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Journal articles on the topic 'Ocular hypertonia'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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1

Tenouri, Soukaina, Hind Saidi, Ahmed Bennis, Fouad Chraibi, Meriem Abdellaoui, and Idriss Benatiya Andaloussi. "Impact of Hypotonizing Treatment in Patients with Glaucoma on the Ocular Surface: About 52 Patients." Scholars Journal of Medical Case Reports 9, no. 12 (2021): 1119–23. http://dx.doi.org/10.36347/sjmcr.2021.v09i12.001.

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Ocular surface disease (OSD) and glaucoma are common comorbidities in the same patient. Thus, the long-term instillation of topical intraocular pressure lowering drops induces changes in the ocular surface. For this, we have realized a cross-sectional study of 52 patients, collected at the ophthalmology department of the Hassan II University Hospital Center at Fez, Morocco. We have included all patients with glaucoma or ocular hypertonia on hypotonizing treatment for at least 06 months. They have answered the Ocular Surface Disease Index (OSDI) after their consent. All patients have been then

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2

Meriem, Elakhdari, Bardi Chaimaa, Akannour Younes, Serghini Louai, and Berraho Amina. "Urrets-Zavalia Syndrome after Penetrating Keratoplasty: A Case Report." Ophthalmology Research: An International Journal 18, no. 3 (2023): 27–29. http://dx.doi.org/10.9734/or/2023/v18i3388.

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Urrets-Zavalia syndrome is a dreaded complication of anterior segment surgery and the most common in patients undergoing corneal transplantation. Avoiding the prescription of mydriatic eye drops during the postoperative period is the mainstay of its therapy, which focuses on prevention. One month after having keratoplasty in the left eye, a 42-year-old man presented with Urrets-Zavalia syndrome and ocular hypertonia at 35 mmgh. Medical and surgical management were required in this situation. The purpose of our presentation is to describe a real-world instance of Urrets-Zavalia syndrome.

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3

Talbi, C., H. Taouti, S. Belghmaidi, I. Hajji, and A. Moutaouakil. "The Prevalence of Ocular Surface Disease in Glaucoma Patients." SAS Journal of Surgery 9, no. 02 (2023): 99–102. http://dx.doi.org/10.36347/sasjs.2023.v09i02.008.

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Introduction: Glaucoma is the third cause of blindness in developed countries after age-related macular degeneration (AMD) and diabetic retinopathy. The aim of this study was to determine the prevalence of ocular surface disease in patients treated for glaucoma or ocular hypertonia. Patients and methods: We conducted a one-year observational study including glaucoma patients older than 15 years, diagnosed and treated for more than six months. The impact of anti-glaucoma treatment on the ocular surface was assessed by a questionnaire derived from the Ocular Surface Disease Index (OSDI). Results

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4

Nouvellon, Emmanuel, Joel L’Hermite, Arnaud Chaumeron, et al. "Ophthalmic Regional Anesthesia." Anesthesiology 100, no. 2 (2004): 370–74. http://dx.doi.org/10.1097/00000542-200402000-00028.

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Background The purpose of this study was to evaluate the efficacy and safety of episcleral single-injection anesthesia in a large number of patients. Methods Over a period of 5 yr, in four institutions, anesthesiologists involved in this prospective study completed a standardized form to evaluate single-injection medial canthus high-volume episcleral anesthesia. The success rate of the block was rated according to an akinesia score. The study parameters included demographic data, surgical procedure, and anesthetic management. All patients were followed up at least until postoperative day 1, an

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5

John, Prescilla D., Erica F. Weiss, Diana Bronshteyn, David M. Masur, John J. McGinley, and Ronda F. Facchini. "A-81 Neuropsychological Profile of Child with Spinocerebellar Ataxia, Type 29." Archives of Clinical Neuropsychology 36, no. 6 (2021): 1126. http://dx.doi.org/10.1093/arclin/acab062.99.

