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1

Tomkins, Andrew. "Nutritional Deficiencies during Famine." Tropical Doctor 21, no. 1_suppl (January 1991): 43–46. http://dx.doi.org/10.1177/00494755910210s110.

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2

Hathcock, John N. "Drug-Induced Nutritional Deficiencies." Journal of Nutrition 116, no. 11 (November 1, 1986): 2311. http://dx.doi.org/10.1093/jn/116.11.2311.

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3

De, Dipankar, and Divya Seshadri. "Nails in nutritional deficiencies." Indian Journal of Dermatology, Venereology, and Leprology 78, no. 3 (2012): 237. http://dx.doi.org/10.4103/0378-6323.95437.

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4

Coelho, V. A. T., M. A. Figueiredo, C. L. Rodas, A. R. B. Bastos, L. C. Coelho, and J. G. Carvalho. "INERAL NUTRITION OF ORNAMENTAL PEPPER IN NUTRITIONAL DEFICIENCIES." Nucleus 11, no. 1 (April 30, 2014): 169–77. http://dx.doi.org/10.3738/1982.2278.875.

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5

Kaur, Damanpreet, Prasad Rasane, Jyoti Singh, Sawinder Kaur, Vikas Kumar, Dipendra Kumar Mahato, Anirban Dey, Kajal Dhawan, and Sudhir Kumar. "Nutritional Interventions for Elderly and Considerations for the Development of Geriatric Foods." Current Aging Science 12, no. 1 (September 25, 2019): 15–27. http://dx.doi.org/10.2174/1874609812666190521110548.

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The process of aging is characterized by numerous changes in the body which has an overall negative effect on the health and lifestyle of elderly. Nutrition deserves special attention as an individual reaches old age. It plays a vital role in affecting the quality of life, including physical, mental and social health. The physiological decline in food intake is very common among older age and this result in nutritional deficiencies. These increased nutritional deficiencies are the major risk factors for certain chronic diseases and deteriorated age related health. Thus, the adoption of nutritional intervention can be a measure to tackle the current situation of nutritional deficiencies and promote a healthy lifestyle.
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6

Menon, Aathira, Apthadevi, and P. R. Roshni. "Nutritional Deficiencies after Bariatric Surgery." Research Journal of Pharmacology and Pharmacodynamics 11, no. 3 (2019): 120. http://dx.doi.org/10.5958/2321-5836.2019.00021.1.

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7

Pereira, Gilberto R., and Amy H. Zucker. "Nutritional Deficiencies in the Neonate." Clinics in Perinatology 13, no. 1 (March 1986): 175–89. http://dx.doi.org/10.1016/s0095-5108(18)30845-5.

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8

Gimenez, María S., Liliana B. Oliveros, and Nidia N. Gomez. "Nutritional Deficiencies and Phospholipid Metabolism." International Journal of Molecular Sciences 12, no. 4 (April 6, 2011): 2408–33. http://dx.doi.org/10.3390/ijms12042408.

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9

Mehta, Nilesh M., and Christopher P. Duggan. "Nutritional Deficiencies During Critical Illness." Pediatric Clinics of North America 56, no. 5 (October 2009): 1143–60. http://dx.doi.org/10.1016/j.pcl.2009.06.007.

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10

Suskind, David L. "Nutritional Deficiencies During Normal Growth." Pediatric Clinics of North America 56, no. 5 (October 2009): 1035–53. http://dx.doi.org/10.1016/j.pcl.2009.07.004.

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11

Mziray-Andrew, Charmaine H., and Timothy A. Sentongo. "Nutritional Deficiencies in Intestinal Failure." Pediatric Clinics of North America 56, no. 5 (October 2009): 1185–200. http://dx.doi.org/10.1016/j.pcl.2009.07.005.

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12

Kumar, Neeraj. "Neurologic Presentations of Nutritional Deficiencies." Neurologic Clinics 28, no. 1 (February 2010): 107–70. http://dx.doi.org/10.1016/j.ncl.2009.09.006.

