Relevant bibliographies by topics / Ochlar manifestations of general diseases

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Ochlar manifestations of general diseases'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "Ochlar manifestations of general diseases"

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Macovei, Luana Andreea, V. Cristescu, Mihaela Debita, and C. A. Dinu. "Oral Manifestations of Osteoarticular Diseases." Revista de Chimie 68, no. 10 (November 15, 2017): 2440–42. http://dx.doi.org/10.37358/rc.17.10.5901.

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Human body acts as a whole and this leads to an increased occurrence of oral manifestations during the onset and development of systemic diseases. Therefore, oral pathological manifestations play an important role in the diagnosis, prognosis and treatment of diseases with osteoarticular involvement. General pathology has a strong impact on oral health which in turn is influenced by systemic factors. Oral manifestations can be used for making an early diagnosis of a serious condition that can be cured with appropriate therapy. Oral lesions can be explained by pathological processes and correlated with systemic diseases. When oral symptoms and signs are present, the disease is already in an advanced stage, due to the fact that the oral cavity is usually affected by a generalized disease. The mouth is part of the digestive system, but it has various symptoms caused by diseases located in other regions. 98 patients with oral manifestations of diseases with osteoarticular involvement were followed up in a private dental clinic from Iasi over a period of 3 years between June 2013 and July 2016. 47 patients were male (47.95%) and 51 were female (52.05%), aged between 18 and 65 years old, with a peak incidence between the ages of 45 and 60. The complex structure of the oral cavity was thoroughly explored (lips, lip mucosa, the corners of the mouth, gingival labial sulcus, oral vestibule, the internal face of the cheeks, the outer surface of gums, the floor and the ceiling of the oral cavity or the hard and the soft palate). The health status of the body is reflected by the health of the oral mucosa, which can be seen as a marker of various systemic diseases.
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Ash, M. M., and C. M. Phillips. "Parasitic Diseases With Cutaneous Manifestations." North Carolina Medical Journal 77, no. 5 (September 1, 2016): 350–54. http://dx.doi.org/10.18043/ncm.77.5.350.

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Robson, Kristi J., and Warren W. Piette. "CUTANEOUS MANIFESTATIONS OF SYSTEMIC DISEASES." Medical Clinics of North America 82, no. 6 (November 1998): 1359–79. http://dx.doi.org/10.1016/s0025-7125(05)70419-3.

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Rao, Devika. "Pulmonary Manifestations of Pediatric Diseases." JAMA 304, no. 10 (September 8, 2010): 1127. http://dx.doi.org/10.1001/jama.2010.1306.

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Koulaouzidis;, A., S. Bhat;, and J. Moschos. "Skin manifestations of liver diseases." Annals of Hepatology 6, no. 3 (July 2007): 181–84. http://dx.doi.org/10.1016/s1665-2681(19)31926-x.

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Baldassano, Vincent F. "Ocular manifestations of rheumatic diseases." Current Opinion in Ophthalmology 9, no. 6 (December 1998): 85–88. http://dx.doi.org/10.1097/00055735-199812000-00015.

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Hostein, J., and J. Fournet. "Gastrointestinal Manifestations of Collagen Diseases." Digestive Diseases 4, no. 4 (1986): 240–52. http://dx.doi.org/10.1159/000171154.

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Gran, Jan Tore, and Gunnar Husby. "Joint Manifestations in Gastrointestinal Diseases." Digestive Diseases 10, no. 5 (1992): 274–94. http://dx.doi.org/10.1159/000171365.

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Gran, Jan Tore, and Gunnar Husby. "Joint Manifestations in Gastrointestinal Diseases." Digestive Diseases 10, no. 5 (1992): 295–312. http://dx.doi.org/10.1159/000171366.

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Brieva, Joaquin C. "Mucocutaneous Manifestations of Viral Diseases." Archives of Dermatology 139, no. 5 (May 1, 2003): 678. http://dx.doi.org/10.1001/archderm.139.5.678-b.

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Dissertations / Theses on the topic "Ochlar manifestations of general diseases"

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Yiu, Kai-hang, and 姚啟恆. "Cardiovascular manifestations in systemic inflammatory diseases." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B48541011.