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Abstract Objective Spinocerebellar ataxia 29 (SCA29) is a rare autosomal dominant disorder resulting in infantile-onset hypertonia, gross motor delay and cognitive impairment. SCA29 causes cerebellar dysfunction and atrophy manifested by poor hand coordination, instability of gait, impaired articulation, poor hand-eye coordination, language delay, and a wide legged, unsteady walk. Slow improvement across childhood is common, but the limited available research suggests significant clinical heterogeneity among individuals diagnosed with SCA29. We report the case of a six-year-old first grade gir

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Cap, Caitlyn J., Madeline Manning, and Kayleen Ball. "A-266 Neuropsychological Profile of a 5-Year-Old Boy with Gould Syndrome." Archives of Clinical Neuropsychology 37, no. 6 (2022): 1416. http://dx.doi.org/10.1093/arclin/acac060.266.

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Abstract Objective: Gould syndrome (COL4A1/A2-related disorders) is an extremely rare, genetic disorder characterized by cerebral vasculature defects, ocular dysgenesis, myopathy, and cerebral cortical abnormalities. Many individuals with COL4A1 mutations exhibit seizures, stroke, visual field defects, and hydrocephalus. To date, research regarding neuropsychological sequelae of Gould syndrome has been sparse. Therefore, this presentation aims to present clinical considerations for pediatric patients with Gould syndrome. Method: Patient is a 5-year-old Hispanic boy born at 35-weeks following p

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7

Jahan, Sultana. "Can Low Dose Sertraline Cause Serotonin Syndrome in Pediatric Patients? 2 Case Reports." CNS Spectrums 26, no. 2 (2021): 155. http://dx.doi.org/10.1017/s1092852920002473.

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AbstractBackgroundSerotonin syndrome is a potentially life-threatening condition associated with increased serotonergic activity in the central nervous system. Serotonin syndrome is underreported complication of pharmacotherapy. The Hunter Criteria for serotonin syndrome (SS) are fulfilled if the patient has taken a serotonergic agent and has one of the following symptoms: 1) spontaneous clonus, 2) inducible clonus and agitation or diaphoresis, 3) ocular clonus and agitation or diaphoresis, 4) tremor and hyperreflexia, 5) hypertonia, or 6) temperature above 38 C and ocular clonus or inducible

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8

Bresson-Dumont, H. "Hypertonie oculaire simple." EMC - Ophtalmologie 38, no. 4 (2021): 1–8. https://doi.org/10.1016/s0246-0343(21)42932-1.

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9

BARDI, Chaimaa, Ihsane HASNAOUI, Salma HASSINA, et al. "When Phacomorphic Glaucoma Becomes Phacolytic: A Case Report." International Journal of Science, Applications and Prosperity 1, no. 1 (2023): 2–5. https://doi.org/10.5281/zenodo.10439641.

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10

Motta, M., C. M. Pereira, C. Azevedo, M. Dias, L. Fernandes, and A. Alves. "183 Hypertonie oculaire après rétinopexie." Journal Français d'Ophtalmologie 30 (April 2007): 2S203. http://dx.doi.org/10.1016/s0181-5512(07)79995-3.

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11

Lacharme, T., J. P. Romanet, and S. Halimi. "Diabète, hypertonie oculaire et glaucome." Médecine des Maladies Métaboliques 3, no. 2 (2009): 165–68. http://dx.doi.org/10.1016/s1957-2557(09)71630-x.

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12

Kossi, Dzidzinyo, Djagnikpo Akouete, Ayena Koffi Didier, et al. "Resultats A Court Terme De La Trabeculoplastie Selective Au Laser Chez Les Patients Togolais." European Scientific Journal, ESJ 13, no. 30 (2017): 271. http://dx.doi.org/10.19044/esj.2017.v13n30p271.