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13

Balint, Jane P. "PHYSICAL FINDINGS IN NUTRITIONAL DEFICIENCIES." Pediatric Clinics of North America 45, no. 1 (February 1998): 245–60. http://dx.doi.org/10.1016/s0031-3955(05)70592-7.

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14

Williams, Faren H. "Neuromuscular Complications of Nutritional Deficiencies." Physical Medicine and Rehabilitation Clinics of North America 19, no. 1 (February 2008): 125–48. http://dx.doi.org/10.1016/j.pmr.2007.10.006.

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15

Davies, D. J., J. M. Baxter, and J. N. Baxter. "Nutritional deficiencies after bariatric surgery." Obesity Surgery 17, no. 9 (September 2007): 1150–58. http://dx.doi.org/10.1007/s11695-007-9208-x.

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16

Bal, Bikram S., Frederick C. Finelli, Timothy R. Shope, and Timothy R. Koch. "Nutritional deficiencies after bariatric surgery." Nature Reviews Endocrinology 8, no. 9 (April 24, 2012): 544–56. http://dx.doi.org/10.1038/nrendo.2012.48.

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17

Yu, Jennifer C., Ksenya Shliakhtsitsava, YunZu M. Wang, Megan Paul, Lauge Farnaes, Victor Wong, Jenny Kim, and Courtney D. Thornburg. "Hematologic Manifestations of Nutritional Deficiencies." Journal of Pediatric Hematology/Oncology 41, no. 3 (April 2019): e182-e185. http://dx.doi.org/10.1097/mph.0000000000001338.

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18

MacDonald, A., and A. Forsyth. "Nutritional deficiencies and the skin." Clinical and Experimental Dermatology 30, no. 4 (July 2005): 388–90. http://dx.doi.org/10.1111/j.1365-2230.2005.01787.x.

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19

Salvati, Lisa M. "Nutritional Deficiencies in HIV Disease." AIDS Patient Care 7, no. 6 (December 1993): 312–13. http://dx.doi.org/10.1089/apc.1993.7.312.

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20

Purandare, C. N. "Maternal Nutritional Deficiencies and Interventions." Journal of Obstetrics and Gynecology of India 62, no. 6 (December 2012): 621–23. http://dx.doi.org/10.1007/s13224-013-0347-9.

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21

Barton, Susan H., Darlene G. Kelly, and Joseph A. Murray. "Nutritional Deficiencies in Celiac Disease." Gastroenterology Clinics of North America 36, no. 1 (March 2007): 93–108. http://dx.doi.org/10.1016/j.gtc.2007.01.006.

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22

Davies, D. J., J. M. Baxter, and J. N. Baxter. "Nutritional deficiencies after bariatric surgery." Obesity Surgery 17, no. 9 (September 2007): 1150–58. http://dx.doi.org/10.1007/bf02802083.

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23

Robbins, Karen A., and Burcin Uygungil. "Nutritional Deficiencies and Food Allergy." Journal of Allergy and Clinical Immunology: In Practice 5, no. 2 (March 2017): 528–29. http://dx.doi.org/10.1016/j.jaip.2017.01.007.

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24

Twersky, Yitzhak, and Simmy Bank. "Nutritional Deficiencies in Chronic Pancreatitis." Gastroenterology Clinics of North America 18, no. 3 (September 1989): 543–65. http://dx.doi.org/10.1016/s0889-8553(21)00641-5.

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25

Zahodnik, Tenell. "Evaluating Patients for Nutritional Deficiencies." Physician Assistant Clinics 6, no. 4 (October 2021): 581–92. http://dx.doi.org/10.1016/j.cpha.2021.05.003.

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26

Truswell, A. S. "ABC of nutrition. Other nutritional deficiencies in affluent communities." BMJ 291, no. 6505 (November 9, 1985): 1333–37. http://dx.doi.org/10.1136/bmj.291.6505.1333.

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27

Raju, G., and Ch Archana. "Prevalence of Nutritional Deficiencies in Undernourished Hospital Visiting Paediatric Patients." Pediatric Education and Research 6, no. 1 (2018): 21–25. http://dx.doi.org/10.21088/per.2321.1644.6118.4.

