Relevant bibliographies by topics / Ombrello

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters

Academic literature on the topic 'Ombrello'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Ombrello"

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Al-Obeidi, A. F., A. Cavers, Y. Ozguler, O. Manches, H. Zhong, B. Yurttas, B. Ueberheide, G. Hatemi, M. Kugler, and J. Nowatzky. "OP0032 ERAP1-MEDIATED IMMUNOGENICITY AND IMMUNE-PHENOTYPES IN HLA-B51+ BEHÇET’S DISEASE POINT TO PATHOGENIC CD8 T CELL EFFECTOR RESPONSES." Annals of the Rheumatic Diseases 79, Suppl 1 (June 2020): 22.2–23. http://dx.doi.org/10.1136/annrheumdis-2020-eular.3885.

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Background:HLA-B51 is a definite risk factor for Behçet’s disease (BD). A coding variant of ERAP1, Hap10 – with low peptide-trimming activity – vastly potentiates this risk, but is mechanistically unclear1,2).Objectives:To test the hypothesis that low or absent ERAP1 activity alters CD8 T cell immunogenicity through changes in the HLA-B51 peptidome and shapes the CD8 T cell immune response in affected subjects.Methods:We generated HLA-B51+ERAP1 KO LCL clones using CRISPR-Cas9, performed mass spectrometry of the immunoprecipitated MHC-class I peptidome with subsequent computational deconvolution for HLA-B51-binding peptides. We then assessed single cell (ICS), bulk (ELISA) and proliferative (CFSE) CD8 effector (IFNg, granzyme B, perforin) T cell responses through stimulation of allogeneic donor cells with WT vs KO LCL and determined ERAP1 haplotypes in 49 untreated Turkish BD subjects with ocular and/ or major vascular involvement as well as healthy donors (HD) whose PBMC were profiled using 6 multicolour flow cytometry panels.Results:WT and KO peptidomes differed significantly (p<0.0005 Fisher’s exact test) with a distinctive shift of peptide length frequencies exceeding 9-mer (binding optimum) in the KO vs WT. This held true for computationally deconvoluted HLA-B51 binders. IFNg secretion from CD8 T cells stimulated with KO LCL was significantly different from WT (ICS, p=0.0006; ELISA, p=0.0059) as were CD8 T cell proliferation and ICS of perforin/granzyme B+CD8 T cells. Analysis of 133 T, B, NK and monocyte cell populations revealed predominance of CD8 T and NKT cell subset in HLA-B51+/Hap10+ BD vs HLA-B51+/Hap10- BD and HD, accounting for 80% of all populations reaching significance (p<0.05, Mann-Whitney). Naive and effector memory CD8 T cell subsets were inversely correlated. Cohen’s effect sizes were large (>0.8) or very large (>1.2).Conclusion:We show that absence of functional ERAP1 alters human CD8 T cell immunogenicity. This is mediated by an HLA-class I peptidome with propensity for longer peptides above 9mer and suggests loss or de-novo presentation of peptide-HLA-B51 complexes to cognate CD8 TCR. The reciprocal changes in antigen- experienced vs naive CD8 T cell subsets in affected subjects point to biologic significance of HLA-B51/Hap10 in BD. Collectively, our findings suggest that an altered HLA-B51 peptidome modulates immunogenicity of CD8 effector T cells in ERAP1-Hap10 carriers with BD and identify targets for future drug development.References:[1]Kirino, Y., G. Bertsias, Y. Ishigatsubo, N. Mizuki, I. Tugal-Tutkun, E. Seyahi, Y. Ozyazgan, F. S. Sacli, B. Erer, H. Inoko, Z. Emrence, A. Cakar, N. Abaci, D. Ustek, C. Satorius, A. Ueda, M. Takeno, Y. Kim, G. M. Wood, M. J. Ombrello, A. Meguro, A. Gul, E. F. Remmers, and D. L. Kastner. 2013. ‘Genome-wide association analysis identifies new susceptibility loci for Behcet’s disease and epistasis between HLA-B*51 and ERAP1’,Nat Genet, 45: 202-7.[2]Takeuchi, M., M. J. Ombrello, Y. Kirino, B. Erer, I. Tugal-Tutkun, E. Seyahi, Y. Ozyazgan, N. R. Watts, A. Gul, D. L. Kastner, and E. F. Remmers. 2016. ‘A single endoplasmic reticulum aminopeptidase-1 protein allotype is a strong risk factor for Behcet’s disease in HLA-B*51 carriers’,Ann Rheum Dis, 75: 2208-11.Disclosure of Interests:Arshed F. Al-Obeidi: None declared, Ann Cavers: None declared, Yesim Ozguler: None declared, Olivier Manches: None declared, Hua Zhong: None declared, Berna Yurttas: None declared, Beatrix Ueberheide: None declared, Gulen Hatemi Grant/research support from: BMS, Celgene Corporation, Silk Road Therapeutics – grant/research support, Consultant of: Bayer, Eli Lilly – consultant, Speakers bureau: AbbVie, Mustafa Nevzat, Novartis, UCB – speaker, Matthias Kugler: None declared, Johannes Nowatzky: None declared
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Wang, W., T. Zhang, L. Wang, and H. Song. "FRI0472 DIAGNOSIS AND MANAGEMENT OF ADENOSINE DEAMINASE 2 DEFICIENCY CHILDREN: THE EXPERIENCE FROM CHINA." Annals of the Rheumatic Diseases 79, Suppl 1 (June 2020): 833.1–833. http://dx.doi.org/10.1136/annrheumdis-2020-eular.3481.