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Aim: To check the short-term tonometric results of SLT in the treatment of primary glaucoma at the open angle and in charge of ocular hypertonias in Togolese people. Methods: A retrospective study was carried out in an ophthalmology center. The first 130 eyes of 72 patients benefited from the SLT laser procedure. The tonometric controls work object focus on follow-up at 1, 3, and 6 months post laser treatment. Results: 130 eyes of 72 patients were collected. The average age of the patients was 49.74 years (± 17.45) and the ages vary between 10 and 85 years. The average IOP of the laser before

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13

Serra, A., I. Zucca, A. Tanda, V. Piras, and M. Fossarello. "Blue-yellow perimetry in patients with ocular hypertone." Acta Ophthalmologica Scandinavica 76, S227 (2009): 24–27. http://dx.doi.org/10.1111/j.1600-0420.1998.tb00870.x.

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14

Zucca, I., M. Nessi, M. Fossarello, and A. Serra. "Evaluation of frequency doubling perimetry in ocular hypertone." Acta Ophthalmologica Scandinavica 78, S232 (2000): 69–70. http://dx.doi.org/10.1111/j.1600-0420.2000.tb01114.x.

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15

Nasri, H., M. Cheour, K. Eleuch, A. Kasri, and A. Kraiem. "545 Hypertonie oculaire post-kératoplastie transfixiante." Journal Français d'Ophtalmologie 30 (April 2007): 2S306. http://dx.doi.org/10.1016/s0181-5512(07)80358-5.

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16

Girerd, X. "Hypertension artérielle-hypertonie oculaire, les parallèles." Journal Français d'Ophtalmologie 30, no. 5 (2007): 9–13. http://dx.doi.org/10.1016/s0181-5512(07)89644-6.

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17

Nordmann, J. P. "Hypertension artérielle-hypertonie oculaire, les parallèles." Journal Français d'Ophtalmologie 30, no. 5 (2007): 14–17. http://dx.doi.org/10.1016/s0181-5512(07)89645-8.

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18

Renard, J. P. "Hypertension artérielle-hypertonie oculaire, les parallèles." Journal Français d'Ophtalmologie 30, no. 5 (2007): 18–21. http://dx.doi.org/10.1016/s0181-5512(07)89646-x.

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19

Fayol, N., O. Abitbol, S. Doan, M. H. Duong, T. Hoang-Xuan, and D. Gatinel. "167 Étude de l’hystérésis cornéenne par Ocular Response Analyzer chez les sujets normaux, glaucomateux, et ayant une hypertonie oculaire." Journal Français d'Ophtalmologie 30 (April 2007): 2S199. http://dx.doi.org/10.1016/s0181-5512(07)79979-5.

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20

Ni, Yang, Baisheng Xu, Lan Wu, et al. "Assessment of Full-Eye Response to Osmotic Stress in Mouse Model In Vivo Using Optical Coherence Tomography." Journal of Ophthalmology 2015 (2015): 1–8. http://dx.doi.org/10.1155/2015/568509.

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NaCl based solutions were applied as osmotic stress agents to alter the hydration state of the mouse eye. Full-eye responses to these osmotic challenges were monitored in vivo using a custom-built optical coherence tomography (OCT) with an extended imaging range of 12.38 mm. Dynamic changes in the mouse eye were quantified based on the OCT images using several parameters, including the central corneal thickness (CCT), the anterior chamber depth (ACD), the crystalline lens thickness (LT), the cornea-retina distance (CRD), the iris curvature (IC), and the lens scattering intensity (LSI). Apparen

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21

Crochelet, O., B. Valero, J. M. Giraud, et al. "525 Une hypertonie oculaire maligne peu ordinaire." Journal Français d'Ophtalmologie 28 (March 2005): 295. http://dx.doi.org/10.1016/s0181-5512(05)73645-7.

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22

Nizamova, Shakhnoza Zariboy Kizi. "CORRELATIONS BETWEEN CLINICAL, NEUROPHYSIOLOGICAL AND HEMODYNAMIC PARAMETERS IN PATIENTS WITH FIRST OPEN OVERALL GLAUCOMA IN PATIENTS WITH HYPERTONIC DEAD." CURRENT APPROACHES AND NEW RESEARCH IN MODERN SCIENCES 2, no. 3 (2023): 95–97. https://doi.org/10.5281/zenodo.7793688.

