Academic literature on the topic 'Nutritional Deficiency'

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Journal articles on the topic "Nutritional Deficiency"

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Altaf, Benash, Mohammad Behram Khan, Rana Khurram Aftaab, Shireen Jawed, Rana Muhammad Tahir Salam, and Farah Amir. "NUTRITIONAL DEFICIENCY ANEMIA;." Professional Medical Journal 25, no. 07 (July 11, 2018): 1018–23. http://dx.doi.org/10.29309/tpmj/18.4449.

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Wharton, Brian, and Pamela Wharton. "Nutrition in Adolescence." Nutrition and Health 4, no. 4 (January 1987): 195–203. http://dx.doi.org/10.1177/026010608700400403.

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This review discusses five questions concerning nutrition in adolescence. Do adolescents have special nutritional needs? What are the recommended dietary intakes? What do adolescent schoolchildren eat? What nutritional disorders occur at this age? The prevalence of iron deficiency anaemia, rickets, coeliac disease, iodine deficiency, dental caries, weight disorders, zinc deficiency and pellagra are discussed. What is the role of school meals or other food supplementation programmes? The primary school years (5–11 in Britain) are relatively quiet nutritionally. Growth occurs at a steady rate neither accelerating nor decelerating, nutritional diseases are less common and in the majority no profound biological events occur. Nutritional interest is in the secondary school child (11–18 years in Britain) particularly in the events of puberty and adolescence. This review aims to determine five points concerning adolescents. Do they have special nutritional needs? What are their recommended dietary intakes? What do they eat? What nutritional disorders occur at this age? What is the role of school meals or other food supplementation programmes?
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Zimmermann, Michael B., and Richard F. Hurrell. "Nutritional iron deficiency." Lancet 370, no. 9586 (August 2007): 511–20. http://dx.doi.org/10.1016/s0140-6736(07)61235-5.

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Stanbury, John B. "Nutritional Deficiency Disorders." Food and Nutrition Bulletin 7, no. 2 (June 1985): 1–8. http://dx.doi.org/10.1177/156482658500700201.

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Altaf, Benash, Mohammad Behram Khan, Rana Khurram Aftaab, Shireen Jawed, Rana Muhammad Tahir Salam, and Farah Amir. "NUTRITIONAL DEFICIENCY ANEMIA." Professional Medical Journal 25, no. 07 (July 10, 2018): 1018–23. http://dx.doi.org/10.29309/tpmj/2018.25.07.115.

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Nutritional anemia is emerging health issue affecting all the age groups but moreprevalent among the youngsters due to poor eating habits and consumption of more junkfood which lacks essential ingredients. The aim is to determine the frequency of anemia andto find its association with junk food among medical students. Study Design: Cross sectionalstudy. Setting: Comprises of 100 students was conducted at Aziz Fatimah Medical and DentalCollege Faisalabad. Period: Six months, 01-01-2017 to 30-06-2017. Methodology: After ethicalapproval all relevant information including age, height, BMI, consumption of junk food andother dietary habits were taken on predesigned proforma. Hb levels were checked by Sahil’smethod in department of Physiology to evaluate the relation of anemia with junk food. Data wasanalyzed by SPPS 20. Results: Study also revealed that 55(49.1%) of the study participants wereconsuming junk food frequently. Among the junk food consumers 69.1% were anemic and theirhemoglobin levels were lower than non –consumers of junk food. Regression analysis showsnegative association between junk food and hemoglobin levels (P value 0.009). Conclusion:Anemia is frequently found in junk food consumers then non junk food consumers.
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Kravtsova, E. I., I. I. Kutsenko, I. O. Borovikov, and S. K. Batmen. "Nutrition and nutritional support during pregnancy." Russian Journal of Woman and Child Health 3, no. 4 (2020): 233–41. http://dx.doi.org/10.32364/2618-8430-2020-3-4-233-241.

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Increased nutrient needs during pregnancy are accounted for by physiological changes in the maternal organism and requirements of the developing and growing baby. They are realized through a number of physiological adaptations in nutrient metabolism. Physiological pregnancy and adequate fetal development are impossible in deficient nutrients, vitamins, and microelements. Randomized clinical trials in pregnant women have demonstrated that nutrient deficiency in the maternal organism results in pregnancy complications and health problems in the newborn but also accounts for a wide range of chronic disorders among the offspring. Probiotics improve digestive health in pregnant women and affect epigenetic programming of human homeostasis that predisposes to dysbiosis, allergies, and lipid and carbohydrate metabolism disorders. Prevention of vitamin and probiotic deficiency during pre-pregnancy planning, pregnancy, and breastfeeding focuses on ensuring that vitamin intake from food meets the requirements for these nutrients. Complex vitamin supplements containing probiotics treat alimentary deficiency and contribute to the favorable course of pregnancy and fetal development. KEYWORDS: nutrition, woman’s health, pregnancy, vitamins, probiotics, nutrient deficiency. FOR CITATION: Kravtsova E.I., Kutsenko I.I., Borovikov I.O., Batmen S.K. Nutrition and nutritional support during pregnancy. Russian Journal of Woman and Child Health. 2020;3(4):233–240. DOI: 10.32364/2618-8430-2020-3-4-233-241.
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Akhmadeeva, L. R., M. V. Naprienko, O. S. Lazovaya, G. S. Zagidullina, A. F. Timirova, R. Kh Gizatullin, and L. V. Smekalkina. "Rehabilitation after stroke: nutritional status and its correction." Medical alphabet 1, no. 11 (July 16, 2020): 17–20. http://dx.doi.org/10.33667/2078-5631-2020-11-17-20.

