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Relevant bibliographies by topics / Onset of diseases

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Academic literature on the topic 'Onset of diseases'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Onset of diseases"

1

van Hoorn, Bastiaan T., Suzanne C. Wilkens, and David Ring. "Gradual Onset Diseases: Misperception of Disease Onset." Journal of Hand Surgery 42, no. 12 (2017): 971–77. http://dx.doi.org/10.1016/j.jhsa.2017.07.021.

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Pfliegler, György, Erzsébet Kovács, György Kovács, Krisztián Urbán, Valéria Nagy, and Boglárka Brúgós. "Adult-onset rare diseases." Orvosi Hetilap 155, no. 9 (2014): 334–40. http://dx.doi.org/10.1556/oh.2014.29857.

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The present paper is focusing on rare diseases manifesting in late childhood or adulthood. A part of these syndromes are not of genetic origin, such as relatively or absolutely rare infections, autoimmune diseases, tumours, or diseases due to rare environmental toxic agents. In addition, even a large proportion of genetic disorders may develop in adulthood or may have adult forms as well, affecting are almost each medical specialization. Examples are storage disorders (e.g. adult form of Tay-Sachs disease, Gaucher-disease), enzyme deficiencies (e.g. ornithin-transcarbamylase deficiency of the

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Asano, Koichiro, Shigeharu Ueki, Mayumi Tamari, Yoshimasa Imoto, Shigeharu Fujieda, and Masami Taniguchi. "Adult‐onset eosinophilic airway diseases." Allergy 75, no. 12 (2020): 3087–99. http://dx.doi.org/10.1111/all.14620.

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Mandia, Daniele, Natalia Shor, Jean-François Benoist, and Yann Nadjar. "Adolescent-Onset and Adult-Onset Vitamin-Responsive Neurogenetic Diseases." JAMA Neurology 78, no. 4 (2021): 483. http://dx.doi.org/10.1001/jamaneurol.2020.4911.

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Robinson, I., and S. Neilson. "Prenatal diagnosis of late onset diseases." BMJ 306, no. 6884 (1993): 1065. http://dx.doi.org/10.1136/bmj.306.6884.1065-a.

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JANCIN, BRUCE. "Early-, Late-Onset Psoriasis: Distinct Diseases." Rheumatology News 10, no. 8 (2011): 44. https://doi.org/10.1016/s1541-9800(11)70538-5.

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Fernández-Eulate, Gorka, Christophe Carreau, Jean-François Benoist, et al. "Diagnostic approach in adult-onset neurometabolic diseases." Journal of Neurology, Neurosurgery & Psychiatry 93, no. 4 (2022): 413–21. http://dx.doi.org/10.1136/jnnp-2021-328045.

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Neurometabolic diseases are a group of individually rare but numerous and heterogeneous genetic diseases best known to paediatricians. The more recently reported adult forms may present with phenotypes strikingly different from paediatric ones and may mimic other more common neurological disorders in adults. Furthermore, unlike most neurogenetic diseases, many neurometabolic diseases are treatable, with both conservative and more recent innovative therapeutics. However, the phenotypical complexity of this group of diseases and the growing number of specialised biochemical tools account for a s

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Shchigoleva, A. E., P. V. Shumilov, and А. Р. Shumilov. "INFLAMMATORY BOWEL DISEASES WITH VERY EARLY ONSET." Pediatria. Journal named after G.N. Speransky 97, no. 6 (2018): 141–46. http://dx.doi.org/10.24110/0031-403x-2018-97-6-141-146.

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Hersh, Aimee, Emily von Scheven, and Ed Yelin. "Adult outcomes of childhood-onset rheumatic diseases." Nature Reviews Rheumatology 7, no. 5 (2011): 290–95. http://dx.doi.org/10.1038/nrrheum.2011.38.

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Halvorsen, Matt, Slavé Petrovski, Renée Shellhaas, et al. "Mosaic mutations in early-onset genetic diseases." Genetics in Medicine 18, no. 7 (2015): 746–49. http://dx.doi.org/10.1038/gim.2015.155.

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Dissertations / Theses on the topic "Onset of diseases"

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Hennig, Branwen Johanna Wanda. "Genetic polymorphisms and early-onset periodontal diseases." Thesis, University of Newcastle Upon Tyne, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311107.

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Mallbris, Lotus. "Psoriasis : studies of phenotype at onset and of associated cardiovascular morbidity /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-414-7/.

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Upstill-Goddard, Rosanna. "Genetic dissection of early-onset breast cancer and other genetic diseases." Thesis, University of Southampton, 2015. https://eprints.soton.ac.uk/386938/.