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Normal tension glaucoma is a form of multifactorial optical neuropathy, the etiology of which is still actively debated. To date, no single factor can fully explain its pathogenesis. The main factors contributing to glaucomatous optical neuropathy (ON) associated with GNN include vascular factors and ocular blood flow, translaminar pressure gradient as well as immune and genetic factors. These factors can ocular blood flow cause axonal damage in several ways.

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23

Meyer, Benjamin I., Naomi E. Gutkind, Marissa K. Shoji, and Andrew J. Rong. "Bilateral Thermal Keratopathy Due to Plasma Skin Regeneration." Ophthalmic Plastic & Reconstructive Surgery 40, no. 3 (2024): e89-e91. http://dx.doi.org/10.1097/iop.0000000000002618.

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A 40-year-old woman underwent periocular plasma skin regeneration, a cosmetic treatment for periorbital rejuvenation. She subsequently developed bilateral thermal keratitis, manifesting as blurred vision, irritation, and redness, with a vision decrease to 20/60 and 20/50 in her OD and OS, respectively. Examination demonstrated bilateral large, irregular corneal epithelial defects and edema, necessitating treatment with amniotic membrane grafts, bandage contact lenses, and hypertonic saline. One year posttreatment, her visual acuity improved to 20/20 and 20/25, albeit with ongoing symptomatic d

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24

Tchabi-Hounnou, S., L. Agbahoungba, I. Sounouvou, et al. "Hypertonie oculaire et glaucome primitif à angle ouvert." Journal Français d'Ophtalmologie 35, no. 7 (2012): 523–28. http://dx.doi.org/10.1016/j.jfo.2011.10.008.

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25

Farukhi Ahmed, Sarah, Audrey Xi Tai, Mason Schmutz, John Combs, and Sameh Mosaed. "Acute Onset Ocular Hypotony after Coronary Artery Bypass Surgery." Case Reports in Ophthalmology 8, no. 1 (2017): 200–207. http://dx.doi.org/10.1159/000468143.

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Importance: The purpose of this case report is to evaluate risk factors associated with post-coronary artery bypass graft (CABG) ocular hypotony compared to post-CABG ischemic optic neuropathy. Observations: The patient described here is a single case at the University of California, Irvine Medical Center, from July 2016. This case demonstrates the rare incidence of acute post-CABG ocular hypotony and vision loss in a patient with prior history of optic atrophy. Both vision loss and hypotony resolved completely to baseline without intervention within 3 days postoperatively. Conclusions and Rel

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26

Ben Amar-Ketata, A., Z. Ben Zina, S. Abid, et al. "468 Syndrome de Vogt-Koyanagi-Harada et hypertonie oculaire." Journal Français d'Ophtalmologie 30 (April 2007): 2S286. http://dx.doi.org/10.1016/s0181-5512(07)80281-6.

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27

Masrour, H., S. Bouchnafati, R. Hanini, et al. "Hypertonie oculaire et uvéite : à propos de 52 cas." La Revue de Médecine Interne 44 (June 2023): A181—A182. http://dx.doi.org/10.1016/j.revmed.2023.04.103.

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28

Zaman, Khadiza, Vien Nguyen, Katalin Prokai-Tatrai, and Laszlo Prokai. "Proteomics-Based Identification of Retinal Protein Networks Impacted by Elevated Intraocular Pressure in the Hypertonic Saline Injection Model of Experimental Glaucoma." International Journal of Molecular Sciences 24, no. 16 (2023): 12592. http://dx.doi.org/10.3390/ijms241612592.

