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Relevant bibliographies by topics / Number of glial cells

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Academic literature on the topic 'Number of glial cells'

Author: Grafiati

Published: 4 June 2021

Last updated: 12 February 2022

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Journal articles on the topic "Number of glial cells"

1

Badenhorst, Paul. "Tramtrack controls glial number and identity in the Drosophila embryonic CNS." Development 128, no. 20 (2001): 4093–101. http://dx.doi.org/10.1242/dev.128.20.4093.

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Neurons and glia are often derived from common multipotent stem cells. In Drosophila, neural identity appears to be the default fate of these precursors. Stem cells that generate either neurons or glia transiently express neural stem cell-specific markers. Further development as glia requires the activation of glial-specific regulators. However, this must be accompanied by simultaneous repression of the alternate neural fate. I show that the Drosophila transcriptional repressor Tramtrack is a key repressor of neuronal fates. It is expressed at high levels in all mature glia of the embryonic ce

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Corty, Megan M., and Marc R. Freeman. "Architects in neural circuit design: Glia control neuron numbers and connectivity." Journal of Cell Biology 203, no. 3 (2013): 395–405. http://dx.doi.org/10.1083/jcb.201306099.

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Glia serve many important functions in the mature nervous system. In addition, these diverse cells have emerged as essential participants in nearly all aspects of neural development. Improved techniques to study neurons in the absence of glia, and to visualize and manipulate glia in vivo, have greatly expanded our knowledge of glial biology and neuron–glia interactions during development. Exciting studies in the last decade have begun to identify the cellular and molecular mechanisms by which glia exert control over neuronal circuit formation. Recent findings illustrate the importance of glial

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Hidalgo, A. "Neuron–glia interactions during axon guidance in Drosophila." Biochemical Society Transactions 31, no. 1 (2003): 50–55. http://dx.doi.org/10.1042/bst0310050.

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Axons navigate to trace stereotypic trajectories over an environment often rich in glial cells. Once axonal trajectories are defined, their structuring proceeds through multiple fasciculation and defasciculation events, to finally establish the mature bundles. Fasciculation and ensheathment also proceed in close association between axons and glial cells, and ultimately require glia. The cross-talk between axons and glia during axon guidance is manifested in: (i) axonal fasciculation and bundling, promoted by glia; (ii) growth cone guidance, as glia function as guidepost cells at choice points;

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Halter, D. A., J. Urban, C. Rickert, et al. "The homeobox gene repo is required for the differentiation and maintenance of glia function in the embryonic nervous system of Drosophila melanogaster." Development 121, no. 2 (1995): 317–32. http://dx.doi.org/10.1242/dev.121.2.317.

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We describe the cloning, expression and phenotypic characterisation of repo, a gene from Drosophila melanogaster that is essential for the differentiation and maintenance of glia function. It is not, however, required for the initial determination of glial cells. In the embryo, the gene, which encodes a homeodomain protein, is expressed exclusively in all developing glia and closely related cells in both the central and peripheral nervous systems. The only observed exceptions in the CNS are the midline glia derived from the mesectoderm and two of three segmental nerve root glial cells. Using a

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Zhang, Albert, Kentaro Noma, and Dong Yan. "Regulation of Gliogenesis by lin-32/Atoh1 in Caenorhabditis elegans." G3 Genes|Genomes|Genetics 10, no. 9 (2020): 3271–78. http://dx.doi.org/10.1534/g3.120.401547.

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Abstract The regulation of gliogenesis is a fundamental process for nervous system development, as the appropriate glial number and identity is required for a functional nervous system. To investigate the molecular mechanisms involved in gliogenesis, we used C. elegans as a model and identified the function of the proneural gene lin-32/Atoh1 in gliogenesis. We found that lin-32 functions during embryonic development to negatively regulate the number of AMsh glia. The ectopic AMsh cells at least partially arise from cells originally fated to become CEPsh glia, suggesting that lin-32 is involved

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Booth, G. E., E. F. Kinrade, and A. Hidalgo. "Glia maintain follower neuron survival during Drosophila CNS development." Development 127, no. 2 (2000): 237–44. http://dx.doi.org/10.1242/dev.127.2.237.

