Relevant bibliographies by topics / Onchocerca

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Onchocerca'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 February 2022

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Journal articles on the topic "Onchocerca"

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Richard-Lenoble, Dominique. "Onchocerca volvulus." EMC - Biologie Médicale 1, no. 1 (2006): 1–8. http://dx.doi.org/10.1016/s2211-9698(06)76283-5.

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Truant, Allan L., Irma Palazzo, Byungse Suh, and Francis Au. "Onchocerca volvulus." Clinical Microbiology Newsletter 20, no. 4 (1998): 29–30. http://dx.doi.org/10.1016/s0196-4399(01)80048-9.

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Hall, Laurie R., and Eric Pearlman. "Pathogenesis of Onchocercal Keratitis (River Blindness)." Clinical Microbiology Reviews 12, no. 3 (1999): 445–53. http://dx.doi.org/10.1128/cmr.12.3.445.

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SUMMARY Onchocerciasis is a major cause of blindness. Although the World Health Organization has been successful in reducing onchocerciasis as a public health problem in parts of West Africa, there remain an estimated 17 million people infected with Onchocerca volvulus, the parasite that causes this disease. Ocular pathology can be manifested in any part of the eye, although disease manifestations are frequently characterized as either posterior or anterior eye disease. This review focuses on onchocerca-mediated keratitis that results from an inflammatory response in the anterior portion of the eye and summarizes what is currently known about human disease. This review also describes studies with experimental models that have been established to determine the immunological mechanisms underlying interstitial keratitis. The pathogenesis of keratitis is thought to be due to the host inflammatory response to degenerating parasites in the eye; therefore, the primary clinical symptoms of onchocercal keratitis (corneal opacification and neovascularization) are induced after injection of soluble O. volvulus antigens into the corneal stroma. Experimental approaches have demonstrated an essential role for sensitized T helper cells and shown that cytokines can regulate the severity of keratitis by controlling recruitment of inflammatory cells into the cornea. Chemokines are also important in inflammatory cell recruitment to the cornea, and their role in onchocerciasis is being examined. Further understanding of the molecular basis of the development of onchocercal keratitis may lead to novel approaches to immunologically based intervention.
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Guderian, Ronald H., Mariela Anselmi, Philip J. Cooper, and Martha E. Chico. "Macrofilaricidal effects of chloroquine on adult Onchocerca volvulus by local infiltration of palpable onchocercal nodules." Revista da Sociedade Brasileira de Medicina Tropical 30, no. 6 (1997): 469–73. http://dx.doi.org/10.1590/s0037-86821997000600005.

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The macrofilaricidal effects of local infiltration of high concentrations of chloroquine into the capsule of onchocercal nodules on adult worms of Onchocerca volvulus was determined. Six weeks post infiltration, histological examination of single nodules showed all adult worms to be dead. With nodule conglomerates, there was localized action of chloroquine only on the adult worms in the infiltrated nodule, with no diffusion of the drug to adjacent nodules. Chloroquine infiltration of young, recently formed nodules to reduce the adult worm load of infected individuals may be an alternative method to costly nodulectomy.
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Dunn, T. S., P. S. Raines, J. Barrett, and P. E. Butterworth. "Carbohydrate metabolism in Onchocerca gutturosa and Onchocerca lienalis (Nematoda:Filarioidea)." International Journal for Parasitology 18, no. 1 (1988): 21–26. http://dx.doi.org/10.1016/0020-7519(88)90031-8.

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Agatsuma, Takeshi, and Mamoru Ito. "Isozyme Study of Onchocerca volvulus and Onchocerca gutturosa in Guatemala." Journal of Parasitology 71, no. 3 (1985): 370. http://dx.doi.org/10.2307/3282023.

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Shah, Jyotsna S., Willy F. Piessens, Dyann F. Wirth, and Marc Karam. "Characterization of an Onchocerca-Specific DNA Clone from Onchocerca Volvulus." American Journal of Tropical Medicine and Hygiene 37, no. 2 (1987): 376–84. http://dx.doi.org/10.4269/ajtmh.1987.37.376.

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Brydon, L. J., G. W. Gooday, L. H. Chappell, and T. P. King. "Chitin in egg shells of Onchocerca gibsoni and Onchocerca volvulus." Molecular and Biochemical Parasitology 25, no. 3 (1987): 267–72. http://dx.doi.org/10.1016/0166-6851(87)90090-9.