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This article presents the results of our study of the nutritional status of patients after cerebral stroke who were hospitalized to the medical rehabilitation unit. Among all patients, 16 % had an increased risk of nutritional deficiency by all scales with the highest numbers in patients over 65 (44 % had a risk of nutritional deficiency on all scales). Among patients with swallowing disorders, 100 % of patients had a risk of nutritional deficiency. Out of all anthropometric indicators, only the circumference of the shoulder muscles was significantly lower in patients with nutritional deficiency. Assessment and correction of malnutrition during rehabilitation after stroke is recommended during the acute treatment and follow-up. Special clinical nutrition products can be a good support for balanced feeding for more rapid rehabilitation.
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Coodley, Gregg. "Nutritional Deficiency and AIDS." Annals of Internal Medicine 113, no. 10 (November 15, 1990): 807. http://dx.doi.org/10.7326/0003-4819-113-10-807_2.

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Hansen, Ronald C. "Dermatitis and Nutritional Deficiency." Archives of Dermatology 128, no. 10 (October 1, 1992): 1389. http://dx.doi.org/10.1001/archderm.1992.01680200099017.

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HANSEN, RONALD M. "Dermatitis and Nutritional Deficiency." American Journal of Diseases of Children 147, no. 9 (September 1, 1993): 940. http://dx.doi.org/10.1001/archpedi.1993.02160330030011.

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Dissertations / Theses on the topic "Nutritional Deficiency"

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Main, A. N. H. "Clinical and experimental studies of nutritional deficiency and nutritional therapy in Crohn's disease." Thesis, University of Edinburgh, 1989. http://hdl.handle.net/1842/19094.

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雷志鵬 and Chi-pang Lui. "Nutritional zinc-deficiency and nitrosamine-induced carcinogenesis in the rat." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1986. http://hub.hku.hk/bib/B31207820.

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Lui, Chi-pang. "Nutritional zinc-deficiency and nitrosamine-induced carcinogenesis in the rat /." [Hong Kong : University of Hong Kong], 1986. http://sunzi.lib.hku.hk/hkuto/record.jsp?B12326550.

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Moreno, Reyes Mario Rodrigo. "Clinical and experimental studies of nutritional selenium and iodine deficiency." Doctoral thesis, Universite Libre de Bruxelles, 2002. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/211435.

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Sibani, Sahar. "Genetic and nutritional folate deficiency : implications for homocystinuria and intestinal neoplasia." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31539.

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Folate deficiency, a prevalent vitamin deficiency in America, can stem from environmental and/or genetic causes. The most common inborn error of folate metabolism is deficiency of methylenetetrahydrofolate reductase (MTHFR), which catalyzes the reduction of 5,10-methylenetetrahydrofolate to 5-methyltetrahydrofolate. Severe MTHFR deficiency results in hyperhomocysteinemia and homocystinuria; patients present with developmental delay, and various neurological and vascular disorders. This thesis describes three mutations identified in the MTHFR locus in patients with severe deficiency: 1025T→C (M→T), 1027T→G (W→G), and 1768G→A (E→K). Genotype-phenotype correlations are described, along with biochemical characterization of three mutations (983A→G (N→S), 1025T→C, 1027T→G). All three mutations exert their effect by decreasing Vmax without changing the enzyme's affinity for its substrate, 5-methyltetrahydrofolate. The 983A→G variant also conferred decreased affinity for FAD, a cofactor.
The more common and mild deficiency observed in the general healthy population is probably due in part to insufficient dietary intake of folate. Folate deficiency has been associated with increased risk for colon cancer. In a pilot study presented here, the impact of altered folate intake on tumor multiplicity in the Min mouse, a model for multiple intestinal neoplasia, was assessed. Folate deficient diets did not produce a consistent change in tumor numbers. However, a linear correlation between S-adenosylmethionine and S-adenosylhomocysteine content of preneoplastic tissue and tumor multiplicity was identified.
This thesis contributes to our understanding of the impact of genetic- and/or dietary-induced folate deficiency on cellular and organismal functions.
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Hughmark, Christine Ann. "Changes in indicators of nutritional status during hospitalization." Thesis, Virginia Tech, 1990. http://hdl.handle.net/10919/41926.

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The medical records of 263 patients hospitalized between 14 and 28 days in a 405-bed, acute care, community hospital were reviewed before 100 records were obtained with complete data to determine percent recommended body weight (RBW), serum albumin, total lymphocyte count (TLC), and hematocrit on admission and near discharge. Serum albumin and hematocrit were the only indicators that changed significantly during hospitalization, with both decreasing from admission to discharge. Considering the four indicators together, changes in nutritional status of the patients during hospitalization were determined. Eighty three patients were found to be at least at moderate potential for nutritional risk on admission, and 95 were found to be at least at moderate potential for nutritional risk near discharge. No patients who were judged to be potentially at severe nutritional risk on admission improved their nutritional status during hospitalization. Paired comparisons of the four parameters indicated that hematocrit and serum albumin seemed to be measuring changes in potential nutritional status more similarly than any other pair of assessment indicators.

Sixty-three of the patients did not receive what was judged to be adequate kilocalories and protein during hospitalization. Twelve of these patients were found to have an increase in potential nutritional risk status.

Of the 163 medical records that were reviewed but did not have all indicators recorded, height was most often missing on admission, and weight and serum albumin were most often missing near discharge.
Master of Science

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Power, Harold Michael. "A study of iron nutrition and immunity in infancy." Master's thesis, University of Cape Town, 1988. http://hdl.handle.net/11427/25837.

