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1

Fantus, I. George. Insulin resistance and cancer: Epidemiology, cellular and molecular mechanisms, and clinical implications. Springer, 2011.

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2

Brown, Kristy A., and Evan R. Simpson. Obesity and Breast Cancer. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4899-8002-1.

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3

Kaufmann, M. Therapeutic management of metastatic breast cancer. Walter de Gruyter & Co, 1989.

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4

McGahan, Lynda. Surveillance mammography after treatment for primary breast cancer. CCOHTA, 2000.

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5

Swanton, Charles, and Stephen R. D. Johnston. Handbook of metastatic breast cancer. 2nd ed. Informa Healthcare, 2011.

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6

Petrén-Mallmin, Marianne. Clinical and experimental imaging of breast cancer metastases in the spine. Munksgaard, 1994.

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7

1952-, Dickson Robert B., and Lippman Marc E. 1945-, eds. Mammary tumor cell cycle, differentiation, and metastasis: Advances in cellular and molecular biology of breast cancer. Kluwer Academic Publishers, 1996.

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8

1922-, Dao Thomas L., Brodie Angela, Ip Clement, and Organ Systems Program (National Cancer Institute). Breast Cancer Working Group., eds. Tumor markers and their significance in the management of breast cancer: Proceedings of a workshop held in Bethesda, Maryland, March 6, 1985. Liss, 1986.

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9

Lawrence, Paszat, and Canadian Coordinating Office for Health Technology Assessment., eds. A population-based cohort study of surveillance mammography after treatment of primary breast cancer. Canadian Coordinating Office for Health Technology Assessment, 2001.

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10

Julia, Gee, Nicholson Robert I, and SpringerLink (Online service), eds. Therapeutic Resistance to Anti-Hormonal Drugs in Breast Cancer: New Molecular Aspects and their Potential as Targets. Springer Netherlands, 2009.

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11

Obesity And Breast Cancer The Role Of Dysregulated Estrogen Metabolism. Springer-Verlag New York Inc., 2013.

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12

Fantus, I. George. Insulin Resistance and Cancer: Epidemiology, Cellular and Molecular Mechanisms and Clinical Implications. Springer, 2011.

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13

Fantus, I. George. Insulin Resistance and Cancer: Epidemiology, Cellular and Molecular Mechanisms and Clinical Implications. Springer, 2013.

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14

Henderson, I. Craig. Breast Cancer. Oxford University Press, Incorporated, 2015.

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15

Yu, Dihua, and Mien-Chie Hung. Breast Cancer Chemosensitivity. Springer, 2010.

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16

Yu, Dihua, and Mien-Chie Hung. Breast Cancer Chemosensitivity. Springer London, Limited, 2009.

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17

Johnston, Stephen, and Charles Swanton. Handbook of Metastatic Breast Cancer. Informa Healthcare, 2006.

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18

Swanton, Charles, and Stephen R. D. Johnston. Handbook of Metastatic Breast Cancer. Taylor & Francis Group, 2019.

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19

Swanton, Charles, and Stephen R. D. Johnston. Handbook of Metastatic Breast Cancer. Taylor & Francis Group, 2011.

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20

Swanton, Charles, and Stephen Johnston. Handbook of Metastatic Breast Cancer. Taylor & Francis Group, 2006.

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21

Swanton, Charles, and Stephen R. D. Johnston. Handbook of Metastatic Breast Cancer. Taylor & Francis Group, 2011.

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22

Breast Cancer Metastasis And Drug Resistance Progress And Prospects. Springer, 2012.

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23

Minimal Residual Disease And Circulating Tumor Cells In Breast Cancer. Springer, 2012.

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24

Pantel, Klaus, Michail Ignatiadis, and Christos Sotiriou. Minimal Residual Disease and Circulating Tumor Cells in Breast Cancer. Springer London, Limited, 2012.

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25

Brown, Kristy A., and Evan R. Simpson. Obesity and Breast Cancer: The Role of Dysregulated Estrogen Metabolism. Springer, 2013.

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26

Diagnostic Pathology: Breast. Saunders Company, W. B., 2016.

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27

Lester, Susan C., and David G. Hicks. Diagnostic Pathology: Breast. Elsevier, 2021.

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28

Rygiel, Katarzyna. Obesity as a Common Denominator in Breast Cancer and Cardiovascular Disease. Cambridge Scholars Publishing, 2019.

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29

Lippman, Marc E., and Robert B. Dickson. Mammary Tumor Cell Cycle, Differentiation, and Metastasis: Advances in Cellular and Molecular Biology of Breast Cancer. Springer, 2012.