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Background:Adenosine deaminase 2 deficiency (DADA2) is a rare antoinflammatory disease caused by mutations in ADA2 gene, few Chinese cases have been reported.Objectives:To describe and compare the clinical features, genotypes, and treatments of Chinese DADA2 patients and foreign cases.Methods:Primary immunodeficiency disease Panel or Whole Exome Sequencing was performed to suspected subjects, and assays for adenosine deaminase 2(ADA2) enzyme activity were also carried out to them and their parents. Case reports of Chinese and foreign patients with DADA2 were searched from PubMed and Chinese domestic databases.Results:Seven unrelated DADA2 children from China were included in our study, 5 were identified at Peking union medical college hospital and 2 had been reported previously (1 on PubMed and 1 in Chinese literatures). 14 mutations in ADA2 were identified, and 9 of which have not been found in other countries. Four children receiving enzymatic analysis had lower ADA2 enzyme activity compared to their parents. Phenotypic manifestations included fever, skin symptoms, vasculitis, neurologic involvement, et al. The treatments varying from steroids, immunosuppressants, and tocilizumab, anti-TNF therapy and hematopoietic stem cell transplantation (HSCT) were effective depending on different phenotype and severity.Conclusion:This study includes the biggest number of Chinese DADA2 patients at present. We recommend combination of enzymatic analysis with gene screening to confirm the diagnosis. Genotypes of patients from China were some different, the clinical manifestations were similar. We suggest anti-TNF therapy may not be necessary for mild case and HSCT should be considered even without hematological phenotype.References:[1]Zhou Q, Yang D, Ombrello AK, Zavialov AV, Toro C, Zavialov AV, et al. Early-onset stroke and vasculopathy associated with mutations in ADA2. N Engl J Med. 2014;370:911-920.[2]Meyts I, Aksentijevich I. Deficiency of Adenosine Deaminase 2 (DADA2): Updates on the Phenotype, Genetics, Pathogenesis, and Treatment. J Clin Immunol. 2018;38:569-578.[3]Wang XN, Zhou ZX, Li SN, Lai JM, Su GX, Kang M, et al. A case report of DADA2. Chin J Rheumatol. 2019;23:476-478.[4]Liu L, Wang W, Wang Y, Hou J, Ying W, Hui X, et al. A Chinese DADA2 patient: report of two novel mutations and successful HSCT. Immunogenetics. 2019;71:299-305.Disclosure of Interests:None declared
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Van der Pas, H., M. Thiery, H. Van Kets, and W. Parewijck. "The Ombrelle-380 IUD: a pilot study." Advances in Contraception 3, no. 1 (March 1987): 37–41. http://dx.doi.org/10.1007/bf01849251.

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Piéchaud, J. F., A. Delogu, J. Kachaner, L. Iserin, Y. Aggoun, S. Giusti, D. Bonnet, and D. Sidi. "Fermeture percutanée du canal artériel par la double ombrelle de Rashkind." Archives de Pédiatrie 2, no. 12 (December 1995): 1149–55. http://dx.doi.org/10.1016/0929-693x(96)89915-5.