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Elevated intraocular pressure is considered a major cause of glaucomatous retinal neurodegeneration. To facilitate a better understanding of the underlying molecular processes and mechanisms, we report a study focusing on alterations of the retina proteome by induced ocular hypertension in a rat model of the disease. Glaucomatous processes were modeled through sclerosing the aqueous outflow routes of the eyes by hypertonic saline injections into an episcleral vein. Mass spectrometry-based quantitative retina proteomics using a label-free shotgun methodology identified over 200 proteins signifi

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29

Faure, C., G. Caputo, J. A. Sahel, and M. Paques. "Hypertonie oculaire aiguë tardive compliquant une rétinopathie de la prématurité." Journal Français d'Ophtalmologie 31, no. 5 (2008): 535.e1–535.e3. http://dx.doi.org/10.1016/s0181-5512(08)72474-4.

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30

Ashman, A., J. H. Norris, and K. Chaidas. "Hypertonie oculaire par pneumorbite secondaire à un minime traumatisme fermé." Annales françaises d'Oto-rhino-laryngologie et de Pathologie Cervico-faciale 137, no. 3 (2020): 205–7. http://dx.doi.org/10.1016/j.aforl.2019.06.001.

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31

Pralon, N., E. Privat, C. Arndt, S. Navarre, and M. Villain. "486 Hypertonie oculaire compliquant une endarteriectomie chez une patiente avec un syndrome d’ischémie oculaire bilatéral." Journal Français d'Ophtalmologie 31 (April 2008): 156. http://dx.doi.org/10.1016/s0181-5512(08)71084-2.

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32

Inborr, Pia Katriina, Mika Harju, and Tero T. Kivelä. "Intravenous Hypertonic Saline to Lower Intraocular Pressure in Ocular Hypertension and Primary Open-angle and Exfoliation Glaucoma." Journal of Glaucoma 27, no. 7 (2018): 638–42. http://dx.doi.org/10.1097/ijg.0000000000000979.

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33

Villain, M., J. P. Nordmann, J. P. Renard, et al. "270 Enquête Française Glaucome et Hypertonie Oculaire 1 Jour (EFGH1J). Nouvelles données." Journal Français d'Ophtalmologie 28 (March 2005): 226. http://dx.doi.org/10.1016/s0181-5512(05)74668-4.

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34

Hnin, Hay Marn, Einar Stefánsson, Thorsteinn Loftsson, Rathapon Asasutjarit, Dusadee Charnvanich та Phatsawee Jansook. "Physicochemical and Stability Evaluation of Topical Niosomal Encapsulating Fosinopril/γ-Cyclodextrin Complex for Ocular Delivery". Pharmaceutics 14, № 6 (2022): 1147. http://dx.doi.org/10.3390/pharmaceutics14061147.

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This study aimed to develop a chemically stable niosomal eye drop containing fosinopril (FOS) for lowering intraocular pressure. The effects of cyclodextrin (CD), surfactant types and membrane stabilizer/charged inducers on physiochemical and chemical properties of niosome were evaluated. The pH value, average particle size, size distribution and zeta potentials were within the acceptable range. All niosomal formulations were shown to be slightly hypertonic with low viscosity. Span® 60/dicetyl phosphate niosomes in the presence and absence of γCD were selected as the optimum formulations accor

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35

Villain, M., J. P. Renard, J. F. Rouland, N. Sandid, and E. Leutenegger. "225 Glaucome, hypertonie oculaire et facteurs de risque vasculaire. À propos d’un cas." Journal Français d'Ophtalmologie 31 (April 2008): 80. http://dx.doi.org/10.1016/s0181-5512(08)70822-2.

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36

Denion, E., F. Metge, and G. Caputo. "546 Rétinopathie de décompression après hypertonie oculaire traitée médicalement : présentation de 2 cas." Journal Français d'Ophtalmologie 32 (April 2009): 1S166. http://dx.doi.org/10.1016/s0181-5512(09)73670-8.

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37

Dot, C., H. El Chehab, A. Russo, and E. Agard. "Hypertonie oculaire après injections intra-vitréennes de corticoïdes, actualisation des connaissances en 2015." Journal Français d'Ophtalmologie 38, no. 7 (2015): 656–64. http://dx.doi.org/10.1016/j.jfo.2015.03.002.