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While survival of CNS neurons appears to depend on multiple neuronal and non-neuronal factors, it remains largely unknown how neuronal survival is controlled during development. Here we show that glia regulate neuronal survival during formation of the Drosophila embryonic CNS. When glial function is impaired either by mutation of the glial cells missing gene, which transforms glia toward a neuronal fate, or by targeted genetic glial ablation, neuronal death is induced non-autonomously. Pioneer neurons, which establish the first longitudinal axon fascicles, are insensitive to glial depletion wh

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Krawczyk, Aleksandra Ewa, and Jadwiga Jaworska-Adamu. "The immunoreactivity of satellite glia of the spinal ganglia of rats treated with monosodium glutamate." Acta Veterinaria Brno 85, no. 4 (2016): 337–41. http://dx.doi.org/10.2754/avb201685040337.

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Satellite glia of the peripheral nervous system ganglia provide metabolic protection to the neurons. The aim of this study was to determine the effects of monosodium glutamate administered parenterally to rats on the expression of glial fibrillary acidic protein, S-100β protein and Ki-67 antigen in the satellite glial cells. Adult, 60-day-old male rats received monosodium glutamate at two doses of 2 g/kg b.w. (group 1) and 4 g/kg b.w. (group 2) subcutaneously for 3 consecutive days. Animals in the control group (group C) were treated with corresponding doses of 0.9% sodium chloride. Immediatel

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Annunziato, Lucio, Francesca Boscia, and Giuseppe Pignataro. "Ionic Transporter Activity in Astrocytes, Microglia, and Oligodendrocytes During Brain Ischemia." Journal of Cerebral Blood Flow & Metabolism 33, no. 7 (2013): 969–82. http://dx.doi.org/10.1038/jcbfm.2013.44.

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Glial cells constitute a large percentage of cells in the nervous system. During recent years, a large number of studies have critically attributed to glia a new role which no longer reflects the long-held view that glia constitute solely a silent and passive supportive scaffolding for brain cells. Indeed, it has been hypothesized that glia, partnering neurons, have a much more actively participating role in brain function. Alteration of intraglial ionic homeostasis in response to ischemic injury has a crucial role in inducing and maintaining glial responses in the ischemic brain. Therefore, g

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Gray, G. E., and J. R. Sanes. "Lineage of radial glia in the chicken optic tectum." Development 114, no. 1 (1992): 271–83. http://dx.doi.org/10.1242/dev.114.1.271.

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In many parts of the central nervous system, the elongated processes of radial glial cells are believed to guide immature neurons from the ventricular zone to their sites of differentiation. To study the clonal relationships of radial glia to other neural cell types, we used a recombinant retrovirus to label precursor cells in the chick optic tectum with a heritable marker, the E. coli lacZ gene. The progeny of the infected cells were detected at later stages of development with a histochemical stain for the lacZ gene product. Radial glia were identified in a substantial fraction of clones, an

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Parker, Robert J., and Vanessa J. Auld. "Signaling in glial development: differentiation migration and axon guidance." Biochemistry and Cell Biology 82, no. 6 (2004): 694–707. http://dx.doi.org/10.1139/o04-119.

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Glial cells have diverse functions that are necessary for the proper development and function of complex nervous systems. During development, a variety of reciprocal signaling interactions between glia and neurons dictate all parts of nervous system development. Glia may provide attractive, repulsive, or contact-mediated cues to steer neuronal growth cones and ensure that neurons find their appropriate synaptic targets. In fact, both neurons and glia may act as migrational substrates for one another at different times during development. Also, the exchange of trophic signals between glia and n

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Dissertations / Theses on the topic "Number of glial cells"

1

Förster, Bettina Ulrike. "Talin in glial cells." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612772.

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Schuliga, Michael, and michael schuliga@deakin edu au. "Steroidogenesis in cultured mammalian glial cells." Deakin University. School of Biological and Chemical Sciences, 1998. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20061207.154152.