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Ferrada, M., K. Sikora, Y. Lou, K. Wells, B. Patel, D. Ospina Cardona, E. Rose, et al. "OP0090 CLASSIFICATION OF PATIENTS WITH RELAPSING POLYCHONDRITIS BASED ON SOMATIC MUTATIONS IN UBA1." Annals of the Rheumatic Diseases 80, Suppl 1 (May 19, 2021): 49. http://dx.doi.org/10.1136/annrheumdis-2021-eular.3422.

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Background:Somatic mutations in ubiquitin activating enzyme 1 (UBA1) cause a newly defined syndrome known as VEXAS. [1] More than fifty percent of patients currently identified with VEXAS meet diagnostic criteria for relapsing polychondritis (RP).Objectives:To determine the prevalence VEXAS within a cohort of patients with RP, to compare their clinical, laboratory, and immunologic features and to develop a clinical algorithm to inform genetic screening for VEXAS among patients with RP.Methods:Exome and targeted sequencing of the UBA1 gene was performed in a prospective observational cohort of patients with RP. Clinical and immunological characteristics of patients with RP were compared based on presence or absence of UBA1 mutations. Random forest was used to derive a clinical algorithm to identify patients with UBA1 mutations. Immune populations were quantified by multipanel flow cytometry. Categorical and continuous variables were compared using the chi square or Kruskal-Wallis test. P<0.05 defined statistical significance.Results:Seven of 92 patients with RP (7.6%) were confirmed to have UBA1 mutations (VEXAS-RP). Six additional patients with VEXAS-RP from other cohorts were included for subsequent analyses. Patients with VEXAS-RP were all male, older at disease onset, and commonly had fever, ear chondritis, skin involvement, deep vein thrombosis, and pulmonary infiltrates. Patients with RP as compared with VEXAS-RP had a significantly higher prevalence of airway chondritis, costochondritis and tenosynovitis/arthralgias. (Table). Mortality was significantly greater in VEXAS-RP than RP (27% vs 2% p=0.01). Maximum ESR, CRP, and mean corpuscular volume (MCV) values were significantly greater in VEXAS-RP. Absolute monocyte, lymphocyte, and platelet counts were significantly lower in VEXAS-RP. A decision tree based on male sex, MCV>100 fl and Platelet count<200 K/ul classified between VEXAS-RP and RP with 100% sensitivity and 96% specificity.Table 1.Clinical Characteristics of patients with RP vs VEXAS-RPAll Patientsn=98RPn=85VEXAS-RPn=13p valueDemographic CharacteristicsRace, White n (%)90 (92)77 (91)13 (100)0.59Sex, Male n (%)26 (27)13 (15)13 (100)<0.0001Age, Symptom onset, years, Median (IQR)38 (30-47)37 (28-43)56 (54-64)<0.0001Clinical SymptomsFever n (%)33 (34)20 (24)13 (100)<0.0001Ear chondritis n (%)61 (62)48 (56)13 (100)0.0015Nose chondritis n (%)83 (85)71 (84)12 (92)0.68Airway chondritis n (%)37 (38)37 (44)0 (0)0.0015Tenosynovitis/arthalgias n (%)83 (85)77 (91)6 (46)0.0005Skin involvement n (%)33 (34)22 (2611 (85)<0.0001Laboratory ValuesESR, mm/hr, median (IQR)12 (6-22)11 (5-19)66.5 (42-110)<0.0001CRP, mg/L, median (IQR)2.9 (0.8-9.6)1.9 (0.6-6.3)17.7 (9.6-99.5)<0.0001Platelet count (k/uL)246(201-299)258 (227-312)145 (100-169)<0.0001MCV fL93.05 (90-98)92.2 (89-95)105 (102-115)<0.0001Absolute lymphocyte count1.6 (1.1-2.3)1.78(1.4-2.4)0.92 (0.5-1.2)<0.0001CT scan abnormalitiesPulmonary infiltrates n (%)16 (16.33)6 (7.06)10 (77)<0.0001ComplicationsDeath n (%)6 (6)3 (4)3 (23)0.029Unprovoked DVT12 (12)4 (5)8 (62)<0.0001N number; IQR = interquartile rangeConclusion:Mutations in UBA1 are causal for disease in a subset of patients with RP. These patients are defined by disease onset in the fifth decade of life or later, male sex, ear/nose chondritis and hematologic abnormalities. Early identification is important in VEXAS given the associated high mortality rate.References:[1]Beck DB, Ferrada MA, Sikora KA, Ombrello AK, Collins JC, Pei W, Balanda N, Ross DL, Ospina Cardona D, Wu Z et al: Somatic Mutations in UBA1 and Severe Adult-Onset Autoinflammatory Disease. N Engl J Med 2020, 383(27):2628-2638.Disclosure of Interests:None declared
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Mucumbitsi, J. "Occlusion percutanée du canal artériel persistant par ombrelle de Rashkind: évaluation clinique et financière." Archives de Pédiatrie 2, no. 4 (April 1995): 400–401. http://dx.doi.org/10.1016/0929-693x(95)90210-t.