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28

Bertani, Lorenzo, Davide Giuseppe Ribaldone, Massimo Bellini, Maria Gloria Mumolo, and Francesco Costa. "Inflammatory Bowel Diseases: Is There a Role for Nutritional Suggestions?" Nutrients 13, no. 4 (April 20, 2021): 1387. http://dx.doi.org/10.3390/nu13041387.

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Nutrition has an important impact on inflammatory bowel diseases (IBD). In particular, several studies have addressed its role in their pathogenesis, showing how the incidence of IBD significantly increased in recent years. Meanwhile, nutrition should be considered a component of the treatment of the disease, both as a therapy itself, and especially in the perspective of correcting the various nutritional deficiencies shown by these patients. In this perspective, nutritional suggestions are very important even in the most severe forms of IBD, requiring hospitalization or surgical treatment. Although current knowledge about nutrition in IBD is increasing over time, nutritional suggestions are often underestimated by clinicians. This narrative review is an update summary of current knowledge on nutritional suggestions in IBD, in order to address the impact of nutrition on pathogenesis, micro- and macro-nutrients deficiencies (especially in the case of sarcopenia and obesity), as well as in hospitalized patients.
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29

Galimberti, Fabrizio, and Natasha A. Mesinkovska. "Skin findings associated with nutritional deficiencies." Cleveland Clinic Journal of Medicine 83, no. 10 (October 2016): 731–39. http://dx.doi.org/10.3949/ccjm.83a.15061.

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30

Carney, Michael W. P. "Neuropsychiatric Disorders Associated With Nutritional Deficiencies." CNS Drugs 3, no. 4 (April 1995): 279–90. http://dx.doi.org/10.2165/00023210-199503040-00005.

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31

Zetu, Cornelia, Rubin Munteanu, Raluca Parasca, and Constantin Ionescu-Tîrgovişte. "Nutritional Deficiencies Associated to Bariatric Surgery." Romanian Journal of Diabetes Nutrition and Metabolic Diseases 20, no. 2 (June 1, 2013): 157–64. http://dx.doi.org/10.2478/rjdnmd-2013-0020.

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Abstract Obesity is an epidemic disease, increasingly addressed through surgical options for weight loss. Benefits of these surgical procedures, such as weight loss and improvement of obesity-related co-morbidities, are well established. However, postoperative complications do occur. Deficiencies in micronutrients, which include water and fat-soluble vitamins and minerals, are common after bariatric surgery. Recognition of the clinical signs and symptoms of micronutrient deficiencies is important to minimize long-term adverse effects.
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32

Grantham-McGregor, S. M., S. P. Walker, and S. Chang. "Nutritional deficiencies and later behavioural development." Proceedings of the Nutrition Society 59, no. 1 (February 2000): 47–54. http://dx.doi.org/10.1017/s0029665100000069.

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The literature on the long-term effects of nutritional deficiencies in early life is reviewed. The severity and duration of the deficiency, the stage of the children’s development, the biological condition of the children and the socio-cultural context may all modify the effect. There is substantial evidence that reduced breast-feeding, small-for-gestational-age birth weight, Fe and I deficiency, and protein-energy malnutrition (PEM) are associated with long-term deficits in cognition and school achievement. However, all these conditions are associated with poverty and poor health, which may account for the association. It is difficult to establish that the long-term relationship is causal, as it requires a randomized treatment trial with long-term follow-up. Such studies are only available for I deficiency in utero and early childhood PEM. Results from these studies indicate that I deficiency has a long-term effect and PEM probably has a long-term effect.
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33

Yehuda, S., S. Rabinovitz, and DI Mostofsky. "Nutritional Deficiencies in Learning and Cognition." Journal of Pediatric Gastroenterology and Nutrition 43, Suppl 3 (December 2006): S22—S25. http://dx.doi.org/10.1097/01.mpg.0000255847.77034.a4.