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Motivation and study design: Iron deficiency is a common condition in infancy, particularly in lower socio-economic groups. In Cape Town it remains a problem in spite of public health measures taken against it: a recent survey found a prevalence of iron deficiency anaemia of 34% in healthy 1-year old term infants who had ready access to a municipal health clinic where iron fortified milk formula is sold at subsidized prices. The consequences of iron deficiency extend beyond anaemia- to involve all organ systems including the immune system. Since Helen Mackay's report in 1928 of a striking decrease in incidents of infection in infants treated with iron, clinicians have assumed that iron deficiency predisposes to infection. Despite a sound theoretical basis for this belief, the clinical evidence for the assumption is poor as studies to date have displayed methodological deficiencies. On the other hand, iron is also essential for the growth of micro-organisms. As such, supplemental iron may predispose to infection. Indeed, there is much laboratory and clinical evidence to show that excess iron can result in the recrudescence of quiescent infections and increase the virulence of newly acquired infections. Thus, the competition between host and parasite may sometimes hinge on the relative availability of iron and it has been speculated that excess iron in infant milk formula may increase susceptibility to infectious diarrhoeal disease. The problem addressed by this thesis was to determine the utility of increasing the level of iron fortification of infant milk formula. Three questions were posed: Does increasing the level of iron fortification of conventional infant milk formula improve the iron nutrition of normal infants fed on the formula? Does increased iron fortification of infant milk formula alter immunity as reflected by incidence of infection and laboratory tests of immune function? Are there any handful effects of increasing the quantity of iron in conventional infant milk formula? A double blind randomized trial was carried out in 1983 and 1984 to answer these questions. A group of 149 healthy, well-nourished infants from a lower socio-economic community of so called Cape Coloureds were followed from the age of 3 months to 1 year. Half of the infants, the Control group, were given a commercially available infant milk formula (Lactogen Full Protein) which has 8.3 mg Fe/ 100 g formula and 37 mg ascorbic acid/ 100 g. The other half of subjects, the Test group, were given the same milk formula but fortified with iron to a concentration of 40 mg Fe/ 100 g. The children were examined every 3 or 4 weeks and any infection or history of infection was noted. Laboratory tests were done at the start of the trial and again on completion. During the trial, laboratory tests were performed only if clinically indicated. The tests included full blood count and differential analysis, red cell zinc protoporphyrin, plasma ferritin, plasma and hair zinc and lymphocyte subtyping with monoclonal antibodies. Within each group, half of the infants were randomly selected for assay of neutrophil bactericidal activity. The other half were assayed for lymphocyte blastogenic response to stimulation with phytohaemagglutinin. Tests of delayed cutaneous hypersensitivity to Candida antigen and PPD were done and all children and their mothers had antibodies to tetanus and polio determined. Results: 74 infants in the Control group started the trial and 62 completed it. In the Test group, 75 infants began and 70 completed the study. Intake of milk and solid foods was not quantified, but the ages of weaning and of introduction of new foods were determined. The Control and Test groups did not differ significantly on any test item. The mean age of completion of weaning was 3.60 months for the Control group and 4.04 months for the Test group. The Control group was first given meat or fish at a mean age of 5.19 months; the Test. group had meat or fish introduced to their diets at a mean age of 4.36 months. These differences were not statistically significant. The children in the Control group were lighter and shorter than the Test group at the end of the year. Mean standard deviation scores for weight were 0.23 and 0.48 respectively (P = 20%), while for length the SD scores were -0.13 and 0.06 (P = 20%).
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Chan, Jessica See Wen 1984. "The role of 5,10-methylenetetrahydrofolate reductase and nutritional deficiencies in cardiac development /." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111553.

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Disruptions in folate metabolism are known to increase the risk for neural tube defects (NTD) and this is preventable by folic acid supplementation. However, the relationship between folate metabolism and cardiac development remains unclear. The interaction between other folate pathway nutrients, choline and riboflavin, and folate metabolism was studied in a murine model of methylenetetrahydrofolate reductase (MTHFR) deficiency. Maternal choline deficiency, riboflavin deficiency and MTHFR deficiency adversely affected embryonic or heart development. The promoters of MTHFR were also examined for interactions with GATA-4, TBX5, MEF2A and NKX-2.5, known transcription factors of cardiac development. Upstream promoter activity was increased in the presence of GATA-4 and this interaction was further enhanced upon the addition of MEF2A. TBX5 appeared to decrease upstream promoter activity. GATA-4 modestly increased downstream promoter activity. These results highlight the importance of adequate nutrient intake during pregnancy and provide a link between folate metabolism and cardiac development.
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Asch, Ruth H. "Neurodevelopmental Consequences of Maternal Omega-3 Fatty Acid Deficiency." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1583154907162725.

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Streeter, Tania C. "Role of Zn nutritional status on infection of Medicago species by Rhizoctonia solani /." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09APSP/09apsps915.pdf.

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Books on the topic "Nutritional Deficiency"

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Environment, nutritional deficiency and its improvement. New Delhi: Northern Book Centre, 1990.

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Task Force Sight and Life, ed. Nutritional anemia. Basel, Switzerland: Sight and Life Press, 2007.

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Nutrition and the eyes: How to keep your eyes health naturally. Montclair, CA: Health Spectrum Publishers, 1994.

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Lorenc, R. S. Nutritional requirements for intestinal calcium and phosphate transport: Final annual report, July 1, 1985 - June 30, 1988. Warsaw, Poland: Hospital-Monument Child's Health Center, 1988.

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Fernandez, Renate Lellep. A simple matter of salt: An ethnography of nutritional deficiency in Spain. Berkeley: University of California Press, 1990.

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Herbers, Mary Ruth. Vitamin A: An urgent nutritional need for the world's children. Washington: U.S. G.P.O., 1985.

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Herbers, Mary Ruth. Vitamin A: An urgent nutritional need for the world's children. Washington: U.S. G.P.O., 1985.

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Herbers, Mary Ruth. Vitamin A: An urgent nutritional need for the world's children. Washington: U.S. G.P.O., 1985.