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Systemic inflammatory diseases, including those of rheumatology and dermatology, are associated with increased cardiovascular events. Evidence demonstrates that the chronic systemic inflammation associated with these diseases plays a pivotal role in all stages of atherosclerotic plaque formation, from initiation of the fatty streak to plaque rupture and consequent acute coronary syndrome. Although a number of studies have evaluated the cardiovascular manifestations in systemic inflammatory disease, this thesis offers additional observations, including the vascular atherosclerotic pattern, the pathogenesis of premature atherosclerosis and the use of coronary calcification as a predictor of adverse cardiovascular outcome. The work is divided into four sections. Section I provides an overview of the cardiovascular manifestation of systemic inflammatory diseases and the patients and methods of the current thesis. The objective of Section II is to evaluate the pattern of cardiovascular manifestation, in particular systemic vascular calcification and cardiac valve calcification in patients with rheumatoid arthritis (RA), systemic lupus erythematosus (SLE) and psoriasis, using multi-detector computed tomography (MDCT) and carotid intima-media thickness (c-IMT). It was found that both patients with RA, SLE and psoriasis had a greater prevalence and extent of vascular calcification compared with age and gender matched controls. Moreover, both aortic valve calcification (AVC), mitral valve calcification (MVC) was found to be more prevalent in patients with RA and SLE than controls. Interestingly, the presence of MVC, but not AVC, independently predicted the occurrence of premature atherosclerosis with arterial calcification in these patients. Section III evaluates the potential underlying mechanisms that lead to cardiovascular manifestations in patients with systemic inflammatory disease. Bone marrow-derived endothelial progenitor cells (EPCs) play an important role in the maintenance of endothelial integrity and hemostasis. The relationship between the circulating EPCs and subclinical coronary atherosclerosis as determined by coronary calcification in RA patients nonetheless remains unclear. The study results demonstrated that RA patients with coronary atherosclerosis have significantly lower levels of CD133/KDR+ and CD133+ EPCs than those without. In addition to older age, lower levels of circulating CD133/KDR+ EPCs also predicted occurrence of coronary atherosclerosis. As with RA and SLE, psoriasis is associated with premature atherosclerosis, although the underlying mechanism remains unclear. The aim of the study was therefore to investigate the relationship of disease activity and systemic inflammation with macrovascular and microvascular function in patients with psoriasis. The results demonstrated that patients with psoriasis have increased arterial stiffness, but not microvascular dysfunction compared with healthy controls. More importantly, high-sensitivity C reactive protein positively correlated with, and independently predicted, arterial stiffness. Section IV explores the prognostic value of a surrogate marker of atherosclerosis, coronary calcium score (CCS), in patients with RA and SLE. A total of 152 patients with RA (n=85) and SLE (n=69), and 106 healthy controls underwent MDCT to measure CCS. All patients were prospectively followed up for major cardiovascular events for a mean period of 4.3 years. The result demonstrated that presence of CCS >100 predicted the occurrence of a major cardiovascular event independent of other risk factors in RA and SLE patients.
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Medicine
Master
Doctor of Medicine
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Whalan, Comus John. "Mechanisms of post-operative sepsis and renal impairment in obstructive jaundice /." Title page, abstract, contents and introduction only, 1998. http://web4.library.adelaide.edu.au/theses/09MD/09mdw552.pdf.

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Sanchez, Phyllis Nancy. "Psychiatric diagnosis vs medical diagnosis: Are mental health professionals aware?" Diss., The University of Arizona, 1989. http://hdl.handle.net/10150/184826.

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For years research has demonstrated a varying incidence of medical disorders manifesting with psychiatric symptoms. A relatively conservative estimate of such so called "medical masquerades" is around 10%. It is important to ascertain whether health care professionals are aware of possible medical masquerades perhaps most especially in a mental health center outpatient setting where non-medically trained clinicians are the first line therapists for treatment in the majority of cases. This study set about to find out how aware three types of health care clinicians (psychiatrists, nonpsychiatrically trained medical doctors, and non-medically trained mental health psychotherapists) are of the prevalence of medical masquerades, and whether these three types of clinicians perform differently on three types of clinical vignettes (psychiatric, somatoform, and medical masquerades). Results revealed that all health care professionals surveyed are aware that there are a percentage of medical masquerades in the clinical population. Results also revealed that the three types of clinicians performed differently on the case vignettes.
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Tsang, Chiu-shun Peter, and 曾昭舜. "Oral biology of human immunodeficiency virus-infected individuals in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31237770.