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34

Molina, J. A., F. Bermejo, T. Ser, F. J. Jiménez-Jiménez, A. Herranz, P. Fernández-Calle, B. Ortuño, C. Villanueva, and M. J. Sainz. "Alcoholic cognitive deterioration and nutritional deficiencies." Acta Neurologica Scandinavica 89, no. 5 (January 29, 2009): 384–90. http://dx.doi.org/10.1111/j.1600-0404.1994.tb02651.x.

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35

Mirtallo, Jay M. "Book Review: Drug-Induced Nutritional Deficiencies." Drug Intelligence & Clinical Pharmacy 20, no. 9 (September 1986): 722. http://dx.doi.org/10.1177/106002808602000928.

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36

Krause, John R. "The Bone Marrow in Nutritional Deficiencies." Hematology/Oncology Clinics of North America 2, no. 4 (December 1988): 557–66. http://dx.doi.org/10.1016/s0889-8588(18)30584-7.

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37

Fung, Ellen B. "Nutritional deficiencies in patients with thalassemia." Annals of the New York Academy of Sciences 1202, no. 1 (August 2010): 188–96. http://dx.doi.org/10.1111/j.1749-6632.2010.05578.x.

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38

Stenram, U. "5-FU toxicity and nutritional deficiencies." British Journal of Cancer 67, no. 5 (May 1993): 1157. http://dx.doi.org/10.1038/bjc.1993.214.

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39

Schweiger, Chaya, Ram Weiss, Elliot Berry, and Andrei Keidar. "Nutritional Deficiencies in Bariatric Surgery Candidates." Obesity Surgery 20, no. 2 (October 30, 2009): 193–97. http://dx.doi.org/10.1007/s11695-009-0008-3.

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40

Hiew, Fu-Liong. "SY7.7. Neuropathy due to nutritional deficiencies." Clinical Neurophysiology 132, no. 8 (August 2021): e50-e51. http://dx.doi.org/10.1016/j.clinph.2021.02.063.

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41

Dembiński, Łukasz, Artur Mazur, Mariusz Dąbrowski, Teresa Jackowska, and Aleksandra Banaszkiewicz. "Knowledge of Medical Students and Medical Professionals Regarding Nutritional Deficiencies in Patients with Celiac Disease." Nutrients 13, no. 6 (May 22, 2021): 1771. http://dx.doi.org/10.3390/nu13061771.

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A gluten-free diet provides relief from symptoms for patients with celiac disease, although there is still a risk of nutritional deficiencies. These patients can potentially consume an excessive amount of fat and insufficient amounts of fiber, iron, vitamin D, and calcium. This study aimed to assess the knowledge of medical students and healthcare professionals in Poland regarding nutritional deficiencies and the prevention of such deficiencies in patients with celiac disease who are on a gluten-free diet. Of the 430 survey participants, 46% did not realize the risk of nutritional deficiencies in patients with celiac disease. The knowledge of the participants was lowest regarding the risk of being overweight or obese. Among the healthcare professionals, an acceptable level of correct answers was provided by only 37% of individuals and was highest for the dietitians’ group. Our results demonstrate the need to improve the education of healthcare professionals concerning nutrition in patients with celiac disease.
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42

Gana, Wassim, Arnaud De Luca, Camille Debacq, Fanny Poitau, Pierre Poupin, Amal Aidoud, and Bertrand Fougère. "Analysis of the Impact of Selected Vitamins Deficiencies on the Risk of Disability in Older People." Nutrients 13, no. 9 (September 10, 2021): 3163. http://dx.doi.org/10.3390/nu13093163.