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A protocol for culturing mammalian type 1 astrocytic cells, using female post-natal rat cerebral cortical tissue, was established and refined for use in steroidogenic metabolic studies incorporating progestin radioisotopes. Cultures were characterised for homogeneity using standard morphological and immunostaining techniques. Qualitative and quantitative studies were conducted to characterise the progesterone (P) metabolic pathways present in astrocytes in vitro. Of particular interest was the formation of the P metabolite, 5á-pregnan-3á-ol-20-one (THP). THP is a GABA(A) receptor agonist,

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Mellor, Robert. "Neurochemical studies on cultured glial cells." Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.300038.

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Nutt, Catherine L. "Mechanisms of drug resistance in glial cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq28512.pdf.

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Nieweg, Katja Pfrieger Frank. "Cholesterol biosynthesis in neurons and glial cells." Strasbourg : Université de Strasbourg, 2009. http://eprints-scd-ulp.u-strasbg.fr:8080/1048/01/NIEWEG_Katja_2007.pdf.

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Nieweg, Katja. "Cholesterol biosynthesis in neurons and glial cells." Université Louis Pasteur (Strasbourg) (1971-2008), 2007. https://publication-theses.unistra.fr/public/theses_doctorat/2007/NIEWEG_Katja_2007.pdf.

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Cette étude confirme l’hypothèse d’une dépendance des neurones en cholesterol astrocytaire au stade postnatal. Nos travaux montrent que les neurones ne peuvent assurer leurs besoins en cholestérol: L’accumulation de lanostérol et la lente conversion de stérols intermédiaires indiquent que les neurones produisent du cholesterol de façon moins efficace que les cellules gliales. La diminution le taux de cholestérol dans les neurones n’induit pas d’augmentation de l’expression des enzymes de sa voie de biosynthèse. L’absence de synthèse d’ester de cholesterol et d’organelles de stockage de cholest

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Gao, Yuanqing. "Hypothalamic Glial Cells in Diet Induced Obesity." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1447071648.

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Burne, Julia Fiona. "The role of retinal ganglion cell axions in the regulation of glial cell numbers in the rodent optic nerve." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.264697.

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Cotterill, Claire Louise. "Semliki Forest virus-induced apoptosis in glial cells." Thesis, University of Edinburgh, 2005. http://hdl.handle.net/1842/29712.

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Multiple sclerosis (MS) is the most common neurological disease affecting young adults. The incidence rates in Scotland are among the highest in the world. Virus infection may have a precipitating role, or exacerbate symptoms. That viruses can produce inflammatory central nervous system (CNS) demyelinating disease has been well established from study of several natural and experimental infections; one model system is Semliki Forest virus (SFV) infection of mice. SFV infection of BALB/c mice infected at less than 12 days of age (P12) causes fulminant encephalitis, characterised by apoptotic dea

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Göritz, Christian. "Influence of glial cells on postnatal differentiation of rat retinal ganglion cells." [S.l. : s.n.], 2005. http://www.diss.fu-berlin.de/2005/65/index.html.

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Books on the topic "Number of glial cells"

1

Castellano, Bernardo, Berta González, and Manuel Nieto-Sampedro, eds. Understanding Glial Cells. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5737-1.

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Jeserich, Gunnar, Hans H. Althaus, Christiane Richter-Landsberg, and Rolf Heumann, eds. Molecular Signaling and Regulation in Glial Cells. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60669-4.

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3

Bignami, A. Glial cells in the central nervous system. Published by Elsevier for the Foundation for the Study of the Nervous System, 1992.

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von Bernhardi, Rommy, ed. Glial Cells in Health and Disease of the CNS. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40764-7.

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Matsas, Rebecca, and Marco Tsacopoulos, eds. The Functional Roles of Glial Cells in Health and Disease. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4685-6.

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Köritzer, Julia. Biophysical Effects of Cold Atmospheric Plasma on Glial Tumor Cells. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-06224-2.

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Alvarez-Leefmans, Francisco J., and John M. Russell, eds. Chloride Channels and Carriers in Nerve, Muscle, and Glial Cells. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-9685-8.

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8

Doris, Dahl, ed. Glial cells in the central nervous system and their reaction to injury. R.G. Landes, 1994.

