Relevant bibliographies by topics / Oncogenes

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Oncogenes'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Oncogenes"

1

Ito, Reina E., Chitose Oneyama, and Kazuhiro Aoki. "Oncogenic mutation or overexpression of oncogenic KRAS or BRAF is not sufficient to confer oncogene addiction." PLOS ONE 16, no. 4 (2021): e0249388. http://dx.doi.org/10.1371/journal.pone.0249388.

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Oncogene addiction is a cellular property by which cancer cells become highly dependent on the expression of oncogenes for their survival. Oncogene addiction can be exploited to design molecularly targeted drugs that kill only cancer cells by inhibiting the specific oncogenes. Genes and cell lines exhibiting oncogene addiction, as well as the mechanisms by which cell death is induced when addicted oncogenes are suppressed, have been extensively studied. However, it is still not fully understood how oncogene addiction is acquired in cancer cells. Here, we take a synthetic biology approach to in
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Martín-Lorenzo, Alberto, Inés Gonzalez-Herrero, Guillermo Rodríguez-Hernández, Idoia García-Ramírez, Carolina Vicente-Dueñas, and Isidro Sánchez-García. "Early epigenetic cancer decisions." Biological Chemistry 395, no. 11 (2014): 1315–20. http://dx.doi.org/10.1515/hsz-2014-0185.

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Abstract A cancer dogma states that inactivation of oncogene(s) can cause cancer remission, implying that oncogenes are the Achilles’ heel of cancers. This current model of cancer has kept oncogenes firmly in focus as therapeutic targets and is in agreement with the fact that in human cancers all cancerous cells, with independence of the cellular heterogeneity existing within the tumour, carry the same oncogenic genetic lesions. However, recent studies of the interactions between an oncogene and its target cell have shown that oncogenes contribute to cancer development via developmental reprog
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Der, C. J. "Cellular oncogenes and human carcinogenesis." Clinical Chemistry 33, no. 5 (1987): 641–46. http://dx.doi.org/10.1093/clinchem/33.5.641.

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Abstract Experimental studies over the past decade have identified 30 or so cellular genes as potential oncogenes. The genetic events that lead to cellular oncogene activation may result in the excessive or inappropriate expression of the gene, or the expression of an aberrant gene product. Although the involvement of these putative cellular oncogenes in human oncogenesis has not been proven, the accumulation of considerable experimental evidence strongly implicates some role of these genes in the malignant process. The inactivation of certain genetic loci (suppressor genes) may also contribut
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Vasudevan, D. M. "Oncogenes and oncogenic viruses." Indian Journal of Clinical Biochemistry 11, no. 1 (1996): 3–6. http://dx.doi.org/10.1007/bf02868403.

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Clark, SS, Y. Liang, CK Reedstrom, and SQ Wu. "Nonrandom cytogenetic changes accompany malignant progression in clonal lines abelson virus-infected lymphocytes." Blood 84, no. 12 (1994): 4301–9. http://dx.doi.org/10.1182/blood.v84.12.4301.bloodjournal84124301.

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Initially, lymphoid cells transformed by v-abl or BCR/ABL oncogenes are poorly oncogenic but progress to full transformation over time. Although expression of the oncogene is necessary to initiate and maintain transformation, other molecular mechanisms are thought to be required for full transformation. To determine whether tumor progression in ABL oncogene-transformed lymphoid cells has a genetic basis, we examined whether progression of the malignant phenotype of transformed clones correlates with particular cytogenetic abnormalities. A modified in vitro bone marrow transformation model was
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Karlina, I. S., E. S. Gorozhanina, and I. V. Ulasov. "THE PROSPECT OF USING ONCOGENES’ INHA, DLL4 AND MMP2 ROLE IN DIAGNOSIS AND TREATMENT OF ONCOLOGICAL DISEASE." Russian Journal of Biotherapy 20, no. 1 (2021): 8–15. http://dx.doi.org/10.17650/1726-9784-2021-20-1-8-15.