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Genetic variation in the genome of an individual plays a key role in susceptibility to many human diseases. Analysis of the genetic variants harboured by individuals presenting with disease phenotypes is crucial for unravelling the genetic landscape of human disease. The methods that are now available for the characterisation of genetic variants, including single nucleotide polymorphism (SNP) microarrays and next generation sequencing, make it possible to explore all genetic variants harboured within an individual with a specific disease phenotype, allowing for tailoring of treatments. This th

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Gaffney, Jessica. "The Benefits of Nutritional Treatments for Very Early Onset Inflammatory Bowel Disease (VEO-IBD) Patients." Scholarship @ Claremont, 2018. http://scholarship.claremont.edu/cmc_theses/1808.

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Inflammatory bowel disease (IBD) is a group of diseases in the gastrointestinal field that is becoming more commonly diagnosed among patients. IBD is usually characterized as a group of chronic diseases affecting the digestive tract that are caused by a multitude of factors including genetic, environmental, mucosal, and immune contributors. One of the subgroups of IBD is very early onset IBD (VEO-IBD), which is diagnosed in children under the age of 6. VEO-IBD is a rare yet unique case of IBD, which reports poor response to conventional adult-onset IBD treatments. Nutrition is an alternative t

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Giunta, Michele. "Exosomal protein deficiencies : how abnormal RNA metabolism results in childhood-onset neurological diseases." Thesis, University of Newcastle upon Tyne, 2017. http://hdl.handle.net/10443/3669.

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RNA metabolism is of critical importance for normal cellular functions and needs to be finely tuned in order to maintain stable conditions within the cell. The exosome complex is the most important RNA processing machinery, responsible for the correct processing of many different types of RNAs and interacting with different co-factors which bind and carry specific subtypes of RNA for degradation to the complex. Mutations in exosome complex subunits (EXOSC3, EXOSC8) were reported to cause severe childhood onset complex neurological disorders presenting with pontocerebellar hypoplasia type 1 (PC

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Rudan, Igor. "Effects of inbreeding on human quantitative traits and complex common diseases of late-onset." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/29345.

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In a historic sample of 2,761 examinees from isolate village populations of the islands of Brac, Hvar and Korcula, Croatia, collected through field work undertaken in 1970’s and 1980’s, individual inbreeding coefficients were computed based on genealogical records. Inbreeding showed strong positive effect on blood pressure and negative on cortical index. The 14 villages were revisited in 2000 to assess the prevalence of learning disability and of common complex diseases of late onset. A cohort study and an ecological study, after appropriate standardization, both showed that inbreeding increas

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Gaddam, Sathvika, Momani Laith Al, Ali Bokhari, and Melania Bochis. "CRITICAL UPPER LIMB ISCHEMIA IN A PATIENT WITH NEW-ONSET ATRIAL FIBRILLATION." Digital Commons @ East Tennessee State University, 2018. https://dc.etsu.edu/asrf/2018/schedule/70.

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Atrial fibrillation is the most common type of serious dysrhythmia, with increasing prevalence in older age groups. Thromboembolism is a serious complication seen with atrial fibrillation and can range from ischemic stroke, mesenteric ischemia to acute limb ischemia. The annual incidence of acute limb ischemia secondary to atrial fibrillation is 0.14%[1]. Here we report a case of critical limb ischemia with brachial artery occlusion due to an embolus in a patient with new onset atrial fibrillation. A 90 year-old female patient presented to the hospital with complaints of shortness of breath on

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Liang, Chengya. "High Saturated Fat Diet Induces Gestational Diabetes, Perinatal Skeletal Malformation and Adult-Onset Chronic Diseases." Diss., Virginia Tech, 2009. http://hdl.handle.net/10919/26700.

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Adult exposure to high fat diet (HFD) has been linked to increased risk of musculoskeletal, cardiovascular, and metabolic diseases; however, the contribution of gestational HFD to elevated oxidative stress (OS), perinatal cardiovascular, skeletal, and metabolic dysfunction as well as long-term effects on adult offspring are incompletely understood. Pathophysiologic mechanisms linking gestational HFD, OS, and insulin resistance to perinatal development and adult-onset chronic diseases are explored in the present study, and maternal antioxidant (quercetin) is offered as a potential preventive d

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Griffith, Abigail Susanne. "Changes in Activity and Milk Components Around the Onset of Peripartum Diseases in Dairy Cows." Thesis, Virginia Tech, 2014. http://hdl.handle.net/10919/50414.