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Tacon, Albert G. J. Nutritional fish pathology: Morphological signs of nutrient deficiency and toxicity in farmed fish. Rome: Food and Agriculture Organization of the United Nations, 1992.

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West, Keith P. Delivery of oral doses of vitamin A to prevent vitamin A deficiency and nutritional blindness: A state-of-the-art review. Baltimore, Md: International Center for Epidemiologic and Preventive Opthalmology, 1987.

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Book chapters on the topic "Nutritional Deficiency"

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Froehlich, Stephan J., Carlo A. Lackerbauer, Guenter Rudolph, Jan Rémi, Soheyl Noachtar, Werner J. Heppt, Annette Cryer, et al. "Nutritional Deficiency." In Encyclopedia of Molecular Mechanisms of Disease, 1503. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6164.

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Said, Moheb S., and Khanjan H. Nagarsheth. "Nutritional Deficiency/Starvation." In Encyclopedia of Trauma Care, 1095–100. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-29613-0_142.

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Moy, Robert J. D. "Nutritional Iron Deficiency in Early Childhood." In Iron Deficiency and Overload, 313–26. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-462-9_18.

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Misra, U. K., and J. Kalita. "Niacin deficiency: Pellagra." In Neurological Consequences of Nutritional Disorders, 47–57. First edition. | Boca Raton, FL : Taylor & Francis, 2021.: CRC Press, 2021. http://dx.doi.org/10.1201/9780429316401-4.

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Misra, U. K., and J. Kalita. "Vitamin D deficiency." In Neurological Consequences of Nutritional Disorders, 89–101. First edition. | Boca Raton, FL : Taylor & Francis, 2021.: CRC Press, 2021. http://dx.doi.org/10.1201/9780429316401-7.

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Kayden, Herbert J., and Maret G. Traber. "Clinical, Nutritional and Biochemical Consequences of Apolipoprotein B Deficiency." In Lipoprotein Deficiency Syndromes, 67–81. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1262-8_7.

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Rosenthal, Mark J., and James S. Goodwin. "Cognitive Effects of Nutritional Deficiency." In Advances in Nutritional Research, 71–100. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2529-1_3.

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Aboussaleh, Youssef, and Samir Bikri. "Cognitive and Behavioral Consequences of Brain Iron Deficiency." In Nutritional Neurosciences, 131–42. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-6467-0_8.

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Misra, U. K., and J. Kalita. "Thiamine deficiency neurological disorders." In Neurological Consequences of Nutritional Disorders, 29–46. First edition. | Boca Raton, FL : Taylor & Francis, 2021.: CRC Press, 2021. http://dx.doi.org/10.1201/9780429316401-3.

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Keen, Carl L., Sheri Zidenberg-Cherr, and Bo Lönnerdal. "Dietary Manganese Toxicity and Deficiency." In Nutritional Bioavailability of Manganese, 21–34. Washington, DC: American Chemical Society, 1987. http://dx.doi.org/10.1021/bk-1987-0354.ch003.

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Conference papers on the topic "Nutritional Deficiency"

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Chushkin, Mikhail, Veronika Beketova, Lydia Popova, Anna Egorova, Vladimir Romanov, Natalya Karpina, and Atadzhan Ergeshov. "Nutritional deficiency in patients with pulmonary tuberculosis." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa2282.

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Srinadi, Ni Luh, Theresia Puspitawati, and Nonik Ayu Wantini. "Relationship of Family Economic Status with Chronic Energy Deficiency in Pregnant Women in Jetis Community Health Center, Yogyakarta." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.03.47.

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ABSTRACT Background: Four nutritional problems are still major health problem in Indonesia, namely Chronic Energy Deficiency (KEK), Iron Nutritional Anemia (AGB), Disorders Due to Iodine Deficiency (IDD), and Vitamin A Deficiency (KVA). This study aimed to determine the relationship of family economic status with chronic energy deficiency in pregnant women in Jetis community health center, Yogyakarta. Subjects and Method: This was a cross sectional study conducted at Jetis community health center, Yogyakarta. A sample of 73 was selected by Accidental Sampling. The data were collected by questionnaire and analyzed by Chi Square. Results: The incidence of chronic energy deficiency with low-income families (71.2%), and in the middle economic status the incidence of chronic energy deficiency in pregnant women (92.6%). It was statistically significant (p< 0.001) Conclusion: There is a relationship between families’ economic status and the incidence of chronic energy deficiency (CED) among pregnant women visiting Community Health Center of Jetis, Yogyakarta. Keywords: economic status, incidence of chronic energy deficiency (CED), pregnant women. Correspondence: Theresia Puspitawati. Study program of Public Health, Faculty of Health Sciences, Universitas Respati Yogyakarta. E-mail: thpuspitawati@gmail.com. Mobile: +628122719110. DOI: https://doi.org/10.26911/the7thicph.03.47
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Dhawale, Chitra Anil, Sanjay Misra, Sonika Thakur, and Navin Dattatraya Jambhekar. "Analysis of nutritional deficiency in citrus species tree leaf using image processing." In 2016 International Conference on Advances in Computing, Communications and Informatics (ICACCI). IEEE, 2016. http://dx.doi.org/10.1109/icacci.2016.7732386.

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Wardani, Endah Kusuma, Nurul Eko Widiyastuti, Lutvia Dwi Rofika, and Wahyu Adri Wirawati. "Factors Affecting Stunting among Children Under Five Years of Age in Banyuwangi, East Java." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.03.80.