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30

Mammary Tumorigenesis and Malignant Progression: Advances in Cellular and Molecular Biology of Breast Cancer (Cancer Treatment and Research). Springer, 1994.

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31

Lippman, Marc E., and Robert B. Dickson. Mammary Tumorigenesis and Malignant Progression: Advances in Cellular and Molecular Biology of Breast Cancer. Springer, 2012.

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32

Hiscox, Stephen, Julia Gee, and Robert I. Nicholson. Therapeutic Resistance to Anti-hormonal Drugs in Breast Cancer: New Molecular Aspects and their Potential as Targets. Hiscox Stephen Gee Julia Nicholson Robert I, 2010.

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33

Diagnostic Pathology: Intraoperative Consultation. Elsevier - Health Sciences Division, 2018.

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34

Harrison, Beth T., and Susan C. Lester. Diagnostic Pathology: Intraoperative Consultation. Elsevier, 2023.

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35

Keum, NaNa, Mingyang Song, Edward L. Giovannucci, and A. Heather Eliassen. Obesity and Body Composition. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0020.

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In 2014, an estimated 1.9 billion adults worldwide were either overweight (BMI 25–29.9) or obese (BMI ≥30). The so-called obesity epidemic began in high-income, English-speaking countries in the early 1970s, but soon spread globally; more than one-third (38%) of all adults and 600,000 children under age five are overweight or obese, as are two-thirds (69%) of adults in the United States. Excessive body fat is a major cause of type 2 diabetes, hypertension, cardiovascular and liver disease, among other disorders, and has been designated a definite cause of at least fourteen cancer sites: breast (postmenopausal), colorectum, endometrium, esophagus (adenocarcinoma), gallbladder, kidney (renal cell), pancreas, gastric cardia, liver, ovary, prostate (advanced tumors), multiple myeloma, thyroid, and meningioma. Mechanisms by which adipose tissue are thought to promote tumor growth include the endocrine and metabolic effects of fat on sex hormones, growth factors, and inflammation, as well as local chemical or mechanical injury of gastrointestinal organs.
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36

Wot, Osha. Plant-Based Diet : Plant-Based Diet with Delicious Recipes,Organic Diet, Vegetarian Diet, Plant Cookbook: Asthma Benefits,Reduce Risk of Breast Cancer, Kidney Stones, Obesity Control. Independently Published, 2019.

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37

Thun, Michael J., Martha S. Linet, James R. Cerhan, Christopher Haiman, and David Schottenfeld. Primary Prevention of Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0062.

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Primary prevention has enormous potential to reduce the human, social, and economic costs of cancer worldwide. The following sections discuss the development and application of preventive interventions in six broad areas of public health: tobacco control, the prevention of obesity and physical inactivity, prevention of infection-related cancers, protection against excessive exposure to ultraviolet light, preventive drug therapies (chemoprevention), and the regulation of carcinogenic exposures. All of these areas affect multiple types of cancer and massive numbers of people. Different interventions are at varying stages of development. For example, effective, evidence-based approaches have been developed over several decades to reduce tobacco use, prevent chronic infection with hepatitis B virus, protect children from excessive sun exposure, regulate exposures in high-income countries, and reduce breast cancer incidence and recurrence in high-risk women. More recent efforts are seeking to identify upstream measures to prevent excessive weight gain, reduce caloric intake, and increase physical activity.
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38

Grant, Warren, and Martin Scott-Brown. Prevention of cancer. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0350.