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Vitamin deficiencies have a serious impact on healthy aging in older people. Many age-related disorders have a direct or indirect impact on nutrition, both in terms of nutrient assimilation and food access, which may result in vitamin deficiencies and may lead to or worsen disabilities. Frailty is characterized by reduced functional abilities, with a key role of malnutrition in its pathogenesis. Aging is associated with various changes in body composition that lead to sarcopenia. Frailty, aging, and sarcopenia all favor malnutrition, and poor nutritional status is a major cause of geriatric morbidity and mortality. In the present narrative review, we focused on vitamins with a significant risk of deficiency in high-income countries: D, C, and B (B6/B9/B12). We also focused on vitamin E as the main lipophilic antioxidant, synergistic to vitamin C. We first discuss the role and needs of these vitamins, the prevalence of deficiencies, and their causes and consequences. We then look at how these vitamins are involved in the biological pathways associated with sarcopenia and frailty. Lastly, we discuss the critical early diagnosis and management of these deficiencies and summarize potential ways of screening malnutrition. A focused nutritional approach might improve the diagnosis of nutritional deficiencies and the initiation of appropriate clinical interventions for reducing the risk of frailty. Further comprehensive research programs on nutritional interventions are needed, with a view to lowering deficiencies in older people and thus decreasing the risk of frailty and sarcopenia.
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43

Pavithran, Sreeshma, and D. D. Bant. "Nutritional status of adolescent school girls residing in rural areas of Dharwad district, India: a cross sectional study." International Journal Of Community Medicine And Public Health 5, no. 7 (June 22, 2018): 2761. http://dx.doi.org/10.18203/2394-6040.ijcmph20182417.

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Background: Adolescence is a period with rapid growth drive and demands higher nutrition. Improving Nutritional status of rural adolescent girls becomes the cornerstone of community’s nutritional status and target intervention for breaking intergeneration cycle of nutritional deficiencies. The objective of the study was to determine nutritional status and study the demographic factors affecting nutritional status of rural adolescent girls in Dharwad district.Methods: A cross-sectional study was carried out among 350 adolescent school girls studying in eight high schools in rural area of Dharwad district during July – September 2017. Demographic profile and dietary pattern was obtained. Anthropometric measurement and clinical examination was done. Statistical analysis was done using SSPS package.Results: Study found 14.9% of rural adolescent girls were under- weight for their age. Based on BMI, 25.2% of girls were under-nourished and 3.7% were over nourished. Significant relation with age, type of diet and age of menarche was found at p value <0.05.Conclusions: There is high prevalence of under nutrition among adolescent girls and is under nutrition is associated with micronutrient deficiencies like anaemia. There is need to create awareness to improve the nutritional needs of adolescent girls in rural areas.
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44

Ayers, Emmeline, and Joe Verghese. "Locomotion, cognition and influences of nutrition in ageing." Proceedings of the Nutrition Society 73, no. 2 (November 1, 2013): 302–8. http://dx.doi.org/10.1017/s0029665113003716.

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Gait and cognitive impairments in older adults can reflect the simultaneous existence of two syndromes that affect certain brain substrates and pathologies. Nutritional deficiencies, which are extremely common among elderly population worldwide, have potential to impact the existence and rehabilitation of both syndromes. Gait and cognition are controlled by brain circuits which are vulnerable to multiple age-related pathologies such as vascular diseases, inflammation and dementias that may be caused or accentuated by poor nutrition or deficiencies that lead to cognitive, gait or combined cognitive and gait impairments. The following review aims to link gait and cognitive classifications and provide an overview of the potential impact of nutritional deficiencies on both neurological and gait dysfunctions. The identification of common modifiable risk factors, such as poor nutrition, may serve as an important preventative strategy to reduce cognitive and mobility impairments and moderate the growing burden of dementia and disability worldwide.
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45

Rice, Marylynne A., and Richard H. Haas. "The Nutritional Aspects of Rett Syndrome." Journal of Child Neurology 3, no. 1_suppl (January 1988): S35—S42. http://dx.doi.org/10.1177/0883073888003001s08.