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ABSTRACT Background: Stunting, chronic malnutrition, results from the exposure of the fetus and young child to nutritional deficiency and infectious disease. In Indonesia, 30.8% of children were stunted, in which 26.2% was in East Java and 8.1% Banyuwangi Regency. This study aimed to investigate the factors affecting stunting among children under five years of age in Banyuwangi, East Java. Subjects and Method: This was a cross-sectional study conducted at Klatak and Wonosobo Community Health Centers, Central Java. a sample of 60 children under five years of age was selected for this study. The study variables were child’s gender, maternal age at pregnancy, maternal education, maternal work status, iron intake, history of chronic energy deficiency, exclusive breastfeeding, supplementary feeding, and history of infectious disease. The frequency distribution data were reported descriptively. Results: The majority of stunted children under study were male (53.3%). Most of the women were at age 20 to 34 years during pregnancy (58.3%). As many as 73.3% mothers were low educated. Most of the mothers were housewives (85%). 78.3% of women took iron supplement during pregnancy. Most of the children did not have the history of chronic energy deficiency (60%). Most of the children received exclusive breastfeeding (61.7%) and supplementary feeding (65%). Only a few children had the history of infectious disease (6.7%). Conclusion: The characteristics of subjects under study vary with maternal age at pregnancy, maternal education, maternal work status, iron intake, history of chronic energy deficiency, exclusive breastfeeding, supplementary feeding, and history of infectious disease. Keywords: stunting, children under five years of age, factors Correspondence: Endah Kusuma Wardani. Midwifery Program, School of Health Sciences Banyuwangi. Jl. Letkol Istiqlah No. 109, Banyuwangi, East Java, 68422. Email: qsuma89@yahoo.com. Mobile: +6282257193736. DOI: https://doi.org/10.26911/the7thicph.03.80
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Soares, Felipe A. L., Efrem E. O. Lousada, Tiago B. Silveira, Raquel A. F. Mini, Luis E. Zárate, and Henrique C. Freitas. "Analysis and Prediction of Childhood Pneumonia Deaths using Machine Learning Algorithms." In Symposium on Knowledge Discovery, Mining and Learning. Sociedade Brasileira de Computação - SBC, 2021. http://dx.doi.org/10.5753/kdmile.2021.17456.

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Acute Respiratory Tract Infections are among the leading causes of child mortality worldwide. Specifically, community-acquired pneumonia has different causes, such as: passive smoking, air pollution, poor hygiene, cardiac insufficiency, oropharyngeal colonization, nutritional deficiency, immunosuppression, and environmental, economic and social factors. Due to the variation of these causes, knowledge discovery in this area of health has been a great challenge for researchers. Thus, this paper presents the steps for the construction of a database and evaluation results applied to the analysis and prediction of potential deaths caused by childhood pneumonia using the Pictorea method. For this, the Random Forest and Artificial Neural Network algorithms were used, and after comparison, the Neural Network algorithm showed higher accuracy by up to 87.57%. This algorithm was used to analyze and predict the number of deaths from pneumonia in children up to 5 years old, and the results were presented using Root Mean Square Error and scatter plots. A domain specialist validated the results and defined that the pattern found is relevant for future studies in the medical field, helping to analyze the behavior of countries and predict future scenarios.
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Machin, S. J., H. Cohen, I. J. Mackie, M. Shearer, and S. D. Scott. "SERUM VITAMIN K1 LEVELS AS AN EARLY INDICATOR OF HYPOPROTHROMBINAEMIA ASSOCIATED WITH ANTIBIOTIC THERAPY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644340.

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The prothrombin time is an insensitive indicator of early vitamin K deficiency and serum vitamin K1 levels may correlate with liver stores. A random non-fasting range of serum vitamin K1 was established in 45 healthy adults of 150-1,530 pg/ml (mean 412 pg/ml). Nine well nourished patients, with normal serum vitamin K1 levels, (mean 546, range 310-1,350 pg/ml), maintained normal prothrombin times and factor VII clotting activity throughout 7 days therapy with cefotetan disodium, an NMTT-containing cephalosporin antibiotic. However, 11 of 20 patients with acute intra-abdominal sepsis and an initially normal prothrombin time who underwent emergency surgery, developed a raised prothrombin time (INR 1.4-3.1) associated with reduction in factor VII activity (0.74 to 0.38 iu/ml) after 3-7 days of antibiotic therapy and the presence of PIVKA II by crossed-immunoelectrophoresis. Nine of these 11 patients had clinical evidence of malnutrition by anthropometric assessment and subnormal serum vitamin K1 (mean 119, range 43-354 pg/ml) levels on admission. Seven received cefotetan but 4 were treated with other non-NMTT containing antibiotics. The 9 patients who maintained normal prothrombin times and factor VII levels had normal nutritional status and normal serum vitamin K1 levels (mean 279, range 103-915 pg/ml) at presentation. A low serum vitamin K1 level was associated with a high incidence of hypopro-thrombinaemia developing following antibiotic therapy and would appear a more sensitive indicator of reduced vitamin K stores than the prothrombin time.
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Silva Júnior, Mário Luciano de Mélo, Roberta Lorena de Farias Souza, Marcos Vinícius de Souza Vilanova, and Marcos Holmes Carvalho. "Subacute combined degeneration of spinal cord vs Miller-Fisher syndrome: a diagnostic challenge." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.635.