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Saalman, Howard, Livio Volpi Ghirardini, and Anthony Law. "Recent Excavations under the "Ombrellone" of Sant'Andrea in Mantua: Preliminary Report." Journal of the Society of Architectural Historians 51, no. 4 (December 1, 1992): 357–76. http://dx.doi.org/10.2307/990735.

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Recent excavations have revealed the existence of an integrated complex of vaulted rooms, stairs, and passages under the so-called ombrellone of the western portico of Sant'Andrea in Mantua. The authors suggest that this complex and its adjacent rooms may have been intended to serve for the exposition of the relics of the Most Precious Blood of Christ, preserved in Sant'Andrea, possibly in times of danger and plague, and for the guardian brotherhood of the relics, the Venerable Company of the Most Precious Blood, established in Sant'Andrea by Pius II in 1462. The adjacent rooms over the first side chapels of the nave may have served for "ritual habitation" by members of the ruling Gonzaga family, particularly for the patron, the marchese Ludovico Gonzaga.
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Rey, C., F. Godart, R. Gutierrez, G. Vaksmann, and G. M. Brévière. "Résultats comparatifs entre la double ombrelle de Rashkind et le disque boutonné de Sideris dans l'occlusion du canal artériel." Archives de Pédiatrie 4, no. 6 (June 1997): 591. http://dx.doi.org/10.1016/s0929-693x(97)87601-4.

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Béal, Jean-Claude, Valérie Bel, and Christine Bonnet. "À propos de trois ombrelles antiques en contexte funéraire dans la vallée du Rhône." Revue archéologique de Narbonnaise 48, no. 1 (2015): 181–92. http://dx.doi.org/10.3406/ran.2015.1914.

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Wang, W., S. Yang, Z. Yu, M. Wei, L. Zhong, and H. Song. "AB1067 CASE OR FAMILY?FROM 2 CHINESE FCAS3 CHILDREN WITH PLCG2 MUTATION TO THEIR FAMILIES." Annals of the Rheumatic Diseases 79, Suppl 1 (June 2020): 1822.3–1823. http://dx.doi.org/10.1136/annrheumdis-2020-eular.862.