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Carlow, C. K. S., and A. E. Bianco. "Resistance to Onchocerca lienalis microfilariae in mice conferred by egg antigens of homologous and heterologous Onchocerca species." Parasitology 94, no. 3 (1987): 485–96. http://dx.doi.org/10.1017/s0031182000055839.

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Embryonic stages of various Onchocerca species have been used to stimulate resistance in CBA mice to challenge injections with the microfilariae of Onchocerca lienalis. Comparable levels of resistance to challenge (29–37% reductions) were conferred by living, freeze-killed, or sonicated organisms administered with Freunds‘ Complete Adjuvant (FCA). Antigens extracted in saline, or with the detergent sodium deoxycholate, were also protective. Adjuvants enhanced the protective effect, particularly FCA (78% reduction), Freunds‘ Incomplete Adjuvant (74% reduction), aluminium hydroxide (70% reduction) and Bordetella pertussis (70% reduction). Detergent extracts prepared from intact embryos with n-octyl glucoside also stimulated significant levels of protection against microfilarial challenge when given with FCA (37–45% reductions). Levels of resistance induced by immunizations with intact organisms were greatest following subcutaneous (s.c.) injection over the neck or by intramuscular inoculation. Soluble extracts were also particially effective given by s.c. inguinal or intraperitoneal injection. A time-interval of greater than 3 weeks between the completion of immunization and challenge was required for the expression of immunity. Cross-protection against challenge with O. lienalis microfilariae was also afforded to mice by immunization with intact embryos or detergent extracts of Onchocerca gutturosa (45 and 34% reductions), Onchocerca gibsoni (66 and 47% reductions) or Onchocerca volvulus (58 and 41% reductions). It is concluded that the embryonic stages of both human and animal parasites provide a source of cross-protective antigens of value in studies on resistance to Onchocerca microfilariae in experimental hosts.
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Koehsler, Martina, Afschin Soleiman, Horst Aspöck, Herbert Auer, and Julia Walochnik. "Onchocerca jakutensisFilariasis in Humans." Emerging Infectious Diseases 13, no. 11 (2007): 1749–52. http://dx.doi.org/10.3201/eid1311.070017.

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Dissertations / Theses on the topic "Onchocerca"

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Silva, Verônica Marchon da. "Estudo epidemiológico para a avaliação da eliminação da Oncocercose em áreas sentinelas da Região Amazônica, Brasil." reponame:Repositório Institucional da FIOCRUZ, 2015. http://www.arca.fiocruz.br/handle/icict/15057.

