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Relevant bibliographies by topics / Ocular Pathologies: Retinopathies (Maculodystrophy

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Journal articles

Academic literature on the topic 'Ocular Pathologies: Retinopathies (Maculodystrophy'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Ocular Pathologies: Retinopathies (Maculodystrophy"

1

Rivera, José Carlos, Rabah Dabouz, Baraa Noueihed, Samy Omri, Houda Tahiri, and Sylvain Chemtob. "Ischemic Retinopathies: Oxidative Stress and Inflammation." Oxidative Medicine and Cellular Longevity 2017 (2017): 1–16. http://dx.doi.org/10.1155/2017/3940241.

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Ischemic retinopathies (IRs), such as retinopathy of prematurity (ROP), diabetic retinopathy (DR), and (in many cases) age-related macular degeneration (AMD), are ocular disorders characterized by an initial phase of microvascular changes that results in ischemia, followed by a second phase of abnormal neovascularization that may culminate into retinal detachment and blindness. IRs are complex retinal conditions in which several factors play a key role during the development of the different pathological stages of the disease. Increasing evidence reveals that oxidative stress and inflammatory

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2

Trinquet, Laure, Suzon Ajasse, Frédéric Chavane, et al. "Uncovering the Characteristics of Pupil Cycle Time (PCT) in Neuropathies and Retinopathies." Vision 9, no. 3 (2025): 51. https://doi.org/10.3390/vision9030051.

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Pupil cycle time (PCT) estimates the dynamics of a biofeedback loop established between pupil size and stimulus luminance, size or colour. The PCT is useful for probing the functional integrity of the retinopupillary circuits, and is therefore potentially applicable for assessing the effects of damage due to retinopathies or neuropathies. In previous studies, PCT was measured by manually counting the number of pupil oscillations during a fixed period to calculate the PCT. This method is scarce, requires a good expertise and cannot be used to estimate several PCT parameters, such as the oscilla

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3

Dayhaw-Barker, Pierrette. "Retinal Pigment Epithelium Melanin and Ocular Toxicity." International Journal of Toxicology 21, no. 6 (2002): 451–54. http://dx.doi.org/10.1080/10915810290169873.

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Ocular morphology is specifically directed at facilitating the transmittance of visible light to the retina for the purposes of photoreceptor absorption and phototransduction, thereby initiating the process of vision. By absorbing excess radiation, melanin significantly enhances this process. It can also act as a photoprotector by quenching reactive oxygen species and other radicals produced as a result of the high oxygen dependency of the retina for its metabolism. However, melanin also binds numerous pharmaceuticals, a process that can result in ocular toxicity. Although our understanding of

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4

Santonocito, Manuela, Cristina Zappulla, Santa Viola, et al. "Assessment of a New Nanostructured Microemulsion System for Ocular Delivery of Sorafenib to Posterior Segment of the Eye." International Journal of Molecular Sciences 22, no. 9 (2021): 4404. http://dx.doi.org/10.3390/ijms22094404.

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Eye drop formulations allowing topical treatment of retinal pathologies have long been sought as alternatives to intravitreal administration. This study aimed to assess whether a novel nanostructured microemulsions system (NaMESys) could be usefully employed to deliver sorafenib to the retina following topical instillation. NaMESys carrying 0.3% sorafenib (NaMESys-SOR) proved to be cytocompatible in vitro on rabbit corneal cells, and well-tolerated following b.i.d. ocular administration to rabbits during a 3-month study. In rats subject to retinal ischemia-reperfusion, NaMESys-SOR significantl

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Babizhayev, MA. "Drug Design of Mitochondria-Targeted Antioxidants, Action, Metabolism and Perspectives for Ophthalmic Therapeutics: N-acetylcarnosine Codrug Treatment Platform." April 22, 2017. https://doi.org/10.19070/2332-290X-1700062.

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Maintaining the redox balance within the mitochondria is critical for cellular homeostasis in the eye since the mitochondria host the energy producing systems of the cell and it is widely recognized that damage to the mitochondria plays a key role in sight threatening age-related eye disorders, including retinopathies (maculodystrophy, retinitis pigmentosa , hereditary optic neuropathy), as well as glaucoma, cataract, and autoimmune uveitis. Reactive oxygen species (ROS) are generated as by-products of cellular metabolism, primarily in the mitochondria. Although ROS are essential participants

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Patel, Riya, Smriti Nair, Hassaam Choudhry, Mustafa Jaffry, and Mohammad Dastjerdi. "Ocular manifestations of liver disease: an important diagnostic aid." International Ophthalmology 44, no. 1 (2024). http://dx.doi.org/10.1007/s10792-024-03103-y.

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Abstract Purpose This review examined existing literature to determine various ocular manifestations of liver pathologies, with a focus on metabolic deficiencies as well as viral and immune liver conditions. Methods Recent data were compiled from PubMed from 2000 to 2020 using keywords that were relevant to the assessed pathologies. Ocular presentations of several liver pathologies were researched and then summarized in a comprehensive form. Results Several ocular manifestations of liver disease were related to vitamin A deficiency, as liver disease is associated with impaired vitamin A homeos

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7

Becker, Kolja, Carina M. Weigelt, Holger Fuchs та ін. "Transcriptome analysis of AAV-induced retinopathy models expressing human VEGF, TNF-α, and IL-6 in murine eyes". Scientific Reports 12, № 1 (2022). http://dx.doi.org/10.1038/s41598-022-23065-4.

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AbstractRetinopathies are multifactorial diseases with complex pathologies that eventually lead to vision loss. Animal models facilitate the understanding of the pathophysiology and identification of novel treatment options. However, each animal model reflects only specific disease aspects and understanding of the specific molecular changes in most disease models is limited. Here, we conducted transcriptome analysis of murine ocular tissue transduced with recombinant Adeno-associated viruses (AAVs) expressing either human VEGF-A, TNF-α, or IL-6. VEGF expression led to a distinct regulation of

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