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A large role in the development of malignant tumors is played by a genetic predisposition. Risk factors for cancer include the presence of mutations in oncogenes‑genes that cause the development of tumors. They were first found in the genome of viruses, and their analogs, called proto‑oncogenes, were found in humans. The study of the work of oncogenes is a promising direction in the development of new methods for the diagnosis and treatment of oncological diseases. The discovery and research of oncogenes of all classes are necessary not only to understand the mechanisms of neoplasm development
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Cooper, H. L., N. Feuerstein, M. Noda, and R. H. Bassin. "Suppression of tropomyosin synthesis, a common biochemical feature of oncogenesis by structurally diverse retroviral oncogenes." Molecular and Cellular Biology 5, no. 5 (1985): 972–83. http://dx.doi.org/10.1128/mcb.5.5.972-983.1985.

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To identify proteins whose production may be altered as a common event in the expression of structurally diverse oncogenes, we compared two-dimensional electropherograms of newly synthesized proteins from NIH/3T3 cell lines transformed by a variety of retroviral oncogenes, from cellular revertant lines, and from a line (433.3) which expresses the v-ras oncogene in response to corticosteroids. Most alterations in the synthesis of specific proteins detected by this approach appeared to be the result of selection during prolonged cultivation and were probably unrelated to the transformation proce
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Cooper, H. L., N. Feuerstein, M. Noda, and R. H. Bassin. "Suppression of tropomyosin synthesis, a common biochemical feature of oncogenesis by structurally diverse retroviral oncogenes." Molecular and Cellular Biology 5, no. 5 (1985): 972–83. http://dx.doi.org/10.1128/mcb.5.5.972.

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To identify proteins whose production may be altered as a common event in the expression of structurally diverse oncogenes, we compared two-dimensional electropherograms of newly synthesized proteins from NIH/3T3 cell lines transformed by a variety of retroviral oncogenes, from cellular revertant lines, and from a line (433.3) which expresses the v-ras oncogene in response to corticosteroids. Most alterations in the synthesis of specific proteins detected by this approach appeared to be the result of selection during prolonged cultivation and were probably unrelated to the transformation proce
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Moore, Patrick S. "KSHV-encoded oncogenes and oncogenesis." Journal of Acquired Immune Deficiency Syndromes & Human Retrovirology 14, no. 4 (1997): A14. http://dx.doi.org/10.1097/00042560-199704010-00033.

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Rey, Federica, Letizia Messa, Cecilia Pandini, et al. "Transcriptome Analysis of Subcutaneous Adipose Tissue from Severely Obese Patients Highlights Deregulation Profiles in Coding and Non-Coding Oncogenes." International Journal of Molecular Sciences 22, no. 4 (2021): 1989. http://dx.doi.org/10.3390/ijms22041989.

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Obesity is a major risk factor for a large number of secondary diseases, including cancer. Specific insights into the role of gender differences and secondary comorbidities, such as type 2 diabetes (T2D) and cancer risk, are yet to be fully identified. The aim of this study is thus to find a correlation between the transcriptional deregulation present in the subcutaneous adipose tissue of obese patients and the oncogenic signature present in multiple cancers, in the presence of T2D, and considering gender differences. The subcutaneous adipose tissue (SAT) of five healthy, normal-weight women,
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Dissertations / Theses on the topic "Oncogenes"

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SCHAFFHAUSER, CHRISTOPHE. "Les oncogenes viraux." Strasbourg 1, 1987. http://www.theses.fr/1987STR10692.

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Pandey, Vijay. "Secreted oncogenes in endometrial carcinoma." Thesis, University of Auckland, 2010. http://hdl.handle.net/2292/8195.