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Submitted by Angelo Silva (asilva@icict.fiocruz.br) on 2016-07-13T18:31:00Z No. of bitstreams: 2 license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) 71824.pdf: 2172041 bytes, checksum: 706d176d456c37e0c405bbdd3df5101a (MD5)<br>Approved for entry into archive by Anderson Silva (avargas@icict.fiocruz.br) on 2016-07-21T19:07:35Z (GMT) No. of bitstreams: 2 71824.pdf: 2172041 bytes, checksum: 706d176d456c37e0c405bbdd3df5101a (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5)<br>Made available in DSpace on 2016-07-21T19:07:35Z (GMT). No. of bitstreams: 2 71824.pdf: 2172041 bytes, checksum: 706d176d456c37e0c405bbdd3df5101a (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) Previous issue date: 2015<br>Fundação Oswaldo Cruz. Instituto Oswaldo Cruz. Rio de Janeiro, RJ, Brasil<br>No Brasil, a área endêmica para oncocercose está restrita a Amazônia, em Terra Indígena Yanomami. O Programa Brasileiro de Eliminação da Oncocercose (PBEO) adota como principal estratégia de controle o tratamento em massa dessa população com Ivermectina, estando em consonância com o Programa para Eliminação da Oncocercose nas Américas (OEPA) e os outros cinco países endêmicos na América Latina \2013 México, Guatemala, Equador, Colômbia e Venezuela. De acordo com estratégias de monitoramento definidas pela Organização Mundial da Saúde (OMS), foi realizado esse estudo visando à disponibilização de ferramentas para avaliação do estado epidemiológico da oncocercose baseadas em análises de parâmetros entomológicos, fornecendo subsídios para medidas e as ações efetivas de controle. Foram realizadas, de 2009 a 2011, avaliações entomológicas, moleculares e epidemiológicas nos três polos base sentinelas para o PBEO \2013 Xitei (subpolo Ketaa e Watatase), Balawau (subpolo Maxapapi e Wanapiu) e Toototobi (subpolo Xiroxiropiu) para o monitoramento do impacto das estratégias de controle do PBEO/OEPA na transmissão da oncocercose no Brasil. Para análise dos parâmetros entomológicos foram realizadas capturas sistemáticas mensais de simulídeos, de 4-8 dias consecutivos de 7-18h Para análise molecular, grupos de até 50 fêmeas foram organizados de acordo com a espécie e outros parâmetros, separados em cabeças e corpos e cada grupo foi testado para a presença de DNA do parasito por PCR-ELISA. Do total de 74.397 simulídeos capturados (54% S. guianense; 40% S. incrustatum; 6% S. oyapockense), 16.971 foram coletados em Xitei/Watatase, 22.910 em Xitei/Ketaa, 2.301 em Balawaú/Maxapapi, 10.986 em Balawaú/Wanapiu e 21.229 de Toototobi. Do total de 1559 pools examinados (821 de Xitei, 302 de Balawaú e 436 de Toototobi), a prevalência estimada de S. guianense infectado foi de 1,0/2.000 simulídeos (95% LSIC \2013 2,2) no polo base Toototobi e 0,5/2.000 simulídeos (95% LSIC \2013 1,4) no polo base Balawaú. Simulim incrustatum e S. oyapockense não apresentaram nenhum pool considerado positivo. Neste estudo, o uso de diagnóstico molecular combinado com a identificação e aspectos bionômicos das espécies vetores e análises epidemiológicas possibilitou avaliar as informações sobre o estado da transmissão da oncocercose após a intervenção de 15 anos de tratamento com ivermectina. Com base nos parâmetros entomológicos de prevalência da infectividade (TI) e o potencial de transmissão anual (PTA) detectado abaixo do preconizado para as áreas sentinela foi possível verificar para período estudado a supressão da transmissão da oncocercose nas três áreas sentinela \2013 Xitei, Balawaú e Toototobi<br>Abstract: In Brazil, the onchocerciasis endemic area is restricted to the Amazon region, in the Yanomami area. The Brazilian Program to Eliminate Onchocerciasis (PBEO) adopts as the most important strategy to control the mass treatment of this population with Ivermectina, and so does the Onchocerciasis Elimination Program for the Americas (OEPA), as well as the other five endemic countries in Latin America - Mexico, Guatemala, Equador, Colombia and Venezuela. According to the strategies defined by the World Health Organization (WHO) this study was done aiming the availability of tools for the evaluation of the epidemiological status onchocerciasis based on entomologic parameters, offering subsidies for the study and effective controlling actions. Were conducted, from 2009 to 2011, entomological, molecular and epidemiological evaluations in three sentinels areas under surveillance of PBEO \2013 Xitei (Ketaa and Watatase), Balawau (Maxapapi and Wanapiu) and Toototobi (Xiroxiropiu) to monitor the PBEO/OEPA controlling strategies impact in the onchocerciasis transmission in Brazil. To analyze the entomological parameters, monthly, simulids captures from 4-8 consecutive days, between the hours of 7am\20136pm were done. For the molecular analysis, groups of up to 50 females were organized according to species and other parameters, separated in heads and bodies and each group was tested through PCR-ELISA, for the presence of the DNA parasite From a total of 74,397 captured simulids (54% S. guianense, 40% S. incrustatum; 3% S. oyapockense); 16,971 were captured from Xitei/ Watatase, 22,910 from Xitei/Ketaa, 2,301 from Balawau/Maxapapi, 10,986 from Balawau/Wanapiu and 21,229 from Toototobi. In a total of 1,559 pools examined (821 from Xitei, 302 from Balawau and 436 from Toototobi), the estimated prevalence of infected S. guianense was 1.0/2,000 simulids (95% L SIC\20132.2) in the Toototobi area, and 0.5/2,000, simulids (95% L SIC \2013 1.4) in the Balawau area. Simulium incrustatum and S. oyapockense did not present any pool considered positive. In this study, the use of molecular diagnostic combined with the identification and bionomic aspects of the vector species, and epidemiologic analysis, assisted in the evaluation of the Onchocerciasis transmission after 15 years of Ivermectina treatment, there are presenting an updated epidemiological profile of the disease in the Brazilian Amazon. Based on entomological parameters of prevalence of infectivity (IT) and the potential for transmission (PTA) detected below levels considered the sentinel areas was verified for the period studied the suppression of onchocerciasis transmission in the three sentinel areas\2013 Xitei, Balawaú e Toototobi
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Tree, Timothy Ian Martin. "Molecular characterisation of Onchocerca volvulus antigens." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.321651.