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38

Delcourt, C., A. Bron, C. Baudouin, et al. "Prévalence et description du traitement par hypotonisants pour glaucome et hypertonie oculaire en France." Journal Français d'Ophtalmologie 29, no. 10 (2006): 1098–106. http://dx.doi.org/10.1016/s0181-5512(06)73905-5.

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39

Courjaret, J. C., D. Denis, M. C. Rojat-Habib, F. Barlési, and F. Matonti. "Hypertonie oculaire secondaire à une métastase de l’angle irido-cornéen d’un carcinome bronchique anaplasique." Journal Français d'Ophtalmologie 37, no. 4 (2014): e61-e63. http://dx.doi.org/10.1016/j.jfo.2013.09.008.

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40

Lajmi, H., and M. A. El Afrit. "Prise en charge thérapeutique d’une hypertonie oculaire post-kératoplastie transfixiante : à propos de 73 cas." Journal Français d'Ophtalmologie 37, no. 3 (2014): 220–25. http://dx.doi.org/10.1016/j.jfo.2013.06.006.

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41

Fowler, Nicholas, Romulo Albuquerque, Jooyoung Cho, Nicholas Bell, Paras Vora, and Greg Botzet. "3494 Naltrexone as a Diagnostic Tool in Ocular Neuropathic Pain." Journal of Clinical and Translational Science 3, s1 (2019): 16–17. http://dx.doi.org/10.1017/cts.2019.41.

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OBJECTIVES/SPECIFIC AIMS: The study aims to track and correlate ocular neuropathic symptoms, corneal sensitivity and dry-eye like pain, after scleral buckle and posterior vitrectomy surgeries. The goal is to identify a population of patients that receive these retinal surgeries that experience ocular neuropathic pain. METHODS/STUDY POPULATION: Methods - Prospective and Retrospective cohort studies were designed with the follow cohorts: scleral buckle, posterior vitrectomy, and control. Typical follow up for SB/PV surgeries are: 1 day, 1 week, 1, 3, 6, 12 months post surgery. CS and DELP metric

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42

Lachkar, Y. "Calculateur de risque de développer un glaucome à partir d’une hypertonie oculaire : attention au risque de confusion." Journal Français d'Ophtalmologie 29, no. 4 (2006): 441–42. http://dx.doi.org/10.1016/s0181-5512(06)77709-9.

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43

Van Went, C., H. Alalwani, E. Brasnu, et al. "Évaluation de la sensibilité cornéenne chez les patients traités médicalement pour un glaucome ou une hypertonie oculaire." Journal Français d'Ophtalmologie 34, no. 10 (2011): 684–90. http://dx.doi.org/10.1016/j.jfo.2011.07.011.

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44

Miray, L. G., R. Rafanomezantsoa, A. L. Rachid, T. F. Andriamampionona, and L. Raobela. "Epidemiological and Clinical Aspects of Primary Open Angle Glaucoma in Fianarantsoa Madagascar." Advances in Social Sciences Research Journal 11, no. 1 (2024): 258–66. http://dx.doi.org/10.14738/assrj.111.16321.

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Introduction : Primary open-angle glaucoma (POAG) is a chronic, progressive, bilateral optic neuropathy corresponding to a loss of retinal ganglion cells and characterised by morphological changes in the optic disc associated with typical visual field damage. Methods : This was a prospective, cross-sectional, descriptive, monocentric study conducted over a 5-month period from September 2020 to February 2021 in the ophthalmology department of the university hospital of Tambohobe Fianarantsoa Madagascar. The purpose of this study was to evaluate the prevalence of primary open-angle glaucoma and

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45

Housni, K., I. Hanafi, C. Baqadir, et al. "THYROID-ASSOCIATED ORBITOPATHY : RETROSPECTIVE STUDY OF 6 CASES." International Journal of Advanced Research 9, no. 06 (2021): 268–73. http://dx.doi.org/10.21474/ijar01/13009.