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Tala, Hazel Marie Bituin Linan. "The oral health status and dental awareness of young Hong Kong Chinese with insulin dependent diabetes mellitus (IDDM)." Click to view the E-thesis via HKUTO, 2000. http://sunzi.lib.hku.hk/hkuto/record/B31954145.

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La, Cock Charles J. R. "Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia." Thesis, Cape Technikon, 1996. http://hdl.handle.net/20.500.11838/2251.

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Thesis (MTech (Medical Technology))--Cape Technikon, 1996.
Haematological defects observed in clonal deletions of mtDNA and the inhibition of mitochondrial function by benzene and chloramphenicol, suggest a role for mtDNA in the pathogenesis of radiation - induced preleukaemia (MDS). The fact that leukaemia cells contain abnormal mitochondria and abnormally structured mtDNA, makes it reasonable to assume mtDNA mutations could be central to the pathogenesis of both MDS and leukaemia. It was decided to examine MDS patients for the presence of mtDNA length mutations (dimers and cocantameres). Such topological forms have already been reported in the literature in association with human leukaemia. These steric considerations suggest that mtDNA dimers are probably non-functional due to supercoiling. Thus, it was felt that a progressive accumulation of non-functional dimers in the haematopoietic compartment could account for many of the clinical features associated with MDS. Transmission electron microscopy was used to examine haematopoietic mtDNA in the bone marrow of six patients with MDS. Abnormal mtDNA dimer formation was found in all instances. The proportional number of these dimers were found to roughly correlate with the Myeloid/ Erythroid cell ratio in the bone marrow, and it appeared likely that the dimers were generated in the myeloid compartment during early MDS.
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Saunders, Lucy C. (Lucy Chloe). "Psychological and endocrine indicators of stress : health and management implications." Thesis, Stellenbosch : Stellenbosch University, 2003. http://hdl.handle.net/10019.1/53563.