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Endometrial carcinoma is the most common malignancy of the female reproductive tract and the incidence in developed countries is rising. Poor survival of late stage and recurrent endometrial carcinoma patients, particularly with an aggressive histologic subtype, necessitate the development of new therapeutic modalities for advanced stage and recurrent endometrial carcinoma. Recent published data have demonstrated elevated levels of human growth hormone (hGH) in endometriosis and endometrial adenocarcinoma. Herein, I demonstrate that autocrine production of hGH can enhance the in vitro a
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Scully, Jaqueline Susan. "Insertion of oncogenes into mouse mammary epithelium." Thesis, University of Cambridge, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.315287.

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Williams, Alistair Robert William. "Expression of oncogenes in human colorectal neoplasms." Thesis, University of Edinburgh, 1988. http://hdl.handle.net/1842/19415.

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Imler, Jean-Luc. "Identification d'une cible transcriptionnelle pour les oncogenes." Université Louis Pasteur (Strasbourg) (1971-2008), 1988. http://www.theses.fr/1988STR13193.

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Gerlinger, Emmanuel. "Proto-oncogenes et developpement embryonnaire : etude bibliographique." Université Louis Pasteur (Strasbourg) (1971-2008), 1989. http://www.theses.fr/1989STR1M202.

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Jaggi, Rolf. "Interaction of oncogenes with the glucocorticoid receptor /." Bern, 1989. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Kemble, David J. "A biochemical study on the regulation of the SRC and FGFR family of protein tyrosine kinases /." View online ; access limited to URI, 2009. http://0-digitalcommons.uri.edu.helin.uri.edu/dissertations/AAI3367994.

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Jenkins, Brendan John. "Activating point mutations in the common ?gb?s[beta]-subunit of the human GM-CSF, IL-3 and IL-5 receptors : implications for receptor function and role in disease / by Brendan John Jenkins." Title page, contents and summary only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phj518.pdf.

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Bartel, Courtney A. "Novel Roles for FAM83 Oncogenes in Breast Cancer." Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1512682785418426.

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Books on the topic "Oncogenes"

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1.

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Benz, Christopher, and Edison Liu, eds. Oncogenes. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-1599-5.

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Vogt, Peter K., ed. Oncogenes. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74697-0.

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M, Glover David, and Hames B. D, eds. Oncogenes. IRL Press, 1989.

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S, Papas Takis, and Vande Woude George F, eds. Oncogenes. Elsevier, 1986.

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Christopher, Benz, and Liu Edison T, eds. Oncogenes. Kluwer Academic Publishers, 1989.

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Cancer, Research Workshop (8th 1989 Grenoble France). Growth factors and oncogenes =: Facteurs de croissance et oncogènes. J. Libbey Eurotext ; Paris : Editions INSERM, 1989.

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Spandidos, Demetrios, ed. ras Oncogenes. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4757-1235-3.

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NATO Advanced Research Workshop on Ras Oncogenes (1988 Athens, Greece). Ras oncogenes. Plenum Press, 1989.

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W, Alt Frederick, Harlow Edward, Ziff Edward, and Cold Spring Harbor Laboratory, eds. Nuclear oncogenes. Cold Spring Harbor Laboratory, 1987.

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Book chapters on the topic "Oncogenes"

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Introduction." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_1.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "myc and Other Nuclear Oncogenes." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_10.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Additional Oncogenes." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_11.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Transgenic Mice: Direct In Vivo Assay for Oncogenes." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_12.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Potential Diagnostic Uses of Oncogenes." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_13.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Potential Therapeutic Applications of Oncogenes." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_14.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Oncogene Paradigm: Contribution to a Fundamental Understanding of Malignancy." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_15.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Assays: Tools of the New Biology." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_2.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Viruses and Oncogenes." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_3.

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Burck, Kathy B., Edison T. Liu, and James W. Larrick. "Human T Cell Lymphotropic/Leukemia Viruses." In Oncogenes. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3718-1_4.

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Conference papers on the topic "Oncogenes"

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Young, Richard A. "Abstract IA01: Transcriptional and epigenetic control of oncogenes." In Abstracts: AACR Special Conference on Chromatin and Epigenetics in Cancer - June 19-22, 2013; Atlanta, GA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.cec13-ia01.