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Context: Ataxic syndromes in the emergency department have a vast differential diagnosis, including immune, nutritional, and other etiologies. Case report: Man, 67yo, alcoholic, but previously healthy, came to ED due to complaints of falls and progressive dysesthesia in lower limbs which started 7 days from admission. He denied other toxic exposures or infectious history, except covid-19 one month before the current symptoms. Physical examination revealed mild hoarseness, left hemipalate paresis, tactile and position sense hypoesthesia, no sensory level, absence of profound reflexes; right hypotonia, global ataxia, and he was unable to walk. Neuroaxis MRI was normal. CSF obtained at D10 had 2 lymphocytes/mm3, 96mg/dL of proteins and no other abnormalities. Hb=13g/ dL, VCM=103fL, B12=424pg/mL, LDH and homocysteine were high. Negative anti-HTLV and -transglutaminase; covid-19 rapid test was positive, but rt-PCR was negative. ENMG revealed moderate sensory-motor axonal polyneuropathy. We hypothesized that the main differential diagnoses were subacute combined degeneration of spinal cord (although high level of protein in CSF) and Miller-Fisher syndrome (although ophthalmoparesis was absent). Considering that there was no motor nor respiratory symptoms, we did not offer pulse therapy and opted for B12 vitamin replacement. The patient had a favorable response to therapies and was able to walk at two weeks of treatment. Conclusions: We did not define the etiology of this case, however, it is important to keep B12 deficiency in mind (even when it is into the lab reference range) and to pay attention to post-viral atypical manifestations, mostly in the context of the current covid-19 pandemic.
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Latifah, Leny, Yusi Dwi Nurcahyani, Suryati Kumorowulan, and Diah Yunitawati. "Iodine Deficiency Associated with Adolescent Cognitive Performance in Endemic Iodine Deficiency Endemic Area." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.05.24.

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ABSTRACT Background: Iodine deficiency is one of the most important public health problems in the world today, especially among children and pregnant women who are considered to be the highest-risk groups. These diseases hinder the socio-economic development of the affected areas. This study aimed to determine iodine deficiency associated with adolescent cognitive performance in endemic iodine deficiency endemic area. Subjects and Method: This is a cross-sectional study conducted in Bulu district, Temanggung. A sample of 120 adolescents range age 11-14 years old was selected by cluster sampling. The dependent variable was Cognitive performance. The independent variable was Iodine deficiency. The data were collected by questionnaire, measurement and assessment. This study was analyzed by multiple logistic regression. Results: On cognitive performance. Adolescent IQs are below average (69.2%), mathematics tests (64.2%), and Indonesian language tests are below average (47.5%). A total of (30.8%) adolescents showed insufficient iodine intake, goiter enlargement (30.2%), stunting (35%), and anemia (20.8%). The linear regression model on IQ showed goiter enlargement as a factor associated with decreased IQ, even after accounting for confounding factors (p = 0.013; R2 = 0.07). Subjects with goiter enlargement had lower mean IQ (Mean = 10.32). IQ was related to Indonesian achievement (p = 0.031; R2 = 0.13), while UIE (p = 0.031), father’s education (p = 0.011), and IQ (p <0.001) were identified as factors related to math test scores (R2 = 0.23). after accounting for confounding factors. Conclusion: Iodine deficiency is negatively associated with cognitive performance in areas of endemic infested iodine deficiency. Goiter enlargement, which is an indicator of long-term iodine deficiency status, correlates with IQ. UIE, which reflects current indicators of iodine deficiency status, affects academic achievement. The cognitive impact of iodine deficiency lasts into adolescence. Therefore, ensuring adequate iodine intake in adolescents is essential to optimize their cognitive performance. Keywords: cognitive performance, iodine deficiency, early adolescence, nutrition, nutrition Correspondence: Leny Latifah. Research and Development Center for Health, Magelang, Central Java, Indonesia. Email: lenylatifah1@gmail.com DOI: https://doi.org/10.26911/the7thicph.05.24
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Tejaswini, H., M. M. Manohara Pai, Radhika M. Pai, Girija Attigeri, and Revathi P. Shenoy. "An ontology-based decision support system for nutrition deficiency." In 2020 IEEE International Conference on Distributed Computing, VLSI, Electrical Circuits and Robotics (DISCOVER). IEEE, 2020. http://dx.doi.org/10.1109/discover50404.2020.9278069.

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Muhammad Asraf Hairuddin, Nooritawati Md Tahir, and Shah Rizam Shah Baki. "Representation of Elaeis Guineensis nutrition deficiency based on image processing approach." In 2011 IEEE International Conference on Computer Applications and Industrial Electronics (ICCAIE). IEEE, 2011. http://dx.doi.org/10.1109/iccaie.2011.6162206.

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Reports on the topic "Nutritional Deficiency"

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Schultz-Nielsen, Marie Louise, Erdal Tekin, and Jane Greve. Labor Market Effects of Intrauterine Exposure to Nutritional Deficiency: Evidence from Administrative Data on Muslim Immigrants in Denmark. Cambridge, MA: National Bureau of Economic Research, December 2014. http://dx.doi.org/10.3386/w20723.

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Ibdah, Mwafaq, Dorothea Tholl, and Philipp W. Simon. How temperature stress changes carrot flavor: Elucidating the genetic determinants of undesired taste in carrots. United States Department of Agriculture, January 2014. http://dx.doi.org/10.32747/2014.7598171.bard.

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Global climate change and warming temperatures represent the greatest future challenge for global food production and quality. In this project, we will define the genetic factors of climate-associated taste deficiencies in carrot. Carrot is considered one of the leading horticultural crops in the world in terms of its nutritional value, health benefits, and unique flavor based on its high content of carotenoids and volatile aroma compounds. In recent years, carrot genotypes of different color with improved nutraceutical attributes have been developed. When exposed to high growth temperatures, carrots develop an undesired harsh and bitter taste caused by the accumulation of terpene metabolites. This taste deficiency represents a quality defect to carrot breeders and large- scale growers and needs to be minimized for successful marketing of carrot crops. Surprisingly, the genetic determinants of bitter and harsh flavor in carrot and their response to temperature stress are not well characterized. We started to elucidate these factors in different carrot cultivars by investigating the biosynthesis of volatile terpenes, which represent the predominant flavor compounds in carrots. Also, up to date we identified and characterized two terpenesynthase enzymes, one of which produces (E)-β- caryophyllene, a major terpene component in carrot root. Both TPSs may contribute to the observed variation in volatile terpene formation.
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Mirindi, Patrice L., Mousumi Das, Patrick N. Mirindi, and Suresh Chandra Babu. Constructing a nutrition deficiency index: Applications for the Democratic Republic of the Congo under a decade of humanitarian crises. Washington, DC: International Food Policy Research Institute, 2021. http://dx.doi.org/10.2499/p15738coll2.134251.