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Krause, Stephanie. "Untersuchungen zur Genstruktur und Genexpression an Glutathion-S-Transferasen der Nematoden Onchocerca volvulus (Leukart 1893) und Caenorhabditis elegans (Maupas 1900)." [S.l. : s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=965211924.

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Eng, Jeffrey K. L. "Genetic selection by ivermectin on Onchocerca volvulus." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111844.

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Onchocerca volvulus is a parasitic filarial nematode responsible for human onchocerciasis, a disease commonly known as "River Blindness". Although there are no well documented cases of ivermectin resistance in O. volvulus, reports of suboptimal responses to ivermectin have appeared. The purpose of this thesis was to examine genetic polymorphisms in O. volvulus and to determine whether there was genetic evidence of ivermectin selection on O. volvulus genes. Analysis of 17 genes from O. volvulus was undertaken in two populations of worms, either from ivermectin-naive patients or from patients who had been repeatedly treated with ivermectin annually. In 14 of the genes no differences in genetic polymorphism were found (although polymorphisms were identified). However, chi square analysis (chi2=0.05) indicated significant differences in allele frequencies for a P-glycoprotein, a beta-tubulin and a putative dyf=8 gene. Analysis of the O. volvulusbeta-tubulin alleles identified three amino acid substitutions in the H3 region with ivermectin selection. Microtubules play a key structural role in the formation of neurons, and in ivermectin-resistant Haemonchus contortus, amphidial neurons show distorted microtubule bundles. Polymerization and depolymerization assays of the recombinant O. volvulus beta-tubulin alleles showed interesting differences between the polymerized tubulin using the two different alleles. It is speculated that similar differences could cause the disorganization of the microtubules identified in the amphidial neurons in ivermectin resistant H. contortus. In addition to the coding mutations, a 24 bp deletion in the adjacent intron to the H3 was detected. A PCR diagnostic assay was developed to genotype individual macro- and microfilariae. Further analyses were conducted to investigate the possibility of a direct relationship between ivermectin and beta-tubulin. Data obtained from equilibrium dialysis experiments indicated that BODIPY FL ivermectin bound to purified O. volvulus alpha- and beta-tubulins. More interesting, non-fluorescent ivermectin and taxol competed with the BODIPY FL ivermectin. The work presented in this thesis provides evidence of genetic selection by ivermectin on O. volvulus and suggests a putative binding site for ivermectin on tubulin. These data provide novel information on ivermectin selection in O. volvulus and on the possible involvement of tubulin in ivermectin resistance.
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Jenkins, Rosalind Elspeth. "Characterization od recombinant antigens of Onchocerca volvulus." Thesis, University of Liverpool, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240476.

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Bourguinat, Catherine. "Effects of ivermectin on Onchocerca volvulus adult worms." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111889.