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Shrestha, Yashaswi, Eric J. Schafer, Barbara Weir, Jesse Boehm, Sapana Thomas, and William C. Hahn. "Abstract A38: Human kinase screen for breast cancer oncogenes." In Abstracts: First AACR International Conference on Frontiers in Basic Cancer Research--Oct 8–11, 2009; Boston MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.fbcr09-a38.

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Yue, Xiao, Lei Han, Fengming Lan та ін. "Abstract 5003: β-catenin regulates multiple oncogenes in glioma". У Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-5003.

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Slotkin, Emily, Elisa de Stanchina, Luca Cartegni, Marc Ladanyi, and Lee Spraggon. "Abstract B26: Therapeutic targeting of sarcomas driven by EWSR1 fusion oncogenes by modulation of the fusion oncogene pre-mRNA." In Abstracts: AACR Special Conference: Advances in Pediatric Cancer Research: From Mechanisms and Models to Treatment and Survivorship; November 9-12, 2015; Fort Lauderdale, Florida. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.pedca15-b26.

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Huang, Bin, Weijia Zhang, Winfried Edelmann, and Yuxun Wang. "Abstract 2219: Identification of oncogenes in mutant Rpa1 associated tumorigenesis." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2219.

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Salcedo, Adriana, John D. Watson, Hilary Racher, et al. "Abstract 3771: Oncogenes and tumour-suppressors drive differential retinoblastoma evolution." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-3771.

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Salcedo, Adriana, John D. Watson, Hilary Racher, et al. "Abstract 3771: Oncogenes and tumour-suppressors drive differential retinoblastoma evolution." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-3771.

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Abraham, Brian J., Nicholas Kwiatkowski, Abraham S. Weintraub, Denes Hnisz, Nancy Hannett, and Richard A. Young. "Abstract PR14: Nucleation of transcriptional super-enhancers at tumor oncogenes." In Abstracts: AACR Special Conference: Translation of the Cancer Genome; February 7-9, 2015; San Francisco, CA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.transcagen-pr14.

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Abraham, Brian J., Nicholas Kwiatkowski, Abraham S. Weintraub, Denes Hnisz, Nancy Hannett, and Richard A. Young. "Abstract PR06: Nucleation of transcriptional super-enhancers at tumor oncogenes." In Abstracts: AACR Special Conference: Computational and Systems Biology of Cancer; February 8-11, 2015; San Francisco, CA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.compsysbio-pr06.

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Guest, Stephen, Ramsi Haddad, Joe Gray, and Stephen Ethier. "Abstract 5125: Functional genomic strategies to identify oncogenes in breast cancer." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5125.

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Reports on the topic "Oncogenes"

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Shrestha, Yashaswi. Identifying Breast Cancer Oncogenes. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada545002.

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Shrestha, Yashaswi. Identifying Breast Cancer Oncogenes. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada555898.

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Fiordalisi, James J., and Channing Der. Novel Oncogenes in Breast Cancer Development. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada403648.

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Shields, Janiel, and Der Channing. Novel Oncogenes in Breast Cancer Development. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada390710.

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Lopez-Diego, Rocio S., and Gregory M. Shackleford. Identification of Oncogenes Cooperating in Murine Mammary Tumorigenesis. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada396673.

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Kao, Jessica Y. Characterizing Candidate Oncogenes at 8q21 in Breast Cancer. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada485724.

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Lopez-Diego, Rocio S., and Gregory M. Shackleford. Identification of Oncogenes Cooperating in Murine Mammary Tumorigenesis. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada409479.

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Kao, Jessica. Characterizing Candidate Oncogenes at 8q21 in Breast Cancer. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada469206.

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Der, Channing J. Cloning of Novel Oncogenes Involved in Human Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada406359.

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Der, Channing J. Cloning of Novel Oncogenes Involved in Human Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada418063.

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