo and in-vitro. Nevertheless, the sclareol showed no change of NO, possibly due to the method used
Burgi, Katia. "Tirosina hidroxilase como marcador da atividade simpática em artérias musculares esqueléticas e renais de ratos normotensos: efeitos do treinamento fisico." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-19102007-122123/.
Full textExercise training (T) does not reduce pressure in normotensive, but causes peripheral and central adjustments on cardiovascular system, mediated by autonomic nervous system. In this study we investigated whether T (55% maximal exercise capacity) is able to change vascular sympathetic drive to skeletal muscle (locomotor, non-locomotor), kidney and adrenals, using immunohistochemistry and western blot for tyrosine hydroxylase (TH). T improved performance (+0,07±0,06 Km/h) and reduced resting HR (from 327±7 to 308±10 bpm) without changing MAP (126mmHg) and arterioles wall/ lumen ratio in any tissue. T caused 48% decrease on TH-immunoreactivity of exercised muscles arterioles, without changes on non-exercised tissues. Norepinephrine concentration on femoral arteries was reduced after T. There was no change on adrenal. TH content. Decreased sympathetic drive to skeletal muscles arterioles (without changes on plasma cathecolamines) is a beneficial, tissue-specific effect in normotensive individuals.
Carvalho, Carolina da Silva. "Identificação dos neurotransmissores das fibras mielínicas e amielínicas do nervo depressor aórtico de ratos: uma abordagem imunohistoquímica." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/17/17140/tde-06012017-115951/.
Full textThe aortic depressor nerve (ADN) is primarily a set of afferent fibers that transmit derived information of changes in arterial blood pressure (BP) from arterial baroreceptors (mechanoreceptors located in the aortic arch and carotid sinus) to sites of cardiovascular control located into central nervous system (CNS). This mechanism is responsible for the reflex regulation of cardiac and vascular function, promoting adjustments of vasoconstrictor and vasodilator centers, simultaneously acting on the sympathetic and parasympathetic systems. In addition, contributes to increased cardiac vagal activity and inhibition of sympathetic discharges to vessels and heart, ensuring the maintenance of blood pressure levels within the normal range. Many neurotransmitters have been described operating in cardio-circulatory control centers located in the brainstem, more specifically in the bulb, participating in the regulation of BP. In these central regions, the neurotransmitters glutamate, GABA (Gamma Aminobutyric Acid) and substance P (SP) have been widely investigated. However, none of these works was carried out a detailed study, investigating the presence of SP in aortic depressor nerves directly, and this information is still unknown. It is believed that SP can be a transmitter at the synapse of the baroreceptor reflex, operating in the modulation of this circuit in an attempt to attenuate elevation of blood pressure. Therefore, there is a need to investigate a morphological and immunohistochemical approach in order to promote the clarification on the present neurotransmitters into ADN. The phrenic nerves were used as a positive control, already as substance P (SP) is characterized in this territory. There have been numerous studies describing the existence of SP in phrenic nerves, a fact that justifies the application of the nerve as control of the ADN, study focus of this project. Based on these requirements, the aim of the present study is two-fold. Firstly, it attempts to promote the standardization of the immunohistochemical (IHC) technique as well as the verification of the feasibility of using glutaraldehyde fixative as a primary, assisting in the identification of neurotransmitters in the peripheral nervous system (PNS). Subsequently, the identification and quantification of SP immunoreactivity in the ADN of normotensive rats by indirect immunohistochemical method (3,3\'-Diaminobenzidine \"DAB\") were done. The study was developed in two stages. The first part corresponds to standardization and optimization of immunohistochemical technique in phrenic nerves of Wistar rats through location and characterization of the SP and enzyme choline acetyltransferase (ChAT). The second phase is about the identification and quantification of the SP into ADN, being a possible neurotransmitter or neuromodulator from the baroreceptor reflex. For this study we used a total of 38 Wistar rats (Rattus norvegicus), normotensive, 20 weeks old, male and female. From this total, 16 male animals were used for standardization of IHC technique in the phrenic nerves. Nonetheless, for the characterization and quantification of SP in ADN were used 22 Wistar rats, 12 males and 10 females. Our results showed an unprecedented manner the presence of SP in unmyelinated fibers (type C) and small diameter fibers (A-delta) into ADN, being quite focused on proximal segments and diffuse distally, suggesting the existence of subsets of unmyelinated