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9

Stewart, Kirsten Nicola. Retrovirally encoded alpha-L-iduronidase produced by NIH 3T3 fibroblasts is taken up intoneuronal and glial cells in vitro. University of Manchester, 1996.

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10

Weinstein, Marc A. Inhibition of malignant glial cell growth by estramustine, an estrogen based antimicrotubule agent, and synthesized analogs of estrone, and the basis for the use of estramustine in combination chemotherapy. s.n.], 1993.

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Book chapters on the topic "Number of glial cells"

1

Dobson, Katharine L., and Tomas C. Bellamy. "Glial Cells." In Essentials of Cerebellum and Cerebellar Disorders. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-24551-5_27.

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Pais, Teresa Faria. "Glial Cells." In Encyclopedia of Inflammatory Diseases. Springer Basel, 2015. http://dx.doi.org/10.1007/978-3-0348-0620-6_111-1.

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Kettenmann, Helmut, and Alex Verkhratsky. "Glial Cells." In Neuroscience in the 21st Century. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1997-6_19.

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Pais, Teresa Faria. "Glial Cells." In Compendium of Inflammatory Diseases. Springer Basel, 2016. http://dx.doi.org/10.1007/978-3-7643-8550-7_111.

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Williams, Matthew, Claire Macdonald, and Mario Cordero. "Glial Cells." In The Neuropathology of Schizophrenia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68308-5_12.

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Castejón, Orlando J. "Cerebellar Glial Cells." In Scanning Electron Microscopy of Cerebellar Cortex. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0159-6_12.

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Kettenmann, Helmut, and Alex Verkhratsky. "Glial Cells: Neuroglia." In Neuroscience in the 21st Century. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3474-4_19.

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Lara, Juan M., Almudena Velasco, Concepción Lillo, David Jimeno, and José Aijón. "Characterization of the Glial Cells in the Teleost Visual Pathway." In Understanding Glial Cells. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5737-1_1.

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del Carmen Fernández-Galaz, María, Julie Ann Chowen, and Luis Miguel García-Segura. "Role of Astroglia in the Neural Effects of Sex Hormones and Neuroactive Steroids." In Understanding Glial Cells. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5737-1_10.

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Molina-Holgado, Francisco, Eduardo Molina-Holgado, Alberto Lledó, and Carmen Guaza. "Cytokines in Astroglial Cells: Functions and Mechanisms of Action." In Understanding Glial Cells. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5737-1_11.

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Conference papers on the topic "Number of glial cells"

1

Suzuki, Kei, Toshihiko Shiraishi, Shin Morishita, and Hiroshi Kanno. "Effects of Mechanical Vibration on Proliferation and Differentiation of Neural Stem Cells." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-66831.

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Neural stem cells have been studied to promote neurogenesis in regenerative therapy. The control of differentiation of neural stem cells to nerve cells and the increase of the number of nerve cells are needed. For the purpose of them, it is important to investigate not only chemical factors but also mechanical factors such as hydrostatic pressure in brain and mechanical vibration in walking. In this study, sinusoidal inertia force was applied to cultured neural stem cells and the effects of mechanical vibration on the cells were investigated. After the cells were cultured in culture plates for

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2

Pasupathy, Parameshwaran, Robert De Simone, and Assimina A. Pelegri. "Numerical Simulation of Stress States in White Matter via a Continuum Model of 3D Axons Tethered to Glia." In ASME 2020 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/imece2020-24667.

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Abstract A new finite element approach is proposed to study the propagation of stress in axons in the central nervous system (CNS) white matter. The axons are embedded in an extra cellular matrix (ECM) and are subjected to tensile loads under purely non-affine kinematic boundary conditions. The axons and the ECM are described by the Ogden hyperelastic material model. The effect of tethering of the axons by oligodendrocytes is investigated using the finite element model. Glial cells are often thought of as the “glue” that hold the axons together. More specifically, oligodendrocytes bond multipl

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Shiraishi, Toshihiko, Kei Suzuki, Shin Morishita, and Hiroshi Kanno. "Control of Apoptosis and Differentiation of Cultured Neural Stem Cells by Mechanical Vibration." In ASME 2009 International Mechanical Engineering Congress and Exposition. ASMEDC, 2009. http://dx.doi.org/10.1115/imece2009-11154.