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Background:Familial cold autoinflammatory syndrome 3 (FCAS3) is an autoinflammatory disease (AID) caused by mutation of the PLCG2 gene, which has not been reported in China. We will report 2 cases of Chinese FCAS3 patients with no claimed family history, but we found the same mutations in a parent during their genetic analysis. After further inquiry of the parent’s medical history, we confirmed that actually, they were two FCAS3 families. Through a literature review, we found that the clinical features of Chinese patients are milder than foreign countries, and their symptoms are concealed and may be ignored, resulting in mistakes in family history collecting.Objectives:To summarize the genetic and clinical features of Chinese FCAS3 patients and to provide diagnostic recommendations for the disease.Methods:Two suspected AID children with recurrent fever and urticaria were enrolled in this study. Clinical data and family history were collected, and genetic analysis was performed by next-generation sequencing (PID panel or WES) and Sanger-based validation. Literature was reviewed from PubMed, CNKI, and Wanfang Database.Results:The two children were both diagnosed to be FCAS3 with PLCG2 mutation. The clinical manifestations of 2 children were recurrent fever, urticaria, and increased ESR and CRP. Case 1 has a paternal, and Case 2 has a maternal heterozygous mutation in the PLCG2 gene, while both had claimed without a family history. Further inquiry showed the two parents used to have a fever with urticaria. By comparing with foreign literature, we found our patients were milder than abroad patients. Large fragment deletions were relatively more common in foreign patients.Conclusion:We reported the case of FCAS3 in China for the first time. Their genotype and phenotype were different from foreign patients. Their symptoms are mild, and heterozygous mutations are more common than foreign patients, which are the main differences. The difference in mutation type may be the reason for different clinical manifestations. Besides, both two families showed a trend of more severe clinical features in the next generation. As the symptoms of the elders were not obvious and may be ignored, it caused trouble for the genetic diagnosis. Therefore, family history should be collected carefully. For rashes and fevers, which are not too severe in overall symptoms, care should be taken about the possibility of AIDs. Genetic testing can help to make a definite diagnosis.Table 1.Descriptive charecteristics of the patients with FMF, n=474VariableCompliant(n=230)Noncompliant (n=244)P valueGender of patient (F)142(61.7)147(60.2)0.73Age, years*35(28-42.5)34(27-44.2)0.88Being Married152(66.1)146(59.8)0.15Disease duration, years*22(14-31)22(15-31)0.71Number of index flare*within last 12-month6.7(1-10)5(3-10)<0.001Family historyof parents54(23.5)39(16.0)0.04Family historyof sibling73(32.9)102(43.4)0.02Comorbid disease presence73(31.7)55(22.5)0.02Treatment<0.001Colchicine230 (94.1)180(78.6)Anakinra&Canakinumab134(5.3)49(21.4)Colchicine response presence127(55.2)126(52.3)0.52Drug using except FMF74(32.2)44(18.0)<0.001Presence of 2 attacks except fever90 (39.1)68(27.9)0.009Chronic peripheral arthritis16(7.0)7(2.9)0.03Amyloidosis18(7.8)9(3.7)0.05Proteinuria23(10.8)8(3.6)0.004Adequate medical care161(70.0)132(54.8)<0.001ISSF severity score*3(2-4)3(2-4)0.02ADDI index*1(0-1)1(0-1)0.05References:[1]Pathak S, Mcdermott M F, Savic S. Autoinflammatory diseases: update on classification diagnosis and management[J]. Journal of Clinical Pathology, 2017, 70(1):1-8.[2]Broderick, L., Hereditary Autoinflammatory Disorders: Recognition and Treatment. Immunol Allergy Clin North Am, 2019. 39(1):13-29.[3]Milner, Joshua D. PLAID: A Syndrome of Complex Patterns of Disease and Unique Phenotypes[J]. Journal of Clinical Immunology, 2015, 35(6):527-530.[4]Picard C, Gaspar H B, Al-Herz W, et al. International Union of Immunological Societies: 2017 Primary Immunodeficiency Diseases Committee Report on Inborn Errors of Immunity[J]. Journal of Clinical Immunology, 2017, 38(Suppl 1):96-128.[5]Ombrello M J, Remmers E F, Sun G, et al. Cold Urticaria, Immunodeficiency, and Autoimmunity Related to PLCG2 Deletions[J]. New England Journal of Medicine, 2012, 366(4):330-8.[6]Zhou Q, Lee GS, Brady J, et al. A Hypermorphic Missense Mutation in PLCG2, Encoding Phospholipase Cγ2, Causes a Dominantly Inherited Autoinflammatory Disease with Immunodeficiency[J]. American Journal of Human Genetics, 2012, 91(4).[7]Neves, J.F., et al., Novel PLCG2 Mutation in a Patient with APLAID and Cutis Laxa. Front Immunol, 2018. 9: 2863.[8]Mcdermott M F, Aksentijevich I, Galon J, et al. Germline mutations in the extracellular domains of the 55 kDa TNF receptor, TNFR1, define a family of dominantly inherited autoinflammatory syndromes[J]. Cell, 1999, 97(1):133-144.Disclosure of Interests:None declared
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Dissertations / Theses on the topic "Ombrello"

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Nalon, Filippo. "Analisi sperimentale sull'utilizzo di etanolo (E85) in un motore per Formula SAE." Master's thesis, Alma Mater Studiorum - Università di Bologna, 2017. http://amslaurea.unibo.it/13146/.