fibers. These findings confirm numerous assumptions that the SP acts as a neurotransmitter from afferent baroreceptor and may participate in the modulation of the Autonomic Nervous System (ANS), since it is located in centers responsible for regulating reflex of BP. Further, an analysis of the percentage of positive SP staining between genders, presented an apparent predominance of SP in males but no significant difference between the groups were found. Similarly, IHC standardization in transverse and longitudinal sections of phrenic nerves showed a positive random and immunostaining of SP in sets of unmyelinated fibers (type C) and small diameter fibers located near the periphery of endoneural space, corroborating location reported on morphological and ultrastructural studies, ensuring the specificity and reproducibility of the method. Distinctly, the fibers of large and medium diameters (A-alpha, beta and gamma), considered myelinated fibers of fast conducting, were immunoreactive to ChAT in phrenic nerves. Finally, it is expected that the identification of neuropeptide serve as a trigger for that future studies involving the release of neurotransmitters into afferent baroreceptors be explored. These results could contribute to the aggregation of relevant information for the modulation and transmission of neural information, thus providing better understanding of communication and baroreflex activities associated with cardiovascular mechanisms.
Arantes, Ana Carolina. "Efeitos da redução da ingestão de sal sobre a pressão arterial em normotensos, pré-hipertensos e normotensos." Universidade Federal de Goiás, 2017. http://repositorio.bc.ufg.br/tede/handle/tede/7904.
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Introduction: The effects of salt intake reduction on casual blood pressure (BP) among hypertensive patients have been widely studied. However, there are few data about this issue in the context of arterial stiffness, as well as in normotensive and pre-hypertensive subjects. Objective:To evaluate the effects of progressive reduction in intake of the addition salt (from 6 grams / day to 4 grams/day) on peripheral and central pressure values as well as arterial stiffness in normotensive, prehypertensive and hypertensive. Methodology: This is an interventional, single-blind clinical trial, with 13 weeks of follow-up and 4 weeks interval between evaluations, performed with technical-administrative workers and teachers from a public university. Inclusion criteria were age between 20 and 60 years, both genders and which meal at home at least 4 times per week. Exclusion criteria were BP ≥160/100 mmHg, using two or more antihypertensive drugs, secondary hypertension; diabetes, history of myocardial infarction or stroke in the last 6 months, and special need diet. This study was approved by the institution’s Ethics Committee and all the participants signed the informed consent form. Recruitment was performed at the workplace, with a questionnaire about dietary habits, anthropometric and casual BP measurement (OMRON 705CP automatic device). According to the BP levels, participants were classified as normotensive (≤130/85 mmHg), pre-hypertensive (130-139/85-89 mmHg) and hypertensive stage I (≥140- 159/90-99 mmHg). The other evaluated variables were home blood pressure monitoring (HBPM), central blood pressure (CBP) measurement by tonometry (SphygmoCor® system), 24-hour urinarysodium, and dietary salt intake. The participants were instructed to reduce the consumption of foods with high sodium content. The addition of salt used during the follow-up was controlled by delivering individual packages for daily family consumption. The protocol adherence was evaluated by controlling the returned packages at each visit. The data were structured and analyzed using Stata software (version 12). Descriptive analysis was performed using relative and absolute frequencies, as well as median and interquartile range or mean and standard deviation for distribution of categorical and quantitative variables, respectively. For comparison between groups at visit 1, Kruskal-Wallis and Fisher's Exact tests were used. The comparison between the groups was done before and after the intervention using Wilcoxon test and paired Student T test. The correlation between BP values and urinary sodium levels was performed using the Spearman test. For all tests, the significance level was set at 5% and the confidence interval was 95%. Results: Fifty-five participants were evaluated, 32 males (median age 48 years). According to BP values, 18 were normotensive, 15 pre-hypertensive and 22 hypertensive. The salt of addition was reduced at each visit from 6 to 4 g/day. The groups are similar in relation to age and sex. There was no difference between blood pressure measurements and sodium excretion before and after the intervention. The parameters of arterial stiffness also did not suffer. Conclusion: Gradual reduction of addition salt intake in a 13-week follow-up is not able to reduce the loss of danger and mean values of blood pressure.