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Thesis (MSc)--University of Stellenbosch, 2003.
ENGLISH ABSTRACT: Introduction: This is a multidisciplinary investigation of stress in working males. The physiological aspect of the stress response is focused primarily on the hypothalamic-pituitary-adrenal axis; the health outcomes focus on cardiovascular risk and the psychological aspect focuses on both personality and state. Aims: To assess psychological and physiological measures of stress and determine if they are associated. To assess the effect of massage as a relaxation intervention on these measures. Abstract: Section 1 This study consisted of 16 working, stressed males. Certain measures of allostatic load were determined, on 2 occasions 1 week apart with appropriate control (serum cortisol, dehydroepiandrosterone-sulphate (DHEAs), total cholesterol, low-density lipoprotein (LDL), high-density lipoprotein) as well as perceived stress levels (State Anxiety, Profile of Mood (POMS), Job Satisfaction (JS), Symptoms of Stress (SYMP». Measurements of personality (Trait Anxiety and Hardiness, with subs cores Commitment, Control and Challenge) and anthropometric measurements were obtained. Repeatability was assessed and then data was averaged. Correlation analysis was done between baseline physiological and psychological parameters. (All r values reported in this abstract have P<0.05, unless otherwise stated). Section 2 11 of the 16 subjects then had 1 week of intensive massage (5 x Ihr each), after which a 3rd measurement was taken. Findings: Section 1 Certain personalities had differing perceptions of stress (Commitment correlated positively with Vigour, r= 0.59; and negatively with Fatigue and POMS, r= -0.51 and r= -0.54 respectively). Certain personalities influenced JS (Commitment correlated positively to JS, r= 0.55; Trait Anxiety correlated negatively to JS, r= -0.54). Certain personalities manifested stress physiologically (Control correlated negatively with CortisollDHEAs, r= -0.53). Increased perception of stress also resulted in an increased likelihood of experiencing physiological symptoms of stress (e.g. State Anxiety correlated with SYMP, r= 0.50). In contrast, lower perceived stress was associated with increased job satisfaction (POMS correlated with JS, r= -0.64; PAFRIKAANSE OPSOMMING: Inleiding: Hierdie studie is 'n multi-dissiplinêre ondersoek na stres in werkende mans. Die fisiologiese aspek van die stresrespons fokus primêr op die hipotalamo-pituitêre-adrenale as, terwyl die gesondsheidsuitkomste op die kardiovaskulêre risiko, en die psigologiese aspek op beide persoonlikheid en status fokus. Doel: Om psigiese en fisiologiese parameters van stress te bepaal, en om vas te stelof hulle met mekaar korrelleer. Om die effek van massering, as ontspanningsterapie, op hierdie parameters te bepaal. Afdeling 1 Sestien gespanne, werkende mans het aan hierdie studie deelgeneem. Sekere parameters van allostatiese lading (serum kortisol, dehidroepiandrosteroon-sulfaat (DHEAs), totale cholesterol, lae digtheid- (LDL) en hoë digtheid lipoproteïene (HDL) is twee maal bepaal, met 'n tussenpose van een week, met gepaste kontrole. Terselfdertyd is subjektiewe ervaring van stres (Toestand Angs, Profiel van Gemoedstemming (POMS), Werksbevrediging (JS), Simptome van Stres (SYMP», metings van persoonlikheid (Trekangs en Hardiness, i.e. Gebondenheid, Beheer en Uitdaging) en antropometriese metings ook verkry. Herhaalbaarheid is getoets en daarna is die gemiddelde van hierdie twee basislyn bepalings gebruik. Korrelasie analises is gedoen tussen basislyn fisiologiese en psigiese parameters. (Alle r-waardes gerapporteer in hierdie samevatting het P< 0.05, behalwe waar anders aangedui.) Afdeling 2 Elf van die sestien proefpersone het 1 week van intensiewe masseerterapie (5 x 1 uur elk) ondergaan, waarna 'n derde meting geneem is. Resultate: Afdeling 1 Sekere persoonlikhede het verskillende ervarings van stres getoon (Toegewydheid het positief gekorreleer met lewenslus, r= 0.59; en negatief met uitputting en POMS, r= -0.51 en r= -0.54 onderskeidelik). Sekere persoonlikhede het werksbevrediging (JS) beïnvloed (Toegewydheid het positief gekorreleer met JS, r = 0.55; Trait anxiety het negatief gekorreleer met JS, r= -0.54). Sekere persoonlikhede het ook fisiologiese geneigdheid tot stres getoon (Beheer het negatief gekorreleer met CIDHEAs, r= -0.53). Verhoogde ervaring van stres het 'n verhoogde waarskynlikheid vir ervaring van fisiologiese simptome van stres tot gevolg gehad (bv. Toestand Angs het positief gekorreleer met SYMP, r= 0.50). In teenstelling, is laer ervaring van stres geassosieer met verhoogde werksbevrediging (POMS het positief gekorreleer met JS, r= -0.64; P
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8

Tsang, Chiu-shun Peter. "Oral biology of human immunodeficiency virus-infected individuals in Hong Kong /." [Hong Kong : Faculty of Dentistry, University of Hong Kong], 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19900661.

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Presnell, Scott. "Whatever the individual says it is : a phenomenological analysis of chronic pain in people with Human Immunodeficiency Virus-associated distal symmetrical polyneuropathy /." [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18066.pdf.

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La, Cock Charles JR. "Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia." Thesis, Cape Technikon, 1996. http://hdl.handle.net/20.500.11838/1493.