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Questo lavoro di tesi tratta della conversione di un motore a combustione interna ad accensione comandata PFI dall’utilizzo di benzina RON95 all’utilizzo di etanolo E85. L’applicazione dello studio riguarda il propulsore della vettura Formula SAE dell’Università di Bologna. In una fase iniziale dello studio si valutano dal punto di vista teorico gli aspetti inerenti il cambiamento di combustibile: confronto proprietà fisico-chimiche (calore latente di vaporizzazione, tonalità termica, numero di ottano), calcolo delle emissioni di CO2, rapporto di compressione, confronto strozzature da regolamento, stima della capacità del serbatoio. Nella parte centrale si approfondiscono le modalità con cui sono stati effettuati gli interventi in vettura e sul propulsore: incremento del rapporto di compressione, simulazione e progettazione venturi e valvola a farfalla, scelta degli iniettori, strategia di controllo pompa carburante, compatibilità dei materiali. Nella parte conclusiva vengono descritte le modalità di calibrazione del sistema di controllo in sala prova motori: calibrazione base dei rendimenti volumetrici e dell’anticipo di accensione, calibrazione avanzata e generazione curve ad ombrello. Le conclusioni riassumono pro e contro dello studio.
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FOURNIE, OHRESSER MARIE-LAURENCE. "Prevention de l'embolie pulmonaire : developpement et etude clinique de l'ombrelle vascor : a propos de 50 cas." Aix-Marseille 2, 1993. http://www.theses.fr/1993AIX20050.

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CHARLIER, SERGERE LAURENCE. "Fermeture du canal arteriel par catheterisme interventionnel avec une double ombrelle de rashkind : a propos de 25 cas." Clermont-Ferrand 1, 1994. http://www.theses.fr/1994CLF1M011.

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Godart, François. "Fermeture percutanee du canal arteriel par le dispositif de rashkind : etude prospective sur 31 patients." Lille 2, 1992. http://www.theses.fr/1992LIL2M160.

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Msadaa, Nesrine. "L' extensibilité de la marque : définition, déterminants et mesures." Aix-Marseille 3, 2009. http://www.theses.fr/2009AIX32050.

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Bouffard, Patrick. "Occlusion percutanée du canal artériel persistant par la technique de rashkind : étude prospective au sujet de 35 cas." Bordeaux 2, 1993. http://www.theses.fr/1993BOR23062.

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Montali, Margherita. "La rivoluzione degli ombrelli di Hong Kong attraverso la stampa. Introduzione alla storia di Hong Kong e breve analisi terminologica di un corpus in lingua cinese." Bachelor's thesis, Alma Mater Studiorum - Università di Bologna, 2016. http://amslaurea.unibo.it/9807/.

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Questo lavoro nasce dalla mia curiosità di comprendere le vicende che hanno sconvolto Hong Kong da settembre 2014 fino ad oggi con l’obiettivo di cogliere le cause principali che hanno innescato le proteste degli abitanti dell’isola. Il mio elaborato è composto da due capitoli: il primo, riguardante la storia di Hong Kong, ne ripercorre tutte le fasi principali: dalla scoperta dell’isola da parte delle navi inglesi fino alla cosiddetta “Rivoluzione degli Ombrelli”, passando per il 1997, anno in cui Hong Kong ritorna alla Cina; il secondo capitolo è dedicato all’analisi terminologica condotta su un corpus di 177,671 token e 15,832 types creato il 5 Maggio 2015 utilizzando i software BootCat e Antconc. L’analisi terminologica ha come scopo quello di analizzare espressioni e termini riguardanti le proteste di Hong Kong, scoppiate il 28 Settembre 2014, per osservare in che modo il linguaggio politico e i simboli della protesta abbiano condizionato la percezione di questi avvenimenti da parte dell’opinione pubblica cinese ed internazionale. Infine, ho organizzato i termini più significativi in schede terminologiche che ho inserito nell’appendice. Gli obiettivi del mio elaborato sono quelli di presentare le recenti rivolte di Hong Kong che, a mio avviso, non hanno avuto una grande risonanza nel nostro paese, cercando di approfondirne gli aspetti più salienti. Inoltre ho scelto di trattare questo argomento perché analizzare le relazioni tra la Cina e una delle proprie “aree periferiche” permette di osservare la politica, la società e la cultura cinese da un punto di vista affascinante e inusuale.
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Palazuelos, Marina. "Image de marque et extension de marque : le rôle des émotions." Paris 2, 2010. http://www.theses.fr/2010PA020103.