Introdução: Os efeitos da redução na ingestão de sal sobre a pressão arterial casual de hipertensos já foram amplamente estudados, entretanto essa análise ainda é escassa no contexto da rigidez arterial e em indivíduos normotensos e pré-hipertensos. Objetivo: Avaliar os efeitos da redução progressiva na ingestão do sal de adição (de 6 gramas/dia para 4 gramas/dia) sobre os valores dapressão periférica e central assim como a rigidez arterial em normotensos, pré-hipertensos e hipertensos. Metodologia: Ensaio clínico, simples cego, com 13 semanas de seguimento e intervalo de 4 semanas entre as consultas, realizado com trabalhadores técnico-administrativos e docentes universitários. Critérios de inclusão: idade entre 20 a 60 anos, para ambos os sexos e refeição principal (almoço e/ou jantar) em casa no mínimo 4x/semana. Critérios de exclusão: pressão arterial (PA) ≥160/100 mmHg, uso de dois ou mais medicamentos anti-hipertensivos, hipertensão secundária; diabéticos, história de infarto do miocárdio ou acidente vascular cerebral nos últimos 6 meses e necessidade de alguma dieta especial. Este estudo foi aprovado pelo Comitê de Ética em Pesquisa da instituição e os participantes assinaram o Termo de Consentimento Livre e Esclarecido. O recrutamento foi realizado no local de trabalho, aplicando-se questionários sobre hábitos alimentares, medidas antropométricas e medida casual da PA com o aparelho automático OMRON 705CP. Os participantes foram classificados, de acordo com a medida casual da pressão arterial, em normotensos (≤130/85 mmHg), pré-hipertensos (130-139/85-89 mmHg) e hipertensos estágio I (≥140-159/90-99 mmHg). As variáveis avaliadas foram a medida casual, monitorização residencial da pressão arterial (MRPA), medida central da pressão arterial (PAC) por tonometria com o aparelho sphygmocor, sódio urinário de 24h e mensuração do sal de adição. Os participantes foram orientados a reduzir o consumo de alimentos com alto teor de sódio e o sal de adição utilizado no período do seguimento foi controlado com a entrega em embalagens individuais, para o consumo diário familiar. A adesão ao protocolo foi avaliada pelo controle das embalagens e pesagem do sal de adição retornado em cada visita. Os dados foram estruturados e analisados no programa Stata versão 12. Foram utilizadas frequências absolutas e relativas para análise estatística descritiva e mediana e intervalo inter-quartil ou média e desvio padrão, para as variáveis categóricas e quantitivas. A distribuição dos dados foi analisada pelo teste Shapiro-Wilk; para a comparação entre grupos na V1B foram realizados os testes de Kruskal-Wallis e Exato de Fisher; comparação intra grupo foi feita antes e após a intervenção pelos testes de Wilcoxon e t-Student pareado; correlação entre valores de PA e níveis de sódio urinário via teste de Spearman. Para todos os testes considerou-se o nível de significância em 5% e intervalo de confiança de 95%. Resultados: Foram avaliados 55 participantes com intervalos de 04 semanas, 32 do sexo masculino (mediana de 48 anos) sendo 18 normotensos, 15 pré-hipertensos e 22 hipertensos, redução progressiva no sal de adição a cada visita de 6 até 4 gramas ao dia. Os grupos foram semelhantes em relação a idade e sexo. Não houve diferença entre medidas de pressão arterial e excreção de sódio antes e depois da intervenção. Os parâmetros de rigidez arterial também não sofreram alterações significativas. Conclusão: A redução gradativa da ingestão de sal de adição num seguimento de 13 semanas não foi capaz de reduzir de maneira significativa os valores periféricos e centrais da pressão arterial.