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Activity and milk components were examined around disease onset in the periparturient period. Cows were monitored for daily rest bouts, rest duration, rest time, and step activity from -21 to +30 d relative to calving. Lactose concentration, fat to protein ratio (F:P), and milk yield were monitored. Diseases analyzed were assisted calving, mastitis, subclinical ketosis (SK), and a multiple disease category (MULTI) for animals who experienced more than one disease. Rest bouts decreased (d 0), step activity increased (d 0), and rest duration decreased (d -1) in animals that experienced assisted

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Lynch, Marcus Phillip. "Evaluation of peptide based vaccines and inhibitors to prevent the onset of HTLV-1 associated diseases." Columbus, Ohio : Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1164739126.

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Books on the topic "Onset of diseases"

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Zoeteweij, M. W., and P. S. Harper. Prenatal Testing for Late-Onset Neurogenetic Diseases. Edited by G. Evers-Kiebooms. Taylor & Francis, 2002. http://dx.doi.org/10.4324/9780203450291.

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G, Evers-Kiebooms, Zoeteweij M. W, and Harper Peter S, eds. Prenatal testing for late-onset neurogenetic diseases. BIOS Scientific, distributed exclusively in the United States and Canada by Springer-Verlag New York, 2002.

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1927-, Butler Robert N., and Brody Jacob A. 1931-, eds. Delaying the onset of late-life dysfunction. Springer Pub. Co., 1995.

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Smith, David G. Understanding M.E.: The phenomenon of myalgic encephalomyelitis and acute onset post viral fatigue syndrome. Robinson Publishing, 1989.

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Smith, David G. Understanding M.E.: The phenomenon of myalgic encephalomyelitis and acute onset post viral fatigue syndrome. Robinson Publishing, 1991.

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Melissa, Pearrow, and Jimerson Shane R, eds. Identifying, assessing, and treating early onset schizophrenia at school. Springer, 2010.

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Parker, James N., and Philip M. Parker. Infantile-onset ascending hereditary spastic paralysis: A bibliography and dictionary for physicians, patients, and genome researchers [to internet references]. ICON Health Publications, 2007.

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Wang, Lemin. The Origin and Onset of Thrombus Disease. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-7344-1.

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Raymond, Levy, ed. Clinical diversity in late onset Alzheimer's disease. Oxford University Press, 1992.

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Durno, Carol Anne. Mode of inheritance and demographics of pediatric-onset inflammatory bowel disease. National Library of Canada, 1999.

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Book chapters on the topic "Onset of diseases"

1

Esdaile, John M. "Adult-Onset Still’s Disease." In Primer on the Rheumatic Diseases. Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-68566-3_23.

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Ghani, Mahdi, Christiane Reitz, Peter St George-Hyslop, and Ekaterina Rogaeva. "Genetic Complexity of Early-Onset Alzheimer’s Disease." In Neurodegenerative Diseases. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-72938-1_3.

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Shimoda, Shinji. "The Onset Mechanism of Primary Biliary Cirrhosis." In Autoimmune Liver Diseases. Springer Japan, 2014. http://dx.doi.org/10.1007/978-4-431-54789-1_12.

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Efthimiou, Petros V., Manil Kukar, and Olga Petryna. "Complex Situations in Patients with Adult-Onset Still’s Disease." In Autoimmune Diseases. Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-358-9_14.

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Schumacher, Axel, Syed Bihaqi, and Nasser H. Zawia. "Epigenetics and Late-Onset Alzheimer’s Disease." In Epigenetic Aspects of Chronic Diseases. Springer London, 2011. http://dx.doi.org/10.1007/978-1-84882-644-1_12.

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Menard, Sandrine, Robert Gniadecki, and Mohammed Osman. "Skin Manifestations of Adult-Onset Immunodeficiencies." In Diseases of Blood and Skin. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-65292-9_16.

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Jayasinghe, Ruwan, and Wanninayake M. Tilakaratne. "Multiple White Lesions of Recent Onset: Candidosis." In Clinicopathological Correlation of Oral Diseases. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-24408-7_36.

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Samardzija, Marijana, Klaudija Masarini, Marius Ueffing, and Dragana Trifunović. "HDAC Inhibition Prevents Primary Cone Degeneration Even After the Onset of Degeneration." In Retinal Degenerative Diseases. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-27378-1_63.

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Zeitler, Philip. "18. Obese Adolescent with New-Onset Diabetes." In A Clinical Approach to Endocrine & Metabolic Diseases. The Endocrine Society, 2012. http://dx.doi.org/10.1210/caem2.9781936704613.ch18.

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Hanson, Robert L., and William C. Knowler. "Type 2 diabetes mellitus and maturity-onset diabetes of the young." In Analysis of Multifactorial Diseases. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579960-7.