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Nutrition is a major problem for the Rett patient. We have studied 21 girls with Rett syndrome (19 typical, two atypical). We report our experience in this population with the nutritional aspects of Rett syndrome, the typical dietary habits, and various nutritional deficiencies. Further experience with the use of high fat diets is reported. (J Child Neurol 1988;3(Suppl):S35-S42).
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46

Siddiqui, Mohamed Tausif, Megan Maisano, Amandeep Singh, Wael A. Al-Yaman, Brad Dworkin, and Donald F. Kirby. "Su1742 IMPACT OF NUTRITIONAL DEFICIENCIES IN GASTROPARESIS." Gastroenterology 158, no. 6 (May 2020): S—628—S—629. http://dx.doi.org/10.1016/s0016-5085(20)32270-8.

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47

Combet, Emilie, and Christina Buckton. "Micronutrient deficiencies, vitamin pills and nutritional supplements." Medicine 43, no. 2 (February 2015): 66–72. http://dx.doi.org/10.1016/j.mpmed.2014.11.002.

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48

Combet, Emilie, and Christina Buckton. "Micronutrient deficiencies, vitamin pills and nutritional supplements." Medicine 47, no. 3 (March 2019): 145–51. http://dx.doi.org/10.1016/j.mpmed.2018.12.004.

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49

Kirby, Midge, and Elaine Danner. "Nutritional Deficiencies in Children on Restricted Diets." Pediatric Clinics of North America 56, no. 5 (October 2009): 1085–103. http://dx.doi.org/10.1016/j.pcl.2009.07.003.

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50

Claster, Susan, John Wood, Susan Carson, Thomas Hofstra, Anne Nord, Rachna Khanna, and Thomas Coates. "Nutritional Deficiencies in Chronically Transfused Hemoglobinopathy Patients." Blood 110, no. 11 (November 16, 2007): 1735. http://dx.doi.org/10.1182/blood.v110.11.1735.1735.

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Abstract Vitamin and mineral deficiencies have been well described in non-transfused patients with sickle cell disease (SCD). Levels of biomarkers of oxidant damage are increased in both SCD and thalassemia major (TM), and levels of antioxidants have been shown to be lower in both diseases. Hypermetabolism, increased hemolysis and denaturation of unpaired globin chains contribute to oxidative stress in SCD and TM. Chronic transfusions, initially thought to be protective against oxidation by marrow suppression, may have the opposite effect due to the well-known propensity of iron to promote free radical generation. Methods: To test this hypothesis, we measured levels of vitamins A, B1, B6, C, D, E as well as selenium, zinc, copper and carnitine and compared them to liver iron concentrations in chronically transfused SCD and TM patients. All patients were required to fast overnight and to hold their iron chelator for 24 hours prior to blood draws. Samples were processed by routine clinical processing (Quest Nichols Institute, San Juan Capistrano, CA). Results were expressed as the percentage of patients outside the reference range for the referral laboratory. Twenty four patients with TM and 43 patients with SCD were studied. Abnormal Values In Transfused Patients SC (%abn) TM (%abn) A 73.7 52.4 B1 38.5 37.5 B6 34.2 34.8 Folate 28.6 37.5 C 56.7 66.7 D25 74.4 50.0 D1, 25 48.6 39.1 Se 67.5 75.0 Zn 24.3 8.3 Carnitine (Free) 22.2 41.7 Results: The results in the table demonstrate that over half the patients were deficient in A, C, D and selenium. A third of the patients had low levels of B vitamins and folate (despite replacement doses in the latter), as well as carnitine. B12 levels(not shown) were normal. E levels (not shown) were low in only a few patients. Deficiency profiles were remarkably similar between the two diseases: only copper was significantly higher in the SCD patients (34.2% elevated vs 0% in TM). B6 and folate were weakly correlated with hepatic iron concentration (r2 = −0.12, −0.18). B6, folate, D25-OH, and D1,25 OH levels declined with age. These results demonstrate that chronically transfused patients have significant deficiencies in nutrients which are involved in buffering oxidant stress, regardless of their diagnosis. Severity of iron overload was not a strong predictor of these abnormalities; although the precise explanation is not known, chronic anemia may be the unifying factor. The magnitude of B1, B6, C and D deficiencies in some patients was well into the range known to cause serious complications. These combined abnormalities may contribute to the morbidity of chronically transfused patients with SCD and TM.
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