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Chen, Yona, Jeffrey Buyer, and Yitzhak Hadar. Microbial Activity in the Rhizosphere in Relation to the Iron Nutrition of Plants. United States Department of Agriculture, October 1993. http://dx.doi.org/10.32747/1993.7613020.bard.

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Iron is the fourth most abundant element in the soil, but since it forms insoluble hydroxides at neutral and basic pH, it often falls short of meeting the basic requirements of plants and microorganisms. Most aerobic and facultative aerobic microorganisms possess a high-affinity Fe transport system in which siderophores are excreted and the consequent Fe complex is taken up via a cognate specific receptor and a transport pathway. The role of the siderophore in Fe uptake by plants and microorganisms was the focus of this study. In this research Rhizopus arrhizus was found to produce a novel siderophore named Rhizoferrin when grown under Fe deficiency. This compound was purified and its chemical structure was elucidated. Fe-Rhizoferrin was found to alleviate Fe deficiency when applied to several plants grown in nutrient solutions. It was concluded that Fe-Rhizoferrin is the most efficient Fe source for plants when compared with other among microbial siderophores known to date and its activity equals that of the most efficient synthetic commercial iron fertilizer-Fe EDDHA. Siderophores produced by several rhizosphere organisms including Rhizopus Pseudomonas were purified. Monoclonal antibodies were produced and used to develop a method for detection of the siderophores produced by plant-growth-promoting microorganisms in barley rhizosphere. The presence of an Fe-ferrichrome uptake in fluorescent Pseudomonas spp. was demonstrated, and its structural requirements were mapped in P. putida with the help of biomimetic ferrichrome analogs. Using competition experiments, it was shown that FOB, Cop B and FC share at least one common determinant in their uptake pathway. Since FC analogs did not affect FOB or Cop-mediated 55Fe uptake, it could be concluded that these siderophores make use of a different receptor(s) than FC. Therefore, recognition of Cop, FOB and FC proceeds through different receptors having different structural requirements. On the other hand, the phytosiderophores mugineic acid (MA and DMA), were utilized indirectly via ligand exchange by P. putida. Receptors from different biological systems seem to differ in their structural requirements for siderophore recognition and uptake. The design of genus- or species-specific drugs, probes or chemicals, along with an understanding of plant-microbe and microbe-microbe relationships as well as developing methods to detect siderophores using monoclonal antibodies are useful for manipulating the composition of the rhizosphere microbial population for better plant growth, Fe-nutrition and protection from diseases.
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Poverenov, Elena, Tara McHugh, and Victor Rodov. Waste to Worth: Active antimicrobial and health-beneficial food coating from byproducts of mushroom industry. United States Department of Agriculture, January 2014. http://dx.doi.org/10.32747/2014.7600015.bard.