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In this study, sinusoidal inertia force was applied to cultured neural stem cells and the effects of mechanical vibration on the cells were investigated. Neural stem cells which were obtained from the hippocampus of an adult Fischer rat were seeded in culture plates at the density of 2.5 × 105 cells/ml. After cells were cultured for one day and adhered on the cultured plate, vibration groups of the culture plates were set on the aluminum plate of the experimental setup and cultured under sinusoidal excitation in another CO2 incubator separated from non-vibration groups of the culture plates. A

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Kremer, AE, L. Gebhardt, J. Robering, H. Kühn, K. Wolf, and MMJ Fischer. "Lysophosphatidic acid activates peripheral glial cells." In 35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0038-1677169.

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Toy, Muhammed Fatih, Burcu Kurt Vatandaslar, and Bilal Ersen Kerman. "Refractive index tomography of myelinating glial cells." In Quantitative Phase Imaging V, edited by Gabriel Popescu and YongKeun Park. SPIE, 2019. http://dx.doi.org/10.1117/12.2512706.

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Yamanaka, Koji. "Active roles of glial cells in neurodegenrative disease." In 2010 International Conference on Systems in Medicine and Biology (ICSMB). IEEE, 2010. http://dx.doi.org/10.1109/icsmb.2010.5735340.

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Salakhutdinov, Ildar F., Pamela VandeVord, Olena Palyvoda, Howard T. W. Matthew, Golam Newaz, and Gregory W. Auner. "Polymer gratings for protein and glial cells adsorption." In Laser Science. OSA, 2008. http://dx.doi.org/10.1364/ls.2008.lthd5.

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Stingl, Andreas, Patricia M. A. Farias, Raquel Milani, Arnaldo Andrade, and Andre Galembeck. "Long term imaging of living brain glial cancer cells." In Neural Imaging and Sensing 2018, edited by Qingming Luo and Jun Ding. SPIE, 2018. http://dx.doi.org/10.1117/12.2290330.

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Bacola, Gregory, Simon Vales, Alice Prigent, et al. "Abstract 119: Enteric glial cells promote chemoresistance in ATM-expressing cancer stem cells." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-119.

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Kolosov, M. S., E. Duz, and A. B. Uzdensky. "Photodynamic damage of glial cells in crayfish ventral nerve cord." In Sartov Fall Meeting 2010, edited by Valery V. Tuchin and Elina A. Genina. SPIE, 2010. http://dx.doi.org/10.1117/12.889355.

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Reports on the topic "Number of glial cells"

1

Rothstein, Jeffrey D., and Betty Diamond. The Role of NG2 Glial Cells in ALS Pathogenesis. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada598910.

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Rothstein, Jeffrey D. The Role of NG2 Glial Cells in ALS Pathogenesis. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada618869.

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Zong, Hui, and Betty Diamond. Social Behavior in Medulloblastoma: Functional Analysis of Tumor-Supporting Glial Cells. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613317.

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Zong, Hui. Social Behavior in Medulloblastoma: Functional Analysis of Tumor-Supporting Glial Cells. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada566929.

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Stern, Michael. Signaling Pathways Controlling the Growth and Proliferation of Drosophilae Perineural Glial Cells. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada437242.

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Stern, Michael. Signaling Pathways Controlling the Growth and Proliferation of Drosophila Perineurial Glial Cells. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada428460.

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Sterm, Michael. Signaling Pathways Controlling the Growth and Proliferation of Drosophila Perineurial Glial Cells. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada458973.

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Stern, Michael. Signaling Pathways Controlling the Growth and Proliferation of Drosophilae Perineurial Glial Cells. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada416605.

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Harris, B., M. Marden, and C. J. Park. The Distribution of the Number of Empty Cells in a Generalized Random Allocation Scheme. Defense Technical Information Center, 1985. http://dx.doi.org/10.21236/ada154806.

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Trichopoulos, Dimitrios. Early Life Processes, Endocrine Mediators and Number of Susceptible Cells in Relation to Breast Cancer Risk. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada485736.

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