Bloos, Frank. "Coronary circulatory reserve in normotensive hyperdynamic sepsis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0011/NQ40242.pdf.
Full textMatoso, Juliana Magalhães Duarte. "Desempenho cognitivo em idosos hipertensos e normotensos." Universidade do Estado do Rio de Janeiro, 2012. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=6174.
Full textA população de idosos cresce rapidamente no Brasil. A prevalência de hipertensão arterial sistêmica (HAS) e distúrbios cognitivos é elevada nesta população. Testamos a hipótese de que a HAS reduz o desempenho cognitivos em idosos. Foram selecionamos idosos hipertensos e normotensos com idade ≥ 60 < 80. O desempenho cognitivo foi avaliado pelo Cambridge Cognitive Examination Revised (CAMCOG-R), por subtestes do Wechsler Adult Intelligence Scale v.3 (WAIS III), além do Rey Auditory Verbal Learning Test (RAVLT), e o dos Trail Making Tests A/B (TMT-A/B). O desempenho cognitivo avaliado pelo escore global do CAMCOG-R e do QI estimado do WAIS III está reduzido nos idosos hipertensos mesmo quando controlado pela escolaridade, depressão, estado geral de saúde e qualidade de vida. O desempenho cognitivo em diversos domínios específicos controlados para a escolaridade, depressão, estado geral de saúde e qualidade de vida, e avaliados pelo CAMCOG-R, WAIS III, TMT-A e RAVLT também está reduzido nos idosos hipertensos. O presente estudo sugere que a HAS está associada ao declínio do desempenho cognitivo global em idosos. Notadamente, o desempenho das funções executivas está reduzido nos idosos hipertensos. Especula-se que a HAS seja um fator de risco para o declínio progressivo do desempenho cognitivo e, portanto, para o desenvolvimento de demência.
The elderly population is growing rapidly in Brazil. The prevalence of hypertension and cognitive disorders is high in this population. We tested the hypothesis that hypertension is associated with decreased cognitive performance in the elderly. We selected normotensive and hypertensive subjects aged ≥ 60 <80 years old. Cognition was assessed through Cambridge Cognitive Examination Revised (CAMCOG-R), subtests of the Wechsler Adult Intelligence Scale v.3 (WAIS III), the Rey Auditory Verbal Learning Test (RAVLT), and the Trail Making Tests A/B (TMT-A/B). The cognitive performance assessed by the CAMCOG-R global score and the estimated WAIS III. QI is reduced in elderly hypertensives even when controlled for education, depression, health status and quality of life. The cognitive performance in diverse cognitive domains assessed through CAMCOG-R, WAIS III, TMT-A e RAVLT is also reduced in elderly hypertensives when contolled for education, depression, health status and quality of life. These results suggest that hypertension is associated with reduced global cognitive performance in eldely hypertensives. Notably, executive functions are reduced in eldely hypertensives. We speculate that hypertension might be a risk factor for the progressive decline in cognitive function and, therefore, for the development of dementia.
Klein, Maria. "Stressreagibilität bei jungen salzsensitiven und salzresistenten normotensiven Frauen /." Berlin, 2008. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000253703.
Full textMorgan, Jennifer A. "Leucocyte sodium transport in normotensive and hypertensive pregnancy." Thesis, University of Leeds, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236321.