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Thesis (MTech(Medical Technology))--Cape Technikon, Cape Town, 1996
Haematol.ogical defects observed in cl.onal deletions of mtDNA and the inhibition of mitochondrial function by benzene and chloramphenicol., suggest a role for mtDNA in the pathogenesis of radiation - induced preleukaemia (MDS). The fact that leukaemia cell.s contain abnormal mitochondria and abnormally structured mtDNA, makes it reasonable to assume mtDNA mutations could be central to the pathogenesis of both MDS and l.eukaemia. It was decided to examine MDS patients for the presence of mtDNA length mutations (dimers and cocantameres). Such topol.ogical forms have already been reported in the literature in association with human leukaemia. These steric considerations suggest that mtDNA dimers are probably non-functional due to supercoil.ing. Thus, it was felt that a progressive accumulation of non-functional dimers in the haematopoietic compartment could account for many of the cl.inical. features associated with MDS. Transmission electron microscopy was used to examine haematopoietic mtDNA in the bone marrow of six patients with MDS. Abnormal mtDNA dimer formation was found in al.l instances. The proportional. number of these dimers were found to roughly correlate with the Myeloid/ Erythroid cell. ratio in the bone marrow, and it appeared likely that the dimers were generated in the myeloid compartment during early MDS. Control.l.ed radiation studies were performed on 20 wistar rats in an attempt to elucidate the approximate time when abnormal mtDNA dimer formation occurred, fol.l.owing fractionated gamma or gamma-neutron irradiation. Gamma-irradiated rats demonstrated abnormal mtDNA dimer formation at the time hypoplastic marrow recovery was first observed.
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Books on the topic "Ochlar manifestations of general diseases"

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Jackson, C. R. S. The eye in general practice. 8th ed. Edinburgh: Churchill Livingstone, 1985.

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D, Finlay R., ed. The eye in general practice. 9th ed. Edinburgh: Churchill Livingstone, 1991.

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Walker, Sara, and Luis J. Jara. Endocrine manifestations of systemic autoimmune diseases. Amsterdam: Elsevier Science, 2008.

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Cutaneous manifestations of systemic disease. London: Gower Medical, 1986.

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Scully, Crispian. An atlas of stomatology: Oral diseases and manifestations of systemic diseases. London: M. Dunitz, 1989.

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Jonathan, Shepherd, ed. Slide interpretation in oral diseases and the oral manifestations of systemic diseases. Oxford: Oxford University Press, 1986.

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S, Anhalt Todd, ed. Handbook of skin clues of systemic diseases. Philadelphia: Lea & Febiger, 1987.

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Jacobs, Paul H. Handbook of skin clues of systemic diseases. 2nd ed. Philadelphia: Lea & Febiger, 1992.

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Chan, Heng Leong. Cutaneous clues to systemic diseases, with illustrations in color. Singapore: Singapore University Press, 1986.

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Yitzchak, Frank, ed. Neurological manifestations of systemic diseases in children. New York: Raven Press, 1993.

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Book chapters on the topic "Ochlar manifestations of general diseases"

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Seppänen, Mikko, and Nima Rezaei. "General Considerations." In Pulmonary Manifestations of Primary Immunodeficiency Diseases, 1–36. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-00880-2_1.

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Marks, Rory M. "Systemic Autoimmune Diseases." In The Saint-Chopra Guide to Inpatient Medicine, edited by Sanjay Saint and Vineet Chopra, 425–42. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190862800.003.0075.

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J. Cantres-Fonseca, Onix. "Thoracic Extrapulmonary Tuberculosis in the Millennial Era." In Update in Respiratory Diseases. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.90376.

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Mycobacterium tuberculosis is one of the most pathogenic infectious organisms, usually known for causing cavitary lung infection. But this mycobacterium is also capable of causing masked involvement in any organ of the body. Its clinical manifestation can mimic other conditions according to the organ affected. Extrapulmonary infection is defined as any manifestation caused by tuberculosis in tissues outside the airway or the pulmonary parenchyma. Despite it being a well-known infectious organism throughout decades, tuberculosis continues to be causing great morbidity and mortality in this millennium. This chapter will discuss the clinical manifestations of extrapulmonary tuberculosis (EPTB), when the mycobacteria invade extrapulmonary tissues inside the thorax. We discuss and review the literature about the clinical manifestations, diagnosis and evaluation, and general treatment.
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Weng, Patricia L., and Katherine Wesseling Perry. "Nephrology and Urology." In General Pediatrics Board Review, 419–62. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190848712.003.0020.