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Introduction : Lorbitopathie dysthyroidienne est une pathologie inflammatoire de lorbite dorigine dysimmunitaire, survenant le plus souvent dans un contexte dhyperthyroidie, rarement dhypothyroidie, et constitue la premiere cause dexophtalmie chez ladulte. Materiel et methode : Nous avons mene une etude retrospective sur 6 patients presentant une ophtalmopathie dysthyroidienne moderee et severe, admis au service dophtalmologie de lHopital 20 Aout 1953 de Casablanca sur une periode setalant entre 2017 et 2019. Resultats : Lage moyen des patients etait de 45 ans, avec un sexe ratio H/F de 2.Deux

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46

Lanzi, Cecilia, Laura Lucarini, Mariaconcetta Durante, et al. "Role of Histamine H3 Receptor Antagonists on Intraocular Pressure Reduction in Rabbit Models of Transient Ocular Hypertension and Glaucoma." International Journal of Molecular Sciences 20, no. 4 (2019): 981. http://dx.doi.org/10.3390/ijms20040981.

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Intraocular pressure (IOP) has a tendency to fluctuate throughout the day, reaching its peak in the early morning in healthy subjects or glaucoma patients. Likewise, histamine tone also fluctuates over time, being lower at nighttime. Numerous studies have demonstrated a correlation between short-term IOP fluctuation and glaucoma progression; however, it has not yet been determined whether histamine plays a role in IOP fluctuations. The aim of this research was to establish the distribution of the histamine receptor proteins and respective mRNAs in the eye by western blot, immunohistochemistry

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47

Bron, A., C. Baudouin, P. Denis, et al. "Satisfaction et observance des patients présentant une hypertonie oculaire ou un glaucome à angle ouvert après instauration d’une bithérapie fixe." Journal Français d'Ophtalmologie 31, no. 7 (2008): 659–65. http://dx.doi.org/10.1016/s0181-5512(08)74379-1.

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48

Laplace, O. "Ophtalmologie - La pression intra-oculaire cible, nouveau paramètre de prise en charge des hypertonies oculaires et des glaucomes." La Presse Médicale 35, no. 7-8 (2006): 1199. http://dx.doi.org/10.1016/s0755-4982(06)74782-0.

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Chen, Jingyao, Weijia Zhang, Yixin Zheng, and Yanze Xu. "Ameliorative Potential of Resveratrol in Dry Eye Disease by Restoring Mitochondrial Function." Evidence-Based Complementary and Alternative Medicine 2022 (May 26, 2022): 1–10. http://dx.doi.org/10.1155/2022/1013444.

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Abstract:

Background and Significance. Dry eye disease (DED) is a prevalent optic surface illness with a high incidence worldwide that is caused by a variety of factors, including mitochondrial dysfunction. Resveratrol has been confirmed to protect the eye surface in DED, and as an antioxidant, resveratrol can maintain mitochondrial function. Therefore, we investigated whether resveratrol can improve DED by restoring mitochondrial function. Methods. The mitochondrial dysfunction of HCE-2 human corneal epithelial cells was induced by high osmotic pressure exposure and treated with resveratrol (50 μM). We

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Ivanovska Adjievska, Biljana, and Violeta Buckoska. "CORNEAL EDEMA AFTER CATARACT SURGERY - CHANGES IN CORNEAL ENDOTHELIUM CELL CHARACTERISTICS." MEDIS – International Journal of Medical Sciences and Research 2, no. 2 (2023): 37–40. http://dx.doi.org/10.35120/medisij020237i.

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Abstract:

 Corneal edema is a common complication of cataract surgery, although tremendous improvements have been made in the surgical techniques, which decreased surgical eye trauma and complication rates. Normal endothelial cell density is 2000-3000 cells/mm2 in older individuals, which maintains the corneal clarity. Even ‘perfect’ cataract surgery does some damage to the endothelium. A significant postoperative endothelium density decrease can impair its ability to maintain corneal clarity, resulting in corneal edema, blurring of vision and ocular pain. Aggressive topical treatment in the first

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