Full textBooks on the topic "Normotension"
Akpogomeh, Benjamin Agbayemie. A Study of alpha-adrenoceptors involved in the neural control of sodium reabsorption by the kidney in normotension, genetic and two models of experimental hypertension. University of Birmingham, 1989.
Davis, Gerard. Reflex neural regulation of renal function in normotensive and hypertensive rats. University of Birmingham, 1991.
Laine, Krista. Intraocular pressure lowering activities of endogenous cannabinoids, and their uptake and enzyme hydrolysis inhibitors in normotensive rabbits. University of Kuopio, 2004.
Yang, Zhuo. The influence of the paraventricular nucleus of the hypothalamus on cardiovascular neurones in the ventrolateral medulla in normotensive and hypertensive rats. University of Birmingham, 1999.
Jacoby, Anne Catherine. CARDIOVASCULAR CIRCADIAN RHYTHMS IN NORMOTENSIVE AND HYPERTENSIVE ADOLESCENTS. 1988.
Banasik, Jacquelyn Lou. Endothelin binding in brain of normotensive and spontaneously hypertensive rats. 1991.
Broege, Phyllis Anne. THE BLOOD PRESSURE RESPONSE TO DAILY STRESS IN NORMOTENSIVE FEMALE NURSES. 1994.
Moore, R. Leigh Hill. Hypotensive effect of isometric exercise on resting blood pressure in high normotensive subjects. 1986.
Kelly, Frecia Marie. THE EFFECTS OF ANGER UPON BLOOD PRESSURE REACTIVITY IN NORMOTENSIVE AND HYPERTENSIVE SUBJECTS. 1986.
Terapia manual del sistema miofascial : la terapia normotensiva aplicada a las disfunciones del sistema musculoesqueletico. Masson, 2002.
Book chapters on the topic "Normotension"
Suzuki, Takahiro, Kyuzo Aoki, and Koichi Miyagawa. "Pressor Effects of Calcium Infusion in the Absence and Presence of a Calcium Antagonist in Subjects with Normotension and Hypertension." In Essential Hypertension 2. Springer Japan, 1989. http://dx.doi.org/10.1007/978-4-431-68090-1_24.
Full textRost, R., H. Heck, and K. Völker. "Ergometrisches Follow-up des Blutdruckverhaltens bei normotensiven Erwachsenen." In Belastungsblutdruck bei Hochdruckkranken. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77975-6_11.
Full textNaranjo, C. A., K. E. Bremner, P. Sanhueza, and E. M. Sellers. "Enalapril Does not Decrease Alcohol Intake in Normotensive Alcoholics." In Novel Pharmacological Interventions for Alcoholism. Springer New York, 1992. http://dx.doi.org/10.1007/978-1-4612-2878-3_17.
Full textPittman, Quentin J. "Brain Vasopressin and Cardiovascular Regulation in Normotensive and Hypertensive Animals." In Neuropeptides and Stress. Springer New York, 1989. http://dx.doi.org/10.1007/978-1-4612-3514-9_11.
Full textPrager, G., R. Prager, and P. Klein. "Age-Dependent Variation of Diurnal Blood Pressure Profile in Normotensive Subjects." In Temporal Variations of the Cardiovascular System. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-662-02748-6_25.
Full textMcCarron, R. M., Y. Yoshihide, M. Spatz, and J. Hallenbeck. "Hypertensive Versus Normotensive Monocyte Adhesion to Cultured Cerebral Microvascular Endothelial Cells." In Biology and Physiology of the Blood-Brain Barrier. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_48.
Full textEarle, M. L., T. Horn, N. Kasting, R. Landgraf, and Q. J. Pittman. "Ventral Septal AVP Release in the Febrile Hypertensive and Normotensive Rat." In Thermal Balance in Health and Disease. Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-7429-8_54.