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In the UK, the four commonest cancers—lung cancer, breast cancer, colon cancer, and prostate cancer—result in around 62 000 deaths every year. Although deaths from cancer have fallen in the UK over the last 20 years, the UK still suffers from higher cancer death rates than many other countries in Western Europe. In 1999, the UK government produced a White Paper called Saving Lives: Our Healthier Nation that outlined a national target to reduce the death rate from cancer by at least 20% in people under 75 by 2010. The subsequent NHS Cancer Plan of 2000 designed a framework by which to achieve this target through effective prevention, screening, and treatment programmes as well as restructuring and developing new diagnostic and treatment facilities. But do we know enough about the biology of the development of cancer for government health policies alone to force dramatic changes in survival? The science behind the causes of cancer tells us that its origin lies in acquired or inherited genetic abnormalities. Inherited gene mutation syndromes and exposure to environmental mutagens cause cancer, largely through abnormalities in DNA repair mechanisms, leading to uncontrolled cell proliferation. Although screening those thought to be at highest risk, and regulating exposure to environmental carcinogens such as tobacco or ionizing radiation, have reduced, and will continue to reduce, cancer deaths, there are many other environmental factors that have been shown to increase the population risk of cancer. These will be outlined in this chapter. However, the available evidence is largely from retrospective and cross-sectional population-based studies and therefore limits the ability to apply this knowledge to the risk of the individual patient who may been seen in clinic. Although we may be able to put him or her into a high-, intermediate-, or low-risk category, the question ‘will I get cancer, doc?’ is one that we cannot answer with certainty. The NHS Cancer Plan of 2000, designed to reduce cancer deaths in this country and to bring UK treatment results in line with those other countries in Europe, focuses on preventing malignancy as part of its comprehensive cancer management strategy. It highlights that the rich are less likely to develop cancer, and will survive longer if they are diagnosed than those who live in poverty. This may reflect available treatment options, but is more likely to be related to the lifestyle of those with regular work, as they may be more health aware. The Cancer Plan, however, suggests that relieving poverty may be more labour intensive and less rewarding than encouraging positive risk-reducing behaviour in all members of the population. Eating well can reduce the risk of developing many cancers, particularly of the stomach and bowel. The Cancer Plan outlines the ‘Five-a-Day’ programme which was rolled out in 2002 and encouraged people to eat at least five portions of fruit and vegetables per day. Obese people are also at higher risk of cancers, in particular endometrial cancer. A good diet and regular exercise not only reduce obesity but are also independent risk-reducing factors. Alcohol misuse is thought to be a major risk factor in around 3% of all cancers, with the highest risk for cancers of the mouth and throat. As part of the Cancer Plan, the Department of Health promotes physical activity and general health programmes, as well as alcohol and smoking programmes, particularly in deprived areas. Focusing on these healthy lifestyle points can potentially reduce an individual lifetime risk of all cancers. However, our knowledge of the biology of four cancers in particular has led to the development of specific life-saving interventions. Outlined in this chapter are details regarding ongoing prevention strategies for carcinomas of the lung, the breast, the bowel, and the cervix.
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39

Omega Six The Devils Fat Why Excess Omega 6 And Lack Of Omega 3 Promotes Chd Aggression Depression Adhd Weight Gain Obesity Poor Sleep Pcos Breast Cancer Infertility Arthritis And Western Illnesses. Les Creux Limited, 2008.

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40

Salud y educación: Estudios sobre realidades plurales con perspectiva de género. Universidad de Colima, 2016.

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41

Collins, Catherine Fisher, ed. African American Women’s Life Issues Today. ABC-CLIO, LLC, 2013. http://dx.doi.org/10.5040/9798400607929.

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After decades of research devoted to women's health, a federal agency focused on women's health, and millions of dollars allocated to address women's health disparities, African American women are still the sickest American citizens. This book examines why. Written by an all-female, all-African American team of health experts that include nurse practitioners, registered nurses, educators, and psychologists, this book focuses on the diseases and related social issues that cause the greatest harm and pose the greatest threat to African American women today. Its chapters address topics as varied as heart disease, cancer, sexually transmitted diseases, domestic violence, cervical and breast cancers, obesity, depression, mental illness, dementia/Alzheimer's, and incarcerated women's health care. A chapter is dedicated to identifying the social, cultural, and environmental barriers that block African American women from experiencing the best possible lives. Providing comprehensive coverage of the topic from an Afrocentric perspective, this text will be of great interest to medical and psychological health professionals and professors; social workers, counselors, and students in these fields; as well as African American women seeking current and expert information on these health threats.
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42

Kelly, Evelyn B., ed. Encyclopedia of Human Genetics and Disease. ABC-CLIO, LLC, 2013. http://dx.doi.org/10.5040/9798400667251.

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This two-volume encyclopedia examines the history, characteristics, causes, and treatment of genetic disease, as well as the science of genetics itself. Modern science has unlocked many of the mysteries of genetics, providing a blueprint for understanding the origins behind previously mysterious ailments and conditions, both common and uncommon. A complete understanding remains elusive, however: geneticists are still refining theories about what causes chromosomes to mutate, and genetic diseases remain difficult to diagnose and challenging to treat. This fascinating reference explores the scientific and human aspects of this complex field of science. Encyclopedia of Human Genetics and Diseasefeatures nearly 400 entries, including well-known genetic diseases, rare and lesser-known genetic diseases, and the genetic factors that may contribute to common diseases and health conditions, such as breast cancer and obesity. The author presents in-depth discussions of concepts essential to understanding genetic disease in 18 entries that provide background on key topics, such as "Genetics 101," the genome and the foundations of genetics, genetic counseling, and newborn screening. Each of the 355 disorders profiled provides the history of the condition, its prevalence, causes, treatment (if any), and further reading. Interesting sidebars and compelling photos that help inform content accompany many entries.
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