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Dissertations / Theses on the topic 'NPM1 mutation'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 February 2022

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1

Seibl, Marlene. "Quantifizierung von minimaler Resterkrankung bei akuter myeloischer Leukämie mit NPM1 Mutation mittels Real-Time-PCR." Diss., lmu, 2011. http://nbn-resolving.de/urn:nbn:de:bvb:19-126943.

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2

Gille, Christine [Verfasser], and Karsten [Akademischer Betreuer] Spiekermann. "MRD-Monitoring bei AML-Patienten mittels Hochdurchsatz-Amplikonsequenzierung (NPM1 Mutation A) / Christine Gille ; Betreuer: Karsten Spiekermann." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2021. http://d-nb.info/1229835466/34.

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3

Jensen, Kai-Ole [Verfasser]. "Monitoring minimaler Resterkrankung (MRD) bei der akuten myeloischen Leukämie (AML) mit NPM1 Mutation / Kai-Ole Jensen." Ulm : Universität Ulm, 2016. http://d-nb.info/1110969465/34.

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4

Gourvest, Morgane. "Etude des longs ARNs non codants dans les leucémies aiguës myéloïdes : relevance clinique et caractérisation fonctionnelle." Thesis, Toulouse 3, 2020. http://www.theses.fr/2020TOU30117.

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Les longs ARNs non codants (lncRNAs) sont définis comme des transcrits ayant une taille supérieure à 200 nucléotides et dépourvus de potentiel codant. Longtemps considérés comme inutiles, leur étude récente a démontré qu’ils jouent un rôle important dans l’expression de nos gènes. On compte d’ailleurs de plus en plus d’exemples de ces lncRNAs dérégulés dans les cancers. Notre étude visait à évaluer l’existence de profils d’expression particuliers de lncRNAs au sein des leucémies aiguës myéloïdes à caryotype normal (LAM-CN), dont l’implication dans cette pathologie n’est que peu décrite. Le séq
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5

Lüker, Jakob [Verfasser]. "Nachweis von NPM1- und FLT3-Mutationen bei Patienten mit AML / Jakob Lüker." Lübeck : Zentrale Hochschulbibliothek Lübeck, 2011. http://d-nb.info/1009752928/34.

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6

Leung, Siu-yung, and 梁小容. "NPM1 and FLT3-ITD mutations in cytogenetically normal acute myeloid leukaemia patients of Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44671143.

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7

Graul, Katja [Verfasser]. "Untersuchungen von NPM1-Mutationen bei Patienten mit akuter myeloischer Leukämie bei Diagnosestellung und im Verlauf / Katja Graul." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2014. http://d-nb.info/1057869686/34.

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8

Aydin, Isabella [Verfasser]. "Inzidenz und prognostische Bedeutung von NPM1-Mutationen bei der akuten myeloischen Leukämie des Erwachsenen: Analysen im Rahmen der AML HD98-A Studie / Isabella Aydin." Ulm : Universität Ulm. Medizinische Fakultät, 2014. http://d-nb.info/1052585922/34.

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9

Krebs, Bernhard Peter [Verfasser]. "Inzidenz und prognostische Bedeutung von Mutationen des Nucleophosmin (NPM1) Gens bei der akuten myeloischen Leukämie (AML) des älteren Patienten (> 60 Jahre) / Bernhard Peter Krebs." Ulm : Universität Ulm. Medizinische Fakultät, 2012. http://d-nb.info/1023728656/34.

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10

Dörr, Alexandra [Verfasser]. "Molekulargenetische Analysen zur Inzidenz von NPM1- und FLT3-Mutationen bei der akuten myeloischen Leukämie des älteren Patienten und Evaluation ihrer Bedeutung als prognostische Marker / Alexandra Dörr." Ulm : Universität Ulm, 2018. http://d-nb.info/1161008578/34.

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11

Garz, Anne-Kathrin [Verfasser], Katharina S. [Akademischer Betreuer] Götze, Katharina S. [Gutachter] Götze, and Bernhard [Gutachter] Küster. "Azacitidine combined with crenolanib abrogates niche protection and expansion of residual leukemic stem cells (LSC) in FLT3-ITD+ acute myeloid leukemia (AML) with concurrent gene mutations in NPM1, DNMT3A or TET2 / Anne-Kathrin Garz ; Gutachter: Katharina S. Götze, Bernhard Küster ; Betreuer: Katharina S. Götze." München : Universitätsbibliothek der TU München, 2017. http://d-nb.info/1148650075/34.

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12

Seibl, Marlene [Verfasser]. "Quantifizierung von minimaler Resterkrankung bei akuter myeloischer Leukämie mit NPM1-Mutation mittels Real-Time-PCR / vorgelegt von Marlene Seibl." 2011. http://d-nb.info/1010976001/34.

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13

Lin, Tsung-Chin, and 林宗縉. "A novel fluorescence-based multiplex PCR assay for rapid simultaneous detection of CEBPA mutations, NPM mutations, and FLT3/ITD in patients with acute myeloid leukemia." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/57350234693352410408.

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碩士<br>國立臺灣大學<br>醫學檢驗暨生物技術學研究所<br>94<br>Acute myeloid leukemias (AML) are clonal disorders that are characterized by acquired somatic mutations in hematopoietic progenitors. Mutations of CCAAT/enhancer binding protein α (CEBPA), nucleophosmin (NPM), and Fms-like tyrosine kinase-3 (FLT3) genes have been reported as the most frequent genetic variations in AML patients, and they have the important value in predicting prognosis, especially in those with normal karyotype. Due to the reappearances of the same CEBPA mutation and NPM mutation at relapse, these mutations are suitable as the biomarkers f
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14

Naidoo, Horacia. "The study of the impact of selected mutations in FMS-like Tyrosine Kinase III (FLT3) and Nucleophosmin (NPM1) - and HIV status on patients with acute Myeloid Leukemia and their response to induction therapy." Thesis, 2012. http://hdl.handle.net/10413/9430.

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Acute Myeloid Leukemia (AML), the most common form of acute leukemia in adults, is only curable in approximately 30% of all cases. Despite prognostic risk stratification using sub-typing and cytogenetic analysis to direct therapy, the mortality and relapse rate remains high. AML patients with normal karyotypes are defined as intermediate risk and are the most challenging to treat. Somatic mutations may be the key in refining prognostic stratification and providing useful therapeutic targets. The FMS-like tyrosine kinase 3 (FLT3) and Nucleophosmin (NPM1) genes have common mutated forms that are
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15

Marshall, Robyn Cara. "Assessment for the presence of fms-like tyrosine kinase 3 (FLT3) and nucleophosmin protein-1 (NPM1) mutations and their association with immunophenotypic markers in de novo Acute Myeloid Leukaemia (AML) diagnosed at a single academic centre." Thesis, 2013. http://hdl.handle.net/10539/12486.

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Introduction: Acute Myeloid Leukaemia (AML) is known to be a heterogeneous clonal disorder of haemopoietic progenitor cells. Recently, the molecular pathogenesis of AML has become more apparent, with the description of two commonly found mutations, namely NPM1 (Nucleophosmin protein-1) gene mutation and the FLT3-ITD (FMS-like tyrosine kinase 3 internal tandem duplication) mutation. These mutations are now routinely assessed and used in both prognostication and treatment decisions in the first world. The primary aim of this project was to assess for the presence of NPM1 and FLT3- ITD mu
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