Full textRossi, Gian Paolo, Teresa Maria Seccia, and Paola Caielli. "Primary Aldosteronism: Rare or Common Condition in Hypertensive Patients and Normotensive Individuals?" In Special Issues in Hypertension. Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-2601-8_4.
Full textPodos, S. M., C. B. Camras, J. B. Serle, and P. Y. Lee. "Pharmacologic Alteration of Aqueous Humor Dynamics in Normotensive and Glaucomatous Monkey Eyes." In Glaucoma Update III. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71785-7_33.
Full textWang, Duo H., and Russell L. Prewitt. "Reduced Aortic and Arteriolar Growth by Captopril in Normotensive and Renal Hypertensive Rats." In Advances in Experimental Medicine and Biology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-6015-5_19.
Full textConference papers on the topic "Normotension"
Lande, K., I. O. S. S. E. Kieldsen, A. Westheim, et al. "PATIENTS WITH MILD ESSENTIAL HYPERTENSION HAVE INCREASED PLATELET SIZE AND RELEASE REACTION ANO SHOW INCREASED RECEPTOR RESPONSE TO INFUSED ADRENALINE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644261.
Full textSolverson, Kevin, Christopher Humphreys, Zhiying Liang, et al. "Refining risk in normotensive acute pulmonary embolism." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3646.
Full textBoekhoven, R. W., L. Marais, M. C. M. Rutten, F. N. vd Vosse, P. Boutouyrie, and R. G. P. Lopata. "Non-invasive strain imaging in normotensive and hypertensive patients." In 2014 IEEE International Ultrasonics Symposium (IUS). IEEE, 2014. http://dx.doi.org/10.1109/ultsym.2014.0448.
Full textTsamis, Alkiviadis, Nikos Stergiopulos, and Alexander Rachev. "A Structure-Based Model of Arterial Remodeling in Response to Sustained Hypertension." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206338.
Full textBodzenta-Lukaszvk, A., K. Krupiński, and M. Bielawiec. "PLATELET FUNCTION AND KALLIKREIN SYSTEM IN PATIENTS WITH ESSENTIAL HYPERTENSION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644258.
Full textXi, Shou-min, San-qiang Li, and Wei-dong Yin. "Topical Application of Melatonin Analogue Neu-P11 Reduces Intraocular Pressure in Normotensive Rabbit." In 2012 International Conference on Biomedical Engineering and Biotechnology (iCBEB). IEEE, 2012. http://dx.doi.org/10.1109/icbeb.2012.451.
Full textAli, S., L. Alnimer, H. A. Rauf, and J. R. Heffernan. "PRES in a Normotensive Patient with Sickle Cell Disease: Low Pressure, Alarming Consequences." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7211.
Full textSilveri, Giulia, Lorenzo Pascazio, and Agostino Accardo. "Relationship Between Blood Pressure and Heart Rate Circadian Rhythms in Normotensive and Hypertensive Subjects." In 2018 Computing in Cardiology Conference. Computing in Cardiology, 2018. http://dx.doi.org/10.22489/cinc.2018.273.
Full textAl-Jahawsheh, Shibley, Basheer Na'amneh, Duha Al-Batayha, and Mohammad Al-Abed. "Analysis of Corrected QT Interval Dispersion (QTcd) in Normotensive Subjects Performing Breath-hold Challenge." In 2020 IEEE 5th Middle East and Africa Conference on Biomedical Engineering (MECBME). IEEE, 2020. http://dx.doi.org/10.1109/mecbme47393.2020.9265167.
Full textKouchaki, Zahra, Mark Butlin, and Alberto P. Avolio. "Hemodynamics changes with acute carotid baroreceptor field stimulation are age-dependent in normotensive rats*." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7318790.
Full textReports on the topic "Normotension"
Kramer, George C. Closed-Loop Resuscitation of Hemorrhagic Shock: Novel Solutions Infused to Hypotensive and Normotensive Endpoints. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada470719.
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