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This chapter on pediatric nephrology and urology examines normal renal physiology and urological anatomy along with common manifestations of renal and urological dysfunction in children. It reviews the clinical presentation, diagnosis, and management of fluid, electrolyte, and acid-base disorders. It describes the presentation and management of glomerular diseases associated with proteinuria, hematuria, and chronic kidney disease. Inherited diseases and syndromes affecting the kidney, bladder, and urinary tract are described. In addition, age-related changes in glomerular filtration, common pathogens associated with urinary tract infection, the diagnosis and management of inguinal masses and vaginal discharge, and the management of acute kidney injury are discussed. This chapter is written for pediatric residents in training.
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Longridge, Nicholas, Pete Clarke, Raheel Aftab, and Tariq Ali. "General Medicine." In Oxford Assess and Progress: Clinical Dentistry, edited by Katharine Boursicot and David Sales. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198825173.003.0020.

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The oral cavity is the largest and most used orifice in the human body. It is the opening of the aerodigestive tract, as well as a region of the body that is heavily involved in both sensing the outside world and communi­cating with it. Simply put, our mouths are complex and deeply intimate structures that can act as windows into the health of the rest of the body. Many disease processes that are systemically invisible may display quite overt oral manifestations. There are deep associations between bodily diseases and oral dis­ease counterparts, with considerable and mounting evidence to show that oral health may have an impact on systemic wellness. It is there­fore important to have an understanding of the wider human anatomy, physiology, and pathology. Understand and treat the patient as a whole, and think about all aspects of their health, whether it be routine pre­ventative treatment for periodontally compromised diabetic patients or polypharmacy patients requiring secondary dental care. Often at times, patients can be unclear about their own health condi­tion; having a fundamental understanding of general medicine will help to make those difficult choices regarding your patients a little easier and clarify when and whom to refer. Key topics are not included for this chapter, as it is a vast topic, and not the main focus of the dental undergraduate curriculum. It is, however, important to have a good basic knowledge of general human diseases, how they might interact with dental treatment, and the role that dentists can play in both diagnosis and management
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Hysing, Mari, and Astri J. Lundervold. "General Effects of Pediatric Diseases on Cognition and Behavior." In Cognitive and Behavioral Abnormalities of Pediatric Diseases. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195342680.003.0006.

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In this chapter, pediatric diseases are defined as “illnesses that affect a person for an extended period of time, often for life, and that require medical care attention above and beyond the normal requirements for a child or adolescent” (American Academy of Pediatrics 1993). The prevalence of such chronic illnesses has steadily increased over the past 20–30 years (Downs et al. 2001; Vincer et al. 2006; Marelli et al. 2007), with estimated rates ranging from below 1% to 44% depending on the definition, method, and sample included in the different studies (van der Lee et al. 2007). Several theoretical frameworks and models have been presented to explain how diagnosis-specific effects and generic factors across disorders affect cognition and behavior. The transactional stress and coping model (TSC) is one of the most cited theories on psychological adaption in children with a chronic illness, encompassing both specific and generic effects (Thompson and Gustafson 1996). Dennis (2000) has presented a similar model of factors affecting cognitive outcome in chronically ill children. The models describe how disease-related variables interact with child characteristics, developmental level, family resources, and peer relationships, and provide a useful framework to the present chapter. Several epidemiological and clinical studies have documented that children with pediatric diseases have an increased risk of emotional and behavioral problems (Lavigne and Faier-Routman 1992; Glazebrook et al. 2003; Hysing et al. 2007). Symptoms of internalizing and externalizing disorders are frequently found across the diseases but illness-specific manifestations also exist. Externalizing symptoms of hyperactivity and social problems are most often shown by children with neurological disorders (Rodenburg et al. 2005), while the rate of emotional problems has been reported to be especially high in children with asthma (Vila et al. 2003). Peer interaction and social functions are often affected in children with pediatric disorders. Investigations of the impact of social functioning is a growing research field in pediatric psychology, as part of a general trend in developmental psychology that focuses on close peer relationship as a moderator of nonoptimal functioning (Bukowski and Adams 2005; Burt et al. 2008).
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Fisher, Simon, and Serena Ledwidge. "Surgery in tropical diseases." In Oxford Assess and Progress: Clinical Surgery. Oxford University Press, 2014. http://dx.doi.org/10.1093/oso/9780199696420.003.0026.