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Conference papers on the topic "Onset of diseases"

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S, Prabagar, Tina Babu, and Rekha R. Nair. "Predicting the Onset of Lifestyle Diseases Using Machine Learning Techniques." In 2024 4th International Conference on Ubiquitous Computing and Intelligent Information Systems (ICUIS). IEEE, 2024. https://doi.org/10.1109/icuis64676.2024.10866310.

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Faria, Gustavo Hugo de Souza. "The impact of epigenetics on the development of neurodegenerative diseases." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.654.

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Introduction: Neurodegenerative diseases affect thousands of people in Brazil and have been increasing in frequency with the aging population. However, little is known about the molecular mechanisms and biomarkers of these diseases, which leads to a medical approach based on symptomatic and unresolving characteristics. Epigenetics, including DNA methylation, histone modifications, and changes in regulatory RNAs, emerges as a tool for prevention of neurodegenerative diseases. Objectives: To review studies that discuss the role of epigenetics in the development of neurodegenerative diseases. Met

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Collar, Giovanna Carello, Marco Antônio De Bastiani, and Eduardo R. Zimmer. "HUNTINGTON’S DISEASE AND EARLYONSET ALZHEIMER’S DISEASE SHARE A TRANSCRIPTOMIC SIGNATURE." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda082.

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Background: Neurodegenerative diseases share progressive loss of neurons and protein misfolding, which ultimately culminates in dementia; many diseases have been identified as causes of early-onset dementia (< 65 years of age) such as Huntington’s disease (HD) and early-onset Alzheimer’s disease (EOAD). Importantly, disease-specific genetic mutations have already been identified for HD and EOAD. Thus, one could suggest that the molecular link between these diseases may arise from alterations at the transcriptomic level, which is yet to be determined. Objective: We aimed at identifying trans

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Montilla, CA, E. Cuevas, J. Abraham-Simón, et al. "AB0030 Biopsy findings in adult-onset still disease." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.935.

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Rensink, Max, Ineke Bolt, and Maartje Schermer. "G006 Predicting age of onset and progression of disease in late-onset neurodegenerative genetic diseases: an ethics review and research agenda." In EHDN and Enroll-HD 2024 abstracts. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/jnnp-2024-ehdn.228.

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Hardy, John. "Genetic risk for late onset neurodegenerative diseases largely reflects failure of protein clearance pathways." In Rijeka Forum on Neurodegenerative Diseases (2 ; 2018 ; Rijeka). Hrvatska akademija znanosti i umjetnosti, 2019. http://dx.doi.org/10.21857/mnlqgc07ny.

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Santos, Gustavo de Carvalho, Pedro Delghingaro Forti, Matheus Azolin Camargo, et al. "ADULT-ONSET STILL S DISEASE AND KIKUCHI-FUJIMOTO: AN UNCOMMON ASSOCIATION OF AUTOINFLAMMATORY DISEASES." In XLI Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2024. https://doi.org/10.47660/cbr.2024.2163.

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Kerimovic-Morina, DK, and D. Jablanovic. "SAT0028 Late onset ankylosing spondylitis-clinical and radiological manifestations." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.380.

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Satybaldyev, AM, TF Akimova, and MM Ivanova. "AB0037 Complicated ra course with onset in elderly age." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.1002.

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Lopez-Montilla, MD, A. Escudero, M. Romero, et al. "SAT0042 Clinical features of psoriatic arthritis with a delayed onset." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.394.

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Reports on the topic "Onset of diseases"

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Chen, Cheng-Che, and Chung-Jen Chen. New-Onset Inflammatory Arthritis After Covid-19 Vaccination. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.7.0128.

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Review question / Objective: Investigate the new-onset inflammatory arthritis after Covid-19 vaccination in patients without pre-existing autoimmune nor rheumatic diseases and analyze their clinical patterns. Condition being studied: To help the readers to understand the clinical patterns of new-onset inflammatory arthritis after Covid-19 vaccination in patients without pre-existing autoimmune nor rheumatic diseases. Eligibility criteria: Inclusion criteria: publications of new-onset inflammatory arthritis after Covid-19 vaccination in patients without pre-existing autoimmune nor rheumatic dis

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Dubief, Jessie, Edith Sky Gross, and Fatoumata Faye. Voices on newborn screening: the opinion of people living with a rare disease. EURORDIS - Rare Diseases Europe, 2024. http://dx.doi.org/10.70790/nlmc2114.