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La littérature sur la marque et sur les émotions a déjà fait l’objet de nombreux articles et travaux de recherche depuis près de trois décennies ; celle qui lie la marque aux émotions est cependant plus récente. Parallèlement, la compréhension des émotions évolue sans cesse notamment grâce aux neurosciences, dont l’apport aux sciences de gestion est discuté dans divers articles. L’objectif de ce travail doctoral est d’étudier la problématique de l’évolution de l’image de marque – à travers la stratégie d’extension de marque – en s’intéressant au rôle des émotions dans ce processus. Une méthodologie qualitative a été mobilisée ; celle-ci s’appuie sur le cas d’une marque, Ladurée, et de son extension de marque dans une catégorie de produits, celle des produits de beauté, opérée à travers deux gammes de produits distinctes (une sous-licence, l’autre sous co-branding).
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Magnoni, Fanny. "Extension verticale de gamme vers le bas : impact sur l'attitude envers la marque et la relation marque-consommateur." Aix-Marseille 3, 2009. http://www.theses.fr/2009AIX32052.

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Duval, Florence. "Attitude de l'enfant-consommateur face aux extensions de marque : influence de la largeur de la marque-mère et de la nature des ses produits originels." Caen, 2008. http://www.theses.fr/2008CAEN0655.

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Les enfants sont, aujourd'hui, des consommateurs à part entière et leur intérêt pour les marques est incontestable. C'est pourquoi, certaines entreprises tentent de séduire ces jeunes consommateurs en utilisant la stratégie d'extension de marque. Toutefois cette stratégie n'est pas sans risque. D'un point de vue académique, très peu de chercheurs se sont intéressés à la problématique des extensions de marque sur le marché de la consommation enfantine. L'objet de cette thèse est donc d'étudier l'attitude des enfants âgés de 8 à 12 ans envers les extensions de marque. Elle examine plus précisément le rôle de trois variables identifiées à l'issue d'une pré-enquête menée auprès de 82 enfants : la largeur de la marque-mère, la nature de ses produits originels et la cohérence perçue par le jeune consommateur entre la marque et le nouveau produit. Pour ce faire, une expérimentation permettant de manipuler la largeur de la marque-mère et la nature de ses produits originels a été réalisée. Les résultats de l'étude quantitative menée auprès de 298 enfants, sur quatre expressions fictives, montrent un un impact direct et positif de la largeur de la marque sur l'attitude du jeune consommateur envers l'extension de marque et révèle le rôle médiateur de la cohérence perçue. En conclusion, cette recherche atteste que le lien logique perçu par l'enfant entre la marque-mère et le nouveau produit contribue au succès de la stratégie d'extension de marque.
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Books on the topic "Ombrello"

1

Il poeta con ombrello rosso. Mantova: Corraini, 2000.

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L' ombrello di Noè: [memorie e conversazioni sul teatro]. Milano: Rizzoli, 2002.

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Camilleri, Andrea. L' ombrello di Noè: [memorie e conversazioni sul teatro]. Milano: Rizzoli, 2002.

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4

L' ombrello di Freshfield: Relazioni di viaggio e storia dell'esplorazione nelle Alpi Apuane, 1865-1905. Pisa: Giardini, 1986.

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Gli ombrelli =: Umbrellas. Milano: BE-MA, 1990.

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Decampe, Jean. L' ombrelle écarlate. [Paris]: Flammarion, 1991.

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L' ombrellino di Amleto. Forlì: Forum, 1986.

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Fabrice, Neaud, and Eudeline Patrick, eds. Mrs Ombrella et les Musées du désert. Paris: Éditions Scali, 2007.

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Les ombrelles de Versailles: Roman. [Paris]: Flammarion, 1999.

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Tisserand, Thierry. Valse des magiciennes, valse des ombrelles. Saint-Romuald, QC: Productions d'Oz, 2003.

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Book chapters on the topic "Ombrello"

1

Audebert, A. J. M. "Clinical Results of a New Contraceptive Intra-uterine Device: Ombrelle 250." In Female Contraception, 336–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73790-9_32.

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"4. Mademoiselle Ombrelle: Shielding the Fair Sex." In Accessories to Modernity, 107–44. University of Pennsylvania Press, 2010. http://dx.doi.org/10.9783/9780812205336.107.

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