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Ivermectin (IVM) is the only safe drug for mass-treatment of onchocerciasis. IVM-resistance has been reported in gastrointestinal nematode parasites of animals. A reduction in response to IVM in Onchocerca volvulus could have significant consequences for the onchocerciasis control programs. Over the last few years, studies have reported genetic selection or reduced responses to IVM in some O. volvulus populations. The risk of a recrudescence of the disease was recently reported with the emergence of resistant adult parasite population in Ghana. It is important to understand the effects of IVM on O. volvulus populations to be able to identify genetic markers to follow IVM selection in the field. In this study, O. volvulus samples were derived from a clinical trial in Cameroon, in which patients were sampled before, and following three years (1994-1997) of IVM treatments. There were four treatment groups: 150mug/kg (1xp.a. or 4xp.a.) and 800mug/kg (1xp.a. or 4xp.a.). DNA from macrofilariae was genotyped for beta-tubulin and P-glycoprotein-like protein (PLP) gene, as well as two control genes and other loci. Reproductive organs of female worms were analyzed by microscopy. A correlation was established between the reproductive status of the female worms and beta-tubulin genotype with the beta-tubulin heterozygous female worms being less fertile than the homozygous female worms. This disadvantage in fertility seemed to disappear after repeated exposure with IVM. We have found evidence that repeated IVM treatment selects for specific alleles of beta-tubulin and PLP. We observed that IVM selection pressure was higher in the female worms than in the male worms. Additionally, loss of polymorphism and selection pressure were higher following thirteen three-monthly doses of IVM compared to annual doses of IVM. Moreover, we found evidence of excess of homozygosity in O. volvulus population, that may be caused by non-random mating and/or subdivision population, which may have implication for the control program. PLP and beta-tubulin genes appear to be promising DNA markers for field use to follow IVM selection. In this perspective, alternative control measures could be considered locally in regions where gene selection is apparent, reducing the likelihood that IVM resistance would develop further and spread.
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Halstead, Meredith. "Putative glutamate-gated chloride channels from Onchocerca volvulus." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=29439.

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Onchocerca volvulus, a filarial nematode, is the causative agent of onchocerciasis.<br>O. volvulus is a human parasite with no animal model host and is endemic in the tropics. O. volvulus material is scarce and must be conserved as part of the Onchocerciasis Control Program. A genomic library was constructed to provide a substantial source of renewable genetic material, in place of original parasite DNA.<br>Currently there is only one glutamate-gated chloride channel that has been sequenced from O. volvulus, but this has not yet been characterized. This GluClx partial cDNA sequence isolated by Cully et al., 1997, may be found in GenBank, accession number U59745. Specific primers were designed to amplify this gene from the genomic library. A fragment of this gene was isolated but the primers were non-specific, amplifying genes in addition to GluClx.<br>A motif is a short recognition sequence within a protein that may allow the modification of the protein. The cysteine loop in the N-terminal of all the ligand-gated ion channels is interesting because it contains the neurotransmitter-gated ion channel signature sequence. (Abstract shortened by UMI.)
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Murray, Katherine Alice. "Characterisation of repetitive DNA sequences of Onchocerca species." Thesis, University of Liverpool, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.280851.

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Van, Eldik Annamaria Johanna. "Synthesis of glutathione conjugates as selective inhibitors for parasitic glutathione S transferases." Thesis, De Montfort University, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246521.

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Cabrera, Z. Y. "Characterisation of Onchocerca antigens and their application to diagnosis." Thesis, Brunel University, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233303.

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Books on the topic "Onchocerca"

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Cabrera, Zully Yanira. Characterization of Onchocerca antigens and their application to diagnosis. Brunel University, 1988.

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Demiaszkiewicz, Aleksander W. Badania nad nicieniami tkankowymi z podrodziny Onchocercinae Leiper, 1911 występującymi u dzikich i domowych przeżuwaczy w Polsce. SGGW, 1995.

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Kim, Aehyung. Health and labor productivity: The economic impact of onchocercal skin disease. World Bank, Africa Human Development Department, Onchocerciasis Coordination Unit : [Addis Ababa], 1997.

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Matthews, Philippa C. Nematodes (roundworms). Edited by Philippa C. Matthews. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198737773.003.0014.

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This chapter consists of short notes, diagrams, maps, and tables to summarize human nematode (‘roundworm’) infections, starting with a classification of relevant organisms. The chapter then goes on to cover Ascaris, Trichinella, Enterobius (‘pin worm’), Trichuris (‘whip worm’), Necator and Ancylostoma (‘hook worms’), and Strongyloides (‘thread worm’). The chapter concludes with a section dedicated to filarial infection, including lymphatic filariasis, Loa Loa (‘eye worm’), Onchocerca volvulus (‘river blindness’), and Dracunculus (‘Guinea worm’). For ease of reference, each topic is broken down into sections, including classification, epidemiology, microbiology, pathophysiology, clinical syndromes, diagnosis, treatment and prevention.
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Paintal, Harman S., and Rajinder K. Chitkara. Zoonotic infections with filarial nematodes. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0067.