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This report presents the European results of the survey ‘Voices on newborn screening: the opinion of people living with a rare disease’, conducted by Rare Barometer within the framework of the European Screen4Care research project. This study gathered the views of more than 6,179 people living with a rare disease and family members worldwide, 5,569 of whom were living in Europe with more than 1,300 distinct rare diseases, hence representing the diversity of the rare disease community. Respondents’ answers confirm the strong support for newborn screening from the rare disease community. They al

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Wongpiyabovorn, Jongkonnee, Nattiya Hirankarn, Yingyos Avihingsanon, Tewin Tencomnao, Yong Poovorawan, and Kriangsak Ruchusatsawat. The association between immunogenetics and genetic susceptibility of psoriasis in Thai population. Chulalongkorn University, 2006. https://doi.org/10.58837/chula.res.2006.27.

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Psoriasis is T-cell-mediated skin autoimmunity, required environmental triggers and genetic susceptibility factors to become manifested. Psoriasis is a chronic skin disease characterized by the abnormal hyperproliferation and differentiation of the epidermis, elongated and prominent blood vessels and a thick perivascular lymphocytic infiltrate. Vascular endothelial growth factor (VEGF) gene play important role in pathogenesis of various diseases with angiogenic basis such as breast cancer and autoimmune disease including psoriasis. Many studies analyzed the association of VEGF gene polymorphis

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Wang, Xiao, Hong Shen, Yujie Liang, Yixin Wang, Meiqi Zhang, and Hongtao Ma. Effects of physical activity interventions for post-COVID-19 patients: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0036.

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Review question / Objective: Coronavirus disease 2019 (COVID-19) is a novel infectious disease caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), which has caused a huge impact in many countries and has attracted great attention from countries around the world. However, since the outbreak of the COVID-19 pandemic, most attention has focused on containing transmission and addressing the surge of critically ill patients in acute care settings. As we enter the second phase of the pandemic, emphasis must evolve to post care of COVID-19 survivors. A variety of persistent sympto

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LI, jianhong, Zhuang LI, Yalin SHE, and Guohua LIN. Assessment of acupuncture for treating herpes zoster:a protocol for an umbrella systematic review and meta analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.4.0010.

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Review question / Objective: Patients who suffer from HZ in line with the consensus of Chinese experts will be included, regardless of sex, race and time of onset . Those who diagnosed with PHN, auto-immune diseases, pregnant women will be excluded.Acupuncture, electroacupuncture, fire needle, skin acupuncture, plum blossom needle, auriculo-acupuncture all these such therapies in treating herpes zoster will be included.The control group’s treatment includes drug therapy (such as antiviral acyclovir nutritional nerve medicine or traditional Chinese medicine, etc.) ，sham acupuncture, placebo, no

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Schat, Karel Antoni, Irit Davidson, and Dan Heller. Chicken infectious anemia virus: immunosuppression, transmission and impact on other diseases. United States Department of Agriculture, 2008. http://dx.doi.org/10.32747/2008.7695591.bard.

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1. Original Objectives. The original broad objectives of the grant were to determine A) the impact of CAV on the generation of cytotoxic T lymphocytes (CTL) to reticuloendotheliosis virus (REV) (CU), B). the interactions between chicken anemia virus (CAV) and Marek’s disease virus (MDV) with an emphasis on horizontal spread of CAV through feathers (KVI), and C) the impact of CAV infection on Salmonella typhimurium (STM) (HUJI). During the third year and the one year no cost extension the CU group included some work on the development of an antigen-antibody complex vaccine for CAV, which was pa

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Sessa, Guido, and Gregory Martin. Role of GRAS Transcription Factors in Tomato Disease Resistance and Basal Defense. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7696520.bard.

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The research problem: Bacterial spot and bacterial speck diseases of tomato are causedby strains of Xanthomonas campestris pv. vesicatoria (Xcv) and Pseudomonas syringae pv.tomato (Pst), respectively. These bacteria colonize aerial parts of the plant and causesignificant losses in tomato production worldwide. Protection against Xcv and Pst bycultural practices or chemical control has been unsuccessful and there are only limitedsources of genetic resistance to these pathogens. In previous research supported in part byBARD IS-3237-01, we extensively characterized changes in tomato gene expressio

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Pericak-Vance, Margaret A. Whole Exome Analysis of Early Onset Alzheimer's Disease. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada602412.

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Pericak-Vance, Margaret A. Whole Exome Analysis of Early Onset Alzheimer's Disease. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada603027.

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Sonia Vallabh & Eric Minikel, Sonia Vallabh &. Eric Minikel. Can anle138b delay the onset of genetic prion disease? Experiment, 2013. http://dx.doi.org/10.18258/0558.

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