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Background. In this proposal we suggest developing a common solution for three seemingly unrelated acute problems: (1) improving sustainability of fast-growing mushroom industry producing worldwide millions of tons of underutilized leftovers; (2) alleviating the epidemic of vitamin D deficiency adversely affecting the public health in both countries and in other regions; (3) reducing spoilage of perishable fruit and vegetable products leading to food wastage. Based on our previous experience we propose utilizing appropriately processed mushroom byproducts as a source of two valuable bioactive materials: antimicrobial and wholesome polysaccharide chitosan and health-strengthening nutrient ergocalciferol⁽ᵛⁱᵗᵃᵐⁱⁿ ᴰ2⁾. ᴬᵈᵈⁱᵗⁱᵒⁿᵃˡ ᵇᵉⁿᵉᶠⁱᵗ ᵒᶠ ᵗʰᵉˢᵉ ᵐᵃᵗᵉʳⁱᵃˡˢ ⁱˢ ᵗʰᵉⁱʳ ᵒʳⁱᵍⁱⁿ ᶠʳᵒᵐ ⁿᵒⁿ⁻ᵃⁿⁱᵐᵃˡ ᶠᵒᵒᵈ⁻ᵍʳᵃᵈᵉ source. We proposed using chitosan and vitamin D as ingredients in active edible coatings on two model foods: highly perishable fresh-cut melon and less perishable health bars. Objectives and work program. The general aim of the project is improving storability, safety and health value of foods by developing and applying a novel active edible coating based on utilization of mushroom industry leftovers. The work plan includes the following tasks: (a) optimizing the UV-B treatment of mushroom leftover stalks to enrich them with vitamin D without compromising chitosan quality - Done; (b) developing effective extraction procedures to yield chitosan and vitamin D from the stalks - Done; (c) utilizing LbL approach to prepare fungal chitosan-based edible coatings with optimal properties - Done; (d) enrichment of the coating matrix with fungal vitamin D utilizing molecular encapsulation and nano-encapsulation approaches - Done, it was found that no encapsulation methods are needed to enrich chitosan matrix with vitamin D; (e) testing the performance of the coating for controlling spoilage of fresh cut melons - Done; (f) testing the performance of the coating for nutritional enhancement and quality preservation of heath bars - Done. Achievements. In this study numerous results were achieved. Mushroom waste, leftover stalks, was treated ʷⁱᵗʰ ᵁⱽ⁻ᴮ ˡⁱᵍʰᵗ ᵃⁿᵈ ᵗʳᵉᵃᵗᵐᵉⁿᵗ ⁱⁿᵈᵘᶜᵉˢ ᵃ ᵛᵉʳʸ ʰⁱᵍʰ ᵃᶜᶜᵘᵐᵘˡᵃᵗⁱᵒⁿ ᵒᶠ ᵛⁱᵗᵃᵐⁱⁿ ᴰ2, ᶠᵃʳ ᵉˣᶜᵉᵉᵈⁱⁿᵍ any other dietary vitamin D source. The straightforward vitamin D extraction procedure and ᵃ ˢⁱᵐᵖˡⁱᶠⁱᵉᵈ ᵃⁿᵃˡʸᵗⁱᶜᵃˡ ᵖʳᵒᵗᵒᶜᵒˡ ᶠᵒʳ ᵗⁱᵐᵉ⁻ᵉᶠᶠⁱᶜⁱᵉⁿᵗ ᵈᵉᵗᵉʳᵐⁱⁿᵃᵗⁱᵒⁿ ᵒᶠ ᵗʰᵉ ᵛⁱᵗᵃᵐⁱⁿ ᴰ2 ᶜᵒⁿᵗᵉⁿᵗ suitable for routine product quality control were developed. Concerning the fungal chitosan extraction, new freeze-thawing protocol was developed, tested on three different mushroom sources and compared to the classic protocol. The new protocol resulted in up to 2-fold increase in the obtained chitosan yield, up to 3-fold increase in its deacetylation degree, high whitening index and good antimicrobial activity. The fungal chitosan films enriched with Vitamin D were prepared and compared to the films based on animal origin chitosan demonstrating similar density, porosity and water vapor permeability. Layer-by-layer chitosan-alginate electrostatic deposition was used to coat fruit bars. The coatings helped to preserve the quality and increase the shelf-life of fruit bars, delaying degradation of ascorbic acid and antioxidant capacity loss as well as reducing bar softening. Microbiological analyses also showed a delay in yeast and fungal growth when compared with single layer coatings of fungal or animal chitosan or alginate. Edible coatings were also applied on fresh-cut melons and provided significant improvement of physiological quality (firmness, weight ˡᵒˢˢ⁾, ᵐⁱᶜʳᵒᵇⁱᵃˡ ˢᵃᶠᵉᵗʸ ⁽ᵇᵃᶜᵗᵉʳⁱᵃ, ᵐᵒˡᵈ, ʸᵉᵃˢᵗ⁾, ⁿᵒʳᵐᵃˡ ʳᵉˢᵖⁱʳᵃᵗⁱᵒⁿ ᵖʳᵒᶜᵉˢˢ ⁽Cᴼ2, ᴼ²⁾ ᵃⁿᵈ ᵈⁱᵈ not cause off-flavor (EtOH). It was also found that the performance of edible coating from fungal stalk leftovers does not concede to the chitosan coatings sourced from animal or good quality mushrooms. Implications. The proposal helped attaining triple benefit: valorization of mushroom industry byproducts; improving public health by fortification of food products with vitamin D from natural non-animal source; and reducing food wastage by using shelf- life-extending antimicrobial edible coatings. New observations with scientific impact were found. The program resulted in 5 research papers. Several effective and straightforward procedures that can be adopted by mushroom growers and food industries were developed. BARD Report - Project 4784
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Eshel, Amram, Jonathan P. Lynch, and Kathleen M. Brown. Physiological Regulation of Root System Architecture: The Role of Ethylene and Phosphorus. United States Department of Agriculture, December 2001. http://dx.doi.org/10.32747/2001.7585195.bard.

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Specific Objectives and Related Results: 1) Determine the effect of phosphorus availability on ethylene production by roots. Test the hypothesis that phosphorus availability regulates ethylene production Clear differences were found between the two plants that were studied. In beans ethylene production is affected by P nutrition, tissue type, and stage of development. There are genotypic differences in the rate of ethylene production by various root types and in the differential in ethylene production when P treatments are compared. The acceleration in ethylene production with P deficiency increases with time. These findings support the hypothesis that ethylene production may be enhanced by phosphorus deficiency, and that the degree of enhancement varies with genotype. In tomatoes the low-P level did not enhance significantly ethylene production by the roots. Wildtype cultivars and ethylene insensitive mutants behaved similarly in that respect. 2) Characterize the effects of phosphorus availability and ethylene on the architecture of whole root systems. Test the hypothesis that both ethylene and low phosphorus availability modify root architecture. In common bean, the basal roots give rise to a major fraction of the whole root system. Unlike other laterals these roots respond to gravitropic stimulation. Their growth angle determines the proportion of the root length in the shallow layers of the soil. A correlation between ethylene production and basal root angle was found in shallow rooted but not deep-rooted genotypes, indicating that acceleration of ethylene synthesis may account for the change in basal root angle in genotypes demonstrating a plastic response to P availability. Short-time gravitropic response of the tap roots of young bean seedlings was not affected by P level in the nutrient solution. Low phosphorus specifically increases root hair length and root hair density in Arabidopsis. We tested 7 different mutants in ethylene perception and response and in each case, the response to low P was lower than that of the wild-type. The extent of reduction in P response varied among the mutants, but every mutant retained some responsiveness to changes in P concentration. The increase in root hair density was due to the increase in the number of trichoblast cell files under low P and was not mediated by ethylene. Low P did not increase the number of root hairs forming from atrichoblasts. This is in contrast to ethylene treatment, which increased the number of root hairs partly by causing root hairs to form on atrichoblasts. 3) Assess the adaptive value of root architectural plasticity in response to phosphorus availability. A simulation study indicated that genetic variation for root architecture in common bean may be related to adaptation to diverse competitive environments. The fractal dimension of tomato root system was directly correlated with P level.
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