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Whether you exercise a desire to experience surgery abroad during your student elective or whether you want to take the opportunity to enhance your career aspirations in far-flung regions, knowledge of common surgi­cal presentations of tropical disease is a prerequisite. Moreover, with the ability conferred by modern jet travel to translocate to almost any area of the globe within 48h, you may well encounter these conditions in your local general hospital clinic or emergency department, in visitors to the country or in travellers returning home from foreign excursions. This chapter tests you on your knowledge of presentations, investi­gations, and management of diseases such as malaria, schistosomiasis, amoebiasis, and filariasis, conditions native to a variety of geographical locations. Some diseases such as hydatid still remain endemic in regions closer to the UK. A sound understanding of parasitic life cycles, modes of exposure to, and manifestations of infection, and principles of treatment, will help you to diagnose and manage these conditions whether they are encountered unexpectedly, or prevalent in your geographical location.
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Sterling, Timothy R., and Richard E. Chaisson. "General Clinical Manifestations of Human Immunodeficiency Virus Infection (Including Acute Retroviral Syndrome and Oral, Cutaneous, Renal, Ocular, Metabolic, and Cardiac Diseases)." In Mandell, Douglas, and Bennett's Principles and Practice of Infectious Diseases, 1541–57. Elsevier, 2015. http://dx.doi.org/10.1016/b978-1-4557-4801-3.00124-7.

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STERLING, TIMOTHY R., and RICHARD E. CHAISSON. "General Clinical Manifestations of Human Immunodeficiency Virus Infection (Including the Acute Retroviral Syndrome and Oral, Cutaneous, Renal, Ocular, Metabolic, and Cardiac Diseases)." In Mandell, Douglas, and Bennett's Principles and Practice of Infectious Diseases, 1705–25. Elsevier, 2010. http://dx.doi.org/10.1016/b978-0-443-06839-3.00121-1.

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Cox, Timothy M., and Richard W. E. Watts. "The inborn errors of metabolism: General aspects." In Oxford Textbook of Medicine, edited by Timothy M. Cox, 1929–41. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0225.

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The inborn errors of metabolism are those inherited diseases in which the phenotype includes a characteristic constellation of biochemical abnormalities related to an alteration in the catalytic activity of a single specific enzyme, activator, or transport protein. Mechanism of diseases—mutations in the proteins giving rise to the inborn errors of metabolism affect primary, secondary, tertiary, or quaternary structure. This can lead to an enormous variety of consequences. Clinical presentation—the manifestations of metabolic disease are protean and may seem nondescript, especially in adults, hence a high level of suspicion may be required to make a correct diagnosis. Prevention and screening—there is a strong case for mass population screening for some inborn errors of metabolism at the presymptomatic stage to allow early detection and introduction of proven treatment before irreversible damage occurs. Management—definitive cure of the underlying abnormality is available for a few disorders, but precise characterization of the biochemical disturbance often permits rational treatment to be organized and provides the basis for further therapeutic endeavours. General approaches include (1) restriction of a substrate that cannot be metabolized including molecules derived from the diet; (2) replacement of a missing metabolic product; (3) removal of poisonous metabolites or rebalancing overproduction of toxic intermediates; (4) administering pharmacological doses of a cofactor, sometimes a vitamin, that may also stabilize a mutant enzyme; (5) replacement of a missing gene product, usually by enzymatic augmentation therapy or pharmacological chaperones, to prevent premature aggregation and denaturation; (6) repression of an overproduced protein or metabolite by stable RNA inhibition; (7) transplantation of cells or organs as a ‘gene replacement therapy’; and (8) activation of a poorly functioning protein.
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Conference papers on the topic "Ochlar manifestations of general diseases"

1

Reitblat, TI, E. Dorodnikov, I. Polishchuk, D. Zamir, and E. Kahana. "FRI0177 The spectrum of clinical manifestations of antiphospholipid antibody syndrome in the general hospital." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.246.

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