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Filarial nematodes have been known to cause human disease for many centuries. Lymphatic filariasis is a common disease in the developing part of the world and much has been written about diagnosis and treatment of this scourge. Wuchereria , Brugia and Onchocerca (especially O. volvulus) have a wide pattern of distribution with severe morbidity. Given the years of scientific work in this field, many drugs that work against these parasites are available today and are attempting to control these infections. In this chapter, the focus is on those filarial nematodes that do not have humans as their primary host. Instead, the filarial organisms that usually parasitize other animals and cause human infection due to a variety of factors are discussed. These factors include: 1. Proximity of humans to the primary host, 2. Proximity of humans to the vector, 3. Changing ecology with introduction of different animals (both host and vector) into new environments, 4. Increasing human mobility, 5. Special scenarios concerning humans, including altered immune function (immunosuppressed due to drugs, auto-immune illness, immunosuppressive diseases), There has been a recent interest in this field because newer diagnostic techniques, including polymerase chain reaction (PCR) assays, DNA primers and electron microscopy have become widespread in use. This will eventually enhance our understanding of the pathophysiology of infections with these seemingly rare filarial organisms.Much of the early work in this field was done in a few specialized centers. As information about these parasites (through the worldwide web) and diagnostic techniques are now widely available, it is our hope that more work regarding these nematodes will be carried out in the developing countries where these infections are common. In this chapter, we focus on Dirofi laria, Meningonema, Loaina, Dipetalonema and certain species of Onchocerca and Brugia.
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Kim, Aehyung, Ajay Tandon, and Asrat Hailu. Health and Labor Productivity: The Economic Impact of Onchocercal Skin Disease. The World Bank, 1999. http://dx.doi.org/10.1596/1813-9450-1836.

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Book chapters on the topic "Onchocerca"

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Ringelmann, R., and Beate Heym. "Onchocerca volvulus." In Parasiten des Menschen. Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_68.

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Liesenfeld, Oliver. "Onchocerca volvulus." In Lexikon der Infektionskrankheiten des Menschen. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_779.

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Mehlhorn, Heinz. "Onchocerca lupi." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4114.

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Mehlhorn, Heinz. "Onchocerca Interbreeds." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4787.

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Mehlhorn, Heinz. "Onchocerca Interbreeds." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4787-1.

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Mehlhorn, Heinz. "Onchocerca lupi." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4114-1.

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Woodward, Michelle, and Cherie Pucheu-Haston. "Onchocerca Preparation." In Manual of Clinical Procedures in the Horse. John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118939956.ch72.

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Despommier, Dickson D., Robert W. Gwadz, and Peter J. Hotez. "Onchocerca volvulus (Leuckart 1893)." In Parasitic Diseases. Springer New York, 1995. http://dx.doi.org/10.1007/978-1-4612-2476-1_8.

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Bianco, Albert E., and Peter J. Ham. "Infection of blackflies with Onchocerca." In The Molecular Biology of Insect Disease Vectors. Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-009-1535-0_11.

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Pearlman, Eric, and Illona Gillette-Ferguson. "Onchocerca volvulus, Wolbachia and River Blindness." In Immune Response and the Eye. KARGER, 2007. http://dx.doi.org/10.1159/000099276.

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Conference papers on the topic "Onchocerca"

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Planat-Chrétien, Anne, Michel Berger, Belen Pedrique, and Samuel Wanji. "Diffuse reflectance spectroscopy for Onchocerca Volvulus nodules characterization." In Diffuse Optical Spectroscopy and Imaging, edited by Hamid Dehghani and Heidrun Wabnitz. SPIE, 2019. http://dx.doi.org/10.1117/12.2526862.

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Mauprivez, C., P. Harlay, and F. Gruffaz. "A calcified onchocercal nodule in the infratemporal fossa. An uncommon case." In 63ème Congrès de la SFCO, edited by S. Boisramé, S. Cousty, J. C. Deschaumes, et al. EDP Sciences, 2015. http://dx.doi.org/10.1051/sfco/20156302034.

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Reports on the topic "Onchocerca"

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GlaxoSmithKline Published Kinase Inhibitor Set 2 Onchocerca lienalis Screening Data. EMBL-EBI, 2018. http://dx.doi.org/10.6019/chembl3988181.

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